PUBLICACIÓN

Axial stretch-dependent cation entry in dystrophic cardiomyopathy: Involvement of several TRPs channels

ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Aguettaz E.; Lopez J.J.; Krzesiak A.; Lipskaia L.; Adnot S.; Hajjar R.J.; Cognard C.; Constantin B.; Sebille S.

2016 CELL CALCIUM

Cell Biology (Q2), Molecular Biology (Q2), Physiology (Q1)

JCR: 3.707

SJR: 1.831

CITAS

19

DOI

10.1016/j.ceca.2016.01.001

EID

2-s2.0-84956919082

ISSN

0143-4160

EISSN

1532-1991

AUTORES DE LA UEX

José Javier López Barba

OTRAS PUBLICACIONES DE INTERÉS

Two Pathways For Store-Mediated Calcium Entry Differentially Dependent On The Actin Cytoskeleton In Human Platelets
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
Filamin A C-Terminal Fragment Modulates Orai1 Expression By Inhibition Of Protein Degradation
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
Filamin A C-Terminal Fragment Modulates Orai1 Expression By Inhibition Of Protein Degradation
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Filamin A C-Terminal Fragment Modulates Orai1 Expression By Inhibition Of Protein Degradation
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Orai1 Plays A Critical Role In Orai3 Synthesis And Stability In Luminal Breast Cancer Cells
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Orai1 Plays A Critical Role In Orai3 Synthesis And Stability In Luminal Breast Cancer Cells
Stanniocalcin2, A Promising New Target For Identifying Patients With Stroke/ictus
Orai1 Plays A Critical Role In Orai3 Synthesis And Stability In Luminal Breast Cancer Cells
Stanniocalcin2, A Promising New Target For Identifying Patients With Stroke/ictus
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Stanniocalcin2, A Promising New Target For Identifying Patients With Stroke/ictus
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Postbiotics Of Lacticaseibacillus Paracasei Cect 9610 And Lactiplantibacillus Plantarum Cect 9608 Attenuates Store-Operated Calcium Entry And Fak Phosphorylation In Colorectal Cancer Cells
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Postbiotics Of Lacticaseibacillus Paracasei Cect 9610 And Lactiplantibacillus Plantarum Cect 9608 Attenuates Store-Operated Calcium Entry And Fak Phosphorylation In Colorectal Cancer Cells
Extended Synaptotagmins 1 And 2 Are Required For Store-Operated Calcium Entry, Cell Migration And Viability In Breast Cancer Cells
Postbiotics Of Lacticaseibacillus Paracasei Cect 9610 And Lactiplantibacillus Plantarum Cect 9608 Attenuates Store-Operated Calcium Entry And Fak Phosphorylation In Colorectal Cancer Cells
Extended Synaptotagmins 1 And 2 Are Required For Store-Operated Calcium Entry, Cell Migration And Viability In Breast Cancer Cells
Differential Functional Role Of Orai1 Variants In Constitutive Ca2+ Entry And Calcification In Luminal Breast Cancer Cells
Extended Synaptotagmins 1 And 2 Are Required For Store-Operated Calcium Entry, Cell Migration And Viability In Breast Cancer Cells
Differential Functional Role Of Orai1 Variants In Constitutive Ca2+ Entry And Calcification In Luminal Breast Cancer Cells
Role Of Orai-Family Channels In The Activation And Regulation Of Transcriptional Activity
Differential Functional Role Of Orai1 Variants In Constitutive Ca2+ Entry And Calcification In Luminal Breast Cancer Cells
Role Of Orai-Family Channels In The Activation And Regulation Of Transcriptional Activity
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Role Of Orai-Family Channels In The Activation And Regulation Of Transcriptional Activity
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Functional Differences In Agonist-Induced Plasma Membrane Expression Of Orai1α And Orai1β
