PUBLICACIÓN

Resveratrol modulates the phosphoinositide 3-kinase pathway through an estrogen receptor α-dependent mechanism: Relevance in cell proliferation

Logo Open Access

ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Pozo-Guisado, E, Lorenzo-Benayas, MJ, Fernandez-Salguero, PM

2004 International Journal of Cancer

CITAS

143

DOI

10.1002/ijc.11720

EID

2-s2.0-1242342067

ISSN

0020-7136

BIBTEX

@article{RID:0916141122194-29, title = {Resveratrol modulates the phosphoinositide 3-kinase pathway through an estrogen receptor alpha-dependent mechanism: Relevance in cell proliferation}, journal = {International Journal of Cancer}, year = {2004}, author = {Pozo-Guisado, E and Lorenzo-Benayas, MJ and Fernandez-Salguero, PM}, volume = {109}, number = {2}, pages = {167-173} }

AUTORES DE LA UEX

Pedro María Fernández Salguero
Eulalia Pozo Guisado
María Jesús Lorenzo Benayas

OTRAS PUBLICACIONES DE INTERÉS

Ileal Vasoactive Intestinal Peptide (Vip) Levels And Vip Receptor/effector System In Lleal Epithelial Cells After Colectomy In The Rat
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment
Introducción: Carta Del Rector De La Uex
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment
Introducción: Carta Del Rector De La Uex
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
Introducción: Carta Del Rector De La Uex
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
The Ah Receptor From Toxicity To Therapeutics: Report From The 5th Ahr Meeting At Penn State University, Usa, June 2022
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
The Ah Receptor From Toxicity To Therapeutics: Report From The 5th Ahr Meeting At Penn State University, Usa, June 2022
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
The Ah Receptor From Toxicity To Therapeutics: Report From The 5th Ahr Meeting At Penn State University, Usa, June 2022
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Characterization Of Autochthonal Hafnia Spp. Strains Isolated From Spanish Soft Raw Ewe's Milk Pdo Cheeses To Be Used As Adjunct Culture
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Characterization Of Autochthonal Hafnia Spp. Strains Isolated From Spanish Soft Raw Ewe's Milk Pdo Cheeses To Be Used As Adjunct Culture
Title: P38δ Regulates Il6 Expression Modulating Erk Phosphorylation In Preadipocytes
Characterization Of Autochthonal Hafnia Spp. Strains Isolated From Spanish Soft Raw Ewe's Milk Pdo Cheeses To Be Used As Adjunct Culture
Title: P38δ Regulates Il6 Expression Modulating Erk Phosphorylation In Preadipocytes
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Title: P38δ Regulates Il6 Expression Modulating Erk Phosphorylation In Preadipocytes
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function
P53 Regulation By Mdm2 Contributes To Self-Renewal And Differentiation Of Basal Stem Cells In Mouse And Human Airway Epithelium
Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function
P53 Regulation By Mdm2 Contributes To Self-Renewal And Differentiation Of Basal Stem Cells In Mouse And Human Airway Epithelium
Stim1 Deficiency Leads To Specific Down-Regulation Of Itpr3 In Sh-Sy5y Cells
P53 Regulation By Mdm2 Contributes To Self-Renewal And Differentiation Of Basal Stem Cells In Mouse And Human Airway Epithelium
Stim1 Deficiency Leads To Specific Down-Regulation Of Itpr3 In Sh-Sy5y Cells
Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor
Stim1 Deficiency Leads To Specific Down-Regulation Of Itpr3 In Sh-Sy5y Cells
Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Lack Of The Aryl Hydrocarbon Receptor Accelerates Aging In Mice
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Lack Of The Aryl Hydrocarbon Receptor Accelerates Aging In Mice
Vav Proteins Maintain Epithelial Traits In Breast Cancer Cells Using Mir-200c-Dependent And Independent Mechanisms
Lack Of The Aryl Hydrocarbon Receptor Accelerates Aging In Mice
Vav Proteins Maintain Epithelial Traits In Breast Cancer Cells Using Mir-200c-Dependent And Independent Mechanisms
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Vav Proteins Maintain Epithelial Traits In Breast Cancer Cells Using Mir-200c-Dependent And Independent Mechanisms
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Role Of Stim1 In Neurodegeneration
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Role Of Stim1 In Neurodegeneration
Regulation Of Calcium Signaling By Stim1 And Orai1
Role Of Stim1 In Neurodegeneration
Regulation Of Calcium Signaling By Stim1 And Orai1
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Regulation Of Calcium Signaling By Stim1 And Orai1
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value
Identification Of New Activators Of Mitochondrial Fusion Reveals A Link Between Mitochondrial Morphology And Pyrimidine Metabolism
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value
Identification Of New Activators Of Mitochondrial Fusion Reveals A Link Between Mitochondrial Morphology And Pyrimidine Metabolism
Histone H4 Acetylation Regulates Behavioral Inter-Individual Variability In Zebrafish
Identification Of New Activators Of Mitochondrial Fusion Reveals A Link Between Mitochondrial Morphology And Pyrimidine Metabolism
Histone H4 Acetylation Regulates Behavioral Inter-Individual Variability In Zebrafish
The Interplay Between Cytoskeleton And Calcium Dynamics
Histone H4 Acetylation Regulates Behavioral Inter-Individual Variability In Zebrafish
The Interplay Between Cytoskeleton And Calcium Dynamics
Store-Operated Calcium Entry Is Dispensable For The Activation Of Erk1/2 Pathway In Prostate Cancer Cells
The Interplay Between Cytoskeleton And Calcium Dynamics
Store-Operated Calcium Entry Is Dispensable For The Activation Of Erk1/2 Pathway In Prostate Cancer Cells
Jnk Signaling Pathway Regulates Sorbitol-Induced Tau Proteolysis And Apoptosis In Sh-Sy5y Cells By Targeting Caspase-3
Store-Operated Calcium Entry Is Dispensable For The Activation Of Erk1/2 Pathway In Prostate Cancer Cells
Jnk Signaling Pathway Regulates Sorbitol-Induced Tau Proteolysis And Apoptosis In Sh-Sy5y Cells By Targeting Caspase-3
Regulation Of Membrane Ruffling By Polarized Stim1 And Orai1 In Cortactin-Rich Domains
Jnk Signaling Pathway Regulates Sorbitol-Induced Tau Proteolysis And Apoptosis In Sh-Sy5y Cells By Targeting Caspase-3
Regulation Of Membrane Ruffling By Polarized Stim1 And Orai1 In Cortactin-Rich Domains
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Regulation Of Membrane Ruffling By Polarized Stim1 And Orai1 In Cortactin-Rich Domains
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis
A Dioxin-Like Compound Induces Hyperplasia And Branching Morphogenesis In Mouse Mammary Gland, Through Alterations In Tgf-Β1 And Aryl Hydrocarbon Receptor Signaling
Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis
A Dioxin-Like Compound Induces Hyperplasia And Branching Morphogenesis In Mouse Mammary Gland, Through Alterations In Tgf-Β1 And Aryl Hydrocarbon Receptor Signaling
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis.
