PUBLICACIÓN

STIM1-containing contact sites promote direct calcium flux from the endoplasmic reticulum to mitochondria

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ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Orantos-Aguilera Y.; Sanchez-Lopez I.; Pascual-Caro C.; Gomez-Suaga P.; Area-Gomez E.; Pozo-Guisado E.; Montesinos J.; Martin-Romero F.J.

2026 EMBO Journal

CITAS

0

DOI

10.1038/s44318-026-00700-8

EID

2-s2.0-105030074233

ISSN

0261-4189

EISSN

1460-2075

AUTORES DE LA UEX

Patricia María Gómez Suaga
Francisco Javier Martín Romero
Eulalia Pozo Guisado
Irene Sánchez López

OTRAS PUBLICACIONES DE INTERÉS

Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Stim1-Containing Contact Sites Promote Direct Calcium Flux From The Endoplasmic Reticulum To Mitochondria
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
Stim1-Containing Contact Sites Promote Direct Calcium Flux From The Endoplasmic Reticulum To Mitochondria
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
Orai1 Plays A Critical Role In Orai3 Synthesis And Stability In Luminal Breast Cancer Cells
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
Orai1 Plays A Critical Role In Orai3 Synthesis And Stability In Luminal Breast Cancer Cells
Stimulating Vapb-Ptpip51 Er-Mitochondria Tethering Corrects Ftd/als Mutant Tdp43 Linked Ca2+ And Synaptic Defects
Orai1 Plays A Critical Role In Orai3 Synthesis And Stability In Luminal Breast Cancer Cells
Stimulating Vapb-Ptpip51 Er-Mitochondria Tethering Corrects Ftd/als Mutant Tdp43 Linked Ca2+ And Synaptic Defects
Activity-Driven Synaptic Translocation Of Lgi1 Controls Excitatory Neurotransmission
Stimulating Vapb-Ptpip51 Er-Mitochondria Tethering Corrects Ftd/als Mutant Tdp43 Linked Ca2+ And Synaptic Defects
Activity-Driven Synaptic Translocation Of Lgi1 Controls Excitatory Neurotransmission
Truncated Tau Interferes With The Autophagy And Endolysosomal Pathway And Results In Lipid Accumulation
Activity-Driven Synaptic Translocation Of Lgi1 Controls Excitatory Neurotransmission
Truncated Tau Interferes With The Autophagy And Endolysosomal Pathway And Results In Lipid Accumulation
Insight Into Endoplasmic Reticulum-Mitochondria Contacts In Human Amyotrophic Lateral Sclerosis
Truncated Tau Interferes With The Autophagy And Endolysosomal Pathway And Results In Lipid Accumulation
Insight Into Endoplasmic Reticulum-Mitochondria Contacts In Human Amyotrophic Lateral Sclerosis
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
Insight Into Endoplasmic Reticulum-Mitochondria Contacts In Human Amyotrophic Lateral Sclerosis
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
Mitochondrial Dysfunction In Repeat Expansion Diseases
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
Mitochondrial Dysfunction In Repeat Expansion Diseases
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease, Volume Ii
Mitochondrial Dysfunction In Repeat Expansion Diseases
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease, Volume Ii
Editorial
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease, Volume Ii
Editorial
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
Editorial
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
The Presence Of Disease-Associated Tau Leads To Adverse Effects On The Autophagy And Endolysosomal Pathway In A Cell Model Of Tauopathy
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
The Presence Of Disease-Associated Tau Leads To Adverse Effects On The Autophagy And Endolysosomal Pathway In A Cell Model Of Tauopathy
Disruption Of The Vapb-Ptpip51 Er-Mitochondria Tethering Proteins In Post-Mortem Human Amyotrophic Lateral Sclerosis
The Presence Of Disease-Associated Tau Leads To Adverse Effects On The Autophagy And Endolysosomal Pathway In A Cell Model Of Tauopathy
Disruption Of The Vapb-Ptpip51 Er-Mitochondria Tethering Proteins In Post-Mortem Human Amyotrophic Lateral Sclerosis
The Interplay Between Autophagy And Rna Homeostasis: Implications For Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
Disruption Of The Vapb-Ptpip51 Er-Mitochondria Tethering Proteins In Post-Mortem Human Amyotrophic Lateral Sclerosis
The Interplay Between Autophagy And Rna Homeostasis: Implications For Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
Disruption Of Er-Mitochondria Tethering And Signalling In C9orf72-Associated Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
