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- ESTARÁS HERMOSEL
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- MATÍAS ESTARÁS HERMOSEL
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- Melatonin controls cell proliferation and modulates mitochondrial physiology in pancreatic stellate cells
- Melatonin induces apoptosis and modulates cyclin expression and mapk phosphorylation in pancreatic stellate cells subjected to hypoxia
- Melatonin induces reactive oxygen species generation and changes in glutathione levels and reduces viability in human pancreatic stellate cells
- Melatonin modulates proliferation of pancreatic stellate cells through caspase-3 activation and changes in cyclin A and D expression
- Melatonin modulates red-ox state and decreases viability of rat pancreatic stellate cells
- Melatonin modulates the antioxidant defenses and the expression of proinflammatory mediators in pancreatic stellate cells subjected to hypoxia
- Melatonin modulates metabolic adaptation of pancreatic stellate cells subjected to hypoxia
- Membrane Lipid Derivatives: Roles of Arachidonic Acid and Its Metabolites in Pancreatic Physiology and Pathophysiology
- Modulation of cell physiology under hypoxia in pancreatic cancer
- Pancreatic stellate cells exhibit adaptation to oxidative stress evoked by hypoxia
- Targeting NUPR1-dependent stress granules formation to induce synthetic lethality in Kras<sup>G12D</sup>-driven tumors
- The lysine derivative aminoadipic acid, a biomarker of protein oxidation and diabetes-risk, induces production of reactive oxygen species and impairs trypsin secretion in mouse pancreatic acinar cells
- The melatonin receptor antagonist luzindole induces Ca<sup>2+</sup> mobilization, reactive oxygen species generation and impairs trypsin secretion in mouse pancreatic acinar cells
- The melatonin receptor antagonist luzindole induces the activation of cellular stress responses and decreases viability of rat pancreatic stellate cells
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