PUBLICACIÓN

Secreción gástrica

ACCEDER A LA PUBLICACIÓN: Dialnet

Camello Almaraz, María Cristina

2018 Fisiología veterinaria

ISBN

9788473605717

AUTORES DE LA UEX

María Cristina Camello Almaraz

OTRAS PUBLICACIONES DE INTERÉS

Histamine Does Not Potentiate Cyclic Amp-Mediated Amylase Secretion In The Guinea-Pig Pancreatic Acinar Cells
Pharmacological Inhibition Reveals Participation Of The Endocytic Compartment In Positive Feedback Il-6 Secretion In Human Skeletal Myotubes
Secretion Of Interleukin 6 In Human Skeletal Muscle Cultures Depends On Ca2+ Signalling
Pharmacological Inhibition Reveals Participation Of The Endocytic Compartment In Positive Feedback Il-6 Secretion In Human Skeletal Myotubes
Secretion Of Interleukin 6 In Human Skeletal Muscle Cultures Depends On Ca2+ Signalling
Monochloramine Effects On Gallbladder Contractility
Secretion Of Interleukin 6 In Human Skeletal Muscle Cultures Depends On Ca2+ Signalling
Monochloramine Effects On Gallbladder Contractility
Age-Induced Differential Changes In The Central And Colonic Human Circadian Oscillators
Monochloramine Effects On Gallbladder Contractility
Age-Induced Differential Changes In The Central And Colonic Human Circadian Oscillators
Fisiología Hepática
Age-Induced Differential Changes In The Central And Colonic Human Circadian Oscillators
Fisiología Hepática
Bases Neurológicas De La Motilidad
Fisiología Hepática
Bases Neurológicas De La Motilidad
Secreción Gástrica
Bases Neurológicas De La Motilidad
Secreción Gástrica
Octodon Degus, A New Model To Study The Agonist And Plexus-Induced Response In The Urinary Bladder
Secreción Gástrica
Octodon Degus, A New Model To Study The Agonist And Plexus-Induced Response In The Urinary Bladder
Propagation Of Intracellular Ca2+ Signals In Aged Exocrine Cells
Octodon Degus, A New Model To Study The Agonist And Plexus-Induced Response In The Urinary Bladder
Propagation Of Intracellular Ca2+ Signals In Aged Exocrine Cells
Age-Related Changes In Mitochondrial Function Of Mouse Colonic Smooth Muscle: Beneficial Effects Of Melatonin
Propagation Of Intracellular Ca2+ Signals In Aged Exocrine Cells
Age-Related Changes In Mitochondrial Function Of Mouse Colonic Smooth Muscle: Beneficial Effects Of Melatonin
Mtor Pathway And Ca2+ Stores Mobilization In Aged Smooth Muscle Cells
Age-Related Changes In Mitochondrial Function Of Mouse Colonic Smooth Muscle: Beneficial Effects Of Melatonin
Mtor Pathway And Ca2+ Stores Mobilization In Aged Smooth Muscle Cells
Aging Impairs Ca2+ Sensitization Pathways In Gallbladder Smooth Muscle
Mtor Pathway And Ca2+ Stores Mobilization In Aged Smooth Muscle Cells
Aging Impairs Ca2+ Sensitization Pathways In Gallbladder Smooth Muscle
Aging-Induced Alterations In Female Rat Colon Smooth Muscle: The Protective Effects Of Hormonal Therapy
Aging Impairs Ca2+ Sensitization Pathways In Gallbladder Smooth Muscle
Aging-Induced Alterations In Female Rat Colon Smooth Muscle: The Protective Effects Of Hormonal Therapy
Nfatc3 Regulates Trypsinogen Activation, Neutrophil Recruitment, And Tissue Damage In Acute Pancreatitis In Mice
Aging-Induced Alterations In Female Rat Colon Smooth Muscle: The Protective Effects Of Hormonal Therapy
Nfatc3 Regulates Trypsinogen Activation, Neutrophil Recruitment, And Tissue Damage In Acute Pancreatitis In Mice
Melatonin, And To A Lesser Extent Growth Hormone, Restores Colonic Smooth Muscle Physiology In Old Rats
Nfatc3 Regulates Trypsinogen Activation, Neutrophil Recruitment, And Tissue Damage In Acute Pancreatitis In Mice
Melatonin, And To A Lesser Extent Growth Hormone, Restores Colonic Smooth Muscle Physiology In Old Rats
Melatonin Induces Neural Sod2 Expression Independent Of The Nf-Kappab Pathway And Improves The Mitochondrial Population And Function In Old Mice
Melatonin, And To A Lesser Extent Growth Hormone, Restores Colonic Smooth Muscle Physiology In Old Rats