The Interplay Between Autophagy And Rna Homeostasis: Implications For Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
Disruption Of Er-Mitochondria Tethering And Signalling In C9orf72-Associated Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Disruption Of Er-Mitochondria Tethering And Signalling In C9orf72-Associated Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Autophagy And Lysosomal Defects In Cells Expressing Disease-Associated Tau
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Autophagy And Lysosomal Defects In Cells Expressing Disease-Associated Tau
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease
Autophagy And Lysosomal Defects In Cells Expressing Disease-Associated Tau
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease
Disruption Of Endoplasmic Reticulum-Mitochondria Tethering Proteins In Post-Mortem Alzheimer's Disease Brain
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease
Disruption Of Endoplasmic Reticulum-Mitochondria Tethering Proteins In Post-Mortem Alzheimer's Disease Brain
Stim1 Deficiency Leads To Specific Down-Regulation Of Itpr3 In Sh-Sy5y Cells
Disruption Of Endoplasmic Reticulum-Mitochondria Tethering Proteins In Post-Mortem Alzheimer's Disease Brain
Stim1 Deficiency Leads To Specific Down-Regulation Of Itpr3 In Sh-Sy5y Cells
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Stim1 Deficiency Leads To Specific Down-Regulation Of Itpr3 In Sh-Sy5y Cells
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Fluctuation In O-Glcnacylation Inactivates Stim1 To Reduce Store-Operated Calcium Ion Entry Via Down-Regulation Of Ser621phosphorylation
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Fluctuation In O-Glcnacylation Inactivates Stim1 To Reduce Store-Operated Calcium Ion Entry Via Down-Regulation Of Ser621phosphorylation
Lmtk2 Binds To Kinesin Light Chains To Mediate Anterograde Axonal Transport Of Cdk5/p35 And Lmtk2 Levels Are Reduced In Alzheimer's Disease Brains
Fluctuation In O-Glcnacylation Inactivates Stim1 To Reduce Store-Operated Calcium Ion Entry Via Down-Regulation Of Ser621phosphorylation
Lmtk2 Binds To Kinesin Light Chains To Mediate Anterograde Axonal Transport Of Cdk5/p35 And Lmtk2 Levels Are Reduced In Alzheimer's Disease Brains
The Vapb-Ptpip51 Endoplasmic Reticulum-Mitochondria Tethering Proteins Are Present In Neuronal Synapses And Regulate Synaptic Activity
Lmtk2 Binds To Kinesin Light Chains To Mediate Anterograde Axonal Transport Of Cdk5/p35 And Lmtk2 Levels Are Reduced In Alzheimer's Disease Brains
The Vapb-Ptpip51 Endoplasmic Reticulum-Mitochondria Tethering Proteins Are Present In Neuronal Synapses And Regulate Synaptic Activity
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
The Vapb-Ptpip51 Endoplasmic Reticulum-Mitochondria Tethering Proteins Are Present In Neuronal Synapses And Regulate Synaptic Activity
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Role Of Stim1 In Neurodegeneration
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Role Of Stim1 In Neurodegeneration
Regulation Of Calcium Signaling By Stim1 And Orai1
Role Of Stim1 In Neurodegeneration
Regulation Of Calcium Signaling By Stim1 And Orai1
The Teleost Retina As A Model For Regenerative Medicine
Regulation Of Calcium Signaling By Stim1 And Orai1
The Teleost Retina As A Model For Regenerative Medicine
Disruption Of Er-Mitochondria Signalling In Fronto-Temporal Dementia And Related Amyotrophic Lateral Sclerosis
The Teleost Retina As A Model For Regenerative Medicine
Disruption Of Er-Mitochondria Signalling In Fronto-Temporal Dementia And Related Amyotrophic Lateral Sclerosis
Parkinson Disease-Associated Mutations In Lrrk2 Cause Centrosomal Defects Via Rab8a Phosphorylation
Disruption Of Er-Mitochondria Signalling In Fronto-Temporal Dementia And Related Amyotrophic Lateral Sclerosis
Parkinson Disease-Associated Mutations In Lrrk2 Cause Centrosomal Defects Via Rab8a Phosphorylation
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Parkinson Disease-Associated Mutations In Lrrk2 Cause Centrosomal Defects Via Rab8a Phosphorylation
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
The Interplay Between Cytoskeleton And Calcium Dynamics
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
The Interplay Between Cytoskeleton And Calcium Dynamics
Er-Mitochondria Signaling Regulates Autophagy
The Interplay Between Cytoskeleton And Calcium Dynamics
Er-Mitochondria Signaling Regulates Autophagy
Α-Synuclein Binds To The Er–mitochondria Tethering Protein Vapb To Disrupt Ca2+ Homeostasis And Mitochondrial Atp Production
Er-Mitochondria Signaling Regulates Autophagy
Α-Synuclein Binds To The Er–mitochondria Tethering Protein Vapb To Disrupt Ca2+ Homeostasis And Mitochondrial Atp Production
The Er-Mitochondria Tethering Complex Vapb-Ptpip51 Regulates Autophagy
Α-Synuclein Binds To The Er–mitochondria Tethering Protein Vapb To Disrupt Ca2+ Homeostasis And Mitochondrial Atp Production
The Er-Mitochondria Tethering Complex Vapb-Ptpip51 Regulates Autophagy
Store-Operated Calcium Entry Is Dispensable For The Activation Of Erk1/2 Pathway In Prostate Cancer Cells
The Er-Mitochondria Tethering Complex Vapb-Ptpip51 Regulates Autophagy
Store-Operated Calcium Entry Is Dispensable For The Activation Of Erk1/2 Pathway In Prostate Cancer Cells
Regulation Of Membrane Ruffling By Polarized Stim1 And Orai1 In Cortactin-Rich Domains
Store-Operated Calcium Entry Is Dispensable For The Activation Of Erk1/2 Pathway In Prostate Cancer Cells
Regulation Of Membrane Ruffling By Polarized Stim1 And Orai1 In Cortactin-Rich Domains
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
Regulation Of Membrane Ruffling By Polarized Stim1 And Orai1 In Cortactin-Rich Domains
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
Als/ftd-Associated Fus Activates Gsk-3β To Disrupt The Vapb–ptpip51 Interaction And Er–mitochondria Associations
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
Als/ftd-Associated Fus Activates Gsk-3β To Disrupt The Vapb–ptpip51 Interaction And Er–mitochondria Associations
Iron Overload Causes Endolysosomal Deficits Modulated By Naadp-Regulated 2-Pore Channels And Rab7a
Als/ftd-Associated Fus Activates Gsk-3β To Disrupt The Vapb–ptpip51 Interaction And Er–mitochondria Associations
Iron Overload Causes Endolysosomal Deficits Modulated By Naadp-Regulated 2-Pore Channels And Rab7a
There's Something Wrong With My Mam; The Er-Mitochondria Axis And Neurodegenerative Diseases
Iron Overload Causes Endolysosomal Deficits Modulated By Naadp-Regulated 2-Pore Channels And Rab7a
There's Something Wrong With My Mam; The Er-Mitochondria Axis And Neurodegenerative Diseases
Alterations In Late Endocytic Trafficking Related To The Pathobiology Of Lrrk2-Linked Parkinson's Disease
There's Something Wrong With My Mam; The Er-Mitochondria Axis And Neurodegenerative Diseases
Alterations In Late Endocytic Trafficking Related To The Pathobiology Of Lrrk2-Linked Parkinson's Disease
Autophagic Pathology And Calcium Deregulation In Neurodegeneration
Alterations In Late Endocytic Trafficking Related To The Pathobiology Of Lrrk2-Linked Parkinson's Disease
Autophagic Pathology And Calcium Deregulation In Neurodegeneration
Stim1 Phosphorylation Triggered By Epidermal Growth Factor Mediates Cell Migration
Autophagic Pathology And Calcium Deregulation In Neurodegeneration
Stim1 Phosphorylation Triggered By Epidermal Growth Factor Mediates Cell Migration
Lrrk2 Delays Degradative Receptor Trafficking By Impeding Late Endosomal Budding Through Decreasing Rab7 Activity
Stim1 Phosphorylation Triggered By Epidermal Growth Factor Mediates Cell Migration
Lrrk2 Delays Degradative Receptor Trafficking By Impeding Late Endosomal Budding Through Decreasing Rab7 Activity
Upstream Deregulation Of Calcium Signaling In Parkinson's Disease
Lrrk2 Delays Degradative Receptor Trafficking By Impeding Late Endosomal Budding Through Decreasing Rab7 Activity
Upstream Deregulation Of Calcium Signaling In Parkinson's Disease
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Upstream Deregulation Of Calcium Signaling In Parkinson's Disease
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Phospho-Stim1 Is A Downstream Effector That Mediates The Signaling Triggered By Igf-1 In Hek293 Cells
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Phospho-Stim1 Is A Downstream Effector That Mediates The Signaling Triggered By Igf-1 In Hek293 Cells
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
Phospho-Stim1 Is A Downstream Effector That Mediates The Signaling Triggered By Igf-1 In Hek293 Cells
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Regulation Of Stim1 By Phosphorylation
