PUBLICACIÓN

A mesenchymal-like phenotype and expression of CD44 predict lack of apoptotic response to sorafenib in liver tumor cells

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ACCEDER A LA PUBLICACIÓN: Scopus Orcid

2015 INTERNATIONAL JOURNAL OF CANCER

Cancer Research (Q1), Oncology (Q1)

JCR: 5.531

SJR: 2.687

CITAS

107

DOI

10.1002/ijc.29097

EID

2-s2.0-84918815230

ISSN

0020-7136

EISSN

1097-0215

AUTORES DE LA UEX

Pedro María Fernández Salguero
Alberto Manuel Álvarez Barrientos

OTRAS PUBLICACIONES DE INTERÉS

Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Neurotoxic Amyloid Β‐peptide And Tau Produce Cytokine‐like Effects On Pmca In Glioblastoma Cell Lines, Enhancing Its Activity And Isoforms Expression
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment
Neurotoxic Amyloid Β‐peptide And Tau Produce Cytokine‐like Effects On Pmca In Glioblastoma Cell Lines, Enhancing Its Activity And Isoforms Expression
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment
Embryo-Induced Changes In The Protein Profile Of Bovine Oviductal Extracellular Vesicles
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment
Embryo-Induced Changes In The Protein Profile Of Bovine Oviductal Extracellular Vesicles
Deciphering The Dialogue Between The Bovine Blastocyst And The Uterus: Embryo-Induced Alterations In Extracellular Vesicle Protein Content From An Ex Vivo Model And The In Vivo Environment
Embryo-Induced Changes In The Protein Profile Of Bovine Oviductal Extracellular Vesicles
Deciphering The Dialogue Between The Bovine Blastocyst And The Uterus: Embryo-Induced Alterations In Extracellular Vesicle Protein Content From An Ex Vivo Model And The In Vivo Environment
Immunophenotype Profile By Flow Cytometry Reveals Different Subtypes Of Extracellular Vesicles In Porcine Seminal Plasma
Deciphering The Dialogue Between The Bovine Blastocyst And The Uterus: Embryo-Induced Alterations In Extracellular Vesicle Protein Content From An Ex Vivo Model And The In Vivo Environment
Immunophenotype Profile By Flow Cytometry Reveals Different Subtypes Of Extracellular Vesicles In Porcine Seminal Plasma
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
Immunophenotype Profile By Flow Cytometry Reveals Different Subtypes Of Extracellular Vesicles In Porcine Seminal Plasma
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
Editorial: Go With The Vet-Flow! The Current Uses And New Frontiers Of Flow Cytometry In Veterinary Sciences
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
Editorial: Go With The Vet-Flow! The Current Uses And New Frontiers Of Flow Cytometry In Veterinary Sciences
Introducción: Carta Del Rector De La Uex
Editorial: Go With The Vet-Flow! The Current Uses And New Frontiers Of Flow Cytometry In Veterinary Sciences
Introducción: Carta Del Rector De La Uex
The Ah Receptor From Toxicity To Therapeutics: Report From The 5th Ahr Meeting At Penn State University, Usa, June 2022
Introducción: Carta Del Rector De La Uex
The Ah Receptor From Toxicity To Therapeutics: Report From The 5th Ahr Meeting At Penn State University, Usa, June 2022
The Proteome Of Large Or Small Extracellular Vesicles In Pig Seminal Plasma Differs, Defining Sources And Biological Functions
The Ah Receptor From Toxicity To Therapeutics: Report From The 5th Ahr Meeting At Penn State University, Usa, June 2022
The Proteome Of Large Or Small Extracellular Vesicles In Pig Seminal Plasma Differs, Defining Sources And Biological Functions
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
The Proteome Of Large Or Small Extracellular Vesicles In Pig Seminal Plasma Differs, Defining Sources And Biological Functions
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function
Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function
Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor
Obesity Affects Β2 Adrenergic Regulation Of The Inflammatory Profile And Phenotype Of Circulating Monocytes From Exercised Animals
Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor
Obesity Affects Β2 Adrenergic Regulation Of The Inflammatory Profile And Phenotype Of Circulating Monocytes From Exercised Animals
Anti-Inflammatory Effect Of Β2 Adrenergic Stimulation On Circulating Monocytes With A Pro-Inflammatory State In High-Fat Diet-Induced Obesity
Obesity Affects Β2 Adrenergic Regulation Of The Inflammatory Profile And Phenotype Of Circulating Monocytes From Exercised Animals
Anti-Inflammatory Effect Of Β2 Adrenergic Stimulation On Circulating Monocytes With A Pro-Inflammatory State In High-Fat Diet-Induced Obesity
Transcriptome Analysis Reveals That Fertilization With Cryopreserved Sperm Downregulates Genes Relevant For Early Embryo Development In The Horse
Anti-Inflammatory Effect Of Β2 Adrenergic Stimulation On Circulating Monocytes With A Pro-Inflammatory State In High-Fat Diet-Induced Obesity
Transcriptome Analysis Reveals That Fertilization With Cryopreserved Sperm Downregulates Genes Relevant For Early Embryo Development In The Horse
Lack Of The Aryl Hydrocarbon Receptor Accelerates Aging In Mice
Transcriptome Analysis Reveals That Fertilization With Cryopreserved Sperm Downregulates Genes Relevant For Early Embryo Development In The Horse
Lack Of The Aryl Hydrocarbon Receptor Accelerates Aging In Mice
Vav Proteins Maintain Epithelial Traits In Breast Cancer Cells Using Mir-200c-Dependent And Independent Mechanisms
Lack Of The Aryl Hydrocarbon Receptor Accelerates Aging In Mice
Vav Proteins Maintain Epithelial Traits In Breast Cancer Cells Using Mir-200c-Dependent And Independent Mechanisms
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Vav Proteins Maintain Epithelial Traits In Breast Cancer Cells Using Mir-200c-Dependent And Independent Mechanisms
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Extracellular Vesicles Isolated From Porcine Seminal Plasma Exhibit Different Tetraspanin Expression Profiles
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Extracellular Vesicles Isolated From Porcine Seminal Plasma Exhibit Different Tetraspanin Expression Profiles
Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science Doi: 10.1242/jcs.02878)
Extracellular Vesicles Isolated From Porcine Seminal Plasma Exhibit Different Tetraspanin Expression Profiles
Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science Doi: 10.1242/jcs.02878)
Erratum: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains (Journal Of Cell Science (2002) 115 (3119-3130) Doi: 10.1242/jcs.209973)
Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science Doi: 10.1242/jcs.02878)
Erratum: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains (Journal Of Cell Science (2002) 115 (3119-3130) Doi: 10.1242/jcs.209973)
Correction: Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels (Doi:10.1242/jcs.01002).
Erratum: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains (Journal Of Cell Science (2002) 115 (3119-3130) Doi: 10.1242/jcs.209973)
Correction: Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels (Doi:10.1242/jcs.01002).
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Correction: Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels (Doi:10.1242/jcs.01002).
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Variation In Malaria Infection And Immune Defence In Invasive And Endemic House Sparrows
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Variation In Malaria Infection And Immune Defence In Invasive And Endemic House Sparrows
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
Variation In Malaria Infection And Immune Defence In Invasive And Endemic House Sparrows
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value
Identification Of New Activators Of Mitochondrial Fusion Reveals A Link Between Mitochondrial Morphology And Pyrimidine Metabolism
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value
Identification Of New Activators Of Mitochondrial Fusion Reveals A Link Between Mitochondrial Morphology And Pyrimidine Metabolism
Histone H4 Acetylation Regulates Behavioral Inter-Individual Variability In Zebrafish
Identification Of New Activators Of Mitochondrial Fusion Reveals A Link Between Mitochondrial Morphology And Pyrimidine Metabolism
Histone H4 Acetylation Regulates Behavioral Inter-Individual Variability In Zebrafish
Centriole Planar Polarity Assessment In Drosophila Wings
Histone H4 Acetylation Regulates Behavioral Inter-Individual Variability In Zebrafish
Centriole Planar Polarity Assessment In Drosophila Wings
Immune-Neuroendocrine Dysregulation In Patients With Osteoarthritis: A Revision And A Pilot Study
Centriole Planar Polarity Assessment In Drosophila Wings
Immune-Neuroendocrine Dysregulation In Patients With Osteoarthritis: A Revision And A Pilot Study
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Immune-Neuroendocrine Dysregulation In Patients With Osteoarthritis: A Revision And A Pilot Study
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis
A Dioxin-Like Compound Induces Hyperplasia And Branching Morphogenesis In Mouse Mammary Gland, Through Alterations In Tgf-Β1 And Aryl Hydrocarbon Receptor Signaling
Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis
A Dioxin-Like Compound Induces Hyperplasia And Branching Morphogenesis In Mouse Mammary Gland, Through Alterations In Tgf-Β1 And Aryl Hydrocarbon Receptor Signaling
Expression Of Concern: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways. J. Cell Sci. Doi: 10.1242/jcs.02878
A Dioxin-Like Compound Induces Hyperplasia And Branching Morphogenesis In Mouse Mammary Gland, Through Alterations In Tgf-Β1 And Aryl Hydrocarbon Receptor Signaling