A Dioxin-Like Compound Induces Hyperplasia And Branching Morphogenesis In Mouse Mammary Gland, Through Alterations In Tgf-Β1 And Aryl Hydrocarbon Receptor Signaling
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis.
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor.
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis.
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor.
New Trends In Aryl Hydrocarbon Receptor Biology.
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor.
New Trends In Aryl Hydrocarbon Receptor Biology.
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements.
New Trends In Aryl Hydrocarbon Receptor Biology.
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements.
Hyperosmotic Stress Induces Tau Proteolysis By Caspase-3 Activation In Sh-Sy5y Cells
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements.
Hyperosmotic Stress Induces Tau Proteolysis By Caspase-3 Activation In Sh-Sy5y Cells
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
Hyperosmotic Stress Induces Tau Proteolysis By Caspase-3 Activation In Sh-Sy5y Cells
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis
New Trends In Aryl Hydrocarbon Receptor Biology
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis
New Trends In Aryl Hydrocarbon Receptor Biology
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements
New Trends In Aryl Hydrocarbon Receptor Biology
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements
Phospho-Stim1 Is A Downstream Effector That Mediates The Signaling Triggered By Igf-1 In Hek293 Cells
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements
Phospho-Stim1 Is A Downstream Effector That Mediates The Signaling Triggered By Igf-1 In Hek293 Cells
Stim1 Phosphorylation Triggered By Epidermal Growth Factor Mediates Cell Migration
Phospho-Stim1 Is A Downstream Effector That Mediates The Signaling Triggered By Igf-1 In Hek293 Cells
Stim1 Phosphorylation Triggered By Epidermal Growth Factor Mediates Cell Migration
Skin Response To A Carcinogen Involves The Xenobiotic Receptor Pregnane X Receptor
Stim1 Phosphorylation Triggered By Epidermal Growth Factor Mediates Cell Migration
Skin Response To A Carcinogen Involves The Xenobiotic Receptor Pregnane X Receptor
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
Skin Response To A Carcinogen Involves The Xenobiotic Receptor Pregnane X Receptor
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells
Bmi1 Regulates Murine Intestinal Stem Cell Proliferation And Self-Renewal Downstream Of Notch
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells
Bmi1 Regulates Murine Intestinal Stem Cell Proliferation And Self-Renewal Downstream Of Notch
Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg
Bmi1 Regulates Murine Intestinal Stem Cell Proliferation And Self-Renewal Downstream Of Notch
Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg
L-Kynurenine/aryl Hydrocarbon Receptor Pathway Mediates Brain Damage After Experimental Stroke
Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg
L-Kynurenine/aryl Hydrocarbon Receptor Pathway Mediates Brain Damage After Experimental Stroke
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol
L-Kynurenine/aryl Hydrocarbon Receptor Pathway Mediates Brain Damage After Experimental Stroke
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol
Aryl Hydrocarbon Receptor-Dependent Induction Of Liver Fibrosis By Dioxin
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol
Aryl Hydrocarbon Receptor-Dependent Induction Of Liver Fibrosis By Dioxin
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
Aryl Hydrocarbon Receptor-Dependent Induction Of Liver Fibrosis By Dioxin
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis
Aryl Hydrocarbon Receptor Contributes To The Mek/erk-Dependent Maintenance Of The Immature State Of Human Dendritic Cells
The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis
Aryl Hydrocarbon Receptor Contributes To The Mek/erk-Dependent Maintenance Of The Immature State Of Human Dendritic Cells
The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway
Aryl Hydrocarbon Receptor Contributes To The Mek/erk-Dependent Maintenance Of The Immature State Of Human Dendritic Cells
The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway
Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition
The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway
Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition
Oculomotor Deficits In Aryl Hydrocarbon Receptor Null Mouse
Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition
Oculomotor Deficits In Aryl Hydrocarbon Receptor Null Mouse
The Regulation Of Stim1 By Phosphorylation
Oculomotor Deficits In Aryl Hydrocarbon Receptor Null Mouse
The Regulation Of Stim1 By Phosphorylation
Inhibition Of Stim1 Phosphorylation Underlies Resveratrol-Induced Inhibition Of Store-Operated Calcium Entry
The Regulation Of Stim1 By Phosphorylation
Inhibition Of Stim1 Phosphorylation Underlies Resveratrol-Induced Inhibition Of Store-Operated Calcium Entry
Aryl Hydrocarbon Receptor-Induced Adrenomedullin Mediates Cigarette Smoke Carcinogenicity In Humans And Mice
Inhibition Of Stim1 Phosphorylation Underlies Resveratrol-Induced Inhibition Of Store-Operated Calcium Entry
Aryl Hydrocarbon Receptor-Induced Adrenomedullin Mediates Cigarette Smoke Carcinogenicity In Humans And Mice
Extended Embryo Culture Supplementation
Aryl Hydrocarbon Receptor-Induced Adrenomedullin Mediates Cigarette Smoke Carcinogenicity In Humans And Mice
Extended Embryo Culture Supplementation
Calcium Signaling In Mouse Oocyte Maturation: The Roles Of Stim1, Orai1 And Soce
Extended Embryo Culture Supplementation
Calcium Signaling In Mouse Oocyte Maturation: The Roles Of Stim1, Orai1 And Soce
Role Of Store-Operated Calcium Entry During Meiotic Progression And Fertilization Of Mammalian Oocytes
Calcium Signaling In Mouse Oocyte Maturation: The Roles Of Stim1, Orai1 And Soce
Role Of Store-Operated Calcium Entry During Meiotic Progression And Fertilization Of Mammalian Oocytes
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Role Of Store-Operated Calcium Entry During Meiotic Progression And Fertilization Of Mammalian Oocytes
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Antimicrobial Resistance Determinants Among Anaerobic Bacteria Isolated From Footrot
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Antimicrobial Resistance Determinants Among Anaerobic Bacteria Isolated From Footrot
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Antimicrobial Resistance Determinants Among Anaerobic Bacteria Isolated From Footrot
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Cómo Se Vive Con Un Artista
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Cómo Se Vive Con Un Artista
Dioxin Receptor And Slug Transcription Factors Regulate The Insulator Activity Of B1 Sine Retrotransposons Via An Rna Polymerase Switch
Cómo Se Vive Con Un Artista
Dioxin Receptor And Slug Transcription Factors Regulate The Insulator Activity Of B1 Sine Retrotransposons Via An Rna Polymerase Switch
The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling
Dioxin Receptor And Slug Transcription Factors Regulate The Insulator Activity Of B1 Sine Retrotransposons Via An Rna Polymerase Switch
The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling
Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse
The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling
Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse
Transcriptional Factor Aryl Hydrocarbon Receptor (Ahr) Controls Cardiovascular And Respiratory Functions By Regulating The Expression Of The Vav3 Proto-Oncogene
Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse
Transcriptional Factor Aryl Hydrocarbon Receptor (Ahr) Controls Cardiovascular And Respiratory Functions By Regulating The Expression Of The Vav3 Proto-Oncogene
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells
Transcriptional Factor Aryl Hydrocarbon Receptor (Ahr) Controls Cardiovascular And Respiratory Functions By Regulating The Expression Of The Vav3 Proto-Oncogene
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells
A Remarkable New Target Gene For The Dioxin Receptor: The Vav3 Proto-Oncogene Links Ahr To Adhesion And Migration
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells
A Remarkable New Target Gene For The Dioxin Receptor: The Vav3 Proto-Oncogene Links Ahr To Adhesion And Migration
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
A Remarkable New Target Gene For The Dioxin Receptor: The Vav3 Proto-Oncogene Links Ahr To Adhesion And Migration
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Role Of Transforming Growth Factor Β In Cancer Microenvironment
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Role Of Transforming Growth Factor Β In Cancer Microenvironment
Dioxin Receptor Deficiency Impairs Angiogenesis By A Mechanism Involving Vegf-A Depletion In The Endothelium And Transforming Growth Factor-Β Overexpression In The Stroma
Role Of Transforming Growth Factor Β In Cancer Microenvironment
Dioxin Receptor Deficiency Impairs Angiogenesis By A Mechanism Involving Vegf-A Depletion In The Endothelium And Transforming Growth Factor-Β Overexpression In The Stroma
Regulation Of Cell Survival By Resveratrol Involves Inhibition Of Nfκb-Regulated Gene Expression In Prostate Cancer Cells
Dioxin Receptor Deficiency Impairs Angiogenesis By A Mechanism Involving Vegf-A Depletion In The Endothelium And Transforming Growth Factor-Β Overexpression In The Stroma
Regulation Of Cell Survival By Resveratrol Involves Inhibition Of Nfκb-Regulated Gene Expression In Prostate Cancer Cells
The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion
Regulation Of Cell Survival By Resveratrol Involves Inhibition Of Nfκb-Regulated Gene Expression In Prostate Cancer Cells
The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion
Fitting A Xenobiotic Receptor Into Cell Homeostasis: How The Dioxin Receptor Interacts With Tgfβ Signaling
The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion
Fitting A Xenobiotic Receptor Into Cell Homeostasis: How The Dioxin Receptor Interacts With Tgfβ Signaling
Relocalization Of Stim1 In Mouse Oocytes At Fertilization: Early Involvement Of Store-Operated Calcium Entry
Fitting A Xenobiotic Receptor Into Cell Homeostasis: How The Dioxin Receptor Interacts With Tgfβ Signaling
Relocalization Of Stim1 In Mouse Oocytes At Fertilization: Early Involvement Of Store-Operated Calcium Entry
Gene Context And Dna Rearrangements In The Carbapenemase Locus Of Division Ii Strains Of Bacteroides Fragilis
Relocalization Of Stim1 In Mouse Oocytes At Fertilization: Early Involvement Of Store-Operated Calcium Entry
Gene Context And Dna Rearrangements In The Carbapenemase Locus Of Division Ii Strains Of Bacteroides Fragilis
Loss Of Dioxin-Receptor Expression Accelerates Wound Healing In Vivo By A Mechanism Involving Tgfβ
Gene Context And Dna Rearrangements In The Carbapenemase Locus Of Division Ii Strains Of Bacteroides Fragilis
Loss Of Dioxin-Receptor Expression Accelerates Wound Healing In Vivo By A Mechanism Involving Tgfβ
Recruitment Of Creb1 And Histone Deacetylase 2 (Hdac2) To The Mouse Ltbp-1 Promoter Regulates Its Constitutive Expression In A Dioxin Receptor-Dependent Manner
Loss Of Dioxin-Receptor Expression Accelerates Wound Healing In Vivo By A Mechanism Involving Tgfβ
Recruitment Of Creb1 And Histone Deacetylase 2 (Hdac2) To The Mouse Ltbp-1 Promoter Regulates Its Constitutive Expression In A Dioxin Receptor-Dependent Manner
Genome-Wide B1 Retrotransposon Binds The Transcription Factors Dioxin Receptor And Slug And Regulates Gene Expression In Vivo
Recruitment Of Creb1 And Histone Deacetylase 2 (Hdac2) To The Mouse Ltbp-1 Promoter Regulates Its Constitutive Expression In A Dioxin Receptor-Dependent Manner
Genome-Wide B1 Retrotransposon Binds The Transcription Factors Dioxin Receptor And Slug And Regulates Gene Expression In Vivo
Genetic Determinants For Cfxa Expression In Bacteroides Strains Isolated From Human Infections
Genome-Wide B1 Retrotransposon Binds The Transcription Factors Dioxin Receptor And Slug And Regulates Gene Expression In Vivo
Genetic Determinants For Cfxa Expression In Bacteroides Strains Isolated From Human Infections
Non-Genomic Action Of Resveratrol On Androgen And Oestrogen Receptors In Prostate Cancer: Modulation Of The Phosphoinositide 3-Kinase Pathway
Genetic Determinants For Cfxa Expression In Bacteroides Strains Isolated From Human Infections
Non-Genomic Action Of Resveratrol On Androgen And Oestrogen Receptors In Prostate Cancer: Modulation Of The Phosphoinositide 3-Kinase Pathway
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
Non-Genomic Action Of Resveratrol On Androgen And Oestrogen Receptors In Prostate Cancer: Modulation Of The Phosphoinositide 3-Kinase Pathway
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
The Aryl Hydrocarbon Receptor, More Than A Xenobiotic-Interacting Protein
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
The Aryl Hydrocarbon Receptor, More Than A Xenobiotic-Interacting Protein
C-Jun N-Terminal Protein Kinase Signalling Pathway Mediates Lovastatin-Induced Rat Brain Neuroblast Apoptosis
The Aryl Hydrocarbon Receptor, More Than A Xenobiotic-Interacting Protein
C-Jun N-Terminal Protein Kinase Signalling Pathway Mediates Lovastatin-Induced Rat Brain Neuroblast Apoptosis
Lovastatin Inhibits The Extracellular-Signal-Regulated Kinase Pathway In Immortalized Rat Brain Neuroblasts
C-Jun N-Terminal Protein Kinase Signalling Pathway Mediates Lovastatin-Induced Rat Brain Neuroblast Apoptosis
Lovastatin Inhibits The Extracellular-Signal-Regulated Kinase Pathway In Immortalized Rat Brain Neuroblasts
Regulation Of Activity And Localization Of The Wnk1 Protein Kinase By Hyperosmotic Stress
Lovastatin Inhibits The Extracellular-Signal-Regulated Kinase Pathway In Immortalized Rat Brain Neuroblasts
Regulation Of Activity And Localization Of The Wnk1 Protein Kinase By Hyperosmotic Stress
Cck1 And 2 Receptors Are Expressed In Immortalized Rat Brain Neuroblasts: Intracellular Signals After Cholecystokinin Stimulation
Regulation Of Activity And Localization Of The Wnk1 Protein Kinase By Hyperosmotic Stress
