PUBLICACIÓN

Agonist-stimulated pathways of calcium signaling in pancreatic acinar cells

ACCEDER A LA PUBLICACIÓN: Scopus

1999 Biological Chemistry

CITAS

18

DOI

10.1515/bc.1999.111

EID

2-s2.0-0032883188

ISSN

1431-6730

AUTORES DE LA UEX

Antonio González Mateos

OTRAS PUBLICACIONES DE INTERÉS

Two Sesquiterpene Lactones From Centaurea Canariensis
Melatonin Interplay In Physiology And Disease—the Fountain Of Eternal Youth Revisited
In Vitro Digestion And Culture In Caco-2 Cells To Assess The Bioavailability Of Fatty Acids: A Case Study In Meat Matrix Enriched With Ω-3 Microcapsules
Melatonin Interplay In Physiology And Disease—the Fountain Of Eternal Youth Revisited
In Vitro Digestion And Culture In Caco-2 Cells To Assess The Bioavailability Of Fatty Acids: A Case Study In Meat Matrix Enriched With Ω-3 Microcapsules
Membrane Lipid Derivatives: Roles Of Arachidonic Acid And Its Metabolites In Pancreatic Physiology And Pathophysiology
In Vitro Digestion And Culture In Caco-2 Cells To Assess The Bioavailability Of Fatty Acids: A Case Study In Meat Matrix Enriched With Ω-3 Microcapsules
Membrane Lipid Derivatives: Roles Of Arachidonic Acid And Its Metabolites In Pancreatic Physiology And Pathophysiology
Special Issue: 2022 Pancreatic Cancer And The Role Of The Stroma In The Pathophysiology Of The Gland
Membrane Lipid Derivatives: Roles Of Arachidonic Acid And Its Metabolites In Pancreatic Physiology And Pathophysiology
Special Issue: 2022 Pancreatic Cancer And The Role Of The Stroma In The Pathophysiology Of The Gland
Melatonin Controls Cell Proliferation And Modulates Mitochondrial Physiology In Pancreatic Stellate Cells
Special Issue: 2022 Pancreatic Cancer And The Role Of The Stroma In The Pathophysiology Of The Gland
Melatonin Controls Cell Proliferation And Modulates Mitochondrial Physiology In Pancreatic Stellate Cells
La Fisiología: Base De Las Ciencias Médicas Y Biológicas
Melatonin Controls Cell Proliferation And Modulates Mitochondrial Physiology In Pancreatic Stellate Cells
La Fisiología: Base De Las Ciencias Médicas Y Biológicas
Melatonin Modulates Metabolic Adaptation Of Pancreatic Stellate Cells Subjected To Hypoxia
La Fisiología: Base De Las Ciencias Médicas Y Biológicas
Melatonin Modulates Metabolic Adaptation Of Pancreatic Stellate Cells Subjected To Hypoxia
Melatonin Modulates The Antioxidant Defenses And The Expression Of Proinflammatory Mediators In Pancreatic Stellate Cells Subjected To Hypoxia
Melatonin Modulates Metabolic Adaptation Of Pancreatic Stellate Cells Subjected To Hypoxia
Melatonin Modulates The Antioxidant Defenses And The Expression Of Proinflammatory Mediators In Pancreatic Stellate Cells Subjected To Hypoxia
Modulation Of Cell Physiology Under Hypoxia In Pancreatic Cancer
Melatonin Modulates The Antioxidant Defenses And The Expression Of Proinflammatory Mediators In Pancreatic Stellate Cells Subjected To Hypoxia
Modulation Of Cell Physiology Under Hypoxia In Pancreatic Cancer
Melatonin Induces Apoptosis And Modulates Cyclin Expression And Mapk Phosphorylation In Pancreatic Stellate Cells Subjected To Hypoxia
Modulation Of Cell Physiology Under Hypoxia In Pancreatic Cancer
Melatonin Induces Apoptosis And Modulates Cyclin Expression And Mapk Phosphorylation In Pancreatic Stellate Cells Subjected To Hypoxia
Antioxidants And Neuron-Astrocyte Interplay In Brain Physiology: Melatonin, A Neighbor To Rely On
Melatonin Induces Apoptosis And Modulates Cyclin Expression And Mapk Phosphorylation In Pancreatic Stellate Cells Subjected To Hypoxia
Antioxidants And Neuron-Astrocyte Interplay In Brain Physiology: Melatonin, A Neighbor To Rely On