Expression Of Concern: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways. J. Cell Sci. Doi: 10.1242/jcs.02878
Expression Of Concern: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains. J. Cell Sci. (2002). 115, 3119-3130.
Expression Of Concern: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways. J. Cell Sci. Doi: 10.1242/jcs.02878
Expression Of Concern: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains. J. Cell Sci. (2002). 115, 3119-3130.
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
Expression Of Concern: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains. J. Cell Sci. (2002). 115, 3119-3130.
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis
New Trends In Aryl Hydrocarbon Receptor Biology
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis
New Trends In Aryl Hydrocarbon Receptor Biology
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements
New Trends In Aryl Hydrocarbon Receptor Biology
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis.
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis.
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor.
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis.
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor.
New Trends In Aryl Hydrocarbon Receptor Biology.
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor.
New Trends In Aryl Hydrocarbon Receptor Biology.
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements.
New Trends In Aryl Hydrocarbon Receptor Biology.
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements.
Skin Response To A Carcinogen Involves The Xenobiotic Receptor Pregnane X Receptor
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements.
Skin Response To A Carcinogen Involves The Xenobiotic Receptor Pregnane X Receptor
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
Skin Response To A Carcinogen Involves The Xenobiotic Receptor Pregnane X Receptor
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells
Bmi1 Regulates Murine Intestinal Stem Cell Proliferation And Self-Renewal Downstream Of Notch
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells
Bmi1 Regulates Murine Intestinal Stem Cell Proliferation And Self-Renewal Downstream Of Notch
Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg
Bmi1 Regulates Murine Intestinal Stem Cell Proliferation And Self-Renewal Downstream Of Notch
Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg
Erratum To Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans [Fungal Genet. Biol. 74 (2015) 10-20] Doi: 10.1016/j.fgb.2014.10.016
Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg
Erratum To Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans [Fungal Genet. Biol. 74 (2015) 10-20] Doi: 10.1016/j.fgb.2014.10.016
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans
Erratum To Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans [Fungal Genet. Biol. 74 (2015) 10-20] Doi: 10.1016/j.fgb.2014.10.016
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans
L-Kynurenine/aryl Hydrocarbon Receptor Pathway Mediates Brain Damage After Experimental Stroke
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans
L-Kynurenine/aryl Hydrocarbon Receptor Pathway Mediates Brain Damage After Experimental Stroke
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol
L-Kynurenine/aryl Hydrocarbon Receptor Pathway Mediates Brain Damage After Experimental Stroke
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol
Aryl Hydrocarbon Receptor-Dependent Induction Of Liver Fibrosis By Dioxin
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol
Aryl Hydrocarbon Receptor-Dependent Induction Of Liver Fibrosis By Dioxin
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells.
Aryl Hydrocarbon Receptor-Dependent Induction Of Liver Fibrosis By Dioxin
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells.
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans.
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells.
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans.
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans.
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol.
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol.
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol.
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis
Aryl Hydrocarbon Receptor Contributes To The Mek/erk-Dependent Maintenance Of The Immature State Of Human Dendritic Cells
The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis
Aryl Hydrocarbon Receptor Contributes To The Mek/erk-Dependent Maintenance Of The Immature State Of Human Dendritic Cells
The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway
Aryl Hydrocarbon Receptor Contributes To The Mek/erk-Dependent Maintenance Of The Immature State Of Human Dendritic Cells
The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway
Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition
The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway
Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition
Oculomotor Deficits In Aryl Hydrocarbon Receptor Null Mouse
Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition
Oculomotor Deficits In Aryl Hydrocarbon Receptor Null Mouse
Effect Of High Pressure Thermal Processing On Some Essential Nutrients And Immunological Components Present In Breast Milk
Oculomotor Deficits In Aryl Hydrocarbon Receptor Null Mouse
Effect Of High Pressure Thermal Processing On Some Essential Nutrients And Immunological Components Present In Breast Milk
Aryl Hydrocarbon Receptor-Induced Adrenomedullin Mediates Cigarette Smoke Carcinogenicity In Humans And Mice
Effect Of High Pressure Thermal Processing On Some Essential Nutrients And Immunological Components Present In Breast Milk
Aryl Hydrocarbon Receptor-Induced Adrenomedullin Mediates Cigarette Smoke Carcinogenicity In Humans And Mice
Cell Fusion Reprogramming Leads To A Specific Hepatic Expression Pattern During Mouse Bone Marrow Derived Hepatocyte Formation In Vivo
Aryl Hydrocarbon Receptor-Induced Adrenomedullin Mediates Cigarette Smoke Carcinogenicity In Humans And Mice
Cell Fusion Reprogramming Leads To A Specific Hepatic Expression Pattern During Mouse Bone Marrow Derived Hepatocyte Formation In Vivo
270 Cd44+/epcam− Phenotype In Hepatocellular Carcinoma (Hcc) Cells Correlates With Increased Autocrine Tgf-Beta And Mesenchymal Traits
Cell Fusion Reprogramming Leads To A Specific Hepatic Expression Pattern During Mouse Bone Marrow Derived Hepatocyte Formation In Vivo
270 Cd44+/epcam− Phenotype In Hepatocellular Carcinoma (Hcc) Cells Correlates With Increased Autocrine Tgf-Beta And Mesenchymal Traits
Dioxin Receptor And Slug Transcription Factors Regulate The Insulator Activity Of B1 Sine Retrotransposons Via An Rna Polymerase Switch
270 Cd44+/epcam− Phenotype In Hepatocellular Carcinoma (Hcc) Cells Correlates With Increased Autocrine Tgf-Beta And Mesenchymal Traits
Dioxin Receptor And Slug Transcription Factors Regulate The Insulator Activity Of B1 Sine Retrotransposons Via An Rna Polymerase Switch
The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling
Dioxin Receptor And Slug Transcription Factors Regulate The Insulator Activity Of B1 Sine Retrotransposons Via An Rna Polymerase Switch
The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling
Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse
The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling
Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse
Transcriptional Factor Aryl Hydrocarbon Receptor (Ahr) Controls Cardiovascular And Respiratory Functions By Regulating The Expression Of The Vav3 Proto-Oncogene
Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse
Transcriptional Factor Aryl Hydrocarbon Receptor (Ahr) Controls Cardiovascular And Respiratory Functions By Regulating The Expression Of The Vav3 Proto-Oncogene
Active Commuting To School And Cognitive Performance In Adolescents: The Avena Study.
Transcriptional Factor Aryl Hydrocarbon Receptor (Ahr) Controls Cardiovascular And Respiratory Functions By Regulating The Expression Of The Vav3 Proto-Oncogene
Active Commuting To School And Cognitive Performance In Adolescents: The Avena Study.
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells
Active Commuting To School And Cognitive Performance In Adolescents: The Avena Study.
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells
A Remarkable New Target Gene For The Dioxin Receptor: The Vav3 Proto-Oncogene Links Ahr To Adhesion And Migration
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells
A Remarkable New Target Gene For The Dioxin Receptor: The Vav3 Proto-Oncogene Links Ahr To Adhesion And Migration
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
A Remarkable New Target Gene For The Dioxin Receptor: The Vav3 Proto-Oncogene Links Ahr To Adhesion And Migration
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Comparative Analysis Of Carmellose 0.5% Versus Hyaluronate 0.15% In Dry Eye: A Flow Cytometric Study
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Comparative Analysis Of Carmellose 0.5% Versus Hyaluronate 0.15% In Dry Eye: A Flow Cytometric Study
The Effect Of Preservative-Free Hp-Guar On Dry Eye After Phacoemulsification: A Flow Cytometric Study.
Comparative Analysis Of Carmellose 0.5% Versus Hyaluronate 0.15% In Dry Eye: A Flow Cytometric Study
The Effect Of Preservative-Free Hp-Guar On Dry Eye After Phacoemulsification: A Flow Cytometric Study.
Role Of Transforming Growth Factor Β In Cancer Microenvironment
The Effect Of Preservative-Free Hp-Guar On Dry Eye After Phacoemulsification: A Flow Cytometric Study.
Role Of Transforming Growth Factor Β In Cancer Microenvironment
