Portal Investigación UEx

Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite

3d Printing Of Carbonaceous Hydrogel Electrodes For Energy Applications

Study Of Printable And Biocompatible Alginate–carbon Hydrogels For Sensor Applications: Mechanical, Electrical, And Cytotoxicity Evaluation

3d Printing Of Carbonaceous Hydrogel Electrodes For Energy Applications

Study Of Printable And Biocompatible Alginate–carbon Hydrogels For Sensor Applications: Mechanical, Electrical, And Cytotoxicity Evaluation

The Potent Human Car Activator Citco Is A Non-Genotoxic Hepatic Tumour-Promoting Agent In Humanised Constitutive Androstane Receptor Mice But Not In Wild-Type Animals

Study Of Printable And Biocompatible Alginate–carbon Hydrogels For Sensor Applications: Mechanical, Electrical, And Cytotoxicity Evaluation

The Potent Human Car Activator Citco Is A Non-Genotoxic Hepatic Tumour-Promoting Agent In Humanised Constitutive Androstane Receptor Mice But Not In Wild-Type Animals

Integration Of In Vivo Stress Reporters With Transcription Profiling To Define Chemical Mixtures Mechanisms Of Toxicity, Including Trans-Generational Effects

The Potent Human Car Activator Citco Is A Non-Genotoxic Hepatic Tumour-Promoting Agent In Humanised Constitutive Androstane Receptor Mice But Not In Wild-Type Animals

Integration Of In Vivo Stress Reporters With Transcription Profiling To Define Chemical Mixtures Mechanisms Of Toxicity, Including Trans-Generational Effects

Ck2 Phosphorylation Of Cmtr1 Promotes Rna Cap Formation And Influenza Virus Infection

Integration Of In Vivo Stress Reporters With Transcription Profiling To Define Chemical Mixtures Mechanisms Of Toxicity, Including Trans-Generational Effects

Ck2 Phosphorylation Of Cmtr1 Promotes Rna Cap Formation And Influenza Virus Infection

Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als

Ck2 Phosphorylation Of Cmtr1 Promotes Rna Cap Formation And Influenza Virus Infection

Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als

Comunicación Del Riesgo Para La Salud Humana Causado Por La Contaminación Atmosférica

Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als

Comunicación Del Riesgo Para La Salud Humana Causado Por La Contaminación Atmosférica

Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers

Comunicación Del Riesgo Para La Salud Humana Causado Por La Contaminación Atmosférica

Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers

Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies

Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers

Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies

Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als.

Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies

Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als.

In Vivo Stress Reporters As Early Biomarkers Of The Cellular Changes Associated With Progeria

Novel In Vivo Tdp-43 Stress Reporter Models To Accelerate Drug Development In Als.

In Vivo Stress Reporters As Early Biomarkers Of The Cellular Changes Associated With Progeria

Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies [Version 1; Peer Review: 2 Approved]

In Vivo Stress Reporters As Early Biomarkers Of The Cellular Changes Associated With Progeria

Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies [Version 1; Peer Review: 2 Approved]

Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers

Potential Of In Vivo Stress Reporter Models To Reduce Animal Use And Provide Mechanistic Insights In Toxicity Studies [Version 1; Peer Review: 2 Approved]

Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers

Application Of The In Vivo Oxidative Stress Reporter Hmox1 As Mechanistic Biomarker Of Arsenic Toxicity

Defining The In Vivo Mechanism Of Air Pollutant Toxicity Using Murine Stress Response Biomarkers

Application Of The In Vivo Oxidative Stress Reporter Hmox1 As Mechanistic Biomarker Of Arsenic Toxicity

Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation

Application Of The In Vivo Oxidative Stress Reporter Hmox1 As Mechanistic Biomarker Of Arsenic Toxicity

Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation

Regulation And Function Of Cmtr1-Dependent Mrna Cap Methylation

Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation

Regulation And Function Of Cmtr1-Dependent Mrna Cap Methylation

Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation

Regulation And Function Of Cmtr1-Dependent Mrna Cap Methylation

Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation

Cdk1-Cyclin B1 Activates Rnmt, Coordinating Mrna Cap Methylation With G1 Phase Transcription

