PUBLICACIÓN

Modulation of sarcoplasmic reticulum Ca2+-ATPase by chronic and acute exposure to peroxynitrite

ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Gutiérrez-Martín, Y., Gutiérrez-Merino, C., Henao, F., Iñesta-Vaquera, F.A., Martín-Romero, F.J.

2004 European Journal of Biochemistry

CITAS

57

DOI

10.1111/j.1432-1033.2004.04193.x

EID

2-s2.0-3042803928

ISSN

0014-2956

BIBTEX

@article { martinromero2004,title = {Modulation of sarcoplasmic reticulum Ca2+-ATPase by chronic and acute exposure to peroxynitrite},journal = {European Journal of Biochemistry},year = {2004},volume = {271},number = {13},pages = {2647-2657},author = {Gutiérrez-Martín, Y. and Martín-Romero, F.J. and Iñesta-Vaquera, F.A. and Gutiérrez-Merino, C. and Henao, F.}}

AUTORES DE LA UEX

Francisco De Asís Iñesta Vaquera
Francisco Javier Martín Romero

OTRAS PUBLICACIONES DE INTERÉS

Regulation Of Phosphorylase B By Amp: The Effects Of Enzyme Concentration
3d Printing Of Carbonaceous Hydrogel Electrodes For Energy Applications
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
3d Printing Of Carbonaceous Hydrogel Electrodes For Energy Applications
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
The Potent Human Car Activator Citco Is A Non-Genotoxic Hepatic Tumour-Promoting Agent In Humanised Constitutive Androstane Receptor Mice But Not In Wild-Type Animals
A Subset Of Orai1α And Orai1β Subunits Heteromerizes To Form Crac Channels
The Potent Human Car Activator Citco Is A Non-Genotoxic Hepatic Tumour-Promoting Agent In Humanised Constitutive Androstane Receptor Mice But Not In Wild-Type Animals
Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als
The Potent Human Car Activator Citco Is A Non-Genotoxic Hepatic Tumour-Promoting Agent In Humanised Constitutive Androstane Receptor Mice But Not In Wild-Type Animals
Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als
Comunicación Del Riesgo Para La Salud Humana Causado Por La Contaminación Atmosférica
Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als
Comunicación Del Riesgo Para La Salud Humana Causado Por La Contaminación Atmosférica
Ck2 Phosphorylation Of Cmtr1 Promotes Rna Cap Formation And Influenza Virus Infection
Comunicación Del Riesgo Para La Salud Humana Causado Por La Contaminación Atmosférica
Ck2 Phosphorylation Of Cmtr1 Promotes Rna Cap Formation And Influenza Virus Infection
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
Ck2 Phosphorylation Of Cmtr1 Promotes Rna Cap Formation And Influenza Virus Infection
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
An Update Of Kaempferol Protection Against Brain Damage Induced By Ischemia-Reperfusion And By 3-Nitropropionic Acid
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
An Update Of Kaempferol Protection Against Brain Damage Induced By Ischemia-Reperfusion And By 3-Nitropropionic Acid
Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als.
An Update Of Kaempferol Protection Against Brain Damage Induced By Ischemia-Reperfusion And By 3-Nitropropionic Acid
Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als.
Store-Operated Calcium Entry Inhibition And Plasma Membrane Calcium Pump Upregulation Contribute To The Maintenance Of Resting Cytosolic Calcium Concentration In A1-Like Astrocytes
Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als.
Store-Operated Calcium Entry Inhibition And Plasma Membrane Calcium Pump Upregulation Contribute To The Maintenance Of Resting Cytosolic Calcium Concentration In A1-Like Astrocytes
Brain Hydrophobic Peptides Antagonists Of Neurotoxic Amyloid Β Peptide Monomers/oligomers–protein Interactions
Store-Operated Calcium Entry Inhibition And Plasma Membrane Calcium Pump Upregulation Contribute To The Maintenance Of Resting Cytosolic Calcium Concentration In A1-Like Astrocytes
Brain Hydrophobic Peptides Antagonists Of Neurotoxic Amyloid Β Peptide Monomers/oligomers–protein Interactions
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease, Volume Ii
Brain Hydrophobic Peptides Antagonists Of Neurotoxic Amyloid Β Peptide Monomers/oligomers–protein Interactions
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease, Volume Ii
Hexa-Histidine, A Peptide With Versatile Applications In The Study Of Amyloid-Β(1–42) Molecular Mechanisms Of Action
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease, Volume Ii
Hexa-Histidine, A Peptide With Versatile Applications In The Study Of Amyloid-Β(1–42) Molecular Mechanisms Of Action
Are There Lipid Membrane-Domain Subtypes In Neurons With Different Roles In Calcium Signaling?
Hexa-Histidine, A Peptide With Versatile Applications In The Study Of Amyloid-Β(1–42) Molecular Mechanisms Of Action
Are There Lipid Membrane-Domain Subtypes In Neurons With Different Roles In Calcium Signaling?
Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers
Are There Lipid Membrane-Domain Subtypes In Neurons With Different Roles In Calcium Signaling?
Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers
Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies
Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers
Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies
Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers
Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies
Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers
Biochemical And Biophysical Characterization Of The Caveolin-2 Interaction With Membranes And Analysis Of The Protein Structural Alteration By The Presence Of Cholesterol
Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers
Biochemical And Biophysical Characterization Of The Caveolin-2 Interaction With Membranes And Analysis Of The Protein Structural Alteration By The Presence Of Cholesterol
Structural Features Of Cytochrome B5 –cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Biochemical And Biophysical Characterization Of The Caveolin-2 Interaction With Membranes And Analysis Of The Protein Structural Alteration By The Presence Of Cholesterol
Structural Features Of Cytochrome B5 –cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Inhibition Of Serca And Pmca Ca2+-Atpase Activities By Polyoxotungstates
Structural Features Of Cytochrome B5 –cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Inhibition Of Serca And Pmca Ca2+-Atpase Activities By Polyoxotungstates
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Inhibition Of Serca And Pmca Ca2+-Atpase Activities By Polyoxotungstates
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Special Issue “molecular And Cellular Mechanisms Of Action Of Markers Of Tissue Degeneration”
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Special Issue “molecular And Cellular Mechanisms Of Action Of Markers Of Tissue Degeneration”
Design And Experimental Evaluation Of A Peptide Antagonist Against Amyloid Β(1–42) Interactions With Calmodulin And Calbindin-D28k
Special Issue “molecular And Cellular Mechanisms Of Action Of Markers Of Tissue Degeneration”
