PUBLICACIÓN

ACCEDER A LA PUBLICACIÓN: Scopus Orcid Dialnet

Cell Biology (Q4), Developmental Biology (Q4), Medicine (miscellaneous) (Q3)

JCR: 1.587

SJR: 0.482

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Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Modulatory Effects Of Cnnm4 On Protein-L-Isoaspartyl-O-Methyltransferase Repair Function During Alcohol-Induced Hepatic Damage
Sumoylation Controls Hu Antigen R Posttranscriptional Activity In Liver Cancer
Modulatory Effects Of Cnnm4 On Protein-L-Isoaspartyl-O-Methyltransferase Repair Function During Alcohol-Induced Hepatic Damage
Sumoylation Controls Hu Antigen R Posttranscriptional Activity In Liver Cancer
Phosphorylation At The Disordered N-End Makes Hur Accumulate And Dimerize In The Cytoplasm
Sumoylation Controls Hu Antigen R Posttranscriptional Activity In Liver Cancer
Phosphorylation At The Disordered N-End Makes Hur Accumulate And Dimerize In The Cytoplasm
Lactate Limits Cns Autoimmunity By Stabilizing Hif-1α In Dendritic Cells
Phosphorylation At The Disordered N-End Makes Hur Accumulate And Dimerize In The Cytoplasm
Lactate Limits Cns Autoimmunity By Stabilizing Hif-1α In Dendritic Cells
Anti-Mir-873-5p Improves Alcohol-Related Liver Disease By Enhancing Hepatic Deacetylation Via Sirt1
Lactate Limits Cns Autoimmunity By Stabilizing Hif-1α In Dendritic Cells
Anti-Mir-873-5p Improves Alcohol-Related Liver Disease By Enhancing Hepatic Deacetylation Via Sirt1
Identification Of Environmental Factors That Promote Intestinal Inflammation
Anti-Mir-873-5p Improves Alcohol-Related Liver Disease By Enhancing Hepatic Deacetylation Via Sirt1
Identification Of Environmental Factors That Promote Intestinal Inflammation
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Identification Of Environmental Factors That Promote Intestinal Inflammation
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Modulatory Effects Of Cnnm4 On Protein-L-Isoaspartyl-O-Methyltransferase Repair Function During Alcohol-Induced Hepatic Damage
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