PUBLICACIÓN

The content of glycogen phosphorylase and glycogen in preparations of sarcoplasmic reticulum - Glycogenolytic complex is enhanced in diabetic rat skeletal muscle

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ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Garduño, E., Nogues, M., Merino, J.M., Gutiérrez-Merino, C., Henao, F.

2001 Diabetologia

CITAS

11

DOI

10.1007/s001250100637

EID

2-s2.0-0034775987

ISSN

0012-186X

BIBTEX

@article{Merino2001,title = {The content of glycogen phosphorylase and glycogen in preparations of sarcoplasmic reticulum - Glycogenolytic complex is enhanced in diabetic rat skeletal muscle},journal = {Diabetologia},year = {2001},volume = {44},number = {10},pages = {1238-1246},author = {Gardu{\~n}o, E. and Nogues, M. and Merino, J.M. and Guti{\'e}rrez-Merino, C. and Henao, F.}}

AUTORES DE LA UEX

Jaime María Merino Fernández

OTRAS PUBLICACIONES DE INTERÉS

Regulation Of Phosphorylase B By Amp: The Effects Of Enzyme Concentration
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment
An Update Of Kaempferol Protection Against Brain Damage Induced By Ischemia-Reperfusion And By 3-Nitropropionic Acid
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment
An Update Of Kaempferol Protection Against Brain Damage Induced By Ischemia-Reperfusion And By 3-Nitropropionic Acid
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment.
An Update Of Kaempferol Protection Against Brain Damage Induced By Ischemia-Reperfusion And By 3-Nitropropionic Acid
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment.
Store-Operated Calcium Entry Inhibition And Plasma Membrane Calcium Pump Upregulation Contribute To The Maintenance Of Resting Cytosolic Calcium Concentration In A1-Like Astrocytes
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment.
Store-Operated Calcium Entry Inhibition And Plasma Membrane Calcium Pump Upregulation Contribute To The Maintenance Of Resting Cytosolic Calcium Concentration In A1-Like Astrocytes
Brain Hydrophobic Peptides Antagonists Of Neurotoxic Amyloid Β Peptide Monomers/oligomers–protein Interactions
Store-Operated Calcium Entry Inhibition And Plasma Membrane Calcium Pump Upregulation Contribute To The Maintenance Of Resting Cytosolic Calcium Concentration In A1-Like Astrocytes
Brain Hydrophobic Peptides Antagonists Of Neurotoxic Amyloid Β Peptide Monomers/oligomers–protein Interactions
Hexa-Histidine, A Peptide With Versatile Applications In The Study Of Amyloid-Β(1–42) Molecular Mechanisms Of Action
Brain Hydrophobic Peptides Antagonists Of Neurotoxic Amyloid Β Peptide Monomers/oligomers–protein Interactions
Hexa-Histidine, A Peptide With Versatile Applications In The Study Of Amyloid-Β(1–42) Molecular Mechanisms Of Action
Are There Lipid Membrane-Domain Subtypes In Neurons With Different Roles In Calcium Signaling?
Hexa-Histidine, A Peptide With Versatile Applications In The Study Of Amyloid-Β(1–42) Molecular Mechanisms Of Action
Are There Lipid Membrane-Domain Subtypes In Neurons With Different Roles In Calcium Signaling?
Biochemical And Biophysical Characterization Of The Caveolin-2 Interaction With Membranes And Analysis Of The Protein Structural Alteration By The Presence Of Cholesterol
Are There Lipid Membrane-Domain Subtypes In Neurons With Different Roles In Calcium Signaling?
Biochemical And Biophysical Characterization Of The Caveolin-2 Interaction With Membranes And Analysis Of The Protein Structural Alteration By The Presence Of Cholesterol
Structural Features Of Cytochrome B5 –cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Biochemical And Biophysical Characterization Of The Caveolin-2 Interaction With Membranes And Analysis Of The Protein Structural Alteration By The Presence Of Cholesterol
Structural Features Of Cytochrome B5 –cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Inhibition Of Serca And Pmca Ca2+-Atpase Activities By Polyoxotungstates
Structural Features Of Cytochrome B5 –cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Inhibition Of Serca And Pmca Ca2+-Atpase Activities By Polyoxotungstates
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Inhibition Of Serca And Pmca Ca2+-Atpase Activities By Polyoxotungstates
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Special Issue “molecular And Cellular Mechanisms Of Action Of Markers Of Tissue Degeneration”
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Special Issue “molecular And Cellular Mechanisms Of Action Of Markers Of Tissue Degeneration”
Design And Experimental Evaluation Of A Peptide Antagonist Against Amyloid Β(1–42) Interactions With Calmodulin And Calbindin-D28k
Special Issue “molecular And Cellular Mechanisms Of Action Of Markers Of Tissue Degeneration”
Design And Experimental Evaluation Of A Peptide Antagonist Against Amyloid Β(1–42) Interactions With Calmodulin And Calbindin-D28k
Kaempferol Prevents The Activation Of Complement C3 Protein And The Generation Of Reactive A1 Astrocytes That Mediate Rat Brain Degeneration Induced By 3-Nitropropionic Acid
Design And Experimental Evaluation Of A Peptide Antagonist Against Amyloid Β(1–42) Interactions With Calmodulin And Calbindin-D28k
Kaempferol Prevents The Activation Of Complement C3 Protein And The Generation Of Reactive A1 Astrocytes That Mediate Rat Brain Degeneration Induced By 3-Nitropropionic Acid
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Kaempferol Prevents The Activation Of Complement C3 Protein And The Generation Of Reactive A1 Astrocytes That Mediate Rat Brain Degeneration Induced By 3-Nitropropionic Acid
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Gold Compounds Inhibit The Ca2+-Atpase Activity Of Brain Pmca And Human Neuroblastoma Sh-Sy5y Cells And Decrease Cell Viability
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Gold Compounds Inhibit The Ca2+-Atpase Activity Of Brain Pmca And Human Neuroblastoma Sh-Sy5y Cells And Decrease Cell Viability
The Relevance Of Amyloid Β-Calmodulin Complexation In Neurons And Brain Degeneration In Alzheimer’s Disease
Gold Compounds Inhibit The Ca2+-Atpase Activity Of Brain Pmca And Human Neuroblastoma Sh-Sy5y Cells And Decrease Cell Viability
The Relevance Of Amyloid Β-Calmodulin Complexation In Neurons And Brain Degeneration In Alzheimer’s Disease
Binding Of Amyloid Β(1–42)‐calmodulin Complexes To Plasma Membrane Lipid Rafts In Cerebellar Granule Neurons Alters Resting Cytosolic Calcium Homeostasis
The Relevance Of Amyloid Β-Calmodulin Complexation In Neurons And Brain Degeneration In Alzheimer’s Disease
Binding Of Amyloid Β(1–42)‐calmodulin Complexes To Plasma Membrane Lipid Rafts In Cerebellar Granule Neurons Alters Resting Cytosolic Calcium Homeostasis
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Binding Of Amyloid Β(1–42)‐calmodulin Complexes To Plasma Membrane Lipid Rafts In Cerebellar Granule Neurons Alters Resting Cytosolic Calcium Homeostasis
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function
Structural Features Of Cytochrome B5–cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function
Structural Features Of Cytochrome B5–cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Human Erythrocytes Exposure To Juglone Leads To An Increase Of Superoxide Anion Production Associated With Cytochrome B5 Reductase Uncoupling
Structural Features Of Cytochrome B5–cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Human Erythrocytes Exposure To Juglone Leads To An Increase Of Superoxide Anion Production Associated With Cytochrome B5 Reductase Uncoupling
Early Reactive A1 Astrocytes Induction By The Neurotoxin 3-Nitropropionic Acid In Rat Brain
Human Erythrocytes Exposure To Juglone Leads To An Increase Of Superoxide Anion Production Associated With Cytochrome B5 Reductase Uncoupling
Early Reactive A1 Astrocytes Induction By The Neurotoxin 3-Nitropropionic Acid In Rat Brain
Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor
Early Reactive A1 Astrocytes Induction By The Neurotoxin 3-Nitropropionic Acid In Rat Brain
Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor
Modulation Of Cyp2c9 Activity And Hydrogen Peroxide Production By Cytochrome B 5
Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor
Modulation Of Cyp2c9 Activity And Hydrogen Peroxide Production By Cytochrome B 5
Ligand Accessibility To Heme Cytochrome B 5 Coordinating Sphere And Enzymatic Activity Enhancement Upon Tyrosine Ionization
Modulation Of Cyp2c9 Activity And Hydrogen Peroxide Production By Cytochrome B 5
Ligand Accessibility To Heme Cytochrome B 5 Coordinating Sphere And Enzymatic Activity Enhancement Upon Tyrosine Ionization
Methylene Blue Blocks And Reverses The Inhibitory Effect Of Tau On Pmca Function
Ligand Accessibility To Heme Cytochrome B 5 Coordinating Sphere And Enzymatic Activity Enhancement Upon Tyrosine Ionization
Methylene Blue Blocks And Reverses The Inhibitory Effect Of Tau On Pmca Function
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Methylene Blue Blocks And Reverses The Inhibitory Effect Of Tau On Pmca Function
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Methyl-Β-Cyclodextrin Impairs The Phosphorylation Of The Β2 Subunit Of L-Type Calcium Channels And Cytosolic Calcium Homeostasis In Mature Cerebellar Granule Neurons
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Methyl-Β-Cyclodextrin Impairs The Phosphorylation Of The Β2 Subunit Of L-Type Calcium Channels And Cytosolic Calcium Homeostasis In Mature Cerebellar Granule Neurons
Creatine Protects Against Cytosolic Calcium Dysregulation, Mitochondrial Depolarization And Increase Of Reactive Oxygen Species Production In Rotenone-Induced Cell Death Of Cerebellar Granule Neurons
Methyl-Β-Cyclodextrin Impairs The Phosphorylation Of The Β2 Subunit Of L-Type Calcium Channels And Cytosolic Calcium Homeostasis In Mature Cerebellar Granule Neurons
Creatine Protects Against Cytosolic Calcium Dysregulation, Mitochondrial Depolarization And Increase Of Reactive Oxygen Species Production In Rotenone-Induced Cell Death Of Cerebellar Granule Neurons
Cytochrome B5 Reductase Is The Component From Neuronal Synaptic Plasma Membrane Vesicles That Generates Superoxide Anion Upon Stimulation By Cytochrome C
Creatine Protects Against Cytosolic Calcium Dysregulation, Mitochondrial Depolarization And Increase Of Reactive Oxygen Species Production In Rotenone-Induced Cell Death Of Cerebellar Granule Neurons
Cytochrome B5 Reductase Is The Component From Neuronal Synaptic Plasma Membrane Vesicles That Generates Superoxide Anion Upon Stimulation By Cytochrome C
Topography Of Human Cytochrome B5/cytochrome B5 Reductase Interacting Domain And Redox Alterations Upon Complex Formation
Cytochrome B5 Reductase Is The Component From Neuronal Synaptic Plasma Membrane Vesicles That Generates Superoxide Anion Upon Stimulation By Cytochrome C
Topography Of Human Cytochrome B5/cytochrome B5 Reductase Interacting Domain And Redox Alterations Upon Complex Formation
Peroxidase-Like Activity Of Cytochrome B5 Is Triggered Upon Hemichrome Formation In Alkaline Ph
Topography Of Human Cytochrome B5/cytochrome B5 Reductase Interacting Domain And Redox Alterations Upon Complex Formation
Peroxidase-Like Activity Of Cytochrome B5 Is Triggered Upon Hemichrome Formation In Alkaline Ph
Methylene Blue Activates The Pmca Activity And Cross-Interacts With Amyloid Β-Peptide, Blocking Aβ-Mediated Pmca Inhibition
Peroxidase-Like Activity Of Cytochrome B5 Is Triggered Upon Hemichrome Formation In Alkaline Ph
Methylene Blue Activates The Pmca Activity And Cross-Interacts With Amyloid Β-Peptide, Blocking Aβ-Mediated Pmca Inhibition
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Methylene Blue Activates The Pmca Activity And Cross-Interacts With Amyloid Β-Peptide, Blocking Aβ-Mediated Pmca Inhibition
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value.
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value.
Phospholipids And Calmodulin Modulate The Inhibition Of Pmca Activity By Tau
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value.
Phospholipids And Calmodulin Modulate The Inhibition Of Pmca Activity By Tau
