PUBLICACIÓN

Furin prodomain ppfurin enhances ca2+ entry through orai and trpc6 channels’ activation in breast cancer cells

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ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Lopez J.J. et al.

2021 Cancers

Cancer Research (Q2), Oncology (Q1)

JCR: 6.575

SJR: 1.349

CITAS

14

DOI

10.3390/cancers13071670

EID

2-s2.0-85103318259

EISSN

2072-6694

BIBTEX

@article{L_pez_2021, doi = {10.3390/cancers13071670}, url = {https://doi.org/10.3390%2Fcancers13071670}, year = 2021, month = {apr}, publisher = {{MDPI} {AG}}, volume = {13}, number = {7}, pages = {1670}, author = {Jose J. L{\'{o}}pez and Geraldine Siegfried and Carlos Cantonero and Fabienne Soulet and Jean Descarpentrie and Tarik Smani and Iker Badiola and Simon Pernot and Serge Evrard and Juan A. Rosado and Abdel-Majid Khatib}, title = {Furin Prodomain {ppFurin} Enhances Ca2$\mathplus$ Entry Through Orai and {TRPC}6 Channels' Activation in Breast Cancer Cells}, journal = {Cancers} }

AUTORES DE LA UEX

José Javier López Barba
Juan Antonio Rosado Dionisio

OTRAS PUBLICACIONES DE INTERÉS

Aten, A Didactic Program To Study Gamma Radiation Attenuation Through Matter
Filamin A C-Terminal Fragment Modulates Orai1 Expression By Inhibition Of Protein Degradation
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Filamin A C-Terminal Fragment Modulates Orai1 Expression By Inhibition Of Protein Degradation
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Reduced Ca2+ Mobilization In Neonatal Human Platelets Involves Saraf And Pannexin-1
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Reduced Ca2+ Mobilization In Neonatal Human Platelets Involves Saraf And Pannexin-1
Postbiotics Of Lacticaseibacillus Paracasei Cect 9610 And Lactiplantibacillus Plantarum Cect 9608 Attenuates Store-Operated Calcium Entry And Fak Phosphorylation In Colorectal Cancer Cells
Reduced Ca2+ Mobilization In Neonatal Human Platelets Involves Saraf And Pannexin-1
Postbiotics Of Lacticaseibacillus Paracasei Cect 9610 And Lactiplantibacillus Plantarum Cect 9608 Attenuates Store-Operated Calcium Entry And Fak Phosphorylation In Colorectal Cancer Cells
The Physiological Role Of Trp Channels In Sleep And Circadian Rhythm
Postbiotics Of Lacticaseibacillus Paracasei Cect 9610 And Lactiplantibacillus Plantarum Cect 9608 Attenuates Store-Operated Calcium Entry And Fak Phosphorylation In Colorectal Cancer Cells
The Physiological Role Of Trp Channels In Sleep And Circadian Rhythm
Inhibition Of Adenylyl Cyclase 8 Prevents The Upregulation Of Orai1 Channel, Which Improves Cardiac Function After Myocardial Infarction
The Physiological Role Of Trp Channels In Sleep And Circadian Rhythm
Inhibition Of Adenylyl Cyclase 8 Prevents The Upregulation Of Orai1 Channel, Which Improves Cardiac Function After Myocardial Infarction
Extended Synaptotagmins 1 And 2 Are Required For Store-Operated Calcium Entry, Cell Migration And Viability In Breast Cancer Cells
Inhibition Of Adenylyl Cyclase 8 Prevents The Upregulation Of Orai1 Channel, Which Improves Cardiac Function After Myocardial Infarction
Extended Synaptotagmins 1 And 2 Are Required For Store-Operated Calcium Entry, Cell Migration And Viability In Breast Cancer Cells
Differential Functional Role Of Orai1 Variants In Constitutive Ca2+ Entry And Calcification In Luminal Breast Cancer Cells
Extended Synaptotagmins 1 And 2 Are Required For Store-Operated Calcium Entry, Cell Migration And Viability In Breast Cancer Cells
Differential Functional Role Of Orai1 Variants In Constitutive Ca2+ Entry And Calcification In Luminal Breast Cancer Cells
Role Of Orai-Family Channels In The Activation And Regulation Of Transcriptional Activity
Differential Functional Role Of Orai1 Variants In Constitutive Ca2+ Entry And Calcification In Luminal Breast Cancer Cells
Role Of Orai-Family Channels In The Activation And Regulation Of Transcriptional Activity
Capn1 (Calpain1)-Dependent Cleavage Of Stim1 (Stromal Interaction Molecule 1) Results In An Enhanced Soce (Store-Operated Calcium Entry) In Human Neonatal Platelets
Role Of Orai-Family Channels In The Activation And Regulation Of Transcriptional Activity
Capn1 (Calpain1)-Dependent Cleavage Of Stim1 (Stromal Interaction Molecule 1) Results In An Enhanced Soce (Store-Operated Calcium Entry) In Human Neonatal Platelets
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Capn1 (Calpain1)-Dependent Cleavage Of Stim1 (Stromal Interaction Molecule 1) Results In An Enhanced Soce (Store-Operated Calcium Entry) In Human Neonatal Platelets
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Functional Differences In Agonist-Induced Plasma Membrane Expression Of Orai1α And Orai1β
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Functional Differences In Agonist-Induced Plasma Membrane Expression Of Orai1α And Orai1β
Secretion Of Interleukin 6 In Human Skeletal Muscle Cultures Depends On Ca2+ Signalling
Functional Differences In Agonist-Induced Plasma Membrane Expression Of Orai1α And Orai1β
Secretion Of Interleukin 6 In Human Skeletal Muscle Cultures Depends On Ca2+ Signalling
New Insights Into The Reparative Angiogenesis After Myocardial Infarction
Secretion Of Interleukin 6 In Human Skeletal Muscle Cultures Depends On Ca2+ Signalling
New Insights Into The Reparative Angiogenesis After Myocardial Infarction
The Ca2+ Sensor Stim In Human Diseases
New Insights Into The Reparative Angiogenesis After Myocardial Infarction
The Ca2+ Sensor Stim In Human Diseases
Thrombotic Alterations Under Perinatal Hypoxic Conditions: Hif And Other Hypoxic Markers
The Ca2+ Sensor Stim In Human Diseases
Thrombotic Alterations Under Perinatal Hypoxic Conditions: Hif And Other Hypoxic Markers
Orai1α And Orai1β Support Calcium Entry And Mammosphere Formation In Breast Cancer Stem Cells
Thrombotic Alterations Under Perinatal Hypoxic Conditions: Hif And Other Hypoxic Markers
Orai1α And Orai1β Support Calcium Entry And Mammosphere Formation In Breast Cancer Stem Cells
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Orai1α And Orai1β Support Calcium Entry And Mammosphere Formation In Breast Cancer Stem Cells
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
The Store-Operated Ca2+ Channel Orai1α Is Required For Agonist-Evoked Nf-Κb Activation By A Mechanism Dependent On Pkcβ2
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
The Store-Operated Ca2+ Channel Orai1α Is Required For Agonist-Evoked Nf-Κb Activation By A Mechanism Dependent On Pkcβ2
Store-Operated Calcium Entry In Breast Cancer Cells Is Insensitive To Orai1 And Stim1 N-Linked Glycosylation
The Store-Operated Ca2+ Channel Orai1α Is Required For Agonist-Evoked Nf-Κb Activation By A Mechanism Dependent On Pkcβ2
Store-Operated Calcium Entry In Breast Cancer Cells Is Insensitive To Orai1 And Stim1 N-Linked Glycosylation
Correction To: Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells (Cancers, (2021), 14, 1, (114), 10.3390/cancers14010114)
Store-Operated Calcium Entry In Breast Cancer Cells Is Insensitive To Orai1 And Stim1 N-Linked Glycosylation
Correction To: Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells (Cancers, (2021), 14, 1, (114), 10.3390/cancers14010114)
Pkc-Mediated Orai1 Channel Phosphorylation Modulates Ca2+ Signaling In Hela Cells
Correction To: Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells (Cancers, (2021), 14, 1, (114), 10.3390/cancers14010114)
Pkc-Mediated Orai1 Channel Phosphorylation Modulates Ca2+ Signaling In Hela Cells
Orai1α, But Not Orai1β, Co-Localizes With Trpc1 And Is Required For Its Plasma Membrane Location And Activation In Hela Cells
Pkc-Mediated Orai1 Channel Phosphorylation Modulates Ca2+ Signaling In Hela Cells
Orai1α, But Not Orai1β, Co-Localizes With Trpc1 And Is Required For Its Plasma Membrane Location And Activation In Hela Cells
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Orai1α, But Not Orai1β, Co-Localizes With Trpc1 And Is Required For Its Plasma Membrane Location And Activation In Hela Cells
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Similarities And Differences Between The Orai1 Variants: Orai1α And Orai1β
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Similarities And Differences Between The Orai1 Variants: Orai1α And Orai1β
Store-Operated Calcium Entry And Its Implications In Cancer Stem Cells
Similarities And Differences Between The Orai1 Variants: Orai1α And Orai1β
Store-Operated Calcium Entry And Its Implications In Cancer Stem Cells
A Fibrin Coating Method Of Polypropylene Meshes Enables The Adhesion Of Menstrual Blood-Derived Mesenchymal Stromal Cells: A New Delivery Strategy For Stem Cell-Based Therapies
Store-Operated Calcium Entry And Its Implications In Cancer Stem Cells
A Fibrin Coating Method Of Polypropylene Meshes Enables The Adhesion Of Menstrual Blood-Derived Mesenchymal Stromal Cells: A New Delivery Strategy For Stem Cell-Based Therapies
The Orai1-Ac8 Interplay: How Breast Cancer Cells Escape From Orai1 Channel Inactivation
A Fibrin Coating Method Of Polypropylene Meshes Enables The Adhesion Of Menstrual Blood-Derived Mesenchymal Stromal Cells: A New Delivery Strategy For Stem Cell-Based Therapies
The Orai1-Ac8 Interplay: How Breast Cancer Cells Escape From Orai1 Channel Inactivation
Furin Prodomain Ppfurin Enhances Ca2+ Entry Through Orai And Trpc6 Channels’ Activation In Breast Cancer Cells
The Orai1-Ac8 Interplay: How Breast Cancer Cells Escape From Orai1 Channel Inactivation
Furin Prodomain Ppfurin Enhances Ca2+ Entry Through Orai And Trpc6 Channels’ Activation In Breast Cancer Cells
Corrigendum: Saraf And Orai1 Contribute To Endothelial Cell Activation And Angiogenesis (Frontiers In Cell And Developmental Biology, (2021), 9, (639952), 10.3389/fcell.2021.639952)
Furin Prodomain Ppfurin Enhances Ca2+ Entry Through Orai And Trpc6 Channels’ Activation In Breast Cancer Cells
Corrigendum: Saraf And Orai1 Contribute To Endothelial Cell Activation And Angiogenesis (Frontiers In Cell And Developmental Biology, (2021), 9, (639952), 10.3389/fcell.2021.639952)
Saraf And Orai1 Contribute To Endothelial Cell Activation And Angiogenesis
Corrigendum: Saraf And Orai1 Contribute To Endothelial Cell Activation And Angiogenesis (Frontiers In Cell And Developmental Biology, (2021), 9, (639952), 10.3389/fcell.2021.639952)
Saraf And Orai1 Contribute To Endothelial Cell Activation And Angiogenesis
Tmem97 Facilitates The Activation Of Soce By Downregulating The Association Of Cholesterol To Orai1 In Mda-Mb-231 Cells
Saraf And Orai1 Contribute To Endothelial Cell Activation And Angiogenesis
Tmem97 Facilitates The Activation Of Soce By Downregulating The Association Of Cholesterol To Orai1 In Mda-Mb-231 Cells
Melatonin Downregulates Trpc6, Impairing Store-Operated Calcium Entry In Triple-Negative Breast Cancer Cells
Tmem97 Facilitates The Activation Of Soce By Downregulating The Association Of Cholesterol To Orai1 In Mda-Mb-231 Cells