Dioxin Receptor Deficiency Impairs Angiogenesis By A Mechanism Involving Vegf-A Depletion In The Endothelium And Transforming Growth Factor-Β Overexpression In The Stroma
Role Of Transforming Growth Factor Β In Cancer Microenvironment
Dioxin Receptor Deficiency Impairs Angiogenesis By A Mechanism Involving Vegf-A Depletion In The Endothelium And Transforming Growth Factor-Β Overexpression In The Stroma
Regulation Of Cell Survival By Resveratrol Involves Inhibition Of Nfκb-Regulated Gene Expression In Prostate Cancer Cells
Dioxin Receptor Deficiency Impairs Angiogenesis By A Mechanism Involving Vegf-A Depletion In The Endothelium And Transforming Growth Factor-Β Overexpression In The Stroma
Regulation Of Cell Survival By Resveratrol Involves Inhibition Of Nfκb-Regulated Gene Expression In Prostate Cancer Cells
The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion
Regulation Of Cell Survival By Resveratrol Involves Inhibition Of Nfκb-Regulated Gene Expression In Prostate Cancer Cells
The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion
Fitting A Xenobiotic Receptor Into Cell Homeostasis: How The Dioxin Receptor Interacts With Tgfβ Signaling
The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion
Fitting A Xenobiotic Receptor Into Cell Homeostasis: How The Dioxin Receptor Interacts With Tgfβ Signaling
Loss Of Dioxin-Receptor Expression Accelerates Wound Healing In Vivo By A Mechanism Involving Tgfβ
Fitting A Xenobiotic Receptor Into Cell Homeostasis: How The Dioxin Receptor Interacts With Tgfβ Signaling
Loss Of Dioxin-Receptor Expression Accelerates Wound Healing In Vivo By A Mechanism Involving Tgfβ
Haematological Reference Values In Spanish Adolescents: The Avena Study.
Loss Of Dioxin-Receptor Expression Accelerates Wound Healing In Vivo By A Mechanism Involving Tgfβ
Haematological Reference Values In Spanish Adolescents: The Avena Study.
Recruitment Of Creb1 And Histone Deacetylase 2 (Hdac2) To The Mouse Ltbp-1 Promoter Regulates Its Constitutive Expression In A Dioxin Receptor-Dependent Manner
Haematological Reference Values In Spanish Adolescents: The Avena Study.
Recruitment Of Creb1 And Histone Deacetylase 2 (Hdac2) To The Mouse Ltbp-1 Promoter Regulates Its Constitutive Expression In A Dioxin Receptor-Dependent Manner
Genome-Wide B1 Retrotransposon Binds The Transcription Factors Dioxin Receptor And Slug And Regulates Gene Expression In Vivo
Recruitment Of Creb1 And Histone Deacetylase 2 (Hdac2) To The Mouse Ltbp-1 Promoter Regulates Its Constitutive Expression In A Dioxin Receptor-Dependent Manner
Genome-Wide B1 Retrotransposon Binds The Transcription Factors Dioxin Receptor And Slug And Regulates Gene Expression In Vivo
Isolation And Characterization Of A Putative Liver Progenitor Population After Treatment Of Fetal Rat Hepatocytes With Tgf-Beta
Genome-Wide B1 Retrotransposon Binds The Transcription Factors Dioxin Receptor And Slug And Regulates Gene Expression In Vivo
Isolation And Characterization Of A Putative Liver Progenitor Population After Treatment Of Fetal Rat Hepatocytes With Tgf-Beta
Ccaat/enhancer Binding Protein Β Deficiency Provides Cerebral Protection Following Excitotoxic Injury
Isolation And Characterization Of A Putative Liver Progenitor Population After Treatment Of Fetal Rat Hepatocytes With Tgf-Beta
Ccaat/enhancer Binding Protein Β Deficiency Provides Cerebral Protection Following Excitotoxic Injury
Deletion Of The Met Tyrosine Kinase In Liver Progenitor Oval Cells Increases Sensitivity To Apoptosis In Vitro
Ccaat/enhancer Binding Protein Β Deficiency Provides Cerebral Protection Following Excitotoxic Injury
Deletion Of The Met Tyrosine Kinase In Liver Progenitor Oval Cells Increases Sensitivity To Apoptosis In Vitro
Bcl6 Represses Nfκb Activity In Diffuse Large B-Cell Lymphomas
Deletion Of The Met Tyrosine Kinase In Liver Progenitor Oval Cells Increases Sensitivity To Apoptosis In Vitro
Bcl6 Represses Nfκb Activity In Diffuse Large B-Cell Lymphomas
Non-Genomic Action Of Resveratrol On Androgen And Oestrogen Receptors In Prostate Cancer: Modulation Of The Phosphoinositide 3-Kinase Pathway
Bcl6 Represses Nfκb Activity In Diffuse Large B-Cell Lymphomas
Non-Genomic Action Of Resveratrol On Androgen And Oestrogen Receptors In Prostate Cancer: Modulation Of The Phosphoinositide 3-Kinase Pathway
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
Non-Genomic Action Of Resveratrol On Androgen And Oestrogen Receptors In Prostate Cancer: Modulation Of The Phosphoinositide 3-Kinase Pathway
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
The Aryl Hydrocarbon Receptor, More Than A Xenobiotic-Interacting Protein
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
The Aryl Hydrocarbon Receptor, More Than A Xenobiotic-Interacting Protein
Methylmalonic Acidaemia Leads To Increased Production Of Reactive Oxygen Species And Induction Of Apoptosis Through The Mitochondrial/caspase Pathway
The Aryl Hydrocarbon Receptor, More Than A Xenobiotic-Interacting Protein
Methylmalonic Acidaemia Leads To Increased Production Of Reactive Oxygen Species And Induction Of Apoptosis Through The Mitochondrial/caspase Pathway
Nitric Oxide Down-Regulates Caveolin-3 Levels Through The Interaction With Myogenin, Its Transcription Factor
Methylmalonic Acidaemia Leads To Increased Production Of Reactive Oxygen Species And Induction Of Apoptosis Through The Mitochondrial/caspase Pathway
Nitric Oxide Down-Regulates Caveolin-3 Levels Through The Interaction With Myogenin, Its Transcription Factor
P38α Mapk Can Positively Or Negatively Regulate Rac-1 Activity Depending On The Presence Of Serum
Nitric Oxide Down-Regulates Caveolin-3 Levels Through The Interaction With Myogenin, Its Transcription Factor
P38α Mapk Can Positively Or Negatively Regulate Rac-1 Activity Depending On The Presence Of Serum
Binding Of Cap70 To Inducible Nitric Oxide Synthase And Implications For The Vectorial Release Of Nitric Oxide In Polarized Cells
P38α Mapk Can Positively Or Negatively Regulate Rac-1 Activity Depending On The Presence Of Serum
Binding Of Cap70 To Inducible Nitric Oxide Synthase And Implications For The Vectorial Release Of Nitric Oxide In Polarized Cells
Mitochondrial Superoxide Mediates Heat-Induced Apoptotic-Like Death In Leishmania Infantum
Binding Of Cap70 To Inducible Nitric Oxide Synthase And Implications For The Vectorial Release Of Nitric Oxide In Polarized Cells
Mitochondrial Superoxide Mediates Heat-Induced Apoptotic-Like Death In Leishmania Infantum
Tlr4-Mediated Survival Of Macrophages Is Myd88 Dependent And Requires Tnf-Α Autocrine Signalling
Mitochondrial Superoxide Mediates Heat-Induced Apoptotic-Like Death In Leishmania Infantum
Tlr4-Mediated Survival Of Macrophages Is Myd88 Dependent And Requires Tnf-Α Autocrine Signalling
Role Of Peroxynitrite In Endothelial Damage Mediated By Cyclosporine A
Tlr4-Mediated Survival Of Macrophages Is Myd88 Dependent And Requires Tnf-Α Autocrine Signalling
Role Of Peroxynitrite In Endothelial Damage Mediated By Cyclosporine A
C-Jun N-Terminal Protein Kinase Signalling Pathway Mediates Lovastatin-Induced Rat Brain Neuroblast Apoptosis
Role Of Peroxynitrite In Endothelial Damage Mediated By Cyclosporine A
C-Jun N-Terminal Protein Kinase Signalling Pathway Mediates Lovastatin-Induced Rat Brain Neuroblast Apoptosis
Lovastatin Inhibits The Extracellular-Signal-Regulated Kinase Pathway In Immortalized Rat Brain Neuroblasts
C-Jun N-Terminal Protein Kinase Signalling Pathway Mediates Lovastatin-Induced Rat Brain Neuroblast Apoptosis
Lovastatin Inhibits The Extracellular-Signal-Regulated Kinase Pathway In Immortalized Rat Brain Neuroblasts
Negative Regulation Of Akt Activity By P38α Map Kinase In Cardiomyocytes Involves Membrane Localization Of Pp2a Through Interaction With Caveolin-1
Lovastatin Inhibits The Extracellular-Signal-Regulated Kinase Pathway In Immortalized Rat Brain Neuroblasts
Negative Regulation Of Akt Activity By P38α Map Kinase In Cardiomyocytes Involves Membrane Localization Of Pp2a Through Interaction With Caveolin-1
The Dioxin Receptor Is Silenced By Promoter Hypermethylation In Human Acute Lymphoblastic Leukemia Through Inhibition Of Sp1 Binding
Negative Regulation Of Akt Activity By P38α Map Kinase In Cardiomyocytes Involves Membrane Localization Of Pp2a Through Interaction With Caveolin-1
The Dioxin Receptor Is Silenced By Promoter Hypermethylation In Human Acute Lymphoblastic Leukemia Through Inhibition Of Sp1 Binding
Ltbp-1 Blockade In Dioxin Receptor-Null Mouse Embryo Fibroblasts Decreases Tgf-Β Activity: Role Of Extracellular Proteases Plasmin And Elastase
The Dioxin Receptor Is Silenced By Promoter Hypermethylation In Human Acute Lymphoblastic Leukemia Through Inhibition Of Sp1 Binding
Ltbp-1 Blockade In Dioxin Receptor-Null Mouse Embryo Fibroblasts Decreases Tgf-Β Activity: Role Of Extracellular Proteases Plasmin And Elastase
Heat-Induced Programmed Cell Death In Leishmania Infantum Is Reverted By Bcl-Xl Expression
Ltbp-1 Blockade In Dioxin Receptor-Null Mouse Embryo Fibroblasts Decreases Tgf-Β Activity: Role Of Extracellular Proteases Plasmin And Elastase
Heat-Induced Programmed Cell Death In Leishmania Infantum Is Reverted By Bcl-Xl Expression
A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c
Heat-Induced Programmed Cell Death In Leishmania Infantum Is Reverted By Bcl-Xl Expression
A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c
Loss Of Apoptosis-Inducing Factor Leads To An Increase In Reactive Oxygen Species, And An Impairment Of Respiration That Can Be Reversed By Antioxidants [4]
A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c
Loss Of Apoptosis-Inducing Factor Leads To An Increase In Reactive Oxygen Species, And An Impairment Of Respiration That Can Be Reversed By Antioxidants [4]