High Affinity Binding Of Amyloid Β-Peptide To Calmodulin: Structural And Functional Implications
Phospholipids And Calmodulin Modulate The Inhibition Of Pmca Activity By Tau
High Affinity Binding Of Amyloid Β-Peptide To Calmodulin: Structural And Functional Implications
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
High Affinity Binding Of Amyloid Β-Peptide To Calmodulin: Structural And Functional Implications
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis
Correlation Between The Potency Of Flavonoids For Cytochrome C Reduction And Inhibition Of Cardiolipin-Induced Peroxidase Activity
Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis
Correlation Between The Potency Of Flavonoids For Cytochrome C Reduction And Inhibition Of Cardiolipin-Induced Peroxidase Activity
High Expression Of Cytochrome B 5 Reductase Isoform 3/cytochrome B 5 System In The Cerebellum And Pyramidal Neurons Of Adult Rat Brain
Correlation Between The Potency Of Flavonoids For Cytochrome C Reduction And Inhibition Of Cardiolipin-Induced Peroxidase Activity
High Expression Of Cytochrome B 5 Reductase Isoform 3/cytochrome B 5 System In The Cerebellum And Pyramidal Neurons Of Adult Rat Brain
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
High Expression Of Cytochrome B 5 Reductase Isoform 3/cytochrome B 5 System In The Cerebellum And Pyramidal Neurons Of Adult Rat Brain
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
Ahr-Dependent 2,3,7,8-Tetrachlorodibenzo-P-Dioxin Toxicity In Human Neuronal Cell Line Shsy5y
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
Ahr-Dependent 2,3,7,8-Tetrachlorodibenzo-P-Dioxin Toxicity In Human Neuronal Cell Line Shsy5y
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Ahr-Dependent 2,3,7,8-Tetrachlorodibenzo-P-Dioxin Toxicity In Human Neuronal Cell Line Shsy5y
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
Cytochrome B5 As A Pleiotropic Metabolic Modulator In Mammalian Cells
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
Cytochrome B5 As A Pleiotropic Metabolic Modulator In Mammalian Cells
The Decrease Of Nad(P)h:quinone Oxidoreductase 1 Activity And Increase Of Ros Production By Nadph Oxidases Are Early Biomarkers In Doxorubicin Cardiotoxicity
Cytochrome B5 As A Pleiotropic Metabolic Modulator In Mammalian Cells
The Decrease Of Nad(P)h:quinone Oxidoreductase 1 Activity And Increase Of Ros Production By Nadph Oxidases Are Early Biomarkers In Doxorubicin Cardiotoxicity
Thymbra Capitata Essential Oil Prevents Cell Death Induced By 4-Hydroxy-2-Nonenal In Neonatal Rat Cardiac Myocytes
The Decrease Of Nad(P)h:quinone Oxidoreductase 1 Activity And Increase Of Ros Production By Nadph Oxidases Are Early Biomarkers In Doxorubicin Cardiotoxicity
Thymbra Capitata Essential Oil Prevents Cell Death Induced By 4-Hydroxy-2-Nonenal In Neonatal Rat Cardiac Myocytes
Apoptotic Cell Death In Cultured Cardiomyocytes Following Exposure To Low Concentrations Of 4-Hydroxy-2-Nonenal
Thymbra Capitata Essential Oil Prevents Cell Death Induced By 4-Hydroxy-2-Nonenal In Neonatal Rat Cardiac Myocytes
Apoptotic Cell Death In Cultured Cardiomyocytes Following Exposure To Low Concentrations Of 4-Hydroxy-2-Nonenal
Caveolin-Rich Lipid Rafts Of The Plasma Membrane Of Mature Cerebellar Granule Neurons Are Microcompartments For Calcium/reactive Oxygen And Nitrogen Species Cross-Talk Signaling
Apoptotic Cell Death In Cultured Cardiomyocytes Following Exposure To Low Concentrations Of 4-Hydroxy-2-Nonenal
Caveolin-Rich Lipid Rafts Of The Plasma Membrane Of Mature Cerebellar Granule Neurons Are Microcompartments For Calcium/reactive Oxygen And Nitrogen Species Cross-Talk Signaling
Purified Nadh-Cytochrome B5 Reductase Is A Novel Superoxide Anion Source Inhibited By Apocynin: Sensitivity To Nitric Oxide And Peroxynitrite
Caveolin-Rich Lipid Rafts Of The Plasma Membrane Of Mature Cerebellar Granule Neurons Are Microcompartments For Calcium/reactive Oxygen And Nitrogen Species Cross-Talk Signaling
Purified Nadh-Cytochrome B5 Reductase Is A Novel Superoxide Anion Source Inhibited By Apocynin: Sensitivity To Nitric Oxide And Peroxynitrite
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis By Disruption Of Intracellular Calcium Homeostasis In Human Neuronal Cell Line Shsy5y
Purified Nadh-Cytochrome B5 Reductase Is A Novel Superoxide Anion Source Inhibited By Apocynin: Sensitivity To Nitric Oxide And Peroxynitrite
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis By Disruption Of Intracellular Calcium Homeostasis In Human Neuronal Cell Line Shsy5y
Stimulation And Clustering Of Cytochrome B 5 Reductase In Caveolin-Rich Lipid Microdomains Is An Early Event In Oxidative Stress-Mediated Apoptosis Of Cerebellar Granule Neurons
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis By Disruption Of Intracellular Calcium Homeostasis In Human Neuronal Cell Line Shsy5y
Stimulation And Clustering Of Cytochrome B 5 Reductase In Caveolin-Rich Lipid Microdomains Is An Early Event In Oxidative Stress-Mediated Apoptosis Of Cerebellar Granule Neurons
L-Type Voltage-Operated Calcium Channels, N-Methyl-D-Aspartate Receptors And Neuronal Nitric-Oxide Synthase Form A Calcium/redox Nano-Transducer Within Lipid Rafts
Stimulation And Clustering Of Cytochrome B 5 Reductase In Caveolin-Rich Lipid Microdomains Is An Early Event In Oxidative Stress-Mediated Apoptosis Of Cerebellar Granule Neurons
L-Type Voltage-Operated Calcium Channels, N-Methyl-D-Aspartate Receptors And Neuronal Nitric-Oxide Synthase Form A Calcium/redox Nano-Transducer Within Lipid Rafts
Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
L-Type Voltage-Operated Calcium Channels, N-Methyl-D-Aspartate Receptors And Neuronal Nitric-Oxide Synthase Form A Calcium/redox Nano-Transducer Within Lipid Rafts
Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
Complex I And Cytochrome C Are Molecular Targets Of Flavonoids That Inhibit Hydrogen Peroxide Production By Mitochondria
Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
Complex I And Cytochrome C Are Molecular Targets Of Flavonoids That Inhibit Hydrogen Peroxide Production By Mitochondria
Neuroprotective Actions Of Flavonoids
Complex I And Cytochrome C Are Molecular Targets Of Flavonoids That Inhibit Hydrogen Peroxide Production By Mitochondria
Neuroprotective Actions Of Flavonoids
The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling
Neuroprotective Actions Of Flavonoids
The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling
Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse
The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling
Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse
Erratum: Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane (Free Radical Biology & Medicine (2011) 50 (323-336))
Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse
Erratum: Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane (Free Radical Biology & Medicine (2011) 50 (323-336))
Inhibition By 4-Hydroxynonenal (Hne) Of Ca 2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
Erratum: Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane (Free Radical Biology & Medicine (2011) 50 (323-336))
Inhibition By 4-Hydroxynonenal (Hne) Of Ca 2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
Early Disruption Of The Actin Cytoskeleton In Cultured Cerebellar Granule Neurons Exposed To 3-Morpholinosydnonimine-Oxidative Stress Is Linked To Alterations Of The Cytosolic Calcium Concentration
Inhibition By 4-Hydroxynonenal (Hne) Of Ca 2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
Early Disruption Of The Actin Cytoskeleton In Cultured Cerebellar Granule Neurons Exposed To 3-Morpholinosydnonimine-Oxidative Stress Is Linked To Alterations Of The Cytosolic Calcium Concentration
Reactivity Of Hydrogen Sulfide With Peroxynitrite And Other Oxidants Of Biological Interest
Early Disruption Of The Actin Cytoskeleton In Cultured Cerebellar Granule Neurons Exposed To 3-Morpholinosydnonimine-Oxidative Stress Is Linked To Alterations Of The Cytosolic Calcium Concentration
Reactivity Of Hydrogen Sulfide With Peroxynitrite And Other Oxidants Of Biological Interest
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells
Reactivity Of Hydrogen Sulfide With Peroxynitrite And Other Oxidants Of Biological Interest
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells
Nerve Growth Factor Increases The Sensitivity To Zinc Toxicity And Induces Cell Cycle Arrest In Pc12 Cells
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells
Nerve Growth Factor Increases The Sensitivity To Zinc Toxicity And Induces Cell Cycle Arrest In Pc12 Cells
Peroxynitrite-Mediated Oxidative Modifications Of Myosin And Implications On Structure And Function
Nerve Growth Factor Increases The Sensitivity To Zinc Toxicity And Induces Cell Cycle Arrest In Pc12 Cells
Peroxynitrite-Mediated Oxidative Modifications Of Myosin And Implications On Structure And Function
L-Type Calcium Channels And Cytochrome B5 Reductase Are Components Of Protein Complexes Tightly Associated With Lipid Rafts Microdomains Of The Neuronal Plasma Membrane
Peroxynitrite-Mediated Oxidative Modifications Of Myosin And Implications On Structure And Function
L-Type Calcium Channels And Cytochrome B5 Reductase Are Components Of Protein Complexes Tightly Associated With Lipid Rafts Microdomains Of The Neuronal Plasma Membrane
Kaempferol Protects Against Rat Striatal Degeneration Induced By 3-Nitropropionic Acid
L-Type Calcium Channels And Cytochrome B5 Reductase Are Components Of Protein Complexes Tightly Associated With Lipid Rafts Microdomains Of The Neuronal Plasma Membrane
Kaempferol Protects Against Rat Striatal Degeneration Induced By 3-Nitropropionic Acid
Role Of Transforming Growth Factor Β In Cancer Microenvironment
Kaempferol Protects Against Rat Striatal Degeneration Induced By 3-Nitropropionic Acid
Role Of Transforming Growth Factor Β In Cancer Microenvironment
The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion
Role Of Transforming Growth Factor Β In Cancer Microenvironment
The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Hydrogen Sulfide Is A Reversible Inhibitor Of The Nadh Oxidase Activity Of Synaptic Plasma Membranes
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Hydrogen Sulfide Is A Reversible Inhibitor Of The Nadh Oxidase Activity Of Synaptic Plasma Membranes
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Hydrogen Sulfide Is A Reversible Inhibitor Of The Nadh Oxidase Activity Of Synaptic Plasma Membranes
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Nmda-Induced Neuroprotection In Hippocampal Neurons Is Mediated Through The Protein Kinase A And Creb (Camp-Response Element-Binding Protein) Pathway
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Nmda-Induced Neuroprotection In Hippocampal Neurons Is Mediated Through The Protein Kinase A And Creb (Camp-Response Element-Binding Protein) Pathway
Sarcoplasmic Reticulum Calcium Atpase Is Inhibited By Organic Vanadium Coordination Compounds: Pyridine-2,6-Dicarboxylatodioxovanadium(V), Bmov, And An Amavadine Analogue
Nmda-Induced Neuroprotection In Hippocampal Neurons Is Mediated Through The Protein Kinase A And Creb (Camp-Response Element-Binding Protein) Pathway
Sarcoplasmic Reticulum Calcium Atpase Is Inhibited By Organic Vanadium Coordination Compounds: Pyridine-2,6-Dicarboxylatodioxovanadium(V), Bmov, And An Amavadine Analogue
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Sarcoplasmic Reticulum Calcium Atpase Is Inhibited By Organic Vanadium Coordination Compounds: Pyridine-2,6-Dicarboxylatodioxovanadium(V), Bmov, And An Amavadine Analogue
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Vanadium And Cadmium In Vivo Effects In Teleost Cardiac Muscle: Metal Accumulation And Oxidative Stress Markers
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Vanadium And Cadmium In Vivo Effects In Teleost Cardiac Muscle: Metal Accumulation And Oxidative Stress Markers
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Vanadium And Cadmium In Vivo Effects In Teleost Cardiac Muscle: Metal Accumulation And Oxidative Stress Markers
