PUBLICACIÓN

Editorial: Molecular Components of Store-Operated Calcium Entry in Health and Disease

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ACCEDER A LA PUBLICACIÓN: Scopus

Gruszczynska-Biegala J., Martin-Romero F.J., Smani T., Secondo A.

2021 Frontiers in Cellular Neuroscience

Cellular and Molecular Neuroscience (Q1)

JCR: 6.147

SJR: 1.661

CITAS

2

DOI

10.3389/fncel.2021.771138

EID

2-s2.0-85117261509

ISSN

1662-5102

AUTORES DE LA UEX

Francisco Javier Martín Romero

OTRAS PUBLICACIONES DE INTERÉS

Role Of Stim1 In Neurodegeneration
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease, Volume Ii
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease, Volume Ii
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease, Volume Ii
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease
Stim1 Deficiency Leads To Specific Down-Regulation Of Itpr3 In Sh-Sy5y Cells
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease
Stim1 Deficiency Leads To Specific Down-Regulation Of Itpr3 In Sh-Sy5y Cells
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Stim1 Deficiency Leads To Specific Down-Regulation Of Itpr3 In Sh-Sy5y Cells
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Fluctuation In O-Glcnacylation Inactivates Stim1 To Reduce Store-Operated Calcium Ion Entry Via Down-Regulation Of Ser<Sup>621</sup>phosphorylation
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Fluctuation In O-Glcnacylation Inactivates Stim1 To Reduce Store-Operated Calcium Ion Entry Via Down-Regulation Of Ser<Sup>621</sup>phosphorylation
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca<Sup>2+</sup> Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Fluctuation In O-Glcnacylation Inactivates Stim1 To Reduce Store-Operated Calcium Ion Entry Via Down-Regulation Of Ser<Sup>621</sup>phosphorylation
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca<Sup>2+</sup> Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Role Of Stim1 In Neurodegeneration
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca<Sup>2+</sup> Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Role Of Stim1 In Neurodegeneration
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
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