The Melatonin Receptor Antagonist Luzindole Induces The Activation Of Cellular Stress Responses And Decreases Viability Of Rat Pancreatic Stellate Cells
Antioxidants And Neuron-Astrocyte Interplay In Brain Physiology: Melatonin, A Neighbor To Rely On
The Melatonin Receptor Antagonist Luzindole Induces The Activation Of Cellular Stress Responses And Decreases Viability Of Rat Pancreatic Stellate Cells
Melatonin Modulates Proliferation Of Pancreatic Stellate Cells Through Caspase-3 Activation And Changes In Cyclin A And D Expression
The Melatonin Receptor Antagonist Luzindole Induces The Activation Of Cellular Stress Responses And Decreases Viability Of Rat Pancreatic Stellate Cells
Melatonin Modulates Proliferation Of Pancreatic Stellate Cells Through Caspase-3 Activation And Changes In Cyclin A And D Expression
Melatonin Modulates Red-Ox State And Decreases Viability Of Rat Pancreatic Stellate Cells
Melatonin Modulates Proliferation Of Pancreatic Stellate Cells Through Caspase-3 Activation And Changes In Cyclin A And D Expression
Melatonin Modulates Red-Ox State And Decreases Viability Of Rat Pancreatic Stellate Cells
Effect Of Tartrazine On Digestive Enzymatic Activities: In Vivo And In Vitro Studies
Melatonin Modulates Red-Ox State And Decreases Viability Of Rat Pancreatic Stellate Cells
Effect Of Tartrazine On Digestive Enzymatic Activities: In Vivo And In Vitro Studies
The Lysine Derivative Aminoadipic Acid, A Biomarker Of Protein Oxidation And Diabetes-Risk, Induces Production Of Reactive Oxygen Species And Impairs Trypsin Secretion In Mouse Pancreatic Acinar Cells
Effect Of Tartrazine On Digestive Enzymatic Activities: In Vivo And In Vitro Studies
The Lysine Derivative Aminoadipic Acid, A Biomarker Of Protein Oxidation And Diabetes-Risk, Induces Production Of Reactive Oxygen Species And Impairs Trypsin Secretion In Mouse Pancreatic Acinar Cells
Noxious Effects Of Selected Food-Occurring Oxidized Amino Acids On Differentiated Caco-2 Intestinal Human Cells
The Lysine Derivative Aminoadipic Acid, A Biomarker Of Protein Oxidation And Diabetes-Risk, Induces Production Of Reactive Oxygen Species And Impairs Trypsin Secretion In Mouse Pancreatic Acinar Cells
Noxious Effects Of Selected Food-Occurring Oxidized Amino Acids On Differentiated Caco-2 Intestinal Human Cells
Pancreatic Stellate Cells Exhibit Adaptation To Oxidative Stress Evoked By Hypoxia
Noxious Effects Of Selected Food-Occurring Oxidized Amino Acids On Differentiated Caco-2 Intestinal Human Cells
Pancreatic Stellate Cells Exhibit Adaptation To Oxidative Stress Evoked By Hypoxia
The Organophosphorus Pesticide Dimethoate Decreases Cell Viability And Induces Changes In Different Biochemical Parameters Of Rat Pancreatic Stellate Cells
Pancreatic Stellate Cells Exhibit Adaptation To Oxidative Stress Evoked By Hypoxia
The Organophosphorus Pesticide Dimethoate Decreases Cell Viability And Induces Changes In Different Biochemical Parameters Of Rat Pancreatic Stellate Cells
The Melatonin Receptor Antagonist Luzindole Induces Ca2+ Mobilization, Reactive Oxygen Species Generation And Impairs Trypsin Secretion In Mouse Pancreatic Acinar Cells
The Organophosphorus Pesticide Dimethoate Decreases Cell Viability And Induces Changes In Different Biochemical Parameters Of Rat Pancreatic Stellate Cells
The Melatonin Receptor Antagonist Luzindole Induces Ca2+ Mobilization, Reactive Oxygen Species Generation And Impairs Trypsin Secretion In Mouse Pancreatic Acinar Cells
Melatonin Induces Reactive Oxygen Species Generation And Changes In Glutathione Levels And Reduces Viability In Human Pancreatic Stellate Cells
The Melatonin Receptor Antagonist Luzindole Induces Ca2+ Mobilization, Reactive Oxygen Species Generation And Impairs Trypsin Secretion In Mouse Pancreatic Acinar Cells