Melatonin Downregulates Trpc6, Impairing Store-Operated Calcium Entry In Triple-Negative Breast Cancer Cells
Role Of Orai3 In The Pathophysiology Of Cancer
Melatonin Downregulates Trpc6, Impairing Store-Operated Calcium Entry In Triple-Negative Breast Cancer Cells
Role Of Orai3 In The Pathophysiology Of Cancer
Cross-Talk Between The Adenylyl Cyclase/camp Pathway And Ca2+ Homeostasis
Role Of Orai3 In The Pathophysiology Of Cancer
Cross-Talk Between The Adenylyl Cyclase/camp Pathway And Ca2+ Homeostasis
Saraf And Efhb Modulate Store-Operated Ca2+ Entry And Are Required For Cell Proliferation, Migration And Viability In Breast Cancer Cells
Cross-Talk Between The Adenylyl Cyclase/camp Pathway And Ca2+ Homeostasis
Saraf And Efhb Modulate Store-Operated Ca2+ Entry And Are Required For Cell Proliferation, Migration And Viability In Breast Cancer Cells
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Saraf And Efhb Modulate Store-Operated Ca2+ Entry And Are Required For Cell Proliferation, Migration And Viability In Breast Cancer Cells
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Pathophysiological Significance Of Store-Operated Calcium Entry In Cardiovascular And Skeletal Muscle Disorders And Angiogenesis
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Pathophysiological Significance Of Store-Operated Calcium Entry In Cardiovascular And Skeletal Muscle Disorders And Angiogenesis
Ela/apela Precursor Cleaved By Furin Displays Tumor Suppressor Function In Renal Cell Carcinoma Through Mtorc1 Activation
Pathophysiological Significance Of Store-Operated Calcium Entry In Cardiovascular And Skeletal Muscle Disorders And Angiogenesis
Ela/apela Precursor Cleaved By Furin Displays Tumor Suppressor Function In Renal Cell Carcinoma Through Mtorc1 Activation
Anticancer Molecular Mechanisms Of Oleocanthal
Ela/apela Precursor Cleaved By Furin Displays Tumor Suppressor Function In Renal Cell Carcinoma Through Mtorc1 Activation
Anticancer Molecular Mechanisms Of Oleocanthal
Trpc Channels: Dysregulation And Ca2+ Mishandling In Ischemic Heart Disease
Anticancer Molecular Mechanisms Of Oleocanthal
Trpc Channels: Dysregulation And Ca2+ Mishandling In Ischemic Heart Disease
Non-Coding Rnas And Ischemic Cardiovascular Diseases
Trpc Channels: Dysregulation And Ca2+ Mishandling In Ischemic Heart Disease
Non-Coding Rnas And Ischemic Cardiovascular Diseases
Furin Prodomain Ppfurin Enhances Soce Through Trpc6 Activation In Breast Cancer Cells
Non-Coding Rnas And Ischemic Cardiovascular Diseases
Furin Prodomain Ppfurin Enhances Soce Through Trpc6 Activation In Breast Cancer Cells
Experiencias De Docencia Virtual En Facultades De Medicina Españolas Durante La Pandemia Covid-19 (I): Anatomía, Fisiología, Fisiopatología, Oncología
Furin Prodomain Ppfurin Enhances Soce Through Trpc6 Activation In Breast Cancer Cells
Experiencias De Docencia Virtual En Facultades De Medicina Españolas Durante La Pandemia Covid-19 (I): Anatomía, Fisiología, Fisiopatología, Oncología
No1, A New Sigma 2 Receptor/tmem97 Fluorescent Ligand, Downregulates Soce And Promotes Apoptosis In The Triple Negative Breast Cancer Cell Lines
Experiencias De Docencia Virtual En Facultades De Medicina Españolas Durante La Pandemia Covid-19 (I): Anatomía, Fisiología, Fisiopatología, Oncología
No1, A New Sigma 2 Receptor/tmem97 Fluorescent Ligand, Downregulates Soce And Promotes Apoptosis In The Triple Negative Breast Cancer Cell Lines
Trpc6 Channel And Its Implications In Breast Cancer: An Overview
No1, A New Sigma 2 Receptor/tmem97 Fluorescent Ligand, Downregulates Soce And Promotes Apoptosis In The Triple Negative Breast Cancer Cell Lines
Trpc6 Channel And Its Implications In Breast Cancer: An Overview
Pgrmc1 Inhibits Progesterone-Evoked Proliferation And Ca2+ Entry Via Stim2 In Mda-Mb-231 Cells
Trpc6 Channel And Its Implications In Breast Cancer: An Overview
Pgrmc1 Inhibits Progesterone-Evoked Proliferation And Ca2+ Entry Via Stim2 In Mda-Mb-231 Cells
Functional Role Of Trpc6 And Stim2 In Cytosolic And Endoplasmic Reticulum Ca2+ Content In Resting Estrogen Receptor-Positive Breast Cancer Cells
Pgrmc1 Inhibits Progesterone-Evoked Proliferation And Ca2+ Entry Via Stim2 In Mda-Mb-231 Cells
Functional Role Of Trpc6 And Stim2 In Cytosolic And Endoplasmic Reticulum Ca2+ Content In Resting Estrogen Receptor-Positive Breast Cancer Cells
Arachidonic Acid Attenuates Cell Proliferation, Migration And Viability By A Mechanism Independent On Calcium Entry
Functional Role Of Trpc6 And Stim2 In Cytosolic And Endoplasmic Reticulum Ca2+ Content In Resting Estrogen Receptor-Positive Breast Cancer Cells
Arachidonic Acid Attenuates Cell Proliferation, Migration And Viability By A Mechanism Independent On Calcium Entry
Trpc Channels In The Soce Scenario
Arachidonic Acid Attenuates Cell Proliferation, Migration And Viability By A Mechanism Independent On Calcium Entry
Trpc Channels In The Soce Scenario
Molecular Basis And Regulation Of Store-Operated Calcium Entry
Trpc Channels In The Soce Scenario
Molecular Basis And Regulation Of Store-Operated Calcium Entry
Trp Channels: Current Perspectives In The Adverse Cardiac Remodeling
Molecular Basis And Regulation Of Store-Operated Calcium Entry
Trp Channels: Current Perspectives In The Adverse Cardiac Remodeling
Phytochemical, Anti-Diabetic And Cardiovascular Properties Of Urtica Dioica L. (Urticaceae): A Review
Trp Channels: Current Perspectives In The Adverse Cardiac Remodeling
Phytochemical, Anti-Diabetic And Cardiovascular Properties Of Urtica Dioica L. (Urticaceae): A Review
Adenylyl Cyclase Type 8 Overexpression Impairs Phosphorylation-Dependent Orai1 Inactivation And Promotes Migration In Mda-Mb-231 Breast Cancer Cells
Phytochemical, Anti-Diabetic And Cardiovascular Properties Of Urtica Dioica L. (Urticaceae): A Review
Adenylyl Cyclase Type 8 Overexpression Impairs Phosphorylation-Dependent Orai1 Inactivation And Promotes Migration In Mda-Mb-231 Breast Cancer Cells
Flavonoids And Platelet-Derived Thrombotic Disorders
Adenylyl Cyclase Type 8 Overexpression Impairs Phosphorylation-Dependent Orai1 Inactivation And Promotes Migration In Mda-Mb-231 Breast Cancer Cells
Flavonoids And Platelet-Derived Thrombotic Disorders
Store-Independent Orai1-Mediated Ca 2+ Entry And Cancer
Flavonoids And Platelet-Derived Thrombotic Disorders
Store-Independent Orai1-Mediated Ca 2+ Entry And Cancer
Stim1 Phosphorylation At Y316 Modulates Its Interaction With Saraf And The Activation Of Soce And Icrac
Store-Independent Orai1-Mediated Ca 2+ Entry And Cancer
Stim1 Phosphorylation At Y316 Modulates Its Interaction With Saraf And The Activation Of Soce And Icrac
(−)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells
Stim1 Phosphorylation At Y316 Modulates Its Interaction With Saraf And The Activation Of Soce And Icrac
(−)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells
Fine-Tuning Of Micrornas In Type 2 Diabetes Mellitus
(−)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells
Fine-Tuning Of Micrornas In Type 2 Diabetes Mellitus
Special Issue On New Cellular, Genetic And Proteomic Tools In The Prevention And Management Of Diabetes Mellitus
Fine-Tuning Of Micrornas In Type 2 Diabetes Mellitus
Special Issue On New Cellular, Genetic And Proteomic Tools In The Prevention And Management Of Diabetes Mellitus
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus
Special Issue On New Cellular, Genetic And Proteomic Tools In The Prevention And Management Of Diabetes Mellitus
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus
Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus
Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Platelet Functions Are Decreased In Obesity And Restored After Weight Loss: Evidence For A Role Of The Serca3-Dependent Adp Secretion Pathway
Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Platelet Functions Are Decreased In Obesity And Restored After Weight Loss: Evidence For A Role Of The Serca3-Dependent Adp Secretion Pathway
Flavonoids And Platelet-Derived Thrombotic Disorders.
Platelet Functions Are Decreased In Obesity And Restored After Weight Loss: Evidence For A Role Of The Serca3-Dependent Adp Secretion Pathway
Flavonoids And Platelet-Derived Thrombotic Disorders.
(-)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells.
Flavonoids And Platelet-Derived Thrombotic Disorders.
(-)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells.
Urocortin-2 Prevents Dysregulation Of Ca2+ Homeostasis And Improves Early Cardiac Remodeling After Ischemia And Reperfusion
(-)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells.
Urocortin-2 Prevents Dysregulation Of Ca2+ Homeostasis And Improves Early Cardiac Remodeling After Ischemia And Reperfusion
The Complex Role Of Store Operated Calcium Entry Pathways And Related Proteins In The Function Of Cardiac, Skeletal And Vascular Smooth Muscle Cells
Urocortin-2 Prevents Dysregulation Of Ca2+ Homeostasis And Improves Early Cardiac Remodeling After Ischemia And Reperfusion
The Complex Role Of Store Operated Calcium Entry Pathways And Related Proteins In The Function Of Cardiac, Skeletal And Vascular Smooth Muscle Cells
Efhb Is A Novel Cytosolic Ca 2+ Sensor That Modulates Stim1-Saraf Interaction
The Complex Role Of Store Operated Calcium Entry Pathways And Related Proteins In The Function Of Cardiac, Skeletal And Vascular Smooth Muscle Cells
Efhb Is A Novel Cytosolic Ca 2+ Sensor That Modulates Stim1-Saraf Interaction
Trp Channels In Angiogenesis And Other Endothelial Functions
Efhb Is A Novel Cytosolic Ca 2+ Sensor That Modulates Stim1-Saraf Interaction
Trp Channels In Angiogenesis And Other Endothelial Functions
Quimiorrecepción
Trp Channels In Angiogenesis And Other Endothelial Functions
Quimiorrecepción
Secreción Intestinal
Quimiorrecepción
Secreción Intestinal
Store-Operated Ca 2+ Entry In Breast Cancer Cells: Remodeling And Functional Role
Secreción Intestinal
Store-Operated Ca 2+ Entry In Breast Cancer Cells: Remodeling And Functional Role
Trpc6 Channels Are Required For Proliferation, Migration And Invasion Of Breast Cancer Cell Lines By Modulation Of Orai1 And Orai3 Surface Exposure
Store-Operated Ca 2+ Entry In Breast Cancer Cells: Remodeling And Functional Role
Trpc6 Channels Are Required For Proliferation, Migration And Invasion Of Breast Cancer Cell Lines By Modulation Of Orai1 And Orai3 Surface Exposure
Fine-Tuning Of Store-Operated Calcium Entry By Fast And Slow Ca2+-Dependent Inactivation: Involvement Of Saraf
Trpc6 Channels Are Required For Proliferation, Migration And Invasion Of Breast Cancer Cell Lines By Modulation Of Orai1 And Orai3 Surface Exposure
Fine-Tuning Of Store-Operated Calcium Entry By Fast And Slow Ca2+-Dependent Inactivation: Involvement Of Saraf
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets
Fine-Tuning Of Store-Operated Calcium Entry By Fast And Slow Ca2+-Dependent Inactivation: Involvement Of Saraf
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets
Fluorescence-Based Measurements Of The Crac Channel Activity In Cell Populations
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets
Fluorescence-Based Measurements Of The Crac Channel Activity In Cell Populations
Involvement Of Stanniocalcins In The Deregulation Of Glycaemia In Obese Mice And Type 2 Diabetic Patients
Fluorescence-Based Measurements Of The Crac Channel Activity In Cell Populations
Involvement Of Stanniocalcins In The Deregulation Of Glycaemia In Obese Mice And Type 2 Diabetic Patients
Filamin A Modulates Platelet Function
Involvement Of Stanniocalcins In The Deregulation Of Glycaemia In Obese Mice And Type 2 Diabetic Patients
Filamin A Modulates Platelet Function
Role Of Stim1 In The Surface Expression Of Saraf.
Filamin A Modulates Platelet Function
Role Of Stim1 In The Surface Expression Of Saraf.
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus.
Role Of Stim1 In The Surface Expression Of Saraf.
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus.
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets.
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus.
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets.
Fine-Tuning Of Micrornas In Type 2 Diabetes Mellitus.
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets.
Fine-Tuning Of Micrornas In Type 2 Diabetes Mellitus.
Trps In Pain Sensation
Fine-Tuning Of Micrornas In Type 2 Diabetes Mellitus.
Trps In Pain Sensation
Orai1 And Orai2 Mediate Store-Operated Calcium Entry That Regulates Hl60 Cell Migration And Fak Phosphorylation
Trps In Pain Sensation
Orai1 And Orai2 Mediate Store-Operated Calcium Entry That Regulates Hl60 Cell Migration And Fak Phosphorylation
Role Of Stim2 In Cell Function And Physiopathology
Orai1 And Orai2 Mediate Store-Operated Calcium Entry That Regulates Hl60 Cell Migration And Fak Phosphorylation
Role Of Stim2 In Cell Function And Physiopathology
Cardiovascular And Hemostatic Disorders: Soce And Ca2+ Handling In Platelet Dysfunction
Role Of Stim2 In Cell Function And Physiopathology
Cardiovascular And Hemostatic Disorders: Soce And Ca2+ Handling In Platelet Dysfunction
Introduction: Overview Of The Pathophysiological Implications Of Store-Operated Calcium Entry In Mammalian Cells
Cardiovascular And Hemostatic Disorders: Soce And Ca2+ Handling In Platelet Dysfunction
Introduction: Overview Of The Pathophysiological Implications Of Store-Operated Calcium Entry In Mammalian Cells
Mir-125a, Mir-139 And Mir-324 Contribute To Urocortin Protection Against Myocardial Ischemia-Reperfusion Injury
Introduction: Overview Of The Pathophysiological Implications Of Store-Operated Calcium Entry In Mammalian Cells
Mir-125a, Mir-139 And Mir-324 Contribute To Urocortin Protection Against Myocardial Ischemia-Reperfusion Injury
Role Of Homocysteine And Folic Acid On The Altered Calcium Homeostasis Of Platelets From Rats With Biliary Cirrhosis
Mir-125a, Mir-139 And Mir-324 Contribute To Urocortin Protection Against Myocardial Ischemia-Reperfusion Injury
Role Of Homocysteine And Folic Acid On The Altered Calcium Homeostasis Of Platelets From Rats With Biliary Cirrhosis
Role Of Stim1 In The Surface Expression Of Saraf
Role Of Homocysteine And Folic Acid On The Altered Calcium Homeostasis Of Platelets From Rats With Biliary Cirrhosis
Role Of Stim1 In The Surface Expression Of Saraf
Dynamic Interaction Of Saraf With Stim1 And Orai1 To Modulate Store-Operated Calcium Entry
Role Of Stim1 In The Surface Expression Of Saraf
Dynamic Interaction Of Saraf With Stim1 And Orai1 To Modulate Store-Operated Calcium Entry
Molecular Modulators Of Store-Operated Calcium Entry
Dynamic Interaction Of Saraf With Stim1 And Orai1 To Modulate Store-Operated Calcium Entry
Molecular Modulators Of Store-Operated Calcium Entry
Historical Overview Of Store-Operated Ca2+ Entry
Molecular Modulators Of Store-Operated Calcium Entry
Historical Overview Of Store-Operated Ca2+ Entry
Store-Operated Ca2+ Entry- Associated Regulatory Factor (Saraf) Plays An Important Role In The Regulation Of Arachidonate-Regulated Ca2+ (Arc) Channels
Historical Overview Of Store-Operated Ca2+ Entry
Store-Operated Ca2+ Entry- Associated Regulatory Factor (Saraf) Plays An Important Role In The Regulation Of Arachidonate-Regulated Ca2+ (Arc) Channels
Saraf Modulates Trpc1, But Not Trpc6, Channel Function In A Stim1-Independent Manner
Store-Operated Ca2+ Entry- Associated Regulatory Factor (Saraf) Plays An Important Role In The Regulation Of Arachidonate-Regulated Ca2+ (Arc) Channels
Saraf Modulates Trpc1, But Not Trpc6, Channel Function In A Stim1-Independent Manner
Data On Calcium Increases Depending On Stretch In Dystrophic Cardiomyocytes
Saraf Modulates Trpc1, But Not Trpc6, Channel Function In A Stim1-Independent Manner
Data On Calcium Increases Depending On Stretch In Dystrophic Cardiomyocytes
Axial Stretch-Dependent Cation Entry In Dystrophic Cardiomyopathy: Involvement Of Several Trps Channels
Data On Calcium Increases Depending On Stretch In Dystrophic Cardiomyocytes
Axial Stretch-Dependent Cation Entry In Dystrophic Cardiomyopathy: Involvement Of Several Trps Channels
Apelin: An Antithrombotic Factor That Inhibits Platelet Function
Axial Stretch-Dependent Cation Entry In Dystrophic Cardiomyopathy: Involvement Of Several Trps Channels
Apelin: An Antithrombotic Factor That Inhibits Platelet Function
Stim And Calcium Channel Complexes In Cancer
Apelin: An Antithrombotic Factor That Inhibits Platelet Function
Stim And Calcium Channel Complexes In Cancer
Regulation Of Platelet Function By Orai, Stim And Trp
Stim And Calcium Channel Complexes In Cancer
Regulation Of Platelet Function By Orai, Stim And Trp
Stim And Orai1 Variants In Store-Operated Calcium Entry
Regulation Of Platelet Function By Orai, Stim And Trp
Stim And Orai1 Variants In Store-Operated Calcium Entry
Orai1 And Trpc1 Proteins Co-Localize With Cav1.2 Channels To Form A Signal Complex In Vascular Smooth Muscle Cells
Stim And Orai1 Variants In Store-Operated Calcium Entry
Orai1 And Trpc1 Proteins Co-Localize With Cav1.2 Channels To Form A Signal Complex In Vascular Smooth Muscle Cells
Medicinal Plants With Antiplatelet Activity
Orai1 And Trpc1 Proteins Co-Localize With Cav1.2 Channels To Form A Signal Complex In Vascular Smooth Muscle Cells
Medicinal Plants With Antiplatelet Activity
Second Messenger-Operated Calcium Entry Through Trpc6
Medicinal Plants With Antiplatelet Activity
Second Messenger-Operated Calcium Entry Through Trpc6
Phospholipase A2 As A Molecular Determinant Of Store-Operated Calcium Entry
Second Messenger-Operated Calcium Entry Through Trpc6
Phospholipase A2 As A Molecular Determinant Of Store-Operated Calcium Entry
Sigma-1 Receptors: A New Pathway For The Modulation Of Store-Operated Calcium Entry
Phospholipase A2 As A Molecular Determinant Of Store-Operated Calcium Entry
Sigma-1 Receptors: A New Pathway For The Modulation Of Store-Operated Calcium Entry
Stim And Orai1 Variants In Store-Operated Calcium Entry.
Sigma-1 Receptors: A New Pathway For The Modulation Of Store-Operated Calcium Entry
Stim And Orai1 Variants In Store-Operated Calcium Entry.
Relationship Between Calcium Mobilization And Platelet Α- And Δ-Granule Secretion. A Role For Trpc6 In Thrombin-Evoked Δ-Granule Exocytosis
Stim And Orai1 Variants In Store-Operated Calcium Entry.
Relationship Between Calcium Mobilization And Platelet Α- And Δ-Granule Secretion. A Role For Trpc6 In Thrombin-Evoked Δ-Granule Exocytosis
Fkbp25 And Fkbp38 Regulate Non-Capacitative Calcium Entry Through Trpc6
Relationship Between Calcium Mobilization And Platelet Α- And Δ-Granule Secretion. A Role For Trpc6 In Thrombin-Evoked Δ-Granule Exocytosis
Fkbp25 And Fkbp38 Regulate Non-Capacitative Calcium Entry Through Trpc6
Evaluation Of The Antiaggregant Activity Of Ascorbyl Phenolic Esters With Antioxidant Properties
Fkbp25 And Fkbp38 Regulate Non-Capacitative Calcium Entry Through Trpc6
Evaluation Of The Antiaggregant Activity Of Ascorbyl Phenolic Esters With Antioxidant Properties
Homer Proteins Mediate The Interaction Between Stim1 And Cav1.2 Channels
Evaluation Of The Antiaggregant Activity Of Ascorbyl Phenolic Esters With Antioxidant Properties
Homer Proteins Mediate The Interaction Between Stim1 And Cav1.2 Channels
Role Of Mtor1 And Mtor2 Complexes In Meg-01 Cell Physiology
Homer Proteins Mediate The Interaction Between Stim1 And Cav1.2 Channels
Role Of Mtor1 And Mtor2 Complexes In Meg-01 Cell Physiology
Regulators Of G-Protein-Signaling Proteins: Negative Modulators Of G-Protein-Coupled Receptor Signaling
Role Of Mtor1 And Mtor2 Complexes In Meg-01 Cell Physiology
Regulators Of G-Protein-Signaling Proteins: Negative Modulators Of G-Protein-Coupled Receptor Signaling
Store-Operated Calcium Entry: Unveiling The Calcium Handling Signalplex
Regulators Of G-Protein-Signaling Proteins: Negative Modulators Of G-Protein-Coupled Receptor Signaling
Store-Operated Calcium Entry: Unveiling The Calcium Handling Signalplex
Functional And Physiopathological Implications Of Trp Channels
Store-Operated Calcium Entry: Unveiling The Calcium Handling Signalplex
Functional And Physiopathological Implications Of Trp Channels
Physiology: A Practical Textbook
Functional And Physiopathological Implications Of Trp Channels
Physiology: A Practical Textbook
Stim1 Regulates Trpc6 Heteromultimerization And Subcellular Location
Physiology: A Practical Textbook
Stim1 Regulates Trpc6 Heteromultimerization And Subcellular Location
Trpc6 Participates In The Regulation Of Cytosolic Basal Calcium Concentration In Murine Resting Platelets
Stim1 Regulates Trpc6 Heteromultimerization And Subcellular Location
Trpc6 Participates In The Regulation Of Cytosolic Basal Calcium Concentration In Murine Resting Platelets
The Canonical Transient Receptor Potential 6 (Trpc6) Channel Is Sensitive To Extracellular Ph In Mouse Platelets
Trpc6 Participates In The Regulation Of Cytosolic Basal Calcium Concentration In Murine Resting Platelets
The Canonical Transient Receptor Potential 6 (Trpc6) Channel Is Sensitive To Extracellular Ph In Mouse Platelets
Synergistic Role Of Protein Phosphatase Inhibitor 1 And Sarco/endoplasmic Reticulum Ca2+-Atpase In The Acquisition Of The Contractile Phenotype Of Arterial Smooth Muscle Cells
The Canonical Transient Receptor Potential 6 (Trpc6) Channel Is Sensitive To Extracellular Ph In Mouse Platelets
Synergistic Role Of Protein Phosphatase Inhibitor 1 And Sarco/endoplasmic Reticulum Ca2+-Atpase In The Acquisition Of The Contractile Phenotype Of Arterial Smooth Muscle Cells
Cytoskeletal And Scaffolding Proteins As Structural And Functional Determinants Of Trp Channels
Synergistic Role Of Protein Phosphatase Inhibitor 1 And Sarco/endoplasmic Reticulum Ca2+-Atpase In The Acquisition Of The Contractile Phenotype Of Arterial Smooth Muscle Cells
Cytoskeletal And Scaffolding Proteins As Structural And Functional Determinants Of Trp Channels
The Trpv1 Ion Channel Is Expressed In Human But Not Mouse Platelets
