PUBLICACIÓN

Identification of environmental factors that promote intestinal inflammation

ACCEDER A LA PUBLICACIÓN: Orcid

Liliana M. Sanmarco et al.

2022 Nature

DOI

10.1038/s41586-022-05308-6

AUTORES DE LA UEX

Claudia María Rejano Gordillo

OTRAS PUBLICACIONES DE INTERÉS

The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Modulatory Effects Of Cnnm4 On Protein-L-Isoaspartyl-O-Methyltransferase Repair Function During Alcohol-Induced Hepatic Damage
Role Of Cnnm4 In The Progression Of Cholangiocarcinoma: Implications For Ferroptosis And Therapeutic Potential
Modulatory Effects Of Cnnm4 On Protein-L-Isoaspartyl-O-Methyltransferase Repair Function During Alcohol-Induced Hepatic Damage
Role Of Cnnm4 In The Progression Of Cholangiocarcinoma: Implications For Ferroptosis And Therapeutic Potential
Effect Of Protease-Activated Receptor 2 Inhibition By 1piperidinepropionic Acid In Lipid Accumulation, Inflammation And Hepatocellular Carcinoma Development
Role Of Cnnm4 In The Progression Of Cholangiocarcinoma: Implications For Ferroptosis And Therapeutic Potential
Effect Of Protease-Activated Receptor 2 Inhibition By 1piperidinepropionic Acid In Lipid Accumulation, Inflammation And Hepatocellular Carcinoma Development
Metabolic Dysfunction-Associated Steatotic Liver Disease Alters Brain Function And Behavior: Insights From Liver-Targeted Sirna Therapy
Effect Of Protease-Activated Receptor 2 Inhibition By 1piperidinepropionic Acid In Lipid Accumulation, Inflammation And Hepatocellular Carcinoma Development
Metabolic Dysfunction-Associated Steatotic Liver Disease Alters Brain Function And Behavior: Insights From Liver-Targeted Sirna Therapy
Sumoylation Controls Hu Antigen R Posttranscriptional Activity In Liver Cancer
Metabolic Dysfunction-Associated Steatotic Liver Disease Alters Brain Function And Behavior: Insights From Liver-Targeted Sirna Therapy
Sumoylation Controls Hu Antigen R Posttranscriptional Activity In Liver Cancer
Phosphorylation At The Disordered N-End Makes Hur Accumulate And Dimerize In The Cytoplasm
Sumoylation Controls Hu Antigen R Posttranscriptional Activity In Liver Cancer
Phosphorylation At The Disordered N-End Makes Hur Accumulate And Dimerize In The Cytoplasm
Neddylation Inhibition Prevents Acetaminophen-Induced Liver Damage By Enhancing The Anabolic Cardiolipin Pathway
Phosphorylation At The Disordered N-End Makes Hur Accumulate And Dimerize In The Cytoplasm
Neddylation Inhibition Prevents Acetaminophen-Induced Liver Damage By Enhancing The Anabolic Cardiolipin Pathway
Lactate Limits Cns Autoimmunity By Stabilizing Hif-1α In Dendritic Cells
Neddylation Inhibition Prevents Acetaminophen-Induced Liver Damage By Enhancing The Anabolic Cardiolipin Pathway
Lactate Limits Cns Autoimmunity By Stabilizing Hif-1α In Dendritic Cells
Anti-Mir-873-5p Improves Alcohol-Related Liver Disease By Enhancing Hepatic Deacetylation Via Sirt1
Lactate Limits Cns Autoimmunity By Stabilizing Hif-1α In Dendritic Cells
Anti-Mir-873-5p Improves Alcohol-Related Liver Disease By Enhancing Hepatic Deacetylation Via Sirt1
Engineered Probiotics Limit Cns Autoimmunity By Stabilizing Hif-1α In Dendritic Cells
Anti-Mir-873-5p Improves Alcohol-Related Liver Disease By Enhancing Hepatic Deacetylation Via Sirt1
Engineered Probiotics Limit Cns Autoimmunity By Stabilizing Hif-1α In Dendritic Cells
Identification Of Environmental Factors That Promote Intestinal Inflammation
Engineered Probiotics Limit Cns Autoimmunity By Stabilizing Hif-1α In Dendritic Cells
Identification Of Environmental Factors That Promote Intestinal Inflammation
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Identification Of Environmental Factors That Promote Intestinal Inflammation
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Modulatory Effects Of Cnnm4 On Protein-L-Isoaspartyl-O-Methyltransferase Repair Function During Alcohol-Induced Hepatic Damage
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