Melatonin Induces Reactive Oxygen Species Generation And Changes In Glutathione Levels And Reduces Viability In Human Pancreatic Stellate Cells
Ingesta
Melatonin Induces Reactive Oxygen Species Generation And Changes In Glutathione Levels And Reduces Viability In Human Pancreatic Stellate Cells
Ingesta
Secreción Salival
Ingesta
Secreción Salival
Inactivation Of Nupr1 Promotes Cell Death By Coupling Er-Stress Responses With Necrosis
Secreción Salival
Inactivation Of Nupr1 Promotes Cell Death By Coupling Er-Stress Responses With Necrosis
Sulfanilic Acid Increases Intracellular Free-Calcium Concentration, Induces Reactive Oxygen Species Production And Impairs Trypsin Secretion In Pancreatic Ar42j Cells
Inactivation Of Nupr1 Promotes Cell Death By Coupling Er-Stress Responses With Necrosis
Sulfanilic Acid Increases Intracellular Free-Calcium Concentration, Induces Reactive Oxygen Species Production And Impairs Trypsin Secretion In Pancreatic Ar42j Cells
Ebselen Impairs Cellular Oxidative State And Induces Endoplasmic Reticulum Stress And Activation Of Crucial Mitogen-Activated Protein Kinases In Pancreatic Tumour Ar42j Cells
Sulfanilic Acid Increases Intracellular Free-Calcium Concentration, Induces Reactive Oxygen Species Production And Impairs Trypsin Secretion In Pancreatic Ar42j Cells
Ebselen Impairs Cellular Oxidative State And Induces Endoplasmic Reticulum Stress And Activation Of Crucial Mitogen-Activated Protein Kinases In Pancreatic Tumour Ar42j Cells
Interferences Of Resveratrol With Fura-2-Derived Fluorescence In Intracellular Free-Ca2+ Concentration Determinations
Ebselen Impairs Cellular Oxidative State And Induces Endoplasmic Reticulum Stress And Activation Of Crucial Mitogen-Activated Protein Kinases In Pancreatic Tumour Ar42j Cells
Interferences Of Resveratrol With Fura-2-Derived Fluorescence In Intracellular Free-Ca2+ Concentration Determinations
Ebselen Alters Cellular Oxidative Status And Induces Endoplasmic Reticulum Stress In Rat Hippocampal Astrocytes
Interferences Of Resveratrol With Fura-2-Derived Fluorescence In Intracellular Free-Ca2+ Concentration Determinations
Ebselen Alters Cellular Oxidative Status And Induces Endoplasmic Reticulum Stress In Rat Hippocampal Astrocytes
Manual Básico Para Usuarios De Animales En La Experimentación En Ciencias Biomédicas
Ebselen Alters Cellular Oxidative Status And Induces Endoplasmic Reticulum Stress In Rat Hippocampal Astrocytes
Manual Básico Para Usuarios De Animales En La Experimentación En Ciencias Biomédicas
El Principio De Las Tres Erres: Reemplazo, Reducción Y Refinamiento
Manual Básico Para Usuarios De Animales En La Experimentación En Ciencias Biomédicas
El Principio De Las Tres Erres: Reemplazo, Reducción Y Refinamiento
Astrocyte Nmda Receptors' Activity Sustains Neuronal Survival Through A Cdk5-Nrf2 Pathway
El Principio De Las Tres Erres: Reemplazo, Reducción Y Refinamiento
Astrocyte Nmda Receptors' Activity Sustains Neuronal Survival Through A Cdk5-Nrf2 Pathway
Modulation Of Copper Accumulation And Copper-Induced Toxicity By Antioxidants And Copper Chelators In Cultured Primary Brain Astrocytes
Astrocyte Nmda Receptors' Activity Sustains Neuronal Survival Through A Cdk5-Nrf2 Pathway
Modulation Of Copper Accumulation And Copper-Induced Toxicity By Antioxidants And Copper Chelators In Cultured Primary Brain Astrocytes
Melatonin Induces The Expression Of Nrf2-Regulated Antioxidant Enzymes Via Pkc And Ca2+ Influx Activation In Mouse Pancreatic Acinar Cells
Modulation Of Copper Accumulation And Copper-Induced Toxicity By Antioxidants And Copper Chelators In Cultured Primary Brain Astrocytes
Melatonin Induces The Expression Of Nrf2-Regulated Antioxidant Enzymes Via Pkc And Ca2+ Influx Activation In Mouse Pancreatic Acinar Cells