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Functional Differences In Agonist-Induced Plasma Membrane Expression Of Orai1α And Orai1β
The Ca2+ Sensor Stim In Human Diseases
Functional Differences In Agonist-Induced Plasma Membrane Expression Of Orai1α And Orai1β
The Ca2+ Sensor Stim In Human Diseases
Orai1α And Orai1β Support Calcium Entry And Mammosphere Formation In Breast Cancer Stem Cells
The Ca2+ Sensor Stim In Human Diseases
Orai1α And Orai1β Support Calcium Entry And Mammosphere Formation In Breast Cancer Stem Cells
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Orai1α And Orai1β Support Calcium Entry And Mammosphere Formation In Breast Cancer Stem Cells
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
The Store-Operated Ca2+ Channel Orai1α Is Required For Agonist-Evoked Nf-Κb Activation By A Mechanism Dependent On Pkcβ2
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
The Store-Operated Ca2+ Channel Orai1α Is Required For Agonist-Evoked Nf-Κb Activation By A Mechanism Dependent On Pkcβ2
Store-Operated Calcium Entry In Breast Cancer Cells Is Insensitive To Orai1 And Stim1 N-Linked Glycosylation
The Store-Operated Ca2+ Channel Orai1α Is Required For Agonist-Evoked Nf-Κb Activation By A Mechanism Dependent On Pkcβ2
Store-Operated Calcium Entry In Breast Cancer Cells Is Insensitive To Orai1 And Stim1 N-Linked Glycosylation
Correction To: Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells (Cancers, (2021), 14, 1, (114), 10.3390/cancers14010114)
Store-Operated Calcium Entry In Breast Cancer Cells Is Insensitive To Orai1 And Stim1 N-Linked Glycosylation
Correction To: Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells (Cancers, (2021), 14, 1, (114), 10.3390/cancers14010114)
Orai1α, But Not Orai1β, Co-Localizes With Trpc1 And Is Required For Its Plasma Membrane Location And Activation In Hela Cells
Correction To: Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells (Cancers, (2021), 14, 1, (114), 10.3390/cancers14010114)
Orai1α, But Not Orai1β, Co-Localizes With Trpc1 And Is Required For Its Plasma Membrane Location And Activation In Hela Cells
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Orai1α, But Not Orai1β, Co-Localizes With Trpc1 And Is Required For Its Plasma Membrane Location And Activation In Hela Cells
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Similarities And Differences Between The Orai1 Variants: Orai1α And Orai1β
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Similarities And Differences Between The Orai1 Variants: Orai1α And Orai1β
Store-Operated Calcium Entry And Its Implications In Cancer Stem Cells
Similarities And Differences Between The Orai1 Variants: Orai1α And Orai1β
Store-Operated Calcium Entry And Its Implications In Cancer Stem Cells
A Fibrin Coating Method Of Polypropylene Meshes Enables The Adhesion Of Menstrual Blood-Derived Mesenchymal Stromal Cells: A New Delivery Strategy For Stem Cell-Based Therapies
Store-Operated Calcium Entry And Its Implications In Cancer Stem Cells
A Fibrin Coating Method Of Polypropylene Meshes Enables The Adhesion Of Menstrual Blood-Derived Mesenchymal Stromal Cells: A New Delivery Strategy For Stem Cell-Based Therapies
The Orai1-Ac8 Interplay: How Breast Cancer Cells Escape From Orai1 Channel Inactivation
A Fibrin Coating Method Of Polypropylene Meshes Enables The Adhesion Of Menstrual Blood-Derived Mesenchymal Stromal Cells: A New Delivery Strategy For Stem Cell-Based Therapies
The Orai1-Ac8 Interplay: How Breast Cancer Cells Escape From Orai1 Channel Inactivation
Furin Prodomain Ppfurin Enhances Ca2+ Entry Through Orai And Trpc6 Channels’ Activation In Breast Cancer Cells
The Orai1-Ac8 Interplay: How Breast Cancer Cells Escape From Orai1 Channel Inactivation
Furin Prodomain Ppfurin Enhances Ca2+ Entry Through Orai And Trpc6 Channels’ Activation In Breast Cancer Cells
Saraf And Efhb Modulate Store-Operated Ca2+ Entry And Are Required For Cell Proliferation, Migration And Viability In Breast Cancer Cells
Furin Prodomain Ppfurin Enhances Ca2+ Entry Through Orai And Trpc6 Channels’ Activation In Breast Cancer Cells
Saraf And Efhb Modulate Store-Operated Ca2+ Entry And Are Required For Cell Proliferation, Migration And Viability In Breast Cancer Cells
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Saraf And Efhb Modulate Store-Operated Ca2+ Entry And Are Required For Cell Proliferation, Migration And Viability In Breast Cancer Cells
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Role Of Orai3 In The Pathophysiology Of Cancer
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Role Of Orai3 In The Pathophysiology Of Cancer
Cross-Talk Between The Adenylyl Cyclase/camp Pathway And Ca2+ Homeostasis
Role Of Orai3 In The Pathophysiology Of Cancer
Cross-Talk Between The Adenylyl Cyclase/camp Pathway And Ca2+ Homeostasis
Ela/apela Precursor Cleaved By Furin Displays Tumor Suppressor Function In Renal Cell Carcinoma Through Mtorc1 Activation
Cross-Talk Between The Adenylyl Cyclase/camp Pathway And Ca2+ Homeostasis
Ela/apela Precursor Cleaved By Furin Displays Tumor Suppressor Function In Renal Cell Carcinoma Through Mtorc1 Activation
Furin Prodomain Ppfurin Enhances Soce Through Trpc6 Activation In Breast Cancer Cells
Ela/apela Precursor Cleaved By Furin Displays Tumor Suppressor Function In Renal Cell Carcinoma Through Mtorc1 Activation
Furin Prodomain Ppfurin Enhances Soce Through Trpc6 Activation In Breast Cancer Cells
Functional Role Of Trpc6 And Stim2 In Cytosolic And Endoplasmic Reticulum Ca2+ Content In Resting Estrogen Receptor-Positive Breast Cancer Cells
Furin Prodomain Ppfurin Enhances Soce Through Trpc6 Activation In Breast Cancer Cells
Functional Role Of Trpc6 And Stim2 In Cytosolic And Endoplasmic Reticulum Ca2+ Content In Resting Estrogen Receptor-Positive Breast Cancer Cells
Arachidonic Acid Attenuates Cell Proliferation, Migration And Viability By A Mechanism Independent On Calcium Entry
Functional Role Of Trpc6 And Stim2 In Cytosolic And Endoplasmic Reticulum Ca2+ Content In Resting Estrogen Receptor-Positive Breast Cancer Cells
Arachidonic Acid Attenuates Cell Proliferation, Migration And Viability By A Mechanism Independent On Calcium Entry
Trpc Channels In The Soce Scenario
Arachidonic Acid Attenuates Cell Proliferation, Migration And Viability By A Mechanism Independent On Calcium Entry
Trpc Channels In The Soce Scenario
Molecular Basis And Regulation Of Store-Operated Calcium Entry
Trpc Channels In The Soce Scenario
Molecular Basis And Regulation Of Store-Operated Calcium Entry
Adenylyl Cyclase Type 8 Overexpression Impairs Phosphorylation-Dependent Orai1 Inactivation And Promotes Migration In Mda-Mb-231 Breast Cancer Cells
Molecular Basis And Regulation Of Store-Operated Calcium Entry
Adenylyl Cyclase Type 8 Overexpression Impairs Phosphorylation-Dependent Orai1 Inactivation And Promotes Migration In Mda-Mb-231 Breast Cancer Cells
Flavonoids And Platelet-Derived Thrombotic Disorders
Adenylyl Cyclase Type 8 Overexpression Impairs Phosphorylation-Dependent Orai1 Inactivation And Promotes Migration In Mda-Mb-231 Breast Cancer Cells
Flavonoids And Platelet-Derived Thrombotic Disorders
Store-Independent Orai1-Mediated Ca 2+ Entry And Cancer
Flavonoids And Platelet-Derived Thrombotic Disorders
Store-Independent Orai1-Mediated Ca 2+ Entry And Cancer
(−)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells
Store-Independent Orai1-Mediated Ca 2+ Entry And Cancer
(−)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus
(−)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus
Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus
Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Platelet Functions Are Decreased In Obesity And Restored After Weight Loss: Evidence For A Role Of The Serca3-Dependent Adp Secretion Pathway
Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Platelet Functions Are Decreased In Obesity And Restored After Weight Loss: Evidence For A Role Of The Serca3-Dependent Adp Secretion Pathway
Efhb Is A Novel Cytosolic Ca 2+ Sensor That Modulates Stim1-Saraf Interaction
Platelet Functions Are Decreased In Obesity And Restored After Weight Loss: Evidence For A Role Of The Serca3-Dependent Adp Secretion Pathway
Efhb Is A Novel Cytosolic Ca 2+ Sensor That Modulates Stim1-Saraf Interaction
Flavonoids And Platelet-Derived Thrombotic Disorders.
Efhb Is A Novel Cytosolic Ca 2+ Sensor That Modulates Stim1-Saraf Interaction
Flavonoids And Platelet-Derived Thrombotic Disorders.
(-)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells.
Flavonoids And Platelet-Derived Thrombotic Disorders.
(-)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells.
Store-Operated Ca 2+ Entry In Breast Cancer Cells: Remodeling And Functional Role
(-)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells.
Store-Operated Ca 2+ Entry In Breast Cancer Cells: Remodeling And Functional Role
Trpc6 Channels Are Required For Proliferation, Migration And Invasion Of Breast Cancer Cell Lines By Modulation Of Orai1 And Orai3 Surface Exposure
Store-Operated Ca 2+ Entry In Breast Cancer Cells: Remodeling And Functional Role
Trpc6 Channels Are Required For Proliferation, Migration And Invasion Of Breast Cancer Cell Lines By Modulation Of Orai1 And Orai3 Surface Exposure
Fine-Tuning Of Store-Operated Calcium Entry By Fast And Slow Ca2+-Dependent Inactivation: Involvement Of Saraf
Trpc6 Channels Are Required For Proliferation, Migration And Invasion Of Breast Cancer Cell Lines By Modulation Of Orai1 And Orai3 Surface Exposure
Fine-Tuning Of Store-Operated Calcium Entry By Fast And Slow Ca2+-Dependent Inactivation: Involvement Of Saraf
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets
Fine-Tuning Of Store-Operated Calcium Entry By Fast And Slow Ca2+-Dependent Inactivation: Involvement Of Saraf
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets
Involvement Of Stanniocalcins In The Deregulation Of Glycaemia In Obese Mice And Type 2 Diabetic Patients
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets
Involvement Of Stanniocalcins In The Deregulation Of Glycaemia In Obese Mice And Type 2 Diabetic Patients
Filamin A Modulates Platelet Function
Involvement Of Stanniocalcins In The Deregulation Of Glycaemia In Obese Mice And Type 2 Diabetic Patients
Filamin A Modulates Platelet Function
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus.
Filamin A Modulates Platelet Function
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus.
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets.
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus.
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets.
Trps In Pain Sensation
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets.
Trps In Pain Sensation
Cardiovascular And Hemostatic Disorders: Soce And Ca2+ Handling In Platelet Dysfunction
Trps In Pain Sensation
Cardiovascular And Hemostatic Disorders: Soce And Ca2+ Handling In Platelet Dysfunction
Role Of Stim1 In The Surface Expression Of Saraf
Cardiovascular And Hemostatic Disorders: Soce And Ca2+ Handling In Platelet Dysfunction
Role Of Stim1 In The Surface Expression Of Saraf
Dynamic Interaction Of Saraf With Stim1 And Orai1 To Modulate Store-Operated Calcium Entry
Role Of Stim1 In The Surface Expression Of Saraf
Dynamic Interaction Of Saraf With Stim1 And Orai1 To Modulate Store-Operated Calcium Entry
Molecular Modulators Of Store-Operated Calcium Entry
Dynamic Interaction Of Saraf With Stim1 And Orai1 To Modulate Store-Operated Calcium Entry
Molecular Modulators Of Store-Operated Calcium Entry
Historical Overview Of Store-Operated Ca2+ Entry
Molecular Modulators Of Store-Operated Calcium Entry
Historical Overview Of Store-Operated Ca2+ Entry
Store-Operated Ca2+ Entry- Associated Regulatory Factor (Saraf) Plays An Important Role In The Regulation Of Arachidonate-Regulated Ca2+ (Arc) Channels
Historical Overview Of Store-Operated Ca2+ Entry
Store-Operated Ca2+ Entry- Associated Regulatory Factor (Saraf) Plays An Important Role In The Regulation Of Arachidonate-Regulated Ca2+ (Arc) Channels
Saraf Modulates Trpc1, But Not Trpc6, Channel Function In A Stim1-Independent Manner
Store-Operated Ca2+ Entry- Associated Regulatory Factor (Saraf) Plays An Important Role In The Regulation Of Arachidonate-Regulated Ca2+ (Arc) Channels
Saraf Modulates Trpc1, But Not Trpc6, Channel Function In A Stim1-Independent Manner
Data On Calcium Increases Depending On Stretch In Dystrophic Cardiomyocytes
Saraf Modulates Trpc1, But Not Trpc6, Channel Function In A Stim1-Independent Manner
Data On Calcium Increases Depending On Stretch In Dystrophic Cardiomyocytes
Axial Stretch-Dependent Cation Entry In Dystrophic Cardiomyopathy: Involvement Of Several Trps Channels
Data On Calcium Increases Depending On Stretch In Dystrophic Cardiomyocytes
Axial Stretch-Dependent Cation Entry In Dystrophic Cardiomyopathy: Involvement Of Several Trps Channels
Apelin: An Antithrombotic Factor That Inhibits Platelet Function
Axial Stretch-Dependent Cation Entry In Dystrophic Cardiomyopathy: Involvement Of Several Trps Channels
Apelin: An Antithrombotic Factor That Inhibits Platelet Function
Role Of Mtor1 And Mtor2 Complexes In Meg-01 Cell Physiology
Apelin: An Antithrombotic Factor That Inhibits Platelet Function
Role Of Mtor1 And Mtor2 Complexes In Meg-01 Cell Physiology
Trpc6 Participates In The Regulation Of Cytosolic Basal Calcium Concentration In Murine Resting Platelets
Role Of Mtor1 And Mtor2 Complexes In Meg-01 Cell Physiology
Trpc6 Participates In The Regulation Of Cytosolic Basal Calcium Concentration In Murine Resting Platelets
Synergistic Role Of Protein Phosphatase Inhibitor 1 And Sarco/endoplasmic Reticulum Ca2+-Atpase In The Acquisition Of The Contractile Phenotype Of Arterial Smooth Muscle Cells
Trpc6 Participates In The Regulation Of Cytosolic Basal Calcium Concentration In Murine Resting Platelets
Synergistic Role Of Protein Phosphatase Inhibitor 1 And Sarco/endoplasmic Reticulum Ca2+-Atpase In The Acquisition Of The Contractile Phenotype Of Arterial Smooth Muscle Cells
Cytoskeletal And Scaffolding Proteins As Structural And Functional Determinants Of Trp Channels
Synergistic Role Of Protein Phosphatase Inhibitor 1 And Sarco/endoplasmic Reticulum Ca2+-Atpase In The Acquisition Of The Contractile Phenotype Of Arterial Smooth Muscle Cells
Cytoskeletal And Scaffolding Proteins As Structural And Functional Determinants Of Trp Channels
Transient Receptor Potential Ankyrin-1 (Trpa1) Modulates Store-Operated Ca2+ Entry By Regulation Of Stim1-Orai1 Association
Cytoskeletal And Scaffolding Proteins As Structural And Functional Determinants Of Trp Channels
Transient Receptor Potential Ankyrin-1 (Trpa1) Modulates Store-Operated Ca2+ Entry By Regulation Of Stim1-Orai1 Association
Pharmacology Of Trp Channels In The Vasculature
Transient Receptor Potential Ankyrin-1 (Trpa1) Modulates Store-Operated Ca2+ Entry By Regulation Of Stim1-Orai1 Association
Pharmacology Of Trp Channels In The Vasculature
Homer Proteins In Ca2+ Entry
Pharmacology Of Trp Channels In The Vasculature
Homer Proteins In Ca2+ Entry
The Polybasic Lysine-Rich Domain Of Plasma Membrane-Resident Stim1 Is Essential For The Modulation Of Store-Operated Divalent Cation Entry By Extracellular Calcium
Homer Proteins In Ca2+ Entry
The Polybasic Lysine-Rich Domain Of Plasma Membrane-Resident Stim1 Is Essential For The Modulation Of Store-Operated Divalent Cation Entry By Extracellular Calcium
Crucial Role For Endoplasmic Reticulum Stress During Megakaryocyte Maturation
The Polybasic Lysine-Rich Domain Of Plasma Membrane-Resident Stim1 Is Essential For The Modulation Of Store-Operated Divalent Cation Entry By Extracellular Calcium
Crucial Role For Endoplasmic Reticulum Stress During Megakaryocyte Maturation
Benefit Of Serca2a Gene Transfer To Vascular Endothelial And Smooth Muscle Cells: A New Aspect In Therapy Of Cardiovascular Diseases
Crucial Role For Endoplasmic Reticulum Stress During Megakaryocyte Maturation
Benefit Of Serca2a Gene Transfer To Vascular Endothelial And Smooth Muscle Cells: A New Aspect In Therapy Of Cardiovascular Diseases
Cardiovascular System Soce And Ca2+ Handling In Platelet Dysfunction
Benefit Of Serca2a Gene Transfer To Vascular Endothelial And Smooth Muscle Cells: A New Aspect In Therapy Of Cardiovascular Diseases
Cardiovascular System Soce And Ca2+ Handling In Platelet Dysfunction
Two-Pore Channel 2 (Tpc2) Modulates Store-Operated Ca2+ Entry
Cardiovascular System Soce And Ca2+ Handling In Platelet Dysfunction
Two-Pore Channel 2 (Tpc2) Modulates Store-Operated Ca2+ Entry
Acidic Naadp-Releasable Ca2+ Compartments In The Megakaryoblastic Cell Line Meg01
Two-Pore Channel 2 (Tpc2) Modulates Store-Operated Ca2+ Entry
Acidic Naadp-Releasable Ca2+ Compartments In The Megakaryoblastic Cell Line Meg01
Stim1 And Stim2 Are Located In The Acidic Ca2+ Stores And Associates With Orai1 Upon Depletion Of The Acidic Stores In Human Platelets
Acidic Naadp-Releasable Ca2+ Compartments In The Megakaryoblastic Cell Line Meg01
Stim1 And Stim2 Are Located In The Acidic Ca2+ Stores And Associates With Orai1 Upon Depletion Of The Acidic Stores In Human Platelets
Attenuated Store-Operated Divalent Cation Entry And Association Between Stim1, Orai1, Htrpc1 And Htrpc6 In Platelets From Type 2 Diabetic Patients
Stim1 And Stim2 Are Located In The Acidic Ca2+ Stores And Associates With Orai1 Upon Depletion Of The Acidic Stores In Human Platelets
Attenuated Store-Operated Divalent Cation Entry And Association Between Stim1, Orai1, Htrpc1 And Htrpc6 In Platelets From Type 2 Diabetic Patients
Serca2a Controls The Mode Of Agonist-Induced Intracellular Ca2+ Signal, Transcription Factor Nfat And Proliferation In Human Vascular Smooth Muscle Cells
Attenuated Store-Operated Divalent Cation Entry And Association Between Stim1, Orai1, Htrpc1 And Htrpc6 In Platelets From Type 2 Diabetic Patients
Serca2a Controls The Mode Of Agonist-Induced Intracellular Ca2+ Signal, Transcription Factor Nfat And Proliferation In Human Vascular Smooth Muscle Cells
Trpc3 Regulates Agonist-Stimulated Ca2+ Mobilization By Mediating The Interaction Between Type I Inositol 1,4,5-Trisphosphate Receptor, Rack1, And Orai1
Serca2a Controls The Mode Of Agonist-Induced Intracellular Ca2+ Signal, Transcription Factor Nfat And Proliferation In Human Vascular Smooth Muscle Cells
Trpc3 Regulates Agonist-Stimulated Ca2+ Mobilization By Mediating The Interaction Between Type I Inositol 1,4,5-Trisphosphate Receptor, Rack1, And Orai1
Homocysteine Induces Caspase Activation By Endoplasmic Reticulum Stress In Platelets From Type 2 Diabetics And Healthy Donors
Trpc3 Regulates Agonist-Stimulated Ca2+ Mobilization By Mediating The Interaction Between Type I Inositol 1,4,5-Trisphosphate Receptor, Rack1, And Orai1
Homocysteine Induces Caspase Activation By Endoplasmic Reticulum Stress In Platelets From Type 2 Diabetics And Healthy Donors
Caspase-3 And -9 Are Activated In Human Myeloid Hl-60 Cells By Calcium Signal
Homocysteine Induces Caspase Activation By Endoplasmic Reticulum Stress In Platelets From Type 2 Diabetics And Healthy Donors
Caspase-3 And -9 Are Activated In Human Myeloid Hl-60 Cells By Calcium Signal
Melatonin, As An Adjuvant-Like Agent, Enhances Platelet Responsiveness
Caspase-3 And -9 Are Activated In Human Myeloid Hl-60 Cells By Calcium Signal
Melatonin, As An Adjuvant-Like Agent, Enhances Platelet Responsiveness
Store-Operated Ca2+ Entry Is Sensitive To The Extracellular Ca2+ Concentration Through Plasma Membrane Stim1
Melatonin, As An Adjuvant-Like Agent, Enhances Platelet Responsiveness
Store-Operated Ca2+ Entry Is Sensitive To The Extracellular Ca2+ Concentration Through Plasma Membrane Stim1
Enhanced Expression Of Stim1/orai1 And Trpc3 In Platelets From Patients With Type 2 Diabetes Mellitus
Store-Operated Ca2+ Entry Is Sensitive To The Extracellular Ca2+ Concentration Through Plasma Membrane Stim1
Enhanced Expression Of Stim1/orai1 And Trpc3 In Platelets From Patients With Type 2 Diabetes Mellitus
N,n,n′,n′-Tetrakis(2-Pyridylmethyl)ethylenediamine Induces Apoptosis Through The Activation Of Caspases-3 And -8 In Human Platelets. A Role For Endoplasmic Reticulum Stress
Enhanced Expression Of Stim1/orai1 And Trpc3 In Platelets From Patients With Type 2 Diabetes Mellitus
N,n,n′,n′-Tetrakis(2-Pyridylmethyl)ethylenediamine Induces Apoptosis Through The Activation Of Caspases-3 And -8 In Human Platelets. A Role For Endoplasmic Reticulum Stress
Role Of Intracellular Calcium On Hydrogen Peroxide-Induced Apoptosis In Rat Pancreatic Acinar Ar42j Cells
N,n,n′,n′-Tetrakis(2-Pyridylmethyl)ethylenediamine Induces Apoptosis Through The Activation Of Caspases-3 And -8 In Human Platelets. A Role For Endoplasmic Reticulum Stress
Role Of Intracellular Calcium On Hydrogen Peroxide-Induced Apoptosis In Rat Pancreatic Acinar Ar42j Cells
Orai1 Mediates The Interaction Between Stim1 And Htrpc1 And Regulates The Mode Of Activation Of Htrpc1-Forming Ca2+ Channels
Role Of Intracellular Calcium On Hydrogen Peroxide-Induced Apoptosis In Rat Pancreatic Acinar Ar42j Cells
Orai1 Mediates The Interaction Between Stim1 And Htrpc1 And Regulates The Mode Of Activation Of Htrpc1-Forming Ca2+ Channels
Intracellular Ca2+ Store Depletion Induces The Formation Of Macromolecular Complexes Involving Htrpc1, Htrpc6, The Type Ii Ip3 Receptor And Serca3 In Human Platelets
Orai1 Mediates The Interaction Between Stim1 And Htrpc1 And Regulates The Mode Of Activation Of Htrpc1-Forming Ca2+ Channels
Intracellular Ca2+ Store Depletion Induces The Formation Of Macromolecular Complexes Involving Htrpc1, Htrpc6, The Type Ii Ip3 Receptor And Serca3 In Human Platelets
Stim1 Regulates Acidic Ca2+ Store Refilling By Interaction With Serca3 In Human Platelets
Intracellular Ca2+ Store Depletion Induces The Formation Of Macromolecular Complexes Involving Htrpc1, Htrpc6, The Type Ii Ip3 Receptor And Serca3 In Human Platelets
Stim1 Regulates Acidic Ca2+ Store Refilling By Interaction With Serca3 In Human Platelets
Cinnamtannin B-1 As An Antioxidant And Platelet Aggregation Inhibitor
Stim1 Regulates Acidic Ca2+ Store Refilling By Interaction With Serca3 In Human Platelets
Cinnamtannin B-1 As An Antioxidant And Platelet Aggregation Inhibitor
Functional Relevance Of The De Novo Coupling Between Htrpc1 And Type Ii Ip3 Receptor In Store-Operated Ca2+ Entry In Human Platelets
Cinnamtannin B-1 As An Antioxidant And Platelet Aggregation Inhibitor