Infiltration Of Inflammatory Cells Plays An Important Role In Matrix Metalloproteinase Expression And Activation In The Heart During Sepsis
Loss Of Apoptosis-Inducing Factor Leads To An Increase In Reactive Oxygen Species, And An Impairment Of Respiration That Can Be Reversed By Antioxidants [4]
Infiltration Of Inflammatory Cells Plays An Important Role In Matrix Metalloproteinase Expression And Activation In The Heart During Sepsis
Hematopoietic Mobilization In Mice Increases The Presence Of Bone Marrow-Derived Hepatocytes Via In Vivo Cell Fusion
Infiltration Of Inflammatory Cells Plays An Important Role In Matrix Metalloproteinase Expression And Activation In The Heart During Sepsis
Hematopoietic Mobilization In Mice Increases The Presence Of Bone Marrow-Derived Hepatocytes Via In Vivo Cell Fusion
Gastrin Induces Leukocyte-Endothelial Cell Interactions In Vivo And Contributes To The Inflammation Caused By Helicobacter Pylori
Hematopoietic Mobilization In Mice Increases The Presence Of Bone Marrow-Derived Hepatocytes Via In Vivo Cell Fusion
Gastrin Induces Leukocyte-Endothelial Cell Interactions In Vivo And Contributes To The Inflammation Caused By Helicobacter Pylori
Autocrine Production Of Tgf-Β Confers Resistance To Apoptosis After An Epithelial-Mesenchymal Transition Process In Hepatocytes: Role Of Egf Receptor Ligands
Gastrin Induces Leukocyte-Endothelial Cell Interactions In Vivo And Contributes To The Inflammation Caused By Helicobacter Pylori
Autocrine Production Of Tgf-Β Confers Resistance To Apoptosis After An Epithelial-Mesenchymal Transition Process In Hepatocytes: Role Of Egf Receptor Ligands
N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways
Autocrine Production Of Tgf-Β Confers Resistance To Apoptosis After An Epithelial-Mesenchymal Transition Process In Hepatocytes: Role Of Egf Receptor Ligands
N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways
Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science (2006) Vol. 119 (1558-1596))
N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways
Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science (2006) Vol. 119 (1558-1596))
Erratum: A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c (Genes And Development (2006) 20 (307-320))
Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science (2006) Vol. 119 (1558-1596))
Erratum: A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c (Genes And Development (2006) 20 (307-320))
Improving Cancer Therapeutics By Molecular Profiling
Erratum: A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c (Genes And Development (2006) 20 (307-320))
Improving Cancer Therapeutics By Molecular Profiling
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Improving Cancer Therapeutics By Molecular Profiling
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Identification Of A Nuclear Export Signal In The Kshv Latent Protein Lana2 Mediating Its Export From The Nucleus
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Identification Of A Nuclear Export Signal In The Kshv Latent Protein Lana2 Mediating Its Export From The Nucleus
Discrepancies Between Nitroglycerin And No-Releasing Drugs On Mitochondrial Oxygen Consumption, Vasoactivity, And The Release Of No
Identification Of A Nuclear Export Signal In The Kshv Latent Protein Lana2 Mediating Its Export From The Nucleus
Discrepancies Between Nitroglycerin And No-Releasing Drugs On Mitochondrial Oxygen Consumption, Vasoactivity, And The Release Of No
Direct Interaction Between The Reductase Domain Of Endothelial Nitric Oxide Synthase And The Ryanodine Receptor
Discrepancies Between Nitroglycerin And No-Releasing Drugs On Mitochondrial Oxygen Consumption, Vasoactivity, And The Release Of No
Direct Interaction Between The Reductase Domain Of Endothelial Nitric Oxide Synthase And The Ryanodine Receptor
Pgc-1α Regulates The Mitochondrial Antioxidant Defense System In Vascular Endothelial Cells
Direct Interaction Between The Reductase Domain Of Endothelial Nitric Oxide Synthase And The Ryanodine Receptor
Pgc-1α Regulates The Mitochondrial Antioxidant Defense System In Vascular Endothelial Cells
Matrix Metalloproteinase 13 Mediates Nitric Oxide Activation Of Endothelial Cell Migration
Pgc-1α Regulates The Mitochondrial Antioxidant Defense System In Vascular Endothelial Cells
Matrix Metalloproteinase 13 Mediates Nitric Oxide Activation Of Endothelial Cell Migration
Resveratrol Modulates The Phosphoinositide 3-Kinase Pathway Through An Estrogen Receptor Α-Dependent Mechanism: Relevance In Cell Proliferation
Matrix Metalloproteinase 13 Mediates Nitric Oxide Activation Of Endothelial Cell Migration
Resveratrol Modulates The Phosphoinositide 3-Kinase Pathway Through An Estrogen Receptor Α-Dependent Mechanism: Relevance In Cell Proliferation
Overexpression Of Latent Transforming Growth Factor-Β Binding Protein 1 (Ltbp-1) In Dioxin Receptor-Null Mouse Embryo Fibroblasts
Resveratrol Modulates The Phosphoinositide 3-Kinase Pathway Through An Estrogen Receptor Α-Dependent Mechanism: Relevance In Cell Proliferation
Overexpression Of Latent Transforming Growth Factor-Β Binding Protein 1 (Ltbp-1) In Dioxin Receptor-Null Mouse Embryo Fibroblasts
Synthesis Of Nitric Oxide In Postganglionic Myenteric Neurons During Endotoxemia: Implications For Gastric Motor Function In Rats.
Overexpression Of Latent Transforming Growth Factor-Β Binding Protein 1 (Ltbp-1) In Dioxin Receptor-Null Mouse Embryo Fibroblasts
Synthesis Of Nitric Oxide In Postganglionic Myenteric Neurons During Endotoxemia: Implications For Gastric Motor Function In Rats.
Source Of Early Reactive Oxygen Species In The Apoptosis Induced By Transforming Growth Factor-Β In Fetal Rat Hepatocytes
Synthesis Of Nitric Oxide In Postganglionic Myenteric Neurons During Endotoxemia: Implications For Gastric Motor Function In Rats.
Source Of Early Reactive Oxygen Species In The Apoptosis Induced By Transforming Growth Factor-Β In Fetal Rat Hepatocytes
Resistance To Tgf-Beta-Induced Apoptosis In Regenerating Hepatocytes.
Source Of Early Reactive Oxygen Species In The Apoptosis Induced By Transforming Growth Factor-Β In Fetal Rat Hepatocytes
Resistance To Tgf-Beta-Induced Apoptosis In Regenerating Hepatocytes.
Palmitoylation Of Inducible Nitric-Oxide Synthase At Cys-3 Is Required For Proper Intracellular Traffic And Nitric Oxide Synthesis.
Resistance To Tgf-Beta-Induced Apoptosis In Regenerating Hepatocytes.
Palmitoylation Of Inducible Nitric-Oxide Synthase At Cys-3 Is Required For Proper Intracellular Traffic And Nitric Oxide Synthesis.
The Effect Of Milk Fermented By Yogurt Cultures Plus Lactobacillus Casei Dn-114001 On The Immune Response Of Subjects Under Academic Examination Stress.
Palmitoylation Of Inducible Nitric-Oxide Synthase At Cys-3 Is Required For Proper Intracellular Traffic And Nitric Oxide Synthesis.
The Effect Of Milk Fermented By Yogurt Cultures Plus Lactobacillus Casei Dn-114001 On The Immune Response Of Subjects Under Academic Examination Stress.
Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels.
The Effect Of Milk Fermented By Yogurt Cultures Plus Lactobacillus Casei Dn-114001 On The Immune Response Of Subjects Under Academic Examination Stress.
Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels.
P38 Alpha Mitogen-Activated Protein Kinase Sensitizes Cells To Apoptosis Induced By Different Stimuli.
Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels.
P38 Alpha Mitogen-Activated Protein Kinase Sensitizes Cells To Apoptosis Induced By Different Stimuli.
Liver Portal Fibrosis In Dioxin Receptor-Null Mice That Overexpress The Latent Transforming Growth Factor-Β-Binding Protein-1
P38 Alpha Mitogen-Activated Protein Kinase Sensitizes Cells To Apoptosis Induced By Different Stimuli.
Liver Portal Fibrosis In Dioxin Receptor-Null Mice That Overexpress The Latent Transforming Growth Factor-Β-Binding Protein-1
Proteasome Inhibition Induces Nuclear Translocation Of The Dioxin Receptor Through An Sp1 And Protein Kinase C-Dependent Pathway
Liver Portal Fibrosis In Dioxin Receptor-Null Mice That Overexpress The Latent Transforming Growth Factor-Β-Binding Protein-1
Proteasome Inhibition Induces Nuclear Translocation Of The Dioxin Receptor Through An Sp1 And Protein Kinase C-Dependent Pathway
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Proteasome Inhibition Induces Nuclear Translocation Of The Dioxin Receptor Through An Sp1 And Protein Kinase C-Dependent Pathway
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Thioridazine Steady-State Plasma Concentrations Are Influenced By Tobacco Smoking And Cyp2d6, But Not By The Cyp2c9 Genotype
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Thioridazine Steady-State Plasma Concentrations Are Influenced By Tobacco Smoking And Cyp2d6, But Not By The Cyp2c9 Genotype
Carcinogenesis Of The Food Mutagen Phip In Mice Is Dependent Of Cyp1a2
Thioridazine Steady-State Plasma Concentrations Are Influenced By Tobacco Smoking And Cyp2d6, But Not By The Cyp2c9 Genotype
Carcinogenesis Of The Food Mutagen Phip In Mice Is Dependent Of Cyp1a2
Polycyclic Aromatic Hydrocarbon-Inducible Dna Adducts: Evidence By 32p-Postlabeling And Use Of Knockout Mice For Ah Receptor-Independent Mechanisms Of Metabolic Activation In Vivo
Carcinogenesis Of The Food Mutagen Phip In Mice Is Dependent Of Cyp1a2
Polycyclic Aromatic Hydrocarbon-Inducible Dna Adducts: Evidence By 32p-Postlabeling And Use Of Knockout Mice For Ah Receptor-Independent Mechanisms Of Metabolic Activation In Vivo
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
Polycyclic Aromatic Hydrocarbon-Inducible Dna Adducts: Evidence By 32p-Postlabeling And Use Of Knockout Mice For Ah Receptor-Independent Mechanisms Of Metabolic Activation In Vivo