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Biological Effects Of Decavanadate: Muscle Contraction, In Vivo Oxidative Stress, And Mitochondrial Toxicity
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Biological Effects Of Decavanadate: Muscle Contraction, In Vivo Oxidative Stress, And Mitochondrial Toxicity
Vanadium Distribution, Lipid Peroxidation And Oxidative Stress Markers Upon Decavanadate In Vivo Administration
Biological Effects Of Decavanadate: Muscle Contraction, In Vivo Oxidative Stress, And Mitochondrial Toxicity
Vanadium Distribution, Lipid Peroxidation And Oxidative Stress Markers Upon Decavanadate In Vivo Administration
Mitochondria As A Target For Decavanadate Toxicity In Sparus Aurata Heart
Vanadium Distribution, Lipid Peroxidation And Oxidative Stress Markers Upon Decavanadate In Vivo Administration
Mitochondria As A Target For Decavanadate Toxicity In Sparus Aurata Heart
Decavanadate Induces Mitochondrial Membrane Depolarization And Inhibits Oxygen Consumption
Mitochondria As A Target For Decavanadate Toxicity In Sparus Aurata Heart
Decavanadate Induces Mitochondrial Membrane Depolarization And Inhibits Oxygen Consumption
Binding Modes Of Decavanadate To Myosin And Inhibition Of The Actomyosin Atpase Activity
Decavanadate Induces Mitochondrial Membrane Depolarization And Inhibits Oxygen Consumption
Binding Modes Of Decavanadate To Myosin And Inhibition Of The Actomyosin Atpase Activity
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Binding Modes Of Decavanadate To Myosin And Inhibition Of The Actomyosin Atpase Activity
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Decavanadate Interactions With Actin: Inhibition Of G-Actin Polymerization And Stabilization Of Decameric Vanadate
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Decavanadate Interactions With Actin: Inhibition Of G-Actin Polymerization And Stabilization Of Decameric Vanadate
Peroxynitrite Induces F-Actin Depolymerization And Blockade Of Myosin Atpase Stimulation
Decavanadate Interactions With Actin: Inhibition Of G-Actin Polymerization And Stabilization Of Decameric Vanadate
Peroxynitrite Induces F-Actin Depolymerization And Blockade Of Myosin Atpase Stimulation
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Peroxynitrite Induces F-Actin Depolymerization And Blockade Of Myosin Atpase Stimulation
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Store-Operated Calcium Entry In Differentiated C2c12 Skeletal Muscle Cells
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Store-Operated Calcium Entry In Differentiated C2c12 Skeletal Muscle Cells
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Store-Operated Calcium Entry In Differentiated C2c12 Skeletal Muscle Cells
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Vanadate Oligomers: In Vivo Effects In Hepatic Vanadium Accumulation And Stress Markers
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Vanadate Oligomers: In Vivo Effects In Hepatic Vanadium Accumulation And Stress Markers
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Vanadate Oligomers: In Vivo Effects In Hepatic Vanadium Accumulation And Stress Markers
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Small Peptides Patterned After The N-Terminus Domain Of Snap25 Inhibit Snare Complex Assembly And Regulated Exocytosis
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Small Peptides Patterned After The N-Terminus Domain Of Snap25 Inhibit Snare Complex Assembly And Regulated Exocytosis
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Small Peptides Patterned After The N-Terminus Domain Of Snap25 Inhibit Snare Complex Assembly And Regulated Exocytosis
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Decavanadate Binding To A High Affinity Site Near The Myosin Catalytic Centre Inhibits F-Actin-Stimulated Myosin Atpase Activity
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Decavanadate Binding To A High Affinity Site Near The Myosin Catalytic Centre Inhibits F-Actin-Stimulated Myosin Atpase Activity
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Decavanadate Binding To A High Affinity Site Near The Myosin Catalytic Centre Inhibits F-Actin-Stimulated Myosin Atpase Activity
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Identification Of Snare Complex Modulators That Inhibit Exocytosis From An Α-Helix-Constrained Combinatorial Library
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Identification Of Snare Complex Modulators That Inhibit Exocytosis From An Α-Helix-Constrained Combinatorial Library
Projection Map Of Covalently Phosphorylated Ca-Atpase From Tubular Crystals
Identification Of Snare Complex Modulators That Inhibit Exocytosis From An Α-Helix-Constrained Combinatorial Library
Projection Map Of Covalently Phosphorylated Ca-Atpase From Tubular Crystals
The Trapping Of The Oh Radical By Coenzyme Q. A Theoretical And Experimental Study
Projection Map Of Covalently Phosphorylated Ca-Atpase From Tubular Crystals
The Trapping Of The Oh Radical By Coenzyme Q. A Theoretical And Experimental Study
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
The Trapping Of The Oh Radical By Coenzyme Q. A Theoretical And Experimental Study
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
A Novel N-Methyl-D-Aspartate Receptor Open Channel Blocker With In Vivo Neuroprotectant Activity
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
A Novel N-Methyl-D-Aspartate Receptor Open Channel Blocker With In Vivo Neuroprotectant Activity
Quenching Of Myosin Intrinsic Fluorescence Unravels The Existence Of A High Affinity Binding Site For Decavanadate
A Novel N-Methyl-D-Aspartate Receptor Open Channel Blocker With In Vivo Neuroprotectant Activity
Quenching Of Myosin Intrinsic Fluorescence Unravels The Existence Of A High Affinity Binding Site For Decavanadate
Sphingomyelin Composition And Physical Asymmetries In Native Acetylcholine Receptor-Rich Membranes
Quenching Of Myosin Intrinsic Fluorescence Unravels The Existence Of A High Affinity Binding Site For Decavanadate
