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- ENDOCRINOLOGÍA MOLECULAR
- ACTIVIDADES FÍSICAS EN PERSONAS CON PROBLEMAS DE SALUD CRÓNICOS: NEUROMUSCULARES, RESPIRATORIAS, DOL
- ACTIVIDADES FÍSICAS PERSONAS PROBLEMAS SALUD CRÓNICOS: NEUROMUSCULARES, RESPIRATORIAS, DOLOR, CANCER
- ACTIVIDADES FÍSICAS PERSONAS PROBLEMAS SALUD CRÓNICOS: NEUROMUSCULARES, RESPIRATORIAS, DOLOR, CANCER
- ACTIVIDADES FÍSICAS PERSONAS PROBLEMAS SALUD CRÓNICOS: NEUROMUSCULARES, RESPIRATORIAS, DOLOR, CANCER
- ENDOCRINOLOGÍA MOLECULAR
- ENDOCRINOLOGÍA Y REGULACIÓN METABÓLICA
- ENDOCRINOLOGÍA Y REGULACIÓN METABÓLICA
- FISIOLOGÍA HUMANA
- FISIOLOGÍA HUMANA
- FISIOLOGÍA HUMANA
- FISIOLOGÍA HUMANA BÁSICA
- HEMATOLOGÍA Y HEMOTERAPIA
- HEMATOLOGÍA Y HEMOTERAPIA
- HEMATOLOGÍA Y HEMOTERAPIA
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- (-)‑Oleocanthal inhibits proliferation and migration by modulating Ca2+ entry through TRPC6 in breast cancer cells.
- (−)‑Oleocanthal inhibits proliferation and migration by modulating Ca2+ entry through TRPC6 in breast cancer cells
- A fibrin coating method of polypropylene meshes enables the adhesion of menstrual blood-derived mesenchymal stromal cells: A new delivery strategy for stem cell-based therapies
- A subset of Orai1α and Orai1β subunits heteromerizes to form CRAC channels
- Acidic NAADP-releasable Ca2+ compartments in the megakaryoblastic cell line MEG01
- Adenylyl cyclase type 8 overexpression impairs phosphorylation-dependent orai1 inactivation and promotes migration in MDA-MB-231 breast cancer cells
- Antiaggregant effects of Arbutus unedo extracts in human platelets
- Apelin: an antithrombotic factor that inhibits platelet function
- Arachidonic acid attenuates cell proliferation, migration and viability by a mechanism independent on calcium entry
- Attenuated store-operated divalent cation entry and association between STIM1, Orai1, hTRPC1 and hTRPC6 in platelets from type 2 diabetic patients
- Axial stretch-dependent cation entry in dystrophic cardiomyopathy: Involvement of several TRPs channels
- Benefit of SERCA2a gene transfer to vascular endothelial and smooth muscle cells: A new aspect in therapy of cardiovascular diseases
- Ca2+ accumulation into acidic organelles mediated by Ca 2+- and vacuolar H+-ATPases in human platelets
- Cardiovascular and hemostatic disorders: SOCE and Ca2+ handling in platelet dysfunction
- Cardiovascular system SOCE and Ca2+ handling in platelet dysfunction
- Caspase-3 and -9 are activated in human myeloid HL-60 cells by calcium signal
- Cinnamtannin B-1 as an antioxidant and platelet aggregation inhibitor
- Correction to: Orai2 Modulates Store-Operated Ca2+ Entry and Cell Cycle Progression in Breast Cancer Cells (Cancers, (2021), 14, 1, (114), 10.3390/cancers14010114)
- Cross-Talk Between the Adenylyl Cyclase/cAMP Pathway and Ca2+ Homeostasis
- Crucial role for endoplasmic reticulum stress during megakaryocyte maturation
- Cytoskeletal and scaffolding proteins as structural and functional determinants of TRP channels
- Data from Inactivation of Proprotein Convertases in T Cells Inhibits PD-1 Expression and Creates a Favorable Immune Microenvironment in Colorectal Cancer
- Data from Inactivation of Proprotein Convertases in T Cells Inhibits PD-1 Expression and Creates a Favorable Immune Microenvironment in Colorectal Cancer
- Data on calcium increases depending on stretch in dystrophic cardiomyocytes
- Differential functional role of Orai1 variants in constitutive Ca2+ entry and calcification in luminal breast cancer cells
- Dynamic interaction of SARAF with STIM1 and Orai1 to modulate store-operated calcium entry
- EFHB is a novel cytosolic Ca 2+ sensor that modulates STIM1-SARAF interaction
- ELA/APELA precursor cleaved by furin displays tumor suppressor function in renal cell carcinoma through mTORC1 activation
- Early caspase-3 activation independent of apoptosis is required for cellular function
- Enhanced expression of STIM1/Orai1 and TRPC3 in platelets from patients with type 2 diabetes mellitus
- Extended Synaptotagmins 1 and 2 Are Required for Store-Operated Calcium Entry, Cell Migration and Viability in Breast Cancer Cells
- Feedback modulation of Orai1α and Orai1β protein content mediated by STIM proteins
- Feedback modulation of Orai1α and Orai1β protein content mediated by STIM proteins
- Figures S1-S4 from Inactivation of Proprotein Convertases in T Cells Inhibits PD-1 Expression and Creates a Favorable Immune Microenvironment in Colorectal Cancer
- Figures S1-S4 from Inactivation of Proprotein Convertases in T Cells Inhibits PD-1 Expression and Creates a Favorable Immune Microenvironment in Colorectal Cancer
- Filamin A C-terminal fragment modulates Orai1 expression by inhibition of protein degradation
- Filamin A Modulates Store-Operated Ca2+ Entry by Regulating STIM1 (Stromal Interaction Molecule 1)-Orai1 Association in Human Platelets
- Filamin A Modulates Store-Operated Ca2+ Entry by Regulating STIM1 (Stromal Interaction Molecule 1)-Orai1 Association in Human Platelets.