Design And Experimental Evaluation Of A Peptide Antagonist Against Amyloid Β(1–42) Interactions With Calmodulin And Calbindin-D28k
Kaempferol Prevents The Activation Of Complement C3 Protein And The Generation Of Reactive A1 Astrocytes That Mediate Rat Brain Degeneration Induced By 3-Nitropropionic Acid
Design And Experimental Evaluation Of A Peptide Antagonist Against Amyloid Β(1–42) Interactions With Calmodulin And Calbindin-D28k
Kaempferol Prevents The Activation Of Complement C3 Protein And The Generation Of Reactive A1 Astrocytes That Mediate Rat Brain Degeneration Induced By 3-Nitropropionic Acid
In Vivo Stress Reporters As Early Biomarkers Of The Cellular Changes Associated With Progeria
Kaempferol Prevents The Activation Of Complement C3 Protein And The Generation Of Reactive A1 Astrocytes That Mediate Rat Brain Degeneration Induced By 3-Nitropropionic Acid
In Vivo Stress Reporters As Early Biomarkers Of The Cellular Changes Associated With Progeria
Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies [Version 1; Peer Review: 2 Approved]
In Vivo Stress Reporters As Early Biomarkers Of The Cellular Changes Associated With Progeria
Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies [Version 1; Peer Review: 2 Approved]
Gold Compounds Inhibit The Ca2+-Atpase Activity Of Brain Pmca And Human Neuroblastoma Sh-Sy5y Cells And Decrease Cell Viability
Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies [Version 1; Peer Review: 2 Approved]
Gold Compounds Inhibit The Ca2+-Atpase Activity Of Brain Pmca And Human Neuroblastoma Sh-Sy5y Cells And Decrease Cell Viability
The Relevance Of Amyloid Β-Calmodulin Complexation In Neurons And Brain Degeneration In Alzheimer’s Disease
Gold Compounds Inhibit The Ca2+-Atpase Activity Of Brain Pmca And Human Neuroblastoma Sh-Sy5y Cells And Decrease Cell Viability
The Relevance Of Amyloid Β-Calmodulin Complexation In Neurons And Brain Degeneration In Alzheimer’s Disease
Binding Of Amyloid Β(1–42)‐calmodulin Complexes To Plasma Membrane Lipid Rafts In Cerebellar Granule Neurons Alters Resting Cytosolic Calcium Homeostasis
The Relevance Of Amyloid Β-Calmodulin Complexation In Neurons And Brain Degeneration In Alzheimer’s Disease
Binding Of Amyloid Β(1–42)‐calmodulin Complexes To Plasma Membrane Lipid Rafts In Cerebellar Granule Neurons Alters Resting Cytosolic Calcium Homeostasis
Application Of The In Vivo Oxidative Stress Reporter Hmox1 As Mechanistic Biomarker Of Arsenic Toxicity
Binding Of Amyloid Β(1–42)‐calmodulin Complexes To Plasma Membrane Lipid Rafts In Cerebellar Granule Neurons Alters Resting Cytosolic Calcium Homeostasis
Application Of The In Vivo Oxidative Stress Reporter Hmox1 As Mechanistic Biomarker Of Arsenic Toxicity
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease
Application Of The In Vivo Oxidative Stress Reporter Hmox1 As Mechanistic Biomarker Of Arsenic Toxicity
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease
Structural Features Of Cytochrome B5–cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Editorial: Molecular Components Of Store-Operated Calcium Entry In Health And Disease
Structural Features Of Cytochrome B5–cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Human Erythrocytes Exposure To Juglone Leads To An Increase Of Superoxide Anion Production Associated With Cytochrome B5 Reductase Uncoupling
Structural Features Of Cytochrome B5–cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Human Erythrocytes Exposure To Juglone Leads To An Increase Of Superoxide Anion Production Associated With Cytochrome B5 Reductase Uncoupling
Stim1 Deficiency Leads To Specific Down-Regulation Of Itpr3 In Sh-Sy5y Cells
Human Erythrocytes Exposure To Juglone Leads To An Increase Of Superoxide Anion Production Associated With Cytochrome B5 Reductase Uncoupling
Stim1 Deficiency Leads To Specific Down-Regulation Of Itpr3 In Sh-Sy5y Cells
Application Of The In Vivo Oxidative Stress Reporter Hmox1 As Mechanistic Biomarker Of Arsenic Toxicity
Stim1 Deficiency Leads To Specific Down-Regulation Of Itpr3 In Sh-Sy5y Cells
Application Of The In Vivo Oxidative Stress Reporter Hmox1 As Mechanistic Biomarker Of Arsenic Toxicity
Early Reactive A1 Astrocytes Induction By The Neurotoxin 3-Nitropropionic Acid In Rat Brain
Application Of The In Vivo Oxidative Stress Reporter Hmox1 As Mechanistic Biomarker Of Arsenic Toxicity
Early Reactive A1 Astrocytes Induction By The Neurotoxin 3-Nitropropionic Acid In Rat Brain
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Early Reactive A1 Astrocytes Induction By The Neurotoxin 3-Nitropropionic Acid In Rat Brain
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Fluctuation In O-Glcnacylation Inactivates Stim1 To Reduce Store-Operated Calcium Ion Entry Via Down-Regulation Of Ser621phosphorylation
Rac1-Dependent Orai1 Translocation To The Leading Edge Supports Lamellipodia Formation And Directional Persistence
Fluctuation In O-Glcnacylation Inactivates Stim1 To Reduce Store-Operated Calcium Ion Entry Via Down-Regulation Of Ser621phosphorylation
Modulation Of Cyp2c9 Activity And Hydrogen Peroxide Production By Cytochrome B 5
Fluctuation In O-Glcnacylation Inactivates Stim1 To Reduce Store-Operated Calcium Ion Entry Via Down-Regulation Of Ser621phosphorylation
Modulation Of Cyp2c9 Activity And Hydrogen Peroxide Production By Cytochrome B 5
Ligand Accessibility To Heme Cytochrome B 5 Coordinating Sphere And Enzymatic Activity Enhancement Upon Tyrosine Ionization
Modulation Of Cyp2c9 Activity And Hydrogen Peroxide Production By Cytochrome B 5
Ligand Accessibility To Heme Cytochrome B 5 Coordinating Sphere And Enzymatic Activity Enhancement Upon Tyrosine Ionization
Methylene Blue Blocks And Reverses The Inhibitory Effect Of Tau On Pmca Function
Ligand Accessibility To Heme Cytochrome B 5 Coordinating Sphere And Enzymatic Activity Enhancement Upon Tyrosine Ionization
Methylene Blue Blocks And Reverses The Inhibitory Effect Of Tau On Pmca Function
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Methylene Blue Blocks And Reverses The Inhibitory Effect Of Tau On Pmca Function
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Methyl-Β-Cyclodextrin Impairs The Phosphorylation Of The Β2 Subunit Of L-Type Calcium Channels And Cytosolic Calcium Homeostasis In Mature Cerebellar Granule Neurons
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Methyl-Β-Cyclodextrin Impairs The Phosphorylation Of The Β2 Subunit Of L-Type Calcium Channels And Cytosolic Calcium Homeostasis In Mature Cerebellar Granule Neurons
Creatine Protects Against Cytosolic Calcium Dysregulation, Mitochondrial Depolarization And Increase Of Reactive Oxygen Species Production In Rotenone-Induced Cell Death Of Cerebellar Granule Neurons
Methyl-Β-Cyclodextrin Impairs The Phosphorylation Of The Β2 Subunit Of L-Type Calcium Channels And Cytosolic Calcium Homeostasis In Mature Cerebellar Granule Neurons
