PUBLICACIÓN

ACCEDER A LA PUBLICACIÓN: Scopus Orcid Dialnet

Molecular Medicine (Q1)

JCR: 8.3

SJR: 3.571

CITAS

DOI

EID

ISSN

EISSN

ISBN

BIBTEX

AUTORES DE LA UEX

OTRAS PUBLICACIONES DE INTERÉS

Melatonin Induces Reactive Oxygen Species Generation And Changes In Glutathione Levels And Reduces Viability In Human Pancreatic Stellate Cells
In Vitro Digestion And Culture In Caco-2 Cells To Assess The Bioavailability Of Fatty Acids: A Case Study In Meat Matrix Enriched With Ω-3 Microcapsules
Targeting Nupr1-Dependent Stress Granules Formation To Induce Synthetic Lethality In Krasg12d-Driven Tumors
In Vitro Digestion And Culture In Caco-2 Cells To Assess The Bioavailability Of Fatty Acids: A Case Study In Meat Matrix Enriched With Ω-3 Microcapsules
Targeting Nupr1-Dependent Stress Granules Formation To Induce Synthetic Lethality In Krasg12d-Driven Tumors
Membrane Lipid Derivatives: Roles Of Arachidonic Acid And Its Metabolites In Pancreatic Physiology And Pathophysiology
Targeting Nupr1-Dependent Stress Granules Formation To Induce Synthetic Lethality In Krasg12d-Driven Tumors
Membrane Lipid Derivatives: Roles Of Arachidonic Acid And Its Metabolites In Pancreatic Physiology And Pathophysiology
Melatonin Controls Cell Proliferation And Modulates Mitochondrial Physiology In Pancreatic Stellate Cells
Membrane Lipid Derivatives: Roles Of Arachidonic Acid And Its Metabolites In Pancreatic Physiology And Pathophysiology
Melatonin Controls Cell Proliferation And Modulates Mitochondrial Physiology In Pancreatic Stellate Cells
Melatonin Modulates Metabolic Adaptation Of Pancreatic Stellate Cells Subjected To Hypoxia
Melatonin Controls Cell Proliferation And Modulates Mitochondrial Physiology In Pancreatic Stellate Cells
Melatonin Modulates Metabolic Adaptation Of Pancreatic Stellate Cells Subjected To Hypoxia
Melatonin Controls Cell Proliferation And Modulates Mitochondrial Physiology In Pancreatic Stellate Cells.
Melatonin Modulates Metabolic Adaptation Of Pancreatic Stellate Cells Subjected To Hypoxia
Melatonin Controls Cell Proliferation And Modulates Mitochondrial Physiology In Pancreatic Stellate Cells.
Melatonin Modulates The Antioxidant Defenses And The Expression Of Proinflammatory Mediators In Pancreatic Stellate Cells Subjected To Hypoxia
Melatonin Controls Cell Proliferation And Modulates Mitochondrial Physiology In Pancreatic Stellate Cells.
Melatonin Modulates The Antioxidant Defenses And The Expression Of Proinflammatory Mediators In Pancreatic Stellate Cells Subjected To Hypoxia
Modulation Of Cell Physiology Under Hypoxia In Pancreatic Cancer
Melatonin Modulates The Antioxidant Defenses And The Expression Of Proinflammatory Mediators In Pancreatic Stellate Cells Subjected To Hypoxia
Modulation Of Cell Physiology Under Hypoxia In Pancreatic Cancer
Melatonin Induces Apoptosis And Modulates Cyclin Expression And Mapk Phosphorylation In Pancreatic Stellate Cells Subjected To Hypoxia
Modulation Of Cell Physiology Under Hypoxia In Pancreatic Cancer
Melatonin Induces Apoptosis And Modulates Cyclin Expression And Mapk Phosphorylation In Pancreatic Stellate Cells Subjected To Hypoxia
The Melatonin Receptor Antagonist Luzindole Induces The Activation Of Cellular Stress Responses And Decreases Viability Of Rat Pancreatic Stellate Cells
Melatonin Induces Apoptosis And Modulates Cyclin Expression And Mapk Phosphorylation In Pancreatic Stellate Cells Subjected To Hypoxia
The Melatonin Receptor Antagonist Luzindole Induces The Activation Of Cellular Stress Responses And Decreases Viability Of Rat Pancreatic Stellate Cells
Melatonin Modulates Proliferation Of Pancreatic Stellate Cells Through Caspase-3 Activation And Changes In Cyclin A And D Expression
The Melatonin Receptor Antagonist Luzindole Induces The Activation Of Cellular Stress Responses And Decreases Viability Of Rat Pancreatic Stellate Cells
Melatonin Modulates Proliferation Of Pancreatic Stellate Cells Through Caspase-3 Activation And Changes In Cyclin A And D Expression
Melatonin Modulates Red-Ox State And Decreases Viability Of Rat Pancreatic Stellate Cells
Melatonin Modulates Proliferation Of Pancreatic Stellate Cells Through Caspase-3 Activation And Changes In Cyclin A And D Expression
Melatonin Modulates Red-Ox State And Decreases Viability Of Rat Pancreatic Stellate Cells
The Lysine Derivative Aminoadipic Acid, A Biomarker Of Protein Oxidation And Diabetes-Risk, Induces Production Of Reactive Oxygen Species And Impairs Trypsin Secretion In Mouse Pancreatic Acinar Cells
Melatonin Modulates Red-Ox State And Decreases Viability Of Rat Pancreatic Stellate Cells
The Lysine Derivative Aminoadipic Acid, A Biomarker Of Protein Oxidation And Diabetes-Risk, Induces Production Of Reactive Oxygen Species And Impairs Trypsin Secretion In Mouse Pancreatic Acinar Cells
Pancreatic Stellate Cells Exhibit Adaptation To Oxidative Stress Evoked By Hypoxia
The Lysine Derivative Aminoadipic Acid, A Biomarker Of Protein Oxidation And Diabetes-Risk, Induces Production Of Reactive Oxygen Species And Impairs Trypsin Secretion In Mouse Pancreatic Acinar Cells
Pancreatic Stellate Cells Exhibit Adaptation To Oxidative Stress Evoked By Hypoxia
The Melatonin Receptor Antagonist Luzindole Induces Ca2+ Mobilization, Reactive Oxygen Species Generation And Impairs Trypsin Secretion In Mouse Pancreatic Acinar Cells
Pancreatic Stellate Cells Exhibit Adaptation To Oxidative Stress Evoked By Hypoxia
The Melatonin Receptor Antagonist Luzindole Induces Ca2+ Mobilization, Reactive Oxygen Species Generation And Impairs Trypsin Secretion In Mouse Pancreatic Acinar Cells
Melatonin Induces Reactive Oxygen Species Generation And Changes In Glutathione Levels And Reduces Viability In Human Pancreatic Stellate Cells
The Melatonin Receptor Antagonist Luzindole Induces Ca2+ Mobilization, Reactive Oxygen Species Generation And Impairs Trypsin Secretion In Mouse Pancreatic Acinar Cells
Melatonin Induces Reactive Oxygen Species Generation And Changes In Glutathione Levels And Reduces Viability In Human Pancreatic Stellate Cells
In Vitro Digestion And Culture In Caco-2 Cells To Assess The Bioavailability Of Fatty Acids: A Case Study In Meat Matrix Enriched With Ω-3 Microcapsules
WEB ACCESIBLE