PUBLICACIÓN

New insights into adipokines as potential biomarkers for type-2 diabetes mellitus.

ACCEDER A LA PUBLICACIÓN: Orcid

2017 Current medicinal chemistry

DOI

10.2174/0929867325666171205162248

AUTORES DE LA UEX

Pedro Cosme Redondo Liberal
José Javier López Barba

OTRAS PUBLICACIONES DE INTERÉS

Evidence For Secretion-Like Coupling Involving Pp60src In The Activation And Maintenance Of Store-Mediated Ca2+ Entry In Mouse Pancreatic Acinar Cells
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
Filamin A C-Terminal Fragment Modulates Orai1 Expression By Inhibition Of Protein Degradation
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
Filamin A C-Terminal Fragment Modulates Orai1 Expression By Inhibition Of Protein Degradation
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Filamin A C-Terminal Fragment Modulates Orai1 Expression By Inhibition Of Protein Degradation
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Effects Of Twelve Weeks Of Square Stepping Exercises On Physical And Cognitive Function And Plasma Content Of Smp30: A Randomised Control Trial
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Effects Of Twelve Weeks Of Square Stepping Exercises On Physical And Cognitive Function And Plasma Content Of Smp30: A Randomised Control Trial
Efcab13 Is A Novel Age-Dependent Promotor Of Soce In Blood Platelets And Megakaryoblastic Meg-01 Cells
Effects Of Twelve Weeks Of Square Stepping Exercises On Physical And Cognitive Function And Plasma Content Of Smp30: A Randomised Control Trial
Efcab13 Is A Novel Age-Dependent Promotor Of Soce In Blood Platelets And Megakaryoblastic Meg-01 Cells
Orai1 Plays A Critical Role In Orai3 Synthesis And Stability In Luminal Breast Cancer Cells
Efcab13 Is A Novel Age-Dependent Promotor Of Soce In Blood Platelets And Megakaryoblastic Meg-01 Cells
Orai1 Plays A Critical Role In Orai3 Synthesis And Stability In Luminal Breast Cancer Cells
Stanniocalcin2, A Promising New Target For Identifying Patients With Stroke/ictus
Orai1 Plays A Critical Role In Orai3 Synthesis And Stability In Luminal Breast Cancer Cells
Stanniocalcin2, A Promising New Target For Identifying Patients With Stroke/ictus
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Stanniocalcin2, A Promising New Target For Identifying Patients With Stroke/ictus
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Saraf Overexpression Impairs Thrombin-Induced Ca2+ Homeostasis In Neonatal Platelets
Feedback Modulation Of Orai1α And Orai1β Protein Content Mediated By Stim Proteins
Saraf Overexpression Impairs Thrombin-Induced Ca2+ Homeostasis In Neonatal Platelets
Postbiotics Of Lacticaseibacillus Paracasei Cect 9610 And Lactiplantibacillus Plantarum Cect 9608 Attenuates Store-Operated Calcium Entry And Fak Phosphorylation In Colorectal Cancer Cells
Saraf Overexpression Impairs Thrombin-Induced Ca2+ Homeostasis In Neonatal Platelets
Postbiotics Of Lacticaseibacillus Paracasei Cect 9610 And Lactiplantibacillus Plantarum Cect 9608 Attenuates Store-Operated Calcium Entry And Fak Phosphorylation In Colorectal Cancer Cells
Extended Synaptotagmins 1 And 2 Are Required For Store-Operated Calcium Entry, Cell Migration And Viability In Breast Cancer Cells
Postbiotics Of Lacticaseibacillus Paracasei Cect 9610 And Lactiplantibacillus Plantarum Cect 9608 Attenuates Store-Operated Calcium Entry And Fak Phosphorylation In Colorectal Cancer Cells
Extended Synaptotagmins 1 And 2 Are Required For Store-Operated Calcium Entry, Cell Migration And Viability In Breast Cancer Cells
Differential Functional Role Of Orai1 Variants In Constitutive Ca2+ Entry And Calcification In Luminal Breast Cancer Cells
Extended Synaptotagmins 1 And 2 Are Required For Store-Operated Calcium Entry, Cell Migration And Viability In Breast Cancer Cells
Differential Functional Role Of Orai1 Variants In Constitutive Ca2+ Entry And Calcification In Luminal Breast Cancer Cells
Role Of Orai-Family Channels In The Activation And Regulation Of Transcriptional Activity
Differential Functional Role Of Orai1 Variants In Constitutive Ca2+ Entry And Calcification In Luminal Breast Cancer Cells
Role Of Orai-Family Channels In The Activation And Regulation Of Transcriptional Activity
Capn1 (Calpain1)-Dependent Cleavage Of Stim1 (Stromal Interaction Molecule 1) Results In An Enhanced Soce (Store-Operated Calcium Entry) In Human Neonatal Platelets
Role Of Orai-Family Channels In The Activation And Regulation Of Transcriptional Activity
Capn1 (Calpain1)-Dependent Cleavage Of Stim1 (Stromal Interaction Molecule 1) Results In An Enhanced Soce (Store-Operated Calcium Entry) In Human Neonatal Platelets
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Capn1 (Calpain1)-Dependent Cleavage Of Stim1 (Stromal Interaction Molecule 1) Results In An Enhanced Soce (Store-Operated Calcium Entry) In Human Neonatal Platelets
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S2 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Table S1 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Supplementary Materials And Methods From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Functional Differences In Agonist-Induced Plasma Membrane Expression Of Orai1α And Orai1β
Figures S1-S4 From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Functional Differences In Agonist-Induced Plasma Membrane Expression Of Orai1α And Orai1β
The Ca2+ Sensor Stim In Human Diseases
Functional Differences In Agonist-Induced Plasma Membrane Expression Of Orai1α And Orai1β
The Ca2+ Sensor Stim In Human Diseases
Thrombotic Alterations Under Perinatal Hypoxic Conditions: Hif And Other Hypoxic Markers
The Ca2+ Sensor Stim In Human Diseases
Thrombotic Alterations Under Perinatal Hypoxic Conditions: Hif And Other Hypoxic Markers
Orai1α And Orai1β Support Calcium Entry And Mammosphere Formation In Breast Cancer Stem Cells
Thrombotic Alterations Under Perinatal Hypoxic Conditions: Hif And Other Hypoxic Markers
Orai1α And Orai1β Support Calcium Entry And Mammosphere Formation In Breast Cancer Stem Cells
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Orai1α And Orai1β Support Calcium Entry And Mammosphere Formation In Breast Cancer Stem Cells
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
The Store-Operated Ca2+ Channel Orai1α Is Required For Agonist-Evoked Nf-Κb Activation By A Mechanism Dependent On Pkcβ2
Data From Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
The Store-Operated Ca2+ Channel Orai1α Is Required For Agonist-Evoked Nf-Κb Activation By A Mechanism Dependent On Pkcβ2
Store-Operated Calcium Entry In Breast Cancer Cells Is Insensitive To Orai1 And Stim1 N-Linked Glycosylation
The Store-Operated Ca2+ Channel Orai1α Is Required For Agonist-Evoked Nf-Κb Activation By A Mechanism Dependent On Pkcβ2
Store-Operated Calcium Entry In Breast Cancer Cells Is Insensitive To Orai1 And Stim1 N-Linked Glycosylation