Cytoskeletal And Scaffolding Proteins As Structural And Functional Determinants Of Trp Channels
The Trpv1 Ion Channel Is Expressed In Human But Not Mouse Platelets
Endothelial Cytoskeleton
The Trpv1 Ion Channel Is Expressed In Human But Not Mouse Platelets
Endothelial Cytoskeleton
Preface
Endothelial Cytoskeleton
Preface
Transient Receptor Potential Ankyrin-1 (Trpa1) Modulates Store-Operated Ca2+ Entry By Regulation Of Stim1-Orai1 Association
Preface
Transient Receptor Potential Ankyrin-1 (Trpa1) Modulates Store-Operated Ca2+ Entry By Regulation Of Stim1-Orai1 Association
The Membrane Potential Modulates Thrombin-Stimulated Ca2+ Mobilization And Platelet Aggregation
Transient Receptor Potential Ankyrin-1 (Trpa1) Modulates Store-Operated Ca2+ Entry By Regulation Of Stim1-Orai1 Association
The Membrane Potential Modulates Thrombin-Stimulated Ca2+ Mobilization And Platelet Aggregation
Pharmacology Of Trp Channels In The Vasculature
The Membrane Potential Modulates Thrombin-Stimulated Ca2+ Mobilization And Platelet Aggregation
Pharmacology Of Trp Channels In The Vasculature
Homer Proteins In Ca2+ Entry
Pharmacology Of Trp Channels In The Vasculature
Homer Proteins In Ca2+ Entry
Long-Term Mtor Inhibitors Administration Evokes Altered Calcium Homeostasis And Platelet Dysfunction In Kidney Transplant Patients
Homer Proteins In Ca2+ Entry
Long-Term Mtor Inhibitors Administration Evokes Altered Calcium Homeostasis And Platelet Dysfunction In Kidney Transplant Patients
Immunophilins Are Involved In The Altered Platelet Aggregation Observed In Patients With Type 2 Diabetes Mellitus
Long-Term Mtor Inhibitors Administration Evokes Altered Calcium Homeostasis And Platelet Dysfunction In Kidney Transplant Patients
Immunophilins Are Involved In The Altered Platelet Aggregation Observed In Patients With Type 2 Diabetes Mellitus
The Polybasic Lysine-Rich Domain Of Plasma Membrane-Resident Stim1 Is Essential For The Modulation Of Store-Operated Divalent Cation Entry By Extracellular Calcium
Immunophilins Are Involved In The Altered Platelet Aggregation Observed In Patients With Type 2 Diabetes Mellitus
The Polybasic Lysine-Rich Domain Of Plasma Membrane-Resident Stim1 Is Essential For The Modulation Of Store-Operated Divalent Cation Entry By Extracellular Calcium
Fkbp52 Is Involved In The Regulation Of Soce Channels In The Human Platelets And Meg 01 Cells
The Polybasic Lysine-Rich Domain Of Plasma Membrane-Resident Stim1 Is Essential For The Modulation Of Store-Operated Divalent Cation Entry By Extracellular Calcium
Fkbp52 Is Involved In The Regulation Of Soce Channels In The Human Platelets And Meg 01 Cells
Molecular Interplay Between Platelets And The Vascular Wall In Thrombosis And Hemostasis
Fkbp52 Is Involved In The Regulation Of Soce Channels In The Human Platelets And Meg 01 Cells
Molecular Interplay Between Platelets And The Vascular Wall In Thrombosis And Hemostasis
Crucial Role For Endoplasmic Reticulum Stress During Megakaryocyte Maturation
Molecular Interplay Between Platelets And The Vascular Wall In Thrombosis And Hemostasis
Crucial Role For Endoplasmic Reticulum Stress During Megakaryocyte Maturation
Benefit Of Serca2a Gene Transfer To Vascular Endothelial And Smooth Muscle Cells: A New Aspect In Therapy Of Cardiovascular Diseases
Crucial Role For Endoplasmic Reticulum Stress During Megakaryocyte Maturation
Benefit Of Serca2a Gene Transfer To Vascular Endothelial And Smooth Muscle Cells: A New Aspect In Therapy Of Cardiovascular Diseases
Introduction
Benefit Of Serca2a Gene Transfer To Vascular Endothelial And Smooth Muscle Cells: A New Aspect In Therapy Of Cardiovascular Diseases
Introduction
Urotensin-Ii Promotes Vascular Smooth Muscle Cell Proliferation Through Store-Operated Calcium Entry And Egfr Transactivation
Introduction
Urotensin-Ii Promotes Vascular Smooth Muscle Cell Proliferation Through Store-Operated Calcium Entry And Egfr Transactivation
Recent Advances In Cardiovascular And Circulatory Signalling
Urotensin-Ii Promotes Vascular Smooth Muscle Cell Proliferation Through Store-Operated Calcium Entry And Egfr Transactivation
Recent Advances In Cardiovascular And Circulatory Signalling
The Store-Operated Calcium Entry Pathway
Recent Advances In Cardiovascular And Circulatory Signalling
The Store-Operated Calcium Entry Pathway
Vaccination Prepartum Enhances The Beneficial Effects Of Melatonin On The Immune Response And Reduces Platelet Responsiveness In Sheep
The Store-Operated Calcium Entry Pathway
Vaccination Prepartum Enhances The Beneficial Effects Of Melatonin On The Immune Response And Reduces Platelet Responsiveness In Sheep
Unraveling Stim2 Function
Vaccination Prepartum Enhances The Beneficial Effects Of Melatonin On The Immune Response And Reduces Platelet Responsiveness In Sheep
Unraveling Stim2 Function
Cardiovascular System Soce And Ca2+ Handling In Platelet Dysfunction
Unraveling Stim2 Function
Cardiovascular System Soce And Ca2+ Handling In Platelet Dysfunction
Two-Pore Channel 2 (Tpc2) Modulates Store-Operated Ca2+ Entry
Cardiovascular System Soce And Ca2+ Handling In Platelet Dysfunction
Two-Pore Channel 2 (Tpc2) Modulates Store-Operated Ca2+ Entry
Capacitative And Non-Capacitative Signaling Complexes In Human Platelets
Two-Pore Channel 2 (Tpc2) Modulates Store-Operated Ca2+ Entry
Capacitative And Non-Capacitative Signaling Complexes In Human Platelets
Homers Regulate Calcium Entry And Aggregation In Human Platelets: A Role For Homers In The Association Between Stim1 And Orai1
Capacitative And Non-Capacitative Signaling Complexes In Human Platelets
Homers Regulate Calcium Entry And Aggregation In Human Platelets: A Role For Homers In The Association Between Stim1 And Orai1
Stim1 Tyrosine-Phosphorylation Is Required For Stim1-Orai1 Association In Human Platelets
Homers Regulate Calcium Entry And Aggregation In Human Platelets: A Role For Homers In The Association Between Stim1 And Orai1
Stim1 Tyrosine-Phosphorylation Is Required For Stim1-Orai1 Association In Human Platelets
Transient Receptor Potential Channels In Human Platelets: Expression And Functional Role
Stim1 Tyrosine-Phosphorylation Is Required For Stim1-Orai1 Association In Human Platelets
Transient Receptor Potential Channels In Human Platelets: Expression And Functional Role
Store-Operated Ca 2+ Entry
Transient Receptor Potential Channels In Human Platelets: Expression And Functional Role
Store-Operated Ca 2+ Entry
Urotensin-Ii Signaling Mechanism In Rat Coronary Artery: Role Of Stim1 And Orai1-Dependent Store Operated Calcium Influx In Vasoconstriction
Store-Operated Ca 2+ Entry
Urotensin-Ii Signaling Mechanism In Rat Coronary Artery: Role Of Stim1 And Orai1-Dependent Store Operated Calcium Influx In Vasoconstriction
Orais And Stims: Physiological Mechanisms And Disease
Urotensin-Ii Signaling Mechanism In Rat Coronary Artery: Role Of Stim1 And Orai1-Dependent Store Operated Calcium Influx In Vasoconstriction
Orais And Stims: Physiological Mechanisms And Disease
The Trpc Ion Channels: Association With Orai1 And Stim1 Proteins And Participation In Capacitative And Non-Capacitative Calcium Entry
Orais And Stims: Physiological Mechanisms And Disease
The Trpc Ion Channels: Association With Orai1 And Stim1 Proteins And Participation In Capacitative And Non-Capacitative Calcium Entry
Functional Role Of The Calmodulin- And Inositol 1,4,5-Trisphosphate Receptor-Binding (Cirb) Site Of Trpc6 In Human Platelet Activation
The Trpc Ion Channels: Association With Orai1 And Stim1 Proteins And Participation In Capacitative And Non-Capacitative Calcium Entry
Functional Role Of The Calmodulin- And Inositol 1,4,5-Trisphosphate Receptor-Binding (Cirb) Site Of Trpc6 In Human Platelet Activation
The Cytoskeleton Plays A Modulatory Role In The Association Between Stim1 And The Ca2+ Channel Subunits Orai1 And Trpc1
Functional Role Of The Calmodulin- And Inositol 1,4,5-Trisphosphate Receptor-Binding (Cirb) Site Of Trpc6 In Human Platelet Activation
The Cytoskeleton Plays A Modulatory Role In The Association Between Stim1 And The Ca2+ Channel Subunits Orai1 And Trpc1
Acidic Naadp-Releasable Ca2+ Compartments In The Megakaryoblastic Cell Line Meg01
The Cytoskeleton Plays A Modulatory Role In The Association Between Stim1 And The Ca2+ Channel Subunits Orai1 And Trpc1
Acidic Naadp-Releasable Ca2+ Compartments In The Megakaryoblastic Cell Line Meg01
Ca2+ Leakage Rate From Agonist-Sensitive Intracellular Pools Is Altered In Platelets From Patients With Type 2 Diabetes
Acidic Naadp-Releasable Ca2+ Compartments In The Megakaryoblastic Cell Line Meg01
Ca2+ Leakage Rate From Agonist-Sensitive Intracellular Pools Is Altered In Platelets From Patients With Type 2 Diabetes
Stim1 And Stim2 Are Located In The Acidic Ca2+ Stores And Associates With Orai1 Upon Depletion Of The Acidic Stores In Human Platelets
Ca2+ Leakage Rate From Agonist-Sensitive Intracellular Pools Is Altered In Platelets From Patients With Type 2 Diabetes
Stim1 And Stim2 Are Located In The Acidic Ca2+ Stores And Associates With Orai1 Upon Depletion Of The Acidic Stores In Human Platelets
Two Distinct Calcium Pools In The Endoplasmic Reticulum Of Hek-293t Cells
Stim1 And Stim2 Are Located In The Acidic Ca2+ Stores And Associates With Orai1 Upon Depletion Of The Acidic Stores In Human Platelets
Two Distinct Calcium Pools In The Endoplasmic Reticulum Of Hek-293t Cells
Attenuated Store-Operated Divalent Cation Entry And Association Between Stim1, Orai1, Htrpc1 And Htrpc6 In Platelets From Type 2 Diabetic Patients
Two Distinct Calcium Pools In The Endoplasmic Reticulum Of Hek-293t Cells
Attenuated Store-Operated Divalent Cation Entry And Association Between Stim1, Orai1, Htrpc1 And Htrpc6 In Platelets From Type 2 Diabetic Patients
Lipid Rafts Are Essential For The Regulation Of Soce By Plasma Membrane Resident Stim1 In Human Platelets
Attenuated Store-Operated Divalent Cation Entry And Association Between Stim1, Orai1, Htrpc1 And Htrpc6 In Platelets From Type 2 Diabetic Patients
Lipid Rafts Are Essential For The Regulation Of Soce By Plasma Membrane Resident Stim1 In Human Platelets
Immunophilins And Thrombotic Disorders
Lipid Rafts Are Essential For The Regulation Of Soce By Plasma Membrane Resident Stim1 In Human Platelets
Immunophilins And Thrombotic Disorders
Editorial: A Role For Immunophilins In Cellular Signalling In Health And Disease
Immunophilins And Thrombotic Disorders
Editorial: A Role For Immunophilins In Cellular Signalling In Health And Disease
Immunophilin Dysfunction And Neuropathology
Editorial: A Role For Immunophilins In Cellular Signalling In Health And Disease
Immunophilin Dysfunction And Neuropathology
Serca2a Controls The Mode Of Agonist-Induced Intracellular Ca2+ Signal, Transcription Factor Nfat And Proliferation In Human Vascular Smooth Muscle Cells
Immunophilin Dysfunction And Neuropathology
Serca2a Controls The Mode Of Agonist-Induced Intracellular Ca2+ Signal, Transcription Factor Nfat And Proliferation In Human Vascular Smooth Muscle Cells