Creatine Protects Against Cytosolic Calcium Dysregulation, Mitochondrial Depolarization And Increase Of Reactive Oxygen Species Production In Rotenone-Induced Cell Death Of Cerebellar Granule Neurons
Cytochrome B5 Reductase Is The Component From Neuronal Synaptic Plasma Membrane Vesicles That Generates Superoxide Anion Upon Stimulation By Cytochrome C
Creatine Protects Against Cytosolic Calcium Dysregulation, Mitochondrial Depolarization And Increase Of Reactive Oxygen Species Production In Rotenone-Induced Cell Death Of Cerebellar Granule Neurons
Cytochrome B5 Reductase Is The Component From Neuronal Synaptic Plasma Membrane Vesicles That Generates Superoxide Anion Upon Stimulation By Cytochrome C
Topography Of Human Cytochrome B5/cytochrome B5 Reductase Interacting Domain And Redox Alterations Upon Complex Formation
Cytochrome B5 Reductase Is The Component From Neuronal Synaptic Plasma Membrane Vesicles That Generates Superoxide Anion Upon Stimulation By Cytochrome C
Topography Of Human Cytochrome B5/cytochrome B5 Reductase Interacting Domain And Redox Alterations Upon Complex Formation
Peroxidase-Like Activity Of Cytochrome B5 Is Triggered Upon Hemichrome Formation In Alkaline Ph
Topography Of Human Cytochrome B5/cytochrome B5 Reductase Interacting Domain And Redox Alterations Upon Complex Formation
Peroxidase-Like Activity Of Cytochrome B5 Is Triggered Upon Hemichrome Formation In Alkaline Ph
Role Of Stim1 In Neurodegeneration
Peroxidase-Like Activity Of Cytochrome B5 Is Triggered Upon Hemichrome Formation In Alkaline Ph
Role Of Stim1 In Neurodegeneration
Methylene Blue Activates The Pmca Activity And Cross-Interacts With Amyloid Β-Peptide, Blocking Aβ-Mediated Pmca Inhibition
Role Of Stim1 In Neurodegeneration
Methylene Blue Activates The Pmca Activity And Cross-Interacts With Amyloid Β-Peptide, Blocking Aβ-Mediated Pmca Inhibition
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Methylene Blue Activates The Pmca Activity And Cross-Interacts With Amyloid Β-Peptide, Blocking Aβ-Mediated Pmca Inhibition
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation
The Interplay Between Cytoskeleton And Calcium Dynamics
Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation
The Interplay Between Cytoskeleton And Calcium Dynamics
Phospholipids And Calmodulin Modulate The Inhibition Of Pmca Activity By Tau
The Interplay Between Cytoskeleton And Calcium Dynamics
Phospholipids And Calmodulin Modulate The Inhibition Of Pmca Activity By Tau
High Affinity Binding Of Amyloid Β-Peptide To Calmodulin: Structural And Functional Implications
Phospholipids And Calmodulin Modulate The Inhibition Of Pmca Activity By Tau
High Affinity Binding Of Amyloid Β-Peptide To Calmodulin: Structural And Functional Implications
Store-Operated Calcium Entry Is Dispensable For The Activation Of Erk1/2 Pathway In Prostate Cancer Cells
High Affinity Binding Of Amyloid Β-Peptide To Calmodulin: Structural And Functional Implications
Store-Operated Calcium Entry Is Dispensable For The Activation Of Erk1/2 Pathway In Prostate Cancer Cells
Regulation Of Membrane Ruffling By Polarized Stim1 And Orai1 In Cortactin-Rich Domains
Store-Operated Calcium Entry Is Dispensable For The Activation Of Erk1/2 Pathway In Prostate Cancer Cells
Regulation Of Membrane Ruffling By Polarized Stim1 And Orai1 In Cortactin-Rich Domains
Regulation And Function Of Cmtr1-Dependent Mrna Cap Methylation
Regulation Of Membrane Ruffling By Polarized Stim1 And Orai1 In Cortactin-Rich Domains
Regulation And Function Of Cmtr1-Dependent Mrna Cap Methylation
Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation
Regulation And Function Of Cmtr1-Dependent Mrna Cap Methylation
Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation
Correlation Between The Potency Of Flavonoids For Cytochrome C Reduction And Inhibition Of Cardiolipin-Induced Peroxidase Activity
Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation
Correlation Between The Potency Of Flavonoids For Cytochrome C Reduction And Inhibition Of Cardiolipin-Induced Peroxidase Activity
High Expression Of Cytochrome B 5 Reductase Isoform 3/cytochrome B 5 System In The Cerebellum And Pyramidal Neurons Of Adult Rat Brain
Correlation Between The Potency Of Flavonoids For Cytochrome C Reduction And Inhibition Of Cardiolipin-Induced Peroxidase Activity
High Expression Of Cytochrome B 5 Reductase Isoform 3/cytochrome B 5 System In The Cerebellum And Pyramidal Neurons Of Adult Rat Brain
Cdk1-Cyclin B1 Activates Rnmt, Coordinating Mrna Cap Methylation With G1 Phase Transcription
High Expression Of Cytochrome B 5 Reductase Isoform 3/cytochrome B 5 System In The Cerebellum And Pyramidal Neurons Of Adult Rat Brain
Cdk1-Cyclin B1 Activates Rnmt, Coordinating Mrna Cap Methylation With G1 Phase Transcription
Human Ifit1 Inhibits Mrna Translation Of Rubulaviruses But Not Other Members Of The Paramyxoviridae Family
Cdk1-Cyclin B1 Activates Rnmt, Coordinating Mrna Cap Methylation With G1 Phase Transcription
Human Ifit1 Inhibits Mrna Translation Of Rubulaviruses But Not Other Members Of The Paramyxoviridae Family
Phospho-Stim1 Is A Downstream Effector That Mediates The Signaling Triggered By Igf-1 In Hek293 Cells
Human Ifit1 Inhibits Mrna Translation Of Rubulaviruses But Not Other Members Of The Paramyxoviridae Family
Phospho-Stim1 Is A Downstream Effector That Mediates The Signaling Triggered By Igf-1 In Hek293 Cells
Stim1 Phosphorylation Triggered By Epidermal Growth Factor Mediates Cell Migration
Phospho-Stim1 Is A Downstream Effector That Mediates The Signaling Triggered By Igf-1 In Hek293 Cells
Stim1 Phosphorylation Triggered By Epidermal Growth Factor Mediates Cell Migration
Cytochrome B5 As A Pleiotropic Metabolic Modulator In Mammalian Cells
Stim1 Phosphorylation Triggered By Epidermal Growth Factor Mediates Cell Migration
Cytochrome B5 As A Pleiotropic Metabolic Modulator In Mammalian Cells
The Decrease Of Nad(P)h:quinone Oxidoreductase 1 Activity And Increase Of Ros Production By Nadph Oxidases Are Early Biomarkers In Doxorubicin Cardiotoxicity
Cytochrome B5 As A Pleiotropic Metabolic Modulator In Mammalian Cells
The Decrease Of Nad(P)h:quinone Oxidoreductase 1 Activity And Increase Of Ros Production By Nadph Oxidases Are Early Biomarkers In Doxorubicin Cardiotoxicity
Thymbra Capitata Essential Oil Prevents Cell Death Induced By 4-Hydroxy-2-Nonenal In Neonatal Rat Cardiac Myocytes
The Decrease Of Nad(P)h:quinone Oxidoreductase 1 Activity And Increase Of Ros Production By Nadph Oxidases Are Early Biomarkers In Doxorubicin Cardiotoxicity
Thymbra Capitata Essential Oil Prevents Cell Death Induced By 4-Hydroxy-2-Nonenal In Neonatal Rat Cardiac Myocytes
Apoptotic Cell Death In Cultured Cardiomyocytes Following Exposure To Low Concentrations Of 4-Hydroxy-2-Nonenal
Thymbra Capitata Essential Oil Prevents Cell Death Induced By 4-Hydroxy-2-Nonenal In Neonatal Rat Cardiac Myocytes
Apoptotic Cell Death In Cultured Cardiomyocytes Following Exposure To Low Concentrations Of 4-Hydroxy-2-Nonenal