Melatonin Induces Calcium Mobilization And Influences Cell Proliferation Independently Of Mt1/mt2 Receptor Activation In Rat Pancreatic Stellate Cells
Melatonin Induces The Expression Of Nrf2-Regulated Antioxidant Enzymes Via Pkc And Ca2+ Influx Activation In Mouse Pancreatic Acinar Cells
Melatonin Induces Calcium Mobilization And Influences Cell Proliferation Independently Of Mt1/mt2 Receptor Activation In Rat Pancreatic Stellate Cells
Melatonin, Mitochondria, And Ca2+ Homeostasis In The Exocrine Pancreas: An Overview
Melatonin Induces Calcium Mobilization And Influences Cell Proliferation Independently Of Mt1/mt2 Receptor Activation In Rat Pancreatic Stellate Cells
Melatonin, Mitochondria, And Ca2+ Homeostasis In The Exocrine Pancreas: An Overview
Pharmacological Dose Of Melatonin Reduces Cytosolic Calcium Load In Response To Cholecystokinin In Mouse Pancreatic Acinar Cells
Melatonin, Mitochondria, And Ca2+ Homeostasis In The Exocrine Pancreas: An Overview
Pharmacological Dose Of Melatonin Reduces Cytosolic Calcium Load In Response To Cholecystokinin In Mouse Pancreatic Acinar Cells
The Seleno-Organic Compound Ebselen Impairs Mitochondrial Physiology And Induces Cell Death In Ar42j Cells
Pharmacological Dose Of Melatonin Reduces Cytosolic Calcium Load In Response To Cholecystokinin In Mouse Pancreatic Acinar Cells
The Seleno-Organic Compound Ebselen Impairs Mitochondrial Physiology And Induces Cell Death In Ar42j Cells
Efecto De La Orquidectomía Sobre El Tamaño Y El Peso De Las Vesículas Seminales De La Rata
The Seleno-Organic Compound Ebselen Impairs Mitochondrial Physiology And Induces Cell Death In Ar42j Cells
Efecto De La Orquidectomía Sobre El Tamaño Y El Peso De Las Vesículas Seminales De La Rata
Diseño De Un Proyecto De Investigación Básico
Efecto De La Orquidectomía Sobre El Tamaño Y El Peso De Las Vesículas Seminales De La Rata
Diseño De Un Proyecto De Investigación Básico
Melatonin Modulates Ca2+ Mobilization And Amylase Release In Response To Cholecystokinin Octapeptide In Mouse Pancreatic Acinar Cells
Diseño De Un Proyecto De Investigación Básico
Melatonin Modulates Ca2+ Mobilization And Amylase Release In Response To Cholecystokinin Octapeptide In Mouse Pancreatic Acinar Cells
Ebselen Alters Mitochondrial Physiology And Reduces Viability Of Rat Hippocampal Astrocytes
Melatonin Modulates Ca2+ Mobilization And Amylase Release In Response To Cholecystokinin Octapeptide In Mouse Pancreatic Acinar Cells
Ebselen Alters Mitochondrial Physiology And Reduces Viability Of Rat Hippocampal Astrocytes
Cinnamtannin B-1, A Natural Antioxidant That Reduces The Effects Of H 2o 2 On Cck-8-Evoked Responses In Mouse Pancreatic Acinar Cells
Ebselen Alters Mitochondrial Physiology And Reduces Viability Of Rat Hippocampal Astrocytes
Cinnamtannin B-1, A Natural Antioxidant That Reduces The Effects Of H 2o 2 On Cck-8-Evoked Responses In Mouse Pancreatic Acinar Cells
Resveratrol Mobilizes Ca2+ From Intracellular Stores And Induces C-Jun N-Terminal Kinase Activation In Tumoral Ar42j Cells
Cinnamtannin B-1, A Natural Antioxidant That Reduces The Effects Of H 2o 2 On Cck-8-Evoked Responses In Mouse Pancreatic Acinar Cells
Resveratrol Mobilizes Ca2+ From Intracellular Stores And Induces C-Jun N-Terminal Kinase Activation In Tumoral Ar42j Cells
Ethanol Reduces Kainate-Evoked Glutamate Secretion In Rat Hippocampal Astrocytes
Resveratrol Mobilizes Ca2+ From Intracellular Stores And Induces C-Jun N-Terminal Kinase Activation In Tumoral Ar42j Cells
Ethanol Reduces Kainate-Evoked Glutamate Secretion In Rat Hippocampal Astrocytes
Melatonin Reduces Pancreatic Tumor Cell Viability By Altering Mitochondrial Physiology