Modulation Of Platelet Function And Signaling By Flavonoids
Serca2a Controls The Mode Of Agonist-Induced Intracellular Ca2+ Signal, Transcription Factor Nfat And Proliferation In Human Vascular Smooth Muscle Cells
Modulation Of Platelet Function And Signaling By Flavonoids
Acidic Ca2+ Stores In Platelets
Modulation Of Platelet Function And Signaling By Flavonoids
Acidic Ca2+ Stores In Platelets
Role Of Oxidant Scavengers In The Prevention Of Ca2+ Homeostasis Disorders
Acidic Ca2+ Stores In Platelets
Role Of Oxidant Scavengers In The Prevention Of Ca2+ Homeostasis Disorders
Lipid Rafts Modulate The Activation But Not The Maintenance Of Store-Operated Ca2+ Entry
Role Of Oxidant Scavengers In The Prevention Of Ca2+ Homeostasis Disorders
Lipid Rafts Modulate The Activation But Not The Maintenance Of Store-Operated Ca2+ Entry
Homocysteine, Intracellular Signaling And Thrombotic Disorders
Lipid Rafts Modulate The Activation But Not The Maintenance Of Store-Operated Ca2+ Entry
Homocysteine, Intracellular Signaling And Thrombotic Disorders
Synthesis And Evaluation Of The Platelet Antiaggregant Properties Of Phenolic Antioxidants Structurally Related To Rosmarinic Acid
Homocysteine, Intracellular Signaling And Thrombotic Disorders
Synthesis And Evaluation Of The Platelet Antiaggregant Properties Of Phenolic Antioxidants Structurally Related To Rosmarinic Acid
Trpc3 Regulates Agonist-Stimulated Ca2+ Mobilization By Mediating The Interaction Between Type I Inositol 1,4,5-Trisphosphate Receptor, Rack1, And Orai1
Synthesis And Evaluation Of The Platelet Antiaggregant Properties Of Phenolic Antioxidants Structurally Related To Rosmarinic Acid
Trpc3 Regulates Agonist-Stimulated Ca2+ Mobilization By Mediating The Interaction Between Type I Inositol 1,4,5-Trisphosphate Receptor, Rack1, And Orai1
Serca2b Activity Is Regulated By Cyclophilins In Human Platelets
Trpc3 Regulates Agonist-Stimulated Ca2+ Mobilization By Mediating The Interaction Between Type I Inositol 1,4,5-Trisphosphate Receptor, Rack1, And Orai1
Serca2b Activity Is Regulated By Cyclophilins In Human Platelets
Homocysteine Induces Caspase Activation By Endoplasmic Reticulum Stress In Platelets From Type 2 Diabetics And Healthy Donors
Serca2b Activity Is Regulated By Cyclophilins In Human Platelets
Homocysteine Induces Caspase Activation By Endoplasmic Reticulum Stress In Platelets From Type 2 Diabetics And Healthy Donors
Melatonin Reduces Apoptosis Induced By Calcium Signaling In Human Leukocytes: Evidence For The Involvement Of Mitochondria And Bax Activation
Homocysteine Induces Caspase Activation By Endoplasmic Reticulum Stress In Platelets From Type 2 Diabetics And Healthy Donors
Melatonin Reduces Apoptosis Induced By Calcium Signaling In Human Leukocytes: Evidence For The Involvement Of Mitochondria And Bax Activation
Caspase-3 And -9 Are Activated In Human Myeloid Hl-60 Cells By Calcium Signal
Melatonin Reduces Apoptosis Induced By Calcium Signaling In Human Leukocytes: Evidence For The Involvement Of Mitochondria And Bax Activation
Caspase-3 And -9 Are Activated In Human Myeloid Hl-60 Cells By Calcium Signal
Melatonin, As An Adjuvant-Like Agent, Enhances Platelet Responsiveness
Caspase-3 And -9 Are Activated In Human Myeloid Hl-60 Cells By Calcium Signal
Melatonin, As An Adjuvant-Like Agent, Enhances Platelet Responsiveness
Melatonin Enhances The Immune Response To Vaccination Against A1 And C Strains Of Dichelobacter Nodosus
Melatonin, As An Adjuvant-Like Agent, Enhances Platelet Responsiveness
Melatonin Enhances The Immune Response To Vaccination Against A1 And C Strains Of Dichelobacter Nodosus
Stim1, Orai1 And Htrpc1 Are Important For Thrombin- And Adp-Induced Aggregation In Human Platelets
Melatonin Enhances The Immune Response To Vaccination Against A1 And C Strains Of Dichelobacter Nodosus
Stim1, Orai1 And Htrpc1 Are Important For Thrombin- And Adp-Induced Aggregation In Human Platelets
Store-Operated Ca2+ Entry Is Sensitive To The Extracellular Ca2+ Concentration Through Plasma Membrane Stim1
Stim1, Orai1 And Htrpc1 Are Important For Thrombin- And Adp-Induced Aggregation In Human Platelets
Store-Operated Ca2+ Entry Is Sensitive To The Extracellular Ca2+ Concentration Through Plasma Membrane Stim1
Enhanced Expression Of Stim1/orai1 And Trpc3 In Platelets From Patients With Type 2 Diabetes Mellitus
Store-Operated Ca2+ Entry Is Sensitive To The Extracellular Ca2+ Concentration Through Plasma Membrane Stim1
Enhanced Expression Of Stim1/orai1 And Trpc3 In Platelets From Patients With Type 2 Diabetes Mellitus
Dynamic Interaction Of Htrpc6 With The Orai1-Stim1 Complex Or Htrpc3 Mediates Its Role In Capacitative Or Non-Capacitative Ca2+ Entry Pathways
Enhanced Expression Of Stim1/orai1 And Trpc3 In Platelets From Patients With Type 2 Diabetes Mellitus
Dynamic Interaction Of Htrpc6 With The Orai1-Stim1 Complex Or Htrpc3 Mediates Its Role In Capacitative Or Non-Capacitative Ca2+ Entry Pathways
N,n,n′,n′-Tetrakis(2-Pyridylmethyl)ethylenediamine Induces Apoptosis Through The Activation Of Caspases-3 And -8 In Human Platelets. A Role For Endoplasmic Reticulum Stress
Dynamic Interaction Of Htrpc6 With The Orai1-Stim1 Complex Or Htrpc3 Mediates Its Role In Capacitative Or Non-Capacitative Ca2+ Entry Pathways
N,n,n′,n′-Tetrakis(2-Pyridylmethyl)ethylenediamine Induces Apoptosis Through The Activation Of Caspases-3 And -8 In Human Platelets. A Role For Endoplasmic Reticulum Stress
Olive Tree Wood Phenolic Compounds With Human Platelet Antiaggregant Properties
N,n,n′,n′-Tetrakis(2-Pyridylmethyl)ethylenediamine Induces Apoptosis Through The Activation Of Caspases-3 And -8 In Human Platelets. A Role For Endoplasmic Reticulum Stress
Olive Tree Wood Phenolic Compounds With Human Platelet Antiaggregant Properties
Hepatitis C Virus Ns5a And Core Proteins Induce Oxidative Stress-Mediated Calcium Signalling Alterations In Hepatocytes
Olive Tree Wood Phenolic Compounds With Human Platelet Antiaggregant Properties
Hepatitis C Virus Ns5a And Core Proteins Induce Oxidative Stress-Mediated Calcium Signalling Alterations In Hepatocytes
Biochemical And Functional Properties Of The Store-Operated Ca2+ Channels
Hepatitis C Virus Ns5a And Core Proteins Induce Oxidative Stress-Mediated Calcium Signalling Alterations In Hepatocytes
Biochemical And Functional Properties Of The Store-Operated Ca2+ Channels
Trpc Channels And Store-Operated Ca2+ Entry
Biochemical And Functional Properties Of The Store-Operated Ca2+ Channels
Trpc Channels And Store-Operated Ca2+ Entry
Acidic-Store Depletion Is Required For Human Platelet Aggregation
Trpc Channels And Store-Operated Ca2+ Entry
Acidic-Store Depletion Is Required For Human Platelet Aggregation
Melatonin Induces Mitochondrial-Mediated Apoptosis In Human Myeloid Hl-60 Cells
Acidic-Store Depletion Is Required For Human Platelet Aggregation
Melatonin Induces Mitochondrial-Mediated Apoptosis In Human Myeloid Hl-60 Cells
Platelet Function In Hypertension
Melatonin Induces Mitochondrial-Mediated Apoptosis In Human Myeloid Hl-60 Cells
Platelet Function In Hypertension
Role Of Calcium Signals On Hydrogen Peroxide-Induced Apoptosis In Human Myeloid Hl-60 Cells
Platelet Function In Hypertension
Role Of Calcium Signals On Hydrogen Peroxide-Induced Apoptosis In Human Myeloid Hl-60 Cells
Role Of Intracellular Calcium On Hydrogen Peroxide-Induced Apoptosis In Rat Pancreatic Acinar Ar42j Cells
Role Of Calcium Signals On Hydrogen Peroxide-Induced Apoptosis In Human Myeloid Hl-60 Cells
Role Of Intracellular Calcium On Hydrogen Peroxide-Induced Apoptosis In Rat Pancreatic Acinar Ar42j Cells
Effect Of Homocysteine On Calcium Mobilization And Platelet Function In Type 2 Diabetes Mellitus
Role Of Intracellular Calcium On Hydrogen Peroxide-Induced Apoptosis In Rat Pancreatic Acinar Ar42j Cells
Effect Of Homocysteine On Calcium Mobilization And Platelet Function In Type 2 Diabetes Mellitus
Orai1 Mediates The Interaction Between Stim1 And Htrpc1 And Regulates The Mode Of Activation Of Htrpc1-Forming Ca2+ Channels
Effect Of Homocysteine On Calcium Mobilization And Platelet Function In Type 2 Diabetes Mellitus
Orai1 Mediates The Interaction Between Stim1 And Htrpc1 And Regulates The Mode Of Activation Of Htrpc1-Forming Ca2+ Channels
Enhanced Exocytotic-Like Insertion Of Orai1 Into The Plasma Membrane Upon Intracellular Ca2+ Store Depletion
Orai1 Mediates The Interaction Between Stim1 And Htrpc1 And Regulates The Mode Of Activation Of Htrpc1-Forming Ca2+ Channels
Enhanced Exocytotic-Like Insertion Of Orai1 Into The Plasma Membrane Upon Intracellular Ca2+ Store Depletion
Intracellular Ca2+ Store Depletion Induces The Formation Of Macromolecular Complexes Involving Htrpc1, Htrpc6, The Type Ii Ip3 Receptor And Serca3 In Human Platelets
Enhanced Exocytotic-Like Insertion Of Orai1 Into The Plasma Membrane Upon Intracellular Ca2+ Store Depletion
Intracellular Ca2+ Store Depletion Induces The Formation Of Macromolecular Complexes Involving Htrpc1, Htrpc6, The Type Ii Ip3 Receptor And Serca3 In Human Platelets
Stim1 Regulates Acidic Ca2+ Store Refilling By Interaction With Serca3 In Human Platelets
Intracellular Ca2+ Store Depletion Induces The Formation Of Macromolecular Complexes Involving Htrpc1, Htrpc6, The Type Ii Ip3 Receptor And Serca3 In Human Platelets
Stim1 Regulates Acidic Ca2+ Store Refilling By Interaction With Serca3 In Human Platelets
Cinnamtannin B-1 As An Antioxidant And Platelet Aggregation Inhibitor
Stim1 Regulates Acidic Ca2+ Store Refilling By Interaction With Serca3 In Human Platelets
Cinnamtannin B-1 As An Antioxidant And Platelet Aggregation Inhibitor
Functional Relevance Of The De Novo Coupling Between Htrpc1 And Type Ii Ip3 Receptor In Store-Operated Ca2+ Entry In Human Platelets
Cinnamtannin B-1 As An Antioxidant And Platelet Aggregation Inhibitor
Functional Relevance Of The De Novo Coupling Between Htrpc1 And Type Ii Ip3 Receptor In Store-Operated Ca2+ Entry In Human Platelets
Intracellular Calcium Release From Human Platelets: Different Messengers For Multiple Stores
Functional Relevance Of The De Novo Coupling Between Htrpc1 And Type Ii Ip3 Receptor In Store-Operated Ca2+ Entry In Human Platelets
Intracellular Calcium Release From Human Platelets: Different Messengers For Multiple Stores
Serca2b And 3 Play A Regulatory Role In Store-Operated Calcium Entry In Human Platelets
Intracellular Calcium Release From Human Platelets: Different Messengers For Multiple Stores
Serca2b And 3 Play A Regulatory Role In Store-Operated Calcium Entry In Human Platelets
Protein Complex Immunological Separation Assay (Procisa): A Technique For Investigating Single Protein Properties
Serca2b And 3 Play A Regulatory Role In Store-Operated Calcium Entry In Human Platelets
Protein Complex Immunological Separation Assay (Procisa): A Technique For Investigating Single Protein Properties
