PUBLICACIÓN

Modulation by phosphorylation of glycogen phosphorylase-sarcoplasmic reticulum interaction

Logo Open Access

ACCEDER A LA PUBLICACIÓN: Scopus

Ana Cuenda, Francisco Centeno, Carlos Gutierrez-Merino

1991 FEBS Letters

CITAS

26

DOI

10.1016/0014-5793(91)80606-4

EID

2-s2.0-0025828189

ISSN

0014-5793

BIBTEX

@article{Cuenda_1991,doi = {10.1016/0014-5793(91)80606-4},url = {https://doi.org/10.1016%2F0014-5793%2891%2980606-4},year = 1991,month = {jun},publisher = {Wiley-Blackwell},volume = {283},number = {2},pages = {273--276},author = {Ana Cuenda and Francisco Centeno and Carlos Gutierrez-Merino},title = {Modulation by phosphorylation of glycogen phosphorylase-sarcoplasmic reticulum interaction},journal = {{FEBS} Letters}}

AUTORES DE LA UEX

Francisco Centeno Velázquez

OTRAS PUBLICACIONES DE INTERÉS

Regulation Of Phosphorylase B By Amp: The Effects Of Enzyme Concentration
Editorial For The Special Issue “mapk In Health And Disease”
Early Atf4 Activity Drives Airway Club And Goblet Cell Differentiation
Editorial For The Special Issue “mapk In Health And Disease”
Early Atf4 Activity Drives Airway Club And Goblet Cell Differentiation
An Update Of Kaempferol Protection Against Brain Damage Induced By Ischemia-Reperfusion And By 3-Nitropropionic Acid
Early Atf4 Activity Drives Airway Club And Goblet Cell Differentiation
An Update Of Kaempferol Protection Against Brain Damage Induced By Ischemia-Reperfusion And By 3-Nitropropionic Acid
Store-Operated Calcium Entry Inhibition And Plasma Membrane Calcium Pump Upregulation Contribute To The Maintenance Of Resting Cytosolic Calcium Concentration In A1-Like Astrocytes
An Update Of Kaempferol Protection Against Brain Damage Induced By Ischemia-Reperfusion And By 3-Nitropropionic Acid
Store-Operated Calcium Entry Inhibition And Plasma Membrane Calcium Pump Upregulation Contribute To The Maintenance Of Resting Cytosolic Calcium Concentration In A1-Like Astrocytes
Brain Hydrophobic Peptides Antagonists Of Neurotoxic Amyloid Β Peptide Monomers/oligomers–protein Interactions
Store-Operated Calcium Entry Inhibition And Plasma Membrane Calcium Pump Upregulation Contribute To The Maintenance Of Resting Cytosolic Calcium Concentration In A1-Like Astrocytes
Brain Hydrophobic Peptides Antagonists Of Neurotoxic Amyloid Β Peptide Monomers/oligomers–protein Interactions
Immunomodulatory Effects Of Inactivated Ligilactobacillus Salivarius Cect 9609 On Respiratory Epithelial Cells
Brain Hydrophobic Peptides Antagonists Of Neurotoxic Amyloid Β Peptide Monomers/oligomers–protein Interactions
Immunomodulatory Effects Of Inactivated Ligilactobacillus Salivarius Cect 9609 On Respiratory Epithelial Cells
Hexa-Histidine, A Peptide With Versatile Applications In The Study Of Amyloid-Β(1–42) Molecular Mechanisms Of Action
Immunomodulatory Effects Of Inactivated Ligilactobacillus Salivarius Cect 9609 On Respiratory Epithelial Cells
Hexa-Histidine, A Peptide With Versatile Applications In The Study Of Amyloid-Β(1–42) Molecular Mechanisms Of Action
Are There Lipid Membrane-Domain Subtypes In Neurons With Different Roles In Calcium Signaling?
Hexa-Histidine, A Peptide With Versatile Applications In The Study Of Amyloid-Β(1–42) Molecular Mechanisms Of Action
Are There Lipid Membrane-Domain Subtypes In Neurons With Different Roles In Calcium Signaling?
Biochemical And Biophysical Characterization Of The Caveolin-2 Interaction With Membranes And Analysis Of The Protein Structural Alteration By The Presence Of Cholesterol
Are There Lipid Membrane-Domain Subtypes In Neurons With Different Roles In Calcium Signaling?
Biochemical And Biophysical Characterization Of The Caveolin-2 Interaction With Membranes And Analysis Of The Protein Structural Alteration By The Presence Of Cholesterol
Structural Features Of Cytochrome B5 –cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Biochemical And Biophysical Characterization Of The Caveolin-2 Interaction With Membranes And Analysis Of The Protein Structural Alteration By The Presence Of Cholesterol