Cck1 And 2 Receptors Are Expressed In Immortalized Rat Brain Neuroblasts: Intracellular Signals After Cholecystokinin Stimulation
Cleavage Of Focal Adhesion Proteins And Pkcδ During Lovastatin-Induced Apoptosis In Spontaneously Immortalized Rat Brain Neuroblasts
Cck1 And 2 Receptors Are Expressed In Immortalized Rat Brain Neuroblasts: Intracellular Signals After Cholecystokinin Stimulation
Cleavage Of Focal Adhesion Proteins And Pkcδ During Lovastatin-Induced Apoptosis In Spontaneously Immortalized Rat Brain Neuroblasts
The Dioxin Receptor Is Silenced By Promoter Hypermethylation In Human Acute Lymphoblastic Leukemia Through Inhibition Of Sp1 Binding
Cleavage Of Focal Adhesion Proteins And Pkcδ During Lovastatin-Induced Apoptosis In Spontaneously Immortalized Rat Brain Neuroblasts
The Dioxin Receptor Is Silenced By Promoter Hypermethylation In Human Acute Lymphoblastic Leukemia Through Inhibition Of Sp1 Binding
Ltbp-1 Blockade In Dioxin Receptor-Null Mouse Embryo Fibroblasts Decreases Tgf-Β Activity: Role Of Extracellular Proteases Plasmin And Elastase
The Dioxin Receptor Is Silenced By Promoter Hypermethylation In Human Acute Lymphoblastic Leukemia Through Inhibition Of Sp1 Binding
Ltbp-1 Blockade In Dioxin Receptor-Null Mouse Embryo Fibroblasts Decreases Tgf-Β Activity: Role Of Extracellular Proteases Plasmin And Elastase
Improving Cancer Therapeutics By Molecular Profiling
Ltbp-1 Blockade In Dioxin Receptor-Null Mouse Embryo Fibroblasts Decreases Tgf-Β Activity: Role Of Extracellular Proteases Plasmin And Elastase
Improving Cancer Therapeutics By Molecular Profiling
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Improving Cancer Therapeutics By Molecular Profiling
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Lovastatin Inhibits The Growth And Survival Pathway Of Phosphoinositide 3-Kinase/protein Kinase B In Immortalized Rat Brain Neuroblasts
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Lovastatin Inhibits The Growth And Survival Pathway Of Phosphoinositide 3-Kinase/protein Kinase B In Immortalized Rat Brain Neuroblasts
Development Of A Pcr-Based Strategy For Cyp2d6 Genotyping Including Gene Multiplication Of Worldwide Potential Use
Lovastatin Inhibits The Growth And Survival Pathway Of Phosphoinositide 3-Kinase/protein Kinase B In Immortalized Rat Brain Neuroblasts
Development Of A Pcr-Based Strategy For Cyp2d6 Genotyping Including Gene Multiplication Of Worldwide Potential Use
Resveratrol Modulates The Phosphoinositide 3-Kinase Pathway Through An Estrogen Receptor Α-Dependent Mechanism: Relevance In Cell Proliferation
Development Of A Pcr-Based Strategy For Cyp2d6 Genotyping Including Gene Multiplication Of Worldwide Potential Use
Resveratrol Modulates The Phosphoinositide 3-Kinase Pathway Through An Estrogen Receptor Α-Dependent Mechanism: Relevance In Cell Proliferation
Overexpression Of Latent Transforming Growth Factor-Β Binding Protein 1 (Ltbp-1) In Dioxin Receptor-Null Mouse Embryo Fibroblasts
Resveratrol Modulates The Phosphoinositide 3-Kinase Pathway Through An Estrogen Receptor Α-Dependent Mechanism: Relevance In Cell Proliferation
Overexpression Of Latent Transforming Growth Factor-Β Binding Protein 1 (Ltbp-1) In Dioxin Receptor-Null Mouse Embryo Fibroblasts
Liver Portal Fibrosis In Dioxin Receptor-Null Mice That Overexpress The Latent Transforming Growth Factor-Β-Binding Protein-1
Overexpression Of Latent Transforming Growth Factor-Β Binding Protein 1 (Ltbp-1) In Dioxin Receptor-Null Mouse Embryo Fibroblasts
Liver Portal Fibrosis In Dioxin Receptor-Null Mice That Overexpress The Latent Transforming Growth Factor-Β-Binding Protein-1
Proteasome Inhibition Induces Nuclear Translocation Of The Dioxin Receptor Through An Sp1 And Protein Kinase C-Dependent Pathway
Liver Portal Fibrosis In Dioxin Receptor-Null Mice That Overexpress The Latent Transforming Growth Factor-Β-Binding Protein-1
Proteasome Inhibition Induces Nuclear Translocation Of The Dioxin Receptor Through An Sp1 And Protein Kinase C-Dependent Pathway
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Proteasome Inhibition Induces Nuclear Translocation Of The Dioxin Receptor Through An Sp1 And Protein Kinase C-Dependent Pathway
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Thioridazine Steady-State Plasma Concentrations Are Influenced By Tobacco Smoking And Cyp2d6, But Not By The Cyp2c9 Genotype
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Thioridazine Steady-State Plasma Concentrations Are Influenced By Tobacco Smoking And Cyp2d6, But Not By The Cyp2c9 Genotype
Carcinogenesis Of The Food Mutagen Phip In Mice Is Dependent Of Cyp1a2
Thioridazine Steady-State Plasma Concentrations Are Influenced By Tobacco Smoking And Cyp2d6, But Not By The Cyp2c9 Genotype
Carcinogenesis Of The Food Mutagen Phip In Mice Is Dependent Of Cyp1a2
Polycyclic Aromatic Hydrocarbon-Inducible Dna Adducts: Evidence By 32p-Postlabeling And Use Of Knockout Mice For Ah Receptor-Independent Mechanisms Of Metabolic Activation In Vivo
Carcinogenesis Of The Food Mutagen Phip In Mice Is Dependent Of Cyp1a2
Polycyclic Aromatic Hydrocarbon-Inducible Dna Adducts: Evidence By 32p-Postlabeling And Use Of Knockout Mice For Ah Receptor-Independent Mechanisms Of Metabolic Activation In Vivo
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
Polycyclic Aromatic Hydrocarbon-Inducible Dna Adducts: Evidence By 32p-Postlabeling And Use Of Knockout Mice For Ah Receptor-Independent Mechanisms Of Metabolic Activation In Vivo
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
Effect Of Thioridazine Dosage On The Debrisoquine Hydroxylation Phenotype In Psychiatric Patients With Different Cyp2d6 Genotypes
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
Effect Of Thioridazine Dosage On The Debrisoquine Hydroxylation Phenotype In Psychiatric Patients With Different Cyp2d6 Genotypes
Targeted Genomic Disruption Of H-Ras And N-Ras, Individually Or In Combination, Reveals The Dispensability Of Both Loci For Mouse Growth And Development
Effect Of Thioridazine Dosage On The Debrisoquine Hydroxylation Phenotype In Psychiatric Patients With Different Cyp2d6 Genotypes
Targeted Genomic Disruption Of H-Ras And N-Ras, Individually Or In Combination, Reveals The Dispensability Of Both Loci For Mouse Growth And Development
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
Targeted Genomic Disruption Of H-Ras And N-Ras, Individually Or In Combination, Reveals The Dispensability Of Both Loci For Mouse Growth And Development
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
Lovastatin Induces Apoptosis Of Spontaneously Immortalized Rat Brain Neuroblasts: Involvement Of Nonsterol Isoprenoid Biosynthesis Inhibition
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
Lovastatin Induces Apoptosis Of Spontaneously Immortalized Rat Brain Neuroblasts: Involvement Of Nonsterol Isoprenoid Biosynthesis Inhibition
Altered Cell Cycle Control At The G2/m Phases In Aryl Hydrocarbon Receptor-Null Embryo Fibroblast
Lovastatin Induces Apoptosis Of Spontaneously Immortalized Rat Brain Neuroblasts: Involvement Of Nonsterol Isoprenoid Biosynthesis Inhibition
