PUBLICACIÓN

Electrophysiological approaches to the study of protein translocation in mitochondria

ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Grigoriev, S.M., Muro, C., Dejean, L.M., Campo, M.L., Martinez-Caballero, S., Kinnally, K.W.

2004 International Review of Cytology

CITAS

27

DOI

10.1016/s0074-7696(04)38005-8

EID

2-s2.0-4444308500

ISSN

0074-7696

BIBTEX

@incollection{Grigoriev_2004, doi = {10.1016/s0074-7696(04)38005-8}, url = {https://doi.org/10.1016%2Fs0074-7696%2804%2938005-8}, year = 2004, publisher = {Elsevier}, pages = {227--274}, author = {Sergey M. Grigoriev and Concepci{\'{o}}n Muro and Laurent M. Dejean and Maria Luisa Campo and Sonia Martinez-Caballero and Kathleen W. Kinnally}, title = {Electrophysiological Approaches to the Study of Protein Translocation in Mitochondria}, booktitle = {International Review of Cytology}}

AUTORES DE LA UEX

María Sonia Martínez Caballero
María Luisa Campo Guinea

OTRAS PUBLICACIONES DE INTERÉS

The Influence Of Fasting And Cortisol Upon Arginosuccinate Synthetase, Arginosuccinase And Arginase Levels In Guinea Pig Liver
Analysis Of Mitochondrial Translocases Tom And Tim By The Patch-Clamping Technique
The Castagnoli–cushman Reaction
Analysis Of Mitochondrial Translocases Tom And Tim By The Patch-Clamping Technique
The Castagnoli–cushman Reaction
A Safe And Green Benzylic Radical Bromination Experiment
The Castagnoli–cushman Reaction
A Safe And Green Benzylic Radical Bromination Experiment
Aprendizaje Basado En Juegos De Rol: Una Aplicación A La Economía Del Cambio Climático
A Safe And Green Benzylic Radical Bromination Experiment
Aprendizaje Basado En Juegos De Rol: Una Aplicación A La Economía Del Cambio Climático
Dual Role Of Mitochondrial Porin In Metabolite Transport Across The Outer Membrane And Protein Transfer To The Inner Membrane
Aprendizaje Basado En Juegos De Rol: Una Aplicación A La Economía Del Cambio Climático
Dual Role Of Mitochondrial Porin In Metabolite Transport Across The Outer Membrane And Protein Transfer To The Inner Membrane
Revisiting Trends On Mitochondrial Mega-Channels For The Import Of Proteins And Nucleic Acids
Dual Role Of Mitochondrial Porin In Metabolite Transport Across The Outer Membrane And Protein Transfer To The Inner Membrane
Revisiting Trends On Mitochondrial Mega-Channels For The Import Of Proteins And Nucleic Acids
A Disulfide Bond In The Tim23 Complex Is Crucial For Voltage Gating And Mitochondrial Protein Import
Revisiting Trends On Mitochondrial Mega-Channels For The Import Of Proteins And Nucleic Acids
A Disulfide Bond In The Tim23 Complex Is Crucial For Voltage Gating And Mitochondrial Protein Import
Autophagy-Related Proteins Are Functionally Active In Human Spermatozoa And May Be Involved In The Regulation Of Cell Survival And Motility
A Disulfide Bond In The Tim23 Complex Is Crucial For Voltage Gating And Mitochondrial Protein Import
Autophagy-Related Proteins Are Functionally Active In Human Spermatozoa And May Be Involved In The Regulation Of Cell Survival And Motility
Bax And Bak Function As The Outer Membrane Component Of The Mitochondrial Permeability Pore In Regulating Necrotic Cell Death In Mice
Autophagy-Related Proteins Are Functionally Active In Human Spermatozoa And May Be Involved In The Regulation Of Cell Survival And Motility
Bax And Bak Function As The Outer Membrane Component Of The Mitochondrial Permeability Pore In Regulating Necrotic Cell Death In Mice
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Bax And Bak Function As The Outer Membrane Component Of The Mitochondrial Permeability Pore In Regulating Necrotic Cell Death In Mice
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Mac And Bcl-2 Family Proteins Conspire In A Deadly Plot
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Mac And Bcl-2 Family Proteins Conspire In A Deadly Plot
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Mac And Bcl-2 Family Proteins Conspire In A Deadly Plot
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Assembly Of The Mitochondrial Apoptosis-Induced Channel, Mac