- Filamin A modulates platelet function
- Fine-tuning of store-operated calcium entry by fast and slow Ca2+-dependent inactivation: Involvement of SARAF
- Flavonoids and platelet-derived thrombotic disorders
- Flavonoids and platelet-derived thrombotic disorders.
- Functional differences in agonist-induced plasma membrane expression of Orai1α and Orai1β
- Functional relevance of the de novo coupling between hTRPC1 and type II IP3 receptor in store-operated Ca2+ entry in human platelets
- Functional role of TRPC6 and STIM2 in cytosolic and endoplasmic reticulum Ca2+ content in resting estrogen receptor-positive breast cancer cells
- Furin Prodomain Ppfurin Enhances Soce Through trpc6 Activation in Breast Cancer Cells
- Furin prodomain ppfurin enhances ca2+ entry through orai and trpc6 channels’ activation in breast cancer cells
- Historical overview of store-operated Ca2+ entry
- Homer proteins in Ca2+ entry
- Homocysteine induces caspase activation by endoplasmic reticulum stress in platelets from type 2 diabetics and healthy donors
- Inactivation of proprotein convertases in T cells inhibits PD-1 expression and creates a favorable immune microenvironment in colorectal cancer
- Interaction of STIM1 with endogenously expressed human canonical TRP1 upon depletion of intracellular Ca2+ stores
- Intracellular Ca2+ homeostasis and aggregation in platelets are impaired by ethanol through the generation of H2O2 and oxidation of sulphydryl groups
- Intracellular Ca2+ store depletion induces the formation of macromolecular complexes involving hTRPC1, hTRPC6, the type II IP3 receptor and SERCA3 in human platelets
- Intracellular Calcium Release from Human Platelets: Different Messengers for Multiple Stores
- Involvement of stanniocalcins in the deregulation of glycaemia in obese mice and type 2 diabetic patients
- Melatonin, as an adjuvant-like agent, enhances platelet responsiveness
- Molecular Basis and Regulation of Store-Operated Calcium Entry
- Molecular modulators of store-operated calcium entry
- N,N,N′,N′-tetrakis(2-pyridylmethyl)ethylenediamine induces apoptosis through the activation of caspases-3 and -8 in human platelets. A role for endoplasmic reticulum stress
- New insights into adipokines as potential biomarkers for type-2 diabetes mellitus
- New insights into adipokines as potential biomarkers for type-2 diabetes mellitus.