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Confocal Visualization Of Nitric Oxide In The Myenteric Plexus Of The Gastric Fundus Obtained From Endotoxin-Treated Rats
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Confocal Visualization Of Nitric Oxide In The Myenteric Plexus Of The Gastric Fundus Obtained From Endotoxin-Treated Rats
Long-Term Treatment With Insulin Induces Apoptosis In Brown Adipocytes: Role Of Oxidative Stress.
Confocal Visualization Of Nitric Oxide In The Myenteric Plexus Of The Gastric Fundus Obtained From Endotoxin-Treated Rats
Long-Term Treatment With Insulin Induces Apoptosis In Brown Adipocytes: Role Of Oxidative Stress.
Sustained Nitric Oxide Delivery Delays Nitric Oxide-Dependent Apoptosis In Macrophages: Contribution To The Physiological Function Of Activated Macrophages.
Long-Term Treatment With Insulin Induces Apoptosis In Brown Adipocytes: Role Of Oxidative Stress.
Sustained Nitric Oxide Delivery Delays Nitric Oxide-Dependent Apoptosis In Macrophages: Contribution To The Physiological Function Of Activated Macrophages.
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
Sustained Nitric Oxide Delivery Delays Nitric Oxide-Dependent Apoptosis In Macrophages: Contribution To The Physiological Function Of Activated Macrophages.
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Evidence For Discoordinate Regulation Of The Hla-Dpb1 Gene
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Evidence For Discoordinate Regulation Of The Hla-Dpb1 Gene
Tace/adam17-Tnf-Α Pathway In Rat Cortical Cultures After Exposure To Oxygen-Glucose Deprivation Or Glutamate
Evidence For Discoordinate Regulation Of The Hla-Dpb1 Gene
Tace/adam17-Tnf-Α Pathway In Rat Cortical Cultures After Exposure To Oxygen-Glucose Deprivation Or Glutamate
Aplidin Induces The Mitochondrial Apoptotic Pathway Via Oxidative Stress-Mediated Jnk And P38 Activation And Protein Kinase C Delta.
Tace/adam17-Tnf-Α Pathway In Rat Cortical Cultures After Exposure To Oxygen-Glucose Deprivation Or Glutamate
Aplidin Induces The Mitochondrial Apoptotic Pathway Via Oxidative Stress-Mediated Jnk And P38 Activation And Protein Kinase C Delta.
Differential Role Of Ppar Gamma In The Regulation Of Ucp-1 And Adipogenesis By Tnf-Alpha In Brown Adipocytes.
Aplidin Induces The Mitochondrial Apoptotic Pathway Via Oxidative Stress-Mediated Jnk And P38 Activation And Protein Kinase C Delta.
Differential Role Of Ppar Gamma In The Regulation Of Ucp-1 And Adipogenesis By Tnf-Alpha In Brown Adipocytes.
Induction Of Apoptosis By Nitric Oxide In Macrophages Is Independent Of Apoptotic Volume Decrease.
Differential Role Of Ppar Gamma In The Regulation Of Ucp-1 And Adipogenesis By Tnf-Alpha In Brown Adipocytes.
Induction Of Apoptosis By Nitric Oxide In Macrophages Is Independent Of Apoptotic Volume Decrease.
In Vitro Fertilizing Capacity And Chromatin Condensation Of Deep Frozen Boar Semen Packaged In 0.5 And 5 Ml Straws.
Induction Of Apoptosis By Nitric Oxide In Macrophages Is Independent Of Apoptotic Volume Decrease.
In Vitro Fertilizing Capacity And Chromatin Condensation Of Deep Frozen Boar Semen Packaged In 0.5 And 5 Ml Straws.
Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains
In Vitro Fertilizing Capacity And Chromatin Condensation Of Deep Frozen Boar Semen Packaged In 0.5 And 5 Ml Straws.
Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
Effect Of Thioridazine Dosage On The Debrisoquine Hydroxylation Phenotype In Psychiatric Patients With Different Cyp2d6 Genotypes
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
Effect Of Thioridazine Dosage On The Debrisoquine Hydroxylation Phenotype In Psychiatric Patients With Different Cyp2d6 Genotypes
Targeted Genomic Disruption Of H-Ras And N-Ras, Individually Or In Combination, Reveals The Dispensability Of Both Loci For Mouse Growth And Development
Effect Of Thioridazine Dosage On The Debrisoquine Hydroxylation Phenotype In Psychiatric Patients With Different Cyp2d6 Genotypes
Targeted Genomic Disruption Of H-Ras And N-Ras, Individually Or In Combination, Reveals The Dispensability Of Both Loci For Mouse Growth And Development
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
Targeted Genomic Disruption Of H-Ras And N-Ras, Individually Or In Combination, Reveals The Dispensability Of Both Loci For Mouse Growth And Development
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
Akt1/pkbalpha Protects Pc12 Cells Against The Parkinsonism-Inducing Neurotoxin 1-Methyl-4-Phenylpyridinium And Reduces The Levels Of Oxygen-Free Radicals.
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
Akt1/pkbalpha Protects Pc12 Cells Against The Parkinsonism-Inducing Neurotoxin 1-Methyl-4-Phenylpyridinium And Reduces The Levels Of Oxygen-Free Radicals.
Use Of Flow Cytometry And Confocal Microscopy Techniques To Investigate Early Cdcl2-Induced Nephrotoxicity In Vitro
Akt1/pkbalpha Protects Pc12 Cells Against The Parkinsonism-Inducing Neurotoxin 1-Methyl-4-Phenylpyridinium And Reduces The Levels Of Oxygen-Free Radicals.
Use Of Flow Cytometry And Confocal Microscopy Techniques To Investigate Early Cdcl2-Induced Nephrotoxicity In Vitro
Lovastatin Induces Apoptosis Of Spontaneously Immortalized Rat Brain Neuroblasts: Involvement Of Nonsterol Isoprenoid Biosynthesis Inhibition
Use Of Flow Cytometry And Confocal Microscopy Techniques To Investigate Early Cdcl2-Induced Nephrotoxicity In Vitro
Lovastatin Induces Apoptosis Of Spontaneously Immortalized Rat Brain Neuroblasts: Involvement Of Nonsterol Isoprenoid Biosynthesis Inhibition
Intracellular Water Motion Decreases In Apoptotic Macrophages After Caspase Activation.
Lovastatin Induces Apoptosis Of Spontaneously Immortalized Rat Brain Neuroblasts: Involvement Of Nonsterol Isoprenoid Biosynthesis Inhibition
Intracellular Water Motion Decreases In Apoptotic Macrophages After Caspase Activation.
Activation Of Caspases Occurs Downstream From Radical Oxygen Species Production, Bcl-Xl Down-Regulation, And Early Cytochrome C Release In Apoptosis Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes.
Intracellular Water Motion Decreases In Apoptotic Macrophages After Caspase Activation.
Activation Of Caspases Occurs Downstream From Radical Oxygen Species Production, Bcl-Xl Down-Regulation, And Early Cytochrome C Release In Apoptosis Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes.
Reactive Oxygen Species (Ros) Mediates The Mitochondrial-Dependent Apoptosis Induced By Transforming Growth Factor (Beta) In Fetal Hepatocytes.
Activation Of Caspases Occurs Downstream From Radical Oxygen Species Production, Bcl-Xl Down-Regulation, And Early Cytochrome C Release In Apoptosis Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes.
Reactive Oxygen Species (Ros) Mediates The Mitochondrial-Dependent Apoptosis Induced By Transforming Growth Factor (Beta) In Fetal Hepatocytes.
Mitochondrial Involvement In Cocaine-Treated Rat Hepatocytes: Effect Of N-Acetylcysteine And Deferoxamine.
Reactive Oxygen Species (Ros) Mediates The Mitochondrial-Dependent Apoptosis Induced By Transforming Growth Factor (Beta) In Fetal Hepatocytes.
Mitochondrial Involvement In Cocaine-Treated Rat Hepatocytes: Effect Of N-Acetylcysteine And Deferoxamine.
Effect Of N-Acetylcysteine And Deferoxamine On Endogenous Antioxidant Defense System Gene Expression In A Rat Hepatocyte Model Of Cocaine Cytotoxicity.
Mitochondrial Involvement In Cocaine-Treated Rat Hepatocytes: Effect Of N-Acetylcysteine And Deferoxamine.
Effect Of N-Acetylcysteine And Deferoxamine On Endogenous Antioxidant Defense System Gene Expression In A Rat Hepatocyte Model Of Cocaine Cytotoxicity.
Fibronectin Regulates Morphology, Cell Organization And Gene Expression Of Rat Fetal Hepatocytes In Primary Culture.
Effect Of N-Acetylcysteine And Deferoxamine On Endogenous Antioxidant Defense System Gene Expression In A Rat Hepatocyte Model Of Cocaine Cytotoxicity.
Fibronectin Regulates Morphology, Cell Organization And Gene Expression Of Rat Fetal Hepatocytes In Primary Culture.
Inhibition Of Pkb/akt1 By C2-Ceramide Involves Activation Of Ceramide-Activated Protein Phosphatase In Pc12 Cells.
Fibronectin Regulates Morphology, Cell Organization And Gene Expression Of Rat Fetal Hepatocytes In Primary Culture.
Inhibition Of Pkb/akt1 By C2-Ceramide Involves Activation Of Ceramide-Activated Protein Phosphatase In Pc12 Cells.
P38 Mitogen-Activated Protein Kinase Mediates Tumor Necrosis Factor-Alpha-Induced Apoptosis In Rat Fetal Brown Adipocytes.
Inhibition Of Pkb/akt1 By C2-Ceramide Involves Activation Of Ceramide-Activated Protein Phosphatase In Pc12 Cells.
P38 Mitogen-Activated Protein Kinase Mediates Tumor Necrosis Factor-Alpha-Induced Apoptosis In Rat Fetal Brown Adipocytes.
Noradrenaline Induces Brown Adipocytes Cell Growth Via Beta-Receptors By A Mechanism Dependent On Erks But Independent Of Camp And Pka.
P38 Mitogen-Activated Protein Kinase Mediates Tumor Necrosis Factor-Alpha-Induced Apoptosis In Rat Fetal Brown Adipocytes.
Noradrenaline Induces Brown Adipocytes Cell Growth Via Beta-Receptors By A Mechanism Dependent On Erks But Independent Of Camp And Pka.
Epidermal Growth Factor Impairs The Cytochrome C/caspase-3 Apoptotic Pathway Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes Via A Phosphoinositide 3-Kinase-Dependent Pathway.
Noradrenaline Induces Brown Adipocytes Cell Growth Via Beta-Receptors By A Mechanism Dependent On Erks But Independent Of Camp And Pka.
Epidermal Growth Factor Impairs The Cytochrome C/caspase-3 Apoptotic Pathway Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes Via A Phosphoinositide 3-Kinase-Dependent Pathway.