Caveolin-Rich Lipid Rafts Of The Plasma Membrane Of Mature Cerebellar Granule Neurons Are Microcompartments For Calcium/reactive Oxygen And Nitrogen Species Cross-Talk Signaling
Apoptotic Cell Death In Cultured Cardiomyocytes Following Exposure To Low Concentrations Of 4-Hydroxy-2-Nonenal
Caveolin-Rich Lipid Rafts Of The Plasma Membrane Of Mature Cerebellar Granule Neurons Are Microcompartments For Calcium/reactive Oxygen And Nitrogen Species Cross-Talk Signaling
Purified Nadh-Cytochrome B5 Reductase Is A Novel Superoxide Anion Source Inhibited By Apocynin: Sensitivity To Nitric Oxide And Peroxynitrite
Caveolin-Rich Lipid Rafts Of The Plasma Membrane Of Mature Cerebellar Granule Neurons Are Microcompartments For Calcium/reactive Oxygen And Nitrogen Species Cross-Talk Signaling
Purified Nadh-Cytochrome B5 Reductase Is A Novel Superoxide Anion Source Inhibited By Apocynin: Sensitivity To Nitric Oxide And Peroxynitrite
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
Purified Nadh-Cytochrome B5 Reductase Is A Novel Superoxide Anion Source Inhibited By Apocynin: Sensitivity To Nitric Oxide And Peroxynitrite
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Regulation Of Stim1 By Phosphorylation
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Regulation Of Stim1 By Phosphorylation
Inhibition Of Stim1 Phosphorylation Underlies Resveratrol-Induced Inhibition Of Store-Operated Calcium Entry
The Regulation Of Stim1 By Phosphorylation
Inhibition Of Stim1 Phosphorylation Underlies Resveratrol-Induced Inhibition Of Store-Operated Calcium Entry
G2019s Leucine-Rich Repeat Kinase 2 Causes Uncoupling Protein-Mediated Mitochondrial Depolarization
Inhibition Of Stim1 Phosphorylation Underlies Resveratrol-Induced Inhibition Of Store-Operated Calcium Entry
G2019s Leucine-Rich Repeat Kinase 2 Causes Uncoupling Protein-Mediated Mitochondrial Depolarization
The Ikappab Kinase Family Phosphorylates The Parkinson's Disease Kinase Lrrk2 At Ser935 And Ser910 During Toll-Like Receptor Signaling
G2019s Leucine-Rich Repeat Kinase 2 Causes Uncoupling Protein-Mediated Mitochondrial Depolarization
The Ikappab Kinase Family Phosphorylates The Parkinson's Disease Kinase Lrrk2 At Ser935 And Ser910 During Toll-Like Receptor Signaling
Discovery Of Potent And Selective Covalent Inhibitors Of Jnk
The Ikappab Kinase Family Phosphorylates The Parkinson's Disease Kinase Lrrk2 At Ser935 And Ser910 During Toll-Like Receptor Signaling
Discovery Of Potent And Selective Covalent Inhibitors Of Jnk
Intracellular Cyclophilin A Is An Important Ca2+ Regulator In Platelets And Critically Involved In Arterial Thrombus Formation
Discovery Of Potent And Selective Covalent Inhibitors Of Jnk
Intracellular Cyclophilin A Is An Important Ca2+ Regulator In Platelets And Critically Involved In Arterial Thrombus Formation
Extended Embryo Culture Supplementation
Intracellular Cyclophilin A Is An Important Ca2+ Regulator In Platelets And Critically Involved In Arterial Thrombus Formation
Extended Embryo Culture Supplementation
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis By Disruption Of Intracellular Calcium Homeostasis In Human Neuronal Cell Line Shsy5y
Extended Embryo Culture Supplementation
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis By Disruption Of Intracellular Calcium Homeostasis In Human Neuronal Cell Line Shsy5y
Calcium Signaling In Mouse Oocyte Maturation: The Roles Of Stim1, Orai1 And Soce
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis By Disruption Of Intracellular Calcium Homeostasis In Human Neuronal Cell Line Shsy5y
Calcium Signaling In Mouse Oocyte Maturation: The Roles Of Stim1, Orai1 And Soce
Role Of Store-Operated Calcium Entry During Meiotic Progression And Fertilization Of Mammalian Oocytes
Calcium Signaling In Mouse Oocyte Maturation: The Roles Of Stim1, Orai1 And Soce
Role Of Store-Operated Calcium Entry During Meiotic Progression And Fertilization Of Mammalian Oocytes
Stimulation And Clustering Of Cytochrome B 5 Reductase In Caveolin-Rich Lipid Microdomains Is An Early Event In Oxidative Stress-Mediated Apoptosis Of Cerebellar Granule Neurons
Role Of Store-Operated Calcium Entry During Meiotic Progression And Fertilization Of Mammalian Oocytes
Stimulation And Clustering Of Cytochrome B 5 Reductase In Caveolin-Rich Lipid Microdomains Is An Early Event In Oxidative Stress-Mediated Apoptosis Of Cerebellar Granule Neurons
L-Type Voltage-Operated Calcium Channels, N-Methyl-D-Aspartate Receptors And Neuronal Nitric-Oxide Synthase Form A Calcium/redox Nano-Transducer Within Lipid Rafts
Stimulation And Clustering Of Cytochrome B 5 Reductase In Caveolin-Rich Lipid Microdomains Is An Early Event In Oxidative Stress-Mediated Apoptosis Of Cerebellar Granule Neurons
L-Type Voltage-Operated Calcium Channels, N-Methyl-D-Aspartate Receptors And Neuronal Nitric-Oxide Synthase Form A Calcium/redox Nano-Transducer Within Lipid Rafts
Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
L-Type Voltage-Operated Calcium Channels, N-Methyl-D-Aspartate Receptors And Neuronal Nitric-Oxide Synthase Form A Calcium/redox Nano-Transducer Within Lipid Rafts
Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
Complex I And Cytochrome C Are Molecular Targets Of Flavonoids That Inhibit Hydrogen Peroxide Production By Mitochondria
Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
Complex I And Cytochrome C Are Molecular Targets Of Flavonoids That Inhibit Hydrogen Peroxide Production By Mitochondria
Neuroprotective Actions Of Flavonoids
Complex I And Cytochrome C Are Molecular Targets Of Flavonoids That Inhibit Hydrogen Peroxide Production By Mitochondria
Neuroprotective Actions Of Flavonoids
Erratum: Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane (Free Radical Biology & Medicine (2011) 50 (323-336))
Neuroprotective Actions Of Flavonoids
Erratum: Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane (Free Radical Biology & Medicine (2011) 50 (323-336))
Inhibition By 4-Hydroxynonenal (Hne) Of Ca 2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
Erratum: Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane (Free Radical Biology & Medicine (2011) 50 (323-336))
Inhibition By 4-Hydroxynonenal (Hne) Of Ca 2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
Early Disruption Of The Actin Cytoskeleton In Cultured Cerebellar Granule Neurons Exposed To 3-Morpholinosydnonimine-Oxidative Stress Is Linked To Alterations Of The Cytosolic Calcium Concentration
Inhibition By 4-Hydroxynonenal (Hne) Of Ca 2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
Early Disruption Of The Actin Cytoskeleton In Cultured Cerebellar Granule Neurons Exposed To 3-Morpholinosydnonimine-Oxidative Stress Is Linked To Alterations Of The Cytosolic Calcium Concentration
Reactivity Of Hydrogen Sulfide With Peroxynitrite And Other Oxidants Of Biological Interest
Early Disruption Of The Actin Cytoskeleton In Cultured Cerebellar Granule Neurons Exposed To 3-Morpholinosydnonimine-Oxidative Stress Is Linked To Alterations Of The Cytosolic Calcium Concentration
