PUBLICACIÓN

2,3,7,8-tetrachlorodibenzo-p-dioxin induces apoptosis by disruption of intracellular calcium homeostasis in human neuronal cell line SHSY5Y

ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Morales-Hernandez A., Sanchez-Martin F.J., Hortigon-Vinagre M.P., Henao F., Merino J.M.

2012 APOPTOSIS

Biochemistry (medical) (Q1), Cancer Research (Q1), Cell Biology (Q2), Clinical Biochemistry (Q1), Pharmaceutical Science (Q1), Pharmacology (Q1)

JCR: 3.949

SJR: 1.963

CITAS

37

DOI

10.1007/s10495-012-0760-z

EID

2-s2.0-84868106291

ISSN

1360-8185

EISSN

1573-675X

BIBTEX

@article{Merino2012,title = {2,3,7,8-tetrachlorodibenzo-p-dioxin induces apoptosis by disruption of intracellular calcium homeostasis in human neuronal cell line SHSY5Y},journal = {Apoptosis},year = {2012},volume = {17},number = {11},pages = {1170-1181},author = {Morales-Hern{\'a}ndez, A. and S{\'a}nchez-Mart{\'i}n, F.J. and Hortig{\'o}n-Vinagre, M.P. and Henao, F. and Merino, J.M.}}