Changes In Antioxidant Defence Systems Induced By Cyclosporine A In Cultures Of Hepatocytes From 2- And 12-Month-Old Rats.
Epidermal Growth Factor Impairs The Cytochrome C/caspase-3 Apoptotic Pathway Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes Via A Phosphoinositide 3-Kinase-Dependent Pathway.
Changes In Antioxidant Defence Systems Induced By Cyclosporine A In Cultures Of Hepatocytes From 2- And 12-Month-Old Rats.
Contribution Of Cyclopentenone Prostaglandins To The Resolution Of Inflammation Through The Potentiation Of Apoptosis In Activated Macrophages.
Changes In Antioxidant Defence Systems Induced By Cyclosporine A In Cultures Of Hepatocytes From 2- And 12-Month-Old Rats.
Contribution Of Cyclopentenone Prostaglandins To The Resolution Of Inflammation Through The Potentiation Of Apoptosis In Activated Macrophages.
Hsp70 Induction By Cyclosporine A In Cultured Rat Hepatocytes: Effect Of Vitamin E Succinate.
Contribution Of Cyclopentenone Prostaglandins To The Resolution Of Inflammation Through The Potentiation Of Apoptosis In Activated Macrophages.
Hsp70 Induction By Cyclosporine A In Cultured Rat Hepatocytes: Effect Of Vitamin E Succinate.
Altered Cell Cycle Control At The G2/m Phases In Aryl Hydrocarbon Receptor-Null Embryo Fibroblast
Hsp70 Induction By Cyclosporine A In Cultured Rat Hepatocytes: Effect Of Vitamin E Succinate.
Altered Cell Cycle Control At The G2/m Phases In Aryl Hydrocarbon Receptor-Null Embryo Fibroblast
Applications Of Confocal Mycroscopy To The Paper Industry: Slime And Deposit Examples
Altered Cell Cycle Control At The G2/m Phases In Aryl Hydrocarbon Receptor-Null Embryo Fibroblast
Applications Of Confocal Mycroscopy To The Paper Industry: Slime And Deposit Examples
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Applications Of Confocal Mycroscopy To The Paper Industry: Slime And Deposit Examples
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Hepatic Fibrosis And Cytochrome P450: Experimental Models Of Fibrosis Compared To Ahr Knockout Mice
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Hepatic Fibrosis And Cytochrome P450: Experimental Models Of Fibrosis Compared To Ahr Knockout Mice
Applications Of Flow Cytometry To Clinical Microbiology
Hepatic Fibrosis And Cytochrome P450: Experimental Models Of Fibrosis Compared To Ahr Knockout Mice
Applications Of Flow Cytometry To Clinical Microbiology
Age-Related Changes On Parameters Of Experimentally-Induced Liver Injury And Regeneration.
Applications Of Flow Cytometry To Clinical Microbiology
Age-Related Changes On Parameters Of Experimentally-Induced Liver Injury And Regeneration.
Apoptotic Response To Tgf-Beta In Fetal Hepatocytes Depends Upon Their State Of Differentiation.
Age-Related Changes On Parameters Of Experimentally-Induced Liver Injury And Regeneration.
Apoptotic Response To Tgf-Beta In Fetal Hepatocytes Depends Upon Their State Of Differentiation.
Nonviral Transfer Of Genes To Pig Primary Keratinocytes. Induction Of Angiogenesis By Composite Grafts Of Modified Keratinocytes Overexpressing Vegf Driven By A Keratin Promoter.
Apoptotic Response To Tgf-Beta In Fetal Hepatocytes Depends Upon Their State Of Differentiation.
Nonviral Transfer Of Genes To Pig Primary Keratinocytes. Induction Of Angiogenesis By Composite Grafts Of Modified Keratinocytes Overexpressing Vegf Driven By A Keratin Promoter.
Cocaine Cytotoxicity In Hepatocyte Cultures From Phenobarbital-Induced Rats: Involvement Of Reactive Oxygen Species And Expression Of Antioxidant Defense Systems.
Nonviral Transfer Of Genes To Pig Primary Keratinocytes. Induction Of Angiogenesis By Composite Grafts Of Modified Keratinocytes Overexpressing Vegf Driven By A Keratin Promoter.
Cocaine Cytotoxicity In Hepatocyte Cultures From Phenobarbital-Induced Rats: Involvement Of Reactive Oxygen Species And Expression Of Antioxidant Defense Systems.
Nitric Oxide Induces Tyrosine Nitration And Release Of Cytochrome C Preceding An Increase Of Mitochondrial Transmembrane Potential In Macrophages.
Cocaine Cytotoxicity In Hepatocyte Cultures From Phenobarbital-Induced Rats: Involvement Of Reactive Oxygen Species And Expression Of Antioxidant Defense Systems.
Nitric Oxide Induces Tyrosine Nitration And Release Of Cytochrome C Preceding An Increase Of Mitochondrial Transmembrane Potential In Macrophages.
Genomic Instability In Gadd45a-Deficient Mice
Nitric Oxide Induces Tyrosine Nitration And Release Of Cytochrome C Preceding An Increase Of Mitochondrial Transmembrane Potential In Macrophages.
Genomic Instability In Gadd45a-Deficient Mice
Cyp1a2 Is Not The Primary Enzyme Responsible For 4-Aminobiphenyl-Induced Hepatocarcinogenesis In Mice
Genomic Instability In Gadd45a-Deficient Mice
Cyp1a2 Is Not The Primary Enzyme Responsible For 4-Aminobiphenyl-Induced Hepatocarcinogenesis In Mice
Targeted Disruption Of The Microsomal Epoxide Hydrolase Gene. Microsomal Epoxide Hydrolase Is Required For The Carcinogenic Activity Of 7,12- Dimethylbenz[A]anthracene
Cyp1a2 Is Not The Primary Enzyme Responsible For 4-Aminobiphenyl-Induced Hepatocarcinogenesis In Mice
Targeted Disruption Of The Microsomal Epoxide Hydrolase Gene. Microsomal Epoxide Hydrolase Is Required For The Carcinogenic Activity Of 7,12- Dimethylbenz[A]anthracene
Expression Of Cyp2a Genes In Human Liver And Extrahepatic Tissues
Targeted Disruption Of The Microsomal Epoxide Hydrolase Gene. Microsomal Epoxide Hydrolase Is Required For The Carcinogenic Activity Of 7,12- Dimethylbenz[A]anthracene
Expression Of Cyp2a Genes In Human Liver And Extrahepatic Tissues
Amelioration Of Tcdd-Induced Teratogenesis In Aryl Hydrocarbon Receptor (Ahr)-Null Mice
Expression Of Cyp2a Genes In Human Liver And Extrahepatic Tissues
Amelioration Of Tcdd-Induced Teratogenesis In Aryl Hydrocarbon Receptor (Ahr)-Null Mice
The Effect Of Non-Genotoxic Carcinogens, Phenobarbital And Clofibrate, On The Relationship Between Reactive Oxygen Species, Antioxidant Enzyme Expression And Apoptosis.
Amelioration Of Tcdd-Induced Teratogenesis In Aryl Hydrocarbon Receptor (Ahr)-Null Mice
The Effect Of Non-Genotoxic Carcinogens, Phenobarbital And Clofibrate, On The Relationship Between Reactive Oxygen Species, Antioxidant Enzyme Expression And Apoptosis.
Influence Of Aminoguanidine On Parameters Of Liver Injury And Regeneration Induced In Rats By A Necrogenic Dose Of Thioacetamide.
The Effect Of Non-Genotoxic Carcinogens, Phenobarbital And Clofibrate, On The Relationship Between Reactive Oxygen Species, Antioxidant Enzyme Expression And Apoptosis.
Influence Of Aminoguanidine On Parameters Of Liver Injury And Regeneration Induced In Rats By A Necrogenic Dose Of Thioacetamide.
Hla-Dpbeta Residue 69 Plays A Crucial Role In Allorecognition.
Influence Of Aminoguanidine On Parameters Of Liver Injury And Regeneration Induced In Rats By A Necrogenic Dose Of Thioacetamide.
Hla-Dpbeta Residue 69 Plays A Crucial Role In Allorecognition.
Transforming Growth Factor-Beta (Tgf-Beta) And Egf Promote Cord-Like Structures That Indicate Terminal Differentiation Of Fetal Hepatocytes In Primary Culture.
Hla-Dpbeta Residue 69 Plays A Crucial Role In Allorecognition.
Transforming Growth Factor-Beta (Tgf-Beta) And Egf Promote Cord-Like Structures That Indicate Terminal Differentiation Of Fetal Hepatocytes In Primary Culture.
P42/p44 Mitogen-Activated Protein Kinases Activation Is Required For The Insulin-Like Growth Factor-I/insulin Induced Proliferation, But Inhibits Differentiation, In Rat Fetal Brown Adipocytes.
Transforming Growth Factor-Beta (Tgf-Beta) And Egf Promote Cord-Like Structures That Indicate Terminal Differentiation Of Fetal Hepatocytes In Primary Culture.
P42/p44 Mitogen-Activated Protein Kinases Activation Is Required For The Insulin-Like Growth Factor-I/insulin Induced Proliferation, But Inhibits Differentiation, In Rat Fetal Brown Adipocytes.
Flow Cytometric Studies Of Nucleoside Transport Regulation In Single Chromaffin Cells.
P42/p44 Mitogen-Activated Protein Kinases Activation Is Required For The Insulin-Like Growth Factor-I/insulin Induced Proliferation, But Inhibits Differentiation, In Rat Fetal Brown Adipocytes.
Flow Cytometric Studies Of Nucleoside Transport Regulation In Single Chromaffin Cells.
Necrogenic And Regenerative Responses Of Liver Of Newly Weaned Rats Against A Sublethal Dose Of Thioacetamide.
Flow Cytometric Studies Of Nucleoside Transport Regulation In Single Chromaffin Cells.
Necrogenic And Regenerative Responses Of Liver Of Newly Weaned Rats Against A Sublethal Dose Of Thioacetamide.
The Aryl Hydrocarbon Receptor. Studies Using The Ahr-Null Mice
Necrogenic And Regenerative Responses Of Liver Of Newly Weaned Rats Against A Sublethal Dose Of Thioacetamide.
The Aryl Hydrocarbon Receptor. Studies Using The Ahr-Null Mice
Aryl-Hydrocarbon Receptor Is An Inhibitory Regulator Of Lipid Synthesis And Of Commitment To Adipogenesis
The Aryl Hydrocarbon Receptor. Studies Using The Ahr-Null Mice
Aryl-Hydrocarbon Receptor Is An Inhibitory Regulator Of Lipid Synthesis And Of Commitment To Adipogenesis
Characterization Of The Human Dihydropyrimidine Dehydrogenase Gene
Aryl-Hydrocarbon Receptor Is An Inhibitory Regulator Of Lipid Synthesis And Of Commitment To Adipogenesis
Characterization Of The Human Dihydropyrimidine Dehydrogenase Gene
Effect Of Phenobarbital On Hepatic Cyp1a1 And Cyp1a2 In The Ahr-Null Mouse
Characterization Of The Human Dihydropyrimidine Dehydrogenase Gene
Effect Of Phenobarbital On Hepatic Cyp1a1 And Cyp1a2 In The Ahr-Null Mouse
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Caucasian Subjects
Effect Of Phenobarbital On Hepatic Cyp1a1 And Cyp1a2 In The Ahr-Null Mouse
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Caucasian Subjects
