PUBLICACIÓN

Canonical transient receptor potential (TRPC) 1 acts as a negative regulator for vanilloid TRPV6-mediated Ca2+ influx

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ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Schindl, R.; Fritsch, R.; Jardin, I.; Frischauf, I.; Kahr, H.; Muik, M.; Riedl, M.C.; Groschner, K.; Romanin, C.

2012 JOURNAL OF BIOLOGICAL CHEMISTRY

Biochemistry (Q1), Cell Biology (Q1), Molecular Biology (Q1)

JCR: 4.651

SJR: 3.396

CITAS

41

DOI

10.1074/jbc.m112.400952

EID

2-s2.0-84867412996

ISSN

0021-9258

EISSN

1083-351X

BIBTEX

@article{Jardín2012,title = {Canonical transient receptor potential (TRPC) 1 acts as a negative regulator for vanilloid TRPV6-mediated Ca2+ influx},journal = {Journal of Biological Chemistry},year = {2012},volume = {287},number = {42},pages = {35612-35620},author = {Schindl, R. and Fritsch, R. and Jardin, I. and Frischauf, I. and Kahr, H. and Muik, M. and Riedl, M.C. and Groschner, K. and Romanin, C.}}

AUTORES DE LA UEX

Isaac Jardín Polo

OTRAS PUBLICACIONES DE INTERÉS

Hydrogen Peroxide And Peroxynitrite Enhance Ca2+ Mobilization And Aggregation In Platelets From Type 2 Diabetic Patients
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
Filamin A C-Terminal Fragment Modulates Orai1 Expression By Inhibition Of Protein Degradation
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
Filamin A C-Terminal Fragment Modulates Orai1 Expression By Inhibition Of Protein Degradation
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Filamin A C-Terminal Fragment Modulates Orai1 Expression By Inhibition Of Protein Degradation
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Orai1 Plays A Critical Role In Orai3 Synthesis And Stability In Luminal Breast Cancer Cells
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Orai1 Plays A Critical Role In Orai3 Synthesis And Stability In Luminal Breast Cancer Cells
Postbiotics Of Lacticaseibacillus Paracasei Cect 9610 And Lactiplantibacillus Plantarum Cect 9608 Attenuates Store-Operated Calcium Entry And Fak Phosphorylation In Colorectal Cancer Cells
Orai1 Plays A Critical Role In Orai3 Synthesis And Stability In Luminal Breast Cancer Cells
Postbiotics Of Lacticaseibacillus Paracasei Cect 9610 And Lactiplantibacillus Plantarum Cect 9608 Attenuates Store-Operated Calcium Entry And Fak Phosphorylation In Colorectal Cancer Cells
Extended Synaptotagmins 1 And 2 Are Required For Store-Operated Calcium Entry, Cell Migration And Viability In Breast Cancer Cells
Postbiotics Of Lacticaseibacillus Paracasei Cect 9610 And Lactiplantibacillus Plantarum Cect 9608 Attenuates Store-Operated Calcium Entry And Fak Phosphorylation In Colorectal Cancer Cells
Extended Synaptotagmins 1 And 2 Are Required For Store-Operated Calcium Entry, Cell Migration And Viability In Breast Cancer Cells
Differential Functional Role Of Orai1 Variants In Constitutive Ca2+ Entry And Calcification In Luminal Breast Cancer Cells
Extended Synaptotagmins 1 And 2 Are Required For Store-Operated Calcium Entry, Cell Migration And Viability In Breast Cancer Cells
Differential Functional Role Of Orai1 Variants In Constitutive Ca2+ Entry And Calcification In Luminal Breast Cancer Cells
Description Of A New Quantitative Method To Assess Mitochondrial Distribution Pattern In Mature Equine Oocytes
Differential Functional Role Of Orai1 Variants In Constitutive Ca2+ Entry And Calcification In Luminal Breast Cancer Cells
Description Of A New Quantitative Method To Assess Mitochondrial Distribution Pattern In Mature Equine Oocytes
Role Of Orai-Family Channels In The Activation And Regulation Of Transcriptional Activity
Description Of A New Quantitative Method To Assess Mitochondrial Distribution Pattern In Mature Equine Oocytes
Role Of Orai-Family Channels In The Activation And Regulation Of Transcriptional Activity