Correction To: Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells (Cancers, (2021), 14, 1, (114), 10.3390/cancers14010114)
Store-Operated Calcium Entry In Breast Cancer Cells Is Insensitive To Orai1 And Stim1 N-Linked Glycosylation
Correction To: Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells (Cancers, (2021), 14, 1, (114), 10.3390/cancers14010114)
Orai1α, But Not Orai1β, Co-Localizes With Trpc1 And Is Required For Its Plasma Membrane Location And Activation In Hela Cells
Correction To: Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells (Cancers, (2021), 14, 1, (114), 10.3390/cancers14010114)
Orai1α, But Not Orai1β, Co-Localizes With Trpc1 And Is Required For Its Plasma Membrane Location And Activation In Hela Cells
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Orai1α, But Not Orai1β, Co-Localizes With Trpc1 And Is Required For Its Plasma Membrane Location And Activation In Hela Cells
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Similarities And Differences Between The Orai1 Variants: Orai1α And Orai1β
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Similarities And Differences Between The Orai1 Variants: Orai1α And Orai1β
Store-Operated Calcium Entry And Its Implications In Cancer Stem Cells
Similarities And Differences Between The Orai1 Variants: Orai1α And Orai1β
Store-Operated Calcium Entry And Its Implications In Cancer Stem Cells
New Evidence On Regucalcin, Body Composition, And Walking Ability Adaptations To Multicomponent Exercise Training In Functionally Limited And Frail Older Adults
Store-Operated Calcium Entry And Its Implications In Cancer Stem Cells
New Evidence On Regucalcin, Body Composition, And Walking Ability Adaptations To Multicomponent Exercise Training In Functionally Limited And Frail Older Adults
A Fibrin Coating Method Of Polypropylene Meshes Enables The Adhesion Of Menstrual Blood-Derived Mesenchymal Stromal Cells: A New Delivery Strategy For Stem Cell-Based Therapies
New Evidence On Regucalcin, Body Composition, And Walking Ability Adaptations To Multicomponent Exercise Training In Functionally Limited And Frail Older Adults
A Fibrin Coating Method Of Polypropylene Meshes Enables The Adhesion Of Menstrual Blood-Derived Mesenchymal Stromal Cells: A New Delivery Strategy For Stem Cell-Based Therapies
The Orai1-Ac8 Interplay: How Breast Cancer Cells Escape From Orai1 Channel Inactivation
A Fibrin Coating Method Of Polypropylene Meshes Enables The Adhesion Of Menstrual Blood-Derived Mesenchymal Stromal Cells: A New Delivery Strategy For Stem Cell-Based Therapies
The Orai1-Ac8 Interplay: How Breast Cancer Cells Escape From Orai1 Channel Inactivation
Furin Prodomain Ppfurin Enhances Ca2+ Entry Through Orai And Trpc6 Channels’ Activation In Breast Cancer Cells
The Orai1-Ac8 Interplay: How Breast Cancer Cells Escape From Orai1 Channel Inactivation
Furin Prodomain Ppfurin Enhances Ca2+ Entry Through Orai And Trpc6 Channels’ Activation In Breast Cancer Cells
Tmem97 Facilitates The Activation Of Soce By Downregulating The Association Of Cholesterol To Orai1 In Mda-Mb-231 Cells
Furin Prodomain Ppfurin Enhances Ca2+ Entry Through Orai And Trpc6 Channels’ Activation In Breast Cancer Cells
Tmem97 Facilitates The Activation Of Soce By Downregulating The Association Of Cholesterol To Orai1 In Mda-Mb-231 Cells
Saraf And Efhb Modulate Store-Operated Ca2+ Entry And Are Required For Cell Proliferation, Migration And Viability In Breast Cancer Cells
Tmem97 Facilitates The Activation Of Soce By Downregulating The Association Of Cholesterol To Orai1 In Mda-Mb-231 Cells
Saraf And Efhb Modulate Store-Operated Ca2+ Entry And Are Required For Cell Proliferation, Migration And Viability In Breast Cancer Cells
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
Saraf And Efhb Modulate Store-Operated Ca2+ Entry And Are Required For Cell Proliferation, Migration And Viability In Breast Cancer Cells
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
New Evidence On Regucalcin, Body Composition, And Walking Ability Adaptations To Multicomponent Exercise Training In Functionally Limited And Frail Older Adults
Orai2 Modulates Store-Operated Ca2+ Entry And Cell Cycle Progression In Breast Cancer Cells
New Evidence On Regucalcin, Body Composition, And Walking Ability Adaptations To Multicomponent Exercise Training In Functionally Limited And Frail Older Adults
Role Of Orai3 In The Pathophysiology Of Cancer
New Evidence On Regucalcin, Body Composition, And Walking Ability Adaptations To Multicomponent Exercise Training In Functionally Limited And Frail Older Adults
Role Of Orai3 In The Pathophysiology Of Cancer
Cross-Talk Between The Adenylyl Cyclase/camp Pathway And Ca2+ Homeostasis
Role Of Orai3 In The Pathophysiology Of Cancer
Cross-Talk Between The Adenylyl Cyclase/camp Pathway And Ca2+ Homeostasis
Pathophysiological Significance Of Store-Operated Calcium Entry In Cardiovascular And Skeletal Muscle Disorders And Angiogenesis
Cross-Talk Between The Adenylyl Cyclase/camp Pathway And Ca2+ Homeostasis
Pathophysiological Significance Of Store-Operated Calcium Entry In Cardiovascular And Skeletal Muscle Disorders And Angiogenesis
Ela/apela Precursor Cleaved By Furin Displays Tumor Suppressor Function In Renal Cell Carcinoma Through Mtorc1 Activation
Pathophysiological Significance Of Store-Operated Calcium Entry In Cardiovascular And Skeletal Muscle Disorders And Angiogenesis
Ela/apela Precursor Cleaved By Furin Displays Tumor Suppressor Function In Renal Cell Carcinoma Through Mtorc1 Activation
Furin Prodomain Ppfurin Enhances Soce Through Trpc6 Activation In Breast Cancer Cells
Ela/apela Precursor Cleaved By Furin Displays Tumor Suppressor Function In Renal Cell Carcinoma Through Mtorc1 Activation
Furin Prodomain Ppfurin Enhances Soce Through Trpc6 Activation In Breast Cancer Cells
No1, A New Sigma 2 Receptor/tmem97 Fluorescent Ligand, Downregulates Soce And Promotes Apoptosis In The Triple Negative Breast Cancer Cell Lines
Furin Prodomain Ppfurin Enhances Soce Through Trpc6 Activation In Breast Cancer Cells
No1, A New Sigma 2 Receptor/tmem97 Fluorescent Ligand, Downregulates Soce And Promotes Apoptosis In The Triple Negative Breast Cancer Cell Lines
Twelve Weeks Of Whole Body Vibration Training Improve Regucalcin, Body Composition And Physical Fitness In Postmenopausal Women: A Pilot Study
No1, A New Sigma 2 Receptor/tmem97 Fluorescent Ligand, Downregulates Soce And Promotes Apoptosis In The Triple Negative Breast Cancer Cell Lines
Twelve Weeks Of Whole Body Vibration Training Improve Regucalcin, Body Composition And Physical Fitness In Postmenopausal Women: A Pilot Study
Pgrmc1 Inhibits Progesterone-Evoked Proliferation And Ca2+ Entry Via Stim2 In Mda-Mb-231 Cells
Twelve Weeks Of Whole Body Vibration Training Improve Regucalcin, Body Composition And Physical Fitness In Postmenopausal Women: A Pilot Study
Pgrmc1 Inhibits Progesterone-Evoked Proliferation And Ca2+ Entry Via Stim2 In Mda-Mb-231 Cells