Reactivity Of Hydrogen Sulfide With Peroxynitrite And Other Oxidants Of Biological Interest
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Reactivity Of Hydrogen Sulfide With Peroxynitrite And Other Oxidants Of Biological Interest
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Erk5 Pathway Regulates The Phosphorylation Of Tumour Suppressor Hdlg During Mitosis
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Erk5 Pathway Regulates The Phosphorylation Of Tumour Suppressor Hdlg During Mitosis
Differential Activation Of P38mapk Isoforms By Mkk6 And Mkk3
Erk5 Pathway Regulates The Phosphorylation Of Tumour Suppressor Hdlg During Mitosis
Differential Activation Of P38mapk Isoforms By Mkk6 And Mkk3
Alternative Erk5 Regulation By Phosphorylation During The Cell Cycle
Differential Activation Of P38mapk Isoforms By Mkk6 And Mkk3
Alternative Erk5 Regulation By Phosphorylation During The Cell Cycle
Peroxynitrite-Mediated Oxidative Modifications Of Myosin And Implications On Structure And Function
Alternative Erk5 Regulation By Phosphorylation During The Cell Cycle
Peroxynitrite-Mediated Oxidative Modifications Of Myosin And Implications On Structure And Function
L-Type Calcium Channels And Cytochrome B5 Reductase Are Components Of Protein Complexes Tightly Associated With Lipid Rafts Microdomains Of The Neuronal Plasma Membrane
Peroxynitrite-Mediated Oxidative Modifications Of Myosin And Implications On Structure And Function
L-Type Calcium Channels And Cytochrome B5 Reductase Are Components Of Protein Complexes Tightly Associated With Lipid Rafts Microdomains Of The Neuronal Plasma Membrane
P38γ Regulates Interaction Of Nuclear Psf And Rna With The Tumour-Suppressor Hdlg In Response To Osmotic Shock
L-Type Calcium Channels And Cytochrome B5 Reductase Are Components Of Protein Complexes Tightly Associated With Lipid Rafts Microdomains Of The Neuronal Plasma Membrane
P38γ Regulates Interaction Of Nuclear Psf And Rna With The Tumour-Suppressor Hdlg In Response To Osmotic Shock
Kaempferol Protects Against Rat Striatal Degeneration Induced By 3-Nitropropionic Acid
P38γ Regulates Interaction Of Nuclear Psf And Rna With The Tumour-Suppressor Hdlg In Response To Osmotic Shock
Kaempferol Protects Against Rat Striatal Degeneration Induced By 3-Nitropropionic Acid
Role Of Transforming Growth Factor Β In Cancer Microenvironment
Kaempferol Protects Against Rat Striatal Degeneration Induced By 3-Nitropropionic Acid
Role Of Transforming Growth Factor Β In Cancer Microenvironment
Relocalization Of Stim1 In Mouse Oocytes At Fertilization: Early Involvement Of Store-Operated Calcium Entry
Role Of Transforming Growth Factor Β In Cancer Microenvironment
Relocalization Of Stim1 In Mouse Oocytes At Fertilization: Early Involvement Of Store-Operated Calcium Entry
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Relocalization Of Stim1 In Mouse Oocytes At Fertilization: Early Involvement Of Store-Operated Calcium Entry
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Hydrogen Sulfide Is A Reversible Inhibitor Of The Nadh Oxidase Activity Of Synaptic Plasma Membranes
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Hydrogen Sulfide Is A Reversible Inhibitor Of The Nadh Oxidase Activity Of Synaptic Plasma Membranes
Proteolysis Of The Tumour Suppressor Hdlg In Response To Osmotic Stress Is Mediated By Caspases And Independent Of Phosphorylation
Hydrogen Sulfide Is A Reversible Inhibitor Of The Nadh Oxidase Activity Of Synaptic Plasma Membranes
Proteolysis Of The Tumour Suppressor Hdlg In Response To Osmotic Stress Is Mediated By Caspases And Independent Of Phosphorylation
Store-Operated Calcium Entry In Human Oocytes And Sensitivity To Oxidative Stress
Proteolysis Of The Tumour Suppressor Hdlg In Response To Osmotic Stress Is Mediated By Caspases And Independent Of Phosphorylation
Store-Operated Calcium Entry In Human Oocytes And Sensitivity To Oxidative Stress
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Store-Operated Calcium Entry In Human Oocytes And Sensitivity To Oxidative Stress
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Contribution Of Culture Media To Oxidative Stress And Its Effect On Human Oocytes
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Contribution Of Culture Media To Oxidative Stress And Its Effect On Human Oocytes
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Contribution Of Culture Media To Oxidative Stress And Its Effect On Human Oocytes
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Sarcoplasmic Reticulum Calcium Atpase Is Inhibited By Organic Vanadium Coordination Compounds: Pyridine-2,6-Dicarboxylatodioxovanadium(V), Bmov, And An Amavadine Analogue
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Sarcoplasmic Reticulum Calcium Atpase Is Inhibited By Organic Vanadium Coordination Compounds: Pyridine-2,6-Dicarboxylatodioxovanadium(V), Bmov, And An Amavadine Analogue
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Sarcoplasmic Reticulum Calcium Atpase Is Inhibited By Organic Vanadium Coordination Compounds: Pyridine-2,6-Dicarboxylatodioxovanadium(V), Bmov, And An Amavadine Analogue
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Vanadium And Cadmium In Vivo Effects In Teleost Cardiac Muscle: Metal Accumulation And Oxidative Stress Markers
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Vanadium And Cadmium In Vivo Effects In Teleost Cardiac Muscle: Metal Accumulation And Oxidative Stress Markers
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Vanadium And Cadmium In Vivo Effects In Teleost Cardiac Muscle: Metal Accumulation And Oxidative Stress Markers
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Alternative P38 Mapk Pathways
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Alternative P38 Mapk Pathways
Biological Effects Of Decavanadate: Muscle Contraction, In Vivo Oxidative Stress, And Mitochondrial Toxicity
Alternative P38 Mapk Pathways
Biological Effects Of Decavanadate: Muscle Contraction, In Vivo Oxidative Stress, And Mitochondrial Toxicity
Vanadium Distribution, Lipid Peroxidation And Oxidative Stress Markers Upon Decavanadate In Vivo Administration
Biological Effects Of Decavanadate: Muscle Contraction, In Vivo Oxidative Stress, And Mitochondrial Toxicity
Vanadium Distribution, Lipid Peroxidation And Oxidative Stress Markers Upon Decavanadate In Vivo Administration
Mitochondria As A Target For Decavanadate Toxicity In Sparus Aurata Heart
Vanadium Distribution, Lipid Peroxidation And Oxidative Stress Markers Upon Decavanadate In Vivo Administration
Mitochondria As A Target For Decavanadate Toxicity In Sparus Aurata Heart
Decavanadate Induces Mitochondrial Membrane Depolarization And Inhibits Oxygen Consumption
Mitochondria As A Target For Decavanadate Toxicity In Sparus Aurata Heart
Decavanadate Induces Mitochondrial Membrane Depolarization And Inhibits Oxygen Consumption
Binding Modes Of Decavanadate To Myosin And Inhibition Of The Actomyosin Atpase Activity