Sphingomyelin Composition And Physical Asymmetries In Native Acetylcholine Receptor-Rich Membranes
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Sphingomyelin Composition And Physical Asymmetries In Native Acetylcholine Receptor-Rich Membranes
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Phosphorylated Ca2+-Atpase Stable Enough For Structural Studies
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Phosphorylated Ca2+-Atpase Stable Enough For Structural Studies
A Remarkably Stable Phosphorylated Form Of Ca2+-Atpase Prepared From Ca2+-Loaded And Fluorescein Isothiocyanate-Labeled Sarcoplasmic Reticulum Vesicles
Phosphorylated Ca2+-Atpase Stable Enough For Structural Studies
A Remarkably Stable Phosphorylated Form Of Ca2+-Atpase Prepared From Ca2+-Loaded And Fluorescein Isothiocyanate-Labeled Sarcoplasmic Reticulum Vesicles
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
A Remarkably Stable Phosphorylated Form Of Ca2+-Atpase Prepared From Ca2+-Loaded And Fluorescein Isothiocyanate-Labeled Sarcoplasmic Reticulum Vesicles
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Identification Of An Aspartic Residue In The P-Loop Of The Vanilloid Receptor That Modulates Pore Properties
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Identification Of An Aspartic Residue In The P-Loop Of The Vanilloid Receptor That Modulates Pore Properties
Arginine-Rich Peptides Are Blockers Of Vr-1 Channels With Analgesic Activity
Identification Of An Aspartic Residue In The P-Loop Of The Vanilloid Receptor That Modulates Pore Properties
Arginine-Rich Peptides Are Blockers Of Vr-1 Channels With Analgesic Activity
The Expression Of Plasma Membrane Ca2+ Pump Isoforms In Cerebellar Granule Neurons Is Modulated By Ca2+
Arginine-Rich Peptides Are Blockers Of Vr-1 Channels With Analgesic Activity
The Expression Of Plasma Membrane Ca2+ Pump Isoforms In Cerebellar Granule Neurons Is Modulated By Ca2+
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
The Expression Of Plasma Membrane Ca2+ Pump Isoforms In Cerebellar Granule Neurons Is Modulated By Ca2+
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Characterization Of A Protease-Resistant Domain Of The Cytosolic Portion Of Sarcoplasmic Reticulum Ca2+-Atpase. Nucleotide- And Metal-Binding Sites
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Characterization Of A Protease-Resistant Domain Of The Cytosolic Portion Of Sarcoplasmic Reticulum Ca2+-Atpase. Nucleotide- And Metal-Binding Sites
Analysis Of Secreted Flavonoids Of Cistus Ladanifer L. By High- Performance Liquid Chromatography-Particle Beam Mass Spectrometry
Characterization Of A Protease-Resistant Domain Of The Cytosolic Portion Of Sarcoplasmic Reticulum Ca2+-Atpase. Nucleotide- And Metal-Binding Sites
Analysis Of Secreted Flavonoids Of Cistus Ladanifer L. By High- Performance Liquid Chromatography-Particle Beam Mass Spectrometry
Selected Peptides Targeted To The Nmda Receptor Channel Protect Neurons From Excitotoxic Death
Analysis Of Secreted Flavonoids Of Cistus Ladanifer L. By High- Performance Liquid Chromatography-Particle Beam Mass Spectrometry
Selected Peptides Targeted To The Nmda Receptor Channel Protect Neurons From Excitotoxic Death
Structural Determinants Of The Blocker Binding Site In Glutamate And Nmda Receptor Channels
Selected Peptides Targeted To The Nmda Receptor Channel Protect Neurons From Excitotoxic Death
Structural Determinants Of The Blocker Binding Site In Glutamate And Nmda Receptor Channels
Dissociation Of Ca2+ From Sarcoplasmic Reticulum Ca2+-Atpase And Changes In Fluorescence Of Optically Selected Trp Residues. Effects Of Kcl And Nacl And Implications For Substeps In Ca2+ Dissociation
Structural Determinants Of The Blocker Binding Site In Glutamate And Nmda Receptor Channels
Dissociation Of Ca2+ From Sarcoplasmic Reticulum Ca2+-Atpase And Changes In Fluorescence Of Optically Selected Trp Residues. Effects Of Kcl And Nacl And Implications For Substeps In Ca2+ Dissociation
Quantitative Variation Of Flavonoids Among Individuals Of A Cistus Ladanifer Population
Dissociation Of Ca2+ From Sarcoplasmic Reticulum Ca2+-Atpase And Changes In Fluorescence Of Optically Selected Trp Residues. Effects Of Kcl And Nacl And Implications For Substeps In Ca2+ Dissociation
Quantitative Variation Of Flavonoids Among Individuals Of A Cistus Ladanifer Population
Role Of Ecological Variables In The Seasonal Variation Of Flavonoid Content Of Cistus Ladanifer Exudate
Quantitative Variation Of Flavonoids Among Individuals Of A Cistus Ladanifer Population
Role Of Ecological Variables In The Seasonal Variation Of Flavonoid Content Of Cistus Ladanifer Exudate
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Role Of Ecological Variables In The Seasonal Variation Of Flavonoid Content Of Cistus Ladanifer Exudate
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Hypothalamic Hypophyseal Inhibitory Factor (Hhif) Increases Intrasynaptosomal Free Calcium Concentration
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Hypothalamic Hypophyseal Inhibitory Factor (Hhif) Increases Intrasynaptosomal Free Calcium Concentration
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
Hypothalamic Hypophyseal Inhibitory Factor (Hhif) Increases Intrasynaptosomal Free Calcium Concentration
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
A Comparative Kinetic Analysis Of The Flavin-Photosensitized Oxidation And Reduction Of Plastocyanin And Cytochrome C6from Different Organisms
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
A Comparative Kinetic Analysis Of The Flavin-Photosensitized Oxidation And Reduction Of Plastocyanin And Cytochrome C6from Different Organisms
Physical State Of Bulk And Protein-Associated Lipid In Nicotinic Acetylcholine Receptor-Rich Membrane Studied By Laurdan Generalized Polarization And Fluorescence Energy Transfer
A Comparative Kinetic Analysis Of The Flavin-Photosensitized Oxidation And Reduction Of Plastocyanin And Cytochrome C6from Different Organisms
Physical State Of Bulk And Protein-Associated Lipid In Nicotinic Acetylcholine Receptor-Rich Membrane Studied By Laurdan Generalized Polarization And Fluorescence Energy Transfer