Altered Cell Cycle Control At The G2/m Phases In Aryl Hydrocarbon Receptor-Null Embryo Fibroblast
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Altered Cell Cycle Control At The G2/m Phases In Aryl Hydrocarbon Receptor-Null Embryo Fibroblast
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Hepatic Fibrosis And Cytochrome P450: Experimental Models Of Fibrosis Compared To Ahr Knockout Mice
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Hepatic Fibrosis And Cytochrome P450: Experimental Models Of Fibrosis Compared To Ahr Knockout Mice
Exocytotic Protein Components In Rat Pituitary Gland After Long-Term Estrogen Administration
Hepatic Fibrosis And Cytochrome P450: Experimental Models Of Fibrosis Compared To Ahr Knockout Mice
Exocytotic Protein Components In Rat Pituitary Gland After Long-Term Estrogen Administration
Genomic Instability In Gadd45a-Deficient Mice
Exocytotic Protein Components In Rat Pituitary Gland After Long-Term Estrogen Administration
Genomic Instability In Gadd45a-Deficient Mice
Cyp1a2 Is Not The Primary Enzyme Responsible For 4-Aminobiphenyl-Induced Hepatocarcinogenesis In Mice
Genomic Instability In Gadd45a-Deficient Mice
Cyp1a2 Is Not The Primary Enzyme Responsible For 4-Aminobiphenyl-Induced Hepatocarcinogenesis In Mice
Targeted Disruption Of The Microsomal Epoxide Hydrolase Gene. Microsomal Epoxide Hydrolase Is Required For The Carcinogenic Activity Of 7,12- Dimethylbenz[A]anthracene
Cyp1a2 Is Not The Primary Enzyme Responsible For 4-Aminobiphenyl-Induced Hepatocarcinogenesis In Mice
Targeted Disruption Of The Microsomal Epoxide Hydrolase Gene. Microsomal Epoxide Hydrolase Is Required For The Carcinogenic Activity Of 7,12- Dimethylbenz[A]anthracene
Expression Of Cyp2a Genes In Human Liver And Extrahepatic Tissues
Targeted Disruption Of The Microsomal Epoxide Hydrolase Gene. Microsomal Epoxide Hydrolase Is Required For The Carcinogenic Activity Of 7,12- Dimethylbenz[A]anthracene
Expression Of Cyp2a Genes In Human Liver And Extrahepatic Tissues
Amelioration Of Tcdd-Induced Teratogenesis In Aryl Hydrocarbon Receptor (Ahr)-Null Mice
Expression Of Cyp2a Genes In Human Liver And Extrahepatic Tissues
Amelioration Of Tcdd-Induced Teratogenesis In Aryl Hydrocarbon Receptor (Ahr)-Null Mice
Direct Action Of Serotonin On Prolactin, Growth Hormone, Corticotropin And Luteinizing Hormone Release In Cocultures Of Anterior And Posterior Pituitary Lobes: Autocrine And/or Paracrine Action Of Vasoactive Intestinal Peptide
Amelioration Of Tcdd-Induced Teratogenesis In Aryl Hydrocarbon Receptor (Ahr)-Null Mice
Direct Action Of Serotonin On Prolactin, Growth Hormone, Corticotropin And Luteinizing Hormone Release In Cocultures Of Anterior And Posterior Pituitary Lobes: Autocrine And/or Paracrine Action Of Vasoactive Intestinal Peptide
The Aryl Hydrocarbon Receptor. Studies Using The Ahr-Null Mice
Direct Action Of Serotonin On Prolactin, Growth Hormone, Corticotropin And Luteinizing Hormone Release In Cocultures Of Anterior And Posterior Pituitary Lobes: Autocrine And/or Paracrine Action Of Vasoactive Intestinal Peptide
The Aryl Hydrocarbon Receptor. Studies Using The Ahr-Null Mice
Aryl-Hydrocarbon Receptor Is An Inhibitory Regulator Of Lipid Synthesis And Of Commitment To Adipogenesis
The Aryl Hydrocarbon Receptor. Studies Using The Ahr-Null Mice
Aryl-Hydrocarbon Receptor Is An Inhibitory Regulator Of Lipid Synthesis And Of Commitment To Adipogenesis
Characterization Of The Human Dihydropyrimidine Dehydrogenase Gene
Aryl-Hydrocarbon Receptor Is An Inhibitory Regulator Of Lipid Synthesis And Of Commitment To Adipogenesis
Characterization Of The Human Dihydropyrimidine Dehydrogenase Gene
Effect Of Phenobarbital On Hepatic Cyp1a1 And Cyp1a2 In The Ahr-Null Mouse
Characterization Of The Human Dihydropyrimidine Dehydrogenase Gene
Effect Of Phenobarbital On Hepatic Cyp1a1 And Cyp1a2 In The Ahr-Null Mouse
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Caucasian Subjects
Effect Of Phenobarbital On Hepatic Cyp1a1 And Cyp1a2 In The Ahr-Null Mouse
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Caucasian Subjects
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Patients With Colorectal Cancer
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Caucasian Subjects
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Patients With Colorectal Cancer
Nomenclature For Human Dpyd Alleles
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Patients With Colorectal Cancer
Nomenclature For Human Dpyd Alleles
The Involvement Of Aryl Hydrocarbon Receptor In The Activation Of Transforming Growth Factor-Β And Apoptosis
Nomenclature For Human Dpyd Alleles
The Involvement Of Aryl Hydrocarbon Receptor In The Activation Of Transforming Growth Factor-Β And Apoptosis
Ret Alternate Splicing Influences The Interaction Of Activated Ret With The Sh2 And Ptb Domains Of Shc, And The Sh2 Domain Of Grb2
The Involvement Of Aryl Hydrocarbon Receptor In The Activation Of Transforming Growth Factor-Β And Apoptosis
Ret Alternate Splicing Influences The Interaction Of Activated Ret With The Sh2 And Ptb Domains Of Shc, And The Sh2 Domain Of Grb2
Aryl Hydrocarbon Receptor Deficient Mice (Ahr-/-) Exhibit An Accumulation Of Liver Retinylpalmitate
Ret Alternate Splicing Influences The Interaction Of Activated Ret With The Sh2 And Ptb Domains Of Shc, And The Sh2 Domain Of Grb2
Aryl Hydrocarbon Receptor Deficient Mice (Ahr-/-) Exhibit An Accumulation Of Liver Retinylpalmitate
Analysis Of The Selenocysteine (Sec) Trna Population In Mouse Embryonic Stem (Es) Cells Heterozygous For The Sec Trna Gene
Aryl Hydrocarbon Receptor Deficient Mice (Ahr-/-) Exhibit An Accumulation Of Liver Retinylpalmitate
Analysis Of The Selenocysteine (Sec) Trna Population In Mouse Embryonic Stem (Es) Cells Heterozygous For The Sec Trna Gene
Aryl Hydrocarbon Receptor Knockout Mice (Ahr(-/-)) Exhibit Liver Retinoid Accumulation And Reduced Retinoic Acid Metabolism
Analysis Of The Selenocysteine (Sec) Trna Population In Mouse Embryonic Stem (Es) Cells Heterozygous For The Sec Trna Gene
Aryl Hydrocarbon Receptor Knockout Mice (Ahr(-/-)) Exhibit Liver Retinoid Accumulation And Reduced Retinoic Acid Metabolism
Expression Of Multiple Endocrine Neoplasia 2b Ret In Neuroblastoma Cells Alters Cell Adhesion In Vitro, Enhances Metastatic Behavior In Vivo, And Activates Jun Kinase
Aryl Hydrocarbon Receptor Knockout Mice (Ahr(-/-)) Exhibit Liver Retinoid Accumulation And Reduced Retinoic Acid Metabolism
Expression Of Multiple Endocrine Neoplasia 2b Ret In Neuroblastoma Cells Alters Cell Adhesion In Vitro, Enhances Metastatic Behavior In Vivo, And Activates Jun Kinase
Cyp2a6 Gene Polymorphism And Risk Of Liver Cancer And Cirrhosis
Expression Of Multiple Endocrine Neoplasia 2b Ret In Neuroblastoma Cells Alters Cell Adhesion In Vitro, Enhances Metastatic Behavior In Vivo, And Activates Jun Kinase
Cyp2a6 Gene Polymorphism And Risk Of Liver Cancer And Cirrhosis
Selenocysteine Trna([Ser]sec) Levels And Selenium-Dependent Glutathione Peroxidase Activity In Mouse Embryonic Stem Cells Heterozygous For A Targeted Mutation In The Trna([Ser]sec) Gene