Melatonin Induces Neural Sod2 Expression Independent Of The Nf-Kappab Pathway And Improves The Mitochondrial Population And Function In Old Mice
Melatonin, A Potential Therapeutic Agent For Smooth Muscle-Related Pathological Conditions And Aging
Melatonin Induces Neural Sod2 Expression Independent Of The Nf-Kappab Pathway And Improves The Mitochondrial Population And Function In Old Mice
Melatonin, A Potential Therapeutic Agent For Smooth Muscle-Related Pathological Conditions And Aging
Capítulo 62. Bases Neurológicas De La Motilidad
Melatonin, A Potential Therapeutic Agent For Smooth Muscle-Related Pathological Conditions And Aging
Capítulo 62. Bases Neurológicas De La Motilidad
Fisiología Hepática
Capítulo 62. Bases Neurológicas De La Motilidad
Fisiología Hepática
Developmental Changes In Ca2+ Homeostasis And Contractility In Gallbladder Smooth Muscle
Fisiología Hepática
Developmental Changes In Ca2+ Homeostasis And Contractility In Gallbladder Smooth Muscle
Age-Related Alterations In Ca2+ Signals And Mitochondrial Membrane Potential In Exocrine Cells Are Prevented By Melatonin
Developmental Changes In Ca2+ Homeostasis And Contractility In Gallbladder Smooth Muscle
Age-Related Alterations In Ca2+ Signals And Mitochondrial Membrane Potential In Exocrine Cells Are Prevented By Melatonin
Calcium Controls Smooth Muscle Trpc Gene Transcription Via The Camk/calcineurin-Dependent Pathways
Age-Related Alterations In Ca2+ Signals And Mitochondrial Membrane Potential In Exocrine Cells Are Prevented By Melatonin
Calcium Controls Smooth Muscle Trpc Gene Transcription Via The Camk/calcineurin-Dependent Pathways
Melatonin Treatment Reverts Age-Related Changes In Guinea Pig Gallbladder Neuromuscular Transmission And Contractility
Calcium Controls Smooth Muscle Trpc Gene Transcription Via The Camk/calcineurin-Dependent Pathways
Melatonin Treatment Reverts Age-Related Changes In Guinea Pig Gallbladder Neuromuscular Transmission And Contractility
Mitochondrial Reactive Oxygen Species And Ca2+ Signaling
Melatonin Treatment Reverts Age-Related Changes In Guinea Pig Gallbladder Neuromuscular Transmission And Contractility
Mitochondrial Reactive Oxygen Species And Ca2+ Signaling
Mitochondrial Production Of Oxidants Is Necessary For Physiological Calcium Oscillations
Mitochondrial Reactive Oxygen Species And Ca2+ Signaling
Mitochondrial Production Of Oxidants Is Necessary For Physiological Calcium Oscillations
Changes In Guinea Pig Gallbladder Smooth Muscle Ca2+ Homeostasis By Acute Acalculous Cholecystitis
Mitochondrial Production Of Oxidants Is Necessary For Physiological Calcium Oscillations
Changes In Guinea Pig Gallbladder Smooth Muscle Ca2+ Homeostasis By Acute Acalculous Cholecystitis
Ca2+ Accumulation Into Acidic Organelles Mediated By Ca 2+- And Vacuolar H+-Atpases In Human Platelets
Changes In Guinea Pig Gallbladder Smooth Muscle Ca2+ Homeostasis By Acute Acalculous Cholecystitis
Ca2+ Accumulation Into Acidic Organelles Mediated By Ca 2+- And Vacuolar H+-Atpases In Human Platelets
Non-Stimulated Ca2+ Leak Pathway In Cerebellar Granule Neurones
Ca2+ Accumulation Into Acidic Organelles Mediated By Ca 2+- And Vacuolar H+-Atpases In Human Platelets
Non-Stimulated Ca2+ Leak Pathway In Cerebellar Granule Neurones
Long Distance Communication Between Muscarinic Receptors And Ca2+ Release Channels Revealed By Carbachol Uncaging In Cell-Attached Patch Pipette
Non-Stimulated Ca2+ Leak Pathway In Cerebellar Granule Neurones
Long Distance Communication Between Muscarinic Receptors And Ca2+ Release Channels Revealed By Carbachol Uncaging In Cell-Attached Patch Pipette
Basal And Physiological Ca2+ Leak From The Endoplasmic Reticulum Of Pancreatic Acinar Cells. Second Messenger-Activated Channels And Translocons
Long Distance Communication Between Muscarinic Receptors And Ca2+ Release Channels Revealed By Carbachol Uncaging In Cell-Attached Patch Pipette