Functional Relevance Of The De Novo Coupling Between Htrpc1 And Type Ii Ip3 Receptor In Store-Operated Ca2+ Entry In Human Platelets
Intracellular Calcium Release From Human Platelets: Different Messengers For Multiple Stores
Functional Relevance Of The De Novo Coupling Between Htrpc1 And Type Ii Ip3 Receptor In Store-Operated Ca2+ Entry In Human Platelets
Intracellular Calcium Release From Human Platelets: Different Messengers For Multiple Stores
Thrombin Induces Activation And Translocation Of Bid, Bax And Bak To The Mitochondria In Human Platelets
Intracellular Calcium Release From Human Platelets: Different Messengers For Multiple Stores
Thrombin Induces Activation And Translocation Of Bid, Bax And Bak To The Mitochondria In Human Platelets
Antiaggregant Effects Of Arbutus Unedo Extracts In Human Platelets
Thrombin Induces Activation And Translocation Of Bid, Bax And Bak To The Mitochondria In Human Platelets
Antiaggregant Effects Of Arbutus Unedo Extracts In Human Platelets
Thrombin Induces Apoptotic Events Through The Generation Of Reactive Oxygen Species In Human Platelets
Antiaggregant Effects Of Arbutus Unedo Extracts In Human Platelets
Thrombin Induces Apoptotic Events Through The Generation Of Reactive Oxygen Species In Human Platelets
Tyrosine Phosphorylation/dephosphorylation Balance Is Involved In Thrombin-Evoked Microtubular Reorganisation In Human Platelets
Thrombin Induces Apoptotic Events Through The Generation Of Reactive Oxygen Species In Human Platelets
Tyrosine Phosphorylation/dephosphorylation Balance Is Involved In Thrombin-Evoked Microtubular Reorganisation In Human Platelets
Early Caspase-3 Activation Independent Of Apoptosis Is Required For Cellular Function
Tyrosine Phosphorylation/dephosphorylation Balance Is Involved In Thrombin-Evoked Microtubular Reorganisation In Human Platelets
Early Caspase-3 Activation Independent Of Apoptosis Is Required For Cellular Function
Interaction Of Stim1 With Endogenously Expressed Human Canonical Trp1 Upon Depletion Of Intracellular Ca2+ Stores
Early Caspase-3 Activation Independent Of Apoptosis Is Required For Cellular Function
Interaction Of Stim1 With Endogenously Expressed Human Canonical Trp1 Upon Depletion Of Intracellular Ca2+ Stores
Intracellular Ca2+ Homeostasis And Aggregation In Platelets Are Impaired By Ethanol Through The Generation Of H2o2 And Oxidation Of Sulphydryl Groups
Interaction Of Stim1 With Endogenously Expressed Human Canonical Trp1 Upon Depletion Of Intracellular Ca2+ Stores
Intracellular Ca2+ Homeostasis And Aggregation In Platelets Are Impaired By Ethanol Through The Generation Of H2o2 And Oxidation Of Sulphydryl Groups
Two Distinct Ca2+ Compartments Show Differential Sensitivity To Thrombin, Adp And Vasopressin In Human Platelets
Intracellular Ca2+ Homeostasis And Aggregation In Platelets Are Impaired By Ethanol Through The Generation Of H2o2 And Oxidation Of Sulphydryl Groups
Two Distinct Ca2+ Compartments Show Differential Sensitivity To Thrombin, Adp And Vasopressin In Human Platelets
Ca2+ Accumulation Into Acidic Organelles Mediated By Ca 2+- And Vacuolar H+-Atpases In Human Platelets
Two Distinct Ca2+ Compartments Show Differential Sensitivity To Thrombin, Adp And Vasopressin In Human Platelets
Ca2+ Accumulation Into Acidic Organelles Mediated By Ca 2+- And Vacuolar H+-Atpases In Human Platelets
Two Pathways For Store-Mediated Calcium Entry Differentially Dependent On The Actin Cytoskeleton In Human Platelets
Ca2+ Accumulation Into Acidic Organelles Mediated By Ca 2+- And Vacuolar H+-Atpases In Human Platelets
Two Pathways For Store-Mediated Calcium Entry Differentially Dependent On The Actin Cytoskeleton In Human Platelets
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
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