Functional Differences In Agonist-Induced Plasma Membrane Expression Of Orai1α And Orai1β
Role Of Orai-Family Channels In The Activation And Regulation Of Transcriptional Activity
Functional Differences In Agonist-Induced Plasma Membrane Expression Of Orai1α And Orai1β
Secretion Of Interleukin 6 In Human Skeletal Muscle Cultures Depends On Ca2+ Signalling
Functional Differences In Agonist-Induced Plasma Membrane Expression Of Orai1α And Orai1β
Secretion Of Interleukin 6 In Human Skeletal Muscle Cultures Depends On Ca2+ Signalling
The Ca2+ Sensor Stim In Human Diseases
Secretion Of Interleukin 6 In Human Skeletal Muscle Cultures Depends On Ca2+ Signalling
The Ca2+ Sensor Stim In Human Diseases
Orai1α And Orai1β Support Calcium Entry And Mammosphere Formation In Breast Cancer Stem Cells
The Ca2+ Sensor Stim In Human Diseases
Orai1α And Orai1β Support Calcium Entry And Mammosphere Formation In Breast Cancer Stem Cells
The Store-Operated Ca2+ Channel Orai1α Is Required For Agonist-Evoked Nf-Κb Activation By A Mechanism Dependent On Pkcβ2
Orai1α And Orai1β Support Calcium Entry And Mammosphere Formation In Breast Cancer Stem Cells
The Store-Operated Ca2+ Channel Orai1α Is Required For Agonist-Evoked Nf-Κb Activation By A Mechanism Dependent On Pkcβ2
Store-Operated Calcium Entry In Breast Cancer Cells Is Insensitive To Orai1 And Stim1 N-Linked Glycosylation
The Store-Operated Ca2+ Channel Orai1α Is Required For Agonist-Evoked Nf-Κb Activation By A Mechanism Dependent On Pkcβ2
Store-Operated Calcium Entry In Breast Cancer Cells Is Insensitive To Orai1 And Stim1 N-Linked Glycosylation
Correction To: Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells (Cancers, (2021), 14, 1, (114), 10.3390/cancers14010114)
Store-Operated Calcium Entry In Breast Cancer Cells Is Insensitive To Orai1 And Stim1 N-Linked Glycosylation
Correction To: Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells (Cancers, (2021), 14, 1, (114), 10.3390/cancers14010114)
Orai1α, But Not Orai1β, Co-Localizes With Trpc1 And Is Required For Its Plasma Membrane Location And Activation In Hela Cells
Correction To: Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells (Cancers, (2021), 14, 1, (114), 10.3390/cancers14010114)
Orai1α, But Not Orai1β, Co-Localizes With Trpc1 And Is Required For Its Plasma Membrane Location And Activation In Hela Cells
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Orai1α, But Not Orai1β, Co-Localizes With Trpc1 And Is Required For Its Plasma Membrane Location And Activation In Hela Cells
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Similarities And Differences Between The Orai1 Variants: Orai1α And Orai1β
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Similarities And Differences Between The Orai1 Variants: Orai1α And Orai1β
Store-Operated Calcium Entry And Its Implications In Cancer Stem Cells
Similarities And Differences Between The Orai1 Variants: Orai1α And Orai1β
Store-Operated Calcium Entry And Its Implications In Cancer Stem Cells
A Fibrin Coating Method Of Polypropylene Meshes Enables The Adhesion Of Menstrual Blood-Derived Mesenchymal Stromal Cells: A New Delivery Strategy For Stem Cell-Based Therapies
Store-Operated Calcium Entry And Its Implications In Cancer Stem Cells
A Fibrin Coating Method Of Polypropylene Meshes Enables The Adhesion Of Menstrual Blood-Derived Mesenchymal Stromal Cells: A New Delivery Strategy For Stem Cell-Based Therapies
The Orai1-Ac8 Interplay: How Breast Cancer Cells Escape From Orai1 Channel Inactivation
A Fibrin Coating Method Of Polypropylene Meshes Enables The Adhesion Of Menstrual Blood-Derived Mesenchymal Stromal Cells: A New Delivery Strategy For Stem Cell-Based Therapies
The Orai1-Ac8 Interplay: How Breast Cancer Cells Escape From Orai1 Channel Inactivation
