PUBLICACIÓN

Potential of in vivo stress reporter models to reduce animal use and provide mechanistic insights in toxicity studies [version 1; peer review: 2 approved]

ACCEDER A LA PUBLICACIÓN: Orcid

Francisco Iñesta Vaquera; Febe Ferro; Michael McMahon; Colin J. Henderson; C. Roland Wolf; Ferro Febe; Henderson Colin J.; Iñesta Vaquera Francisco; McMahon Michael; Wolf C. Roland

2022 F1000Research

Immunology and Microbiology (miscellaneous) (Q2), Medicine (miscellaneous) (Q1), Pharmacology, Toxicology and Pharmaceutics (miscellaneous) (Q1), Biochemistry, Genetics and Molecular Biology (miscellaneous) (Q2)

SJR: 0.871

DOI

10.12688/f1000research.123077.1

ISSN

2046-1402

AUTORES DE LA UEX

Francisco De Asís Iñesta Vaquera

OTRAS PUBLICACIONES DE INTERÉS

Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
3d Printing Of Carbonaceous Hydrogel Electrodes For Energy Applications
Study Of Printable And Biocompatible Alginate–carbon Hydrogels For Sensor Applications: Mechanical, Electrical, And Cytotoxicity Evaluation
3d Printing Of Carbonaceous Hydrogel Electrodes For Energy Applications
Study Of Printable And Biocompatible Alginate–carbon Hydrogels For Sensor Applications: Mechanical, Electrical, And Cytotoxicity Evaluation
The Potent Human Car Activator Citco Is A Non-Genotoxic Hepatic Tumour-Promoting Agent In Humanised Constitutive Androstane Receptor Mice But Not In Wild-Type Animals
Study Of Printable And Biocompatible Alginate–carbon Hydrogels For Sensor Applications: Mechanical, Electrical, And Cytotoxicity Evaluation
The Potent Human Car Activator Citco Is A Non-Genotoxic Hepatic Tumour-Promoting Agent In Humanised Constitutive Androstane Receptor Mice But Not In Wild-Type Animals
Integration Of In Vivo Stress Reporters With Transcription Profiling To Define Chemical Mixtures Mechanisms Of Toxicity, Including Trans-Generational Effects
The Potent Human Car Activator Citco Is A Non-Genotoxic Hepatic Tumour-Promoting Agent In Humanised Constitutive Androstane Receptor Mice But Not In Wild-Type Animals
Integration Of In Vivo Stress Reporters With Transcription Profiling To Define Chemical Mixtures Mechanisms Of Toxicity, Including Trans-Generational Effects
From Medical Imaging To Bioprinted Tissues: The Importance Of Workflow Optimisation For Improved Cell Function
Integration Of In Vivo Stress Reporters With Transcription Profiling To Define Chemical Mixtures Mechanisms Of Toxicity, Including Trans-Generational Effects
From Medical Imaging To Bioprinted Tissues: The Importance Of Workflow Optimisation For Improved Cell Function
Ck2 Phosphorylation Of Cmtr1 Promotes Rna Cap Formation And Influenza Virus Infection
From Medical Imaging To Bioprinted Tissues: The Importance Of Workflow Optimisation For Improved Cell Function
Ck2 Phosphorylation Of Cmtr1 Promotes Rna Cap Formation And Influenza Virus Infection
Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als
Ck2 Phosphorylation Of Cmtr1 Promotes Rna Cap Formation And Influenza Virus Infection
Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als
Comunicación Del Riesgo Para La Salud Humana Causado Por La Contaminación Atmosférica
Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als
Comunicación Del Riesgo Para La Salud Humana Causado Por La Contaminación Atmosférica
Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers
Comunicación Del Riesgo Para La Salud Humana Causado Por La Contaminación Atmosférica
Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers
Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies
Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers
Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies
Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als.
Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies
Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als.
In Vivo Stress Reporters As Early Biomarkers Of The Cellular Changes Associated With Progeria
Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als.
In Vivo Stress Reporters As Early Biomarkers Of The Cellular Changes Associated With Progeria
Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies [Version 1; Peer Review: 2 Approved]
In Vivo Stress Reporters As Early Biomarkers Of The Cellular Changes Associated With Progeria
Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies [Version 1; Peer Review: 2 Approved]
Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers
Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies [Version 1; Peer Review: 2 Approved]
Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers
Application Of The In Vivo Oxidative Stress Reporter Hmox1 As Mechanistic Biomarker Of Arsenic Toxicity
Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers
Application Of The In Vivo Oxidative Stress Reporter Hmox1 As Mechanistic Biomarker Of Arsenic Toxicity
Application Of The In Vivo Oxidative Stress Reporter Hmox1 As Mechanistic Biomarker Of Arsenic Toxicity
Application Of The In Vivo Oxidative Stress Reporter Hmox1 As Mechanistic Biomarker Of Arsenic Toxicity
Application Of The In Vivo Oxidative Stress Reporter Hmox1 As Mechanistic Biomarker Of Arsenic Toxicity
Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation
Application Of The In Vivo Oxidative Stress Reporter Hmox1 As Mechanistic Biomarker Of Arsenic Toxicity
Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation
Regulation And Function Of Cmtr1-Dependent Mrna Cap Methylation
Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation
Regulation And Function Of Cmtr1-Dependent Mrna Cap Methylation
Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation
Regulation And Function Of Cmtr1-Dependent Mrna Cap Methylation
Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation
Cdk1-Cyclin B1 Activates Rnmt, Coordinating Mrna Cap Methylation With G1 Phase Transcription
Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation
Cdk1-Cyclin B1 Activates Rnmt, Coordinating Mrna Cap Methylation With G1 Phase Transcription
Human Ifit1 Inhibits Mrna Translation Of Rubulaviruses But Not Other Members Of The Paramyxoviridae Family
Cdk1-Cyclin B1 Activates Rnmt, Coordinating Mrna Cap Methylation With G1 Phase Transcription
Human Ifit1 Inhibits Mrna Translation Of Rubulaviruses But Not Other Members Of The Paramyxoviridae Family
G2019s Leucine-Rich Repeat Kinase 2 Causes Uncoupling Protein-Mediated Mitochondrial Depolarization
Human Ifit1 Inhibits Mrna Translation Of Rubulaviruses But Not Other Members Of The Paramyxoviridae Family
G2019s Leucine-Rich Repeat Kinase 2 Causes Uncoupling Protein-Mediated Mitochondrial Depolarization
The Ikappab Kinase Family Phosphorylates The Parkinson's Disease Kinase Lrrk2 At Ser935 And Ser910 During Toll-Like Receptor Signaling
G2019s Leucine-Rich Repeat Kinase 2 Causes Uncoupling Protein-Mediated Mitochondrial Depolarization
The Ikappab Kinase Family Phosphorylates The Parkinson's Disease Kinase Lrrk2 At Ser935 And Ser910 During Toll-Like Receptor Signaling
Discovery Of Potent And Selective Covalent Inhibitors Of Jnk
The Ikappab Kinase Family Phosphorylates The Parkinson's Disease Kinase Lrrk2 At Ser935 And Ser910 During Toll-Like Receptor Signaling
Discovery Of Potent And Selective Covalent Inhibitors Of Jnk
Erk5 Pathway Regulates The Phosphorylation Of Tumour Suppressor Hdlg During Mitosis
Discovery Of Potent And Selective Covalent Inhibitors Of Jnk
Erk5 Pathway Regulates The Phosphorylation Of Tumour Suppressor Hdlg During Mitosis
Differential Activation Of P38mapk Isoforms By Mkk6 And Mkk3
Erk5 Pathway Regulates The Phosphorylation Of Tumour Suppressor Hdlg During Mitosis
Differential Activation Of P38mapk Isoforms By Mkk6 And Mkk3
Alternative Erk5 Regulation By Phosphorylation During The Cell Cycle
Differential Activation Of P38mapk Isoforms By Mkk6 And Mkk3
Alternative Erk5 Regulation By Phosphorylation During The Cell Cycle
P38γ Regulates Interaction Of Nuclear Psf And Rna With The Tumour-Suppressor Hdlg In Response To Osmotic Shock
Alternative Erk5 Regulation By Phosphorylation During The Cell Cycle
P38γ Regulates Interaction Of Nuclear Psf And Rna With The Tumour-Suppressor Hdlg In Response To Osmotic Shock
Proteolysis Of The Tumour Suppressor Hdlg In Response To Osmotic Stress Is Mediated By Caspases And Independent Of Phosphorylation
P38γ Regulates Interaction Of Nuclear Psf And Rna With The Tumour-Suppressor Hdlg In Response To Osmotic Shock
Proteolysis Of The Tumour Suppressor Hdlg In Response To Osmotic Stress Is Mediated By Caspases And Independent Of Phosphorylation
Alternative P38 Mapk Pathways
Proteolysis Of The Tumour Suppressor Hdlg In Response To Osmotic Stress Is Mediated By Caspases And Independent Of Phosphorylation
Alternative P38 Mapk Pathways
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Alternative P38 Mapk Pathways
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
3d Printing Of Carbonaceous Hydrogel Electrodes For Energy Applications
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