AUTORES DE LA UEX

Jaime María Merino Fernández

OTRAS PUBLICACIONES DE INTERÉS

Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment.
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment.
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment.
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function
Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function
Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value.
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value.
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value.
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
Ahr-Dependent 2,3,7,8-Tetrachlorodibenzo-P-Dioxin Toxicity In Human Neuronal Cell Line Shsy5y
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
Ahr-Dependent 2,3,7,8-Tetrachlorodibenzo-P-Dioxin Toxicity In Human Neuronal Cell Line Shsy5y
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Ahr-Dependent 2,3,7,8-Tetrachlorodibenzo-P-Dioxin Toxicity In Human Neuronal Cell Line Shsy5y
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
Thymbra Capitata Essential Oil Prevents Cell Death Induced By 4-Hydroxy-2-Nonenal In Neonatal Rat Cardiac Myocytes
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
Thymbra Capitata Essential Oil Prevents Cell Death Induced By 4-Hydroxy-2-Nonenal In Neonatal Rat Cardiac Myocytes
Apoptotic Cell Death In Cultured Cardiomyocytes Following Exposure To Low Concentrations Of 4-Hydroxy-2-Nonenal
Thymbra Capitata Essential Oil Prevents Cell Death Induced By 4-Hydroxy-2-Nonenal In Neonatal Rat Cardiac Myocytes
Apoptotic Cell Death In Cultured Cardiomyocytes Following Exposure To Low Concentrations Of 4-Hydroxy-2-Nonenal
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis By Disruption Of Intracellular Calcium Homeostasis In Human Neuronal Cell Line Shsy5y
Apoptotic Cell Death In Cultured Cardiomyocytes Following Exposure To Low Concentrations Of 4-Hydroxy-2-Nonenal
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis By Disruption Of Intracellular Calcium Homeostasis In Human Neuronal Cell Line Shsy5y
Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis By Disruption Of Intracellular Calcium Homeostasis In Human Neuronal Cell Line Shsy5y
Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling
Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling
Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse
The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling
Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse
Erratum: Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane (Free Radical Biology & Medicine (2011) 50 (323-336))
Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse
Erratum: Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane (Free Radical Biology & Medicine (2011) 50 (323-336))
Inhibition By 4-Hydroxynonenal (Hne) Of Ca 2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
Erratum: Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane (Free Radical Biology & Medicine (2011) 50 (323-336))
Inhibition By 4-Hydroxynonenal (Hne) Of Ca 2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells
Inhibition By 4-Hydroxynonenal (Hne) Of Ca 2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells
Nerve Growth Factor Increases The Sensitivity To Zinc Toxicity And Induces Cell Cycle Arrest In Pc12 Cells
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells
Nerve Growth Factor Increases The Sensitivity To Zinc Toxicity And Induces Cell Cycle Arrest In Pc12 Cells
Role Of Transforming Growth Factor Β In Cancer Microenvironment
Nerve Growth Factor Increases The Sensitivity To Zinc Toxicity And Induces Cell Cycle Arrest In Pc12 Cells
Role Of Transforming Growth Factor Β In Cancer Microenvironment
The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion
Role Of Transforming Growth Factor Β In Cancer Microenvironment
The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion
Nmda-Induced Neuroprotection In Hippocampal Neurons Is Mediated Through The Protein Kinase A And Creb (Camp-Response Element-Binding Protein) Pathway
The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion
Nmda-Induced Neuroprotection In Hippocampal Neurons Is Mediated Through The Protein Kinase A And Creb (Camp-Response Element-Binding Protein) Pathway
Sarcoplasmic Reticulum Calcium Atpase Is Inhibited By Organic Vanadium Coordination Compounds: Pyridine-2,6-Dicarboxylatodioxovanadium(V), Bmov, And An Amavadine Analogue
Nmda-Induced Neuroprotection In Hippocampal Neurons Is Mediated Through The Protein Kinase A And Creb (Camp-Response Element-Binding Protein) Pathway
Sarcoplasmic Reticulum Calcium Atpase Is Inhibited By Organic Vanadium Coordination Compounds: Pyridine-2,6-Dicarboxylatodioxovanadium(V), Bmov, And An Amavadine Analogue
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Sarcoplasmic Reticulum Calcium Atpase Is Inhibited By Organic Vanadium Coordination Compounds: Pyridine-2,6-Dicarboxylatodioxovanadium(V), Bmov, And An Amavadine Analogue
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Store-Operated Calcium Entry In Differentiated C2c12 Skeletal Muscle Cells
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Store-Operated Calcium Entry In Differentiated C2c12 Skeletal Muscle Cells
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Store-Operated Calcium Entry In Differentiated C2c12 Skeletal Muscle Cells
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Small Peptides Patterned After The N-Terminus Domain Of Snap25 Inhibit Snare Complex Assembly And Regulated Exocytosis
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Small Peptides Patterned After The N-Terminus Domain Of Snap25 Inhibit Snare Complex Assembly And Regulated Exocytosis
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Small Peptides Patterned After The N-Terminus Domain Of Snap25 Inhibit Snare Complex Assembly And Regulated Exocytosis
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Identification Of Snare Complex Modulators That Inhibit Exocytosis From An Α-Helix-Constrained Combinatorial Library