A Multi-Layered Fibrin Coating Allows Menstrual Blood-Derived Mesenchymal Stromal Cells Adhesion On Polypropylene Surgical Meshes
The Orai1-Ac8 Interplay: How Breast Cancer Cells Escape From Orai1 Channel Inactivation
A Multi-Layered Fibrin Coating Allows Menstrual Blood-Derived Mesenchymal Stromal Cells Adhesion On Polypropylene Surgical Meshes
Melatonin Downregulates Trpc6, Impairing Store-Operated Calcium Entry In Triple-Negative Breast Cancer Cells
A Multi-Layered Fibrin Coating Allows Menstrual Blood-Derived Mesenchymal Stromal Cells Adhesion On Polypropylene Surgical Meshes
Melatonin Downregulates Trpc6, Impairing Store-Operated Calcium Entry In Triple-Negative Breast Cancer Cells
Saraf And Efhb Modulate Store-Operated Ca2+ Entry And Are Required For Cell Proliferation, Migration And Viability In Breast Cancer Cells
Melatonin Downregulates Trpc6, Impairing Store-Operated Calcium Entry In Triple-Negative Breast Cancer Cells
Saraf And Efhb Modulate Store-Operated Ca2+ Entry And Are Required For Cell Proliferation, Migration And Viability In Breast Cancer Cells
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Saraf And Efhb Modulate Store-Operated Ca2+ Entry And Are Required For Cell Proliferation, Migration And Viability In Breast Cancer Cells
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Role Of Orai3 In The Pathophysiology Of Cancer
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Role Of Orai3 In The Pathophysiology Of Cancer
Cross-Talk Between The Adenylyl Cyclase/camp Pathway And Ca2+ Homeostasis
Role Of Orai3 In The Pathophysiology Of Cancer
Cross-Talk Between The Adenylyl Cyclase/camp Pathway And Ca2+ Homeostasis
Trpc6 Channel And Its Implications In Breast Cancer: An Overview
Cross-Talk Between The Adenylyl Cyclase/camp Pathway And Ca2+ Homeostasis
Trpc6 Channel And Its Implications In Breast Cancer: An Overview
Functional Role Of Trpc6 And Stim2 In Cytosolic And Endoplasmic Reticulum Ca2+ Content In Resting Estrogen Receptor-Positive Breast Cancer Cells
Trpc6 Channel And Its Implications In Breast Cancer: An Overview
Functional Role Of Trpc6 And Stim2 In Cytosolic And Endoplasmic Reticulum Ca2+ Content In Resting Estrogen Receptor-Positive Breast Cancer Cells
Trpc Channels In The Soce Scenario
Functional Role Of Trpc6 And Stim2 In Cytosolic And Endoplasmic Reticulum Ca2+ Content In Resting Estrogen Receptor-Positive Breast Cancer Cells
Trpc Channels In The Soce Scenario
Molecular Basis And Regulation Of Store-Operated Calcium Entry
Trpc Channels In The Soce Scenario
Molecular Basis And Regulation Of Store-Operated Calcium Entry
Adenylyl Cyclase Type 8 Overexpression Impairs Phosphorylation-Dependent Orai1 Inactivation And Promotes Migration In Mda-Mb-231 Breast Cancer Cells
Molecular Basis And Regulation Of Store-Operated Calcium Entry
Adenylyl Cyclase Type 8 Overexpression Impairs Phosphorylation-Dependent Orai1 Inactivation And Promotes Migration In Mda-Mb-231 Breast Cancer Cells
Flavonoids And Platelet-Derived Thrombotic Disorders
Adenylyl Cyclase Type 8 Overexpression Impairs Phosphorylation-Dependent Orai1 Inactivation And Promotes Migration In Mda-Mb-231 Breast Cancer Cells
Flavonoids And Platelet-Derived Thrombotic Disorders
Store-Independent Orai1-Mediated Ca 2+ Entry And Cancer
Flavonoids And Platelet-Derived Thrombotic Disorders
Store-Independent Orai1-Mediated Ca 2+ Entry And Cancer
Stim1 Phosphorylation At Y316 Modulates Its Interaction With Saraf And The Activation Of Soce And Icrac
Store-Independent Orai1-Mediated Ca 2+ Entry And Cancer
Stim1 Phosphorylation At Y316 Modulates Its Interaction With Saraf And The Activation Of Soce And Icrac
(−)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells
Stim1 Phosphorylation At Y316 Modulates Its Interaction With Saraf And The Activation Of Soce And Icrac
(−)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells
Fine-Tuning Of Micrornas In Type 2 Diabetes Mellitus
(−)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells
Fine-Tuning Of Micrornas In Type 2 Diabetes Mellitus
The Complex Role Of Store Operated Calcium Entry Pathways And Related Proteins In The Function Of Cardiac, Skeletal And Vascular Smooth Muscle Cells
Fine-Tuning Of Micrornas In Type 2 Diabetes Mellitus
The Complex Role Of Store Operated Calcium Entry Pathways And Related Proteins In The Function Of Cardiac, Skeletal And Vascular Smooth Muscle Cells
Efhb Is A Novel Cytosolic Ca 2+ Sensor That Modulates Stim1-Saraf Interaction
The Complex Role Of Store Operated Calcium Entry Pathways And Related Proteins In The Function Of Cardiac, Skeletal And Vascular Smooth Muscle Cells
Efhb Is A Novel Cytosolic Ca 2+ Sensor That Modulates Stim1-Saraf Interaction
Flavonoids And Platelet-Derived Thrombotic Disorders.
Efhb Is A Novel Cytosolic Ca 2+ Sensor That Modulates Stim1-Saraf Interaction
Flavonoids And Platelet-Derived Thrombotic Disorders.
(-)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells.
Flavonoids And Platelet-Derived Thrombotic Disorders.
(-)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells.
Store-Operated Ca 2+ Entry In Breast Cancer Cells: Remodeling And Functional Role
(-)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells.
Store-Operated Ca 2+ Entry In Breast Cancer Cells: Remodeling And Functional Role
Trpc6 Channels Are Required For Proliferation, Migration And Invasion Of Breast Cancer Cell Lines By Modulation Of Orai1 And Orai3 Surface Exposure
Store-Operated Ca 2+ Entry In Breast Cancer Cells: Remodeling And Functional Role
Trpc6 Channels Are Required For Proliferation, Migration And Invasion Of Breast Cancer Cell Lines By Modulation Of Orai1 And Orai3 Surface Exposure
Fine-Tuning Of Store-Operated Calcium Entry By Fast And Slow Ca2+-Dependent Inactivation: Involvement Of Saraf
Trpc6 Channels Are Required For Proliferation, Migration And Invasion Of Breast Cancer Cell Lines By Modulation Of Orai1 And Orai3 Surface Exposure
Fine-Tuning Of Store-Operated Calcium Entry By Fast And Slow Ca2+-Dependent Inactivation: Involvement Of Saraf
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets
Fine-Tuning Of Store-Operated Calcium Entry By Fast And Slow Ca2+-Dependent Inactivation: Involvement Of Saraf
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets
Involvement Of Stanniocalcins In The Deregulation Of Glycaemia In Obese Mice And Type 2 Diabetic Patients
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets
Involvement Of Stanniocalcins In The Deregulation Of Glycaemia In Obese Mice And Type 2 Diabetic Patients
Filamin A Modulates Platelet Function
Involvement Of Stanniocalcins In The Deregulation Of Glycaemia In Obese Mice And Type 2 Diabetic Patients
Filamin A Modulates Platelet Function
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets.
Filamin A Modulates Platelet Function
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets.
Fine-Tuning Of Micrornas In Type 2 Diabetes Mellitus.
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets.
Fine-Tuning Of Micrornas In Type 2 Diabetes Mellitus.
Trps In Pain Sensation
Fine-Tuning Of Micrornas In Type 2 Diabetes Mellitus.
Trps In Pain Sensation
Orai1 And Orai2 Mediate Store-Operated Calcium Entry That Regulates Hl60 Cell Migration And Fak Phosphorylation
Trps In Pain Sensation
Orai1 And Orai2 Mediate Store-Operated Calcium Entry That Regulates Hl60 Cell Migration And Fak Phosphorylation
Role Of Stim2 In Cell Function And Physiopathology
Orai1 And Orai2 Mediate Store-Operated Calcium Entry That Regulates Hl60 Cell Migration And Fak Phosphorylation
Role Of Stim2 In Cell Function And Physiopathology
Apelin: An Antithrombotic Factor That Inhibits Platelet Function
Role Of Stim2 In Cell Function And Physiopathology