- Orai1 mediates the interaction between STIM1 and hTRPC1 and regulates the mode of activation of hTRPC1-forming Ca2+ channels
- Orai1α and Orai1β support calcium entry and mammosphere formation in breast cancer stem cells
- Orai1α, but not Orai1β, co-localizes with TRPC1 and is required for its plasma membrane location and activation in HeLa cells
- Orai2 Modulates Store-Operated Ca2+ Entry and Cell Cycle Progression in Breast Cancer Cells
- Orai2 modulates store-operated ca2+ entry and cell cycle progression in breast cancer cells
- Pharmacology of TRP channels in the vasculature
- Platelet Functions are Decreased in Obesity and Restored after Weight Loss: Evidence for a Role of the SERCA3-Dependent ADP Secretion Pathway
- Postbiotics of Lacticaseibacillus paracasei CECT 9610 and Lactiplantibacillus plantarum CECT 9608 attenuates store-operated calcium entry and FAK phosphorylation in colorectal cancer cells
- Role of Orai-family channels in the activation and regulation of transcriptional activity
- Role of STIM1 in the surface expression of SARAF
- Role of intracellular calcium on hydrogen peroxide-induced apoptosis in rat pancreatic acinar AR42J cells
- Role of mTOR1 and mTOR2 complexes in MEG-01 cell physiology
- Role of orai3 in the pathophysiology of cancer
- SARAF and EFHB Modulate Store-Operated Ca2+ Entry and Are Required for Cell Proliferation, Migration and Viability in Breast Cancer Cells
- SARAF modulates TRPC1, but not TRPC6, channel function in a STIM1-independent manner
- SERCA2a controls the mode of agonist-induced intracellular Ca2+ signal, transcription factor NFAT and proliferation in human vascular smooth muscle cells
- STIM1 and STIM2 are located in the acidic Ca2+ stores and associates with Orai1 upon depletion of the acidic stores in human platelets
- STIM1 regulates acidic Ca2+ store refilling by interaction with SERCA3 in human platelets
- Similarities and Differences between the Orai1 Variants: Orai1α and Orai1β
- Store-Operated Calcium Entry and Its Implications in Cancer Stem Cells
- Store-Operated Calcium Entry in Breast Cancer Cells Is Insensitive to Orai1 and STIM1 N-Linked Glycosylation
- Store-independent Orai1-mediated Ca 2+ entry and cancer
- Store-operated Ca 2+ entry in breast cancer cells: Remodeling and functional role
- Store-operated Ca2+ entry- Associated regulatory factor (SARAF) plays an important role in the regulation of arachidonate-regulated Ca2+ (ARC) channels
- Store-operated Ca2+ entry is sensitive to the extracellular Ca2+ concentration through plasma membrane STIM1
- Supplementary Materials and Methods from Inactivation of Proprotein Convertases in T Cells Inhibits PD-1 Expression and Creates a Favorable Immune Microenvironment in Colorectal Cancer
- Supplementary Materials and Methods from Inactivation of Proprotein Convertases in T Cells Inhibits PD-1 Expression and Creates a Favorable Immune Microenvironment in Colorectal Cancer
- Supplementary Table S1 from Inactivation of Proprotein Convertases in T Cells Inhibits PD-1 Expression and Creates a Favorable Immune Microenvironment in Colorectal Cancer
- Supplementary Table S1 from Inactivation of Proprotein Convertases in T Cells Inhibits PD-1 Expression and Creates a Favorable Immune Microenvironment in Colorectal Cancer
- Supplementary Table S2 from Inactivation of Proprotein Convertases in T Cells Inhibits PD-1 Expression and Creates a Favorable Immune Microenvironment in Colorectal Cancer
- Supplementary Table S2 from Inactivation of Proprotein Convertases in T Cells Inhibits PD-1 Expression and Creates a Favorable Immune Microenvironment in Colorectal Cancer
- Synergistic role of protein phosphatase inhibitor 1 and sarco/endoplasmic reticulum Ca2+-ATPase in the acquisition of the contractile phenotype of arterial smooth muscle cells
- TRPC channels in the SOCE scenario
- TRPC3 regulates agonist-stimulated Ca2+ mobilization by mediating the interaction between type I inositol 1,4,5-trisphosphate receptor, RACK1, and Orai1
- TRPC6 participates in the regulation of cytosolic basal calcium concentration in murine resting platelets
- TRPs in pain sensation
- The Ca2+ Sensor STIM in Human Diseases
- The orai1-ac8 interplay: How breast cancer cells escape from orai1 channel inactivation
- The polybasic lysine-rich domain of plasma membrane-resident STIM1 is essential for the modulation of store-operated divalent cation entry by extracellular calcium
- The store-operated Ca2+ channel Orai1α is required for agonist-evoked NF-κB activation by a mechanism dependent on PKCβ2
- Thrombin induces activation and translocation of Bid, Bax and Bak to the mitochondria in human platelets
- Thrombin induces apoptotic events through the generation of reactive oxygen species in human platelets
- Transient receptor potential ankyrin-1 (TRPA1) modulates store-operated Ca2+ entry by regulation of STIM1-Orai1 association
- Trpc6 channels are required for proliferation, migration and invasion of breast cancer cell lines by modulation of orai1 and orai3 surface exposure
- Two distinct Ca2+ compartments show differential sensitivity to thrombin, ADP and vasopressin in human platelets
- Two pathways for store-mediated calcium entry differentially dependent on the actin cytoskeleton in human platelets
- Two-pore channel 2 (TPC2) modulates store-operated Ca2+ entry
- Tyrosine phosphorylation/dephosphorylation balance is involved in thrombin-evoked microtubular reorganisation in human platelets
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