PUBLICACIÓN

Modulation of the sarcoplasmic reticulum (Ca2+ + Mg2+)-ATPase by pentobarbital

ACCEDER A LA PUBLICACIÓN: Scopus

FERNANDEZSALGUERO, P, HENAO, F, LAYNEZ, J, GUTIERREZMERINO, C

1990 BBA - Biomembranes

CITAS

15

DOI

10.1016/0005-2736(90)90397-7

EID

2-s2.0-0025139331

ISSN

0005-2736

BIBTEX

@article{RID:0916141122147-90, title = {MODULATION OF THE SARCOPLASMIC-RETICULUM (CA-2+ + MG-2+)-ATPASE BY PENTOBARBITAL}, journal = {Biochimica Et Biophysica Acta}, year = {1990}, author = {FERNANDEZSALGUERO, P and HENAO, F and LAYNEZ, J and GUTIERREZMERINO, C}, volume = {1022}, number = {1}, pages = {33-40} }

AUTORES DE LA UEX

Pedro María Fernández Salguero

OTRAS PUBLICACIONES DE INTERÉS

Regulation Of Phosphorylase B By Amp: The Effects Of Enzyme Concentration
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment
Introducción: Carta Del Rector De La Uex
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment
Introducción: Carta Del Rector De La Uex
An Update Of Kaempferol Protection Against Brain Damage Induced By Ischemia-Reperfusion And By 3-Nitropropionic Acid
Introducción: Carta Del Rector De La Uex
An Update Of Kaempferol Protection Against Brain Damage Induced By Ischemia-Reperfusion And By 3-Nitropropionic Acid
The Ah Receptor From Toxicity To Therapeutics: Report From The 5th Ahr Meeting At Penn State University, Usa, June 2022
An Update Of Kaempferol Protection Against Brain Damage Induced By Ischemia-Reperfusion And By 3-Nitropropionic Acid
The Ah Receptor From Toxicity To Therapeutics: Report From The 5th Ahr Meeting At Penn State University, Usa, June 2022
Store-Operated Calcium Entry Inhibition And Plasma Membrane Calcium Pump Upregulation Contribute To The Maintenance Of Resting Cytosolic Calcium Concentration In A1-Like Astrocytes
The Ah Receptor From Toxicity To Therapeutics: Report From The 5th Ahr Meeting At Penn State University, Usa, June 2022
Store-Operated Calcium Entry Inhibition And Plasma Membrane Calcium Pump Upregulation Contribute To The Maintenance Of Resting Cytosolic Calcium Concentration In A1-Like Astrocytes
Brain Hydrophobic Peptides Antagonists Of Neurotoxic Amyloid Β Peptide Monomers/oligomers–protein Interactions
Store-Operated Calcium Entry Inhibition And Plasma Membrane Calcium Pump Upregulation Contribute To The Maintenance Of Resting Cytosolic Calcium Concentration In A1-Like Astrocytes
Brain Hydrophobic Peptides Antagonists Of Neurotoxic Amyloid Β Peptide Monomers/oligomers–protein Interactions
Hexa-Histidine, A Peptide With Versatile Applications In The Study Of Amyloid-Β(1–42) Molecular Mechanisms Of Action
Brain Hydrophobic Peptides Antagonists Of Neurotoxic Amyloid Β Peptide Monomers/oligomers–protein Interactions
Hexa-Histidine, A Peptide With Versatile Applications In The Study Of Amyloid-Β(1–42) Molecular Mechanisms Of Action
Are There Lipid Membrane-Domain Subtypes In Neurons With Different Roles In Calcium Signaling?
Hexa-Histidine, A Peptide With Versatile Applications In The Study Of Amyloid-Β(1–42) Molecular Mechanisms Of Action
Are There Lipid Membrane-Domain Subtypes In Neurons With Different Roles In Calcium Signaling?
Biochemical And Biophysical Characterization Of The Caveolin-2 Interaction With Membranes And Analysis Of The Protein Structural Alteration By The Presence Of Cholesterol
Are There Lipid Membrane-Domain Subtypes In Neurons With Different Roles In Calcium Signaling?
Biochemical And Biophysical Characterization Of The Caveolin-2 Interaction With Membranes And Analysis Of The Protein Structural Alteration By The Presence Of Cholesterol
Structural Features Of Cytochrome B5 –cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Biochemical And Biophysical Characterization Of The Caveolin-2 Interaction With Membranes And Analysis Of The Protein Structural Alteration By The Presence Of Cholesterol
Structural Features Of Cytochrome B5 –cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Inhibition Of Serca And Pmca Ca2+-Atpase Activities By Polyoxotungstates
Structural Features Of Cytochrome B5 –cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Inhibition Of Serca And Pmca Ca2+-Atpase Activities By Polyoxotungstates
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Inhibition Of Serca And Pmca Ca2+-Atpase Activities By Polyoxotungstates
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Special Issue “molecular And Cellular Mechanisms Of Action Of Markers Of Tissue Degeneration”
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Special Issue “molecular And Cellular Mechanisms Of Action Of Markers Of Tissue Degeneration”
Design And Experimental Evaluation Of A Peptide Antagonist Against Amyloid Β(1–42) Interactions With Calmodulin And Calbindin-D28k
Special Issue “molecular And Cellular Mechanisms Of Action Of Markers Of Tissue Degeneration”
Design And Experimental Evaluation Of A Peptide Antagonist Against Amyloid Β(1–42) Interactions With Calmodulin And Calbindin-D28k
Kaempferol Prevents The Activation Of Complement C3 Protein And The Generation Of Reactive A1 Astrocytes That Mediate Rat Brain Degeneration Induced By 3-Nitropropionic Acid
Design And Experimental Evaluation Of A Peptide Antagonist Against Amyloid Β(1–42) Interactions With Calmodulin And Calbindin-D28k
Kaempferol Prevents The Activation Of Complement C3 Protein And The Generation Of Reactive A1 Astrocytes That Mediate Rat Brain Degeneration Induced By 3-Nitropropionic Acid
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Kaempferol Prevents The Activation Of Complement C3 Protein And The Generation Of Reactive A1 Astrocytes That Mediate Rat Brain Degeneration Induced By 3-Nitropropionic Acid
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
From Nucleus To Organs: Insights Of Aryl Hydrocarbon Receptor Molecular Mechanisms
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Liver Regeneration After Partial Hepatectomy Is Improved In The Absence Of Aryl Hydrocarbon Receptor
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Aryl Hydrocarbon Receptor: From Homeostasis To Tumor Progression
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Gold Compounds Inhibit The Ca2+-Atpase Activity Of Brain Pmca And Human Neuroblastoma Sh-Sy5y Cells And Decrease Cell Viability
Aryl Hydrocarbon Receptor Blocks Aging-Induced Senescence In The Liver And Fibroblast Cells
Gold Compounds Inhibit The Ca2+-Atpase Activity Of Brain Pmca And Human Neuroblastoma Sh-Sy5y Cells And Decrease Cell Viability
The Relevance Of Amyloid Β-Calmodulin Complexation In Neurons And Brain Degeneration In Alzheimer’s Disease
Gold Compounds Inhibit The Ca2+-Atpase Activity Of Brain Pmca And Human Neuroblastoma Sh-Sy5y Cells And Decrease Cell Viability
The Relevance Of Amyloid Β-Calmodulin Complexation In Neurons And Brain Degeneration In Alzheimer’s Disease
Binding Of Amyloid Β(1–42)‐calmodulin Complexes To Plasma Membrane Lipid Rafts In Cerebellar Granule Neurons Alters Resting Cytosolic Calcium Homeostasis
The Relevance Of Amyloid Β-Calmodulin Complexation In Neurons And Brain Degeneration In Alzheimer’s Disease
Binding Of Amyloid Β(1–42)‐calmodulin Complexes To Plasma Membrane Lipid Rafts In Cerebellar Granule Neurons Alters Resting Cytosolic Calcium Homeostasis
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Binding Of Amyloid Β(1–42)‐calmodulin Complexes To Plasma Membrane Lipid Rafts In Cerebellar Granule Neurons Alters Resting Cytosolic Calcium Homeostasis
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
The Aryl Hydrocarbon Receptor Promotes Differentiation During Mouse Preimplantational Embryo Development
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function
Loss Of Aryl Hydrocarbon Receptor Favors K-Rasg12d-Driven Non-Small Cell Lung Cancer
Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function
Structural Features Of Cytochrome B5–cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Aryl Hydrocarbon Receptor Controls Skin Homeostasis, Regeneration, And Hair Follicle Cycling By Adjusting Epidermal Stem Cell Function
Structural Features Of Cytochrome B5–cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Human Erythrocytes Exposure To Juglone Leads To An Increase Of Superoxide Anion Production Associated With Cytochrome B5 Reductase Uncoupling
Structural Features Of Cytochrome B5–cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Human Erythrocytes Exposure To Juglone Leads To An Increase Of Superoxide Anion Production Associated With Cytochrome B5 Reductase Uncoupling
Early Reactive A1 Astrocytes Induction By The Neurotoxin 3-Nitropropionic Acid In Rat Brain
Human Erythrocytes Exposure To Juglone Leads To An Increase Of Superoxide Anion Production Associated With Cytochrome B5 Reductase Uncoupling
Early Reactive A1 Astrocytes Induction By The Neurotoxin 3-Nitropropionic Acid In Rat Brain
Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor
Early Reactive A1 Astrocytes Induction By The Neurotoxin 3-Nitropropionic Acid In Rat Brain
Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor
Modulation Of Cyp2c9 Activity And Hydrogen Peroxide Production By Cytochrome B 5
Alu Retrotransposons Modulate Nanog Expression Through Dynamic Changes In Regional Chromatin Conformation Via Aryl Hydrocarbon Receptor
Modulation Of Cyp2c9 Activity And Hydrogen Peroxide Production By Cytochrome B 5
Ligand Accessibility To Heme Cytochrome B 5 Coordinating Sphere And Enzymatic Activity Enhancement Upon Tyrosine Ionization
Modulation Of Cyp2c9 Activity And Hydrogen Peroxide Production By Cytochrome B 5