Apelin: An Antithrombotic Factor That Inhibits Platelet Function
Stim And Calcium Channel Complexes In Cancer
Apelin: An Antithrombotic Factor That Inhibits Platelet Function
Stim And Calcium Channel Complexes In Cancer
Regulation Of Platelet Function By Orai, Stim And Trp
Stim And Calcium Channel Complexes In Cancer
Regulation Of Platelet Function By Orai, Stim And Trp
The Stim1: Orai Interaction
Regulation Of Platelet Function By Orai, Stim And Trp
The Stim1: Orai Interaction
Molecular Mechanisms Of Stim/orai Communication
The Stim1: Orai Interaction
Molecular Mechanisms Of Stim/orai Communication
Cholesterol Modulates Orai1 Channel Function
Molecular Mechanisms Of Stim/orai Communication
Cholesterol Modulates Orai1 Channel Function
Stim And Orai1 Variants In Store-Operated Calcium Entry
Cholesterol Modulates Orai1 Channel Function
Stim And Orai1 Variants In Store-Operated Calcium Entry
Regulators Of G-Protein-Signaling Proteins: Negative Modulators Of G-Protein-Coupled Receptor Signaling
Stim And Orai1 Variants In Store-Operated Calcium Entry
Regulators Of G-Protein-Signaling Proteins: Negative Modulators Of G-Protein-Coupled Receptor Signaling
A Calcium-Accumulating Region, Car, In The Channel Orai1 Enhances Ca2+ Permeation And Soce-Induced Gene Transcription
Regulators Of G-Protein-Signaling Proteins: Negative Modulators Of G-Protein-Coupled Receptor Signaling
A Calcium-Accumulating Region, Car, In The Channel Orai1 Enhances Ca2+ Permeation And Soce-Induced Gene Transcription
A Coiled-Coil Clamp Controls Both Conformation And Clustering Of Stromal Interaction Molecule 1 (Stim1)
A Calcium-Accumulating Region, Car, In The Channel Orai1 Enhances Ca2+ Permeation And Soce-Induced Gene Transcription
A Coiled-Coil Clamp Controls Both Conformation And Clustering Of Stromal Interaction Molecule 1 (Stim1)
Cholesterol Regulates Orai1 Function
A Coiled-Coil Clamp Controls Both Conformation And Clustering Of Stromal Interaction Molecule 1 (Stim1)
Cholesterol Regulates Orai1 Function
The Trpv1 Ion Channel Is Expressed In Human But Not Mouse Platelets
Cholesterol Regulates Orai1 Function
The Trpv1 Ion Channel Is Expressed In Human But Not Mouse Platelets
Complex Role Of Stim1 In The Activation Of Store-Independent Orai1/3 Channels
The Trpv1 Ion Channel Is Expressed In Human But Not Mouse Platelets
Complex Role Of Stim1 In The Activation Of Store-Independent Orai1/3 Channels
Trpc 1 Acts As A Negative Regulator For Trpv6 Mediated Ca2+ Influx
Complex Role Of Stim1 In The Activation Of Store-Independent Orai1/3 Channels
Trpc 1 Acts As A Negative Regulator For Trpv6 Mediated Ca2+ Influx
Pharmacology Of Trp Channels In The Vasculature
Trpc 1 Acts As A Negative Regulator For Trpv6 Mediated Ca2+ Influx
Pharmacology Of Trp Channels In The Vasculature
Homer Proteins In Ca2+ Entry
Pharmacology Of Trp Channels In The Vasculature
Homer Proteins In Ca2+ Entry
The Polybasic Lysine-Rich Domain Of Plasma Membrane-Resident Stim1 Is Essential For The Modulation Of Store-Operated Divalent Cation Entry By Extracellular Calcium
Homer Proteins In Ca2+ Entry
The Polybasic Lysine-Rich Domain Of Plasma Membrane-Resident Stim1 Is Essential For The Modulation Of Store-Operated Divalent Cation Entry By Extracellular Calcium
The Extended Transmembrane Orai1 N-Terminal (Eton) Region Combines Binding Interface And Gate For Orai1 Activation By Stim1
The Polybasic Lysine-Rich Domain Of Plasma Membrane-Resident Stim1 Is Essential For The Modulation Of Store-Operated Divalent Cation Entry By Extracellular Calcium
The Extended Transmembrane Orai1 N-Terminal (Eton) Region Combines Binding Interface And Gate For Orai1 Activation By Stim1
The Stim1/orai Signaling Machinery
The Extended Transmembrane Orai1 N-Terminal (Eton) Region Combines Binding Interface And Gate For Orai1 Activation By Stim1