Cyp2a6 Gene Polymorphism And Risk Of Liver Cancer And Cirrhosis
Selenocysteine Trna([Ser]sec) Levels And Selenium-Dependent Glutathione Peroxidase Activity In Mouse Embryonic Stem Cells Heterozygous For A Targeted Mutation In The Trna([Ser]sec) Gene
Lack Of Correlation Between Phenotype And Genotype For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In A Family Of Pakistani Origin
Selenocysteine Trna([Ser]sec) Levels And Selenium-Dependent Glutathione Peroxidase Activity In Mouse Embryonic Stem Cells Heterozygous For A Targeted Mutation In The Trna([Ser]sec) Gene
Lack Of Correlation Between Phenotype And Genotype For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In A Family Of Pakistani Origin
Lesions Of Aryl-Hydrocarbon Receptor-Deficient Mice
Lack Of Correlation Between Phenotype And Genotype For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In A Family Of Pakistani Origin
Lesions Of Aryl-Hydrocarbon Receptor-Deficient Mice
Mutations At Codon 974 Of The Dpyd Gene Are A Rare Event
Lesions Of Aryl-Hydrocarbon Receptor-Deficient Mice
Mutations At Codon 974 Of The Dpyd Gene Are A Rare Event
Regulation Of Somatostatin Gene Expression By Veratridine-Induced Depolarization In Cultured Fetal Cerebrocortical Cells
Mutations At Codon 974 Of The Dpyd Gene Are A Rare Event
Regulation Of Somatostatin Gene Expression By Veratridine-Induced Depolarization In Cultured Fetal Cerebrocortical Cells
Autocrine And/or Paracrine Action Of Vasoactive Intestinal Peptide On Thyrotropin-Releasing Hormone-Induced Prolactin Release
Regulation Of Somatostatin Gene Expression By Veratridine-Induced Depolarization In Cultured Fetal Cerebrocortical Cells
Autocrine And/or Paracrine Action Of Vasoactive Intestinal Peptide On Thyrotropin-Releasing Hormone-Induced Prolactin Release
Autoparacrine Action Of Vasoactive Intestinal Peptide On Dopaminergic Control Of Prolactin Secretion
Autocrine And/or Paracrine Action Of Vasoactive Intestinal Peptide On Thyrotropin-Releasing Hormone-Induced Prolactin Release
Autoparacrine Action Of Vasoactive Intestinal Peptide On Dopaminergic Control Of Prolactin Secretion
Autocrine And/or Paracrine Action Of Vasoactive Intestinal Peptide On Thyrotropin-Releasing Hormone Induced Prolactin Release.
Autoparacrine Action Of Vasoactive Intestinal Peptide On Dopaminergic Control Of Prolactin Secretion
Autocrine And/or Paracrine Action Of Vasoactive Intestinal Peptide On Thyrotropin-Releasing Hormone Induced Prolactin Release.
Autoparacrine Action Of Vasoactive Intestinal Peptide On Dopaminergic Control Of Prolactin Secretion.
Autocrine And/or Paracrine Action Of Vasoactive Intestinal Peptide On Thyrotropin-Releasing Hormone Induced Prolactin Release.
Autoparacrine Action Of Vasoactive Intestinal Peptide On Dopaminergic Control Of Prolactin Secretion.
Piperonyl Butoxide And Acenaphthylene Induce Cytochrome P450 1a2 And 1b1 Mrna In Aromatic Hydrocarbon-Responsive Receptor Knock-Out Mouse Liver
Autoparacrine Action Of Vasoactive Intestinal Peptide On Dopaminergic Control Of Prolactin Secretion.
Piperonyl Butoxide And Acenaphthylene Induce Cytochrome P450 1a2 And 1b1 Mrna In Aromatic Hydrocarbon-Responsive Receptor Knock-Out Mouse Liver
Transcriptional Induction Of The Cytochrome P4501a1 Gene By A Thiazolium Compound, Yh439
Piperonyl Butoxide And Acenaphthylene Induce Cytochrome P450 1a2 And 1b1 Mrna In Aromatic Hydrocarbon-Responsive Receptor Knock-Out Mouse Liver
Transcriptional Induction Of The Cytochrome P4501a1 Gene By A Thiazolium Compound, Yh439
The Role Of The Aryl Hydrocarbon Receptor In Animal Development, Physiological Homeostasis And Toxicity Of Tcdd
Transcriptional Induction Of The Cytochrome P4501a1 Gene By A Thiazolium Compound, Yh439
The Role Of The Aryl Hydrocarbon Receptor In Animal Development, Physiological Homeostasis And Toxicity Of Tcdd
Differential Regulation Of Mouse Ah Receptor Gene Expression In Cell Lines Of Different Tissue Origins
The Role Of The Aryl Hydrocarbon Receptor In Animal Development, Physiological Homeostasis And Toxicity Of Tcdd
Differential Regulation Of Mouse Ah Receptor Gene Expression In Cell Lines Of Different Tissue Origins
Molecular Basis Of The Human Dihydropyrimidine Dehydrogenase Deficiency And 5-Fluorouracil Toxicity
Differential Regulation Of Mouse Ah Receptor Gene Expression In Cell Lines Of Different Tissue Origins
Molecular Basis Of The Human Dihydropyrimidine Dehydrogenase Deficiency And 5-Fluorouracil Toxicity
Role Of Cyp2e1 In The Hepatotoxicity Of Acetaminophen
Molecular Basis Of The Human Dihydropyrimidine Dehydrogenase Deficiency And 5-Fluorouracil Toxicity
Role Of Cyp2e1 In The Hepatotoxicity Of Acetaminophen
Aryl-Hydrocarbon Receptor-Deficient Mice Are Resistant To 2,3,7,8-Tetrachlorodibenzo-P-Dioxin-Induced Toxicity
Role Of Cyp2e1 In The Hepatotoxicity Of Acetaminophen
Aryl-Hydrocarbon Receptor-Deficient Mice Are Resistant To 2,3,7,8-Tetrachlorodibenzo-P-Dioxin-Induced Toxicity
Hepatic Fibrosis In Ahr(-/-) Mice
Aryl-Hydrocarbon Receptor-Deficient Mice Are Resistant To 2,3,7,8-Tetrachlorodibenzo-P-Dioxin-Induced Toxicity
Hepatic Fibrosis In Ahr(-/-) Mice
The T/ebp Null Mouse: Thyroid-Specific Enhancer-Binding Protein Is Essential For The Organogenesis Of The Thyroid, Lung, Ventral Forebrain, And Pituitary
Hepatic Fibrosis In Ahr(-/-) Mice
The T/ebp Null Mouse: Thyroid-Specific Enhancer-Binding Protein Is Essential For The Organogenesis Of The Thyroid, Lung, Ventral Forebrain, And Pituitary
Pyrimidines And Cns Regulation
The T/ebp Null Mouse: Thyroid-Specific Enhancer-Binding Protein Is Essential For The Organogenesis Of The Thyroid, Lung, Ventral Forebrain, And Pituitary
Pyrimidines And Cns Regulation
Targeted Disruption Of Specific Cytochromes P450 And Xenobiotic Receptor Genes
Pyrimidines And Cns Regulation
Targeted Disruption Of Specific Cytochromes P450 And Xenobiotic Receptor Genes
Catalytic Specificity Of Protein-Tyrosine Kinases Is Critical For Selective Signalling
Targeted Disruption Of Specific Cytochromes P450 And Xenobiotic Receptor Genes
Catalytic Specificity Of Protein-Tyrosine Kinases Is Critical For Selective Signalling
Triiodothyronine Regulates Somatostatin Gene Expression In Cultured Fetal Rat Cerebrocortical Cells
Catalytic Specificity Of Protein-Tyrosine Kinases Is Critical For Selective Signalling
Triiodothyronine Regulates Somatostatin Gene Expression In Cultured Fetal Rat Cerebrocortical Cells
Mutation Of The Ret Protooncogene In Sporadic Medullary Thyroid Carcinoma
Triiodothyronine Regulates Somatostatin Gene Expression In Cultured Fetal Rat Cerebrocortical Cells
Mutation Of The Ret Protooncogene In Sporadic Medullary Thyroid Carcinoma
Corticosterone Modulates Growth Hormone-Releasing Factor And Somatostatin In Fetal Rat Hypothalamic Cultures
Mutation Of The Ret Protooncogene In Sporadic Medullary Thyroid Carcinoma
Corticosterone Modulates Growth Hormone-Releasing Factor And Somatostatin In Fetal Rat Hypothalamic Cultures
Diagnostic-Analysis, Clinical Importance And Molecular-Basis Of Dihydropyrimidine Dehydrogenase-Deficiency
Corticosterone Modulates Growth Hormone-Releasing Factor And Somatostatin In Fetal Rat Hypothalamic Cultures