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Assembly Of The Mitochondrial Apoptosis-Induced Channel, Mac
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Assembly Of The Mitochondrial Apoptosis-Induced Channel, Mac
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Arginase I Induction By Modified Lipoproteins In Macrophages: A Peroxisome Proliferator-Activated Receptor-Γ/δ-Mediated Effect That Links Lipid Metabolism And Immunity
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Arginase I Induction By Modified Lipoproteins In Macrophages: A Peroxisome Proliferator-Activated Receptor-Γ/δ-Mediated Effect That Links Lipid Metabolism And Immunity
Apoptosis
Arginase I Induction By Modified Lipoproteins In Macrophages: A Peroxisome Proliferator-Activated Receptor-Γ/δ-Mediated Effect That Links Lipid Metabolism And Immunity
Apoptosis
Awaking Tim22, A Dynamic Ligand-Gated Channel For Protein Insertion In The Mitochondrial Inner Membrane
Apoptosis
Awaking Tim22, A Dynamic Ligand-Gated Channel For Protein Insertion In The Mitochondrial Inner Membrane
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Awaking Tim22, A Dynamic Ligand-Gated Channel For Protein Insertion In The Mitochondrial Inner Membrane
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Erratum: The Nuclear Receptor Interacting Factor-3 Transcriptional Coregulator Mediates Rapid Apoptosis In Breast Cancer Cells Through Direct And Bystander-Mediated Events (Cancer Research (2007) 67, (1775-1782))
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Erratum: The Nuclear Receptor Interacting Factor-3 Transcriptional Coregulator Mediates Rapid Apoptosis In Breast Cancer Cells Through Direct And Bystander-Mediated Events (Cancer Research (2007) 67, (1775-1782))
A Fluorescence Assay For Peptide Translocation Into Mitochondria
Erratum: The Nuclear Receptor Interacting Factor-3 Transcriptional Coregulator Mediates Rapid Apoptosis In Breast Cancer Cells Through Direct And Bystander-Mediated Events (Cancer Research (2007) 67, (1775-1782))
A Fluorescence Assay For Peptide Translocation Into Mitochondria
The Nuclear Receptor Interacting Factor-3 Transcriptional Coregulator Mediates Rapid Apoptosis In Breast Cancer Cells Through Direct And Bystander-Mediated Events
A Fluorescence Assay For Peptide Translocation Into Mitochondria
The Nuclear Receptor Interacting Factor-3 Transcriptional Coregulator Mediates Rapid Apoptosis In Breast Cancer Cells Through Direct And Bystander-Mediated Events
Tim17p Regulates The Twin Pore Structure And Voltage Gating Of The Mitochondrial Protein Import Complex Tim23
The Nuclear Receptor Interacting Factor-3 Transcriptional Coregulator Mediates Rapid Apoptosis In Breast Cancer Cells Through Direct And Bystander-Mediated Events
Tim17p Regulates The Twin Pore Structure And Voltage Gating Of The Mitochondrial Protein Import Complex Tim23
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Tim17p Regulates The Twin Pore Structure And Voltage Gating Of The Mitochondrial Protein Import Complex Tim23
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Is Mac The Knife That Cuts Cytochrome C From Mitochondria During Apoptosis?
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Is Mac The Knife That Cuts Cytochrome C From Mitochondria During Apoptosis?
Regulation Of The Mitochondrial Apoptosis-Induced Channel, Mac, By Bcl-2 Family Proteins
Is Mac The Knife That Cuts Cytochrome C From Mitochondria During Apoptosis?
Regulation Of The Mitochondrial Apoptosis-Induced Channel, Mac, By Bcl-2 Family Proteins
Detection Of The Mitochondrial Apoptosis-Induced Channel (Mac) And Its Regulation By Bcl-2 Family Proteins.
Regulation Of The Mitochondrial Apoptosis-Induced Channel, Mac, By Bcl-2 Family Proteins
Detection Of The Mitochondrial Apoptosis-Induced Channel (Mac) And Its Regulation By Bcl-2 Family Proteins.
Arginase I Induction During Leishmania Major Infection Mediates The Development Of Disease
Detection Of The Mitochondrial Apoptosis-Induced Channel (Mac) And Its Regulation By Bcl-2 Family Proteins.