Structural Features Of Cytochrome B5 –cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
In Silico Research Of New Therapeutics Rotenoids Derivatives Against Leishmania Amazonensis Infection
Structural Features Of Cytochrome B5 –cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
In Silico Research Of New Therapeutics Rotenoids Derivatives Against Leishmania Amazonensis Infection
Inhibition Of Serca And Pmca Ca2+-Atpase Activities By Polyoxotungstates
In Silico Research Of New Therapeutics Rotenoids Derivatives Against Leishmania Amazonensis Infection
Inhibition Of Serca And Pmca Ca2+-Atpase Activities By Polyoxotungstates
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Inhibition Of Serca And Pmca Ca2+-Atpase Activities By Polyoxotungstates
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Special Issue “molecular And Cellular Mechanisms Of Action Of Markers Of Tissue Degeneration”
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Special Issue “molecular And Cellular Mechanisms Of Action Of Markers Of Tissue Degeneration”
Design And Experimental Evaluation Of A Peptide Antagonist Against Amyloid Β(1–42) Interactions With Calmodulin And Calbindin-D28k
Special Issue “molecular And Cellular Mechanisms Of Action Of Markers Of Tissue Degeneration”
Design And Experimental Evaluation Of A Peptide Antagonist Against Amyloid Β(1–42) Interactions With Calmodulin And Calbindin-D28k
Kaempferol Prevents The Activation Of Complement C3 Protein And The Generation Of Reactive A1 Astrocytes That Mediate Rat Brain Degeneration Induced By 3-Nitropropionic Acid
Design And Experimental Evaluation Of A Peptide Antagonist Against Amyloid Β(1–42) Interactions With Calmodulin And Calbindin-D28k
Kaempferol Prevents The Activation Of Complement C3 Protein And The Generation Of Reactive A1 Astrocytes That Mediate Rat Brain Degeneration Induced By 3-Nitropropionic Acid
Title: P38δ Regulates Il6 Expression Modulating Erk Phosphorylation In Preadipocytes
Kaempferol Prevents The Activation Of Complement C3 Protein And The Generation Of Reactive A1 Astrocytes That Mediate Rat Brain Degeneration Induced By 3-Nitropropionic Acid
Title: P38δ Regulates Il6 Expression Modulating Erk Phosphorylation In Preadipocytes
Junctional Adhesion Molecule 3 Expression In The Mouse Airway Epithelium Is Linked To Multiciliated Cells
Title: P38δ Regulates Il6 Expression Modulating Erk Phosphorylation In Preadipocytes
Junctional Adhesion Molecule 3 Expression In The Mouse Airway Epithelium Is Linked To Multiciliated Cells
Gold Compounds Inhibit The Ca2+-Atpase Activity Of Brain Pmca And Human Neuroblastoma Sh-Sy5y Cells And Decrease Cell Viability
Junctional Adhesion Molecule 3 Expression In The Mouse Airway Epithelium Is Linked To Multiciliated Cells
Gold Compounds Inhibit The Ca2+-Atpase Activity Of Brain Pmca And Human Neuroblastoma Sh-Sy5y Cells And Decrease Cell Viability
The Relevance Of Amyloid Β-Calmodulin Complexation In Neurons And Brain Degeneration In Alzheimer’s Disease
Gold Compounds Inhibit The Ca2+-Atpase Activity Of Brain Pmca And Human Neuroblastoma Sh-Sy5y Cells And Decrease Cell Viability
The Relevance Of Amyloid Β-Calmodulin Complexation In Neurons And Brain Degeneration In Alzheimer’s Disease
Binding Of Amyloid Β(1–42)‐calmodulin Complexes To Plasma Membrane Lipid Rafts In Cerebellar Granule Neurons Alters Resting Cytosolic Calcium Homeostasis
The Relevance Of Amyloid Β-Calmodulin Complexation In Neurons And Brain Degeneration In Alzheimer’s Disease
Binding Of Amyloid Β(1–42)‐calmodulin Complexes To Plasma Membrane Lipid Rafts In Cerebellar Granule Neurons Alters Resting Cytosolic Calcium Homeostasis
P53 Regulation By Mdm2 Contributes To Self-Renewal And Differentiation Of Basal Stem Cells In Mouse And Human Airway Epithelium
Binding Of Amyloid Β(1–42)‐calmodulin Complexes To Plasma Membrane Lipid Rafts In Cerebellar Granule Neurons Alters Resting Cytosolic Calcium Homeostasis
P53 Regulation By Mdm2 Contributes To Self-Renewal And Differentiation Of Basal Stem Cells In Mouse And Human Airway Epithelium
Structural Features Of Cytochrome B5–cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
P53 Regulation By Mdm2 Contributes To Self-Renewal And Differentiation Of Basal Stem Cells In Mouse And Human Airway Epithelium
Structural Features Of Cytochrome B5–cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Human Erythrocytes Exposure To Juglone Leads To An Increase Of Superoxide Anion Production Associated With Cytochrome B5 Reductase Uncoupling
Structural Features Of Cytochrome B5–cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Human Erythrocytes Exposure To Juglone Leads To An Increase Of Superoxide Anion Production Associated With Cytochrome B5 Reductase Uncoupling
Early Reactive A1 Astrocytes Induction By The Neurotoxin 3-Nitropropionic Acid In Rat Brain
Human Erythrocytes Exposure To Juglone Leads To An Increase Of Superoxide Anion Production Associated With Cytochrome B5 Reductase Uncoupling
Early Reactive A1 Astrocytes Induction By The Neurotoxin 3-Nitropropionic Acid In Rat Brain
Three Alkaloids From An Apocynaceae Species, Aspidosperma Spruceanum As Antileishmaniasis Agents By In Silico Demo-Case Studies
Early Reactive A1 Astrocytes Induction By The Neurotoxin 3-Nitropropionic Acid In Rat Brain
Three Alkaloids From An Apocynaceae Species, Aspidosperma Spruceanum As Antileishmaniasis Agents By In Silico Demo-Case Studies
Modulation Of Cyp2c9 Activity And Hydrogen Peroxide Production By Cytochrome B 5
Three Alkaloids From An Apocynaceae Species, Aspidosperma Spruceanum As Antileishmaniasis Agents By In Silico Demo-Case Studies
Modulation Of Cyp2c9 Activity And Hydrogen Peroxide Production By Cytochrome B 5
Ligand Accessibility To Heme Cytochrome B 5 Coordinating Sphere And Enzymatic Activity Enhancement Upon Tyrosine Ionization
Modulation Of Cyp2c9 Activity And Hydrogen Peroxide Production By Cytochrome B 5
Ligand Accessibility To Heme Cytochrome B 5 Coordinating Sphere And Enzymatic Activity Enhancement Upon Tyrosine Ionization
Methylene Blue Blocks And Reverses The Inhibitory Effect Of Tau On Pmca Function
Ligand Accessibility To Heme Cytochrome B 5 Coordinating Sphere And Enzymatic Activity Enhancement Upon Tyrosine Ionization
Methylene Blue Blocks And Reverses The Inhibitory Effect Of Tau On Pmca Function
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Methylene Blue Blocks And Reverses The Inhibitory Effect Of Tau On Pmca Function
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Centriole Positioning: Not Just A Little Dot In The Cell
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Centriole Positioning: Not Just A Little Dot In The Cell
Methyl-Β-Cyclodextrin Impairs The Phosphorylation Of The Β2 Subunit Of L-Type Calcium Channels And Cytosolic Calcium Homeostasis In Mature Cerebellar Granule Neurons
Centriole Positioning: Not Just A Little Dot In The Cell
Methyl-Β-Cyclodextrin Impairs The Phosphorylation Of The Β2 Subunit Of L-Type Calcium Channels And Cytosolic Calcium Homeostasis In Mature Cerebellar Granule Neurons
Creatine Protects Against Cytosolic Calcium Dysregulation, Mitochondrial Depolarization And Increase Of Reactive Oxygen Species Production In Rotenone-Induced Cell Death Of Cerebellar Granule Neurons
Methyl-Β-Cyclodextrin Impairs The Phosphorylation Of The Β2 Subunit Of L-Type Calcium Channels And Cytosolic Calcium Homeostasis In Mature Cerebellar Granule Neurons
Creatine Protects Against Cytosolic Calcium Dysregulation, Mitochondrial Depolarization And Increase Of Reactive Oxygen Species Production In Rotenone-Induced Cell Death Of Cerebellar Granule Neurons
Cytochrome B5 Reductase Is The Component From Neuronal Synaptic Plasma Membrane Vesicles That Generates Superoxide Anion Upon Stimulation By Cytochrome C