Role Of Lipid Rafts In The Interaction Between Htrpc1, Orai1 And Stim1
Protein Complex Immunological Separation Assay (Procisa): A Technique For Investigating Single Protein Properties
Role Of Lipid Rafts In The Interaction Between Htrpc1, Orai1 And Stim1
Phosphatidylinositol 4,5-Bisphosphate Enhances Store-Operated Calcium Entry Through Htrpc6 Channel In Human Platelets
Role Of Lipid Rafts In The Interaction Between Htrpc1, Orai1 And Stim1
Phosphatidylinositol 4,5-Bisphosphate Enhances Store-Operated Calcium Entry Through Htrpc6 Channel In Human Platelets
Thrombin Induces Activation And Translocation Of Bid, Bax And Bak To The Mitochondria In Human Platelets
Phosphatidylinositol 4,5-Bisphosphate Enhances Store-Operated Calcium Entry Through Htrpc6 Channel In Human Platelets
Thrombin Induces Activation And Translocation Of Bid, Bax And Bak To The Mitochondria In Human Platelets
Physical Properties Of Two Types Of Calcium Stores And Sercas In Human Platelets
Thrombin Induces Activation And Translocation Of Bid, Bax And Bak To The Mitochondria In Human Platelets
Physical Properties Of Two Types Of Calcium Stores And Sercas In Human Platelets
Chapter 3 Natriuretic Peptides In Vascular Physiology And Pathology
Physical Properties Of Two Types Of Calcium Stores And Sercas In Human Platelets
Chapter 3 Natriuretic Peptides In Vascular Physiology And Pathology
Platelet Signalling Abnormalities In Patients With Type 2 Diabetes Mellitus: A Review
Chapter 3 Natriuretic Peptides In Vascular Physiology And Pathology
Platelet Signalling Abnormalities In Patients With Type 2 Diabetes Mellitus: A Review
Corrigendum To Receptor Subtypes For Vasonatrin Peptide In Renal Glomeruli And Arteries [Regul. Pept. 129 (2005) 183-189] (Doi:10.1016/j.regpep.2005.02.006)
Platelet Signalling Abnormalities In Patients With Type 2 Diabetes Mellitus: A Review
Corrigendum To Receptor Subtypes For Vasonatrin Peptide In Renal Glomeruli And Arteries [Regul. Pept. 129 (2005) 183-189] (Doi:10.1016/j.regpep.2005.02.006)
Corrigendum To Renal Atrial Natriuretic Peptide Receptors Binding Properties And Function Are Resistant To Doca-Salt-Induced Hypertension In Rats [Regul. Pept. 137 (2006) 114-120] (Doi:10.1016/j.regpep.2006.06.003)
Corrigendum To Receptor Subtypes For Vasonatrin Peptide In Renal Glomeruli And Arteries [Regul. Pept. 129 (2005) 183-189] (Doi:10.1016/j.regpep.2005.02.006)
Corrigendum To Renal Atrial Natriuretic Peptide Receptors Binding Properties And Function Are Resistant To Doca-Salt-Induced Hypertension In Rats [Regul. Pept. 137 (2006) 114-120] (Doi:10.1016/j.regpep.2006.06.003)
Effect Of Homocysteine On Calcium Mobilization And Platelet Function In Type 2 Diabetes Mellitus
Corrigendum To Renal Atrial Natriuretic Peptide Receptors Binding Properties And Function Are Resistant To Doca-Salt-Induced Hypertension In Rats [Regul. Pept. 137 (2006) 114-120] (Doi:10.1016/j.regpep.2006.06.003)
Effect Of Homocysteine On Calcium Mobilization And Platelet Function In Type 2 Diabetes Mellitus
Preface
Effect Of Homocysteine On Calcium Mobilization And Platelet Function In Type 2 Diabetes Mellitus
Preface
Antiaggregant Effects Of Arbutus Unedo Extracts In Human Platelets
Preface
Antiaggregant Effects Of Arbutus Unedo Extracts In Human Platelets
Involvement Of Snare Proteins In Thrombin-Induced Platelet Aggregation: Evidence For The Relevance Of Ca2+ Entry
Antiaggregant Effects Of Arbutus Unedo Extracts In Human Platelets
Involvement Of Snare Proteins In Thrombin-Induced Platelet Aggregation: Evidence For The Relevance Of Ca2+ Entry
Characterization Of The Intracellular Mechanisms Involved In The Antiaggregant Properties Of Cinnamtannin B-1 From Bay Wood In Human Platelets
Involvement Of Snare Proteins In Thrombin-Induced Platelet Aggregation: Evidence For The Relevance Of Ca2+ Entry
Characterization Of The Intracellular Mechanisms Involved In The Antiaggregant Properties Of Cinnamtannin B-1 From Bay Wood In Human Platelets
Thrombin Induces Apoptotic Events Through The Generation Of Reactive Oxygen Species In Human Platelets
Characterization Of The Intracellular Mechanisms Involved In The Antiaggregant Properties Of Cinnamtannin B-1 From Bay Wood In Human Platelets
Thrombin Induces Apoptotic Events Through The Generation Of Reactive Oxygen Species In Human Platelets
Cinnamtannin B-1 From Bay Wood Exhibits Antiapoptotic Effects In Human Platelets
Thrombin Induces Apoptotic Events Through The Generation Of Reactive Oxygen Species In Human Platelets
Cinnamtannin B-1 From Bay Wood Exhibits Antiapoptotic Effects In Human Platelets
Cinnamtannin B-1 From Bay Wood Reduces Abnormal Intracellular Ca2+ Homeostasis And Platelet Hyperaggregability In Type 2 Diabetes Mellitus Patients
Cinnamtannin B-1 From Bay Wood Exhibits Antiapoptotic Effects In Human Platelets
Cinnamtannin B-1 From Bay Wood Reduces Abnormal Intracellular Ca2+ Homeostasis And Platelet Hyperaggregability In Type 2 Diabetes Mellitus Patients
Tyrosine Phosphorylation/dephosphorylation Balance Is Involved In Thrombin-Evoked Microtubular Reorganisation In Human Platelets
Cinnamtannin B-1 From Bay Wood Reduces Abnormal Intracellular Ca2+ Homeostasis And Platelet Hyperaggregability In Type 2 Diabetes Mellitus Patients
Tyrosine Phosphorylation/dephosphorylation Balance Is Involved In Thrombin-Evoked Microtubular Reorganisation In Human Platelets
Urtica Dioica Extract Reduces Platelet Hyperaggregability In Type 2 Diabetes Mellitus By Inhibition Of Oxidant Production, Ca2+ Mobilization And Protein Tyrosine Phosphorylation
Tyrosine Phosphorylation/dephosphorylation Balance Is Involved In Thrombin-Evoked Microtubular Reorganisation In Human Platelets
Urtica Dioica Extract Reduces Platelet Hyperaggregability In Type 2 Diabetes Mellitus By Inhibition Of Oxidant Production, Ca2+ Mobilization And Protein Tyrosine Phosphorylation
Differential Involvement Of Thrombin Receptors In Ca2+ Release From Two Different Intracellular Stores In Human Platelets
Urtica Dioica Extract Reduces Platelet Hyperaggregability In Type 2 Diabetes Mellitus By Inhibition Of Oxidant Production, Ca2+ Mobilization And Protein Tyrosine Phosphorylation
Differential Involvement Of Thrombin Receptors In Ca2+ Release From Two Different Intracellular Stores In Human Platelets
Htrpc1-Associated Α-Actinin, And Not Htrpc1 Itself, Is Tyrosine Phosphorylated During Human Platelet Activation
Differential Involvement Of Thrombin Receptors In Ca2+ Release From Two Different Intracellular Stores In Human Platelets
Htrpc1-Associated Α-Actinin, And Not Htrpc1 Itself, Is Tyrosine Phosphorylated During Human Platelet Activation
Dual Role Of Tubulin-Cytoskeleton In Store-Operated Calcium Entry In Human Platelets
Htrpc1-Associated Α-Actinin, And Not Htrpc1 Itself, Is Tyrosine Phosphorylated During Human Platelet Activation
Dual Role Of Tubulin-Cytoskeleton In Store-Operated Calcium Entry In Human Platelets
Apoptotic Events In Blood Cells
Dual Role Of Tubulin-Cytoskeleton In Store-Operated Calcium Entry In Human Platelets
Apoptotic Events In Blood Cells
Recent Advances In Natriuretic Peptide Research: Molecular Medicine
Apoptotic Events In Blood Cells
Recent Advances In Natriuretic Peptide Research: Molecular Medicine
Altered Calcium Signalling In Platelets From Bile-Duct-Ligated Rats
Recent Advances In Natriuretic Peptide Research: Molecular Medicine
Altered Calcium Signalling In Platelets From Bile-Duct-Ligated Rats
Transient Receptor Potential Channels And Intracellular Signaling
Altered Calcium Signalling In Platelets From Bile-Duct-Ligated Rats
Transient Receptor Potential Channels And Intracellular Signaling
Involvement Of Ryanodine-Operated Channels In Tert-Butylhydroperoxide- Evoked Ca2+ Mobilisation In Pancreatic Acinar Cells
Transient Receptor Potential Channels And Intracellular Signaling
Involvement Of Ryanodine-Operated Channels In Tert-Butylhydroperoxide- Evoked Ca2+ Mobilisation In Pancreatic Acinar Cells
Early Caspase-3 Activation Independent Of Apoptosis Is Required For Cellular Function
Involvement Of Ryanodine-Operated Channels In Tert-Butylhydroperoxide- Evoked Ca2+ Mobilisation In Pancreatic Acinar Cells
Early Caspase-3 Activation Independent Of Apoptosis Is Required For Cellular Function
Interaction Of Stim1 With Endogenously Expressed Human Canonical Trp1 Upon Depletion Of Intracellular Ca2+ Stores
Early Caspase-3 Activation Independent Of Apoptosis Is Required For Cellular Function
Interaction Of Stim1 With Endogenously Expressed Human Canonical Trp1 Upon Depletion Of Intracellular Ca2+ Stores
Endogenously Generated Reactive Oxygen Species Reduce Pmca Activity In Platelets From Patients With Non-Insulin-Dependent Diabetes Mellitus
Interaction Of Stim1 With Endogenously Expressed Human Canonical Trp1 Upon Depletion Of Intracellular Ca2+ Stores
Endogenously Generated Reactive Oxygen Species Reduce Pmca Activity In Platelets From Patients With Non-Insulin-Dependent Diabetes Mellitus
Intracellular Ca2+ Homeostasis And Aggregation In Platelets Are Impaired By Ethanol Through The Generation Of H2o2 And Oxidation Of Sulphydryl Groups
Endogenously Generated Reactive Oxygen Species Reduce Pmca Activity In Platelets From Patients With Non-Insulin-Dependent Diabetes Mellitus
Intracellular Ca2+ Homeostasis And Aggregation In Platelets Are Impaired By Ethanol Through The Generation Of H2o2 And Oxidation Of Sulphydryl Groups
Thrombin-Induced Caspases 3 And 9 Translocation To The Cytoskeleton Is Independent Of Changes In Cytosolic Calcium In Human Platelets
Intracellular Ca2+ Homeostasis And Aggregation In Platelets Are Impaired By Ethanol Through The Generation Of H2o2 And Oxidation Of Sulphydryl Groups
Thrombin-Induced Caspases 3 And 9 Translocation To The Cytoskeleton Is Independent Of Changes In Cytosolic Calcium In Human Platelets
Calcium Signalling And Reactive Oxygen Species In Non-Excitable Cells
Thrombin-Induced Caspases 3 And 9 Translocation To The Cytoskeleton Is Independent Of Changes In Cytosolic Calcium In Human Platelets
Calcium Signalling And Reactive Oxygen Species In Non-Excitable Cells
Alterations In Intracellular Calcium Homeostasis And Platelet Aggregation Induced By Ethanol
Calcium Signalling And Reactive Oxygen Species In Non-Excitable Cells
Alterations In Intracellular Calcium Homeostasis And Platelet Aggregation Induced By Ethanol
Two Distinct Ca2+ Compartments Show Differential Sensitivity To Thrombin, Adp And Vasopressin In Human Platelets
Alterations In Intracellular Calcium Homeostasis And Platelet Aggregation Induced By Ethanol
Two Distinct Ca2+ Compartments Show Differential Sensitivity To Thrombin, Adp And Vasopressin In Human Platelets
Roles Of Calcium And Tyrosine Kinases In The Pathogenesis Of Type 2 Diabetes Mellitus
Two Distinct Ca2+ Compartments Show Differential Sensitivity To Thrombin, Adp And Vasopressin In Human Platelets
Roles Of Calcium And Tyrosine Kinases In The Pathogenesis Of Type 2 Diabetes Mellitus