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Patients With Colorectal Cancer
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Caucasian Subjects
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Patients With Colorectal Cancer
Nomenclature For Human Dpyd Alleles
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Patients With Colorectal Cancer
Nomenclature For Human Dpyd Alleles
The Involvement Of Aryl Hydrocarbon Receptor In The Activation Of Transforming Growth Factor-Β And Apoptosis
Nomenclature For Human Dpyd Alleles
The Involvement Of Aryl Hydrocarbon Receptor In The Activation Of Transforming Growth Factor-Β And Apoptosis
Microbiological Induced Corrosion In The Paper Industry
The Involvement Of Aryl Hydrocarbon Receptor In The Activation Of Transforming Growth Factor-Β And Apoptosis
Microbiological Induced Corrosion In The Paper Industry
Tnf-Alpha Induces Apoptosis In Rat Fetal Brown Adipocytes In Primary Culture.
Microbiological Induced Corrosion In The Paper Industry
Tnf-Alpha Induces Apoptosis In Rat Fetal Brown Adipocytes In Primary Culture.
Cycloheximide Prevents Apoptosis, Reactive Oxygen Species Production, And Glutathione Depletion Induced By Transforming Growth Factor Beta In Fetal Rat Hepatocytes In Primary Culture.
Tnf-Alpha Induces Apoptosis In Rat Fetal Brown Adipocytes In Primary Culture.
Cycloheximide Prevents Apoptosis, Reactive Oxygen Species Production, And Glutathione Depletion Induced By Transforming Growth Factor Beta In Fetal Rat Hepatocytes In Primary Culture.
Neuronal Mitochondrial Morphology And Transmembrane Potential Are Severely Altered By Hypothyroidism During Rat Brain Development.
Cycloheximide Prevents Apoptosis, Reactive Oxygen Species Production, And Glutathione Depletion Induced By Transforming Growth Factor Beta In Fetal Rat Hepatocytes In Primary Culture.
Neuronal Mitochondrial Morphology And Transmembrane Potential Are Severely Altered By Hypothyroidism During Rat Brain Development.
Age-Dependent Modifications In Rat Hepatocyte Antioxidant Defense Systems.
Neuronal Mitochondrial Morphology And Transmembrane Potential Are Severely Altered By Hypothyroidism During Rat Brain Development.
Age-Dependent Modifications In Rat Hepatocyte Antioxidant Defense Systems.
Rapid Up-Regulation Of Ikappabbeta And Abrogation Of Nf-Kappab Activity In Peritoneal Macrophages Stimulated With Lipopolysaccharide.
Age-Dependent Modifications In Rat Hepatocyte Antioxidant Defense Systems.
Rapid Up-Regulation Of Ikappabbeta And Abrogation Of Nf-Kappab Activity In Peritoneal Macrophages Stimulated With Lipopolysaccharide.
Phenotypic Characterization Of A Candida Albicans Strain Deficient In Its Major Exoglucanase.
Rapid Up-Regulation Of Ikappabbeta And Abrogation Of Nf-Kappab Activity In Peritoneal Macrophages Stimulated With Lipopolysaccharide.
Phenotypic Characterization Of A Candida Albicans Strain Deficient In Its Major Exoglucanase.
Aryl Hydrocarbon Receptor Deficient Mice (Ahr-/-) Exhibit An Accumulation Of Liver Retinylpalmitate
Phenotypic Characterization Of A Candida Albicans Strain Deficient In Its Major Exoglucanase.
Aryl Hydrocarbon Receptor Deficient Mice (Ahr-/-) Exhibit An Accumulation Of Liver Retinylpalmitate
Analysis Of The Selenocysteine (Sec) Trna Population In Mouse Embryonic Stem (Es) Cells Heterozygous For The Sec Trna Gene
Aryl Hydrocarbon Receptor Deficient Mice (Ahr-/-) Exhibit An Accumulation Of Liver Retinylpalmitate
Analysis Of The Selenocysteine (Sec) Trna Population In Mouse Embryonic Stem (Es) Cells Heterozygous For The Sec Trna Gene
Aryl Hydrocarbon Receptor Knockout Mice (Ahr(-/-)) Exhibit Liver Retinoid Accumulation And Reduced Retinoic Acid Metabolism
Analysis Of The Selenocysteine (Sec) Trna Population In Mouse Embryonic Stem (Es) Cells Heterozygous For The Sec Trna Gene
Aryl Hydrocarbon Receptor Knockout Mice (Ahr(-/-)) Exhibit Liver Retinoid Accumulation And Reduced Retinoic Acid Metabolism
Cyp2a6 Gene Polymorphism And Risk Of Liver Cancer And Cirrhosis
Aryl Hydrocarbon Receptor Knockout Mice (Ahr(-/-)) Exhibit Liver Retinoid Accumulation And Reduced Retinoic Acid Metabolism
Cyp2a6 Gene Polymorphism And Risk Of Liver Cancer And Cirrhosis
Selenocysteine Trna([Ser]sec) Levels And Selenium-Dependent Glutathione Peroxidase Activity In Mouse Embryonic Stem Cells Heterozygous For A Targeted Mutation In The Trna([Ser]sec) Gene
Cyp2a6 Gene Polymorphism And Risk Of Liver Cancer And Cirrhosis
Selenocysteine Trna([Ser]sec) Levels And Selenium-Dependent Glutathione Peroxidase Activity In Mouse Embryonic Stem Cells Heterozygous For A Targeted Mutation In The Trna([Ser]sec) Gene
Lack Of Correlation Between Phenotype And Genotype For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In A Family Of Pakistani Origin
Selenocysteine Trna([Ser]sec) Levels And Selenium-Dependent Glutathione Peroxidase Activity In Mouse Embryonic Stem Cells Heterozygous For A Targeted Mutation In The Trna([Ser]sec) Gene
Lack Of Correlation Between Phenotype And Genotype For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In A Family Of Pakistani Origin
Lesions Of Aryl-Hydrocarbon Receptor-Deficient Mice
Lack Of Correlation Between Phenotype And Genotype For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In A Family Of Pakistani Origin
Lesions Of Aryl-Hydrocarbon Receptor-Deficient Mice
Mutations At Codon 974 Of The Dpyd Gene Are A Rare Event
Lesions Of Aryl-Hydrocarbon Receptor-Deficient Mice
Mutations At Codon 974 Of The Dpyd Gene Are A Rare Event
Epidermal Growth Factor, But Not Hepatocyte Growth Factor, Suppresses The Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocytes In Primary Culture.
Mutations At Codon 974 Of The Dpyd Gene Are A Rare Event
Epidermal Growth Factor, But Not Hepatocyte Growth Factor, Suppresses The Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocytes In Primary Culture.
Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocyte Primary Cultures: Involvement Of Reactive Oxygen Intermediates.
Epidermal Growth Factor, But Not Hepatocyte Growth Factor, Suppresses The Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocytes In Primary Culture.
Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocyte Primary Cultures: Involvement Of Reactive Oxygen Intermediates.
Piperonyl Butoxide And Acenaphthylene Induce Cytochrome P450 1a2 And 1b1 Mrna In Aromatic Hydrocarbon-Responsive Receptor Knock-Out Mouse Liver
Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocyte Primary Cultures: Involvement Of Reactive Oxygen Intermediates.
Piperonyl Butoxide And Acenaphthylene Induce Cytochrome P450 1a2 And 1b1 Mrna In Aromatic Hydrocarbon-Responsive Receptor Knock-Out Mouse Liver
Transcriptional Induction Of The Cytochrome P4501a1 Gene By A Thiazolium Compound, Yh439
Piperonyl Butoxide And Acenaphthylene Induce Cytochrome P450 1a2 And 1b1 Mrna In Aromatic Hydrocarbon-Responsive Receptor Knock-Out Mouse Liver
Transcriptional Induction Of The Cytochrome P4501a1 Gene By A Thiazolium Compound, Yh439
The Role Of The Aryl Hydrocarbon Receptor In Animal Development, Physiological Homeostasis And Toxicity Of Tcdd
Transcriptional Induction Of The Cytochrome P4501a1 Gene By A Thiazolium Compound, Yh439
The Role Of The Aryl Hydrocarbon Receptor In Animal Development, Physiological Homeostasis And Toxicity Of Tcdd
Differential Regulation Of Mouse Ah Receptor Gene Expression In Cell Lines Of Different Tissue Origins
The Role Of The Aryl Hydrocarbon Receptor In Animal Development, Physiological Homeostasis And Toxicity Of Tcdd
Differential Regulation Of Mouse Ah Receptor Gene Expression In Cell Lines Of Different Tissue Origins
Molecular Basis Of The Human Dihydropyrimidine Dehydrogenase Deficiency And 5-Fluorouracil Toxicity
Differential Regulation Of Mouse Ah Receptor Gene Expression In Cell Lines Of Different Tissue Origins
Molecular Basis Of The Human Dihydropyrimidine Dehydrogenase Deficiency And 5-Fluorouracil Toxicity
Role Of Cyp2e1 In The Hepatotoxicity Of Acetaminophen
Molecular Basis Of The Human Dihydropyrimidine Dehydrogenase Deficiency And 5-Fluorouracil Toxicity
Role Of Cyp2e1 In The Hepatotoxicity Of Acetaminophen
Aryl-Hydrocarbon Receptor-Deficient Mice Are Resistant To 2,3,7,8-Tetrachlorodibenzo-P-Dioxin-Induced Toxicity
Role Of Cyp2e1 In The Hepatotoxicity Of Acetaminophen
Aryl-Hydrocarbon Receptor-Deficient Mice Are Resistant To 2,3,7,8-Tetrachlorodibenzo-P-Dioxin-Induced Toxicity
Hepatic Fibrosis In Ahr(-/-) Mice
Aryl-Hydrocarbon Receptor-Deficient Mice Are Resistant To 2,3,7,8-Tetrachlorodibenzo-P-Dioxin-Induced Toxicity
Hepatic Fibrosis In Ahr(-/-) Mice
The T/ebp Null Mouse: Thyroid-Specific Enhancer-Binding Protein Is Essential For The Organogenesis Of The Thyroid, Lung, Ventral Forebrain, And Pituitary
Hepatic Fibrosis In Ahr(-/-) Mice
The T/ebp Null Mouse: Thyroid-Specific Enhancer-Binding Protein Is Essential For The Organogenesis Of The Thyroid, Lung, Ventral Forebrain, And Pituitary
Pyrimidines And Cns Regulation
The T/ebp Null Mouse: Thyroid-Specific Enhancer-Binding Protein Is Essential For The Organogenesis Of The Thyroid, Lung, Ventral Forebrain, And Pituitary
Pyrimidines And Cns Regulation
Targeted Disruption Of Specific Cytochromes P450 And Xenobiotic Receptor Genes
Pyrimidines And Cns Regulation
Targeted Disruption Of Specific Cytochromes P450 And Xenobiotic Receptor Genes
Diagnostic-Analysis, Clinical Importance And Molecular-Basis Of Dihydropyrimidine Dehydrogenase-Deficiency
Targeted Disruption Of Specific Cytochromes P450 And Xenobiotic Receptor Genes
Diagnostic-Analysis, Clinical Importance And Molecular-Basis Of Dihydropyrimidine Dehydrogenase-Deficiency