Creatine Protects Against Cytosolic Calcium Dysregulation, Mitochondrial Depolarization And Increase Of Reactive Oxygen Species Production In Rotenone-Induced Cell Death Of Cerebellar Granule Neurons
Cytochrome B5 Reductase Is The Component From Neuronal Synaptic Plasma Membrane Vesicles That Generates Superoxide Anion Upon Stimulation By Cytochrome C
Topography Of Human Cytochrome B5/cytochrome B5 Reductase Interacting Domain And Redox Alterations Upon Complex Formation
Cytochrome B5 Reductase Is The Component From Neuronal Synaptic Plasma Membrane Vesicles That Generates Superoxide Anion Upon Stimulation By Cytochrome C
Topography Of Human Cytochrome B5/cytochrome B5 Reductase Interacting Domain And Redox Alterations Upon Complex Formation
Peroxidase-Like Activity Of Cytochrome B5 Is Triggered Upon Hemichrome Formation In Alkaline Ph
Topography Of Human Cytochrome B5/cytochrome B5 Reductase Interacting Domain And Redox Alterations Upon Complex Formation
Peroxidase-Like Activity Of Cytochrome B5 Is Triggered Upon Hemichrome Formation In Alkaline Ph
P38α Blocks Brown Adipose Tissue Thermogenesis Through P38δ Inhibition
Peroxidase-Like Activity Of Cytochrome B5 Is Triggered Upon Hemichrome Formation In Alkaline Ph
P38α Blocks Brown Adipose Tissue Thermogenesis Through P38δ Inhibition
Methylene Blue Activates The Pmca Activity And Cross-Interacts With Amyloid Β-Peptide, Blocking Aβ-Mediated Pmca Inhibition
P38α Blocks Brown Adipose Tissue Thermogenesis Through P38δ Inhibition
Methylene Blue Activates The Pmca Activity And Cross-Interacts With Amyloid Β-Peptide, Blocking Aβ-Mediated Pmca Inhibition
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Methylene Blue Activates The Pmca Activity And Cross-Interacts With Amyloid Β-Peptide, Blocking Aβ-Mediated Pmca Inhibition
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Mkk6 Controls T3-Mediated Browning Of White Adipose Tissue
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Mkk6 Controls T3-Mediated Browning Of White Adipose Tissue
Jnk Signaling Pathway Regulates Sorbitol-Induced Tau Proteolysis And Apoptosis In Sh-Sy5y Cells By Targeting Caspase-3
Mkk6 Controls T3-Mediated Browning Of White Adipose Tissue
Jnk Signaling Pathway Regulates Sorbitol-Induced Tau Proteolysis And Apoptosis In Sh-Sy5y Cells By Targeting Caspase-3
Mkk6 Controls T3-Mediated Browning Of White Adipose Tissue
Jnk Signaling Pathway Regulates Sorbitol-Induced Tau Proteolysis And Apoptosis In Sh-Sy5y Cells By Targeting Caspase-3
Mkk6 Controls T3-Mediated Browning Of White Adipose Tissue
Phospholipids And Calmodulin Modulate The Inhibition Of Pmca Activity By Tau
Mkk6 Controls T3-Mediated Browning Of White Adipose Tissue
Phospholipids And Calmodulin Modulate The Inhibition Of Pmca Activity By Tau
High Affinity Binding Of Amyloid Β-Peptide To Calmodulin: Structural And Functional Implications
Phospholipids And Calmodulin Modulate The Inhibition Of Pmca Activity By Tau
High Affinity Binding Of Amyloid Β-Peptide To Calmodulin: Structural And Functional Implications
Jnk Signaling Pathway Regulates Sorbitol-Induced Tau Proteolysis And Apoptosis In Sh-Sy5y Cells By Targeting Caspase-3
High Affinity Binding Of Amyloid Β-Peptide To Calmodulin: Structural And Functional Implications
Jnk Signaling Pathway Regulates Sorbitol-Induced Tau Proteolysis And Apoptosis In Sh-Sy5y Cells By Targeting Caspase-3
Correlation Between The Potency Of Flavonoids For Cytochrome C Reduction And Inhibition Of Cardiolipin-Induced Peroxidase Activity
Jnk Signaling Pathway Regulates Sorbitol-Induced Tau Proteolysis And Apoptosis In Sh-Sy5y Cells By Targeting Caspase-3
Correlation Between The Potency Of Flavonoids For Cytochrome C Reduction And Inhibition Of Cardiolipin-Induced Peroxidase Activity
Hyperosmotic Stress Induces Tau Proteolysis By Caspase-3 Activation In Sh-Sy5y Cells
Correlation Between The Potency Of Flavonoids For Cytochrome C Reduction And Inhibition Of Cardiolipin-Induced Peroxidase Activity
