PUBLICACIÓN

Regulation of activity and localization of the WNK1 protein kinase by hyperosmotic stress

ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Zagórska A. et al.

2007 Journal of Cell Biology

CITAS

170

DOI

10.1083/jcb.200605093

EID

2-s2.0-33845970213

ISSN

0021-9525

EISSN

0021-9525

BIBTEX

@article{Pozo-Guisado2007,title = {Regulation of activity and localization of the WNK1 protein kinase by hyperosmotic stress},journal = {Journal of Cell Biology},year = {2007},volume = {176},number = {1},pages = {89-100},author = {Zag{\'o}rska, A. and Pozo-Guisado, E. and Boudeau, J. and Vitari, A.C. and Rafiqi, F.H. and Thastrup, J. and Deak, M. and Campbell, D.G. and Morrice, N.A. and Prescott, A.R. and Alessi, D.R.}}

AUTORES DE LA UEX

Eulalia Pozo Guisado

OTRAS PUBLICACIONES DE INTERÉS

Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
Stim1 Deficiency Leads To Specific Down-Regulation Of Itpr3 In Sh-Sy5y Cells
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
Stim1 Deficiency Leads To Specific Down-Regulation Of Itpr3 In Sh-Sy5y Cells
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Stim1 Deficiency Leads To Specific Down-Regulation Of Itpr3 In Sh-Sy5y Cells
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Role Of Stim1 In Neurodegeneration
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Role Of Stim1 In Neurodegeneration
Regulation Of Calcium Signaling By Stim1 And Orai1
Role Of Stim1 In Neurodegeneration
Regulation Of Calcium Signaling By Stim1 And Orai1
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Regulation Of Calcium Signaling By Stim1 And Orai1
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
The Interplay Between Cytoskeleton And Calcium Dynamics
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
The Interplay Between Cytoskeleton And Calcium Dynamics
Store-Operated Calcium Entry Is Dispensable For The Activation Of Erk1/2 Pathway In Prostate Cancer Cells
The Interplay Between Cytoskeleton And Calcium Dynamics
Store-Operated Calcium Entry Is Dispensable For The Activation Of Erk1/2 Pathway In Prostate Cancer Cells
Regulation Of Membrane Ruffling By Polarized Stim1 And Orai1 In Cortactin-Rich Domains
Store-Operated Calcium Entry Is Dispensable For The Activation Of Erk1/2 Pathway In Prostate Cancer Cells
Regulation Of Membrane Ruffling By Polarized Stim1 And Orai1 In Cortactin-Rich Domains
Phospho-Stim1 Is A Downstream Effector That Mediates The Signaling Triggered By Igf-1 In Hek293 Cells
Regulation Of Membrane Ruffling By Polarized Stim1 And Orai1 In Cortactin-Rich Domains
Phospho-Stim1 Is A Downstream Effector That Mediates The Signaling Triggered By Igf-1 In Hek293 Cells
Stim1 Phosphorylation Triggered By Epidermal Growth Factor Mediates Cell Migration
Phospho-Stim1 Is A Downstream Effector That Mediates The Signaling Triggered By Igf-1 In Hek293 Cells
Stim1 Phosphorylation Triggered By Epidermal Growth Factor Mediates Cell Migration
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
Stim1 Phosphorylation Triggered By Epidermal Growth Factor Mediates Cell Migration
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Regulation Of Stim1 By Phosphorylation
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Regulation Of Stim1 By Phosphorylation
Inhibition Of Stim1 Phosphorylation Underlies Resveratrol-Induced Inhibition Of Store-Operated Calcium Entry
The Regulation Of Stim1 By Phosphorylation
Inhibition Of Stim1 Phosphorylation Underlies Resveratrol-Induced Inhibition Of Store-Operated Calcium Entry
Extended Embryo Culture Supplementation
Inhibition Of Stim1 Phosphorylation Underlies Resveratrol-Induced Inhibition Of Store-Operated Calcium Entry
Extended Embryo Culture Supplementation
Calcium Signaling In Mouse Oocyte Maturation: The Roles Of Stim1, Orai1 And Soce
Extended Embryo Culture Supplementation
Calcium Signaling In Mouse Oocyte Maturation: The Roles Of Stim1, Orai1 And Soce
Role Of Store-Operated Calcium Entry During Meiotic Progression And Fertilization Of Mammalian Oocytes
Calcium Signaling In Mouse Oocyte Maturation: The Roles Of Stim1, Orai1 And Soce
Role Of Store-Operated Calcium Entry During Meiotic Progression And Fertilization Of Mammalian Oocytes
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Role Of Store-Operated Calcium Entry During Meiotic Progression And Fertilization Of Mammalian Oocytes
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Regulation Of Cell Survival By Resveratrol Involves Inhibition Of Nfκb-Regulated Gene Expression In Prostate Cancer Cells
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Regulation Of Cell Survival By Resveratrol Involves Inhibition Of Nfκb-Regulated Gene Expression In Prostate Cancer Cells
Relocalization Of Stim1 In Mouse Oocytes At Fertilization: Early Involvement Of Store-Operated Calcium Entry
Regulation Of Cell Survival By Resveratrol Involves Inhibition Of Nfκb-Regulated Gene Expression In Prostate Cancer Cells
Relocalization Of Stim1 In Mouse Oocytes At Fertilization: Early Involvement Of Store-Operated Calcium Entry
Non-Genomic Action Of Resveratrol On Androgen And Oestrogen Receptors In Prostate Cancer: Modulation Of The Phosphoinositide 3-Kinase Pathway
Relocalization Of Stim1 In Mouse Oocytes At Fertilization: Early Involvement Of Store-Operated Calcium Entry
Non-Genomic Action Of Resveratrol On Androgen And Oestrogen Receptors In Prostate Cancer: Modulation Of The Phosphoinositide 3-Kinase Pathway
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
Non-Genomic Action Of Resveratrol On Androgen And Oestrogen Receptors In Prostate Cancer: Modulation Of The Phosphoinositide 3-Kinase Pathway
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
Regulation Of Activity And Localization Of The Wnk1 Protein Kinase By Hyperosmotic Stress
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
Regulation Of Activity And Localization Of The Wnk1 Protein Kinase By Hyperosmotic Stress
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Regulation Of Activity And Localization Of The Wnk1 Protein Kinase By Hyperosmotic Stress
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Development Of A Pcr-Based Strategy For Cyp2d6 Genotyping Including Gene Multiplication Of Worldwide Potential Use
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Development Of A Pcr-Based Strategy For Cyp2d6 Genotyping Including Gene Multiplication Of Worldwide Potential Use
Resveratrol Modulates The Phosphoinositide 3-Kinase Pathway Through An Estrogen Receptor Α-Dependent Mechanism: Relevance In Cell Proliferation
Development Of A Pcr-Based Strategy For Cyp2d6 Genotyping Including Gene Multiplication Of Worldwide Potential Use
Resveratrol Modulates The Phosphoinositide 3-Kinase Pathway Through An Estrogen Receptor Α-Dependent Mechanism: Relevance In Cell Proliferation
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Resveratrol Modulates The Phosphoinositide 3-Kinase Pathway Through An Estrogen Receptor Α-Dependent Mechanism: Relevance In Cell Proliferation
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
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