Basal And Physiological Ca2+ Leak From The Endoplasmic Reticulum Of Pancreatic Acinar Cells. Second Messenger-Activated Channels And Translocons
Calcium Leak From Intracellular Store - The Enigma Of Calcium Signalling
Basal And Physiological Ca2+ Leak From The Endoplasmic Reticulum Of Pancreatic Acinar Cells. Second Messenger-Activated Channels And Translocons
Calcium Leak From Intracellular Store - The Enigma Of Calcium Signalling
Role Of Mitochondria In Ca2+ Oscillations And Shape Of Ca2+ Signals In Pancreatic Acinar Cells
Calcium Leak From Intracellular Store - The Enigma Of Calcium Signalling
Role Of Mitochondria In Ca2+ Oscillations And Shape Of Ca2+ Signals In Pancreatic Acinar Cells
Release Of Calcium From Mitochondrial And Nonmitochondrial Intracellular Stores In Mouse Pancreatic Acinar Cells By Hydrogen Peroxide
Role Of Mitochondria In Ca2+ Oscillations And Shape Of Ca2+ Signals In Pancreatic Acinar Cells
Release Of Calcium From Mitochondrial And Nonmitochondrial Intracellular Stores In Mouse Pancreatic Acinar Cells By Hydrogen Peroxide
Differential Involvement Of Vacuolar H+-Atpase In The Refilling Of Thapsigargin- And Agonist-Mobilized Ca2+ Stores
Release Of Calcium From Mitochondrial And Nonmitochondrial Intracellular Stores In Mouse Pancreatic Acinar Cells By Hydrogen Peroxide
Differential Involvement Of Vacuolar H+-Atpase In The Refilling Of Thapsigargin- And Agonist-Mobilized Ca2+ Stores
Role Of Proton Gradients And Vacuolar H+-Atpases In The Refilling Of Intracellular Calcium Stores In Exocrine Cells
Differential Involvement Of Vacuolar H+-Atpase In The Refilling Of Thapsigargin- And Agonist-Mobilized Ca2+ Stores
Role Of Proton Gradients And Vacuolar H+-Atpases In The Refilling Of Intracellular Calcium Stores In Exocrine Cells
Effects Of Antioxidants On Calcium Signal Induced By Cholecystokinin In Mouse Pancreatic Acinar Cells
Role Of Proton Gradients And Vacuolar H+-Atpases In The Refilling Of Intracellular Calcium Stores In Exocrine Cells
Effects Of Antioxidants On Calcium Signal Induced By Cholecystokinin In Mouse Pancreatic Acinar Cells
Egf Stimulates Tyrosine Phosphorylation Of Focal Adhesion Kinase (P125(Fak)) And Paxillin In Rat Pancreatic Acini By A Phospholipase C-Independent Process That Depends On Phosphatidylinositol 3-Kinase, The Small Gtp-Binding Protein, P21(Rho), And The Integrity Of The Actin Cytoskeleton
Effects Of Antioxidants On Calcium Signal Induced By Cholecystokinin In Mouse Pancreatic Acinar Cells
Egf Stimulates Tyrosine Phosphorylation Of Focal Adhesion Kinase (P125(Fak)) And Paxillin In Rat Pancreatic Acini By A Phospholipase C-Independent Process That Depends On Phosphatidylinositol 3-Kinase, The Small Gtp-Binding Protein, P21(Rho), And The Integrity Of The Actin Cytoskeleton
Sequential Activation Of Different Ca2+ Entry Pathways Upon Cholinergic Stimulation In Mouse Pancreatic Acinar Cells
Egf Stimulates Tyrosine Phosphorylation Of Focal Adhesion Kinase (P125(Fak)) And Paxillin In Rat Pancreatic Acini By A Phospholipase C-Independent Process That Depends On Phosphatidylinositol 3-Kinase, The Small Gtp-Binding Protein, P21(Rho), And The Integrity Of The Actin Cytoskeleton
Sequential Activation Of Different Ca2+ Entry Pathways Upon Cholinergic Stimulation In Mouse Pancreatic Acinar Cells
Histamine Does Not Potentiate Cyclic Amp-Mediated Amylase Secretion In The Guinea-Pig Pancreatic Acinar Cells
Sequential Activation Of Different Ca2+ Entry Pathways Upon Cholinergic Stimulation In Mouse Pancreatic Acinar Cells
Histamine Does Not Potentiate Cyclic Amp-Mediated Amylase Secretion In The Guinea-Pig Pancreatic Acinar Cells
Pharmacological Inhibition Reveals Participation Of The Endocytic Compartment In Positive Feedback Il-6 Secretion In Human Skeletal Myotubes
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