Hyperosmotic Stress Induces Tau Proteolysis By Caspase-3 Activation In Sh-Sy5y Cells
High Expression Of Cytochrome B 5 Reductase Isoform 3/cytochrome B 5 System In The Cerebellum And Pyramidal Neurons Of Adult Rat Brain
Hyperosmotic Stress Induces Tau Proteolysis By Caspase-3 Activation In Sh-Sy5y Cells
High Expression Of Cytochrome B 5 Reductase Isoform 3/cytochrome B 5 System In The Cerebellum And Pyramidal Neurons Of Adult Rat Brain
Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg
High Expression Of Cytochrome B 5 Reductase Isoform 3/cytochrome B 5 System In The Cerebellum And Pyramidal Neurons Of Adult Rat Brain
Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg
Cytochrome B5 As A Pleiotropic Metabolic Modulator In Mammalian Cells
Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg
Cytochrome B5 As A Pleiotropic Metabolic Modulator In Mammalian Cells
The Decrease Of Nad(P)h:quinone Oxidoreductase 1 Activity And Increase Of Ros Production By Nadph Oxidases Are Early Biomarkers In Doxorubicin Cardiotoxicity
Cytochrome B5 As A Pleiotropic Metabolic Modulator In Mammalian Cells
The Decrease Of Nad(P)h:quinone Oxidoreductase 1 Activity And Increase Of Ros Production By Nadph Oxidases Are Early Biomarkers In Doxorubicin Cardiotoxicity
Caveolin-Rich Lipid Rafts Of The Plasma Membrane Of Mature Cerebellar Granule Neurons Are Microcompartments For Calcium/reactive Oxygen And Nitrogen Species Cross-Talk Signaling
The Decrease Of Nad(P)h:quinone Oxidoreductase 1 Activity And Increase Of Ros Production By Nadph Oxidases Are Early Biomarkers In Doxorubicin Cardiotoxicity
Caveolin-Rich Lipid Rafts Of The Plasma Membrane Of Mature Cerebellar Granule Neurons Are Microcompartments For Calcium/reactive Oxygen And Nitrogen Species Cross-Talk Signaling
Purified Nadh-Cytochrome B5 Reductase Is A Novel Superoxide Anion Source Inhibited By Apocynin: Sensitivity To Nitric Oxide And Peroxynitrite
Caveolin-Rich Lipid Rafts Of The Plasma Membrane Of Mature Cerebellar Granule Neurons Are Microcompartments For Calcium/reactive Oxygen And Nitrogen Species Cross-Talk Signaling
Purified Nadh-Cytochrome B5 Reductase Is A Novel Superoxide Anion Source Inhibited By Apocynin: Sensitivity To Nitric Oxide And Peroxynitrite
Stimulation And Clustering Of Cytochrome B 5 Reductase In Caveolin-Rich Lipid Microdomains Is An Early Event In Oxidative Stress-Mediated Apoptosis Of Cerebellar Granule Neurons
Purified Nadh-Cytochrome B5 Reductase Is A Novel Superoxide Anion Source Inhibited By Apocynin: Sensitivity To Nitric Oxide And Peroxynitrite
Stimulation And Clustering Of Cytochrome B 5 Reductase In Caveolin-Rich Lipid Microdomains Is An Early Event In Oxidative Stress-Mediated Apoptosis Of Cerebellar Granule Neurons
L-Type Voltage-Operated Calcium Channels, N-Methyl-D-Aspartate Receptors And Neuronal Nitric-Oxide Synthase Form A Calcium/redox Nano-Transducer Within Lipid Rafts
Stimulation And Clustering Of Cytochrome B 5 Reductase In Caveolin-Rich Lipid Microdomains Is An Early Event In Oxidative Stress-Mediated Apoptosis Of Cerebellar Granule Neurons
L-Type Voltage-Operated Calcium Channels, N-Methyl-D-Aspartate Receptors And Neuronal Nitric-Oxide Synthase Form A Calcium/redox Nano-Transducer Within Lipid Rafts
Complex I And Cytochrome C Are Molecular Targets Of Flavonoids That Inhibit Hydrogen Peroxide Production By Mitochondria
L-Type Voltage-Operated Calcium Channels, N-Methyl-D-Aspartate Receptors And Neuronal Nitric-Oxide Synthase Form A Calcium/redox Nano-Transducer Within Lipid Rafts
Complex I And Cytochrome C Are Molecular Targets Of Flavonoids That Inhibit Hydrogen Peroxide Production By Mitochondria
Neuroprotective Actions Of Flavonoids
Complex I And Cytochrome C Are Molecular Targets Of Flavonoids That Inhibit Hydrogen Peroxide Production By Mitochondria
Neuroprotective Actions Of Flavonoids
Early Disruption Of The Actin Cytoskeleton In Cultured Cerebellar Granule Neurons Exposed To 3-Morpholinosydnonimine-Oxidative Stress Is Linked To Alterations Of The Cytosolic Calcium Concentration