Arginase I Induction During Leishmania Major Infection Mediates The Development Of Disease
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase I Induction During Leishmania Major Infection Mediates The Development Of Disease
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
The Role Of The Mitochondrial Apoptosis Induced Channel Mac In Cytochrome C Release
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
The Role Of The Mitochondrial Apoptosis Induced Channel Mac In Cytochrome C Release
Oligomeric Bax Is A Component Of The Putative Cytochrome C Release Channel Mac, Mitochondrial Apoptosis-Induced Channel
The Role Of The Mitochondrial Apoptosis Induced Channel Mac In Cytochrome C Release
Oligomeric Bax Is A Component Of The Putative Cytochrome C Release Channel Mac, Mitochondrial Apoptosis-Induced Channel
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Oligomeric Bax Is A Component Of The Putative Cytochrome C Release Channel Mac, Mitochondrial Apoptosis-Induced Channel
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Some Amphiphilic Cations Block The Mitochondrial Apoptosis-Induced Channel, Mac
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Some Amphiphilic Cations Block The Mitochondrial Apoptosis-Induced Channel, Mac
Electrophysiological Approaches To The Study Of Protein Translocation In Mitochondria
Some Amphiphilic Cations Block The Mitochondrial Apoptosis-Induced Channel, Mac
Electrophysiological Approaches To The Study Of Protein Translocation In Mitochondria
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Electrophysiological Approaches To The Study Of Protein Translocation In Mitochondria
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Comparison Of The Tim And Tom Channel Activities Of The Mitochondrial Protein Import Complexes
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Comparison Of The Tim And Tom Channel Activities Of The Mitochondrial Protein Import Complexes
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Comparison Of The Tim And Tom Channel Activities Of The Mitochondrial Protein Import Complexes
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Lithium Blocks The Pkb And Gsk3 Dephosphorylation Induced By Ceramide Through Protein Phosphatase-2a
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Lithium Blocks The Pkb And Gsk3 Dephosphorylation Induced By Ceramide Through Protein Phosphatase-2a
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Lithium Blocks The Pkb And Gsk3 Dephosphorylation Induced By Ceramide Through Protein Phosphatase-2a
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Lesions Induced By 2,4-D And Chlorpyrifos In Tench (Tinca Tinca L.): Implication In Toxicity Studies
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Lesions Induced By 2,4-D And Chlorpyrifos In Tench (Tinca Tinca L.): Implication In Toxicity Studies
Different Dependence Of Lithium And Valproate On P13k/pkb Pathway
Lesions Induced By 2,4-D And Chlorpyrifos In Tench (Tinca Tinca L.): Implication In Toxicity Studies
Different Dependence Of Lithium And Valproate On P13k/pkb Pathway
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Different Dependence Of Lithium And Valproate On P13k/pkb Pathway
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Ultrastructural Study Of Ovine Pulmonary Pasteurellosis: Involvement Of Neutrophils And Macrophages
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Ultrastructural Study Of Ovine Pulmonary Pasteurellosis: Involvement Of Neutrophils And Macrophages
Glu-256 Is A Main Structural Determinant For Oligomerisation Of Human Arginase I
Ultrastructural Study Of Ovine Pulmonary Pasteurellosis: Involvement Of Neutrophils And Macrophages
Glu-256 Is A Main Structural Determinant For Oligomerisation Of Human Arginase I
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Glu-256 Is A Main Structural Determinant For Oligomerisation Of Human Arginase I
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Inhibition Of Phosphatidylcholine Synthesis Precedes Apoptosis Induced By C2-Ceramide: Protection By Exogenous Phosphatidylcholine