Ligand Accessibility To Heme Cytochrome B 5 Coordinating Sphere And Enzymatic Activity Enhancement Upon Tyrosine Ionization
Methylene Blue Blocks And Reverses The Inhibitory Effect Of Tau On Pmca Function
Ligand Accessibility To Heme Cytochrome B 5 Coordinating Sphere And Enzymatic Activity Enhancement Upon Tyrosine Ionization
Methylene Blue Blocks And Reverses The Inhibitory Effect Of Tau On Pmca Function
Lack Of The Aryl Hydrocarbon Receptor Accelerates Aging In Mice
Methylene Blue Blocks And Reverses The Inhibitory Effect Of Tau On Pmca Function
Lack Of The Aryl Hydrocarbon Receptor Accelerates Aging In Mice
Vav Proteins Maintain Epithelial Traits In Breast Cancer Cells Using Mir-200c-Dependent And Independent Mechanisms
Lack Of The Aryl Hydrocarbon Receptor Accelerates Aging In Mice
Vav Proteins Maintain Epithelial Traits In Breast Cancer Cells Using Mir-200c-Dependent And Independent Mechanisms
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Vav Proteins Maintain Epithelial Traits In Breast Cancer Cells Using Mir-200c-Dependent And Independent Mechanisms
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Methyl-Β-Cyclodextrin Impairs The Phosphorylation Of The Β2 Subunit Of L-Type Calcium Channels And Cytosolic Calcium Homeostasis In Mature Cerebellar Granule Neurons
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Methyl-Β-Cyclodextrin Impairs The Phosphorylation Of The Β2 Subunit Of L-Type Calcium Channels And Cytosolic Calcium Homeostasis In Mature Cerebellar Granule Neurons
Creatine Protects Against Cytosolic Calcium Dysregulation, Mitochondrial Depolarization And Increase Of Reactive Oxygen Species Production In Rotenone-Induced Cell Death Of Cerebellar Granule Neurons
Methyl-Β-Cyclodextrin Impairs The Phosphorylation Of The Β2 Subunit Of L-Type Calcium Channels And Cytosolic Calcium Homeostasis In Mature Cerebellar Granule Neurons
Creatine Protects Against Cytosolic Calcium Dysregulation, Mitochondrial Depolarization And Increase Of Reactive Oxygen Species Production In Rotenone-Induced Cell Death Of Cerebellar Granule Neurons
Cytochrome B5 Reductase Is The Component From Neuronal Synaptic Plasma Membrane Vesicles That Generates Superoxide Anion Upon Stimulation By Cytochrome C
Creatine Protects Against Cytosolic Calcium Dysregulation, Mitochondrial Depolarization And Increase Of Reactive Oxygen Species Production In Rotenone-Induced Cell Death Of Cerebellar Granule Neurons
Cytochrome B5 Reductase Is The Component From Neuronal Synaptic Plasma Membrane Vesicles That Generates Superoxide Anion Upon Stimulation By Cytochrome C
Topography Of Human Cytochrome B5/cytochrome B5 Reductase Interacting Domain And Redox Alterations Upon Complex Formation
Cytochrome B5 Reductase Is The Component From Neuronal Synaptic Plasma Membrane Vesicles That Generates Superoxide Anion Upon Stimulation By Cytochrome C
Topography Of Human Cytochrome B5/cytochrome B5 Reductase Interacting Domain And Redox Alterations Upon Complex Formation
Peroxidase-Like Activity Of Cytochrome B5 Is Triggered Upon Hemichrome Formation In Alkaline Ph
Topography Of Human Cytochrome B5/cytochrome B5 Reductase Interacting Domain And Redox Alterations Upon Complex Formation
Peroxidase-Like Activity Of Cytochrome B5 Is Triggered Upon Hemichrome Formation In Alkaline Ph
Methylene Blue Activates The Pmca Activity And Cross-Interacts With Amyloid Β-Peptide, Blocking Aβ-Mediated Pmca Inhibition
Peroxidase-Like Activity Of Cytochrome B5 Is Triggered Upon Hemichrome Formation In Alkaline Ph
Methylene Blue Activates The Pmca Activity And Cross-Interacts With Amyloid Β-Peptide, Blocking Aβ-Mediated Pmca Inhibition
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Methylene Blue Activates The Pmca Activity And Cross-Interacts With Amyloid Β-Peptide, Blocking Aβ-Mediated Pmca Inhibition
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value
Identification Of New Activators Of Mitochondrial Fusion Reveals A Link Between Mitochondrial Morphology And Pyrimidine Metabolism
The Aryl Hydrocarbon Receptor In The Crossroad Of Signalling Networks With Therapeutic Value
Identification Of New Activators Of Mitochondrial Fusion Reveals A Link Between Mitochondrial Morphology And Pyrimidine Metabolism
Histone H4 Acetylation Regulates Behavioral Inter-Individual Variability In Zebrafish
Identification Of New Activators Of Mitochondrial Fusion Reveals A Link Between Mitochondrial Morphology And Pyrimidine Metabolism
Histone H4 Acetylation Regulates Behavioral Inter-Individual Variability In Zebrafish
Phospholipids And Calmodulin Modulate The Inhibition Of Pmca Activity By Tau
Histone H4 Acetylation Regulates Behavioral Inter-Individual Variability In Zebrafish
Phospholipids And Calmodulin Modulate The Inhibition Of Pmca Activity By Tau
High Affinity Binding Of Amyloid Β-Peptide To Calmodulin: Structural And Functional Implications
Phospholipids And Calmodulin Modulate The Inhibition Of Pmca Activity By Tau
High Affinity Binding Of Amyloid Β-Peptide To Calmodulin: Structural And Functional Implications
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
High Affinity Binding Of Amyloid Β-Peptide To Calmodulin: Structural And Functional Implications
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis
A Dioxin-Like Compound Induces Hyperplasia And Branching Morphogenesis In Mouse Mammary Gland, Through Alterations In Tgf-Β1 And Aryl Hydrocarbon Receptor Signaling
Dioxin Receptor Adjusts Liver Regeneration After Acute Toxic Injury And Protects Against Liver Carcinogenesis
A Dioxin-Like Compound Induces Hyperplasia And Branching Morphogenesis In Mouse Mammary Gland, Through Alterations In Tgf-Β1 And Aryl Hydrocarbon Receptor Signaling
Correlation Between The Potency Of Flavonoids For Cytochrome C Reduction And Inhibition Of Cardiolipin-Induced Peroxidase Activity
A Dioxin-Like Compound Induces Hyperplasia And Branching Morphogenesis In Mouse Mammary Gland, Through Alterations In Tgf-Β1 And Aryl Hydrocarbon Receptor Signaling
Correlation Between The Potency Of Flavonoids For Cytochrome C Reduction And Inhibition Of Cardiolipin-Induced Peroxidase Activity
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis.
Correlation Between The Potency Of Flavonoids For Cytochrome C Reduction And Inhibition Of Cardiolipin-Induced Peroxidase Activity
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis.
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor.
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis.
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor.
New Trends In Aryl Hydrocarbon Receptor Biology.
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor.
New Trends In Aryl Hydrocarbon Receptor Biology.
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements.
New Trends In Aryl Hydrocarbon Receptor Biology.
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements.
High Expression Of Cytochrome B 5 Reductase Isoform 3/cytochrome B 5 System In The Cerebellum And Pyramidal Neurons Of Adult Rat Brain
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements.
High Expression Of Cytochrome B 5 Reductase Isoform 3/cytochrome B 5 System In The Cerebellum And Pyramidal Neurons Of Adult Rat Brain
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
High Expression Of Cytochrome B 5 Reductase Isoform 3/cytochrome B 5 System In The Cerebellum And Pyramidal Neurons Of Adult Rat Brain
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis
New Trends In Aryl Hydrocarbon Receptor Biology
Cd69 Controls The Uptake Of L-Tryptophan Through Lat1-Cd98 And Ahr-Dependent Secretion Of Il-22 In Psoriasis
New Trends In Aryl Hydrocarbon Receptor Biology
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements
New Trends In Aryl Hydrocarbon Receptor Biology
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements
Skin Response To A Carcinogen Involves The Xenobiotic Receptor Pregnane X Receptor
Rna-Seq Analysis To Measure The Expression Of Sine Retroelements
Skin Response To A Carcinogen Involves The Xenobiotic Receptor Pregnane X Receptor
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
Skin Response To A Carcinogen Involves The Xenobiotic Receptor Pregnane X Receptor
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells
Bmi1 Regulates Murine Intestinal Stem Cell Proliferation And Self-Renewal Downstream Of Notch
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells
Bmi1 Regulates Murine Intestinal Stem Cell Proliferation And Self-Renewal Downstream Of Notch
Cytochrome B5 As A Pleiotropic Metabolic Modulator In Mammalian Cells
Bmi1 Regulates Murine Intestinal Stem Cell Proliferation And Self-Renewal Downstream Of Notch
Cytochrome B5 As A Pleiotropic Metabolic Modulator In Mammalian Cells
The Decrease Of Nad(P)h:quinone Oxidoreductase 1 Activity And Increase Of Ros Production By Nadph Oxidases Are Early Biomarkers In Doxorubicin Cardiotoxicity
Cytochrome B5 As A Pleiotropic Metabolic Modulator In Mammalian Cells
The Decrease Of Nad(P)h:quinone Oxidoreductase 1 Activity And Increase Of Ros Production By Nadph Oxidases Are Early Biomarkers In Doxorubicin Cardiotoxicity
L-Kynurenine/aryl Hydrocarbon Receptor Pathway Mediates Brain Damage After Experimental Stroke
The Decrease Of Nad(P)h:quinone Oxidoreductase 1 Activity And Increase Of Ros Production By Nadph Oxidases Are Early Biomarkers In Doxorubicin Cardiotoxicity
L-Kynurenine/aryl Hydrocarbon Receptor Pathway Mediates Brain Damage After Experimental Stroke
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol
L-Kynurenine/aryl Hydrocarbon Receptor Pathway Mediates Brain Damage After Experimental Stroke
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol
Aryl Hydrocarbon Receptor-Dependent Induction Of Liver Fibrosis By Dioxin
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol
Aryl Hydrocarbon Receptor-Dependent Induction Of Liver Fibrosis By Dioxin
Thymbra Capitata Essential Oil Prevents Cell Death Induced By 4-Hydroxy-2-Nonenal In Neonatal Rat Cardiac Myocytes
Aryl Hydrocarbon Receptor-Dependent Induction Of Liver Fibrosis By Dioxin
Thymbra Capitata Essential Oil Prevents Cell Death Induced By 4-Hydroxy-2-Nonenal In Neonatal Rat Cardiac Myocytes
Apoptotic Cell Death In Cultured Cardiomyocytes Following Exposure To Low Concentrations Of 4-Hydroxy-2-Nonenal
Thymbra Capitata Essential Oil Prevents Cell Death Induced By 4-Hydroxy-2-Nonenal In Neonatal Rat Cardiac Myocytes
Apoptotic Cell Death In Cultured Cardiomyocytes Following Exposure To Low Concentrations Of 4-Hydroxy-2-Nonenal
Caveolin-Rich Lipid Rafts Of The Plasma Membrane Of Mature Cerebellar Granule Neurons Are Microcompartments For Calcium/reactive Oxygen And Nitrogen Species Cross-Talk Signaling
Apoptotic Cell Death In Cultured Cardiomyocytes Following Exposure To Low Concentrations Of 4-Hydroxy-2-Nonenal
Caveolin-Rich Lipid Rafts Of The Plasma Membrane Of Mature Cerebellar Granule Neurons Are Microcompartments For Calcium/reactive Oxygen And Nitrogen Species Cross-Talk Signaling
Purified Nadh-Cytochrome B5 Reductase Is A Novel Superoxide Anion Source Inhibited By Apocynin: Sensitivity To Nitric Oxide And Peroxynitrite
Caveolin-Rich Lipid Rafts Of The Plasma Membrane Of Mature Cerebellar Granule Neurons Are Microcompartments For Calcium/reactive Oxygen And Nitrogen Species Cross-Talk Signaling
Purified Nadh-Cytochrome B5 Reductase Is A Novel Superoxide Anion Source Inhibited By Apocynin: Sensitivity To Nitric Oxide And Peroxynitrite
The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis
Purified Nadh-Cytochrome B5 Reductase Is A Novel Superoxide Anion Source Inhibited By Apocynin: Sensitivity To Nitric Oxide And Peroxynitrite
The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis
Aryl Hydrocarbon Receptor Contributes To The Mek/erk-Dependent Maintenance Of The Immature State Of Human Dendritic Cells
The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis
Aryl Hydrocarbon Receptor Contributes To The Mek/erk-Dependent Maintenance Of The Immature State Of Human Dendritic Cells
The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway
Aryl Hydrocarbon Receptor Contributes To The Mek/erk-Dependent Maintenance Of The Immature State Of Human Dendritic Cells
The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway
Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition
The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway
Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition
Oculomotor Deficits In Aryl Hydrocarbon Receptor Null Mouse
Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition
Oculomotor Deficits In Aryl Hydrocarbon Receptor Null Mouse
Aryl Hydrocarbon Receptor-Induced Adrenomedullin Mediates Cigarette Smoke Carcinogenicity In Humans And Mice
Oculomotor Deficits In Aryl Hydrocarbon Receptor Null Mouse
Aryl Hydrocarbon Receptor-Induced Adrenomedullin Mediates Cigarette Smoke Carcinogenicity In Humans And Mice
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis By Disruption Of Intracellular Calcium Homeostasis In Human Neuronal Cell Line Shsy5y
Aryl Hydrocarbon Receptor-Induced Adrenomedullin Mediates Cigarette Smoke Carcinogenicity In Humans And Mice
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis By Disruption Of Intracellular Calcium Homeostasis In Human Neuronal Cell Line Shsy5y
Stimulation And Clustering Of Cytochrome B 5 Reductase In Caveolin-Rich Lipid Microdomains Is An Early Event In Oxidative Stress-Mediated Apoptosis Of Cerebellar Granule Neurons
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis By Disruption Of Intracellular Calcium Homeostasis In Human Neuronal Cell Line Shsy5y
Stimulation And Clustering Of Cytochrome B 5 Reductase In Caveolin-Rich Lipid Microdomains Is An Early Event In Oxidative Stress-Mediated Apoptosis Of Cerebellar Granule Neurons
L-Type Voltage-Operated Calcium Channels, N-Methyl-D-Aspartate Receptors And Neuronal Nitric-Oxide Synthase Form A Calcium/redox Nano-Transducer Within Lipid Rafts
Stimulation And Clustering Of Cytochrome B 5 Reductase In Caveolin-Rich Lipid Microdomains Is An Early Event In Oxidative Stress-Mediated Apoptosis Of Cerebellar Granule Neurons
L-Type Voltage-Operated Calcium Channels, N-Methyl-D-Aspartate Receptors And Neuronal Nitric-Oxide Synthase Form A Calcium/redox Nano-Transducer Within Lipid Rafts
Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
L-Type Voltage-Operated Calcium Channels, N-Methyl-D-Aspartate Receptors And Neuronal Nitric-Oxide Synthase Form A Calcium/redox Nano-Transducer Within Lipid Rafts
Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
Complex I And Cytochrome C Are Molecular Targets Of Flavonoids That Inhibit Hydrogen Peroxide Production By Mitochondria
Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
Complex I And Cytochrome C Are Molecular Targets Of Flavonoids That Inhibit Hydrogen Peroxide Production By Mitochondria
Neuroprotective Actions Of Flavonoids
Complex I And Cytochrome C Are Molecular Targets Of Flavonoids That Inhibit Hydrogen Peroxide Production By Mitochondria
Neuroprotective Actions Of Flavonoids
Dioxin Receptor And Slug Transcription Factors Regulate The Insulator Activity Of B1 Sine Retrotransposons Via An Rna Polymerase Switch
Neuroprotective Actions Of Flavonoids
Dioxin Receptor And Slug Transcription Factors Regulate The Insulator Activity Of B1 Sine Retrotransposons Via An Rna Polymerase Switch
The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling
Dioxin Receptor And Slug Transcription Factors Regulate The Insulator Activity Of B1 Sine Retrotransposons Via An Rna Polymerase Switch
The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling
Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse
The Ahr Regulates Cell Adhesion And Migration By Interacting With Oncogene And Growth Factor-Dependent Signaling
Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse
Transcriptional Factor Aryl Hydrocarbon Receptor (Ahr) Controls Cardiovascular And Respiratory Functions By Regulating The Expression Of The Vav3 Proto-Oncogene
Aryl Hydrocarbon Receptor-Dependent Induction Of Apoptosis By 2,3,7,8-Tetrachlorodibenzo-P-Dioxin In Cerebellar Granule Cells From Mouse
Transcriptional Factor Aryl Hydrocarbon Receptor (Ahr) Controls Cardiovascular And Respiratory Functions By Regulating The Expression Of The Vav3 Proto-Oncogene
Erratum: Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane (Free Radical Biology & Medicine (2011) 50 (323-336))
Transcriptional Factor Aryl Hydrocarbon Receptor (Ahr) Controls Cardiovascular And Respiratory Functions By Regulating The Expression Of The Vav3 Proto-Oncogene
Erratum: Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane (Free Radical Biology & Medicine (2011) 50 (323-336))
Inhibition By 4-Hydroxynonenal (Hne) Of Ca 2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
Erratum: Inhibition By 4-Hydroxynonenal (Hne) Of Ca2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane (Free Radical Biology & Medicine (2011) 50 (323-336))
Inhibition By 4-Hydroxynonenal (Hne) Of Ca 2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
Early Disruption Of The Actin Cytoskeleton In Cultured Cerebellar Granule Neurons Exposed To 3-Morpholinosydnonimine-Oxidative Stress Is Linked To Alterations Of The Cytosolic Calcium Concentration
Inhibition By 4-Hydroxynonenal (Hne) Of Ca 2+ Transport By Serca1a: Low Concentrations Of Hne Open Protein-Mediated Leaks In The Membrane
Early Disruption Of The Actin Cytoskeleton In Cultured Cerebellar Granule Neurons Exposed To 3-Morpholinosydnonimine-Oxidative Stress Is Linked To Alterations Of The Cytosolic Calcium Concentration
Reactivity Of Hydrogen Sulfide With Peroxynitrite And Other Oxidants Of Biological Interest
Early Disruption Of The Actin Cytoskeleton In Cultured Cerebellar Granule Neurons Exposed To 3-Morpholinosydnonimine-Oxidative Stress Is Linked To Alterations Of The Cytosolic Calcium Concentration
Reactivity Of Hydrogen Sulfide With Peroxynitrite And Other Oxidants Of Biological Interest
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells
Reactivity Of Hydrogen Sulfide With Peroxynitrite And Other Oxidants Of Biological Interest
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells
A Remarkable New Target Gene For The Dioxin Receptor: The Vav3 Proto-Oncogene Links Ahr To Adhesion And Migration
2,3,7,8-Tetrachlorodibenzo-P-Dioxin Induces Apoptosis In Neural Growth Factor (Ngf)-Differentiated Pheochromocytoma Pc12 Cells
A Remarkable New Target Gene For The Dioxin Receptor: The Vav3 Proto-Oncogene Links Ahr To Adhesion And Migration
Peroxynitrite-Mediated Oxidative Modifications Of Myosin And Implications On Structure And Function
A Remarkable New Target Gene For The Dioxin Receptor: The Vav3 Proto-Oncogene Links Ahr To Adhesion And Migration
Peroxynitrite-Mediated Oxidative Modifications Of Myosin And Implications On Structure And Function
L-Type Calcium Channels And Cytochrome B5 Reductase Are Components Of Protein Complexes Tightly Associated With Lipid Rafts Microdomains Of The Neuronal Plasma Membrane
Peroxynitrite-Mediated Oxidative Modifications Of Myosin And Implications On Structure And Function
L-Type Calcium Channels And Cytochrome B5 Reductase Are Components Of Protein Complexes Tightly Associated With Lipid Rafts Microdomains Of The Neuronal Plasma Membrane
Kaempferol Protects Against Rat Striatal Degeneration Induced By 3-Nitropropionic Acid
L-Type Calcium Channels And Cytochrome B5 Reductase Are Components Of Protein Complexes Tightly Associated With Lipid Rafts Microdomains Of The Neuronal Plasma Membrane
Kaempferol Protects Against Rat Striatal Degeneration Induced By 3-Nitropropionic Acid
Role Of Transforming Growth Factor Β In Cancer Microenvironment
Kaempferol Protects Against Rat Striatal Degeneration Induced By 3-Nitropropionic Acid
Role Of Transforming Growth Factor Β In Cancer Microenvironment
Dioxin Receptor Deficiency Impairs Angiogenesis By A Mechanism Involving Vegf-A Depletion In The Endothelium And Transforming Growth Factor-Β Overexpression In The Stroma
Role Of Transforming Growth Factor Β In Cancer Microenvironment
Dioxin Receptor Deficiency Impairs Angiogenesis By A Mechanism Involving Vegf-A Depletion In The Endothelium And Transforming Growth Factor-Β Overexpression In The Stroma
Regulation Of Cell Survival By Resveratrol Involves Inhibition Of Nfκb-Regulated Gene Expression In Prostate Cancer Cells
Dioxin Receptor Deficiency Impairs Angiogenesis By A Mechanism Involving Vegf-A Depletion In The Endothelium And Transforming Growth Factor-Β Overexpression In The Stroma
Regulation Of Cell Survival By Resveratrol Involves Inhibition Of Nfκb-Regulated Gene Expression In Prostate Cancer Cells
The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion
Regulation Of Cell Survival By Resveratrol Involves Inhibition Of Nfκb-Regulated Gene Expression In Prostate Cancer Cells
The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion
Fitting A Xenobiotic Receptor Into Cell Homeostasis: How The Dioxin Receptor Interacts With Tgfβ Signaling
The Dioxin Receptor Regulates The Constitutive Expression Of The Vav3 Proto-Oncogene And Modulates Cell Shape And Adhesion
Fitting A Xenobiotic Receptor Into Cell Homeostasis: How The Dioxin Receptor Interacts With Tgfβ Signaling
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Fitting A Xenobiotic Receptor Into Cell Homeostasis: How The Dioxin Receptor Interacts With Tgfβ Signaling
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Loss Of Dioxin-Receptor Expression Accelerates Wound Healing In Vivo By A Mechanism Involving Tgfβ
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Loss Of Dioxin-Receptor Expression Accelerates Wound Healing In Vivo By A Mechanism Involving Tgfβ
Hydrogen Sulfide Is A Reversible Inhibitor Of The Nadh Oxidase Activity Of Synaptic Plasma Membranes
Loss Of Dioxin-Receptor Expression Accelerates Wound Healing In Vivo By A Mechanism Involving Tgfβ
Hydrogen Sulfide Is A Reversible Inhibitor Of The Nadh Oxidase Activity Of Synaptic Plasma Membranes
Recruitment Of Creb1 And Histone Deacetylase 2 (Hdac2) To The Mouse Ltbp-1 Promoter Regulates Its Constitutive Expression In A Dioxin Receptor-Dependent Manner
Hydrogen Sulfide Is A Reversible Inhibitor Of The Nadh Oxidase Activity Of Synaptic Plasma Membranes
Recruitment Of Creb1 And Histone Deacetylase 2 (Hdac2) To The Mouse Ltbp-1 Promoter Regulates Its Constitutive Expression In A Dioxin Receptor-Dependent Manner
Genome-Wide B1 Retrotransposon Binds The Transcription Factors Dioxin Receptor And Slug And Regulates Gene Expression In Vivo
Recruitment Of Creb1 And Histone Deacetylase 2 (Hdac2) To The Mouse Ltbp-1 Promoter Regulates Its Constitutive Expression In A Dioxin Receptor-Dependent Manner
Genome-Wide B1 Retrotransposon Binds The Transcription Factors Dioxin Receptor And Slug And Regulates Gene Expression In Vivo
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Genome-Wide B1 Retrotransposon Binds The Transcription Factors Dioxin Receptor And Slug And Regulates Gene Expression In Vivo
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Sarcoplasmic Reticulum Calcium Atpase Is Inhibited By Organic Vanadium Coordination Compounds: Pyridine-2,6-Dicarboxylatodioxovanadium(V), Bmov, And An Amavadine Analogue
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Sarcoplasmic Reticulum Calcium Atpase Is Inhibited By Organic Vanadium Coordination Compounds: Pyridine-2,6-Dicarboxylatodioxovanadium(V), Bmov, And An Amavadine Analogue
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Sarcoplasmic Reticulum Calcium Atpase Is Inhibited By Organic Vanadium Coordination Compounds: Pyridine-2,6-Dicarboxylatodioxovanadium(V), Bmov, And An Amavadine Analogue
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Vanadium And Cadmium In Vivo Effects In Teleost Cardiac Muscle: Metal Accumulation And Oxidative Stress Markers
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Vanadium And Cadmium In Vivo Effects In Teleost Cardiac Muscle: Metal Accumulation And Oxidative Stress Markers
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Vanadium And Cadmium In Vivo Effects In Teleost Cardiac Muscle: Metal Accumulation And Oxidative Stress Markers
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Non-Genomic Action Of Resveratrol On Androgen And Oestrogen Receptors In Prostate Cancer: Modulation Of The Phosphoinositide 3-Kinase Pathway
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Non-Genomic Action Of Resveratrol On Androgen And Oestrogen Receptors In Prostate Cancer: Modulation Of The Phosphoinositide 3-Kinase Pathway
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
Non-Genomic Action Of Resveratrol On Androgen And Oestrogen Receptors In Prostate Cancer: Modulation Of The Phosphoinositide 3-Kinase Pathway
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
The Aryl Hydrocarbon Receptor, More Than A Xenobiotic-Interacting Protein
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
The Aryl Hydrocarbon Receptor, More Than A Xenobiotic-Interacting Protein
Biological Effects Of Decavanadate: Muscle Contraction, In Vivo Oxidative Stress, And Mitochondrial Toxicity
The Aryl Hydrocarbon Receptor, More Than A Xenobiotic-Interacting Protein
Biological Effects Of Decavanadate: Muscle Contraction, In Vivo Oxidative Stress, And Mitochondrial Toxicity
Vanadium Distribution, Lipid Peroxidation And Oxidative Stress Markers Upon Decavanadate In Vivo Administration
Biological Effects Of Decavanadate: Muscle Contraction, In Vivo Oxidative Stress, And Mitochondrial Toxicity
Vanadium Distribution, Lipid Peroxidation And Oxidative Stress Markers Upon Decavanadate In Vivo Administration
Mitochondria As A Target For Decavanadate Toxicity In Sparus Aurata Heart
Vanadium Distribution, Lipid Peroxidation And Oxidative Stress Markers Upon Decavanadate In Vivo Administration
Mitochondria As A Target For Decavanadate Toxicity In Sparus Aurata Heart
Decavanadate Induces Mitochondrial Membrane Depolarization And Inhibits Oxygen Consumption
Mitochondria As A Target For Decavanadate Toxicity In Sparus Aurata Heart
Decavanadate Induces Mitochondrial Membrane Depolarization And Inhibits Oxygen Consumption
Binding Modes Of Decavanadate To Myosin And Inhibition Of The Actomyosin Atpase Activity
Decavanadate Induces Mitochondrial Membrane Depolarization And Inhibits Oxygen Consumption
Binding Modes Of Decavanadate To Myosin And Inhibition Of The Actomyosin Atpase Activity
The Dioxin Receptor Is Silenced By Promoter Hypermethylation In Human Acute Lymphoblastic Leukemia Through Inhibition Of Sp1 Binding
Binding Modes Of Decavanadate To Myosin And Inhibition Of The Actomyosin Atpase Activity
The Dioxin Receptor Is Silenced By Promoter Hypermethylation In Human Acute Lymphoblastic Leukemia Through Inhibition Of Sp1 Binding
Ltbp-1 Blockade In Dioxin Receptor-Null Mouse Embryo Fibroblasts Decreases Tgf-Β Activity: Role Of Extracellular Proteases Plasmin And Elastase
The Dioxin Receptor Is Silenced By Promoter Hypermethylation In Human Acute Lymphoblastic Leukemia Through Inhibition Of Sp1 Binding
Ltbp-1 Blockade In Dioxin Receptor-Null Mouse Embryo Fibroblasts Decreases Tgf-Β Activity: Role Of Extracellular Proteases Plasmin And Elastase
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Ltbp-1 Blockade In Dioxin Receptor-Null Mouse Embryo Fibroblasts Decreases Tgf-Β Activity: Role Of Extracellular Proteases Plasmin And Elastase
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Decavanadate Interactions With Actin: Inhibition Of G-Actin Polymerization And Stabilization Of Decameric Vanadate
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Decavanadate Interactions With Actin: Inhibition Of G-Actin Polymerization And Stabilization Of Decameric Vanadate
Peroxynitrite Induces F-Actin Depolymerization And Blockade Of Myosin Atpase Stimulation
Decavanadate Interactions With Actin: Inhibition Of G-Actin Polymerization And Stabilization Of Decameric Vanadate
Peroxynitrite Induces F-Actin Depolymerization And Blockade Of Myosin Atpase Stimulation
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Peroxynitrite Induces F-Actin Depolymerization And Blockade Of Myosin Atpase Stimulation
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Improving Cancer Therapeutics By Molecular Profiling
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Improving Cancer Therapeutics By Molecular Profiling
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Improving Cancer Therapeutics By Molecular Profiling
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Store-Operated Calcium Entry In Differentiated C2c12 Skeletal Muscle Cells
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Store-Operated Calcium Entry In Differentiated C2c12 Skeletal Muscle Cells
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Store-Operated Calcium Entry In Differentiated C2c12 Skeletal Muscle Cells
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Vanadate Oligomers: In Vivo Effects In Hepatic Vanadium Accumulation And Stress Markers
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Vanadate Oligomers: In Vivo Effects In Hepatic Vanadium Accumulation And Stress Markers
Resveratrol Modulates The Phosphoinositide 3-Kinase Pathway Through An Estrogen Receptor Α-Dependent Mechanism: Relevance In Cell Proliferation
Vanadate Oligomers: In Vivo Effects In Hepatic Vanadium Accumulation And Stress Markers
Resveratrol Modulates The Phosphoinositide 3-Kinase Pathway Through An Estrogen Receptor Α-Dependent Mechanism: Relevance In Cell Proliferation
Overexpression Of Latent Transforming Growth Factor-Β Binding Protein 1 (Ltbp-1) In Dioxin Receptor-Null Mouse Embryo Fibroblasts
Resveratrol Modulates The Phosphoinositide 3-Kinase Pathway Through An Estrogen Receptor Α-Dependent Mechanism: Relevance In Cell Proliferation
Overexpression Of Latent Transforming Growth Factor-Β Binding Protein 1 (Ltbp-1) In Dioxin Receptor-Null Mouse Embryo Fibroblasts
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Overexpression Of Latent Transforming Growth Factor-Β Binding Protein 1 (Ltbp-1) In Dioxin Receptor-Null Mouse Embryo Fibroblasts
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Decavanadate Binding To A High Affinity Site Near The Myosin Catalytic Centre Inhibits F-Actin-Stimulated Myosin Atpase Activity
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Decavanadate Binding To A High Affinity Site Near The Myosin Catalytic Centre Inhibits F-Actin-Stimulated Myosin Atpase Activity
Liver Portal Fibrosis In Dioxin Receptor-Null Mice That Overexpress The Latent Transforming Growth Factor-Β-Binding Protein-1
Decavanadate Binding To A High Affinity Site Near The Myosin Catalytic Centre Inhibits F-Actin-Stimulated Myosin Atpase Activity
Liver Portal Fibrosis In Dioxin Receptor-Null Mice That Overexpress The Latent Transforming Growth Factor-Β-Binding Protein-1
Proteasome Inhibition Induces Nuclear Translocation Of The Dioxin Receptor Through An Sp1 And Protein Kinase C-Dependent Pathway
Liver Portal Fibrosis In Dioxin Receptor-Null Mice That Overexpress The Latent Transforming Growth Factor-Β-Binding Protein-1
Proteasome Inhibition Induces Nuclear Translocation Of The Dioxin Receptor Through An Sp1 And Protein Kinase C-Dependent Pathway
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Proteasome Inhibition Induces Nuclear Translocation Of The Dioxin Receptor Through An Sp1 And Protein Kinase C-Dependent Pathway
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Thioridazine Steady-State Plasma Concentrations Are Influenced By Tobacco Smoking And Cyp2d6, But Not By The Cyp2c9 Genotype
Down-Regulation Of Cyp1a2 Induction During The Maturation Of Mouse Cerebellar Granule Cells In Culture: Role Of Nitric Oxide Accumulation
Thioridazine Steady-State Plasma Concentrations Are Influenced By Tobacco Smoking And Cyp2d6, But Not By The Cyp2c9 Genotype
Carcinogenesis Of The Food Mutagen Phip In Mice Is Dependent Of Cyp1a2
Thioridazine Steady-State Plasma Concentrations Are Influenced By Tobacco Smoking And Cyp2d6, But Not By The Cyp2c9 Genotype
Carcinogenesis Of The Food Mutagen Phip In Mice Is Dependent Of Cyp1a2
Polycyclic Aromatic Hydrocarbon-Inducible Dna Adducts: Evidence By 32p-Postlabeling And Use Of Knockout Mice For Ah Receptor-Independent Mechanisms Of Metabolic Activation In Vivo
Carcinogenesis Of The Food Mutagen Phip In Mice Is Dependent Of Cyp1a2
Polycyclic Aromatic Hydrocarbon-Inducible Dna Adducts: Evidence By 32p-Postlabeling And Use Of Knockout Mice For Ah Receptor-Independent Mechanisms Of Metabolic Activation In Vivo
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
Polycyclic Aromatic Hydrocarbon-Inducible Dna Adducts: Evidence By 32p-Postlabeling And Use Of Knockout Mice For Ah Receptor-Independent Mechanisms Of Metabolic Activation In Vivo
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
Projection Map Of Covalently Phosphorylated Ca-Atpase From Tubular Crystals
El Resveratrol Presente En El Vino Como Potencial Inhibidor Del Desarrollo De Tumores En Humanos
Projection Map Of Covalently Phosphorylated Ca-Atpase From Tubular Crystals
The Trapping Of The Oh Radical By Coenzyme Q. A Theoretical And Experimental Study
Projection Map Of Covalently Phosphorylated Ca-Atpase From Tubular Crystals
The Trapping Of The Oh Radical By Coenzyme Q. A Theoretical And Experimental Study
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
The Trapping Of The Oh Radical By Coenzyme Q. A Theoretical And Experimental Study
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Neuroprotection Against Excitotoxicity By N-Alkylglycines In Rat Hippocampal Neurons
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
Quenching Of Myosin Intrinsic Fluorescence Unravels The Existence Of A High Affinity Binding Site For Decavanadate
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
Quenching Of Myosin Intrinsic Fluorescence Unravels The Existence Of A High Affinity Binding Site For Decavanadate
Sphingomyelin Composition And Physical Asymmetries In Native Acetylcholine Receptor-Rich Membranes
Quenching Of Myosin Intrinsic Fluorescence Unravels The Existence Of A High Affinity Binding Site For Decavanadate
Sphingomyelin Composition And Physical Asymmetries In Native Acetylcholine Receptor-Rich Membranes
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
Sphingomyelin Composition And Physical Asymmetries In Native Acetylcholine Receptor-Rich Membranes
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
Effect Of Thioridazine Dosage On The Debrisoquine Hydroxylation Phenotype In Psychiatric Patients With Different Cyp2d6 Genotypes
Resveratrol, Una Fitalexina Presente En El Vino, Inhibe Proliferación Celular En Líneas Tumorales Derivadas De Epitelio Mamario Humano
Effect Of Thioridazine Dosage On The Debrisoquine Hydroxylation Phenotype In Psychiatric Patients With Different Cyp2d6 Genotypes
Targeted Genomic Disruption Of H-Ras And N-Ras, Individually Or In Combination, Reveals The Dispensability Of Both Loci For Mouse Growth And Development
Effect Of Thioridazine Dosage On The Debrisoquine Hydroxylation Phenotype In Psychiatric Patients With Different Cyp2d6 Genotypes
Targeted Genomic Disruption Of H-Ras And N-Ras, Individually Or In Combination, Reveals The Dispensability Of Both Loci For Mouse Growth And Development
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
Targeted Genomic Disruption Of H-Ras And N-Ras, Individually Or In Combination, Reveals The Dispensability Of Both Loci For Mouse Growth And Development
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Proteasome Inhibition Induces Nuclear Translocation And Transcriptional Activation Of The Dioxin Receptor In Mouse Embryo Primary Fibroblasts In The Absence Of Xenobiotics
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Phosphorylated Ca2+-Atpase Stable Enough For Structural Studies
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Phosphorylated Ca2+-Atpase Stable Enough For Structural Studies
A Remarkably Stable Phosphorylated Form Of Ca2+-Atpase Prepared From Ca2+-Loaded And Fluorescein Isothiocyanate-Labeled Sarcoplasmic Reticulum Vesicles
Phosphorylated Ca2+-Atpase Stable Enough For Structural Studies
A Remarkably Stable Phosphorylated Form Of Ca2+-Atpase Prepared From Ca2+-Loaded And Fluorescein Isothiocyanate-Labeled Sarcoplasmic Reticulum Vesicles
Altered Cell Cycle Control At The G2/m Phases In Aryl Hydrocarbon Receptor-Null Embryo Fibroblast
A Remarkably Stable Phosphorylated Form Of Ca2+-Atpase Prepared From Ca2+-Loaded And Fluorescein Isothiocyanate-Labeled Sarcoplasmic Reticulum Vesicles
Altered Cell Cycle Control At The G2/m Phases In Aryl Hydrocarbon Receptor-Null Embryo Fibroblast
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Altered Cell Cycle Control At The G2/m Phases In Aryl Hydrocarbon Receptor-Null Embryo Fibroblast
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Hepatic Fibrosis And Cytochrome P450: Experimental Models Of Fibrosis Compared To Ahr Knockout Mice
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Hepatic Fibrosis And Cytochrome P450: Experimental Models Of Fibrosis Compared To Ahr Knockout Mice
The Expression Of Plasma Membrane Ca2+ Pump Isoforms In Cerebellar Granule Neurons Is Modulated By Ca2+
Hepatic Fibrosis And Cytochrome P450: Experimental Models Of Fibrosis Compared To Ahr Knockout Mice
The Expression Of Plasma Membrane Ca2+ Pump Isoforms In Cerebellar Granule Neurons Is Modulated By Ca2+
Genomic Instability In Gadd45a-Deficient Mice
The Expression Of Plasma Membrane Ca2+ Pump Isoforms In Cerebellar Granule Neurons Is Modulated By Ca2+
Genomic Instability In Gadd45a-Deficient Mice
Cyp1a2 Is Not The Primary Enzyme Responsible For 4-Aminobiphenyl-Induced Hepatocarcinogenesis In Mice
Genomic Instability In Gadd45a-Deficient Mice
Cyp1a2 Is Not The Primary Enzyme Responsible For 4-Aminobiphenyl-Induced Hepatocarcinogenesis In Mice
Targeted Disruption Of The Microsomal Epoxide Hydrolase Gene. Microsomal Epoxide Hydrolase Is Required For The Carcinogenic Activity Of 7,12- Dimethylbenz[A]anthracene
Cyp1a2 Is Not The Primary Enzyme Responsible For 4-Aminobiphenyl-Induced Hepatocarcinogenesis In Mice
Targeted Disruption Of The Microsomal Epoxide Hydrolase Gene. Microsomal Epoxide Hydrolase Is Required For The Carcinogenic Activity Of 7,12- Dimethylbenz[A]anthracene
Expression Of Cyp2a Genes In Human Liver And Extrahepatic Tissues
Targeted Disruption Of The Microsomal Epoxide Hydrolase Gene. Microsomal Epoxide Hydrolase Is Required For The Carcinogenic Activity Of 7,12- Dimethylbenz[A]anthracene
Expression Of Cyp2a Genes In Human Liver And Extrahepatic Tissues
Amelioration Of Tcdd-Induced Teratogenesis In Aryl Hydrocarbon Receptor (Ahr)-Null Mice
Expression Of Cyp2a Genes In Human Liver And Extrahepatic Tissues
Amelioration Of Tcdd-Induced Teratogenesis In Aryl Hydrocarbon Receptor (Ahr)-Null Mice
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Amelioration Of Tcdd-Induced Teratogenesis In Aryl Hydrocarbon Receptor (Ahr)-Null Mice
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Characterization Of A Protease-Resistant Domain Of The Cytosolic Portion Of Sarcoplasmic Reticulum Ca2+-Atpase. Nucleotide- And Metal-Binding Sites
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Characterization Of A Protease-Resistant Domain Of The Cytosolic Portion Of Sarcoplasmic Reticulum Ca2+-Atpase. Nucleotide- And Metal-Binding Sites
Analysis Of Secreted Flavonoids Of Cistus Ladanifer L. By High- Performance Liquid Chromatography-Particle Beam Mass Spectrometry
Characterization Of A Protease-Resistant Domain Of The Cytosolic Portion Of Sarcoplasmic Reticulum Ca2+-Atpase. Nucleotide- And Metal-Binding Sites
Analysis Of Secreted Flavonoids Of Cistus Ladanifer L. By High- Performance Liquid Chromatography-Particle Beam Mass Spectrometry
The Aryl Hydrocarbon Receptor. Studies Using The Ahr-Null Mice
Analysis Of Secreted Flavonoids Of Cistus Ladanifer L. By High- Performance Liquid Chromatography-Particle Beam Mass Spectrometry
The Aryl Hydrocarbon Receptor. Studies Using The Ahr-Null Mice
Aryl-Hydrocarbon Receptor Is An Inhibitory Regulator Of Lipid Synthesis And Of Commitment To Adipogenesis
The Aryl Hydrocarbon Receptor. Studies Using The Ahr-Null Mice
Aryl-Hydrocarbon Receptor Is An Inhibitory Regulator Of Lipid Synthesis And Of Commitment To Adipogenesis
Characterization Of The Human Dihydropyrimidine Dehydrogenase Gene
Aryl-Hydrocarbon Receptor Is An Inhibitory Regulator Of Lipid Synthesis And Of Commitment To Adipogenesis
Characterization Of The Human Dihydropyrimidine Dehydrogenase Gene
Effect Of Phenobarbital On Hepatic Cyp1a1 And Cyp1a2 In The Ahr-Null Mouse
Characterization Of The Human Dihydropyrimidine Dehydrogenase Gene
Effect Of Phenobarbital On Hepatic Cyp1a1 And Cyp1a2 In The Ahr-Null Mouse
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Caucasian Subjects
Effect Of Phenobarbital On Hepatic Cyp1a1 And Cyp1a2 In The Ahr-Null Mouse
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Caucasian Subjects
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Patients With Colorectal Cancer
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Caucasian Subjects
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Patients With Colorectal Cancer
Nomenclature For Human Dpyd Alleles
Dihydropyrimidine Dehydrogenase Pharmacogenetics In Patients With Colorectal Cancer
Nomenclature For Human Dpyd Alleles
The Involvement Of Aryl Hydrocarbon Receptor In The Activation Of Transforming Growth Factor-Β And Apoptosis
Nomenclature For Human Dpyd Alleles
The Involvement Of Aryl Hydrocarbon Receptor In The Activation Of Transforming Growth Factor-Β And Apoptosis
Dissociation Of Ca2+ From Sarcoplasmic Reticulum Ca2+-Atpase And Changes In Fluorescence Of Optically Selected Trp Residues. Effects Of Kcl And Nacl And Implications For Substeps In Ca2+ Dissociation
The Involvement Of Aryl Hydrocarbon Receptor In The Activation Of Transforming Growth Factor-Β And Apoptosis
Dissociation Of Ca2+ From Sarcoplasmic Reticulum Ca2+-Atpase And Changes In Fluorescence Of Optically Selected Trp Residues. Effects Of Kcl And Nacl And Implications For Substeps In Ca2+ Dissociation
Quantitative Variation Of Flavonoids Among Individuals Of A Cistus Ladanifer Population
Dissociation Of Ca2+ From Sarcoplasmic Reticulum Ca2+-Atpase And Changes In Fluorescence Of Optically Selected Trp Residues. Effects Of Kcl And Nacl And Implications For Substeps In Ca2+ Dissociation
Quantitative Variation Of Flavonoids Among Individuals Of A Cistus Ladanifer Population
Role Of Ecological Variables In The Seasonal Variation Of Flavonoid Content Of Cistus Ladanifer Exudate
Quantitative Variation Of Flavonoids Among Individuals Of A Cistus Ladanifer Population
Role Of Ecological Variables In The Seasonal Variation Of Flavonoid Content Of Cistus Ladanifer Exudate
Aryl Hydrocarbon Receptor Deficient Mice (Ahr-/-) Exhibit An Accumulation Of Liver Retinylpalmitate
Role Of Ecological Variables In The Seasonal Variation Of Flavonoid Content Of Cistus Ladanifer Exudate
Aryl Hydrocarbon Receptor Deficient Mice (Ahr-/-) Exhibit An Accumulation Of Liver Retinylpalmitate
Analysis Of The Selenocysteine (Sec) Trna Population In Mouse Embryonic Stem (Es) Cells Heterozygous For The Sec Trna Gene
Aryl Hydrocarbon Receptor Deficient Mice (Ahr-/-) Exhibit An Accumulation Of Liver Retinylpalmitate
Analysis Of The Selenocysteine (Sec) Trna Population In Mouse Embryonic Stem (Es) Cells Heterozygous For The Sec Trna Gene
Aryl Hydrocarbon Receptor Knockout Mice (Ahr(-/-)) Exhibit Liver Retinoid Accumulation And Reduced Retinoic Acid Metabolism
Analysis Of The Selenocysteine (Sec) Trna Population In Mouse Embryonic Stem (Es) Cells Heterozygous For The Sec Trna Gene
Aryl Hydrocarbon Receptor Knockout Mice (Ahr(-/-)) Exhibit Liver Retinoid Accumulation And Reduced Retinoic Acid Metabolism
Cyp2a6 Gene Polymorphism And Risk Of Liver Cancer And Cirrhosis
Aryl Hydrocarbon Receptor Knockout Mice (Ahr(-/-)) Exhibit Liver Retinoid Accumulation And Reduced Retinoic Acid Metabolism
Cyp2a6 Gene Polymorphism And Risk Of Liver Cancer And Cirrhosis
Selenocysteine Trna([Ser]sec) Levels And Selenium-Dependent Glutathione Peroxidase Activity In Mouse Embryonic Stem Cells Heterozygous For A Targeted Mutation In The Trna([Ser]sec) Gene
Cyp2a6 Gene Polymorphism And Risk Of Liver Cancer And Cirrhosis
Selenocysteine Trna([Ser]sec) Levels And Selenium-Dependent Glutathione Peroxidase Activity In Mouse Embryonic Stem Cells Heterozygous For A Targeted Mutation In The Trna([Ser]sec) Gene
Lack Of Correlation Between Phenotype And Genotype For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In A Family Of Pakistani Origin
Selenocysteine Trna([Ser]sec) Levels And Selenium-Dependent Glutathione Peroxidase Activity In Mouse Embryonic Stem Cells Heterozygous For A Targeted Mutation In The Trna([Ser]sec) Gene
Lack Of Correlation Between Phenotype And Genotype For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In A Family Of Pakistani Origin
Lesions Of Aryl-Hydrocarbon Receptor-Deficient Mice
Lack Of Correlation Between Phenotype And Genotype For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In A Family Of Pakistani Origin
Lesions Of Aryl-Hydrocarbon Receptor-Deficient Mice
Mutations At Codon 974 Of The Dpyd Gene Are A Rare Event
Lesions Of Aryl-Hydrocarbon Receptor-Deficient Mice
Mutations At Codon 974 Of The Dpyd Gene Are A Rare Event
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Mutations At Codon 974 Of The Dpyd Gene Are A Rare Event
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Hypothalamic Hypophyseal Inhibitory Factor (Hhif) Increases Intrasynaptosomal Free Calcium Concentration
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Hypothalamic Hypophyseal Inhibitory Factor (Hhif) Increases Intrasynaptosomal Free Calcium Concentration
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
Hypothalamic Hypophyseal Inhibitory Factor (Hhif) Increases Intrasynaptosomal Free Calcium Concentration
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
A Comparative Kinetic Analysis Of The Flavin-Photosensitized Oxidation And Reduction Of Plastocyanin And Cytochrome C6from Different Organisms
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
A Comparative Kinetic Analysis Of The Flavin-Photosensitized Oxidation And Reduction Of Plastocyanin And Cytochrome C6from Different Organisms
Physical State Of Bulk And Protein-Associated Lipid In Nicotinic Acetylcholine Receptor-Rich Membrane Studied By Laurdan Generalized Polarization And Fluorescence Energy Transfer
A Comparative Kinetic Analysis Of The Flavin-Photosensitized Oxidation And Reduction Of Plastocyanin And Cytochrome C6from Different Organisms
Physical State Of Bulk And Protein-Associated Lipid In Nicotinic Acetylcholine Receptor-Rich Membrane Studied By Laurdan Generalized Polarization And Fluorescence Energy Transfer
Piperonyl Butoxide And Acenaphthylene Induce Cytochrome P450 1a2 And 1b1 Mrna In Aromatic Hydrocarbon-Responsive Receptor Knock-Out Mouse Liver
Physical State Of Bulk And Protein-Associated Lipid In Nicotinic Acetylcholine Receptor-Rich Membrane Studied By Laurdan Generalized Polarization And Fluorescence Energy Transfer
Piperonyl Butoxide And Acenaphthylene Induce Cytochrome P450 1a2 And 1b1 Mrna In Aromatic Hydrocarbon-Responsive Receptor Knock-Out Mouse Liver
Transcriptional Induction Of The Cytochrome P4501a1 Gene By A Thiazolium Compound, Yh439