Decavanadate Induces Mitochondrial Membrane Depolarization And Inhibits Oxygen Consumption
Binding Modes Of Decavanadate To Myosin And Inhibition Of The Actomyosin Atpase Activity
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Binding Modes Of Decavanadate To Myosin And Inhibition Of The Actomyosin Atpase Activity
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Decavanadate Interactions With Actin: Inhibition Of G-Actin Polymerization And Stabilization Of Decameric Vanadate
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Decavanadate Interactions With Actin: Inhibition Of G-Actin Polymerization And Stabilization Of Decameric Vanadate
Peroxynitrite Induces F-Actin Depolymerization And Blockade Of Myosin Atpase Stimulation
Decavanadate Interactions With Actin: Inhibition Of G-Actin Polymerization And Stabilization Of Decameric Vanadate
Peroxynitrite Induces F-Actin Depolymerization And Blockade Of Myosin Atpase Stimulation
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Peroxynitrite Induces F-Actin Depolymerization And Blockade Of Myosin Atpase Stimulation
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Store-Operated Calcium Entry In Differentiated C2c12 Skeletal Muscle Cells
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Store-Operated Calcium Entry In Differentiated C2c12 Skeletal Muscle Cells
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Store-Operated Calcium Entry In Differentiated C2c12 Skeletal Muscle Cells
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Vanadate Oligomers: In Vivo Effects In Hepatic Vanadium Accumulation And Stress Markers
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Vanadate Oligomers: In Vivo Effects In Hepatic Vanadium Accumulation And Stress Markers
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Vanadate Oligomers: In Vivo Effects In Hepatic Vanadium Accumulation And Stress Markers
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Decavanadate Binding To A High Affinity Site Near The Myosin Catalytic Centre Inhibits F-Actin-Stimulated Myosin Atpase Activity
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Decavanadate Binding To A High Affinity Site Near The Myosin Catalytic Centre Inhibits F-Actin-Stimulated Myosin Atpase Activity
The Drosophila Selenoprotein Bthd Is Required For Survival And Has A Role In Salivary Gland Development
Decavanadate Binding To A High Affinity Site Near The Myosin Catalytic Centre Inhibits F-Actin-Stimulated Myosin Atpase Activity
The Drosophila Selenoprotein Bthd Is Required For Survival And Has A Role In Salivary Gland Development
Projection Map Of Covalently Phosphorylated Ca-Atpase From Tubular Crystals
The Drosophila Selenoprotein Bthd Is Required For Survival And Has A Role In Salivary Gland Development
Projection Map Of Covalently Phosphorylated Ca-Atpase From Tubular Crystals
The Trapping Of The Oh Radical By Coenzyme Q. A Theoretical And Experimental Study
Projection Map Of Covalently Phosphorylated Ca-Atpase From Tubular Crystals
The Trapping Of The Oh Radical By Coenzyme Q. A Theoretical And Experimental Study
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
The Trapping Of The Oh Radical By Coenzyme Q. A Theoretical And Experimental Study
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
Quenching Of Myosin Intrinsic Fluorescence Unravels The Existence Of A High Affinity Binding Site For Decavanadate
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
Quenching Of Myosin Intrinsic Fluorescence Unravels The Existence Of A High Affinity Binding Site For Decavanadate
Sphingomyelin Composition And Physical Asymmetries In Native Acetylcholine Receptor-Rich Membranes
Quenching Of Myosin Intrinsic Fluorescence Unravels The Existence Of A High Affinity Binding Site For Decavanadate
Sphingomyelin Composition And Physical Asymmetries In Native Acetylcholine Receptor-Rich Membranes
Selenium Metabolism In Drosophila. Selenoproteins, Selenoprotein Mrna Expression, Fertility, And Mortality
Sphingomyelin Composition And Physical Asymmetries In Native Acetylcholine Receptor-Rich Membranes
Selenium Metabolism In Drosophila. Selenoproteins, Selenoprotein Mrna Expression, Fertility, And Mortality
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Selenium Metabolism In Drosophila. Selenoproteins, Selenoprotein Mrna Expression, Fertility, And Mortality
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Phosphorylated Ca2+-Atpase Stable Enough For Structural Studies
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Phosphorylated Ca2+-Atpase Stable Enough For Structural Studies
A Remarkably Stable Phosphorylated Form Of Ca2+-Atpase Prepared From Ca2+-Loaded And Fluorescein Isothiocyanate-Labeled Sarcoplasmic Reticulum Vesicles
Phosphorylated Ca2+-Atpase Stable Enough For Structural Studies
A Remarkably Stable Phosphorylated Form Of Ca2+-Atpase Prepared From Ca2+-Loaded And Fluorescein Isothiocyanate-Labeled Sarcoplasmic Reticulum Vesicles
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
A Remarkably Stable Phosphorylated Form Of Ca2+-Atpase Prepared From Ca2+-Loaded And Fluorescein Isothiocyanate-Labeled Sarcoplasmic Reticulum Vesicles
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
The Expression Of Plasma Membrane Ca2+ Pump Isoforms In Cerebellar Granule Neurons Is Modulated By Ca2+
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
The Expression Of Plasma Membrane Ca2+ Pump Isoforms In Cerebellar Granule Neurons Is Modulated By Ca2+
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
The Expression Of Plasma Membrane Ca2+ Pump Isoforms In Cerebellar Granule Neurons Is Modulated By Ca2+
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Characterization Of A Protease-Resistant Domain Of The Cytosolic Portion Of Sarcoplasmic Reticulum Ca2+-Atpase. Nucleotide- And Metal-Binding Sites
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Characterization Of A Protease-Resistant Domain Of The Cytosolic Portion Of Sarcoplasmic Reticulum Ca2+-Atpase. Nucleotide- And Metal-Binding Sites
Analysis Of Secreted Flavonoids Of Cistus Ladanifer L. By High- Performance Liquid Chromatography-Particle Beam Mass Spectrometry
Characterization Of A Protease-Resistant Domain Of The Cytosolic Portion Of Sarcoplasmic Reticulum Ca2+-Atpase. Nucleotide- And Metal-Binding Sites
Analysis Of Secreted Flavonoids Of Cistus Ladanifer L. By High- Performance Liquid Chromatography-Particle Beam Mass Spectrometry
Dissociation Of Ca2+ From Sarcoplasmic Reticulum Ca2+-Atpase And Changes In Fluorescence Of Optically Selected Trp Residues. Effects Of Kcl And Nacl And Implications For Substeps In Ca2+ Dissociation
Analysis Of Secreted Flavonoids Of Cistus Ladanifer L. By High- Performance Liquid Chromatography-Particle Beam Mass Spectrometry
Dissociation Of Ca2+ From Sarcoplasmic Reticulum Ca2+-Atpase And Changes In Fluorescence Of Optically Selected Trp Residues. Effects Of Kcl And Nacl And Implications For Substeps In Ca2+ Dissociation