Functional Role Of Trpc6 And Stim2 In Cytosolic And Endoplasmic Reticulum Ca2+ Content In Resting Estrogen Receptor-Positive Breast Cancer Cells
Pgrmc1 Inhibits Progesterone-Evoked Proliferation And Ca2+ Entry Via Stim2 In Mda-Mb-231 Cells
Functional Role Of Trpc6 And Stim2 In Cytosolic And Endoplasmic Reticulum Ca2+ Content In Resting Estrogen Receptor-Positive Breast Cancer Cells
Arachidonic Acid Attenuates Cell Proliferation, Migration And Viability By A Mechanism Independent On Calcium Entry
Functional Role Of Trpc6 And Stim2 In Cytosolic And Endoplasmic Reticulum Ca2+ Content In Resting Estrogen Receptor-Positive Breast Cancer Cells
Arachidonic Acid Attenuates Cell Proliferation, Migration And Viability By A Mechanism Independent On Calcium Entry
Trpc Channels In The Soce Scenario
Arachidonic Acid Attenuates Cell Proliferation, Migration And Viability By A Mechanism Independent On Calcium Entry
Trpc Channels In The Soce Scenario
Molecular Basis And Regulation Of Store-Operated Calcium Entry
Trpc Channels In The Soce Scenario
Molecular Basis And Regulation Of Store-Operated Calcium Entry
New Insights Into Adipokines As Potential Biomarkers For Typr-2 Diabetes Mellitus
Molecular Basis And Regulation Of Store-Operated Calcium Entry
New Insights Into Adipokines As Potential Biomarkers For Typr-2 Diabetes Mellitus
Adenylyl Cyclase Type 8 Overexpression Impairs Phosphorylation-Dependent Orai1 Inactivation And Promotes Migration In Mda-Mb-231 Breast Cancer Cells
New Insights Into Adipokines As Potential Biomarkers For Typr-2 Diabetes Mellitus
Adenylyl Cyclase Type 8 Overexpression Impairs Phosphorylation-Dependent Orai1 Inactivation And Promotes Migration In Mda-Mb-231 Breast Cancer Cells
Flavonoids And Platelet-Derived Thrombotic Disorders
Adenylyl Cyclase Type 8 Overexpression Impairs Phosphorylation-Dependent Orai1 Inactivation And Promotes Migration In Mda-Mb-231 Breast Cancer Cells
Flavonoids And Platelet-Derived Thrombotic Disorders
Store-Independent Orai1-Mediated Ca 2+ Entry And Cancer
Flavonoids And Platelet-Derived Thrombotic Disorders
Store-Independent Orai1-Mediated Ca 2+ Entry And Cancer
Stim1 Phosphorylation At Y316 Modulates Its Interaction With Saraf And The Activation Of Soce And Icrac
Store-Independent Orai1-Mediated Ca 2+ Entry And Cancer
Stim1 Phosphorylation At Y316 Modulates Its Interaction With Saraf And The Activation Of Soce And Icrac
(−)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells
Stim1 Phosphorylation At Y316 Modulates Its Interaction With Saraf And The Activation Of Soce And Icrac
(−)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells
Special Issue On New Cellular, Genetic And Proteomic Tools In The Prevention And Management Of Diabetes Mellitus
(−)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells
Special Issue On New Cellular, Genetic And Proteomic Tools In The Prevention And Management Of Diabetes Mellitus
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus
Special Issue On New Cellular, Genetic And Proteomic Tools In The Prevention And Management Of Diabetes Mellitus
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus
Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus
Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Platelet Functions Are Decreased In Obesity And Restored After Weight Loss: Evidence For A Role Of The Serca3-Dependent Adp Secretion Pathway
Inactivation Of Proprotein Convertases In T Cells Inhibits Pd-1 Expression And Creates A Favorable Immune Microenvironment In Colorectal Cancer
Platelet Functions Are Decreased In Obesity And Restored After Weight Loss: Evidence For A Role Of The Serca3-Dependent Adp Secretion Pathway
Efhb Is A Novel Cytosolic Ca 2+ Sensor That Modulates Stim1-Saraf Interaction
Platelet Functions Are Decreased In Obesity And Restored After Weight Loss: Evidence For A Role Of The Serca3-Dependent Adp Secretion Pathway
Efhb Is A Novel Cytosolic Ca 2+ Sensor That Modulates Stim1-Saraf Interaction
Fisiología Digestiva De Las Aves
Efhb Is A Novel Cytosolic Ca 2+ Sensor That Modulates Stim1-Saraf Interaction
Fisiología Digestiva De Las Aves
Flavonoids And Platelet-Derived Thrombotic Disorders.
Fisiología Digestiva De Las Aves
Flavonoids And Platelet-Derived Thrombotic Disorders.
(-)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells.
Flavonoids And Platelet-Derived Thrombotic Disorders.
(-)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells.
Store-Operated Ca 2+ Entry In Breast Cancer Cells: Remodeling And Functional Role
(-)‑oleocanthal Inhibits Proliferation And Migration By Modulating Ca2+ Entry Through Trpc6 In Breast Cancer Cells.
Store-Operated Ca 2+ Entry In Breast Cancer Cells: Remodeling And Functional Role
Trpc6 Channels Are Required For Proliferation, Migration And Invasion Of Breast Cancer Cell Lines By Modulation Of Orai1 And Orai3 Surface Exposure
Store-Operated Ca 2+ Entry In Breast Cancer Cells: Remodeling And Functional Role
Trpc6 Channels Are Required For Proliferation, Migration And Invasion Of Breast Cancer Cell Lines By Modulation Of Orai1 And Orai3 Surface Exposure
Stanniocalcin 2 Regulates Non-Capacitative Ca2+ Entry And Aggregation In Mouse Platelets
Trpc6 Channels Are Required For Proliferation, Migration And Invasion Of Breast Cancer Cell Lines By Modulation Of Orai1 And Orai3 Surface Exposure
Stanniocalcin 2 Regulates Non-Capacitative Ca2+ Entry And Aggregation In Mouse Platelets
Fine-Tuning Of Store-Operated Calcium Entry By Fast And Slow Ca2+-Dependent Inactivation: Involvement Of Saraf
Stanniocalcin 2 Regulates Non-Capacitative Ca2+ Entry And Aggregation In Mouse Platelets
Fine-Tuning Of Store-Operated Calcium Entry By Fast And Slow Ca2+-Dependent Inactivation: Involvement Of Saraf
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets
Fine-Tuning Of Store-Operated Calcium Entry By Fast And Slow Ca2+-Dependent Inactivation: Involvement Of Saraf
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets
Mirna And Platelet Genetic Machinery
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets
Mirna And Platelet Genetic Machinery
Fluorescence-Based Measurements Of The Crac Channel Activity In Cell Populations
Mirna And Platelet Genetic Machinery
Fluorescence-Based Measurements Of The Crac Channel Activity In Cell Populations
Involvement Of Stanniocalcins In The Deregulation Of Glycaemia In Obese Mice And Type 2 Diabetic Patients
Fluorescence-Based Measurements Of The Crac Channel Activity In Cell Populations
Involvement Of Stanniocalcins In The Deregulation Of Glycaemia In Obese Mice And Type 2 Diabetic Patients
Filamin A Modulates Platelet Function
Involvement Of Stanniocalcins In The Deregulation Of Glycaemia In Obese Mice And Type 2 Diabetic Patients
Filamin A Modulates Platelet Function
Involvement Of Stanniocalcins In The Deregulation Of Glycaemia In Obese Mice And Type 2 Diabetic Patients.