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
Physical State Of Bulk And Protein-Associated Lipid In Nicotinic Acetylcholine Receptor-Rich Membrane Studied By Laurdan Generalized Polarization And Fluorescence Energy Transfer
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Rate Of Na+/ca2+ Exchange Across The Plasma Membrane Of Synaptosomes Measured Using The Fluorescence Of Chlorotetracycline. Implications To Calcium Homeostasis In Synaptic Terminals
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Rate Of Na+/ca2+ Exchange Across The Plasma Membrane Of Synaptosomes Measured Using The Fluorescence Of Chlorotetracycline. Implications To Calcium Homeostasis In Synaptic Terminals
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Rate Of Na+/ca2+ Exchange Across The Plasma Membrane Of Synaptosomes Measured Using The Fluorescence Of Chlorotetracycline. Implications To Calcium Homeostasis In Synaptic Terminals
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Preferential Distribution Of The Fluorescent Phospholipid Probes Nbd-Phosphatidylcholine And Rhodamine-Phosphatidylethanolamine In The Exofacial Leaflet Of Acetylcholine Receptor-Rich Membranes From Torpedo Marmorata
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Preferential Distribution Of The Fluorescent Phospholipid Probes Nbd-Phosphatidylcholine And Rhodamine-Phosphatidylethanolamine In The Exofacial Leaflet Of Acetylcholine Receptor-Rich Membranes From Torpedo Marmorata
Estudio Sobre Relaciones Estructura-Función En La Ca2+,mg2+-Atpasa Del Retículo Sarcoplásmico Del Músculo Esquelético
Preferential Distribution Of The Fluorescent Phospholipid Probes Nbd-Phosphatidylcholine And Rhodamine-Phosphatidylethanolamine In The Exofacial Leaflet Of Acetylcholine Receptor-Rich Membranes From Torpedo Marmorata
Estudio Sobre Relaciones Estructura-Función En La Ca2+,mg2+-Atpasa Del Retículo Sarcoplásmico Del Músculo Esquelético
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Estudio Sobre Relaciones Estructura-Función En La Ca2+,mg2+-Atpasa Del Retículo Sarcoplásmico Del Músculo Esquelético
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Modulation Of The Ca2+ Pump By The Hypothalamic-Hypophysary Inhibitory Factor
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Modulation Of The Ca2+ Pump By The Hypothalamic-Hypophysary Inhibitory Factor
Modulation Of The Ca 2+,mg2+-Atpase Of Sarcoplasmic Reticulum By The Hypothalamic Hypophyseal Inhibitory Factor
Modulation Of The Ca2+ Pump By The Hypothalamic-Hypophysary Inhibitory Factor
Modulation Of The Ca 2+,mg2+-Atpase Of Sarcoplasmic Reticulum By The Hypothalamic Hypophyseal Inhibitory Factor
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Modulation Of The Ca 2+,mg2+-Atpase Of Sarcoplasmic Reticulum By The Hypothalamic Hypophyseal Inhibitory Factor
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Cancellation Of The Cooperativity Of Ca2+ Binding To Sarcoplasmic Reticulum Ca2+‐atpase By The Non‐ionic Detergent Dodecylmaltoside
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Cancellation Of The Cooperativity Of Ca2+ Binding To Sarcoplasmic Reticulum Ca2+‐atpase By The Non‐ionic Detergent Dodecylmaltoside
Seasonal Variation Of Exudate Of Cistus Ladanifer
Cancellation Of The Cooperativity Of Ca2+ Binding To Sarcoplasmic Reticulum Ca2+‐atpase By The Non‐ionic Detergent Dodecylmaltoside
Seasonal Variation Of Exudate Of Cistus Ladanifer
Glycogen Phosphorolysis Can Form A Metabolic Shuttle To Support Ca2+ Uptake By Sarcoplasmic Reticulum Membranes In Skeletal Muscle
Seasonal Variation Of Exudate Of Cistus Ladanifer
Glycogen Phosphorolysis Can Form A Metabolic Shuttle To Support Ca2+ Uptake By Sarcoplasmic Reticulum Membranes In Skeletal Muscle
Advanced Course On Structure-Function Relationships Of Membrane Proteins: An Assessment Of Current Knowledge
Glycogen Phosphorolysis Can Form A Metabolic Shuttle To Support Ca2+ Uptake By Sarcoplasmic Reticulum Membranes In Skeletal Muscle
Advanced Course On Structure-Function Relationships Of Membrane Proteins: An Assessment Of Current Knowledge
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Advanced Course On Structure-Function Relationships Of Membrane Proteins: An Assessment Of Current Knowledge
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Properties Of The Purified Hypothalamic Pituitary Na/k-Atpase Inhibitor
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Properties Of The Purified Hypothalamic Pituitary Na/k-Atpase Inhibitor
Location Of Functional Centers In The Microsomal Cytochrome P450 System
Properties Of The Purified Hypothalamic Pituitary Na/k-Atpase Inhibitor
Location Of Functional Centers In The Microsomal Cytochrome P450 System
The Metal Sites On Sarcoplasmic Reticulum Membranes That Bind Lanthanide Ions With The Highest Affinity Are Not The Atpase Ca2+ Transport Sites
Location Of Functional Centers In The Microsomal Cytochrome P450 System
The Metal Sites On Sarcoplasmic Reticulum Membranes That Bind Lanthanide Ions With The Highest Affinity Are Not The Atpase Ca2+ Transport Sites
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
The Metal Sites On Sarcoplasmic Reticulum Membranes That Bind Lanthanide Ions With The Highest Affinity Are Not The Atpase Ca2+ Transport Sites
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Binding Of Gadolinium Ions To Sarcoplasmic Reticulum Membranes
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Binding Of Gadolinium Ions To Sarcoplasmic Reticulum Membranes
Kinetic Characterization Of The Perturbation By Dodecylmaltoside Of Sarcoplasmic Reticulum Ca2+ ‐atpase
Binding Of Gadolinium Ions To Sarcoplasmic Reticulum Membranes
Kinetic Characterization Of The Perturbation By Dodecylmaltoside Of Sarcoplasmic Reticulum Ca2+ ‐atpase
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Kinetic Characterization Of The Perturbation By Dodecylmaltoside Of Sarcoplasmic Reticulum Ca2+ ‐atpase