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Inhibition Of Phosphatidylcholine Synthesis Precedes Apoptosis Induced By C2-Ceramide: Protection By Exogenous Phosphatidylcholine
Mcc And Psc, The Putative Protein Import Channels Of Mitochondria
Inhibition Of Phosphatidylcholine Synthesis Precedes Apoptosis Induced By C2-Ceramide: Protection By Exogenous Phosphatidylcholine
Mcc And Psc, The Putative Protein Import Channels Of Mitochondria
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Mcc And Psc, The Putative Protein Import Channels Of Mitochondria
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Signal Presequences Increase Mitochondrial Permeability And Open The Multiple Conductance Channel
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Signal Presequences Increase Mitochondrial Permeability And Open The Multiple Conductance Channel
2,4-D Treatment In Tench (Tinca Tinca L.): Pathological Processes On The Excretory Kidney
Signal Presequences Increase Mitochondrial Permeability And Open The Multiple Conductance Channel
2,4-D Treatment In Tench (Tinca Tinca L.): Pathological Processes On The Excretory Kidney
Th1/th2-Regulated Expression Of Arginase Isoforms In Murine Macrophages And Dendritic Cells
2,4-D Treatment In Tench (Tinca Tinca L.): Pathological Processes On The Excretory Kidney
Th1/th2-Regulated Expression Of Arginase Isoforms In Murine Macrophages And Dendritic Cells
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Th1/th2-Regulated Expression Of Arginase Isoforms In Murine Macrophages And Dendritic Cells
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Acute 2,4-D Poisoning In Tench (Tinca Tinca L.): Lesions In The Hematopoietic Portion Of The Kidney
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Acute 2,4-D Poisoning In Tench (Tinca Tinca L.): Lesions In The Hematopoietic Portion Of The Kidney
Histopathological Lesions In Tench, Tinca Tinca (L.) Kidney Followiong Exposure To Chlorpyrifos
Acute 2,4-D Poisoning In Tench (Tinca Tinca L.): Lesions In The Hematopoietic Portion Of The Kidney
Histopathological Lesions In Tench, Tinca Tinca (L.) Kidney Followiong Exposure To Chlorpyrifos
Anti-Mhv3 State Induced By Ifn Gamma In Macrophages Is Not Related To Arginine Metabolism
Histopathological Lesions In Tench, Tinca Tinca (L.) Kidney Followiong Exposure To Chlorpyrifos
Anti-Mhv3 State Induced By Ifn Gamma In Macrophages Is Not Related To Arginine Metabolism
Effects Of Carbonyl Cyanide Phenylhydrazones On Two Mitochondrial Ion Channel Activities
Anti-Mhv3 State Induced By Ifn Gamma In Macrophages Is Not Related To Arginine Metabolism
Effects Of Carbonyl Cyanide Phenylhydrazones On Two Mitochondrial Ion Channel Activities
Involvement Of Protein Kinase A In The Induction Of Arginase In Murine Bone Marrow-Derived Macrophages
Effects Of Carbonyl Cyanide Phenylhydrazones On Two Mitochondrial Ion Channel Activities
Involvement Of Protein Kinase A In The Induction Of Arginase In Murine Bone Marrow-Derived Macrophages
Oxidation Of N(G)-Hydroxy-L-Arginine To Nitric Oxide Mediated By Respiratory Burst: An Alternative Pathway To No Synthesis
Involvement Of Protein Kinase A In The Induction Of Arginase In Murine Bone Marrow-Derived Macrophages
Oxidation Of N(G)-Hydroxy-L-Arginine To Nitric Oxide Mediated By Respiratory Burst: An Alternative Pathway To No Synthesis
Perspectives On The Mitochondrial Multiple Conductance Channel
Oxidation Of N(G)-Hydroxy-L-Arginine To Nitric Oxide Mediated By Respiratory Burst: An Alternative Pathway To No Synthesis
Perspectives On The Mitochondrial Multiple Conductance Channel
Arginase Induction By Suppressors Of Nitric Oxide Synthesis (Il-4, Il-10 And Pge2) In Murine Bone-Marrow-Derived Macrophages
Perspectives On The Mitochondrial Multiple Conductance Channel
Arginase Induction By Suppressors Of Nitric Oxide Synthesis (Il-4, Il-10 And Pge2) In Murine Bone-Marrow-Derived Macrophages
Reciprocal Regulation Of The Nitric Oxide Synthase/arginase Balance In Mouse Bone Marrow‐derived Macrophages By Th 1 And Th 2 Cytokines