Filamin A Modulates Platelet Function
Involvement Of Stanniocalcins In The Deregulation Of Glycaemia In Obese Mice And Type 2 Diabetic Patients.
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus.
Involvement Of Stanniocalcins In The Deregulation Of Glycaemia In Obese Mice And Type 2 Diabetic Patients.
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus.
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets.
New Insights Into Adipokines As Potential Biomarkers For Type-2 Diabetes Mellitus.
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets.
Trps In Pain Sensation
Filamin A Modulates Store-Operated Ca2+ Entry By Regulating Stim1 (Stromal Interaction Molecule 1)-Orai1 Association In Human Platelets.
Trps In Pain Sensation
Cardiovascular And Hemostatic Disorders: Soce And Ca2+ Handling In Platelet Dysfunction
Trps In Pain Sensation
Cardiovascular And Hemostatic Disorders: Soce And Ca2+ Handling In Platelet Dysfunction
Role Of Stim1 In The Surface Expression Of Saraf
Cardiovascular And Hemostatic Disorders: Soce And Ca2+ Handling In Platelet Dysfunction
Role Of Stim1 In The Surface Expression Of Saraf
Dynamic Interaction Of Saraf With Stim1 And Orai1 To Modulate Store-Operated Calcium Entry
Role Of Stim1 In The Surface Expression Of Saraf
Dynamic Interaction Of Saraf With Stim1 And Orai1 To Modulate Store-Operated Calcium Entry
Molecular Modulators Of Store-Operated Calcium Entry
Dynamic Interaction Of Saraf With Stim1 And Orai1 To Modulate Store-Operated Calcium Entry
Molecular Modulators Of Store-Operated Calcium Entry
Historical Overview Of Store-Operated Ca2+ Entry
Molecular Modulators Of Store-Operated Calcium Entry
Historical Overview Of Store-Operated Ca2+ Entry
Store-Operated Ca2+ Entry- Associated Regulatory Factor (Saraf) Plays An Important Role In The Regulation Of Arachidonate-Regulated Ca2+ (Arc) Channels
Historical Overview Of Store-Operated Ca2+ Entry
Store-Operated Ca2+ Entry- Associated Regulatory Factor (Saraf) Plays An Important Role In The Regulation Of Arachidonate-Regulated Ca2+ (Arc) Channels
Saraf Modulates Trpc1, But Not Trpc6, Channel Function In A Stim1-Independent Manner
Store-Operated Ca2+ Entry- Associated Regulatory Factor (Saraf) Plays An Important Role In The Regulation Of Arachidonate-Regulated Ca2+ (Arc) Channels
Saraf Modulates Trpc1, But Not Trpc6, Channel Function In A Stim1-Independent Manner
Data On Calcium Increases Depending On Stretch In Dystrophic Cardiomyocytes
Saraf Modulates Trpc1, But Not Trpc6, Channel Function In A Stim1-Independent Manner
Data On Calcium Increases Depending On Stretch In Dystrophic Cardiomyocytes
Axial Stretch-Dependent Cation Entry In Dystrophic Cardiomyopathy: Involvement Of Several Trps Channels
Data On Calcium Increases Depending On Stretch In Dystrophic Cardiomyocytes
Axial Stretch-Dependent Cation Entry In Dystrophic Cardiomyopathy: Involvement Of Several Trps Channels
Apelin: An Antithrombotic Factor That Inhibits Platelet Function
Axial Stretch-Dependent Cation Entry In Dystrophic Cardiomyopathy: Involvement Of Several Trps Channels
Apelin: An Antithrombotic Factor That Inhibits Platelet Function
Relationship Between Calcium Mobilization And Platelet Α- And Δ-Granule Secretion. A Role For Trpc6 In Thrombin-Evoked Δ-Granule Exocytosis
Apelin: An Antithrombotic Factor That Inhibits Platelet Function
Relationship Between Calcium Mobilization And Platelet Α- And Δ-Granule Secretion. A Role For Trpc6 In Thrombin-Evoked Δ-Granule Exocytosis
Fkbp25 And Fkbp38 Regulate Non-Capacitative Calcium Entry Through Trpc6
Relationship Between Calcium Mobilization And Platelet Α- And Δ-Granule Secretion. A Role For Trpc6 In Thrombin-Evoked Δ-Granule Exocytosis
Fkbp25 And Fkbp38 Regulate Non-Capacitative Calcium Entry Through Trpc6
Evaluation Of The Antiaggregant Activity Of Ascorbyl Phenolic Esters With Antioxidant Properties
Fkbp25 And Fkbp38 Regulate Non-Capacitative Calcium Entry Through Trpc6
Evaluation Of The Antiaggregant Activity Of Ascorbyl Phenolic Esters With Antioxidant Properties
Role Of Mtor1 And Mtor2 Complexes In Meg-01 Cell Physiology
Evaluation Of The Antiaggregant Activity Of Ascorbyl Phenolic Esters With Antioxidant Properties
Role Of Mtor1 And Mtor2 Complexes In Meg-01 Cell Physiology
Store-Operated Calcium Entry: Unveiling The Calcium Handling Signalplex
Role Of Mtor1 And Mtor2 Complexes In Meg-01 Cell Physiology
Store-Operated Calcium Entry: Unveiling The Calcium Handling Signalplex
Relationship Between Extracellular And Intracelular Calcium Mobilization And Platelet Granule Secretion
Store-Operated Calcium Entry: Unveiling The Calcium Handling Signalplex
Relationship Between Extracellular And Intracelular Calcium Mobilization And Platelet Granule Secretion
Stim1 Regulates Trpc6 Heteromultimerization And Subcellular Location
Relationship Between Extracellular And Intracelular Calcium Mobilization And Platelet Granule Secretion
Stim1 Regulates Trpc6 Heteromultimerization And Subcellular Location
Trpc6 Participates In The Regulation Of Cytosolic Basal Calcium Concentration In Murine Resting Platelets
Stim1 Regulates Trpc6 Heteromultimerization And Subcellular Location
Trpc6 Participates In The Regulation Of Cytosolic Basal Calcium Concentration In Murine Resting Platelets
The Canonical Transient Receptor Potential 6 (Trpc6) Channel Is Sensitive To Extracellular Ph In Mouse Platelets
Trpc6 Participates In The Regulation Of Cytosolic Basal Calcium Concentration In Murine Resting Platelets
The Canonical Transient Receptor Potential 6 (Trpc6) Channel Is Sensitive To Extracellular Ph In Mouse Platelets
Synergistic Role Of Protein Phosphatase Inhibitor 1 And Sarco/endoplasmic Reticulum Ca2+-Atpase In The Acquisition Of The Contractile Phenotype Of Arterial Smooth Muscle Cells
The Canonical Transient Receptor Potential 6 (Trpc6) Channel Is Sensitive To Extracellular Ph In Mouse Platelets
Synergistic Role Of Protein Phosphatase Inhibitor 1 And Sarco/endoplasmic Reticulum Ca2+-Atpase In The Acquisition Of The Contractile Phenotype Of Arterial Smooth Muscle Cells
Cytoskeletal And Scaffolding Proteins As Structural And Functional Determinants Of Trp Channels
Synergistic Role Of Protein Phosphatase Inhibitor 1 And Sarco/endoplasmic Reticulum Ca2+-Atpase In The Acquisition Of The Contractile Phenotype Of Arterial Smooth Muscle Cells