Caspases 3 And 9 Are Translocated To The Cytoskeleton And Activated By Thrombin In Human Platelets. Evidence For The Involvement Of Pkc And The Actin Filament Polymerization
Roles Of Calcium And Tyrosine Kinases In The Pathogenesis Of Type 2 Diabetes Mellitus
Caspases 3 And 9 Are Translocated To The Cytoskeleton And Activated By Thrombin In Human Platelets. Evidence For The Involvement Of Pkc And The Actin Filament Polymerization
A Role For 5,6-Epoxyeicosatrienoic Acid In Calcium Entry By De Novo Conformational Coupling In Human Platelets
Caspases 3 And 9 Are Translocated To The Cytoskeleton And Activated By Thrombin In Human Platelets. Evidence For The Involvement Of Pkc And The Actin Filament Polymerization
A Role For 5,6-Epoxyeicosatrienoic Acid In Calcium Entry By De Novo Conformational Coupling In Human Platelets
A Role For Cofilin In The Activation Of Store-Operated Calcium Entry By De Novo Conformational Coupling In Human Platelets
A Role For 5,6-Epoxyeicosatrienoic Acid In Calcium Entry By De Novo Conformational Coupling In Human Platelets
A Role For Cofilin In The Activation Of Store-Operated Calcium Entry By De Novo Conformational Coupling In Human Platelets
Renal Atrial Natriuretic Peptide Receptors Binding Properties And Function Are Resistant To Doca-Salt-Induced Hypertension In Rats
A Role For Cofilin In The Activation Of Store-Operated Calcium Entry By De Novo Conformational Coupling In Human Platelets
Renal Atrial Natriuretic Peptide Receptors Binding Properties And Function Are Resistant To Doca-Salt-Induced Hypertension In Rats
Discovering The Mechanism Of Capacitative Calcium Entry
Renal Atrial Natriuretic Peptide Receptors Binding Properties And Function Are Resistant To Doca-Salt-Induced Hypertension In Rats
Discovering The Mechanism Of Capacitative Calcium Entry
Different Effect Of Atp On Anp Receptor Guanylyl Cyclase In Spontaneously Hypertensive And Normotensive Rats
Discovering The Mechanism Of Capacitative Calcium Entry
Different Effect Of Atp On Anp Receptor Guanylyl Cyclase In Spontaneously Hypertensive And Normotensive Rats
Inhibitory Effect Of Ca2+ On Atp-Mediated Stimulation Of Npr-A-Coupled Guanylyl Cyclase In Renal Glomeruli From Spontaneously Hypertensive And Normotensive Rats
Different Effect Of Atp On Anp Receptor Guanylyl Cyclase In Spontaneously Hypertensive And Normotensive Rats
Inhibitory Effect Of Ca2+ On Atp-Mediated Stimulation Of Npr-A-Coupled Guanylyl Cyclase In Renal Glomeruli From Spontaneously Hypertensive And Normotensive Rats
Store-Operated Ca2+ Entry: Vesicle Fusion Or Reversible Trafficking And De Novo Conformational Coupling?
Inhibitory Effect Of Ca2+ On Atp-Mediated Stimulation Of Npr-A-Coupled Guanylyl Cyclase In Renal Glomeruli From Spontaneously Hypertensive And Normotensive Rats
Store-Operated Ca2+ Entry: Vesicle Fusion Or Reversible Trafficking And De Novo Conformational Coupling?
Dynamics Of Calcium Fluxes In Human Platelets Assessed In Calcium-Free Medium
Store-Operated Ca2+ Entry: Vesicle Fusion Or Reversible Trafficking And De Novo Conformational Coupling?
Dynamics Of Calcium Fluxes In Human Platelets Assessed In Calcium-Free Medium
Ca2+ Accumulation Into Acidic Organelles Mediated By Ca 2+- And Vacuolar H+-Atpases In Human Platelets
Dynamics Of Calcium Fluxes In Human Platelets Assessed In Calcium-Free Medium
Ca2+ Accumulation Into Acidic Organelles Mediated By Ca 2+- And Vacuolar H+-Atpases In Human Platelets
Hydrogen Peroxide And Peroxynitrite Enhance Ca2+ Mobilization And Aggregation In Platelets From Type 2 Diabetic Patients
Ca2+ Accumulation Into Acidic Organelles Mediated By Ca 2+- And Vacuolar H+-Atpases In Human Platelets
Hydrogen Peroxide And Peroxynitrite Enhance Ca2+ Mobilization And Aggregation In Platelets From Type 2 Diabetic Patients
Collaborative Effect Of Serca And Pmca In Cytosolic Calcium Homeostasis In Human Platelets
Hydrogen Peroxide And Peroxynitrite Enhance Ca2+ Mobilization And Aggregation In Platelets From Type 2 Diabetic Patients
Collaborative Effect Of Serca And Pmca In Cytosolic Calcium Homeostasis In Human Platelets
Ca2+-Independent Activation Of Bruton's Tyrosine Kinase Is Required For Store-Mediated Ca2+ Entry In Human Platelets
Collaborative Effect Of Serca And Pmca In Cytosolic Calcium Homeostasis In Human Platelets
Ca2+-Independent Activation Of Bruton's Tyrosine Kinase Is Required For Store-Mediated Ca2+ Entry In Human Platelets
Cleavage Of Snap-25 And Vamp-2 Impairs Store-Operated Ca2+ Entry In Mouse Pancreatic Acinar Cells
Ca2+-Independent Activation Of Bruton's Tyrosine Kinase Is Required For Store-Mediated Ca2+ Entry In Human Platelets
Cleavage Of Snap-25 And Vamp-2 Impairs Store-Operated Ca2+ Entry In Mouse Pancreatic Acinar Cells
Receptor Subtypes For Vasonatrin Peptide In Renal Glomeruli And Arteries
Cleavage Of Snap-25 And Vamp-2 Impairs Store-Operated Ca2+ Entry In Mouse Pancreatic Acinar Cells
Receptor Subtypes For Vasonatrin Peptide In Renal Glomeruli And Arteries
Characteristics Of The Renal C-Type Natriuretic Peptide Receptor In Hypertrophied And Developing Rat Kidney
Receptor Subtypes For Vasonatrin Peptide In Renal Glomeruli And Arteries
Characteristics Of The Renal C-Type Natriuretic Peptide Receptor In Hypertrophied And Developing Rat Kidney
Disruption Of The Filamentous Actin Cytoskeleton Is Necessary For The Activation Of Capacitative Calcium Entry In Naive Smooth Muscle Cells
Characteristics Of The Renal C-Type Natriuretic Peptide Receptor In Hypertrophied And Developing Rat Kidney
Disruption Of The Filamentous Actin Cytoskeleton Is Necessary For The Activation Of Capacitative Calcium Entry In Naive Smooth Muscle Cells
G-Protein Coupled Receptors And Calcium Signaling In Development.
Disruption Of The Filamentous Actin Cytoskeleton Is Necessary For The Activation Of Capacitative Calcium Entry In Naive Smooth Muscle Cells
G-Protein Coupled Receptors And Calcium Signaling In Development.
Dietary Virgin Olive Oil Enhances Secretagogue-Evoked Calcium Signaling In Rat Pancreatic Acinar Cells
G-Protein Coupled Receptors And Calcium Signaling In Development.
Dietary Virgin Olive Oil Enhances Secretagogue-Evoked Calcium Signaling In Rat Pancreatic Acinar Cells
Store-Operated Ca 2+ Entry And Tyrosine Kinase Pp60 Src Hyperactivity Are Modulated By Hyperglycemia In Platelets From Patients With Non Insulin-Dependent Diabetes Mellitus
Dietary Virgin Olive Oil Enhances Secretagogue-Evoked Calcium Signaling In Rat Pancreatic Acinar Cells
Store-Operated Ca 2+ Entry And Tyrosine Kinase Pp60 Src Hyperactivity Are Modulated By Hyperglycemia In Platelets From Patients With Non Insulin-Dependent Diabetes Mellitus
Two Pathways For Store-Mediated Calcium Entry Differentially Dependent On The Actin Cytoskeleton In Human Platelets
Store-Operated Ca 2+ Entry And Tyrosine Kinase Pp60 Src Hyperactivity Are Modulated By Hyperglycemia In Platelets From Patients With Non Insulin-Dependent Diabetes Mellitus
Two Pathways For Store-Mediated Calcium Entry Differentially Dependent On The Actin Cytoskeleton In Human Platelets
A Role For Snap-25 But Not Vamps In Store-Mediated Ca2+ Entry In Human Platelets
Two Pathways For Store-Mediated Calcium Entry Differentially Dependent On The Actin Cytoskeleton In Human Platelets
A Role For Snap-25 But Not Vamps In Store-Mediated Ca2+ Entry In Human Platelets
Dual Effect Of Hydrogen Peroxide On Store-Mediated Calcium Entry In Human Platelets
A Role For Snap-25 But Not Vamps In Store-Mediated Ca2+ Entry In Human Platelets
Dual Effect Of Hydrogen Peroxide On Store-Mediated Calcium Entry In Human Platelets
Effect Of Hydrogen Peroxide On Ca2+ Mobilisation In Human Platelets Through Sulphydryl Oxidation Dependent And Independent Mechanisms
Dual Effect Of Hydrogen Peroxide On Store-Mediated Calcium Entry In Human Platelets
Effect Of Hydrogen Peroxide On Ca2+ Mobilisation In Human Platelets Through Sulphydryl Oxidation Dependent And Independent Mechanisms
Hydrogen Peroxide Generation Induces Pp60src Activation In Human Platelets: Evidence For The Involvement Of This Pathway In Store-Mediated Calcium Entry
Effect Of Hydrogen Peroxide On Ca2+ Mobilisation In Human Platelets Through Sulphydryl Oxidation Dependent And Independent Mechanisms
Hydrogen Peroxide Generation Induces Pp60src Activation In Human Platelets: Evidence For The Involvement Of This Pathway In Store-Mediated Calcium Entry
G-Protein Coupled Receptors And Calcium Signaling In Development
Hydrogen Peroxide Generation Induces Pp60src Activation In Human Platelets: Evidence For The Involvement Of This Pathway In Store-Mediated Calcium Entry
G-Protein Coupled Receptors And Calcium Signaling In Development
Patterning Of Renal Cgmp Production By The Natriuretic Peptide Receptor Type A And Blood Pressure In Spontaneously Hypertensive Rats
G-Protein Coupled Receptors And Calcium Signaling In Development
Patterning Of Renal Cgmp Production By The Natriuretic Peptide Receptor Type A And Blood Pressure In Spontaneously Hypertensive Rats
Water Deprivation Enhances The Inhibitory Effect Of Natriuretic Peptides On Camp Synthesis In Rat Renal Glomeruli
Patterning Of Renal Cgmp Production By The Natriuretic Peptide Receptor Type A And Blood Pressure In Spontaneously Hypertensive Rats
Water Deprivation Enhances The Inhibitory Effect Of Natriuretic Peptides On Camp Synthesis In Rat Renal Glomeruli
Reduced Plasma Membrane Ca2+-Atpase Function In Platelets From Patients With Non-Insulin-Dependent Diabetes Mellitus
Water Deprivation Enhances The Inhibitory Effect Of Natriuretic Peptides On Camp Synthesis In Rat Renal Glomeruli
Reduced Plasma Membrane Ca2+-Atpase Function In Platelets From Patients With Non-Insulin-Dependent Diabetes Mellitus
Evidence For Secretion-Like Coupling Involving Pp60src In The Activation And Maintenance Of Store-Mediated Ca2+ Entry In Mouse Pancreatic Acinar Cells
Reduced Plasma Membrane Ca2+-Atpase Function In Platelets From Patients With Non-Insulin-Dependent Diabetes Mellitus
Evidence For Secretion-Like Coupling Involving Pp60src In The Activation And Maintenance Of Store-Mediated Ca2+ Entry In Mouse Pancreatic Acinar Cells
Differences Between Natriuretic Peptide Receptors In The Olfactory Bulb And Hypothalamus From Spontaneously Hypertensive And Normotensive Rat Brain
Evidence For Secretion-Like Coupling Involving Pp60src In The Activation And Maintenance Of Store-Mediated Ca2+ Entry In Mouse Pancreatic Acinar Cells
Differences Between Natriuretic Peptide Receptors In The Olfactory Bulb And Hypothalamus From Spontaneously Hypertensive And Normotensive Rat Brain
Effects Of Reactive Oxygen Species On Actin Filament Polymerisation And Amylase Secretion In Mouse Pancreatic Acinar Cells
Differences Between Natriuretic Peptide Receptors In The Olfactory Bulb And Hypothalamus From Spontaneously Hypertensive And Normotensive Rat Brain
Effects Of Reactive Oxygen Species On Actin Filament Polymerisation And Amylase Secretion In Mouse Pancreatic Acinar Cells
Trp Channels And Calcium Entry In Human Platelets [3] (Multiple Letters)