Diagnostic-Analysis, Clinical Importance And Molecular-Basis Of Dihydropyrimidine Dehydrogenase-Deficiency
Organization And Evolution Of The Cytochrome P450 Cyp2a-2b-2f Subfamily Gene Cluster On Human Chromosome 19
Diagnostic-Analysis, Clinical Importance And Molecular-Basis Of Dihydropyrimidine Dehydrogenase-Deficiency
Organization And Evolution Of The Cytochrome P450 Cyp2a-2b-2f Subfamily Gene Cluster On Human Chromosome 19
A Genetic Polymorphism In Coumarin 7-Hydroxylation: Sequence Of The Human Cyp2a Genes And Identification Of Variant Cyp2a6 Alleles
Organization And Evolution Of The Cytochrome P450 Cyp2a-2b-2f Subfamily Gene Cluster On Human Chromosome 19
A Genetic Polymorphism In Coumarin 7-Hydroxylation: Sequence Of The Human Cyp2a Genes And Identification Of Variant Cyp2a6 Alleles
Ret Activation By Germline Men2a And Men2b Mutations
A Genetic Polymorphism In Coumarin 7-Hydroxylation: Sequence Of The Human Cyp2a Genes And Identification Of Variant Cyp2a6 Alleles
Ret Activation By Germline Men2a And Men2b Mutations
Multiple Mrna Isoforms Of The Human Ret Proto-Oncogene Generated By Alternate Splicing
Ret Activation By Germline Men2a And Men2b Mutations
Multiple Mrna Isoforms Of The Human Ret Proto-Oncogene Generated By Alternate Splicing
Comparison Of Substrate Metabolism By Wild Type Cyp2d6 Protein And A Variant Containing Methionine, Not Valine, At Position 374
Multiple Mrna Isoforms Of The Human Ret Proto-Oncogene Generated By Alternate Splicing
Comparison Of Substrate Metabolism By Wild Type Cyp2d6 Protein And A Variant Containing Methionine, Not Valine, At Position 374
Human Polymorphism In Drug Metabolism: Mutation In The Dihydropyrimidine Dehydrogenase Gene Results In Exon Skipping And Thymine Uracilurea
Comparison Of Substrate Metabolism By Wild Type Cyp2d6 Protein And A Variant Containing Methionine, Not Valine, At Position 374
Human Polymorphism In Drug Metabolism: Mutation In The Dihydropyrimidine Dehydrogenase Gene Results In Exon Skipping And Thymine Uracilurea
Genetic Polymorphism Of Cyp2c9 And Its Effect On Warfarin Maintenance Dose Requirement In Patients Undergoing Anticoagulation Therapy
Human Polymorphism In Drug Metabolism: Mutation In The Dihydropyrimidine Dehydrogenase Gene Results In Exon Skipping And Thymine Uracilurea
Genetic Polymorphism Of Cyp2c9 And Its Effect On Warfarin Maintenance Dose Requirement In Patients Undergoing Anticoagulation Therapy
Correlation Between Catalytic Activity And Protein Content For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In Human Lymphocytes
Genetic Polymorphism Of Cyp2c9 And Its Effect On Warfarin Maintenance Dose Requirement In Patients Undergoing Anticoagulation Therapy
Correlation Between Catalytic Activity And Protein Content For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In Human Lymphocytes
Neonatal Lethality Associated With Respiratory Distress In Mice Lacking Cytochrome P450 1a2
Correlation Between Catalytic Activity And Protein Content For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In Human Lymphocytes
Neonatal Lethality Associated With Respiratory Distress In Mice Lacking Cytochrome P450 1a2
Xenobiotic Receptor Knockout Mice
Neonatal Lethality Associated With Respiratory Distress In Mice Lacking Cytochrome P450 1a2
Xenobiotic Receptor Knockout Mice
Immune System Impairment And Hepatic Fibrosis In Mice Lacking The Dioxin-Binding Ah Receptor
Xenobiotic Receptor Knockout Mice
Immune System Impairment And Hepatic Fibrosis In Mice Lacking The Dioxin-Binding Ah Receptor
Targeted Disruption Of The Α Isoform Of The Peroxisome Proliferator- Activated Receptor Gene In Mice Results In Abolishment Of The Pleiotropic Effects Of Peroxisome Proliferators
Immune System Impairment And Hepatic Fibrosis In Mice Lacking The Dioxin-Binding Ah Receptor
Targeted Disruption Of The Α Isoform Of The Peroxisome Proliferator- Activated Receptor Gene In Mice Results In Abolishment Of The Pleiotropic Effects Of Peroxisome Proliferators
Effect Of Potassium-Induced Depolarization On Somatostatin Gene Expression In Cultured Fetal Rat Cerebrocortical Cells
Targeted Disruption Of The Α Isoform Of The Peroxisome Proliferator- Activated Receptor Gene In Mice Results In Abolishment Of The Pleiotropic Effects Of Peroxisome Proliferators
Effect Of Potassium-Induced Depolarization On Somatostatin Gene Expression In Cultured Fetal Rat Cerebrocortical Cells
Induction Of Vasoactive Intestinal Peptide Gene Expression And Prolactin Secretion By Insulin-Like Growth Factor I In Rat Pituitary Cells: Evidence For An Autoparacrine Regulatory System
Effect Of Potassium-Induced Depolarization On Somatostatin Gene Expression In Cultured Fetal Rat Cerebrocortical Cells
Induction Of Vasoactive Intestinal Peptide Gene Expression And Prolactin Secretion By Insulin-Like Growth Factor I In Rat Pituitary Cells: Evidence For An Autoparacrine Regulatory System
Biosynthesis Of Growth Hormone-Releasing Factor By Fetal Rat Cerebrocortical And Hypothalamic Cells
Induction Of Vasoactive Intestinal Peptide Gene Expression And Prolactin Secretion By Insulin-Like Growth Factor I In Rat Pituitary Cells: Evidence For An Autoparacrine Regulatory System
Biosynthesis Of Growth Hormone-Releasing Factor By Fetal Rat Cerebrocortical And Hypothalamic Cells
Cdna Cloning And Chromosome Mapping Of Human Dihydropyrimidine Dehydrogenase, An Enzyme Associated With 5-Fluorouracil Toxicity And Congenital Thymine Uraciluria
Biosynthesis Of Growth Hormone-Releasing Factor By Fetal Rat Cerebrocortical And Hypothalamic Cells
Cdna Cloning And Chromosome Mapping Of Human Dihydropyrimidine Dehydrogenase, An Enzyme Associated With 5-Fluorouracil Toxicity And Congenital Thymine Uraciluria
Assignment Of The Human Dihydropyrimidine Dehydrogenase Gene (Dpyd) To Chromosome Region 1p22 By Fluorescence In Situ Hybridization
Cdna Cloning And Chromosome Mapping Of Human Dihydropyrimidine Dehydrogenase, An Enzyme Associated With 5-Fluorouracil Toxicity And Congenital Thymine Uraciluria
Assignment Of The Human Dihydropyrimidine Dehydrogenase Gene (Dpyd) To Chromosome Region 1p22 By Fluorescence In Situ Hybridization
Potassium-Induced Depolarization Stimulates Somatostatin Gene Expression In Cultured Fetal Rat Cerebrocortical Cells
Assignment Of The Human Dihydropyrimidine Dehydrogenase Gene (Dpyd) To Chromosome Region 1p22 By Fluorescence In Situ Hybridization
Potassium-Induced Depolarization Stimulates Somatostatin Gene Expression In Cultured Fetal Rat Cerebrocortical Cells
Neurotransmitter Regulation Of Somatostatin Secretion By Fetal Rat Cerebral Cortical Cells In Culture
Potassium-Induced Depolarization Stimulates Somatostatin Gene Expression In Cultured Fetal Rat Cerebrocortical Cells
Neurotransmitter Regulation Of Somatostatin Secretion By Fetal Rat Cerebral Cortical Cells In Culture
Growth Hormone-Releasing Factor Regulation By Somatostatin, Growth Hormone And Insulin-Like Growth Factor I In Fetal Rat Hypothalamic-Brain Stem Cell Cocultures
Neurotransmitter Regulation Of Somatostatin Secretion By Fetal Rat Cerebral Cortical Cells In Culture