Cytoskeletal And Scaffolding Proteins As Structural And Functional Determinants Of Trp Channels
Endothelial Cytoskeleton
Cytoskeletal And Scaffolding Proteins As Structural And Functional Determinants Of Trp Channels
Endothelial Cytoskeleton
Endothelial Cells And The Regulation Of Platelet Function
Endothelial Cytoskeleton
Endothelial Cells And The Regulation Of Platelet Function
Preface
Endothelial Cells And The Regulation Of Platelet Function
Preface
Transient Receptor Potential Ankyrin-1 (Trpa1) Modulates Store-Operated Ca2+ Entry By Regulation Of Stim1-Orai1 Association
Preface
Transient Receptor Potential Ankyrin-1 (Trpa1) Modulates Store-Operated Ca2+ Entry By Regulation Of Stim1-Orai1 Association
Pharmacology Of Trp Channels In The Vasculature
Transient Receptor Potential Ankyrin-1 (Trpa1) Modulates Store-Operated Ca2+ Entry By Regulation Of Stim1-Orai1 Association
Pharmacology Of Trp Channels In The Vasculature
Homer Proteins In Ca2+ Entry
Pharmacology Of Trp Channels In The Vasculature
Homer Proteins In Ca2+ Entry
Long-Term Mtor Inhibitors Administration Evokes Altered Calcium Homeostasis And Platelet Dysfunction In Kidney Transplant Patients
Homer Proteins In Ca2+ Entry
Long-Term Mtor Inhibitors Administration Evokes Altered Calcium Homeostasis And Platelet Dysfunction In Kidney Transplant Patients
Immunophilins Are Involved In The Altered Platelet Aggregation Observed In Patients With Type 2 Diabetes Mellitus
Long-Term Mtor Inhibitors Administration Evokes Altered Calcium Homeostasis And Platelet Dysfunction In Kidney Transplant Patients
Immunophilins Are Involved In The Altered Platelet Aggregation Observed In Patients With Type 2 Diabetes Mellitus
The Polybasic Lysine-Rich Domain Of Plasma Membrane-Resident Stim1 Is Essential For The Modulation Of Store-Operated Divalent Cation Entry By Extracellular Calcium
Immunophilins Are Involved In The Altered Platelet Aggregation Observed In Patients With Type 2 Diabetes Mellitus
The Polybasic Lysine-Rich Domain Of Plasma Membrane-Resident Stim1 Is Essential For The Modulation Of Store-Operated Divalent Cation Entry By Extracellular Calcium
Fkbp52 Is Involved In The Regulation Of Soce Channels In The Human Platelets And Meg 01 Cells
The Polybasic Lysine-Rich Domain Of Plasma Membrane-Resident Stim1 Is Essential For The Modulation Of Store-Operated Divalent Cation Entry By Extracellular Calcium
Fkbp52 Is Involved In The Regulation Of Soce Channels In The Human Platelets And Meg 01 Cells
Molecular Interplay Between Platelets And The Vascular Wall In Thrombosis And Hemostasis
Fkbp52 Is Involved In The Regulation Of Soce Channels In The Human Platelets And Meg 01 Cells
Molecular Interplay Between Platelets And The Vascular Wall In Thrombosis And Hemostasis
Crucial Role For Endoplasmic Reticulum Stress During Megakaryocyte Maturation
Molecular Interplay Between Platelets And The Vascular Wall In Thrombosis And Hemostasis
Crucial Role For Endoplasmic Reticulum Stress During Megakaryocyte Maturation
Benefit Of Serca2a Gene Transfer To Vascular Endothelial And Smooth Muscle Cells: A New Aspect In Therapy Of Cardiovascular Diseases
Crucial Role For Endoplasmic Reticulum Stress During Megakaryocyte Maturation
Benefit Of Serca2a Gene Transfer To Vascular Endothelial And Smooth Muscle Cells: A New Aspect In Therapy Of Cardiovascular Diseases
Cardiovascular System Soce And Ca2+ Handling In Platelet Dysfunction
Benefit Of Serca2a Gene Transfer To Vascular Endothelial And Smooth Muscle Cells: A New Aspect In Therapy Of Cardiovascular Diseases
Cardiovascular System Soce And Ca2+ Handling In Platelet Dysfunction
Two-Pore Channel 2 (Tpc2) Modulates Store-Operated Ca2+ Entry
Cardiovascular System Soce And Ca2+ Handling In Platelet Dysfunction
Two-Pore Channel 2 (Tpc2) Modulates Store-Operated Ca2+ Entry
Stim1 Tyrosine-Phosphorylation Is Required For Stim1-Orai1 Association In Human Platelets
Two-Pore Channel 2 (Tpc2) Modulates Store-Operated Ca2+ Entry
Stim1 Tyrosine-Phosphorylation Is Required For Stim1-Orai1 Association In Human Platelets
Transient Receptor Potential Channels In Human Platelets: Expression And Functional Role
Stim1 Tyrosine-Phosphorylation Is Required For Stim1-Orai1 Association In Human Platelets
Transient Receptor Potential Channels In Human Platelets: Expression And Functional Role
Store-Operated Ca 2+ Entry
Transient Receptor Potential Channels In Human Platelets: Expression And Functional Role
Store-Operated Ca 2+ Entry
Acidic Naadp-Releasable Ca2+ Compartments In The Megakaryoblastic Cell Line Meg01
Store-Operated Ca 2+ Entry
Acidic Naadp-Releasable Ca2+ Compartments In The Megakaryoblastic Cell Line Meg01
Stim1 And Stim2 Are Located In The Acidic Ca2+ Stores And Associates With Orai1 Upon Depletion Of The Acidic Stores In Human Platelets
Acidic Naadp-Releasable Ca2+ Compartments In The Megakaryoblastic Cell Line Meg01
Stim1 And Stim2 Are Located In The Acidic Ca2+ Stores And Associates With Orai1 Upon Depletion Of The Acidic Stores In Human Platelets
Two Distinct Calcium Pools In The Endoplasmic Reticulum Of Hek-293t Cells
Stim1 And Stim2 Are Located In The Acidic Ca2+ Stores And Associates With Orai1 Upon Depletion Of The Acidic Stores In Human Platelets
Two Distinct Calcium Pools In The Endoplasmic Reticulum Of Hek-293t Cells
Attenuated Store-Operated Divalent Cation Entry And Association Between Stim1, Orai1, Htrpc1 And Htrpc6 In Platelets From Type 2 Diabetic Patients
Two Distinct Calcium Pools In The Endoplasmic Reticulum Of Hek-293t Cells
Attenuated Store-Operated Divalent Cation Entry And Association Between Stim1, Orai1, Htrpc1 And Htrpc6 In Platelets From Type 2 Diabetic Patients
Immunophilins And Thrombotic Disorders
Attenuated Store-Operated Divalent Cation Entry And Association Between Stim1, Orai1, Htrpc1 And Htrpc6 In Platelets From Type 2 Diabetic Patients
Immunophilins And Thrombotic Disorders
Editorial: A Role For Immunophilins In Cellular Signalling In Health And Disease
Immunophilins And Thrombotic Disorders
Editorial: A Role For Immunophilins In Cellular Signalling In Health And Disease
Immunossupressant And Organ Transplantation: Immunophilins Targeting Agent And Alternative Therapies
Editorial: A Role For Immunophilins In Cellular Signalling In Health And Disease
Immunossupressant And Organ Transplantation: Immunophilins Targeting Agent And Alternative Therapies