Piperonyl Butoxide And Acenaphthylene Induce Cytochrome P450 1a2 And 1b1 Mrna In Aromatic Hydrocarbon-Responsive Receptor Knock-Out Mouse Liver
Transcriptional Induction Of The Cytochrome P4501a1 Gene By A Thiazolium Compound, Yh439
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
Transcriptional Induction Of The Cytochrome P4501a1 Gene By A Thiazolium Compound, Yh439
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
The Role Of The Aryl Hydrocarbon Receptor In Animal Development, Physiological Homeostasis And Toxicity Of Tcdd
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
The Role Of The Aryl Hydrocarbon Receptor In Animal Development, Physiological Homeostasis And Toxicity Of Tcdd
Differential Regulation Of Mouse Ah Receptor Gene Expression In Cell Lines Of Different Tissue Origins
The Role Of The Aryl Hydrocarbon Receptor In Animal Development, Physiological Homeostasis And Toxicity Of Tcdd
Differential Regulation Of Mouse Ah Receptor Gene Expression In Cell Lines Of Different Tissue Origins
Molecular Basis Of The Human Dihydropyrimidine Dehydrogenase Deficiency And 5-Fluorouracil Toxicity
Differential Regulation Of Mouse Ah Receptor Gene Expression In Cell Lines Of Different Tissue Origins
Molecular Basis Of The Human Dihydropyrimidine Dehydrogenase Deficiency And 5-Fluorouracil Toxicity
Role Of Cyp2e1 In The Hepatotoxicity Of Acetaminophen
Molecular Basis Of The Human Dihydropyrimidine Dehydrogenase Deficiency And 5-Fluorouracil Toxicity
Role Of Cyp2e1 In The Hepatotoxicity Of Acetaminophen
Aryl-Hydrocarbon Receptor-Deficient Mice Are Resistant To 2,3,7,8-Tetrachlorodibenzo-P-Dioxin-Induced Toxicity
Role Of Cyp2e1 In The Hepatotoxicity Of Acetaminophen
Aryl-Hydrocarbon Receptor-Deficient Mice Are Resistant To 2,3,7,8-Tetrachlorodibenzo-P-Dioxin-Induced Toxicity
Hepatic Fibrosis In Ahr(-/-) Mice
Aryl-Hydrocarbon Receptor-Deficient Mice Are Resistant To 2,3,7,8-Tetrachlorodibenzo-P-Dioxin-Induced Toxicity
Hepatic Fibrosis In Ahr(-/-) Mice
The T/ebp Null Mouse: Thyroid-Specific Enhancer-Binding Protein Is Essential For The Organogenesis Of The Thyroid, Lung, Ventral Forebrain, And Pituitary
Hepatic Fibrosis In Ahr(-/-) Mice
The T/ebp Null Mouse: Thyroid-Specific Enhancer-Binding Protein Is Essential For The Organogenesis Of The Thyroid, Lung, Ventral Forebrain, And Pituitary
Pyrimidines And Cns Regulation
The T/ebp Null Mouse: Thyroid-Specific Enhancer-Binding Protein Is Essential For The Organogenesis Of The Thyroid, Lung, Ventral Forebrain, And Pituitary
Pyrimidines And Cns Regulation
Targeted Disruption Of Specific Cytochromes P450 And Xenobiotic Receptor Genes
Pyrimidines And Cns Regulation
Targeted Disruption Of Specific Cytochromes P450 And Xenobiotic Receptor Genes
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Targeted Disruption Of Specific Cytochromes P450 And Xenobiotic Receptor Genes
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Rate Of Na+/ca2+ Exchange Across The Plasma Membrane Of Synaptosomes Measured Using The Fluorescence Of Chlorotetracycline. Implications To Calcium Homeostasis In Synaptic Terminals
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Rate Of Na+/ca2+ Exchange Across The Plasma Membrane Of Synaptosomes Measured Using The Fluorescence Of Chlorotetracycline. Implications To Calcium Homeostasis In Synaptic Terminals
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Rate Of Na+/ca2+ Exchange Across The Plasma Membrane Of Synaptosomes Measured Using The Fluorescence Of Chlorotetracycline. Implications To Calcium Homeostasis In Synaptic Terminals
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Preferential Distribution Of The Fluorescent Phospholipid Probes Nbd-Phosphatidylcholine And Rhodamine-Phosphatidylethanolamine In The Exofacial Leaflet Of Acetylcholine Receptor-Rich Membranes From Torpedo Marmorata
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Preferential Distribution Of The Fluorescent Phospholipid Probes Nbd-Phosphatidylcholine And Rhodamine-Phosphatidylethanolamine In The Exofacial Leaflet Of Acetylcholine Receptor-Rich Membranes From Torpedo Marmorata
Diagnostic-Analysis, Clinical Importance And Molecular-Basis Of Dihydropyrimidine Dehydrogenase-Deficiency
Preferential Distribution Of The Fluorescent Phospholipid Probes Nbd-Phosphatidylcholine And Rhodamine-Phosphatidylethanolamine In The Exofacial Leaflet Of Acetylcholine Receptor-Rich Membranes From Torpedo Marmorata
Diagnostic-Analysis, Clinical Importance And Molecular-Basis Of Dihydropyrimidine Dehydrogenase-Deficiency
Organization And Evolution Of The Cytochrome P450 Cyp2a-2b-2f Subfamily Gene Cluster On Human Chromosome 19
Diagnostic-Analysis, Clinical Importance And Molecular-Basis Of Dihydropyrimidine Dehydrogenase-Deficiency
Organization And Evolution Of The Cytochrome P450 Cyp2a-2b-2f Subfamily Gene Cluster On Human Chromosome 19
A Genetic Polymorphism In Coumarin 7-Hydroxylation: Sequence Of The Human Cyp2a Genes And Identification Of Variant Cyp2a6 Alleles
Organization And Evolution Of The Cytochrome P450 Cyp2a-2b-2f Subfamily Gene Cluster On Human Chromosome 19
A Genetic Polymorphism In Coumarin 7-Hydroxylation: Sequence Of The Human Cyp2a Genes And Identification Of Variant Cyp2a6 Alleles
Comparison Of Substrate Metabolism By Wild Type Cyp2d6 Protein And A Variant Containing Methionine, Not Valine, At Position 374
A Genetic Polymorphism In Coumarin 7-Hydroxylation: Sequence Of The Human Cyp2a Genes And Identification Of Variant Cyp2a6 Alleles
Comparison Of Substrate Metabolism By Wild Type Cyp2d6 Protein And A Variant Containing Methionine, Not Valine, At Position 374
Human Polymorphism In Drug Metabolism: Mutation In The Dihydropyrimidine Dehydrogenase Gene Results In Exon Skipping And Thymine Uracilurea
Comparison Of Substrate Metabolism By Wild Type Cyp2d6 Protein And A Variant Containing Methionine, Not Valine, At Position 374
Human Polymorphism In Drug Metabolism: Mutation In The Dihydropyrimidine Dehydrogenase Gene Results In Exon Skipping And Thymine Uracilurea
Genetic Polymorphism Of Cyp2c9 And Its Effect On Warfarin Maintenance Dose Requirement In Patients Undergoing Anticoagulation Therapy
Human Polymorphism In Drug Metabolism: Mutation In The Dihydropyrimidine Dehydrogenase Gene Results In Exon Skipping And Thymine Uracilurea
Genetic Polymorphism Of Cyp2c9 And Its Effect On Warfarin Maintenance Dose Requirement In Patients Undergoing Anticoagulation Therapy
Correlation Between Catalytic Activity And Protein Content For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In Human Lymphocytes
Genetic Polymorphism Of Cyp2c9 And Its Effect On Warfarin Maintenance Dose Requirement In Patients Undergoing Anticoagulation Therapy
Correlation Between Catalytic Activity And Protein Content For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In Human Lymphocytes
Neonatal Lethality Associated With Respiratory Distress In Mice Lacking Cytochrome P450 1a2
Correlation Between Catalytic Activity And Protein Content For The Polymorphically Expressed Dihydropyrimidine Dehydrogenase In Human Lymphocytes
Neonatal Lethality Associated With Respiratory Distress In Mice Lacking Cytochrome P450 1a2
Xenobiotic Receptor Knockout Mice
Neonatal Lethality Associated With Respiratory Distress In Mice Lacking Cytochrome P450 1a2
Xenobiotic Receptor Knockout Mice
Immune System Impairment And Hepatic Fibrosis In Mice Lacking The Dioxin-Binding Ah Receptor
Xenobiotic Receptor Knockout Mice
Immune System Impairment And Hepatic Fibrosis In Mice Lacking The Dioxin-Binding Ah Receptor
Targeted Disruption Of The Α Isoform Of The Peroxisome Proliferator- Activated Receptor Gene In Mice Results In Abolishment Of The Pleiotropic Effects Of Peroxisome Proliferators
Immune System Impairment And Hepatic Fibrosis In Mice Lacking The Dioxin-Binding Ah Receptor
Targeted Disruption Of The Α Isoform Of The Peroxisome Proliferator- Activated Receptor Gene In Mice Results In Abolishment Of The Pleiotropic Effects Of Peroxisome Proliferators
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Targeted Disruption Of The Α Isoform Of The Peroxisome Proliferator- Activated Receptor Gene In Mice Results In Abolishment Of The Pleiotropic Effects Of Peroxisome Proliferators
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Modulation Of The Ca2+ Pump By The Hypothalamic-Hypophysary Inhibitory Factor
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Modulation Of The Ca2+ Pump By The Hypothalamic-Hypophysary Inhibitory Factor
Modulation Of The Ca 2+,mg2+-Atpase Of Sarcoplasmic Reticulum By The Hypothalamic Hypophyseal Inhibitory Factor
Modulation Of The Ca2+ Pump By The Hypothalamic-Hypophysary Inhibitory Factor
Modulation Of The Ca 2+,mg2+-Atpase Of Sarcoplasmic Reticulum By The Hypothalamic Hypophyseal Inhibitory Factor
Cdna Cloning And Chromosome Mapping Of Human Dihydropyrimidine Dehydrogenase, An Enzyme Associated With 5-Fluorouracil Toxicity And Congenital Thymine Uraciluria
Modulation Of The Ca 2+,mg2+-Atpase Of Sarcoplasmic Reticulum By The Hypothalamic Hypophyseal Inhibitory Factor
Cdna Cloning And Chromosome Mapping Of Human Dihydropyrimidine Dehydrogenase, An Enzyme Associated With 5-Fluorouracil Toxicity And Congenital Thymine Uraciluria
Assignment Of The Human Dihydropyrimidine Dehydrogenase Gene (Dpyd) To Chromosome Region 1p22 By Fluorescence In Situ Hybridization
Cdna Cloning And Chromosome Mapping Of Human Dihydropyrimidine Dehydrogenase, An Enzyme Associated With 5-Fluorouracil Toxicity And Congenital Thymine Uraciluria
Assignment Of The Human Dihydropyrimidine Dehydrogenase Gene (Dpyd) To Chromosome Region 1p22 By Fluorescence In Situ Hybridization
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Assignment Of The Human Dihydropyrimidine Dehydrogenase Gene (Dpyd) To Chromosome Region 1p22 By Fluorescence In Situ Hybridization
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Cancellation Of The Cooperativity Of Ca2+ Binding To Sarcoplasmic Reticulum Ca2+‐atpase By The Non‐ionic Detergent Dodecylmaltoside
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Cancellation Of The Cooperativity Of Ca2+ Binding To Sarcoplasmic Reticulum Ca2+‐atpase By The Non‐ionic Detergent Dodecylmaltoside
Seasonal Variation Of Exudate Of Cistus Ladanifer
Cancellation Of The Cooperativity Of Ca2+ Binding To Sarcoplasmic Reticulum Ca2+‐atpase By The Non‐ionic Detergent Dodecylmaltoside
Seasonal Variation Of Exudate Of Cistus Ladanifer
Glycogen Phosphorolysis Can Form A Metabolic Shuttle To Support Ca2+ Uptake By Sarcoplasmic Reticulum Membranes In Skeletal Muscle
Seasonal Variation Of Exudate Of Cistus Ladanifer
Glycogen Phosphorolysis Can Form A Metabolic Shuttle To Support Ca2+ Uptake By Sarcoplasmic Reticulum Membranes In Skeletal Muscle
Advanced Course On Structure-Function Relationships Of Membrane Proteins: An Assessment Of Current Knowledge
Glycogen Phosphorolysis Can Form A Metabolic Shuttle To Support Ca2+ Uptake By Sarcoplasmic Reticulum Membranes In Skeletal Muscle
Advanced Course On Structure-Function Relationships Of Membrane Proteins: An Assessment Of Current Knowledge
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Advanced Course On Structure-Function Relationships Of Membrane Proteins: An Assessment Of Current Knowledge
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Properties Of The Purified Hypothalamic Pituitary Na/k-Atpase Inhibitor
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Properties Of The Purified Hypothalamic Pituitary Na/k-Atpase Inhibitor
Location Of Functional Centers In The Microsomal Cytochrome P450 System
Properties Of The Purified Hypothalamic Pituitary Na/k-Atpase Inhibitor
Location Of Functional Centers In The Microsomal Cytochrome P450 System
The Metal Sites On Sarcoplasmic Reticulum Membranes That Bind Lanthanide Ions With The Highest Affinity Are Not The Atpase Ca2+ Transport Sites
Location Of Functional Centers In The Microsomal Cytochrome P450 System
The Metal Sites On Sarcoplasmic Reticulum Membranes That Bind Lanthanide Ions With The Highest Affinity Are Not The Atpase Ca2+ Transport Sites
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
The Metal Sites On Sarcoplasmic Reticulum Membranes That Bind Lanthanide Ions With The Highest Affinity Are Not The Atpase Ca2+ Transport Sites
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Binding Of Gadolinium Ions To Sarcoplasmic Reticulum Membranes
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Binding Of Gadolinium Ions To Sarcoplasmic Reticulum Membranes
Kinetic Characterization Of The Perturbation By Dodecylmaltoside Of Sarcoplasmic Reticulum Ca2+ ‐atpase
Binding Of Gadolinium Ions To Sarcoplasmic Reticulum Membranes
Kinetic Characterization Of The Perturbation By Dodecylmaltoside Of Sarcoplasmic Reticulum Ca2+ ‐atpase
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Kinetic Characterization Of The Perturbation By Dodecylmaltoside Of Sarcoplasmic Reticulum Ca2+ ‐atpase
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Modulation By Phosphorylation Of Glycogen Phosphorylase-Sarcoplasmic Reticulum Interaction
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Modulation By Phosphorylation Of Glycogen Phosphorylase-Sarcoplasmic Reticulum Interaction
Caracterización Por Técnicas Espectrofotométricas Del Ultravioleta Y Visible Edel Exudado De Cistus Ladanifer L. Como Respuesta Al Estrés Estival
Modulation By Phosphorylation Of Glycogen Phosphorylase-Sarcoplasmic Reticulum Interaction
Caracterización Por Técnicas Espectrofotométricas Del Ultravioleta Y Visible Edel Exudado De Cistus Ladanifer L. Como Respuesta Al Estrés Estival
Hemin And Hemeprotein Bleaching During Linoleic Acid Oxidation By Lipoxygenases
Caracterización Por Técnicas Espectrofotométricas Del Ultravioleta Y Visible Edel Exudado De Cistus Ladanifer L. Como Respuesta Al Estrés Estival
Hemin And Hemeprotein Bleaching During Linoleic Acid Oxidation By Lipoxygenases
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Hemin And Hemeprotein Bleaching During Linoleic Acid Oxidation By Lipoxygenases
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Modulation Of Calcium Fluxes Across Synaptosomal Plasma Membrane By Local Anesthetics
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Modulation Of Calcium Fluxes Across Synaptosomal Plasma Membrane By Local Anesthetics
Modulation Of The Ca2+,mg2+‐atpase Activity Of Synaptosomal Plasma Membrane By The Local Anesthetics Dibucaine And Lidocaine
Modulation Of Calcium Fluxes Across Synaptosomal Plasma Membrane By Local Anesthetics
Modulation Of The Ca2+,mg2+‐atpase Activity Of Synaptosomal Plasma Membrane By The Local Anesthetics Dibucaine And Lidocaine
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Modulation Of The Ca2+,mg2+‐atpase Activity Of Synaptosomal Plasma Membrane By The Local Anesthetics Dibucaine And Lidocaine
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Interaction Of The Local Anesthetics Dibucaine And Tetracaine With Sarcoplasmic Reticulum Membranes. Differential Scanning Calorimetry And Fluorescence Studies
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Interaction Of The Local Anesthetics Dibucaine And Tetracaine With Sarcoplasmic Reticulum Membranes. Differential Scanning Calorimetry And Fluorescence Studies
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Interaction Of The Local Anesthetics Dibucaine And Tetracaine With Sarcoplasmic Reticulum Membranes. Differential Scanning Calorimetry And Fluorescence Studies
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Local Anesthetic-Divalent Cation Binding Center Interaction
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Local Anesthetic-Divalent Cation Binding Center Interaction
Comparative Fluorescence Properties Of Lipoxygenases
Local Anesthetic-Divalent Cation Binding Center Interaction
Comparative Fluorescence Properties Of Lipoxygenases
Effects Of Local Anesthetics On The Passive Permeability Of Sarcoplasmic Reticulum Vesicles To Ca2+ And Mg2+
Comparative Fluorescence Properties Of Lipoxygenases
Effects Of Local Anesthetics On The Passive Permeability Of Sarcoplasmic Reticulum Vesicles To Ca2+ And Mg2+
The Position Of The Atp Binding Site On The (Ca2+ + Mg2+)-Atpase
Effects Of Local Anesthetics On The Passive Permeability Of Sarcoplasmic Reticulum Vesicles To Ca2+ And Mg2+
The Position Of The Atp Binding Site On The (Ca2+ + Mg2+)-Atpase
Long-Wavelength Fluorescence Of Tyrosine And Tryptophan Solutions.
The Position Of The Atp Binding Site On The (Ca2+ + Mg2+)-Atpase
Long-Wavelength Fluorescence Of Tyrosine And Tryptophan Solutions.
Local Anesthetics Inhibit The Ca2+,mg2+‐atpase Activity Of Rat Brain Synaptosomes
Long-Wavelength Fluorescence Of Tyrosine And Tryptophan Solutions.
Local Anesthetics Inhibit The Ca2+,mg2+‐atpase Activity Of Rat Brain Synaptosomes
Dependence Of The Fluorescence Of Fluorescein Labelled (Ca2+, Mg2+)-Atpase Upon The Lipid To Protein Ratio In Sarcoplasmic Reticulum Reconstituted Systems
Local Anesthetics Inhibit The Ca2+,mg2+‐atpase Activity Of Rat Brain Synaptosomes
Dependence Of The Fluorescence Of Fluorescein Labelled (Ca2+, Mg2+)-Atpase Upon The Lipid To Protein Ratio In Sarcoplasmic Reticulum Reconstituted Systems
On The Rate Of Association And Dissociation Processes Of Intrinsic Membrane Proteins
Dependence Of The Fluorescence Of Fluorescein Labelled (Ca2+, Mg2+)-Atpase Upon The Lipid To Protein Ratio In Sarcoplasmic Reticulum Reconstituted Systems
On The Rate Of Association And Dissociation Processes Of Intrinsic Membrane Proteins
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. Ii. Protein Distribution And Aggregation State In Biological Membranes
On The Rate Of Association And Dissociation Processes Of Intrinsic Membrane Proteins
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. Ii. Protein Distribution And Aggregation State In Biological Membranes
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. I. Lateral Phase Separation In Unilamellar Vesicles Formed By Binary Phospholipid Mixtures
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. Ii. Protein Distribution And Aggregation State In Biological Membranes
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. I. Lateral Phase Separation In Unilamellar Vesicles Formed By Binary Phospholipid Mixtures
Relationship Between The Activity Of Pancreatic Phospholipase A2 And The Physical State Of The Phospholipid Substrate.
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. I. Lateral Phase Separation In Unilamellar Vesicles Formed By Binary Phospholipid Mixtures
Relationship Between The Activity Of Pancreatic Phospholipase A2 And The Physical State Of The Phospholipid Substrate.
Regulation Of Phosphorylase B By Amp: The Effects Of Enzyme Concentration
Relationship Between The Activity Of Pancreatic Phospholipase A2 And The Physical State Of The Phospholipid Substrate.
Regulation Of Phosphorylase B By Amp: The Effects Of Enzyme Concentration
Involvement Of Β-Catenin Expression In Hepatocellular Carcinoma Prognosis In A Cohort Of Patients Undergoing Curative Treatment
WEB ACCESIBLE