Organization And Evolution Of The Cytochrome P450 Cyp2a-2b-2f Subfamily Gene Cluster On Human Chromosome 19
Diagnostic-Analysis, Clinical Importance And Molecular-Basis Of Dihydropyrimidine Dehydrogenase-Deficiency
Organization And Evolution Of The Cytochrome P450 Cyp2a-2b-2f Subfamily Gene Cluster On Human Chromosome 19
Transforming Growth Factor Beta Modulates Growth And Differentiation Of Fetal Hepatocytes In Primary Culture.
Organization And Evolution Of The Cytochrome P450 Cyp2a-2b-2f Subfamily Gene Cluster On Human Chromosome 19
Transforming Growth Factor Beta Modulates Growth And Differentiation Of Fetal Hepatocytes In Primary Culture.
Differentiation Of Rat Brown Adipocytes During Late Foetal Development: Role Of Insulin-Like Growth Factor I.
Transforming Growth Factor Beta Modulates Growth And Differentiation Of Fetal Hepatocytes In Primary Culture.
Differentiation Of Rat Brown Adipocytes During Late Foetal Development: Role Of Insulin-Like Growth Factor I.
The Role Of Hla-Dp Beta Residue 69 In The Definition Of Antibody-Binding Epitopes.
Differentiation Of Rat Brown Adipocytes During Late Foetal Development: Role Of Insulin-Like Growth Factor I.
The Role Of Hla-Dp Beta Residue 69 In The Definition Of Antibody-Binding Epitopes.
Relationship Between Antioxidant Systems, Intracellular Thiols And Dna Ploidy In Liver Of Rats During Experimental Cirrhogenesis.
The Role Of Hla-Dp Beta Residue 69 In The Definition Of Antibody-Binding Epitopes.
Relationship Between Antioxidant Systems, Intracellular Thiols And Dna Ploidy In Liver Of Rats During Experimental Cirrhogenesis.
Splenic B Lymphocyte Programmed Cell Death Is Prevented By Nitric Oxide Release Through Mechanisms Involving Sustained Bcl-2 Levels.
Relationship Between Antioxidant Systems, Intracellular Thiols And Dna Ploidy In Liver Of Rats During Experimental Cirrhogenesis.
Splenic B Lymphocyte Programmed Cell Death Is Prevented By Nitric Oxide Release Through Mechanisms Involving Sustained Bcl-2 Levels.
Two Different Subpopulations Of Ig-Bearing Cells In Lymphoid Organs Of Rainbow Trout.
Splenic B Lymphocyte Programmed Cell Death Is Prevented By Nitric Oxide Release Through Mechanisms Involving Sustained Bcl-2 Levels.
Two Different Subpopulations Of Ig-Bearing Cells In Lymphoid Organs Of Rainbow Trout.
A Genetic Polymorphism In Coumarin 7-Hydroxylation: Sequence Of The Human Cyp2a Genes And Identification Of Variant Cyp2a6 Alleles
Two Different Subpopulations Of Ig-Bearing Cells In Lymphoid Organs Of Rainbow Trout.
A Genetic Polymorphism In Coumarin 7-Hydroxylation: Sequence Of The Human Cyp2a Genes And Identification Of Variant Cyp2a6 Alleles
Comparison Of Substrate Metabolism By Wild Type Cyp2d6 Protein And A Variant Containing Methionine, Not Valine, At Position 374
A Genetic Polymorphism In Coumarin 7-Hydroxylation: Sequence Of The Human Cyp2a Genes And Identification Of Variant Cyp2a6 Alleles
Comparison Of Substrate Metabolism By Wild Type Cyp2d6 Protein And A Variant Containing Methionine, Not Valine, At Position 374
Human Polymorphism In Drug Metabolism: Mutation In The Dihydropyrimidine Dehydrogenase Gene Results In Exon Skipping And Thymine Uracilurea
Comparison Of Substrate Metabolism By Wild Type Cyp2d6 Protein And A Variant Containing Methionine, Not Valine, At Position 374
Human Polymorphism In Drug Metabolism: Mutation In The Dihydropyrimidine Dehydrogenase Gene Results In Exon Skipping And Thymine Uracilurea
Genetic Polymorphism Of Cyp2c9 And Its Effect On Warfarin Maintenance Dose Requirement In Patients Undergoing Anticoagulation Therapy
Human Polymorphism In Drug Metabolism: Mutation In The Dihydropyrimidine Dehydrogenase Gene Results In Exon Skipping And Thymine Uracilurea
Genetic Polymorphism Of Cyp2c9 And Its Effect On Warfarin Maintenance Dose Requirement In Patients Undergoing Anticoagulation Therapy
Correlation Between Catalytic Activity And Protein Content For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In Human Lymphocytes
Genetic Polymorphism Of Cyp2c9 And Its Effect On Warfarin Maintenance Dose Requirement In Patients Undergoing Anticoagulation Therapy
Correlation Between Catalytic Activity And Protein Content For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In Human Lymphocytes
Neonatal Lethality Associated With Respiratory Distress In Mice Lacking Cytochrome P450 1a2
Correlation Between Catalytic Activity And Protein Content For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In Human Lymphocytes
Neonatal Lethality Associated With Respiratory Distress In Mice Lacking Cytochrome P450 1a2
Xenobiotic Receptor Knockout Mice
Neonatal Lethality Associated With Respiratory Distress In Mice Lacking Cytochrome P450 1a2
Xenobiotic Receptor Knockout Mice
Immune System Impairment And Hepatic Fibrosis In Mice Lacking The Dioxin-Binding Ah Receptor
Xenobiotic Receptor Knockout Mice
Immune System Impairment And Hepatic Fibrosis In Mice Lacking The Dioxin-Binding Ah Receptor
Targeted Disruption Of The Α Isoform Of The Peroxisome Proliferator- Activated Receptor Gene In Mice Results In Abolishment Of The Pleiotropic Effects Of Peroxisome Proliferators
Immune System Impairment And Hepatic Fibrosis In Mice Lacking The Dioxin-Binding Ah Receptor
Targeted Disruption Of The Α Isoform Of The Peroxisome Proliferator- Activated Receptor Gene In Mice Results In Abolishment Of The Pleiotropic Effects Of Peroxisome Proliferators
Cocaine-Induced Liver Injury In Mice Elicits Specific Changes In Dna Ploidy And Induces Programmed Death Of Hepatocytes
Targeted Disruption Of The Α Isoform Of The Peroxisome Proliferator- Activated Receptor Gene In Mice Results In Abolishment Of The Pleiotropic Effects Of Peroxisome Proliferators
Cocaine-Induced Liver Injury In Mice Elicits Specific Changes In Dna Ploidy And Induces Programmed Death Of Hepatocytes
Yeast Exo-Beta-Glucanases Can Be Used As Efficient And Readily Detectable Reporter Genes In Saccharomyces Cerevisiae.
Cocaine-Induced Liver Injury In Mice Elicits Specific Changes In Dna Ploidy And Induces Programmed Death Of Hepatocytes
Yeast Exo-Beta-Glucanases Can Be Used As Efficient And Readily Detectable Reporter Genes In Saccharomyces Cerevisiae.
Molecular Analysis Of An Hla-Dp Mutant Cell Line Selected For Its Resistance To Killing By Hla-Dpw2-Specific T-Cell Clones.
Yeast Exo-Beta-Glucanases Can Be Used As Efficient And Readily Detectable Reporter Genes In Saccharomyces Cerevisiae.
Molecular Analysis Of An Hla-Dp Mutant Cell Line Selected For Its Resistance To Killing By Hla-Dpw2-Specific T-Cell Clones.
Cdna Cloning And Chromosome Mapping Of Human Dihydropyrimidine Dehydrogenase, An Enzyme Associated With 5-Fluorouracil Toxicity And Congenital Thymine Uraciluria
Molecular Analysis Of An Hla-Dp Mutant Cell Line Selected For Its Resistance To Killing By Hla-Dpw2-Specific T-Cell Clones.
Cdna Cloning And Chromosome Mapping Of Human Dihydropyrimidine Dehydrogenase, An Enzyme Associated With 5-Fluorouracil Toxicity And Congenital Thymine Uraciluria
Assignment Of The Human Dihydropyrimidine Dehydrogenase Gene (Dpyd) To Chromosome Region 1p22 By Fluorescence In Situ Hybridization
Cdna Cloning And Chromosome Mapping Of Human Dihydropyrimidine Dehydrogenase, An Enzyme Associated With 5-Fluorouracil Toxicity And Congenital Thymine Uraciluria
Assignment Of The Human Dihydropyrimidine Dehydrogenase Gene (Dpyd) To Chromosome Region 1p22 By Fluorescence In Situ Hybridization
Relationship Between Genomic Dna Ploidy And Parameters Of Liver Damage During Necrosis And Regeneration Induced By Thioacetamide
Assignment Of The Human Dihydropyrimidine Dehydrogenase Gene (Dpyd) To Chromosome Region 1p22 By Fluorescence In Situ Hybridization
Relationship Between Genomic Dna Ploidy And Parameters Of Liver Damage During Necrosis And Regeneration Induced By Thioacetamide
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Relationship Between Genomic Dna Ploidy And Parameters Of Liver Damage During Necrosis And Regeneration Induced By Thioacetamide
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Differential Proliferative Response Of Cultured Fetal And Regenerating Hepatocytes To Growth Factors And Hormones.
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Differential Proliferative Response Of Cultured Fetal And Regenerating Hepatocytes To Growth Factors And Hormones.
Flow Cytometric Analysis Of African Swine Fever Virus-Induced Plasma Membrane Proteins And Their Humoral Immune Response In Infected Pigs.
Differential Proliferative Response Of Cultured Fetal And Regenerating Hepatocytes To Growth Factors And Hormones.
Flow Cytometric Analysis Of African Swine Fever Virus-Induced Plasma Membrane Proteins And Their Humoral Immune Response In Infected Pigs.
Flow Cytometric Analysis Of Saccharomyces Cerevisiae Autolytic Mutants And Protoplasts.
Flow Cytometric Analysis Of African Swine Fever Virus-Induced Plasma Membrane Proteins And Their Humoral Immune Response In Infected Pigs.
Flow Cytometric Analysis Of Saccharomyces Cerevisiae Autolytic Mutants And Protoplasts.
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Flow Cytometric Analysis Of Saccharomyces Cerevisiae Autolytic Mutants And Protoplasts.
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
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Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Not Available
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
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Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Neurotoxic Amyloid Β‐peptide And Tau Produce Cytokine‐like Effects On Pmca In Glioblastoma Cell Lines, Enhancing Its Activity And Isoforms Expression
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