Immunophilin Dysfunction And Neuropathology
Immunossupressant And Organ Transplantation: Immunophilins Targeting Agent And Alternative Therapies
Immunophilin Dysfunction And Neuropathology
Serca2a Controls The Mode Of Agonist-Induced Intracellular Ca2+ Signal, Transcription Factor Nfat And Proliferation In Human Vascular Smooth Muscle Cells
Immunophilin Dysfunction And Neuropathology
Serca2a Controls The Mode Of Agonist-Induced Intracellular Ca2+ Signal, Transcription Factor Nfat And Proliferation In Human Vascular Smooth Muscle Cells
Trpc3 Regulates Agonist-Stimulated Ca2+ Mobilization By Mediating The Interaction Between Type I Inositol 1,4,5-Trisphosphate Receptor, Rack1, And Orai1
Serca2a Controls The Mode Of Agonist-Induced Intracellular Ca2+ Signal, Transcription Factor Nfat And Proliferation In Human Vascular Smooth Muscle Cells
Trpc3 Regulates Agonist-Stimulated Ca2+ Mobilization By Mediating The Interaction Between Type I Inositol 1,4,5-Trisphosphate Receptor, Rack1, And Orai1
Serca2b Activity Is Regulated By Cyclophilins In Human Platelets
Trpc3 Regulates Agonist-Stimulated Ca2+ Mobilization By Mediating The Interaction Between Type I Inositol 1,4,5-Trisphosphate Receptor, Rack1, And Orai1
Serca2b Activity Is Regulated By Cyclophilins In Human Platelets
Homocysteine Induces Caspase Activation By Endoplasmic Reticulum Stress In Platelets From Type 2 Diabetics And Healthy Donors
Serca2b Activity Is Regulated By Cyclophilins In Human Platelets
Homocysteine Induces Caspase Activation By Endoplasmic Reticulum Stress In Platelets From Type 2 Diabetics And Healthy Donors
Melatonin Reduces Apoptosis Induced By Calcium Signaling In Human Leukocytes: Evidence For The Involvement Of Mitochondria And Bax Activation
Homocysteine Induces Caspase Activation By Endoplasmic Reticulum Stress In Platelets From Type 2 Diabetics And Healthy Donors
Melatonin Reduces Apoptosis Induced By Calcium Signaling In Human Leukocytes: Evidence For The Involvement Of Mitochondria And Bax Activation
Caspase-3 And -9 Are Activated In Human Myeloid Hl-60 Cells By Calcium Signal
Melatonin Reduces Apoptosis Induced By Calcium Signaling In Human Leukocytes: Evidence For The Involvement Of Mitochondria And Bax Activation
Caspase-3 And -9 Are Activated In Human Myeloid Hl-60 Cells By Calcium Signal
Melatonin, As An Adjuvant-Like Agent, Enhances Platelet Responsiveness
Caspase-3 And -9 Are Activated In Human Myeloid Hl-60 Cells By Calcium Signal
Melatonin, As An Adjuvant-Like Agent, Enhances Platelet Responsiveness
Role Of Calcium Signals On Hydrogen Peroxide-Induced Apoptosis In Human Myeloid Hl-60 Cells
Melatonin, As An Adjuvant-Like Agent, Enhances Platelet Responsiveness
Role Of Calcium Signals On Hydrogen Peroxide-Induced Apoptosis In Human Myeloid Hl-60 Cells
Store-Operated Ca2+ Entry Is Sensitive To The Extracellular Ca2+ Concentration Through Plasma Membrane Stim1
Role Of Calcium Signals On Hydrogen Peroxide-Induced Apoptosis In Human Myeloid Hl-60 Cells
Store-Operated Ca2+ Entry Is Sensitive To The Extracellular Ca2+ Concentration Through Plasma Membrane Stim1
Enhanced Expression Of Stim1/orai1 And Trpc3 In Platelets From Patients With Type 2 Diabetes Mellitus
Store-Operated Ca2+ Entry Is Sensitive To The Extracellular Ca2+ Concentration Through Plasma Membrane Stim1
Enhanced Expression Of Stim1/orai1 And Trpc3 In Platelets From Patients With Type 2 Diabetes Mellitus
N,n,n′,n′-Tetrakis(2-Pyridylmethyl)ethylenediamine Induces Apoptosis Through The Activation Of Caspases-3 And -8 In Human Platelets. A Role For Endoplasmic Reticulum Stress
Enhanced Expression Of Stim1/orai1 And Trpc3 In Platelets From Patients With Type 2 Diabetes Mellitus
N,n,n′,n′-Tetrakis(2-Pyridylmethyl)ethylenediamine Induces Apoptosis Through The Activation Of Caspases-3 And -8 In Human Platelets. A Role For Endoplasmic Reticulum Stress
Melatonin Induces Mitochondrial-Mediated Apoptosis In Human Myeloid Hl-60 Cells
N,n,n′,n′-Tetrakis(2-Pyridylmethyl)ethylenediamine Induces Apoptosis Through The Activation Of Caspases-3 And -8 In Human Platelets. A Role For Endoplasmic Reticulum Stress
Melatonin Induces Mitochondrial-Mediated Apoptosis In Human Myeloid Hl-60 Cells
Role Of Calcium Signals On Hydrogen Peroxide-Induced Apoptosis In Human Myeloid Hl-60 Cells
Melatonin Induces Mitochondrial-Mediated Apoptosis In Human Myeloid Hl-60 Cells
Role Of Calcium Signals On Hydrogen Peroxide-Induced Apoptosis In Human Myeloid Hl-60 Cells
Role Of Intracellular Calcium On Hydrogen Peroxide-Induced Apoptosis In Rat Pancreatic Acinar Ar42j Cells
Role Of Calcium Signals On Hydrogen Peroxide-Induced Apoptosis In Human Myeloid Hl-60 Cells
Role Of Intracellular Calcium On Hydrogen Peroxide-Induced Apoptosis In Rat Pancreatic Acinar Ar42j Cells
Orai1 Mediates The Interaction Between Stim1 And Htrpc1 And Regulates The Mode Of Activation Of Htrpc1-Forming Ca2+ Channels
Role Of Intracellular Calcium On Hydrogen Peroxide-Induced Apoptosis In Rat Pancreatic Acinar Ar42j Cells
Orai1 Mediates The Interaction Between Stim1 And Htrpc1 And Regulates The Mode Of Activation Of Htrpc1-Forming Ca2+ Channels
Intracellular Ca2+ Store Depletion Induces The Formation Of Macromolecular Complexes Involving Htrpc1, Htrpc6, The Type Ii Ip3 Receptor And Serca3 In Human Platelets
Orai1 Mediates The Interaction Between Stim1 And Htrpc1 And Regulates The Mode Of Activation Of Htrpc1-Forming Ca2+ Channels
Intracellular Ca2+ Store Depletion Induces The Formation Of Macromolecular Complexes Involving Htrpc1, Htrpc6, The Type Ii Ip3 Receptor And Serca3 In Human Platelets
Stim1 Regulates Acidic Ca2+ Store Refilling By Interaction With Serca3 In Human Platelets
Intracellular Ca2+ Store Depletion Induces The Formation Of Macromolecular Complexes Involving Htrpc1, Htrpc6, The Type Ii Ip3 Receptor And Serca3 In Human Platelets
Stim1 Regulates Acidic Ca2+ Store Refilling By Interaction With Serca3 In Human Platelets
Cinnamtannin B-1 As An Antioxidant And Platelet Aggregation Inhibitor
Stim1 Regulates Acidic Ca2+ Store Refilling By Interaction With Serca3 In Human Platelets
Cinnamtannin B-1 As An Antioxidant And Platelet Aggregation Inhibitor
Functional Relevance Of The De Novo Coupling Between Htrpc1 And Type Ii Ip3 Receptor In Store-Operated Ca2+ Entry In Human Platelets