Arginase Induction By Suppressors Of Nitric Oxide Synthesis (Il-4, Il-10 And Pge2) In Murine Bone-Marrow-Derived Macrophages
Reciprocal Regulation Of The Nitric Oxide Synthase/arginase Balance In Mouse Bone Marrow‐derived Macrophages By Th 1 And Th 2 Cytokines
Spontaneous Bovine Respiratory Syncytial Virus Infection In Goats: Pathological Findings
Reciprocal Regulation Of The Nitric Oxide Synthase/arginase Balance In Mouse Bone Marrow‐derived Macrophages By Th 1 And Th 2 Cytokines
Spontaneous Bovine Respiratory Syncytial Virus Infection In Goats: Pathological Findings
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
Spontaneous Bovine Respiratory Syncytial Virus Infection In Goats: Pathological Findings
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
Determination Of Arginase Activity In Macrophages: A Micromethod
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
Determination Of Arginase Activity In Macrophages: A Micromethod
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Determination Of Arginase Activity In Macrophages: A Micromethod
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
The Immunologic Response To Experimental Infection With Bovine Respiratory Syncytial Virus In Lambs
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
The Immunologic Response To Experimental Infection With Bovine Respiratory Syncytial Virus In Lambs
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
The Immunologic Response To Experimental Infection With Bovine Respiratory Syncytial Virus In Lambs
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
The Effect Of Antimycin A On Mouse Liver Inner Mitochondrial Membrane Channel Activity
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
The Effect Of Antimycin A On Mouse Liver Inner Mitochondrial Membrane Channel Activity
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
The Effect Of Antimycin A On Mouse Liver Inner Mitochondrial Membrane Channel Activity
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Arginine Catabolism In The Phototrophic Bacterium Rhodobacter Capsulatus E1f1: Purification And Properties Of Arginase
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Arginine Catabolism In The Phototrophic Bacterium Rhodobacter Capsulatus E1f1: Purification And Properties Of Arginase
Trypsin Digestion Of Arginase: Evidence For A Stable Conformation Manganese Directed
Arginine Catabolism In The Phototrophic Bacterium Rhodobacter Capsulatus E1f1: Purification And Properties Of Arginase
Trypsin Digestion Of Arginase: Evidence For A Stable Conformation Manganese Directed
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Trypsin Digestion Of Arginase: Evidence For A Stable Conformation Manganese Directed
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
Properties Of Arginase Immobilized In A Fibrin Clot
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
Properties Of Arginase Immobilized In A Fibrin Clot
The Regulation Of Glucose 6-Phosphate Dehydrogenase From Dicentrarchus Labrax (Bass) Liver
Properties Of Arginase Immobilized In A Fibrin Clot
The Regulation Of Glucose 6-Phosphate Dehydrogenase From Dicentrarchus Labrax (Bass) Liver
Mitochondrial Channel Activity Studied By Patch-Clamping Mitoplasts
The Regulation Of Glucose 6-Phosphate Dehydrogenase From Dicentrarchus Labrax (Bass) Liver
Mitochondrial Channel Activity Studied By Patch-Clamping Mitoplasts
Glucose-6-Phosphate Dehydrogenase From Dicentrarchus Labrax Liver: Kinetic Mechanism And Kinetics Of Nadph Inhibition
Mitochondrial Channel Activity Studied By Patch-Clamping Mitoplasts
Glucose-6-Phosphate Dehydrogenase From Dicentrarchus Labrax Liver: Kinetic Mechanism And Kinetics Of Nadph Inhibition
Circadian Rhythms In Enzymatic Activity Of Rat Liver Arginase And Glucose 6-Phosphate Dehydrogenase.
Glucose-6-Phosphate Dehydrogenase From Dicentrarchus Labrax Liver: Kinetic Mechanism And Kinetics Of Nadph Inhibition
Circadian Rhythms In Enzymatic Activity Of Rat Liver Arginase And Glucose 6-Phosphate Dehydrogenase.
Nucleotide Effects On Kinetic Properties Of Mitochondril Atpase
Circadian Rhythms In Enzymatic Activity Of Rat Liver Arginase And Glucose 6-Phosphate Dehydrogenase.
