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<https://opendata.unex.es/recurso/ciencia-tecnologia/investigacion/publicaciones/Publicacion/2018-2828> ou:bibtex "@article{44276430cb4a4eec88c02ffe52663a14, title = 'Disruption of ER-mitochondria signalling in fronto-temporal dementia and related amyotrophic lateral sclerosis', abstract = 'Fronto-temporal dementia (FTD) and amyotrophic lateral sclerosis (ALS) are two related and incurable neurodegenerative diseases. Features of these diseases include pathological protein inclusions in affected neurons with TAR DNA-binding protein 43 (TDP-43), dipeptide repeat proteins derived from the C9ORF72 gene, and fused in sarcoma (FUS) representing major constituent proteins in these inclusions. Mutations in C9ORF72 and the genes encoding TDP-43 and FUS cause familial forms of FTD/ALS which provides evidence to link the pathology and genetics of these diseases. A large number of seemingly disparate physiological functions are damaged in FTD/ALS. However, many of these damaged functions are regulated by signalling between the endoplasmic reticulum and mitochondria, and this has stimulated investigations into the role of endoplasmic reticulum-mitochondria signalling in FTD/ALS disease processes. Here, we review progress on this topic.', author = 'Lau, {Dawn H.W.} and Naomi Hartopp and Welsh, {Natalie J.} and Sarah Mueller and Glennon, {Elizabeth B.} and M{\\'o}rotz, {G{\\'a}bor M.} and Ambra Annibali and Patricia Gomez-Suaga and Radu Stoica and Sebastien Paillusson and Miller, {Christopher C.J.}', year = '2018', month = mar, day = '1', doi = '10.1038/s41419-017-0022-7', language = 'English', volume = '9', journal = 'Cell Death and Disease', issn = '2041-4889', publisher = 'Nature Publishing Group', number = '3', }";
    bibo:doi "10.1038/s41419-017-0022-7";
    ou:eid "2-s2.0-85043278650";
    dcterms:contributor "Lau D.H.W. et al.";
    bibo:volume "9";
    dcterms:title "Disruption of ER-mitochondria signalling in fronto-temporal dementia and related amyotrophic lateral sclerosis";
    bibo:eissn "2041-4889";
    vivo:identifier "2018-2828";
    ou:openaccess "1"^^xsd:boolean;
    vcard:url <https://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=85043278650&origin=inward>;
    dcterms:creator "Lau D.H.W.";
    fabio:hasPublicationYear "2018";
    dcterms:publisher "Cell Death and Disease";
    ou:urlScopus <https://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=85043278650&origin=inward>;
    ou:urlOrcid <https://kclpure.kcl.ac.uk/portal/en/publications/44276430-cb4a-4eec-88c0-2ffe52663a14>;
    dcterms:created "2018-03-01T00:00:00"^^xsd:dateTime;
    ou:vecesCitado "60"^^xsd:integer;
    a ou:Publicacion;
    ou:tipoPublicacion "Review";
    ou:publicadaEnRevista <https://opendata.unex.es/recurso/ciencia-tecnologia/investigacion/revistas/Revista/2018-4851>, <https://opendata.unex.es/recurso/ciencia-tecnologia/investigacion/revistas/Revista/2018-4851>.
<https://opendata.unex.es/recurso/educacion/universidad/personal/03301885b8214f1b43752b6676c63279-patricia-maria-gomez-suaga> ou:tienePublicacion <https://opendata.unex.es/recurso/ciencia-tecnologia/investigacion/publicaciones/Publicacion/2018-2828>.