Growth Hormone-Releasing Factor Regulation By Somatostatin, Growth Hormone And Insulin-Like Growth Factor I In Fetal Rat Hypothalamic-Brain Stem Cell Cocultures
Pituitary And Peripheral Insulin‐like Growth Factor‐i Regulation By Thyroid Hormone
Growth Hormone-Releasing Factor Regulation By Somatostatin, Growth Hormone And Insulin-Like Growth Factor I In Fetal Rat Hypothalamic-Brain Stem Cell Cocultures
Pituitary And Peripheral Insulin‐like Growth Factor‐i Regulation By Thyroid Hormone
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Pituitary And Peripheral Insulin‐like Growth Factor‐i Regulation By Thyroid Hormone
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Regulation Of Somatostatin And Growth Hormone-Releasing Factor By Gonadal Steroids In Fetal Rat Hypothalamic Cells In Culture
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Regulation Of Somatostatin And Growth Hormone-Releasing Factor By Gonadal Steroids In Fetal Rat Hypothalamic Cells In Culture
Role Of Locally Produced Growth Hormone-Releasing Factors In Somatostatin Regulatory By Fetal Rat Brain Cells In Culture
Regulation Of Somatostatin And Growth Hormone-Releasing Factor By Gonadal Steroids In Fetal Rat Hypothalamic Cells In Culture
Role Of Locally Produced Growth Hormone-Releasing Factors In Somatostatin Regulatory By Fetal Rat Brain Cells In Culture
Thyroid Hormones Regulate Release And Content Of Vasoactive Intestinal Peptide In Cultured Fetal Cerebral Cortical Cells
Role Of Locally Produced Growth Hormone-Releasing Factors In Somatostatin Regulatory By Fetal Rat Brain Cells In Culture
Thyroid Hormones Regulate Release And Content Of Vasoactive Intestinal Peptide In Cultured Fetal Cerebral Cortical Cells
Immunocytochemical Evidence Of Hypothalamic Regulation Of Adenohypophyseal Vip In The Male Rat
Thyroid Hormones Regulate Release And Content Of Vasoactive Intestinal Peptide In Cultured Fetal Cerebral Cortical Cells
Immunocytochemical Evidence Of Hypothalamic Regulation Of Adenohypophyseal Vip In The Male Rat
Endogenous Vasoactive Intestinal Peptide (Vip) Regulates Somatostatin Secretion By Cultured Fetal Rat Cerebral Cortical And Hypothalamic Cells
Immunocytochemical Evidence Of Hypothalamic Regulation Of Adenohypophyseal Vip In The Male Rat
Endogenous Vasoactive Intestinal Peptide (Vip) Regulates Somatostatin Secretion By Cultured Fetal Rat Cerebral Cortical And Hypothalamic Cells
Pineal Vasoactive Intestinal Peptide Is Reduced During The Proestrous Stage Of The Rat Estrous Cycle
Endogenous Vasoactive Intestinal Peptide (Vip) Regulates Somatostatin Secretion By Cultured Fetal Rat Cerebral Cortical And Hypothalamic Cells
Pineal Vasoactive Intestinal Peptide Is Reduced During The Proestrous Stage Of The Rat Estrous Cycle
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Pineal Vasoactive Intestinal Peptide Is Reduced During The Proestrous Stage Of The Rat Estrous Cycle
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Neurosecretory And Trophic Action On Fetal Rat Neuroblasts Induced By An Amino Acid Mixture
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Neurosecretory And Trophic Action On Fetal Rat Neuroblasts Induced By An Amino Acid Mixture
Secretions Of Somatostatin And Vip In Cultures Of Fetal Rat Neuroblasts Increased By Amino Acids
Neurosecretory And Trophic Action On Fetal Rat Neuroblasts Induced By An Amino Acid Mixture
Secretions Of Somatostatin And Vip In Cultures Of Fetal Rat Neuroblasts Increased By Amino Acids
Demonstration Of Immunoreactive Vasoactive Intestinal Peptide (Ir-Vip) And Somatostatin (Ir-Som) In Rat Thymus
Secretions Of Somatostatin And Vip In Cultures Of Fetal Rat Neuroblasts Increased By Amino Acids
Demonstration Of Immunoreactive Vasoactive Intestinal Peptide (Ir-Vip) And Somatostatin (Ir-Som) In Rat Thymus
Influence Of Thyroid Hormones On Somatostatin Processing In Cultured Cerebro-Cortical Cells
Demonstration Of Immunoreactive Vasoactive Intestinal Peptide (Ir-Vip) And Somatostatin (Ir-Som) In Rat Thymus
Influence Of Thyroid Hormones On Somatostatin Processing In Cultured Cerebro-Cortical Cells
Depolarizing Influences Regulate Somatostatin Synthesis And Processing In Cultured Cerebral Cortical Cells
Influence Of Thyroid Hormones On Somatostatin Processing In Cultured Cerebro-Cortical Cells
Depolarizing Influences Regulate Somatostatin Synthesis And Processing In Cultured Cerebral Cortical Cells
Regulation Of The Neuropeptides, Somatostatin And Vasoactive Intestinal Peptide (Vip), By Local And External Messengers
Depolarizing Influences Regulate Somatostatin Synthesis And Processing In Cultured Cerebral Cortical Cells
Regulation Of The Neuropeptides, Somatostatin And Vasoactive Intestinal Peptide (Vip), By Local And External Messengers
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Regulation Of The Neuropeptides, Somatostatin And Vasoactive Intestinal Peptide (Vip), By Local And External Messengers
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Divergent Effects Of Acute Depolarization On Somatostatin Release And Protein Synthesis In Cultured Fetal And Neonatal Rat Brain Cells
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Divergent Effects Of Acute Depolarization On Somatostatin Release And Protein Synthesis In Cultured Fetal And Neonatal Rat Brain Cells
Growth Hormone-Releasing Factor Production By Fetal Rat Cerebrocortical And Hypothalamic Cells In Primary Culture
Divergent Effects Of Acute Depolarization On Somatostatin Release And Protein Synthesis In Cultured Fetal And Neonatal Rat Brain Cells
Growth Hormone-Releasing Factor Production By Fetal Rat Cerebrocortical And Hypothalamic Cells In Primary Culture
Synthesis And Secretion Of Vasoactive Intestinal Peptide By Rat Fetal Cerebral Cortical And Hypothalamic Cells In Culture
Growth Hormone-Releasing Factor Production By Fetal Rat Cerebrocortical And Hypothalamic Cells In Primary Culture
Synthesis And Secretion Of Vasoactive Intestinal Peptide By Rat Fetal Cerebral Cortical And Hypothalamic Cells In Culture
Synthesis And Secretion Of Vasoactive Intestinal Peptide And Vip‐prohormone By Fetal Rat Brain Cells In Culture
Synthesis And Secretion Of Vasoactive Intestinal Peptide By Rat Fetal Cerebral Cortical And Hypothalamic Cells In Culture
Synthesis And Secretion Of Vasoactive Intestinal Peptide And Vip‐prohormone By Fetal Rat Brain Cells In Culture
Thyroid Hormone Action On Biosynthesis Of Somatostatin By Fetal Rat Brain Cells In Culture.
Synthesis And Secretion Of Vasoactive Intestinal Peptide And Vip‐prohormone By Fetal Rat Brain Cells In Culture
Thyroid Hormone Action On Biosynthesis Of Somatostatin By Fetal Rat Brain Cells In Culture.
Ileal Vasoactive Intestinal Peptide (Vip) Levels And Vip Receptor/effector System In Lleal Epithelial Cells After Colectomy In The Rat
Thyroid Hormone Action On Biosynthesis Of Somatostatin By Fetal Rat Brain Cells In Culture.
Ileal Vasoactive Intestinal Peptide (Vip) Levels And Vip Receptor/effector System In Lleal Epithelial Cells After Colectomy In The Rat
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment
WEB ACCESIBLE