Nucleotide Effects On Kinetic Properties Of Mitochondril Atpase
Effect Of Nucleotides And Inhibitory Anions On Mitochondrial Atpase. Its Ph Dependence.
Nucleotide Effects On Kinetic Properties Of Mitochondril Atpase
Effect Of Nucleotides And Inhibitory Anions On Mitochondrial Atpase. Its Ph Dependence.
Nucleotide Effects On Kinetic Properties Of Mitochondrial Atpase.
Effect Of Nucleotides And Inhibitory Anions On Mitochondrial Atpase. Its Ph Dependence.
Nucleotide Effects On Kinetic Properties Of Mitochondrial Atpase.
Protonmotive Force And Photophosphorylation In Single Swollen Thylakoid Vesicles
Nucleotide Effects On Kinetic Properties Of Mitochondrial Atpase.
Protonmotive Force And Photophosphorylation In Single Swollen Thylakoid Vesicles
Assay Of Atp Synthesis Using Single Giant Mitochondria
Protonmotive Force And Photophosphorylation In Single Swollen Thylakoid Vesicles
Assay Of Atp Synthesis Using Single Giant Mitochondria
Protonmotive Force In Giant Mitochondria
Assay Of Atp Synthesis Using Single Giant Mitochondria
Protonmotive Force In Giant Mitochondria
Energy Transduction, Ion Transport And Protonmotive Force: Are They Linked?
Protonmotive Force In Giant Mitochondria
Energy Transduction, Ion Transport And Protonmotive Force: Are They Linked?
Effects Of Heating On The Ion-Gating Function And Structural Domains Of The Acetylcholine Receptor
Energy Transduction, Ion Transport And Protonmotive Force: Are They Linked?
Effects Of Heating On The Ion-Gating Function And Structural Domains Of The Acetylcholine Receptor
Controlled Thermal Disruption Of The Ion Gating Function And The Structure Of The Acetylcholine Receptor
Effects Of Heating On The Ion-Gating Function And Structural Domains Of The Acetylcholine Receptor
Controlled Thermal Disruption Of The Ion Gating Function And The Structure Of The Acetylcholine Receptor
Three Catalytic Sites In Mitochondrial Atpase
Controlled Thermal Disruption Of The Ion Gating Function And The Structure Of The Acetylcholine Receptor
Three Catalytic Sites In Mitochondrial Atpase
Reductive Methylation As A Probe Of The Heat-Labile Α-Bungarotoxin-Acetylcholine Receptor Membrane Complex: Evidence For Surface Interactions
Three Catalytic Sites In Mitochondrial Atpase
Reductive Methylation As A Probe Of The Heat-Labile Α-Bungarotoxin-Acetylcholine Receptor Membrane Complex: Evidence For Surface Interactions
Nutritional Regulation Of Glycolysis In Rainbow Trout (Salmo Gairdneri R.)
Reductive Methylation As A Probe Of The Heat-Labile Α-Bungarotoxin-Acetylcholine Receptor Membrane Complex: Evidence For Surface Interactions
Nutritional Regulation Of Glycolysis In Rainbow Trout (Salmo Gairdneri R.)
Physico-Chemical Properties Of Guinea Pig Liver Arginosuccinate Synthetase
Nutritional Regulation Of Glycolysis In Rainbow Trout (Salmo Gairdneri R.)
Physico-Chemical Properties Of Guinea Pig Liver Arginosuccinate Synthetase
Arginine As Competitive Inhibitor Of Arginosuccinate Synthetase From Guinea Pig Liver
Physico-Chemical Properties Of Guinea Pig Liver Arginosuccinate Synthetase
Arginine As Competitive Inhibitor Of Arginosuccinate Synthetase From Guinea Pig Liver
Physico-Chemical Properties Of Guinea Pig Liver Arginase
Arginine As Competitive Inhibitor Of Arginosuccinate Synthetase From Guinea Pig Liver
Physico-Chemical Properties Of Guinea Pig Liver Arginase
Effect Of Ph On The Sensitivity Of Mitochondrial Atpase To Free Atp, Adp And Anions.
Physico-Chemical Properties Of Guinea Pig Liver Arginase
Effect Of Ph On The Sensitivity Of Mitochondrial Atpase To Free Atp, Adp And Anions.
The Influence Of Fasting And Cortisol Upon Arginosuccinate Synthetase, Arginosuccinase And Arginase Levels In Guinea Pig Liver
Effect Of Ph On The Sensitivity Of Mitochondrial Atpase To Free Atp, Adp And Anions.
The Influence Of Fasting And Cortisol Upon Arginosuccinate Synthetase, Arginosuccinase And Arginase Levels In Guinea Pig Liver
Analysis Of Mitochondrial Translocases Tom And Tim By The Patch-Clamping Technique
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