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Regulation Of Stim1 By Phosphorylation
Inhibition Of Stim1 Phosphorylation Underlies Resveratrol-Induced Inhibition Of Store-Operated Calcium Entry
The Regulation Of Stim1 By Phosphorylation
Inhibition Of Stim1 Phosphorylation Underlies Resveratrol-Induced Inhibition Of Store-Operated Calcium Entry
A Link Between Autophagy And The Pathophysiology Of Lrrk2 In Parkinson's Disease
Inhibition Of Stim1 Phosphorylation Underlies Resveratrol-Induced Inhibition Of Store-Operated Calcium Entry
A Link Between Autophagy And The Pathophysiology Of Lrrk2 In Parkinson's Disease
A Link Between Lrrk2, Autophagy And Naadp-Mediated Endolysosomal Calcium Signalling
A Link Between Autophagy And The Pathophysiology Of Lrrk2 In Parkinson's Disease
A Link Between Lrrk2, Autophagy And Naadp-Mediated Endolysosomal Calcium Signalling
Stone-Isolated Carbonatogenic Bacteria As Inoculants In Bioconsolidation Treatments For Historical Limestone
A Link Between Lrrk2, Autophagy And Naadp-Mediated Endolysosomal Calcium Signalling
Stone-Isolated Carbonatogenic Bacteria As Inoculants In Bioconsolidation Treatments For Historical Limestone
Leucine-Rich Repeat Kinase 2 Regulates Autophagy Through A Calcium-Dependent Pathway Involving Naadp
Stone-Isolated Carbonatogenic Bacteria As Inoculants In Bioconsolidation Treatments For Historical Limestone
Leucine-Rich Repeat Kinase 2 Regulates Autophagy Through A Calcium-Dependent Pathway Involving Naadp
Lrrk2 As A Modulator Of Lysosomal Calcium Homeostasis With Downstream Effects On Autophagy
Leucine-Rich Repeat Kinase 2 Regulates Autophagy Through A Calcium-Dependent Pathway Involving Naadp
Lrrk2 As A Modulator Of Lysosomal Calcium Homeostasis With Downstream Effects On Autophagy
Intracellular Cyclophilin A Is An Important Ca2+ Regulator In Platelets And Critically Involved In Arterial Thrombus Formation
Lrrk2 As A Modulator Of Lysosomal Calcium Homeostasis With Downstream Effects On Autophagy
Intracellular Cyclophilin A Is An Important Ca2+ Regulator In Platelets And Critically Involved In Arterial Thrombus Formation
Extended Embryo Culture Supplementation
Intracellular Cyclophilin A Is An Important Ca2+ Regulator In Platelets And Critically Involved In Arterial Thrombus Formation
Extended Embryo Culture Supplementation
Calcium Signaling In Mouse Oocyte Maturation: The Roles Of Stim1, Orai1 And Soce
Extended Embryo Culture Supplementation
Calcium Signaling In Mouse Oocyte Maturation: The Roles Of Stim1, Orai1 And Soce
Role Of Store-Operated Calcium Entry During Meiotic Progression And Fertilization Of Mammalian Oocytes
Calcium Signaling In Mouse Oocyte Maturation: The Roles Of Stim1, Orai1 And Soce
Role Of Store-Operated Calcium Entry During Meiotic Progression And Fertilization Of Mammalian Oocytes
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Role Of Store-Operated Calcium Entry During Meiotic Progression And Fertilization Of Mammalian Oocytes
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Posttranslational Modifications As Versatile Regulators Of Parkin Function
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Posttranslational Modifications As Versatile Regulators Of Parkin Function
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Posttranslational Modifications As Versatile Regulators Of Parkin Function
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Spak/osr1 Regulate Nkcc1 And Wnk Activity: Analysis Of Wnk Isoform Interactions And Activation By T-Loop Trans-Autophosphorylation
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Regulation Of Cell Survival By Resveratrol Involves Inhibition Of Nfκb-Regulated Gene Expression In Prostate Cancer Cells
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Regulation Of Cell Survival By Resveratrol Involves Inhibition Of Nfκb-Regulated Gene Expression In Prostate Cancer Cells
Relocalization Of Stim1 In Mouse Oocytes At Fertilization: Early Involvement Of Store-Operated Calcium Entry
Regulation Of Cell Survival By Resveratrol Involves Inhibition Of Nfκb-Regulated Gene Expression In Prostate Cancer Cells
Relocalization Of Stim1 In Mouse Oocytes At Fertilization: Early Involvement Of Store-Operated Calcium Entry
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Relocalization Of Stim1 In Mouse Oocytes At Fertilization: Early Involvement Of Store-Operated Calcium Entry