Quantitative Variation Of Flavonoids Among Individuals Of A Cistus Ladanifer Population
Dissociation Of Ca2+ From Sarcoplasmic Reticulum Ca2+-Atpase And Changes In Fluorescence Of Optically Selected Trp Residues. Effects Of Kcl And Nacl And Implications For Substeps In Ca2+ Dissociation
Quantitative Variation Of Flavonoids Among Individuals Of A Cistus Ladanifer Population
Role Of Ecological Variables In The Seasonal Variation Of Flavonoid Content Of Cistus Ladanifer Exudate
Quantitative Variation Of Flavonoids Among Individuals Of A Cistus Ladanifer Population
Role Of Ecological Variables In The Seasonal Variation Of Flavonoid Content Of Cistus Ladanifer Exudate
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Role Of Ecological Variables In The Seasonal Variation Of Flavonoid Content Of Cistus Ladanifer Exudate
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Hypothalamic Hypophyseal Inhibitory Factor (Hhif) Increases Intrasynaptosomal Free Calcium Concentration
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Hypothalamic Hypophyseal Inhibitory Factor (Hhif) Increases Intrasynaptosomal Free Calcium Concentration
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
Hypothalamic Hypophyseal Inhibitory Factor (Hhif) Increases Intrasynaptosomal Free Calcium Concentration
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
A Comparative Kinetic Analysis Of The Flavin-Photosensitized Oxidation And Reduction Of Plastocyanin And Cytochrome C6from Different Organisms
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
A Comparative Kinetic Analysis Of The Flavin-Photosensitized Oxidation And Reduction Of Plastocyanin And Cytochrome C6from Different Organisms
Physical State Of Bulk And Protein-Associated Lipid In Nicotinic Acetylcholine Receptor-Rich Membrane Studied By Laurdan Generalized Polarization And Fluorescence Energy Transfer
A Comparative Kinetic Analysis Of The Flavin-Photosensitized Oxidation And Reduction Of Plastocyanin And Cytochrome C6from Different Organisms
Physical State Of Bulk And Protein-Associated Lipid In Nicotinic Acetylcholine Receptor-Rich Membrane Studied By Laurdan Generalized Polarization And Fluorescence Energy Transfer
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
Physical State Of Bulk And Protein-Associated Lipid In Nicotinic Acetylcholine Receptor-Rich Membrane Studied By Laurdan Generalized Polarization And Fluorescence Energy Transfer
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Rate Of Na+/ca2+ Exchange Across The Plasma Membrane Of Synaptosomes Measured Using The Fluorescence Of Chlorotetracycline. Implications To Calcium Homeostasis In Synaptic Terminals
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Rate Of Na+/ca2+ Exchange Across The Plasma Membrane Of Synaptosomes Measured Using The Fluorescence Of Chlorotetracycline. Implications To Calcium Homeostasis In Synaptic Terminals
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Rate Of Na+/ca2+ Exchange Across The Plasma Membrane Of Synaptosomes Measured Using The Fluorescence Of Chlorotetracycline. Implications To Calcium Homeostasis In Synaptic Terminals
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Preferential Distribution Of The Fluorescent Phospholipid Probes Nbd-Phosphatidylcholine And Rhodamine-Phosphatidylethanolamine In The Exofacial Leaflet Of Acetylcholine Receptor-Rich Membranes From Torpedo Marmorata
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Preferential Distribution Of The Fluorescent Phospholipid Probes Nbd-Phosphatidylcholine And Rhodamine-Phosphatidylethanolamine In The Exofacial Leaflet Of Acetylcholine Receptor-Rich Membranes From Torpedo Marmorata
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Preferential Distribution Of The Fluorescent Phospholipid Probes Nbd-Phosphatidylcholine And Rhodamine-Phosphatidylethanolamine In The Exofacial Leaflet Of Acetylcholine Receptor-Rich Membranes From Torpedo Marmorata
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Modulation Of The Ca2+ Pump By The Hypothalamic-Hypophysary Inhibitory Factor
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Modulation Of The Ca2+ Pump By The Hypothalamic-Hypophysary Inhibitory Factor
Modulation Of The Ca 2+,mg2+-Atpase Of Sarcoplasmic Reticulum By The Hypothalamic Hypophyseal Inhibitory Factor
Modulation Of The Ca2+ Pump By The Hypothalamic-Hypophysary Inhibitory Factor
Modulation Of The Ca 2+,mg2+-Atpase Of Sarcoplasmic Reticulum By The Hypothalamic Hypophyseal Inhibitory Factor
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Modulation Of The Ca 2+,mg2+-Atpase Of Sarcoplasmic Reticulum By The Hypothalamic Hypophyseal Inhibitory Factor
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Cancellation Of The Cooperativity Of Ca2+ Binding To Sarcoplasmic Reticulum Ca2+‐atpase By The Non‐ionic Detergent Dodecylmaltoside
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Cancellation Of The Cooperativity Of Ca2+ Binding To Sarcoplasmic Reticulum Ca2+‐atpase By The Non‐ionic Detergent Dodecylmaltoside
Seasonal Variation Of Exudate Of Cistus Ladanifer
Cancellation Of The Cooperativity Of Ca2+ Binding To Sarcoplasmic Reticulum Ca2+‐atpase By The Non‐ionic Detergent Dodecylmaltoside
Seasonal Variation Of Exudate Of Cistus Ladanifer
Glycogen Phosphorolysis Can Form A Metabolic Shuttle To Support Ca2+ Uptake By Sarcoplasmic Reticulum Membranes In Skeletal Muscle
Seasonal Variation Of Exudate Of Cistus Ladanifer
Glycogen Phosphorolysis Can Form A Metabolic Shuttle To Support Ca2+ Uptake By Sarcoplasmic Reticulum Membranes In Skeletal Muscle
Advanced Course On Structure-Function Relationships Of Membrane Proteins: An Assessment Of Current Knowledge
Glycogen Phosphorolysis Can Form A Metabolic Shuttle To Support Ca2+ Uptake By Sarcoplasmic Reticulum Membranes In Skeletal Muscle
Advanced Course On Structure-Function Relationships Of Membrane Proteins: An Assessment Of Current Knowledge
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Advanced Course On Structure-Function Relationships Of Membrane Proteins: An Assessment Of Current Knowledge
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Properties Of The Purified Hypothalamic Pituitary Na/k-Atpase Inhibitor
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Properties Of The Purified Hypothalamic Pituitary Na/k-Atpase Inhibitor
Location Of Functional Centers In The Microsomal Cytochrome P450 System
Properties Of The Purified Hypothalamic Pituitary Na/k-Atpase Inhibitor
Location Of Functional Centers In The Microsomal Cytochrome P450 System
The Metal Sites On Sarcoplasmic Reticulum Membranes That Bind Lanthanide Ions With The Highest Affinity Are Not The Atpase Ca2+ Transport Sites
Location Of Functional Centers In The Microsomal Cytochrome P450 System
The Metal Sites On Sarcoplasmic Reticulum Membranes That Bind Lanthanide Ions With The Highest Affinity Are Not The Atpase Ca2+ Transport Sites