Dhx15 Regulates Cmtr1-Dependent Gene Expression And Cell Proliferation

Cdk1-Cyclin B1 Activates Rnmt, Coordinating Mrna Cap Methylation With G1 Phase Transcription

Human Ifit1 Inhibits Mrna Translation Of Rubulaviruses But Not Other Members Of The Paramyxoviridae Family

Cdk1-Cyclin B1 Activates Rnmt, Coordinating Mrna Cap Methylation With G1 Phase Transcription

Human Ifit1 Inhibits Mrna Translation Of Rubulaviruses But Not Other Members Of The Paramyxoviridae Family

G2019s Leucine-Rich Repeat Kinase 2 Causes Uncoupling Protein-Mediated Mitochondrial Depolarization

Human Ifit1 Inhibits Mrna Translation Of Rubulaviruses But Not Other Members Of The Paramyxoviridae Family

G2019s Leucine-Rich Repeat Kinase 2 Causes Uncoupling Protein-Mediated Mitochondrial Depolarization

The Ikappab Kinase Family Phosphorylates The Parkinson's Disease Kinase Lrrk2 At Ser935 And Ser910 During Toll-Like Receptor Signaling

G2019s Leucine-Rich Repeat Kinase 2 Causes Uncoupling Protein-Mediated Mitochondrial Depolarization

The Ikappab Kinase Family Phosphorylates The Parkinson's Disease Kinase Lrrk2 At Ser935 And Ser910 During Toll-Like Receptor Signaling

Discovery Of Potent And Selective Covalent Inhibitors Of Jnk

The Ikappab Kinase Family Phosphorylates The Parkinson's Disease Kinase Lrrk2 At Ser935 And Ser910 During Toll-Like Receptor Signaling

Discovery Of Potent And Selective Covalent Inhibitors Of Jnk

Erk5 Pathway Regulates The Phosphorylation Of Tumour Suppressor Hdlg During Mitosis

Discovery Of Potent And Selective Covalent Inhibitors Of Jnk

Erk5 Pathway Regulates The Phosphorylation Of Tumour Suppressor Hdlg During Mitosis

Differential Activation Of P38mapk Isoforms By Mkk6 And Mkk3

Erk5 Pathway Regulates The Phosphorylation Of Tumour Suppressor Hdlg During Mitosis

Differential Activation Of P38mapk Isoforms By Mkk6 And Mkk3

Alternative Erk5 Regulation By Phosphorylation During The Cell Cycle

Differential Activation Of P38mapk Isoforms By Mkk6 And Mkk3

Alternative Erk5 Regulation By Phosphorylation During The Cell Cycle

P38γ Regulates Interaction Of Nuclear Psf And Rna With The Tumour-Suppressor Hdlg In Response To Osmotic Shock

Alternative Erk5 Regulation By Phosphorylation During The Cell Cycle

P38γ Regulates Interaction Of Nuclear Psf And Rna With The Tumour-Suppressor Hdlg In Response To Osmotic Shock

Proteolysis Of The Tumour Suppressor Hdlg In Response To Osmotic Stress Is Mediated By Caspases And Independent Of Phosphorylation

P38γ Regulates Interaction Of Nuclear Psf And Rna With The Tumour-Suppressor Hdlg In Response To Osmotic Shock

Proteolysis Of The Tumour Suppressor Hdlg In Response To Osmotic Stress Is Mediated By Caspases And Independent Of Phosphorylation

Alternative P38 Mapk Pathways

Proteolysis Of The Tumour Suppressor Hdlg In Response To Osmotic Stress Is Mediated By Caspases And Independent Of Phosphorylation

Alternative P38 Mapk Pathways

Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite

Alternative P38 Mapk Pathways

Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite

3d Printing Of Carbonaceous Hydrogel Electrodes For Energy Applications