The Stim1/orai Signaling Machinery
Identification And Function Of Exchange Proteins Activated Directly By Cyclic Amp (Epac) In Mammalian Spermatozoa
The Stim1/orai Signaling Machinery
Identification And Function Of Exchange Proteins Activated Directly By Cyclic Amp (Epac) In Mammalian Spermatozoa
Vaccination Prepartum Enhances The Beneficial Effects Of Melatonin On The Immune Response And Reduces Platelet Responsiveness In Sheep
Identification And Function Of Exchange Proteins Activated Directly By Cyclic Amp (Epac) In Mammalian Spermatozoa
Vaccination Prepartum Enhances The Beneficial Effects Of Melatonin On The Immune Response And Reduces Platelet Responsiveness In Sheep
Homers Regulate Calcium Entry And Aggregation In Human Platelets: A Role For Homers In The Association Between Stim1 And Orai1
Vaccination Prepartum Enhances The Beneficial Effects Of Melatonin On The Immune Response And Reduces Platelet Responsiveness In Sheep
Homers Regulate Calcium Entry And Aggregation In Human Platelets: A Role For Homers In The Association Between Stim1 And Orai1
Stim1 Tyrosine-Phosphorylation Is Required For Stim1-Orai1 Association In Human Platelets
Homers Regulate Calcium Entry And Aggregation In Human Platelets: A Role For Homers In The Association Between Stim1 And Orai1
Stim1 Tyrosine-Phosphorylation Is Required For Stim1-Orai1 Association In Human Platelets
Transient Receptor Potential Channels In Human Platelets: Expression And Functional Role
Stim1 Tyrosine-Phosphorylation Is Required For Stim1-Orai1 Association In Human Platelets
Transient Receptor Potential Channels In Human Platelets: Expression And Functional Role
Canonical Transient Receptor Potential (Trpc) 1 Acts As A Negative Regulator For Vanilloid Trpv6-Mediated Ca2+ Influx
Transient Receptor Potential Channels In Human Platelets: Expression And Functional Role
Canonical Transient Receptor Potential (Trpc) 1 Acts As A Negative Regulator For Vanilloid Trpv6-Mediated Ca2+ Influx
The Trpc Ion Channels: Association With Orai1 And Stim1 Proteins And Participation In Capacitative And Non-Capacitative Calcium Entry
Canonical Transient Receptor Potential (Trpc) 1 Acts As A Negative Regulator For Vanilloid Trpv6-Mediated Ca2+ Influx
The Trpc Ion Channels: Association With Orai1 And Stim1 Proteins And Participation In Capacitative And Non-Capacitative Calcium Entry
Ca2+ Leakage Rate From Agonist-Sensitive Intracellular Pools Is Altered In Platelets From Patients With Type 2 Diabetes
The Trpc Ion Channels: Association With Orai1 And Stim1 Proteins And Participation In Capacitative And Non-Capacitative Calcium Entry
Ca2+ Leakage Rate From Agonist-Sensitive Intracellular Pools Is Altered In Platelets From Patients With Type 2 Diabetes
Stim1 And Stim2 Are Located In The Acidic Ca2+ Stores And Associates With Orai1 Upon Depletion Of The Acidic Stores In Human Platelets
Ca2+ Leakage Rate From Agonist-Sensitive Intracellular Pools Is Altered In Platelets From Patients With Type 2 Diabetes
Stim1 And Stim2 Are Located In The Acidic Ca2+ Stores And Associates With Orai1 Upon Depletion Of The Acidic Stores In Human Platelets
Attenuated Store-Operated Divalent Cation Entry And Association Between Stim1, Orai1, Htrpc1 And Htrpc6 In Platelets From Type 2 Diabetic Patients
Stim1 And Stim2 Are Located In The Acidic Ca2+ Stores And Associates With Orai1 Upon Depletion Of The Acidic Stores In Human Platelets
Attenuated Store-Operated Divalent Cation Entry And Association Between Stim1, Orai1, Htrpc1 And Htrpc6 In Platelets From Type 2 Diabetic Patients
Lipid Rafts Are Essential For The Regulation Of Soce By Plasma Membrane Resident Stim1 In Human Platelets
Attenuated Store-Operated Divalent Cation Entry And Association Between Stim1, Orai1, Htrpc1 And Htrpc6 In Platelets From Type 2 Diabetic Patients