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Identification Of Snare Complex Modulators That Inhibit Exocytosis From An Α-Helix-Constrained Combinatorial Library
Projection Map Of Covalently Phosphorylated Ca-Atpase From Tubular Crystals
Identification Of Snare Complex Modulators That Inhibit Exocytosis From An Α-Helix-Constrained Combinatorial Library
Projection Map Of Covalently Phosphorylated Ca-Atpase From Tubular Crystals
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
Projection Map Of Covalently Phosphorylated Ca-Atpase From Tubular Crystals
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
A Novel N-Methyl-D-Aspartate Receptor Open Channel Blocker With In Vivo Neuroprotectant Activity
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
A Novel N-Methyl-D-Aspartate Receptor Open Channel Blocker With In Vivo Neuroprotectant Activity
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
A Novel N-Methyl-D-Aspartate Receptor Open Channel Blocker With In Vivo Neuroprotectant Activity
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Phosphorylated Ca2+-Atpase Stable Enough For Structural Studies
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Phosphorylated Ca2+-Atpase Stable Enough For Structural Studies
A Remarkably Stable Phosphorylated Form Of Ca2+-Atpase Prepared From Ca2+-Loaded And Fluorescein Isothiocyanate-Labeled Sarcoplasmic Reticulum Vesicles
Phosphorylated Ca2+-Atpase Stable Enough For Structural Studies
A Remarkably Stable Phosphorylated Form Of Ca2+-Atpase Prepared From Ca2+-Loaded And Fluorescein Isothiocyanate-Labeled Sarcoplasmic Reticulum Vesicles
Identification Of An Aspartic Residue In The P-Loop Of The Vanilloid Receptor That Modulates Pore Properties
A Remarkably Stable Phosphorylated Form Of Ca2+-Atpase Prepared From Ca2+-Loaded And Fluorescein Isothiocyanate-Labeled Sarcoplasmic Reticulum Vesicles
Identification Of An Aspartic Residue In The P-Loop Of The Vanilloid Receptor That Modulates Pore Properties
Arginine-Rich Peptides Are Blockers Of Vr-1 Channels With Analgesic Activity
Identification Of An Aspartic Residue In The P-Loop Of The Vanilloid Receptor That Modulates Pore Properties
Arginine-Rich Peptides Are Blockers Of Vr-1 Channels With Analgesic Activity
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Arginine-Rich Peptides Are Blockers Of Vr-1 Channels With Analgesic Activity
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Characterization Of A Protease-Resistant Domain Of The Cytosolic Portion Of Sarcoplasmic Reticulum Ca2+-Atpase. Nucleotide- And Metal-Binding Sites
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Characterization Of A Protease-Resistant Domain Of The Cytosolic Portion Of Sarcoplasmic Reticulum Ca2+-Atpase. Nucleotide- And Metal-Binding Sites
Selected Peptides Targeted To The Nmda Receptor Channel Protect Neurons From Excitotoxic Death
Characterization Of A Protease-Resistant Domain Of The Cytosolic Portion Of Sarcoplasmic Reticulum Ca2+-Atpase. Nucleotide- And Metal-Binding Sites
Selected Peptides Targeted To The Nmda Receptor Channel Protect Neurons From Excitotoxic Death
Structural Determinants Of The Blocker Binding Site In Glutamate And Nmda Receptor Channels
Selected Peptides Targeted To The Nmda Receptor Channel Protect Neurons From Excitotoxic Death
Structural Determinants Of The Blocker Binding Site In Glutamate And Nmda Receptor Channels
Dissociation Of Ca2+ From Sarcoplasmic Reticulum Ca2+-Atpase And Changes In Fluorescence Of Optically Selected Trp Residues. Effects Of Kcl And Nacl And Implications For Substeps In Ca2+ Dissociation
Structural Determinants Of The Blocker Binding Site In Glutamate And Nmda Receptor Channels
Dissociation Of Ca2+ From Sarcoplasmic Reticulum Ca2+-Atpase And Changes In Fluorescence Of Optically Selected Trp Residues. Effects Of Kcl And Nacl And Implications For Substeps In Ca2+ Dissociation
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Dissociation Of Ca2+ From Sarcoplasmic Reticulum Ca2+-Atpase And Changes In Fluorescence Of Optically Selected Trp Residues. Effects Of Kcl And Nacl And Implications For Substeps In Ca2+ Dissociation
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Estudio Sobre Relaciones Estructura-Función En La Ca2+,mg2+-Atpasa Del Retículo Sarcoplásmico Del Músculo Esquelético
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Estudio Sobre Relaciones Estructura-Función En La Ca2+,mg2+-Atpasa Del Retículo Sarcoplásmico Del Músculo Esquelético
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Estudio Sobre Relaciones Estructura-Función En La Ca2+,mg2+-Atpasa Del Retículo Sarcoplásmico Del Músculo Esquelético
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Cancellation Of The Cooperativity Of Ca2+ Binding To Sarcoplasmic Reticulum Ca2+‐atpase By The Non‐ionic Detergent Dodecylmaltoside
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Cancellation Of The Cooperativity Of Ca2+ Binding To Sarcoplasmic Reticulum Ca2+‐atpase By The Non‐ionic Detergent Dodecylmaltoside
The Metal Sites On Sarcoplasmic Reticulum Membranes That Bind Lanthanide Ions With The Highest Affinity Are Not The Atpase Ca2+ Transport Sites
Cancellation Of The Cooperativity Of Ca2+ Binding To Sarcoplasmic Reticulum Ca2+‐atpase By The Non‐ionic Detergent Dodecylmaltoside
The Metal Sites On Sarcoplasmic Reticulum Membranes That Bind Lanthanide Ions With The Highest Affinity Are Not The Atpase Ca2+ Transport Sites
Binding Of Gadolinium Ions To Sarcoplasmic Reticulum Membranes
The Metal Sites On Sarcoplasmic Reticulum Membranes That Bind Lanthanide Ions With The Highest Affinity Are Not The Atpase Ca2+ Transport Sites
Binding Of Gadolinium Ions To Sarcoplasmic Reticulum Membranes
Kinetic Characterization Of The Perturbation By Dodecylmaltoside Of Sarcoplasmic Reticulum Ca2+ ‐atpase
Binding Of Gadolinium Ions To Sarcoplasmic Reticulum Membranes
Kinetic Characterization Of The Perturbation By Dodecylmaltoside Of Sarcoplasmic Reticulum Ca2+ ‐atpase
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Kinetic Characterization Of The Perturbation By Dodecylmaltoside Of Sarcoplasmic Reticulum Ca2+ ‐atpase
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment
WEB ACCESIBLE