Cinnamtannin B-1 As An Antioxidant And Platelet Aggregation Inhibitor
Functional Relevance Of The De Novo Coupling Between Htrpc1 And Type Ii Ip3 Receptor In Store-Operated Ca2+ Entry In Human Platelets
Intracellular Calcium Release From Human Platelets: Different Messengers For Multiple Stores
Functional Relevance Of The De Novo Coupling Between Htrpc1 And Type Ii Ip3 Receptor In Store-Operated Ca2+ Entry In Human Platelets
Intracellular Calcium Release From Human Platelets: Different Messengers For Multiple Stores
Serca2b And 3 Play A Regulatory Role In Store-Operated Calcium Entry In Human Platelets
Intracellular Calcium Release From Human Platelets: Different Messengers For Multiple Stores
Serca2b And 3 Play A Regulatory Role In Store-Operated Calcium Entry In Human Platelets
Protein Complex Immunological Separation Assay (Procisa): A Technique For Investigating Single Protein Properties
Serca2b And 3 Play A Regulatory Role In Store-Operated Calcium Entry In Human Platelets
Protein Complex Immunological Separation Assay (Procisa): A Technique For Investigating Single Protein Properties
Phosphatidylinositol 4,5-Bisphosphate Enhances Store-Operated Calcium Entry Through Htrpc6 Channel In Human Platelets
Protein Complex Immunological Separation Assay (Procisa): A Technique For Investigating Single Protein Properties
Phosphatidylinositol 4,5-Bisphosphate Enhances Store-Operated Calcium Entry Through Htrpc6 Channel In Human Platelets
Thrombin Induces Activation And Translocation Of Bid, Bax And Bak To The Mitochondria In Human Platelets
Phosphatidylinositol 4,5-Bisphosphate Enhances Store-Operated Calcium Entry Through Htrpc6 Channel In Human Platelets
Thrombin Induces Activation And Translocation Of Bid, Bax And Bak To The Mitochondria In Human Platelets
Antiaggregant Effects Of Arbutus Unedo Extracts In Human Platelets
Thrombin Induces Activation And Translocation Of Bid, Bax And Bak To The Mitochondria In Human Platelets
Antiaggregant Effects Of Arbutus Unedo Extracts In Human Platelets
Thrombin Induces Apoptotic Events Through The Generation Of Reactive Oxygen Species In Human Platelets
Antiaggregant Effects Of Arbutus Unedo Extracts In Human Platelets
Thrombin Induces Apoptotic Events Through The Generation Of Reactive Oxygen Species In Human Platelets
Tyrosine Phosphorylation/dephosphorylation Balance Is Involved In Thrombin-Evoked Microtubular Reorganisation In Human Platelets
Thrombin Induces Apoptotic Events Through The Generation Of Reactive Oxygen Species In Human Platelets
Tyrosine Phosphorylation/dephosphorylation Balance Is Involved In Thrombin-Evoked Microtubular Reorganisation In Human Platelets
Htrpc1-Associated Α-Actinin, And Not Htrpc1 Itself, Is Tyrosine Phosphorylated During Human Platelet Activation
Tyrosine Phosphorylation/dephosphorylation Balance Is Involved In Thrombin-Evoked Microtubular Reorganisation In Human Platelets
Htrpc1-Associated Α-Actinin, And Not Htrpc1 Itself, Is Tyrosine Phosphorylated During Human Platelet Activation
Dual Role Of Tubulin-Cytoskeleton In Store-Operated Calcium Entry In Human Platelets
Htrpc1-Associated Α-Actinin, And Not Htrpc1 Itself, Is Tyrosine Phosphorylated During Human Platelet Activation
Dual Role Of Tubulin-Cytoskeleton In Store-Operated Calcium Entry In Human Platelets
Early Caspase-3 Activation Independent Of Apoptosis Is Required For Cellular Function
Dual Role Of Tubulin-Cytoskeleton In Store-Operated Calcium Entry In Human Platelets
Early Caspase-3 Activation Independent Of Apoptosis Is Required For Cellular Function
Interaction Of Stim1 With Endogenously Expressed Human Canonical Trp1 Upon Depletion Of Intracellular Ca2+ Stores
Early Caspase-3 Activation Independent Of Apoptosis Is Required For Cellular Function
Interaction Of Stim1 With Endogenously Expressed Human Canonical Trp1 Upon Depletion Of Intracellular Ca2+ Stores
Endogenously Generated Reactive Oxygen Species Reduce Pmca Activity In Platelets From Patients With Non-Insulin-Dependent Diabetes Mellitus
Interaction Of Stim1 With Endogenously Expressed Human Canonical Trp1 Upon Depletion Of Intracellular Ca2+ Stores
Endogenously Generated Reactive Oxygen Species Reduce Pmca Activity In Platelets From Patients With Non-Insulin-Dependent Diabetes Mellitus
Intracellular Ca2+ Homeostasis And Aggregation In Platelets Are Impaired By Ethanol Through The Generation Of H2o2 And Oxidation Of Sulphydryl Groups
Endogenously Generated Reactive Oxygen Species Reduce Pmca Activity In Platelets From Patients With Non-Insulin-Dependent Diabetes Mellitus
Intracellular Ca2+ Homeostasis And Aggregation In Platelets Are Impaired By Ethanol Through The Generation Of H2o2 And Oxidation Of Sulphydryl Groups
Calcium Signalling And Reactive Oxygen Species In Non-Excitable Cells
Intracellular Ca2+ Homeostasis And Aggregation In Platelets Are Impaired By Ethanol Through The Generation Of H2o2 And Oxidation Of Sulphydryl Groups
Calcium Signalling And Reactive Oxygen Species In Non-Excitable Cells
Two Distinct Ca2+ Compartments Show Differential Sensitivity To Thrombin, Adp And Vasopressin In Human Platelets
Calcium Signalling And Reactive Oxygen Species In Non-Excitable Cells
Two Distinct Ca2+ Compartments Show Differential Sensitivity To Thrombin, Adp And Vasopressin In Human Platelets
Roles Of Calcium And Tyrosine Kinases In The Pathogenesis Of Type 2 Diabetes Mellitus
Two Distinct Ca2+ Compartments Show Differential Sensitivity To Thrombin, Adp And Vasopressin In Human Platelets
Roles Of Calcium And Tyrosine Kinases In The Pathogenesis Of Type 2 Diabetes Mellitus
A Role For 5,6-Epoxyeicosatrienoic Acid In Calcium Entry By De Novo Conformational Coupling In Human Platelets
Roles Of Calcium And Tyrosine Kinases In The Pathogenesis Of Type 2 Diabetes Mellitus
A Role For 5,6-Epoxyeicosatrienoic Acid In Calcium Entry By De Novo Conformational Coupling In Human Platelets
A Role For Cofilin In The Activation Of Store-Operated Calcium Entry By De Novo Conformational Coupling In Human Platelets
A Role For 5,6-Epoxyeicosatrienoic Acid In Calcium Entry By De Novo Conformational Coupling In Human Platelets
A Role For Cofilin In The Activation Of Store-Operated Calcium Entry By De Novo Conformational Coupling In Human Platelets
Store-Operated Ca2+ Entry: Vesicle Fusion Or Reversible Trafficking And De Novo Conformational Coupling?