Lipid Rafts Are Essential For The Regulation Of Soce By Plasma Membrane Resident Stim1 In Human Platelets
Role Of Oxidant Scavengers In The Prevention Of Ca2+ Homeostasis Disorders
Lipid Rafts Are Essential For The Regulation Of Soce By Plasma Membrane Resident Stim1 In Human Platelets
Role Of Oxidant Scavengers In The Prevention Of Ca2+ Homeostasis Disorders
Homocysteine, Intracellular Signaling And Thrombotic Disorders
Role Of Oxidant Scavengers In The Prevention Of Ca2+ Homeostasis Disorders
Homocysteine, Intracellular Signaling And Thrombotic Disorders
Trpc3 Regulates Agonist-Stimulated Ca2+ Mobilization By Mediating The Interaction Between Type I Inositol 1,4,5-Trisphosphate Receptor, Rack1, And Orai1
Homocysteine, Intracellular Signaling And Thrombotic Disorders
Trpc3 Regulates Agonist-Stimulated Ca2+ Mobilization By Mediating The Interaction Between Type I Inositol 1,4,5-Trisphosphate Receptor, Rack1, And Orai1
Melatonin, As An Adjuvant-Like Agent, Enhances Platelet Responsiveness
Trpc3 Regulates Agonist-Stimulated Ca2+ Mobilization By Mediating The Interaction Between Type I Inositol 1,4,5-Trisphosphate Receptor, Rack1, And Orai1
Melatonin, As An Adjuvant-Like Agent, Enhances Platelet Responsiveness
Store-Operated Ca2+ Entry Is Sensitive To The Extracellular Ca2+ Concentration Through Plasma Membrane Stim1
Melatonin, As An Adjuvant-Like Agent, Enhances Platelet Responsiveness
Store-Operated Ca2+ Entry Is Sensitive To The Extracellular Ca2+ Concentration Through Plasma Membrane Stim1
Dynamic Interaction Of Htrpc6 With The Orai1-Stim1 Complex Or Htrpc3 Mediates Its Role In Capacitative Or Non-Capacitative Ca2+ Entry Pathways
Store-Operated Ca2+ Entry Is Sensitive To The Extracellular Ca2+ Concentration Through Plasma Membrane Stim1
Dynamic Interaction Of Htrpc6 With The Orai1-Stim1 Complex Or Htrpc3 Mediates Its Role In Capacitative Or Non-Capacitative Ca2+ Entry Pathways
Acidic-Store Depletion Is Required For Human Platelet Aggregation
Dynamic Interaction Of Htrpc6 With The Orai1-Stim1 Complex Or Htrpc3 Mediates Its Role In Capacitative Or Non-Capacitative Ca2+ Entry Pathways
Acidic-Store Depletion Is Required For Human Platelet Aggregation
Effect Of Homocysteine On Calcium Mobilization And Platelet Function In Type 2 Diabetes Mellitus
Acidic-Store Depletion Is Required For Human Platelet Aggregation
Effect Of Homocysteine On Calcium Mobilization And Platelet Function In Type 2 Diabetes Mellitus
Effect Of Homocysteine On Calcium Mobilization And Platelet Function In Type 2 Diabetes Mellitus
Effect Of Homocysteine On Calcium Mobilization And Platelet Function In Type 2 Diabetes Mellitus
Effect Of Homocysteine On Calcium Mobilization And Platelet Function In Type 2 Diabetes Mellitus
Orai1 Mediates The Interaction Between Stim1 And Htrpc1 And Regulates The Mode Of Activation Of Htrpc1-Forming Ca2+ Channels
Effect Of Homocysteine On Calcium Mobilization And Platelet Function In Type 2 Diabetes Mellitus
Orai1 Mediates The Interaction Between Stim1 And Htrpc1 And Regulates The Mode Of Activation Of Htrpc1-Forming Ca2+ Channels
Intracellular Ca2+ Store Depletion Induces The Formation Of Macromolecular Complexes Involving Htrpc1, Htrpc6, The Type Ii Ip3 Receptor And Serca3 In Human Platelets
Orai1 Mediates The Interaction Between Stim1 And Htrpc1 And Regulates The Mode Of Activation Of Htrpc1-Forming Ca2+ Channels
Intracellular Ca2+ Store Depletion Induces The Formation Of Macromolecular Complexes Involving Htrpc1, Htrpc6, The Type Ii Ip3 Receptor And Serca3 In Human Platelets
Stim1 Regulates Acidic Ca2+ Store Refilling By Interaction With Serca3 In Human Platelets
Intracellular Ca2+ Store Depletion Induces The Formation Of Macromolecular Complexes Involving Htrpc1, Htrpc6, The Type Ii Ip3 Receptor And Serca3 In Human Platelets
Stim1 Regulates Acidic Ca2+ Store Refilling By Interaction With Serca3 In Human Platelets
Cinnamtannin B-1 As An Antioxidant And Platelet Aggregation Inhibitor
Stim1 Regulates Acidic Ca2+ Store Refilling By Interaction With Serca3 In Human Platelets
Cinnamtannin B-1 As An Antioxidant And Platelet Aggregation Inhibitor
Functional Relevance Of The De Novo Coupling Between Htrpc1 And Type Ii Ip3 Receptor In Store-Operated Ca2+ Entry In Human Platelets
Cinnamtannin B-1 As An Antioxidant And Platelet Aggregation Inhibitor
Functional Relevance Of The De Novo Coupling Between Htrpc1 And Type Ii Ip3 Receptor In Store-Operated Ca2+ Entry In Human Platelets
Intracellular Calcium Release From Human Platelets: Different Messengers For Multiple Stores
Functional Relevance Of The De Novo Coupling Between Htrpc1 And Type Ii Ip3 Receptor In Store-Operated Ca2+ Entry In Human Platelets
Intracellular Calcium Release From Human Platelets: Different Messengers For Multiple Stores
Role Of Lipid Rafts In The Interaction Between Htrpc1, Orai1 And Stim1
Intracellular Calcium Release From Human Platelets: Different Messengers For Multiple Stores
Role Of Lipid Rafts In The Interaction Between Htrpc1, Orai1 And Stim1
Phosphatidylinositol 4,5-Bisphosphate Enhances Store-Operated Calcium Entry Through Htrpc6 Channel In Human Platelets
Role Of Lipid Rafts In The Interaction Between Htrpc1, Orai1 And Stim1
Phosphatidylinositol 4,5-Bisphosphate Enhances Store-Operated Calcium Entry Through Htrpc6 Channel In Human Platelets
Physical Properties Of Two Types Of Calcium Stores And Sercas In Human Platelets
Phosphatidylinositol 4,5-Bisphosphate Enhances Store-Operated Calcium Entry Through Htrpc6 Channel In Human Platelets
Physical Properties Of Two Types Of Calcium Stores And Sercas In Human Platelets
Involvement Of Snare Proteins In Thrombin-Induced Platelet Aggregation: Evidence For The Relevance Of Ca2+ Entry
Physical Properties Of Two Types Of Calcium Stores And Sercas In Human Platelets
Involvement Of Snare Proteins In Thrombin-Induced Platelet Aggregation: Evidence For The Relevance Of Ca2+ Entry
Urtica Dioica Extract Reduces Platelet Hyperaggregability In Type 2 Diabetes Mellitus By Inhibition Of Oxidant Production, Ca2+ Mobilization And Protein Tyrosine Phosphorylation
Involvement Of Snare Proteins In Thrombin-Induced Platelet Aggregation: Evidence For The Relevance Of Ca2+ Entry
Urtica Dioica Extract Reduces Platelet Hyperaggregability In Type 2 Diabetes Mellitus By Inhibition Of Oxidant Production, Ca2+ Mobilization And Protein Tyrosine Phosphorylation
Differential Involvement Of Thrombin Receptors In Ca2+ Release From Two Different Intracellular Stores In Human Platelets
Urtica Dioica Extract Reduces Platelet Hyperaggregability In Type 2 Diabetes Mellitus By Inhibition Of Oxidant Production, Ca2+ Mobilization And Protein Tyrosine Phosphorylation
Differential Involvement Of Thrombin Receptors In Ca2+ Release From Two Different Intracellular Stores In Human Platelets
Endogenously Generated Reactive Oxygen Species Reduce Pmca Activity In Platelets From Patients With Non-Insulin-Dependent Diabetes Mellitus
Differential Involvement Of Thrombin Receptors In Ca2+ Release From Two Different Intracellular Stores In Human Platelets
Endogenously Generated Reactive Oxygen Species Reduce Pmca Activity In Platelets From Patients With Non-Insulin-Dependent Diabetes Mellitus
Hydrogen Peroxide And Peroxynitrite Enhance Ca2+ Mobilization And Aggregation In Platelets From Type 2 Diabetic Patients
Endogenously Generated Reactive Oxygen Species Reduce Pmca Activity In Platelets From Patients With Non-Insulin-Dependent Diabetes Mellitus
Hydrogen Peroxide And Peroxynitrite Enhance Ca2+ Mobilization And Aggregation In Platelets From Type 2 Diabetic Patients
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
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