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Modulation By Phosphorylation Of Glycogen Phosphorylase-Sarcoplasmic Reticulum Interaction
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Modulation By Phosphorylation Of Glycogen Phosphorylase-Sarcoplasmic Reticulum Interaction
Caracterización Por Técnicas Espectrofotométricas Del Ultravioleta Y Visible Edel Exudado De Cistus Ladanifer L. Como Respuesta Al Estrés Estival
Modulation By Phosphorylation Of Glycogen Phosphorylase-Sarcoplasmic Reticulum Interaction
Caracterización Por Técnicas Espectrofotométricas Del Ultravioleta Y Visible Edel Exudado De Cistus Ladanifer L. Como Respuesta Al Estrés Estival
Hemin And Hemeprotein Bleaching During Linoleic Acid Oxidation By Lipoxygenases
Caracterización Por Técnicas Espectrofotométricas Del Ultravioleta Y Visible Edel Exudado De Cistus Ladanifer L. Como Respuesta Al Estrés Estival
Hemin And Hemeprotein Bleaching During Linoleic Acid Oxidation By Lipoxygenases
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Hemin And Hemeprotein Bleaching During Linoleic Acid Oxidation By Lipoxygenases
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Modulation Of Calcium Fluxes Across Synaptosomal Plasma Membrane By Local Anesthetics
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Modulation Of Calcium Fluxes Across Synaptosomal Plasma Membrane By Local Anesthetics
Modulation Of The Ca2+,mg2+‐atpase Activity Of Synaptosomal Plasma Membrane By The Local Anesthetics Dibucaine And Lidocaine
Modulation Of Calcium Fluxes Across Synaptosomal Plasma Membrane By Local Anesthetics
Modulation Of The Ca2+,mg2+‐atpase Activity Of Synaptosomal Plasma Membrane By The Local Anesthetics Dibucaine And Lidocaine
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Modulation Of The Ca2+,mg2+‐atpase Activity Of Synaptosomal Plasma Membrane By The Local Anesthetics Dibucaine And Lidocaine
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Interaction Of The Local Anesthetics Dibucaine And Tetracaine With Sarcoplasmic Reticulum Membranes. Differential Scanning Calorimetry And Fluorescence Studies
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Interaction Of The Local Anesthetics Dibucaine And Tetracaine With Sarcoplasmic Reticulum Membranes. Differential Scanning Calorimetry And Fluorescence Studies
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Interaction Of The Local Anesthetics Dibucaine And Tetracaine With Sarcoplasmic Reticulum Membranes. Differential Scanning Calorimetry And Fluorescence Studies
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Local Anesthetic-Divalent Cation Binding Center Interaction
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Local Anesthetic-Divalent Cation Binding Center Interaction
Comparative Fluorescence Properties Of Lipoxygenases
Local Anesthetic-Divalent Cation Binding Center Interaction
Comparative Fluorescence Properties Of Lipoxygenases
Effects Of Local Anesthetics On The Passive Permeability Of Sarcoplasmic Reticulum Vesicles To Ca2+ And Mg2+
Comparative Fluorescence Properties Of Lipoxygenases
Effects Of Local Anesthetics On The Passive Permeability Of Sarcoplasmic Reticulum Vesicles To Ca2+ And Mg2+
The Position Of The Atp Binding Site On The (Ca2+ + Mg2+)-Atpase
Effects Of Local Anesthetics On The Passive Permeability Of Sarcoplasmic Reticulum Vesicles To Ca2+ And Mg2+
The Position Of The Atp Binding Site On The (Ca2+ + Mg2+)-Atpase
Long-Wavelength Fluorescence Of Tyrosine And Tryptophan Solutions.
The Position Of The Atp Binding Site On The (Ca2+ + Mg2+)-Atpase
Long-Wavelength Fluorescence Of Tyrosine And Tryptophan Solutions.
Local Anesthetics Inhibit The Ca2+,mg2+‐atpase Activity Of Rat Brain Synaptosomes
Long-Wavelength Fluorescence Of Tyrosine And Tryptophan Solutions.
Local Anesthetics Inhibit The Ca2+,mg2+‐atpase Activity Of Rat Brain Synaptosomes
Dependence Of The Fluorescence Of Fluorescein Labelled (Ca2+, Mg2+)-Atpase Upon The Lipid To Protein Ratio In Sarcoplasmic Reticulum Reconstituted Systems
Local Anesthetics Inhibit The Ca2+,mg2+‐atpase Activity Of Rat Brain Synaptosomes
Dependence Of The Fluorescence Of Fluorescein Labelled (Ca2+, Mg2+)-Atpase Upon The Lipid To Protein Ratio In Sarcoplasmic Reticulum Reconstituted Systems
On The Rate Of Association And Dissociation Processes Of Intrinsic Membrane Proteins
Dependence Of The Fluorescence Of Fluorescein Labelled (Ca2+, Mg2+)-Atpase Upon The Lipid To Protein Ratio In Sarcoplasmic Reticulum Reconstituted Systems
On The Rate Of Association And Dissociation Processes Of Intrinsic Membrane Proteins
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. Ii. Protein Distribution And Aggregation State In Biological Membranes
On The Rate Of Association And Dissociation Processes Of Intrinsic Membrane Proteins
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. Ii. Protein Distribution And Aggregation State In Biological Membranes
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. I. Lateral Phase Separation In Unilamellar Vesicles Formed By Binary Phospholipid Mixtures
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. Ii. Protein Distribution And Aggregation State In Biological Membranes
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. I. Lateral Phase Separation In Unilamellar Vesicles Formed By Binary Phospholipid Mixtures
Relationship Between The Activity Of Pancreatic Phospholipase A2 And The Physical State Of The Phospholipid Substrate.
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. I. Lateral Phase Separation In Unilamellar Vesicles Formed By Binary Phospholipid Mixtures
Relationship Between The Activity Of Pancreatic Phospholipase A2 And The Physical State Of The Phospholipid Substrate.
Regulation Of Phosphorylase B By Amp: The Effects Of Enzyme Concentration
Relationship Between The Activity Of Pancreatic Phospholipase A2 And The Physical State Of The Phospholipid Substrate.
Regulation Of Phosphorylase B By Amp: The Effects Of Enzyme Concentration
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment
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