Neuroprotective Actions Of Flavonoids
Early Disruption Of The Actin Cytoskeleton In Cultured Cerebellar Granule Neurons Exposed To 3-Morpholinosydnonimine-Oxidative Stress Is Linked To Alterations Of The Cytosolic Calcium Concentration
Reactivity Of Hydrogen Sulfide With Peroxynitrite And Other Oxidants Of Biological Interest
Early Disruption Of The Actin Cytoskeleton In Cultured Cerebellar Granule Neurons Exposed To 3-Morpholinosydnonimine-Oxidative Stress Is Linked To Alterations Of The Cytosolic Calcium Concentration
Reactivity Of Hydrogen Sulfide With Peroxynitrite And Other Oxidants Of Biological Interest
Peroxynitrite-Mediated Oxidative Modifications Of Myosin And Implications On Structure And Function
Reactivity Of Hydrogen Sulfide With Peroxynitrite And Other Oxidants Of Biological Interest
Peroxynitrite-Mediated Oxidative Modifications Of Myosin And Implications On Structure And Function
L-Type Calcium Channels And Cytochrome B5 Reductase Are Components Of Protein Complexes Tightly Associated With Lipid Rafts Microdomains Of The Neuronal Plasma Membrane
Peroxynitrite-Mediated Oxidative Modifications Of Myosin And Implications On Structure And Function
L-Type Calcium Channels And Cytochrome B5 Reductase Are Components Of Protein Complexes Tightly Associated With Lipid Rafts Microdomains Of The Neuronal Plasma Membrane
P38γ Regulates Interaction Of Nuclear Psf And Rna With The Tumour-Suppressor Hdlg In Response To Osmotic Shock
L-Type Calcium Channels And Cytochrome B5 Reductase Are Components Of Protein Complexes Tightly Associated With Lipid Rafts Microdomains Of The Neuronal Plasma Membrane
P38γ Regulates Interaction Of Nuclear Psf And Rna With The Tumour-Suppressor Hdlg In Response To Osmotic Shock
Kaempferol Protects Against Rat Striatal Degeneration Induced By 3-Nitropropionic Acid
P38γ Regulates Interaction Of Nuclear Psf And Rna With The Tumour-Suppressor Hdlg In Response To Osmotic Shock
Kaempferol Protects Against Rat Striatal Degeneration Induced By 3-Nitropropionic Acid
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Kaempferol Protects Against Rat Striatal Degeneration Induced By 3-Nitropropionic Acid
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Hydrogen Sulfide Is A Reversible Inhibitor Of The Nadh Oxidase Activity Of Synaptic Plasma Membranes
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Hydrogen Sulfide Is A Reversible Inhibitor Of The Nadh Oxidase Activity Of Synaptic Plasma Membranes
Proteolysis Of The Tumour Suppressor Hdlg In Response To Osmotic Stress Is Mediated By Caspases And Independent Of Phosphorylation
Hydrogen Sulfide Is A Reversible Inhibitor Of The Nadh Oxidase Activity Of Synaptic Plasma Membranes
Proteolysis Of The Tumour Suppressor Hdlg In Response To Osmotic Stress Is Mediated By Caspases And Independent Of Phosphorylation
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Proteolysis Of The Tumour Suppressor Hdlg In Response To Osmotic Stress Is Mediated By Caspases And Independent Of Phosphorylation
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Vanadium And Cadmium In Vivo Effects In Teleost Cardiac Muscle: Metal Accumulation And Oxidative Stress Markers
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Vanadium And Cadmium In Vivo Effects In Teleost Cardiac Muscle: Metal Accumulation And Oxidative Stress Markers
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Vanadium And Cadmium In Vivo Effects In Teleost Cardiac Muscle: Metal Accumulation And Oxidative Stress Markers
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Biological Effects Of Decavanadate: Muscle Contraction, In Vivo Oxidative Stress, And Mitochondrial Toxicity
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Biological Effects Of Decavanadate: Muscle Contraction, In Vivo Oxidative Stress, And Mitochondrial Toxicity
