Portal Investigación UEx

Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle

Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment

Competitiveness Versus Cooperativeness Linked To Leisure Activities In Young University Students In Spain

Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment

Competitiveness Versus Cooperativeness Linked To Leisure Activities In Young University Students In Spain

Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment.

Competitiveness Versus Cooperativeness Linked To Leisure Activities In Young University Students In Spain

Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment.

From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms

Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment.

From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms

Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor

From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms

Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor

Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression

Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor

Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression

Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells

Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression

Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells

The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development

Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells

The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development

Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer

The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development

Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer

Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function

Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer

Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function

Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor

Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function

Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor

Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling

Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor

Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling

The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value

Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling

The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value

Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor

The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value

Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor

Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis

Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor

Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis

The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value.

Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis

The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value.

Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice

The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value.

Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice

Ahr-Dependent 2,3,7,8-Tetrachlorodibenzo-P-Dioxin Toxicity In Human Neuronal Cell Line Shsy5y

Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice

Ahr-Dependent 2,3,7,8-Tetrachlorodibenzo-P-Dioxin Toxicity In Human Neuronal Cell Line Shsy5y

Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor

Ahr-Dependent 2,3,7,8-Tetrachlorodibenzo-P-Dioxin Toxicity In Human Neuronal Cell Line Shsy5y

Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor

Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis

Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor

Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis

2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis By Disruption Of Intracellular Calcium Homeostasis In Human Neuronal Cell Line Shsy5y

Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis

2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis By Disruption Of Intracellular Calcium Homeostasis In Human Neuronal Cell Line Shsy5y

The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling

2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis By Disruption Of Intracellular Calcium Homeostasis In Human Neuronal Cell Line Shsy5y

The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling

Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse

The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling

Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse

2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells

Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse

2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells

Nerve Growth Factor Increases The Sensitivity To Zinc Toxicity And Induces Cell Cycle Arrest In Pc12 Cells

2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells

Nerve Growth Factor Increases The Sensitivity To Zinc Toxicity And Induces Cell Cycle Arrest In Pc12 Cells

The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion

Nerve Growth Factor Increases The Sensitivity To Zinc Toxicity And Induces Cell Cycle Arrest In Pc12 Cells

The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion

Nmda-Induced Neuroprotection In Hippocampal Neurons Is Mediated Through The Protein Kinase A And Creb (Camp-Response Element-Binding Protein) Pathway

The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion

Nmda-Induced Neuroprotection In Hippocampal Neurons Is Mediated Through The Protein Kinase A And Creb (Camp-Response Element-Binding Protein) Pathway

Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))

Nmda-Induced Neuroprotection In Hippocampal Neurons Is Mediated Through The Protein Kinase A And Creb (Camp-Response Element-Binding Protein) Pathway

Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))

Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb

Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))

Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb

Small Peptides Patterned After The N-Terminus Domain Of Snap25 Inhibit Snare Complex Assembly And Regulated Exocytosis

Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb

Small Peptides Patterned After The N-Terminus Domain Of Snap25 Inhibit Snare Complex Assembly And Regulated Exocytosis

Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation

Small Peptides Patterned After The N-Terminus Domain Of Snap25 Inhibit Snare Complex Assembly And Regulated Exocytosis

Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation

Identification Of Snare Complex Modulators That Inhibit Exocytosis From An Α-Helix-Constrained Combinatorial Library

Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation

Identification Of Snare Complex Modulators That Inhibit Exocytosis From An Α-Helix-Constrained Combinatorial Library

Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons

Identification Of Snare Complex Modulators That Inhibit Exocytosis From An Α-Helix-Constrained Combinatorial Library

Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons

A Novel N-Methyl-D-Aspartate Receptor Open Channel Blocker With In Vivo Neuroprotectant Activity

Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons

A Novel N-Methyl-D-Aspartate Receptor Open Channel Blocker With In Vivo Neuroprotectant Activity

The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle

A Novel N-Methyl-D-Aspartate Receptor Open Channel Blocker With In Vivo Neuroprotectant Activity

The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle

Identification Of An Aspartic Residue In The P-Loop Of The Vanilloid Receptor That Modulates Pore Properties

The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle

Identification Of An Aspartic Residue In The P-Loop Of The Vanilloid Receptor That Modulates Pore Properties

Arginine-Rich Peptides Are Blockers Of Vr-1 Channels With Analgesic Activity

Identification Of An Aspartic Residue In The P-Loop Of The Vanilloid Receptor That Modulates Pore Properties

Arginine-Rich Peptides Are Blockers Of Vr-1 Channels With Analgesic Activity

Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes

Arginine-Rich Peptides Are Blockers Of Vr-1 Channels With Analgesic Activity

Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes

Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes

Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes

Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes

Selected Peptides Targeted To The Nmda Receptor Channel Protect Neurons From Excitotoxic Death

Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes

Selected Peptides Targeted To The Nmda Receptor Channel Protect Neurons From Excitotoxic Death

Structural Determinants Of The Blocker Binding Site In Glutamate And Nmda Receptor Channels

Selected Peptides Targeted To The Nmda Receptor Channel Protect Neurons From Excitotoxic Death

Structural Determinants Of The Blocker Binding Site In Glutamate And Nmda Receptor Channels

Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)

Structural Determinants Of The Blocker Binding Site In Glutamate And Nmda Receptor Channels

Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)

Estudio Sobre Relaciones Estructura-Función En La Ca2+,mg2+-Atpasa Del Retículo Sarcoplásmico Del Músculo Esquelético

Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)

Estudio Sobre Relaciones Estructura-Función En La Ca2+,mg2+-Atpasa Del Retículo Sarcoplásmico Del Músculo Esquelético

Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide

Estudio Sobre Relaciones Estructura-Función En La Ca2+,mg2+-Atpasa Del Retículo Sarcoplásmico Del Músculo Esquelético

Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide

Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle

Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide

Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle

Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins

Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle

Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins

Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle

Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins

Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle

Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment