PUBLICACIÓN

Overexpression of latent transforming growth factor-β binding protein 1 (LTBP-1) in dioxin receptor-null mouse embryo fibroblasts

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ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Santiago-Josefat, B, Mulero-Navarro, S, Dallas, SL, Fernandez-Salguero, PM

2004 Journal of Cell Science

CITAS

46

DOI

10.1242/jcs.00932

EID

2-s2.0-1642279296

ISSN

0021-9533

BIBTEX

@article{RID:0916141122194-30, title = {Overexpression of latent transforming growth factor-beta binding protein 1 (LTBP-1) in dioxin receptor-null mouse embryo fibroblasts}, journal = {Journal of Cell Science}, year = {2004}, author = {Santiago-Josefat, B and Mulero-Navarro, S and Dallas, SL and Fernandez-Salguero, PM}, volume = {117}, number = {6}, pages = {849-859} }

AUTORES DE LA UEX

Pedro María Fernández Salguero
Sonia María Mulero Navarro

OTRAS PUBLICACIONES DE INTERÉS

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Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Thioridazine Steady-State Plasma Concentrations Are Influenced By Tobacco Smoking And Cyp2d6, But Not By The Cyp2c9 Genotype
Carcinogenesis Of The Food Mutagen Phip In Mice Is Dependent Of Cyp1a2
Thioridazine Steady-State Plasma Concentrations Are Influenced By Tobacco Smoking And Cyp2d6, But Not By The Cyp2c9 Genotype
Carcinogenesis Of The Food Mutagen Phip In Mice Is Dependent Of Cyp1a2
Polycyclic Aromatic Hydrocarbon-Inducible Dna Adducts: Evidence By 32p-Postlabeling And Use Of Knockout Mice For Ah Receptor-Independent Mechanisms Of Metabolic Activation In Vivo
Carcinogenesis Of The Food Mutagen Phip In Mice Is Dependent Of Cyp1a2
Polycyclic Aromatic Hydrocarbon-Inducible Dna Adducts: Evidence By 32p-Postlabeling And Use Of Knockout Mice For Ah Receptor-Independent Mechanisms Of Metabolic Activation In Vivo
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
Polycyclic Aromatic Hydrocarbon-Inducible Dna Adducts: Evidence By 32p-Postlabeling And Use Of Knockout Mice For Ah Receptor-Independent Mechanisms Of Metabolic Activation In Vivo
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
Effect Of Thioridazine Dosage On The Debrisoquine Hydroxylation Phenotype In Psychiatric Patients With Different Cyp2d6 Genotypes
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
Effect Of Thioridazine Dosage On The Debrisoquine Hydroxylation Phenotype In Psychiatric Patients With Different Cyp2d6 Genotypes
Targeted Genomic Disruption Of H-Ras And N-Ras, Individually Or In Combination, Reveals The Dispensability Of Both Loci For Mouse Growth And Development
Effect Of Thioridazine Dosage On The Debrisoquine Hydroxylation Phenotype In Psychiatric Patients With Different Cyp2d6 Genotypes
Targeted Genomic Disruption Of H-Ras And N-Ras, Individually Or In Combination, Reveals The Dispensability Of Both Loci For Mouse Growth And Development
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
Targeted Genomic Disruption Of H-Ras And N-Ras, Individually Or In Combination, Reveals The Dispensability Of Both Loci For Mouse Growth And Development
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
Altered Cell Cycle Control At The G2/m Phases In Aryl Hydrocarbon Receptor-Null Embryo Fibroblast
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
Altered Cell Cycle Control At The G2/m Phases In Aryl Hydrocarbon Receptor-Null Embryo Fibroblast
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Altered Cell Cycle Control At The G2/m Phases In Aryl Hydrocarbon Receptor-Null Embryo Fibroblast
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Hepatic Fibrosis And Cytochrome P450: Experimental Models Of Fibrosis Compared To Ahr Knockout Mice
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Hepatic Fibrosis And Cytochrome P450: Experimental Models Of Fibrosis Compared To Ahr Knockout Mice
Genomic Instability In Gadd45a-Deficient Mice
Hepatic Fibrosis And Cytochrome P450: Experimental Models Of Fibrosis Compared To Ahr Knockout Mice
Genomic Instability In Gadd45a-Deficient Mice
Cyp1a2 Is Not The Primary Enzyme Responsible For 4-Aminobiphenyl-Induced Hepatocarcinogenesis In Mice
Genomic Instability In Gadd45a-Deficient Mice
Cyp1a2 Is Not The Primary Enzyme Responsible For 4-Aminobiphenyl-Induced Hepatocarcinogenesis In Mice
Targeted Disruption Of The Microsomal Epoxide Hydrolase Gene. Microsomal Epoxide Hydrolase Is Required For The Carcinogenic Activity Of 7,12- Dimethylbenz[A]anthracene
Cyp1a2 Is Not The Primary Enzyme Responsible For 4-Aminobiphenyl-Induced Hepatocarcinogenesis In Mice
Targeted Disruption Of The Microsomal Epoxide Hydrolase Gene. Microsomal Epoxide Hydrolase Is Required For The Carcinogenic Activity Of 7,12- Dimethylbenz[A]anthracene
Expression Of Cyp2a Genes In Human Liver And Extrahepatic Tissues
Targeted Disruption Of The Microsomal Epoxide Hydrolase Gene. Microsomal Epoxide Hydrolase Is Required For The Carcinogenic Activity Of 7,12- Dimethylbenz[A]anthracene
Expression Of Cyp2a Genes In Human Liver And Extrahepatic Tissues
Amelioration Of Tcdd-Induced Teratogenesis In Aryl Hydrocarbon Receptor (Ahr)-Null Mice
Expression Of Cyp2a Genes In Human Liver And Extrahepatic Tissues
Amelioration Of Tcdd-Induced Teratogenesis In Aryl Hydrocarbon Receptor (Ahr)-Null Mice
The Aryl Hydrocarbon Receptor. Studies Using The Ahr-Null Mice
Amelioration Of Tcdd-Induced Teratogenesis In Aryl Hydrocarbon Receptor (Ahr)-Null Mice
The Aryl Hydrocarbon Receptor. Studies Using The Ahr-Null Mice
Aryl-Hydrocarbon Receptor Is An Inhibitory Regulator Of Lipid Synthesis And Of Commitment To Adipogenesis
The Aryl Hydrocarbon Receptor. Studies Using The Ahr-Null Mice
Aryl-Hydrocarbon Receptor Is An Inhibitory Regulator Of Lipid Synthesis And Of Commitment To Adipogenesis
Characterization Of The Human Dihydropyrimidine Dehydrogenase Gene
Aryl-Hydrocarbon Receptor Is An Inhibitory Regulator Of Lipid Synthesis And Of Commitment To Adipogenesis
Characterization Of The Human Dihydropyrimidine Dehydrogenase Gene
Effect Of Phenobarbital On Hepatic Cyp1a1 And Cyp1a2 In The Ahr-Null Mouse
Characterization Of The Human Dihydropyrimidine Dehydrogenase Gene
Effect Of Phenobarbital On Hepatic Cyp1a1 And Cyp1a2 In The Ahr-Null Mouse
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Caucasian Subjects
Effect Of Phenobarbital On Hepatic Cyp1a1 And Cyp1a2 In The Ahr-Null Mouse
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Caucasian Subjects
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Patients With Colorectal Cancer
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Caucasian Subjects
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Patients With Colorectal Cancer
Nomenclature For Human Dpyd Alleles
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Patients With Colorectal Cancer
Nomenclature For Human Dpyd Alleles
The Involvement Of Aryl Hydrocarbon Receptor In The Activation Of Transforming Growth Factor-Β And Apoptosis
Nomenclature For Human Dpyd Alleles
The Involvement Of Aryl Hydrocarbon Receptor In The Activation Of Transforming Growth Factor-Β And Apoptosis
Aryl Hydrocarbon Receptor Deficient Mice (Ahr-/-) Exhibit An Accumulation Of Liver Retinylpalmitate
The Involvement Of Aryl Hydrocarbon Receptor In The Activation Of Transforming Growth Factor-Β And Apoptosis
Aryl Hydrocarbon Receptor Deficient Mice (Ahr-/-) Exhibit An Accumulation Of Liver Retinylpalmitate
Analysis Of The Selenocysteine (Sec) Trna Population In Mouse Embryonic Stem (Es) Cells Heterozygous For The Sec Trna Gene
Aryl Hydrocarbon Receptor Deficient Mice (Ahr-/-) Exhibit An Accumulation Of Liver Retinylpalmitate
Analysis Of The Selenocysteine (Sec) Trna Population In Mouse Embryonic Stem (Es) Cells Heterozygous For The Sec Trna Gene
Aryl Hydrocarbon Receptor Knockout Mice (Ahr(-/-)) Exhibit Liver Retinoid Accumulation And Reduced Retinoic Acid Metabolism
Analysis Of The Selenocysteine (Sec) Trna Population In Mouse Embryonic Stem (Es) Cells Heterozygous For The Sec Trna Gene
Aryl Hydrocarbon Receptor Knockout Mice (Ahr(-/-)) Exhibit Liver Retinoid Accumulation And Reduced Retinoic Acid Metabolism
Cyp2a6 Gene Polymorphism And Risk Of Liver Cancer And Cirrhosis
Aryl Hydrocarbon Receptor Knockout Mice (Ahr(-/-)) Exhibit Liver Retinoid Accumulation And Reduced Retinoic Acid Metabolism
Cyp2a6 Gene Polymorphism And Risk Of Liver Cancer And Cirrhosis
Selenocysteine Trna([Ser]sec) Levels And Selenium-Dependent Glutathione Peroxidase Activity In Mouse Embryonic Stem Cells Heterozygous For A Targeted Mutation In The Trna([Ser]sec) Gene
Cyp2a6 Gene Polymorphism And Risk Of Liver Cancer And Cirrhosis
Selenocysteine Trna([Ser]sec) Levels And Selenium-Dependent Glutathione Peroxidase Activity In Mouse Embryonic Stem Cells Heterozygous For A Targeted Mutation In The Trna([Ser]sec) Gene
Lack Of Correlation Between Phenotype And Genotype For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In A Family Of Pakistani Origin
Selenocysteine Trna([Ser]sec) Levels And Selenium-Dependent Glutathione Peroxidase Activity In Mouse Embryonic Stem Cells Heterozygous For A Targeted Mutation In The Trna([Ser]sec) Gene
Lack Of Correlation Between Phenotype And Genotype For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In A Family Of Pakistani Origin
Lesions Of Aryl-Hydrocarbon Receptor-Deficient Mice
Lack Of Correlation Between Phenotype And Genotype For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In A Family Of Pakistani Origin
Lesions Of Aryl-Hydrocarbon Receptor-Deficient Mice
Mutations At Codon 974 Of The Dpyd Gene Are A Rare Event
Lesions Of Aryl-Hydrocarbon Receptor-Deficient Mice
Mutations At Codon 974 Of The Dpyd Gene Are A Rare Event
Piperonyl Butoxide And Acenaphthylene Induce Cytochrome P450 1a2 And 1b1 Mrna In Aromatic Hydrocarbon-Responsive Receptor Knock-Out Mouse Liver
Mutations At Codon 974 Of The Dpyd Gene Are A Rare Event
Piperonyl Butoxide And Acenaphthylene Induce Cytochrome P450 1a2 And 1b1 Mrna In Aromatic Hydrocarbon-Responsive Receptor Knock-Out Mouse Liver
Transcriptional Induction Of The Cytochrome P4501a1 Gene By A Thiazolium Compound, Yh439
Piperonyl Butoxide And Acenaphthylene Induce Cytochrome P450 1a2 And 1b1 Mrna In Aromatic Hydrocarbon-Responsive Receptor Knock-Out Mouse Liver
Transcriptional Induction Of The Cytochrome P4501a1 Gene By A Thiazolium Compound, Yh439
The Role Of The Aryl Hydrocarbon Receptor In Animal Development, Physiological Homeostasis And Toxicity Of Tcdd
Transcriptional Induction Of The Cytochrome P4501a1 Gene By A Thiazolium Compound, Yh439
The Role Of The Aryl Hydrocarbon Receptor In Animal Development, Physiological Homeostasis And Toxicity Of Tcdd
Differential Regulation Of Mouse Ah Receptor Gene Expression In Cell Lines Of Different Tissue Origins
The Role Of The Aryl Hydrocarbon Receptor In Animal Development, Physiological Homeostasis And Toxicity Of Tcdd
Differential Regulation Of Mouse Ah Receptor Gene Expression In Cell Lines Of Different Tissue Origins
Molecular Basis Of The Human Dihydropyrimidine Dehydrogenase Deficiency And 5-Fluorouracil Toxicity
Differential Regulation Of Mouse Ah Receptor Gene Expression In Cell Lines Of Different Tissue Origins
Molecular Basis Of The Human Dihydropyrimidine Dehydrogenase Deficiency And 5-Fluorouracil Toxicity
Role Of Cyp2e1 In The Hepatotoxicity Of Acetaminophen
Molecular Basis Of The Human Dihydropyrimidine Dehydrogenase Deficiency And 5-Fluorouracil Toxicity
Role Of Cyp2e1 In The Hepatotoxicity Of Acetaminophen
Aryl-Hydrocarbon Receptor-Deficient Mice Are Resistant To 2,3,7,8-Tetrachlorodibenzo-P-Dioxin-Induced Toxicity
Role Of Cyp2e1 In The Hepatotoxicity Of Acetaminophen
Aryl-Hydrocarbon Receptor-Deficient Mice Are Resistant To 2,3,7,8-Tetrachlorodibenzo-P-Dioxin-Induced Toxicity
Hepatic Fibrosis In Ahr(-/-) Mice
Aryl-Hydrocarbon Receptor-Deficient Mice Are Resistant To 2,3,7,8-Tetrachlorodibenzo-P-Dioxin-Induced Toxicity
Hepatic Fibrosis In Ahr(-/-) Mice
The T/ebp Null Mouse: Thyroid-Specific Enhancer-Binding Protein Is Essential For The Organogenesis Of The Thyroid, Lung, Ventral Forebrain, And Pituitary
Hepatic Fibrosis In Ahr(-/-) Mice
The T/ebp Null Mouse: Thyroid-Specific Enhancer-Binding Protein Is Essential For The Organogenesis Of The Thyroid, Lung, Ventral Forebrain, And Pituitary
Pyrimidines And Cns Regulation
The T/ebp Null Mouse: Thyroid-Specific Enhancer-Binding Protein Is Essential For The Organogenesis Of The Thyroid, Lung, Ventral Forebrain, And Pituitary
Pyrimidines And Cns Regulation
Targeted Disruption Of Specific Cytochromes P450 And Xenobiotic Receptor Genes
Pyrimidines And Cns Regulation
Targeted Disruption Of Specific Cytochromes P450 And Xenobiotic Receptor Genes
Diagnostic-Analysis, Clinical Importance And Molecular-Basis Of Dihydropyrimidine Dehydrogenase-Deficiency
Targeted Disruption Of Specific Cytochromes P450 And Xenobiotic Receptor Genes
Diagnostic-Analysis, Clinical Importance And Molecular-Basis Of Dihydropyrimidine Dehydrogenase-Deficiency
Organization And Evolution Of The Cytochrome P450 Cyp2a-2b-2f Subfamily Gene Cluster On Human Chromosome 19
Diagnostic-Analysis, Clinical Importance And Molecular-Basis Of Dihydropyrimidine Dehydrogenase-Deficiency
Organization And Evolution Of The Cytochrome P450 Cyp2a-2b-2f Subfamily Gene Cluster On Human Chromosome 19
A Genetic Polymorphism In Coumarin 7-Hydroxylation: Sequence Of The Human Cyp2a Genes And Identification Of Variant Cyp2a6 Alleles
Organization And Evolution Of The Cytochrome P450 Cyp2a-2b-2f Subfamily Gene Cluster On Human Chromosome 19
A Genetic Polymorphism In Coumarin 7-Hydroxylation: Sequence Of The Human Cyp2a Genes And Identification Of Variant Cyp2a6 Alleles
Comparison Of Substrate Metabolism By Wild Type Cyp2d6 Protein And A Variant Containing Methionine, Not Valine, At Position 374
A Genetic Polymorphism In Coumarin 7-Hydroxylation: Sequence Of The Human Cyp2a Genes And Identification Of Variant Cyp2a6 Alleles
Comparison Of Substrate Metabolism By Wild Type Cyp2d6 Protein And A Variant Containing Methionine, Not Valine, At Position 374
Human Polymorphism In Drug Metabolism: Mutation In The Dihydropyrimidine Dehydrogenase Gene Results In Exon Skipping And Thymine Uracilurea
Comparison Of Substrate Metabolism By Wild Type Cyp2d6 Protein And A Variant Containing Methionine, Not Valine, At Position 374
Human Polymorphism In Drug Metabolism: Mutation In The Dihydropyrimidine Dehydrogenase Gene Results In Exon Skipping And Thymine Uracilurea
Genetic Polymorphism Of Cyp2c9 And Its Effect On Warfarin Maintenance Dose Requirement In Patients Undergoing Anticoagulation Therapy
Human Polymorphism In Drug Metabolism: Mutation In The Dihydropyrimidine Dehydrogenase Gene Results In Exon Skipping And Thymine Uracilurea
Genetic Polymorphism Of Cyp2c9 And Its Effect On Warfarin Maintenance Dose Requirement In Patients Undergoing Anticoagulation Therapy
Correlation Between Catalytic Activity And Protein Content For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In Human Lymphocytes
Genetic Polymorphism Of Cyp2c9 And Its Effect On Warfarin Maintenance Dose Requirement In Patients Undergoing Anticoagulation Therapy
Correlation Between Catalytic Activity And Protein Content For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In Human Lymphocytes
Neonatal Lethality Associated With Respiratory Distress In Mice Lacking Cytochrome P450 1a2
Correlation Between Catalytic Activity And Protein Content For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In Human Lymphocytes
Neonatal Lethality Associated With Respiratory Distress In Mice Lacking Cytochrome P450 1a2
Xenobiotic Receptor Knockout Mice
Neonatal Lethality Associated With Respiratory Distress In Mice Lacking Cytochrome P450 1a2
Xenobiotic Receptor Knockout Mice
Immune System Impairment And Hepatic Fibrosis In Mice Lacking The Dioxin-Binding Ah Receptor
Xenobiotic Receptor Knockout Mice
Immune System Impairment And Hepatic Fibrosis In Mice Lacking The Dioxin-Binding Ah Receptor
Targeted Disruption Of The Α Isoform Of The Peroxisome Proliferator- Activated Receptor Gene In Mice Results In Abolishment Of The Pleiotropic Effects Of Peroxisome Proliferators
Immune System Impairment And Hepatic Fibrosis In Mice Lacking The Dioxin-Binding Ah Receptor
Targeted Disruption Of The Α Isoform Of The Peroxisome Proliferator- Activated Receptor Gene In Mice Results In Abolishment Of The Pleiotropic Effects Of Peroxisome Proliferators
Cdna Cloning And Chromosome Mapping Of Human Dihydropyrimidine Dehydrogenase, An Enzyme Associated With 5-Fluorouracil Toxicity And Congenital Thymine Uraciluria
Targeted Disruption Of The Α Isoform Of The Peroxisome Proliferator- Activated Receptor Gene In Mice Results In Abolishment Of The Pleiotropic Effects Of Peroxisome Proliferators
Cdna Cloning And Chromosome Mapping Of Human Dihydropyrimidine Dehydrogenase, An Enzyme Associated With 5-Fluorouracil Toxicity And Congenital Thymine Uraciluria
Assignment Of The Human Dihydropyrimidine Dehydrogenase Gene (Dpyd) To Chromosome Region 1p22 By Fluorescence In Situ Hybridization
Cdna Cloning And Chromosome Mapping Of Human Dihydropyrimidine Dehydrogenase, An Enzyme Associated With 5-Fluorouracil Toxicity And Congenital Thymine Uraciluria
Assignment Of The Human Dihydropyrimidine Dehydrogenase Gene (Dpyd) To Chromosome Region 1p22 By Fluorescence In Situ Hybridization
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Assignment Of The Human Dihydropyrimidine Dehydrogenase Gene (Dpyd) To Chromosome Region 1p22 By Fluorescence In Situ Hybridization
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment
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