Vanadium Distribution, Lipid Peroxidation And Oxidative Stress Markers Upon Decavanadate In Vivo Administration
Biological Effects Of Decavanadate: Muscle Contraction, In Vivo Oxidative Stress, And Mitochondrial Toxicity
Vanadium Distribution, Lipid Peroxidation And Oxidative Stress Markers Upon Decavanadate In Vivo Administration
Mitochondria As A Target For Decavanadate Toxicity In Sparus Aurata Heart
Vanadium Distribution, Lipid Peroxidation And Oxidative Stress Markers Upon Decavanadate In Vivo Administration
Mitochondria As A Target For Decavanadate Toxicity In Sparus Aurata Heart
Decavanadate Induces Mitochondrial Membrane Depolarization And Inhibits Oxygen Consumption
Mitochondria As A Target For Decavanadate Toxicity In Sparus Aurata Heart
Decavanadate Induces Mitochondrial Membrane Depolarization And Inhibits Oxygen Consumption
Binding Modes Of Decavanadate To Myosin And Inhibition Of The Actomyosin Atpase Activity
Decavanadate Induces Mitochondrial Membrane Depolarization And Inhibits Oxygen Consumption
Binding Modes Of Decavanadate To Myosin And Inhibition Of The Actomyosin Atpase Activity
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Binding Modes Of Decavanadate To Myosin And Inhibition Of The Actomyosin Atpase Activity
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Decavanadate Interactions With Actin: Inhibition Of G-Actin Polymerization And Stabilization Of Decameric Vanadate
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Decavanadate Interactions With Actin: Inhibition Of G-Actin Polymerization And Stabilization Of Decameric Vanadate
Peroxynitrite Induces F-Actin Depolymerization And Blockade Of Myosin Atpase Stimulation
Decavanadate Interactions With Actin: Inhibition Of G-Actin Polymerization And Stabilization Of Decameric Vanadate
Peroxynitrite Induces F-Actin Depolymerization And Blockade Of Myosin Atpase Stimulation
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Peroxynitrite Induces F-Actin Depolymerization And Blockade Of Myosin Atpase Stimulation
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Vanadate Oligomers: In Vivo Effects In Hepatic Vanadium Accumulation And Stress Markers
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Vanadate Oligomers: In Vivo Effects In Hepatic Vanadium Accumulation And Stress Markers
Crystal Structure Of Human Arginase I At 1.29-Å Resolution And Exploration Of Inhibition In The Immune Response
Vanadate Oligomers: In Vivo Effects In Hepatic Vanadium Accumulation And Stress Markers
Crystal Structure Of Human Arginase I At 1.29-Å Resolution And Exploration Of Inhibition In The Immune Response
P38γ Regulates The Localisation Of Sap97 In The Cytoskeleton By Modulating Its Interaction With Gkap
Crystal Structure Of Human Arginase I At 1.29-Å Resolution And Exploration Of Inhibition In The Immune Response
P38γ Regulates The Localisation Of Sap97 In The Cytoskeleton By Modulating Its Interaction With Gkap
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
P38γ Regulates The Localisation Of Sap97 In The Cytoskeleton By Modulating Its Interaction With Gkap
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Canine Pyometra: A Study Of The Urinary Proteins By Sds-Page And Western Blot
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Canine Pyometra: A Study Of The Urinary Proteins By Sds-Page And Western Blot
Decavanadate Binding To A High Affinity Site Near The Myosin Catalytic Centre Inhibits F-Actin-Stimulated Myosin Atpase Activity
Canine Pyometra: A Study Of The Urinary Proteins By Sds-Page And Western Blot
Decavanadate Binding To A High Affinity Site Near The Myosin Catalytic Centre Inhibits F-Actin-Stimulated Myosin Atpase Activity
Stress- And Mitogen-Induced Phosphorylation Of The Synapse-Associated Protein Sap90/psd-95 By Activation Of Sapk3/p38γ And Erk1/erk2
Decavanadate Binding To A High Affinity Site Near The Myosin Catalytic Centre Inhibits F-Actin-Stimulated Myosin Atpase Activity