Ethanol Reduces Kainate-Evoked Glutamate Secretion In Rat Hippocampal Astrocytes
Melatonin Reduces Pancreatic Tumor Cell Viability By Altering Mitochondrial Physiology
Educación Integral Sobre Sexualidad En Adolescentes Por Su Enfermera Comunitaria
Melatonin Reduces Pancreatic Tumor Cell Viability By Altering Mitochondrial Physiology
Educación Integral Sobre Sexualidad En Adolescentes Por Su Enfermera Comunitaria
Regulator Of G-Protein Signaling 14 Protein Modulates Ca2+ Influx Through Cav1 Channels
Educación Integral Sobre Sexualidad En Adolescentes Por Su Enfermera Comunitaria
Regulator Of G-Protein Signaling 14 Protein Modulates Ca2+ Influx Through Cav1 Channels
Effect Of Cinnamtannin B-1 On Cholecystokinin-8-Evoked Responses In Mouse Pancreatic Acinar Cells
Regulator Of G-Protein Signaling 14 Protein Modulates Ca2+ Influx Through Cav1 Channels
Effect Of Cinnamtannin B-1 On Cholecystokinin-8-Evoked Responses In Mouse Pancreatic Acinar Cells
Melatonin Induces Calcium Release From Cck-8- And Thapsigargin-Sensitive Cytosolic Stores In Pancreatic Ar42j Cells
Effect Of Cinnamtannin B-1 On Cholecystokinin-8-Evoked Responses In Mouse Pancreatic Acinar Cells
Melatonin Induces Calcium Release From Cck-8- And Thapsigargin-Sensitive Cytosolic Stores In Pancreatic Ar42j Cells
Increased Calcium Influx In The Presence Of Ethanol In Mouse Pancreatic Acinar Cells
Melatonin Induces Calcium Release From Cck-8- And Thapsigargin-Sensitive Cytosolic Stores In Pancreatic Ar42j Cells
Increased Calcium Influx In The Presence Of Ethanol In Mouse Pancreatic Acinar Cells
Ethanol Alters The Physiology Of Neuron-Glia Communication
Increased Calcium Influx In The Presence Of Ethanol In Mouse Pancreatic Acinar Cells
Ethanol Alters The Physiology Of Neuron-Glia Communication
Ethanol Exerts Dual Effects On Calcium Homeostasis In Cck-8-Stimulated Mouse Pancreatic Acinar Cells
Ethanol Alters The Physiology Of Neuron-Glia Communication
Ethanol Exerts Dual Effects On Calcium Homeostasis In Cck-8-Stimulated Mouse Pancreatic Acinar Cells
Ethanol Impairs Calcium Homeostasis Following Cck-8 Stimulation In Mouse Pancreatic Acinar Cells
Ethanol Exerts Dual Effects On Calcium Homeostasis In Cck-8-Stimulated Mouse Pancreatic Acinar Cells
Ethanol Impairs Calcium Homeostasis Following Cck-8 Stimulation In Mouse Pancreatic Acinar Cells
Ethanol Induces Glutamate Secretion By Ca2+ Mobilization And Ros Generation In Rat Hippocampal Astrocytes
Ethanol Impairs Calcium Homeostasis Following Cck-8 Stimulation In Mouse Pancreatic Acinar Cells
Ethanol Induces Glutamate Secretion By Ca2+ Mobilization And Ros Generation In Rat Hippocampal Astrocytes
Ebselen Increases Cytosolic Free Ca2+ Concentration, Stimulates Glutamate Release And Increases Gfap Content In Rat Hippocampal Astrocytes
Ethanol Induces Glutamate Secretion By Ca2+ Mobilization And Ros Generation In Rat Hippocampal Astrocytes
Ebselen Increases Cytosolic Free Ca2+ Concentration, Stimulates Glutamate Release And Increases Gfap Content In Rat Hippocampal Astrocytes
Role Of Intracellular Calcium On Hydrogen Peroxide-Induced Apoptosis In Rat Pancreatic Acinar Ar42j Cells
Ebselen Increases Cytosolic Free Ca2+ Concentration, Stimulates Glutamate Release And Increases Gfap Content In Rat Hippocampal Astrocytes
Role Of Intracellular Calcium On Hydrogen Peroxide-Induced Apoptosis In Rat Pancreatic Acinar Ar42j Cells
Apoptosis In Nervous Cells
Role Of Intracellular Calcium On Hydrogen Peroxide-Induced Apoptosis In Rat Pancreatic Acinar Ar42j Cells
Apoptosis In Nervous Cells
Ethanol Stimulates Ros Generation By Mitochondria Through Ca2+ Mobilization And Increases Gfap Content In Rat Hippocampal Astrocytes
Apoptosis In Nervous Cells