A Role For Cofilin In The Activation Of Store-Operated Calcium Entry By De Novo Conformational Coupling In Human Platelets
Store-Operated Ca2+ Entry: Vesicle Fusion Or Reversible Trafficking And De Novo Conformational Coupling?
Dynamics Of Calcium Fluxes In Human Platelets Assessed In Calcium-Free Medium
Store-Operated Ca2+ Entry: Vesicle Fusion Or Reversible Trafficking And De Novo Conformational Coupling?
Dynamics Of Calcium Fluxes In Human Platelets Assessed In Calcium-Free Medium
Ca2+ Accumulation Into Acidic Organelles Mediated By Ca 2+- And Vacuolar H+-Atpases In Human Platelets
Dynamics Of Calcium Fluxes In Human Platelets Assessed In Calcium-Free Medium
Ca2+ Accumulation Into Acidic Organelles Mediated By Ca 2+- And Vacuolar H+-Atpases In Human Platelets
Hydrogen Peroxide And Peroxynitrite Enhance Ca2+ Mobilization And Aggregation In Platelets From Type 2 Diabetic Patients
Ca2+ Accumulation Into Acidic Organelles Mediated By Ca 2+- And Vacuolar H+-Atpases In Human Platelets
Hydrogen Peroxide And Peroxynitrite Enhance Ca2+ Mobilization And Aggregation In Platelets From Type 2 Diabetic Patients
Collaborative Effect Of Serca And Pmca In Cytosolic Calcium Homeostasis In Human Platelets
Hydrogen Peroxide And Peroxynitrite Enhance Ca2+ Mobilization And Aggregation In Platelets From Type 2 Diabetic Patients
Collaborative Effect Of Serca And Pmca In Cytosolic Calcium Homeostasis In Human Platelets
Ca2+-Independent Activation Of Bruton's Tyrosine Kinase Is Required For Store-Mediated Ca2+ Entry In Human Platelets
Collaborative Effect Of Serca And Pmca In Cytosolic Calcium Homeostasis In Human Platelets
Ca2+-Independent Activation Of Bruton's Tyrosine Kinase Is Required For Store-Mediated Ca2+ Entry In Human Platelets
Cleavage Of Snap-25 And Vamp-2 Impairs Store-Operated Ca2+ Entry In Mouse Pancreatic Acinar Cells
Ca2+-Independent Activation Of Bruton's Tyrosine Kinase Is Required For Store-Mediated Ca2+ Entry In Human Platelets
Cleavage Of Snap-25 And Vamp-2 Impairs Store-Operated Ca2+ Entry In Mouse Pancreatic Acinar Cells
Dietary Virgin Olive Oil Enhances Secretagogue-Evoked Calcium Signaling In Rat Pancreatic Acinar Cells
Cleavage Of Snap-25 And Vamp-2 Impairs Store-Operated Ca2+ Entry In Mouse Pancreatic Acinar Cells
Dietary Virgin Olive Oil Enhances Secretagogue-Evoked Calcium Signaling In Rat Pancreatic Acinar Cells
Dietary Virgin Olive Oil Enhances Secretagogue-Evoked Calcium Signaling In Rat Pancreatic Acinar Cells
Dietary Virgin Olive Oil Enhances Secretagogue-Evoked Calcium Signaling In Rat Pancreatic Acinar Cells
Dietary Virgin Olive Oil Enhances Secretagogue-Evoked Calcium Signaling In Rat Pancreatic Acinar Cells
Store-Operated Ca 2+ Entry And Tyrosine Kinase Pp60 Src Hyperactivity Are Modulated By Hyperglycemia In Platelets From Patients With Non Insulin-Dependent Diabetes Mellitus
Dietary Virgin Olive Oil Enhances Secretagogue-Evoked Calcium Signaling In Rat Pancreatic Acinar Cells
Store-Operated Ca 2+ Entry And Tyrosine Kinase Pp60 Src Hyperactivity Are Modulated By Hyperglycemia In Platelets From Patients With Non Insulin-Dependent Diabetes Mellitus
Two Pathways For Store-Mediated Calcium Entry Differentially Dependent On The Actin Cytoskeleton In Human Platelets
Store-Operated Ca 2+ Entry And Tyrosine Kinase Pp60 Src Hyperactivity Are Modulated By Hyperglycemia In Platelets From Patients With Non Insulin-Dependent Diabetes Mellitus
Two Pathways For Store-Mediated Calcium Entry Differentially Dependent On The Actin Cytoskeleton In Human Platelets
A Role For Snap-25 But Not Vamps In Store-Mediated Ca2+ Entry In Human Platelets
Two Pathways For Store-Mediated Calcium Entry Differentially Dependent On The Actin Cytoskeleton In Human Platelets
A Role For Snap-25 But Not Vamps In Store-Mediated Ca2+ Entry In Human Platelets
Dual Effect Of Hydrogen Peroxide On Store-Mediated Calcium Entry In Human Platelets
A Role For Snap-25 But Not Vamps In Store-Mediated Ca2+ Entry In Human Platelets
Dual Effect Of Hydrogen Peroxide On Store-Mediated Calcium Entry In Human Platelets
Effect Of Hydrogen Peroxide On Ca2+ Mobilisation In Human Platelets Through Sulphydryl Oxidation Dependent And Independent Mechanisms
Dual Effect Of Hydrogen Peroxide On Store-Mediated Calcium Entry In Human Platelets
Effect Of Hydrogen Peroxide On Ca2+ Mobilisation In Human Platelets Through Sulphydryl Oxidation Dependent And Independent Mechanisms
Hydrogen Peroxide Generation Induces Pp60src Activation In Human Platelets: Evidence For The Involvement Of This Pathway In Store-Mediated Calcium Entry
Effect Of Hydrogen Peroxide On Ca2+ Mobilisation In Human Platelets Through Sulphydryl Oxidation Dependent And Independent Mechanisms
Hydrogen Peroxide Generation Induces Pp60src Activation In Human Platelets: Evidence For The Involvement Of This Pathway In Store-Mediated Calcium Entry
Reduced Plasma Membrane Ca2+-Atpase Function In Platelets From Patients With Non-Insulin-Dependent Diabetes Mellitus
Hydrogen Peroxide Generation Induces Pp60src Activation In Human Platelets: Evidence For The Involvement Of This Pathway In Store-Mediated Calcium Entry
Reduced Plasma Membrane Ca2+-Atpase Function In Platelets From Patients With Non-Insulin-Dependent Diabetes Mellitus
Evidence For Secretion-Like Coupling Involving Pp60src In The Activation And Maintenance Of Store-Mediated Ca2+ Entry In Mouse Pancreatic Acinar Cells
Reduced Plasma Membrane Ca2+-Atpase Function In Platelets From Patients With Non-Insulin-Dependent Diabetes Mellitus
Evidence For Secretion-Like Coupling Involving Pp60src In The Activation And Maintenance Of Store-Mediated Ca2+ Entry In Mouse Pancreatic Acinar Cells
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
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