Effects Of Reactive Oxygen Species On Actin Filament Polymerisation And Amylase Secretion In Mouse Pancreatic Acinar Cells
Trp Channels And Calcium Entry In Human Platelets [3] (Multiple Letters)
Endogenously Expressed Trp1 Is Involved In Store-Mediated Ca2+ Entry By Conformational Coupling In Human Platelets
Trp Channels And Calcium Entry In Human Platelets [3] (Multiple Letters)
Endogenously Expressed Trp1 Is Involved In Store-Mediated Ca2+ Entry By Conformational Coupling In Human Platelets
A-Type Natriuretic Peptide Receptor In The Spontaneously Hypertensive Rat Kidney
Endogenously Expressed Trp1 Is Involved In Store-Mediated Ca2+ Entry By Conformational Coupling In Human Platelets
A-Type Natriuretic Peptide Receptor In The Spontaneously Hypertensive Rat Kidney
The Erk Cascade, A New Pathway Involved In The Activation Of Store-Mediated Calcium Entry In Human Platelets
A-Type Natriuretic Peptide Receptor In The Spontaneously Hypertensive Rat Kidney
The Erk Cascade, A New Pathway Involved In The Activation Of Store-Mediated Calcium Entry In Human Platelets
Dendroaspis Natriuretic Peptide-Like Immunoreactivity And Its Regulation In Rat Aortic Vascular Smooth Muscle
The Erk Cascade, A New Pathway Involved In The Activation Of Store-Mediated Calcium Entry In Human Platelets
Dendroaspis Natriuretic Peptide-Like Immunoreactivity And Its Regulation In Rat Aortic Vascular Smooth Muscle
Expression And Control Of C-Type Natriuretic Peptide In Rat Vascular Smooth Muscle Cells
Dendroaspis Natriuretic Peptide-Like Immunoreactivity And Its Regulation In Rat Aortic Vascular Smooth Muscle
Expression And Control Of C-Type Natriuretic Peptide In Rat Vascular Smooth Muscle Cells
Activation Of Store-Mediated Calcium Entry By Secretion-Like Coupling Between The Inositol 1,4,5-Trisphosphate Receptor Type Ii And Human Transient Receptor Potential (Htrp1) Channels In Human Platelets
Expression And Control Of C-Type Natriuretic Peptide In Rat Vascular Smooth Muscle Cells
Activation Of Store-Mediated Calcium Entry By Secretion-Like Coupling Between The Inositol 1,4,5-Trisphosphate Receptor Type Ii And Human Transient Receptor Potential (Htrp1) Channels In Human Platelets
Role Of The Erk Pathway In The Activation Of Store-Mediated Calcium Entry In Human Platelets
Activation Of Store-Mediated Calcium Entry By Secretion-Like Coupling Between The Inositol 1,4,5-Trisphosphate Receptor Type Ii And Human Transient Receptor Potential (Htrp1) Channels In Human Platelets
Role Of The Erk Pathway In The Activation Of Store-Mediated Calcium Entry In Human Platelets
Cyclic Nucleotides Modulate Store-Mediated Calcium Entry Through The Activation Of Protein-Tyrosine Phosphatases And Altered Actin Polymerization In Human Platelets
Role Of The Erk Pathway In The Activation Of Store-Mediated Calcium Entry In Human Platelets
Cyclic Nucleotides Modulate Store-Mediated Calcium Entry Through The Activation Of Protein-Tyrosine Phosphatases And Altered Actin Polymerization In Human Platelets
Fibrinogen Binding To The Integrin Αiibβ3 Modulates Store-Mediated Calcium Entry In Human Platelets
Cyclic Nucleotides Modulate Store-Mediated Calcium Entry Through The Activation Of Protein-Tyrosine Phosphatases And Altered Actin Polymerization In Human Platelets
Fibrinogen Binding To The Integrin Αiibβ3 Modulates Store-Mediated Calcium Entry In Human Platelets
The Inositol Trisphosphate Receptor Antagonist 2-Aminoethoxydiphenylborate (2-Apb) Blocks Ca2+ Entry Channels In Human Platelets: Cautions For Its Use In Studying Ca2+ Influx
Fibrinogen Binding To The Integrin Αiibβ3 Modulates Store-Mediated Calcium Entry In Human Platelets
The Inositol Trisphosphate Receptor Antagonist 2-Aminoethoxydiphenylborate (2-Apb) Blocks Ca2+ Entry Channels In Human Platelets: Cautions For Its Use In Studying Ca2+ Influx
Tumor Necrosis Factor-Α Inhibits Store-Mediated Ca2+ Entry In The Human Hepatocellular Carcinoma Cell Line Hepg2
The Inositol Trisphosphate Receptor Antagonist 2-Aminoethoxydiphenylborate (2-Apb) Blocks Ca2+ Entry Channels In Human Platelets: Cautions For Its Use In Studying Ca2+ Influx
Tumor Necrosis Factor-Α Inhibits Store-Mediated Ca2+ Entry In The Human Hepatocellular Carcinoma Cell Line Hepg2
Activation Of M3 Muscarinic Receptors Induces Rapid Tyrosine Phosphorylation Of P125(Fak), P130(Cas), And Paxillin In Rat Pancreatic Acini
Tumor Necrosis Factor-Α Inhibits Store-Mediated Ca2+ Entry In The Human Hepatocellular Carcinoma Cell Line Hepg2
Activation Of M3 Muscarinic Receptors Induces Rapid Tyrosine Phosphorylation Of P125(Fak), P130(Cas), And Paxillin In Rat Pancreatic Acini
A Role For Phosphoinositides In Tyrosine Phosphorylation Of P125 Focal Adhesion Kinase In Rat Pancreatic Acini
Activation Of M3 Muscarinic Receptors Induces Rapid Tyrosine Phosphorylation Of P125(Fak), P130(Cas), And Paxillin In Rat Pancreatic Acini
A Role For Phosphoinositides In Tyrosine Phosphorylation Of P125 Focal Adhesion Kinase In Rat Pancreatic Acini
Protein Kinase C Activates Non-Capacitative Calcium Entry In Human Platelets
A Role For Phosphoinositides In Tyrosine Phosphorylation Of P125 Focal Adhesion Kinase In Rat Pancreatic Acini
Protein Kinase C Activates Non-Capacitative Calcium Entry In Human Platelets
A Role For The Actin Cytoskeleton In The Initiation And Maintenance Of Store-Mediated Calcium Entry In Human Platelets
Protein Kinase C Activates Non-Capacitative Calcium Entry In Human Platelets
A Role For The Actin Cytoskeleton In The Initiation And Maintenance Of Store-Mediated Calcium Entry In Human Platelets
Tyrosine Kinases Activate Store-Mediated Ca2+ Entry In Human Platelets Through The Reorganization Of The Actin Cytoskeleton
A Role For The Actin Cytoskeleton In The Initiation And Maintenance Of Store-Mediated Calcium Entry In Human Platelets
Tyrosine Kinases Activate Store-Mediated Ca2+ Entry In Human Platelets Through The Reorganization Of The Actin Cytoskeleton
Coupling Between Inositol 1,4,5-Trisphosphate Receptors And Human Transient Receptor Potential Channel 1 When Intracellular Ca2+ Stores Are Depleted
Tyrosine Kinases Activate Store-Mediated Ca2+ Entry In Human Platelets Through The Reorganization Of The Actin Cytoskeleton
Coupling Between Inositol 1,4,5-Trisphosphate Receptors And Human Transient Receptor Potential Channel 1 When Intracellular Ca2+ Stores Are Depleted
The Actin Cytoskeleton In Store-Mediated Calcium Entry
Coupling Between Inositol 1,4,5-Trisphosphate Receptors And Human Transient Receptor Potential Channel 1 When Intracellular Ca2+ Stores Are Depleted
The Actin Cytoskeleton In Store-Mediated Calcium Entry
Regulation Of Plasma Membrane Ca2+-Atpase By Small Gtpases And Phosphoinositides In Human Platelets
The Actin Cytoskeleton In Store-Mediated Calcium Entry
Regulation Of Plasma Membrane Ca2+-Atpase By Small Gtpases And Phosphoinositides In Human Platelets
Farnesylcysteine Analogues Inhibit Store-Regulated Ca2+ Entry In Human Platelets: Evidence For Involvement Of Small Gtp-Binding Proteins And Actin Cytoskeleton
Regulation Of Plasma Membrane Ca2+-Atpase By Small Gtpases And Phosphoinositides In Human Platelets
Farnesylcysteine Analogues Inhibit Store-Regulated Ca2+ Entry In Human Platelets: Evidence For Involvement Of Small Gtp-Binding Proteins And Actin Cytoskeleton
Phosphoinositides Are Required For Store-Mediated Calcium Entry In Human Platelets
Farnesylcysteine Analogues Inhibit Store-Regulated Ca2+ Entry In Human Platelets: Evidence For Involvement Of Small Gtp-Binding Proteins And Actin Cytoskeleton
Phosphoinositides Are Required For Store-Mediated Calcium Entry In Human Platelets
A Role For The Actin Cytoskeleton In The Initiation And Maintenance Of Store-Mediated Calcium Entry In Human Platelets. Evidence For Conformational Coupling
Phosphoinositides Are Required For Store-Mediated Calcium Entry In Human Platelets
A Role For The Actin Cytoskeleton In The Initiation And Maintenance Of Store-Mediated Calcium Entry In Human Platelets. Evidence For Conformational Coupling
Are Tyrosine Phosphorylation Of P125(Fak) And Paxillin Or The Small Gtp Binding Protein, Rho, Needed For Cck-Stimulated Pancreatic Amylase Secretion?
A Role For The Actin Cytoskeleton In The Initiation And Maintenance Of Store-Mediated Calcium Entry In Human Platelets. Evidence For Conformational Coupling
Are Tyrosine Phosphorylation Of P125(Fak) And Paxillin Or The Small Gtp Binding Protein, Rho, Needed For Cck-Stimulated Pancreatic Amylase Secretion?
Acetylcholine-Evoked Potassium Transport In The Isolated Guinea-Pig Pancreas
Are Tyrosine Phosphorylation Of P125(Fak) And Paxillin Or The Small Gtp Binding Protein, Rho, Needed For Cck-Stimulated Pancreatic Amylase Secretion?
Acetylcholine-Evoked Potassium Transport In The Isolated Guinea-Pig Pancreas
Cck Causes Rapid Tyrosine Phosphorylation Of P125(Fak) Focal Adhesion Kinase And Paxillin In Rat Pancreatic Acini
Acetylcholine-Evoked Potassium Transport In The Isolated Guinea-Pig Pancreas
Cck Causes Rapid Tyrosine Phosphorylation Of P125(Fak) Focal Adhesion Kinase And Paxillin In Rat Pancreatic Acini
Ionic Requirements In Histamine-Evoked Potassium Efflux In Guinea Pig Pancreas
Cck Causes Rapid Tyrosine Phosphorylation Of P125(Fak) Focal Adhesion Kinase And Paxillin In Rat Pancreatic Acini
Ionic Requirements In Histamine-Evoked Potassium Efflux In Guinea Pig Pancreas
Cholecystokinin-Stimulated Tyrosine Phosphorylation Of P125fak And Paxillin Is Mediated By Phospholipase C-Dependent And -Independent Mechanisms And Requires The Integrity Of The Actin Cytoskeleton And Participation Of P21rho
Ionic Requirements In Histamine-Evoked Potassium Efflux In Guinea Pig Pancreas
Cholecystokinin-Stimulated Tyrosine Phosphorylation Of P125fak And Paxillin Is Mediated By Phospholipase C-Dependent And -Independent Mechanisms And Requires The Integrity Of The Actin Cytoskeleton And Participation Of P21rho
Secretin Potentiates Guinea Pig Pancreatic Response To Cholecystokinin By A Cholinergic Mechanism
Cholecystokinin-Stimulated Tyrosine Phosphorylation Of P125fak And Paxillin Is Mediated By Phospholipase C-Dependent And -Independent Mechanisms And Requires The Integrity Of The Actin Cytoskeleton And Participation Of P21rho
Secretin Potentiates Guinea Pig Pancreatic Response To Cholecystokinin By A Cholinergic Mechanism
Histamine-Evoked Potassium Release In The Mouse And Guinea Pig Pancreas
Secretin Potentiates Guinea Pig Pancreatic Response To Cholecystokinin By A Cholinergic Mechanism
Histamine-Evoked Potassium Release In The Mouse And Guinea Pig Pancreas
Aten, A Didactic Program To Study Gamma Radiation Attenuation Through Matter
Histamine-Evoked Potassium Release In The Mouse And Guinea Pig Pancreas
Aten, A Didactic Program To Study Gamma Radiation Attenuation Through Matter
Filamin A C-Terminal Fragment Modulates Orai1 Expression By Inhibition Of Protein Degradation
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