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
The Metal Sites On Sarcoplasmic Reticulum Membranes That Bind Lanthanide Ions With The Highest Affinity Are Not The Atpase Ca2+ Transport Sites
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Binding Of Gadolinium Ions To Sarcoplasmic Reticulum Membranes
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Binding Of Gadolinium Ions To Sarcoplasmic Reticulum Membranes
Kinetic Characterization Of The Perturbation By Dodecylmaltoside Of Sarcoplasmic Reticulum Ca2+ ‐atpase
Binding Of Gadolinium Ions To Sarcoplasmic Reticulum Membranes
Kinetic Characterization Of The Perturbation By Dodecylmaltoside Of Sarcoplasmic Reticulum Ca2+ ‐atpase
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Kinetic Characterization Of The Perturbation By Dodecylmaltoside Of Sarcoplasmic Reticulum Ca2+ ‐atpase
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Modulation By Phosphorylation Of Glycogen Phosphorylase-Sarcoplasmic Reticulum Interaction
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Modulation By Phosphorylation Of Glycogen Phosphorylase-Sarcoplasmic Reticulum Interaction
Caracterización Por Técnicas Espectrofotométricas Del Ultravioleta Y Visible Edel Exudado De Cistus Ladanifer L. Como Respuesta Al Estrés Estival
Modulation By Phosphorylation Of Glycogen Phosphorylase-Sarcoplasmic Reticulum Interaction
Caracterización Por Técnicas Espectrofotométricas Del Ultravioleta Y Visible Edel Exudado De Cistus Ladanifer L. Como Respuesta Al Estrés Estival
Hemin And Hemeprotein Bleaching During Linoleic Acid Oxidation By Lipoxygenases
Caracterización Por Técnicas Espectrofotométricas Del Ultravioleta Y Visible Edel Exudado De Cistus Ladanifer L. Como Respuesta Al Estrés Estival
Hemin And Hemeprotein Bleaching During Linoleic Acid Oxidation By Lipoxygenases
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Hemin And Hemeprotein Bleaching During Linoleic Acid Oxidation By Lipoxygenases
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Modulation Of Calcium Fluxes Across Synaptosomal Plasma Membrane By Local Anesthetics
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Modulation Of Calcium Fluxes Across Synaptosomal Plasma Membrane By Local Anesthetics
Modulation Of The Ca2+,mg2+‐atpase Activity Of Synaptosomal Plasma Membrane By The Local Anesthetics Dibucaine And Lidocaine
Modulation Of Calcium Fluxes Across Synaptosomal Plasma Membrane By Local Anesthetics
Modulation Of The Ca2+,mg2+‐atpase Activity Of Synaptosomal Plasma Membrane By The Local Anesthetics Dibucaine And Lidocaine
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Modulation Of The Ca2+,mg2+‐atpase Activity Of Synaptosomal Plasma Membrane By The Local Anesthetics Dibucaine And Lidocaine
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Interaction Of The Local Anesthetics Dibucaine And Tetracaine With Sarcoplasmic Reticulum Membranes. Differential Scanning Calorimetry And Fluorescence Studies
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Interaction Of The Local Anesthetics Dibucaine And Tetracaine With Sarcoplasmic Reticulum Membranes. Differential Scanning Calorimetry And Fluorescence Studies
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Interaction Of The Local Anesthetics Dibucaine And Tetracaine With Sarcoplasmic Reticulum Membranes. Differential Scanning Calorimetry And Fluorescence Studies
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Local Anesthetic-Divalent Cation Binding Center Interaction
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Local Anesthetic-Divalent Cation Binding Center Interaction
Comparative Fluorescence Properties Of Lipoxygenases
Local Anesthetic-Divalent Cation Binding Center Interaction
Comparative Fluorescence Properties Of Lipoxygenases
Effects Of Local Anesthetics On The Passive Permeability Of Sarcoplasmic Reticulum Vesicles To Ca2+ And Mg2+
Comparative Fluorescence Properties Of Lipoxygenases
Effects Of Local Anesthetics On The Passive Permeability Of Sarcoplasmic Reticulum Vesicles To Ca2+ And Mg2+
The Position Of The Atp Binding Site On The (Ca2+ + Mg2+)-Atpase
Effects Of Local Anesthetics On The Passive Permeability Of Sarcoplasmic Reticulum Vesicles To Ca2+ And Mg2+
The Position Of The Atp Binding Site On The (Ca2+ + Mg2+)-Atpase
Long-Wavelength Fluorescence Of Tyrosine And Tryptophan Solutions.
The Position Of The Atp Binding Site On The (Ca2+ + Mg2+)-Atpase
Long-Wavelength Fluorescence Of Tyrosine And Tryptophan Solutions.
Local Anesthetics Inhibit The Ca2+,mg2+‐atpase Activity Of Rat Brain Synaptosomes
Long-Wavelength Fluorescence Of Tyrosine And Tryptophan Solutions.
Local Anesthetics Inhibit The Ca2+,mg2+‐atpase Activity Of Rat Brain Synaptosomes
Dependence Of The Fluorescence Of Fluorescein Labelled (Ca2+, Mg2+)-Atpase Upon The Lipid To Protein Ratio In Sarcoplasmic Reticulum Reconstituted Systems
Local Anesthetics Inhibit The Ca2+,mg2+‐atpase Activity Of Rat Brain Synaptosomes
Dependence Of The Fluorescence Of Fluorescein Labelled (Ca2+, Mg2+)-Atpase Upon The Lipid To Protein Ratio In Sarcoplasmic Reticulum Reconstituted Systems
On The Rate Of Association And Dissociation Processes Of Intrinsic Membrane Proteins
Dependence Of The Fluorescence Of Fluorescein Labelled (Ca2+, Mg2+)-Atpase Upon The Lipid To Protein Ratio In Sarcoplasmic Reticulum Reconstituted Systems
On The Rate Of Association And Dissociation Processes Of Intrinsic Membrane Proteins
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. Ii. Protein Distribution And Aggregation State In Biological Membranes
On The Rate Of Association And Dissociation Processes Of Intrinsic Membrane Proteins
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. Ii. Protein Distribution And Aggregation State In Biological Membranes
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. I. Lateral Phase Separation In Unilamellar Vesicles Formed By Binary Phospholipid Mixtures
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. Ii. Protein Distribution And Aggregation State In Biological Membranes
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. I. Lateral Phase Separation In Unilamellar Vesicles Formed By Binary Phospholipid Mixtures
Relationship Between The Activity Of Pancreatic Phospholipase A2 And The Physical State Of The Phospholipid Substrate.
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. I. Lateral Phase Separation In Unilamellar Vesicles Formed By Binary Phospholipid Mixtures
Relationship Between The Activity Of Pancreatic Phospholipase A2 And The Physical State Of The Phospholipid Substrate.
Regulation Of Phosphorylase B By Amp: The Effects Of Enzyme Concentration
Relationship Between The Activity Of Pancreatic Phospholipase A2 And The Physical State Of The Phospholipid Substrate.
Regulation Of Phosphorylase B By Amp: The Effects Of Enzyme Concentration
3d Printing Of Carbonaceous Hydrogel Electrodes For Energy Applications
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