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Store-Operated Calcium Entry In Human Oocytes And Sensitivity To Oxidative Stress
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Store-Operated Calcium Entry In Human Oocytes And Sensitivity To Oxidative Stress
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Store-Operated Calcium Entry In Human Oocytes And Sensitivity To Oxidative Stress
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Contribution Of Culture Media To Oxidative Stress And Its Effect On Human Oocytes
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Contribution Of Culture Media To Oxidative Stress And Its Effect On Human Oocytes
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Contribution Of Culture Media To Oxidative Stress And Its Effect On Human Oocytes
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Non-Genomic Action Of Resveratrol On Androgen And Oestrogen Receptors In Prostate Cancer: Modulation Of The Phosphoinositide 3-Kinase Pathway
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Non-Genomic Action Of Resveratrol On Androgen And Oestrogen Receptors In Prostate Cancer: Modulation Of The Phosphoinositide 3-Kinase Pathway
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
Non-Genomic Action Of Resveratrol On Androgen And Oestrogen Receptors In Prostate Cancer: Modulation Of The Phosphoinositide 3-Kinase Pathway
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
Regulation Of Activity And Localization Of The Wnk1 Protein Kinase By Hyperosmotic Stress
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
Regulation Of Activity And Localization Of The Wnk1 Protein Kinase By Hyperosmotic Stress
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Regulation Of Activity And Localization Of The Wnk1 Protein Kinase By Hyperosmotic Stress
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Store-Operated Calcium Entry In Differentiated C2c12 Skeletal Muscle Cells
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Store-Operated Calcium Entry In Differentiated C2c12 Skeletal Muscle Cells
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Store-Operated Calcium Entry In Differentiated C2c12 Skeletal Muscle Cells
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Development Of A Pcr-Based Strategy For Cyp2d6 Genotyping Including Gene Multiplication Of Worldwide Potential Use
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Development Of A Pcr-Based Strategy For Cyp2d6 Genotyping Including Gene Multiplication Of Worldwide Potential Use
Resveratrol Modulates The Phosphoinositide 3-Kinase Pathway Through An Estrogen Receptor Α-Dependent Mechanism: Relevance In Cell Proliferation
Development Of A Pcr-Based Strategy For Cyp2d6 Genotyping Including Gene Multiplication Of Worldwide Potential Use
Resveratrol Modulates The Phosphoinositide 3-Kinase Pathway Through An Estrogen Receptor Α-Dependent Mechanism: Relevance In Cell Proliferation
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Resveratrol Modulates The Phosphoinositide 3-Kinase Pathway Through An Estrogen Receptor Α-Dependent Mechanism: Relevance In Cell Proliferation
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
The Drosophila Selenoprotein Bthd Is Required For Survival And Has A Role In Salivary Gland Development
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
The Drosophila Selenoprotein Bthd Is Required For Survival And Has A Role In Salivary Gland Development
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
The Drosophila Selenoprotein Bthd Is Required For Survival And Has A Role In Salivary Gland Development
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
Selenium Metabolism In Drosophila. Selenoproteins, Selenoprotein Mrna Expression, Fertility, And Mortality
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
Selenium Metabolism In Drosophila. Selenoproteins, Selenoprotein Mrna Expression, Fertility, And Mortality
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Selenium Metabolism In Drosophila. Selenoproteins, Selenoprotein Mrna Expression, Fertility, And Mortality
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Stim1-Containing Contact Sites Promote Direct Calcium Flux From The Endoplasmic Reticulum To Mitochondria
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