PUBLICACIÓN

Quantification of cyclin D1 and D2 proteins in multiple myeloma identifies different expression patterns from those revealed by gene expression profiling

ACCEDER A LA PUBLICACIÓN: Orcid

Ignacio Cardona-Benavides et al.

2023

DOI

10.21203/rs.3.rs-2616507/v1

AUTORES DE LA UEX

Aida María López Guerrero

OTRAS PUBLICACIONES DE INTERÉS

Calcium Signaling In Mouse Oocyte Maturation: The Roles Of Stim1, Orai1 And Soce
Quantification Of Cyclin D1 And D2 Proteins In Multiple Myeloma Identifies Different Expression Patterns From Those Revealed By Gene Expression Profiling
Quantification Of Cyclin D1 And D2 Proteins In Multiple Myeloma Identifies Different Expression Patterns From Those Revealed By Gene Expression Profiling
Quantification Of Cyclin D1 And D2 Proteins In Multiple Myeloma Identifies Different Expression Patterns From Those Revealed By Gene Expression Profiling
Quantification Of Cyclin D1 And D2 Proteins In Multiple Myeloma Identifies Different Expression Patterns From Those Revealed By Gene Expression Profiling
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Quantification Of Cyclin D1 And D2 Proteins In Multiple Myeloma Identifies Different Expression Patterns From Those Revealed By Gene Expression Profiling
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Store-Operated Calcium Entry Is Dispensable For The Activation Of Erk1/2 Pathway In Prostate Cancer Cells
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Store-Operated Calcium Entry Is Dispensable For The Activation Of Erk1/2 Pathway In Prostate Cancer Cells
Regulation Of Membrane Ruffling By Polarized Stim1 And Orai1 In Cortactin-Rich Domains
Store-Operated Calcium Entry Is Dispensable For The Activation Of Erk1/2 Pathway In Prostate Cancer Cells
Regulation Of Membrane Ruffling By Polarized Stim1 And Orai1 In Cortactin-Rich Domains
Phospho-Stim1 Is A Downstream Effector That Mediates The Signaling Triggered By Igf-1 In Hek293 Cells
Regulation Of Membrane Ruffling By Polarized Stim1 And Orai1 In Cortactin-Rich Domains
Phospho-Stim1 Is A Downstream Effector That Mediates The Signaling Triggered By Igf-1 In Hek293 Cells
Stim1 Phosphorylation Triggered By Epidermal Growth Factor Mediates Cell Migration
Phospho-Stim1 Is A Downstream Effector That Mediates The Signaling Triggered By Igf-1 In Hek293 Cells
Stim1 Phosphorylation Triggered By Epidermal Growth Factor Mediates Cell Migration
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
Stim1 Phosphorylation Triggered By Epidermal Growth Factor Mediates Cell Migration
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
Calcium Signaling In Mouse Oocyte Maturation: The Roles Of Stim1, Orai1 And Soce
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
Calcium Signaling In Mouse Oocyte Maturation: The Roles Of Stim1, Orai1 And Soce
Quantification Of Cyclin D1 And D2 Proteins In Multiple Myeloma Identifies Different Expression Patterns From Those Revealed By Gene Expression Profiling
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