Ethanol Stimulates Ros Generation By Mitochondria Through Ca2+ Mobilization And Increases Gfap Content In Rat Hippocampal Astrocytes
Modulation Of Cck-8-Evoked Intracellular Ca2+ Waves By Hydrogen Peroxide In Mouse Pancreatic Acinar Cells
Ethanol Stimulates Ros Generation By Mitochondria Through Ca2+ Mobilization And Increases Gfap Content In Rat Hippocampal Astrocytes
Modulation Of Cck-8-Evoked Intracellular Ca2+ Waves By Hydrogen Peroxide In Mouse Pancreatic Acinar Cells
Ethanol Impairs Cck-8-Evoked Amylase Secretion Through Ca2+-Mediated Ros Generation In Mouse Pancreatic Acinar Cells
Modulation Of Cck-8-Evoked Intracellular Ca2+ Waves By Hydrogen Peroxide In Mouse Pancreatic Acinar Cells
Ethanol Impairs Cck-8-Evoked Amylase Secretion Through Ca2+-Mediated Ros Generation In Mouse Pancreatic Acinar Cells
H2o2 Mobilizes Ca2+ From Agonist- And Thapsigargin-Sensitive And Insensitive Intracellular Stores And Stimulates Glutamate Secretion In Rat Hippocampal Astrocytes
Ethanol Impairs Cck-8-Evoked Amylase Secretion Through Ca2+-Mediated Ros Generation In Mouse Pancreatic Acinar Cells
H2o2 Mobilizes Ca2+ From Agonist- And Thapsigargin-Sensitive And Insensitive Intracellular Stores And Stimulates Glutamate Secretion In Rat Hippocampal Astrocytes
Involvement Of Ryanodine-Operated Channels In Tert-Butylhydroperoxide- Evoked Ca2+ Mobilisation In Pancreatic Acinar Cells
H2o2 Mobilizes Ca2+ From Agonist- And Thapsigargin-Sensitive And Insensitive Intracellular Stores And Stimulates Glutamate Secretion In Rat Hippocampal Astrocytes
Involvement Of Ryanodine-Operated Channels In Tert-Butylhydroperoxide- Evoked Ca2+ Mobilisation In Pancreatic Acinar Cells
Dose-Dependent Effect Of Hydrogen Peroxide On Calcium Mobilization In Mouse Pancreatic Acinar Cells
Involvement Of Ryanodine-Operated Channels In Tert-Butylhydroperoxide- Evoked Ca2+ Mobilisation In Pancreatic Acinar Cells
Dose-Dependent Effect Of Hydrogen Peroxide On Calcium Mobilization In Mouse Pancreatic Acinar Cells
Effect Of H2o2 On Cck-8-Evoked Changes In Mitochondrial Activity In Isolated Mouse Pancreatic Acinar Cells
Dose-Dependent Effect Of Hydrogen Peroxide On Calcium Mobilization In Mouse Pancreatic Acinar Cells
Effect Of H2o2 On Cck-8-Evoked Changes In Mitochondrial Activity In Isolated Mouse Pancreatic Acinar Cells
H2o2-Induced Changes In Mitochondrial Activity In Isolated Mouse Pancreatic Acinar Cells
Effect Of H2o2 On Cck-8-Evoked Changes In Mitochondrial Activity In Isolated Mouse Pancreatic Acinar Cells
H2o2-Induced Changes In Mitochondrial Activity In Isolated Mouse Pancreatic Acinar Cells
Dietary Virgin Olive Oil Enhances Secretagogue-Evoked Calcium Signaling In Rat Pancreatic Acinar Cells
H2o2-Induced Changes In Mitochondrial Activity In Isolated Mouse Pancreatic Acinar Cells
Dietary Virgin Olive Oil Enhances Secretagogue-Evoked Calcium Signaling In Rat Pancreatic Acinar Cells
Generation Of Ros In Response To Cck-8 Stimulation In Mouse Pancreatic Acinar Cells
Dietary Virgin Olive Oil Enhances Secretagogue-Evoked Calcium Signaling In Rat Pancreatic Acinar Cells
Generation Of Ros In Response To Cck-8 Stimulation In Mouse Pancreatic Acinar Cells
Changes In Mitochondrial Activity Evoked By Cholecystokinin In Isolated Mouse Pancreatic Acinar Cells
Generation Of Ros In Response To Cck-8 Stimulation In Mouse Pancreatic Acinar Cells
Changes In Mitochondrial Activity Evoked By Cholecystokinin In Isolated Mouse Pancreatic Acinar Cells
Evidence For Secretion-Like Coupling Involving Pp60src In The Activation And Maintenance Of Store-Mediated Ca2+ Entry In Mouse Pancreatic Acinar Cells
Changes In Mitochondrial Activity Evoked By Cholecystokinin In Isolated Mouse Pancreatic Acinar Cells
