PUBLICACIÓN

Modulatory effects of CNNM4 on protein-l-isoaspartyl-O-methyltransferase repair function during alcohol-induced hepatic damage

ACCEDER A LA PUBLICACIÓN: Orcid

Irene González-Recio et al.

2025 Hepatology

DOI

10.1097/hep.0000000000001156

AUTORES DE LA UEX

Claudia María Rejano Gordillo

OTRAS PUBLICACIONES DE INTERÉS

Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Modulatory Effects Of Cnnm4 On Protein-L-Isoaspartyl-O-Methyltransferase Repair Function During Alcohol-Induced Hepatic Damage
Role Of Cnnm4 In The Progression Of Cholangiocarcinoma: Implications For Ferroptosis And Therapeutic Potential
Modulatory Effects Of Cnnm4 On Protein-L-Isoaspartyl-O-Methyltransferase Repair Function During Alcohol-Induced Hepatic Damage
Role Of Cnnm4 In The Progression Of Cholangiocarcinoma: Implications For Ferroptosis And Therapeutic Potential
Effect Of Protease-Activated Receptor 2 Inhibition By 1piperidinepropionic Acid In Lipid Accumulation, Inflammation And Hepatocellular Carcinoma Development
Role Of Cnnm4 In The Progression Of Cholangiocarcinoma: Implications For Ferroptosis And Therapeutic Potential
Effect Of Protease-Activated Receptor 2 Inhibition By 1piperidinepropionic Acid In Lipid Accumulation, Inflammation And Hepatocellular Carcinoma Development
Sumoylation Controls Hu Antigen R Posttranscriptional Activity In Liver Cancer
Effect Of Protease-Activated Receptor 2 Inhibition By 1piperidinepropionic Acid In Lipid Accumulation, Inflammation And Hepatocellular Carcinoma Development
Sumoylation Controls Hu Antigen R Posttranscriptional Activity In Liver Cancer
Phosphorylation At The Disordered N-End Makes Hur Accumulate And Dimerize In The Cytoplasm
Sumoylation Controls Hu Antigen R Posttranscriptional Activity In Liver Cancer
Phosphorylation At The Disordered N-End Makes Hur Accumulate And Dimerize In The Cytoplasm
Neddylation Inhibition Prevents Acetaminophen-Induced Liver Damage By Enhancing The Anabolic Cardiolipin Pathway
Phosphorylation At The Disordered N-End Makes Hur Accumulate And Dimerize In The Cytoplasm
Neddylation Inhibition Prevents Acetaminophen-Induced Liver Damage By Enhancing The Anabolic Cardiolipin Pathway
Lactate Limits Cns Autoimmunity By Stabilizing Hif-1α In Dendritic Cells
Neddylation Inhibition Prevents Acetaminophen-Induced Liver Damage By Enhancing The Anabolic Cardiolipin Pathway
Lactate Limits Cns Autoimmunity By Stabilizing Hif-1α In Dendritic Cells
Anti-Mir-873-5p Improves Alcohol-Related Liver Disease By Enhancing Hepatic Deacetylation Via Sirt1
Lactate Limits Cns Autoimmunity By Stabilizing Hif-1α In Dendritic Cells
Anti-Mir-873-5p Improves Alcohol-Related Liver Disease By Enhancing Hepatic Deacetylation Via Sirt1
Engineered Probiotics Limit Cns Autoimmunity By Stabilizing Hif-1α In Dendritic Cells
Anti-Mir-873-5p Improves Alcohol-Related Liver Disease By Enhancing Hepatic Deacetylation Via Sirt1
Engineered Probiotics Limit Cns Autoimmunity By Stabilizing Hif-1α In Dendritic Cells
Identification Of Environmental Factors That Promote Intestinal Inflammation
Engineered Probiotics Limit Cns Autoimmunity By Stabilizing Hif-1α In Dendritic Cells
Identification Of Environmental Factors That Promote Intestinal Inflammation
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Identification Of Environmental Factors That Promote Intestinal Inflammation
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Modulatory Effects Of Cnnm4 On Protein-L-Isoaspartyl-O-Methyltransferase Repair Function During Alcohol-Induced Hepatic Damage
WEB ACCESIBLE