Evidence For Secretion-Like Coupling Involving Pp60src In The Activation And Maintenance Of Store-Mediated Ca2+ Entry In Mouse Pancreatic Acinar Cells
Effects Of Reactive Oxygen Species On Actin Filament Polymerisation And Amylase Secretion In Mouse Pancreatic Acinar Cells
Evidence For Secretion-Like Coupling Involving Pp60src In The Activation And Maintenance Of Store-Mediated Ca2+ Entry In Mouse Pancreatic Acinar Cells
Effects Of Reactive Oxygen Species On Actin Filament Polymerisation And Amylase Secretion In Mouse Pancreatic Acinar Cells
Xod-Catalyzed Ros Generation Mobilizes Calcium From Intracellular Stores In Mouse Pancreatic Acinar Cells
Effects Of Reactive Oxygen Species On Actin Filament Polymerisation And Amylase Secretion In Mouse Pancreatic Acinar Cells
Xod-Catalyzed Ros Generation Mobilizes Calcium From Intracellular Stores In Mouse Pancreatic Acinar Cells
Arachidonic Acid Modulates The Spatiotemporal Characteristics Of Agonist-Evoked Ca2+ Waves In Mouse Pancreatic Acinar Cells
Xod-Catalyzed Ros Generation Mobilizes Calcium From Intracellular Stores In Mouse Pancreatic Acinar Cells
Arachidonic Acid Modulates The Spatiotemporal Characteristics Of Agonist-Evoked Ca2+ Waves In Mouse Pancreatic Acinar Cells
Participation Of Mitochondria In Calcium Signalling In The Exocrine Pancreas
Arachidonic Acid Modulates The Spatiotemporal Characteristics Of Agonist-Evoked Ca2+ Waves In Mouse Pancreatic Acinar Cells
Participation Of Mitochondria In Calcium Signalling In The Exocrine Pancreas
Effect Of Xanthine Oxidase-Catalyzed Reactive Oxygen Species Generation On Secretagogue-Evoked Calcium Mobilization In Mouse Pancreatic Acinar Cells
Participation Of Mitochondria In Calcium Signalling In The Exocrine Pancreas
Effect Of Xanthine Oxidase-Catalyzed Reactive Oxygen Species Generation On Secretagogue-Evoked Calcium Mobilization In Mouse Pancreatic Acinar Cells
Agonist-Evoked Mitochondrial Ca2+ Signals In Mouse Pancreatic Acinar Cells
Effect Of Xanthine Oxidase-Catalyzed Reactive Oxygen Species Generation On Secretagogue-Evoked Calcium Mobilization In Mouse Pancreatic Acinar Cells
Agonist-Evoked Mitochondrial Ca2+ Signals In Mouse Pancreatic Acinar Cells
Low Cytoplasmic [Ca2+] Activates I(Crac) Independently Of Global Ca2+ Store Depletion In Rbl-1 Cells
Agonist-Evoked Mitochondrial Ca2+ Signals In Mouse Pancreatic Acinar Cells
Low Cytoplasmic [Ca2+] Activates I(Crac) Independently Of Global Ca2+ Store Depletion In Rbl-1 Cells
Cholecystokinin-Evoked Ca2+ Waves In Isolated Mouse Pancreatic Acinar Cells Are Modulated By Activation Of Cytosolic Phospholipase A2, Phospholipase D, And Protein Kinase C
Low Cytoplasmic [Ca2+] Activates I(Crac) Independently Of Global Ca2+ Store Depletion In Rbl-1 Cells
Cholecystokinin-Evoked Ca2+ Waves In Isolated Mouse Pancreatic Acinar Cells Are Modulated By Activation Of Cytosolic Phospholipase A2, Phospholipase D, And Protein Kinase C
Agonist-Stimulated Pathways Of Calcium Signaling In Pancreatic Acinar Cells
Cholecystokinin-Evoked Ca2+ Waves In Isolated Mouse Pancreatic Acinar Cells Are Modulated By Activation Of Cytosolic Phospholipase A2, Phospholipase D, And Protein Kinase C
Agonist-Stimulated Pathways Of Calcium Signaling In Pancreatic Acinar Cells
Ingesta De Calcio En Escolares De Badajoz
Agonist-Stimulated Pathways Of Calcium Signaling In Pancreatic Acinar Cells
Ingesta De Calcio En Escolares De Badajoz
Effect Of Intracellular Ph On Acetylcholine-Induced Ca2+ Waves In Mouse Pancreatic Acinar Cells
Ingesta De Calcio En Escolares De Badajoz
Effect Of Intracellular Ph On Acetylcholine-Induced Ca2+ Waves In Mouse Pancreatic Acinar Cells
Histamine Does Not Potentiate Cyclic Amp-Mediated Amylase Secretion In The Guinea-Pig Pancreatic Acinar Cells
Effect Of Intracellular Ph On Acetylcholine-Induced Ca2+ Waves In Mouse Pancreatic Acinar Cells
Histamine Does Not Potentiate Cyclic Amp-Mediated Amylase Secretion In The Guinea-Pig Pancreatic Acinar Cells
Trypsinogen Secretion From The Isolated Guinea-Pig Pancreas In Response To Secretagogues
Histamine Does Not Potentiate Cyclic Amp-Mediated Amylase Secretion In The Guinea-Pig Pancreatic Acinar Cells
Trypsinogen Secretion From The Isolated Guinea-Pig Pancreas In Response To Secretagogues
Free Cytosolic Calcium Levels Modify Intracellular Ph In Rat Pancreatic Acini
Trypsinogen Secretion From The Isolated Guinea-Pig Pancreas In Response To Secretagogues
Free Cytosolic Calcium Levels Modify Intracellular Ph In Rat Pancreatic Acini
Intracellular Ph And Calcium Signalling In Rat Pancreatic Acinar Cells
Free Cytosolic Calcium Levels Modify Intracellular Ph In Rat Pancreatic Acini
Intracellular Ph And Calcium Signalling In Rat Pancreatic Acinar Cells
Cholecystokinin-Stimulated Tyrosine Phosphorylation Of P125fak And Paxillin Is Mediated By Phospholipase C-Dependent And -Independent Mechanisms And Requires The Integrity Of The Actin Cytoskeleton And Participation Of P21rho
Intracellular Ph And Calcium Signalling In Rat Pancreatic Acinar Cells
Cholecystokinin-Stimulated Tyrosine Phosphorylation Of P125fak And Paxillin Is Mediated By Phospholipase C-Dependent And -Independent Mechanisms And Requires The Integrity Of The Actin Cytoskeleton And Participation Of P21rho
Stimulus-Secretion Pathway For Histamine In The Exocrine Pancreas
Cholecystokinin-Stimulated Tyrosine Phosphorylation Of P125fak And Paxillin Is Mediated By Phospholipase C-Dependent And -Independent Mechanisms And Requires The Integrity Of The Actin Cytoskeleton And Participation Of P21rho
Stimulus-Secretion Pathway For Histamine In The Exocrine Pancreas
Impairment Of Intracellular Calcium Homoeostasis In The Exocrine Pancreas After Caerulein-Induced Acute Pancreatitis In The Rat.
Stimulus-Secretion Pathway For Histamine In The Exocrine Pancreas
Impairment Of Intracellular Calcium Homoeostasis In The Exocrine Pancreas After Caerulein-Induced Acute Pancreatitis In The Rat.
Impairment Of Intracellular Calcium Homoeostasis In The Exocrine Pancreas After Caerulein-Induced Acute Pancreatitis In The Rat
Impairment Of Intracellular Calcium Homoeostasis In The Exocrine Pancreas After Caerulein-Induced Acute Pancreatitis In The Rat.
Impairment Of Intracellular Calcium Homoeostasis In The Exocrine Pancreas After Caerulein-Induced Acute Pancreatitis In The Rat
Reciprocal Changes In Intracellular And Extracellular Magnesium In Rat Pancreatic Acinar Cells In Response To Different Secretagogues.
Impairment Of Intracellular Calcium Homoeostasis In The Exocrine Pancreas After Caerulein-Induced Acute Pancreatitis In The Rat
Reciprocal Changes In Intracellular And Extracellular Magnesium In Rat Pancreatic Acinar Cells In Response To Different Secretagogues.
Effects Of Phorbol Esters And Secretin On Acetylcholine-Evoked Exocrine Pancreatic Secretion In The Anaesthetized Rat
Reciprocal Changes In Intracellular And Extracellular Magnesium In Rat Pancreatic Acinar Cells In Response To Different Secretagogues.
Effects Of Phorbol Esters And Secretin On Acetylcholine-Evoked Exocrine Pancreatic Secretion In The Anaesthetized Rat
Two Sesquiterpene Lactones From Centaurea Canariensis
Effects Of Phorbol Esters And Secretin On Acetylcholine-Evoked Exocrine Pancreatic Secretion In The Anaesthetized Rat
Two Sesquiterpene Lactones From Centaurea Canariensis
Melatonin Interplay In Physiology And Disease—the Fountain Of Eternal Youth Revisited
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