Stress- And Mitogen-Induced Phosphorylation Of The Synapse-Associated Protein Sap90/psd-95 By Activation Of Sapk3/p38γ And Erk1/erk2
Characterization Of Renal Damage In Canine Leptospirosis By Sodium Dodecyl Sulphate-Polyacrylamide Gel Electrophoresis (Sds-Page) And Western Blotting Of The Urinary Proteins
Stress- And Mitogen-Induced Phosphorylation Of The Synapse-Associated Protein Sap90/psd-95 By Activation Of Sapk3/p38γ And Erk1/erk2
Characterization Of Renal Damage In Canine Leptospirosis By Sodium Dodecyl Sulphate-Polyacrylamide Gel Electrophoresis (Sds-Page) And Western Blotting Of The Urinary Proteins
Sds-Page And Western Blot Of Urinary Proteins In Dogs With Leishmaniasis
Characterization Of Renal Damage In Canine Leptospirosis By Sodium Dodecyl Sulphate-Polyacrylamide Gel Electrophoresis (Sds-Page) And Western Blotting Of The Urinary Proteins
Sds-Page And Western Blot Of Urinary Proteins In Dogs With Leishmaniasis
The Trapping Of The Oh Radical By Coenzyme Q. A Theoretical And Experimental Study
Sds-Page And Western Blot Of Urinary Proteins In Dogs With Leishmaniasis
The Trapping Of The Oh Radical By Coenzyme Q. A Theoretical And Experimental Study
Neuroprotective Effects Of Lithium - Pointing Out Protein Phosphatases As Drug Targets?
The Trapping Of The Oh Radical By Coenzyme Q. A Theoretical And Experimental Study
Neuroprotective Effects Of Lithium - Pointing Out Protein Phosphatases As Drug Targets?
Lithium Blocks The Pkb And Gsk3 Dephosphorylation Induced By Ceramide Through Protein Phosphatase-2a
Neuroprotective Effects Of Lithium - Pointing Out Protein Phosphatases As Drug Targets?
Lithium Blocks The Pkb And Gsk3 Dephosphorylation Induced By Ceramide Through Protein Phosphatase-2a
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
Lithium Blocks The Pkb And Gsk3 Dephosphorylation Induced By Ceramide Through Protein Phosphatase-2a
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
Quenching Of Myosin Intrinsic Fluorescence Unravels The Existence Of A High Affinity Binding Site For Decavanadate
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
Quenching Of Myosin Intrinsic Fluorescence Unravels The Existence Of A High Affinity Binding Site For Decavanadate
Sphingomyelin Composition And Physical Asymmetries In Native Acetylcholine Receptor-Rich Membranes
Quenching Of Myosin Intrinsic Fluorescence Unravels The Existence Of A High Affinity Binding Site For Decavanadate
Sphingomyelin Composition And Physical Asymmetries In Native Acetylcholine Receptor-Rich Membranes
Different Dependence Of Lithium And Valproate On P13k/pkb Pathway
Sphingomyelin Composition And Physical Asymmetries In Native Acetylcholine Receptor-Rich Membranes
Different Dependence Of Lithium And Valproate On P13k/pkb Pathway
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Different Dependence Of Lithium And Valproate On P13k/pkb Pathway
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Glu-256 Is A Main Structural Determinant For Oligomerisation Of Human Arginase I
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Glu-256 Is A Main Structural Determinant For Oligomerisation Of Human Arginase I
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Glu-256 Is A Main Structural Determinant For Oligomerisation Of Human Arginase I
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
The Expression Of Plasma Membrane Ca2+ Pump Isoforms In Cerebellar Granule Neurons Is Modulated By Ca2+
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
The Expression Of Plasma Membrane Ca2+ Pump Isoforms In Cerebellar Granule Neurons Is Modulated By Ca2+
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
The Expression Of Plasma Membrane Ca2+ Pump Isoforms In Cerebellar Granule Neurons Is Modulated By Ca2+
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Th1/th2-Regulated Expression Of Arginase Isoforms In Murine Macrophages And Dendritic Cells
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Th1/th2-Regulated Expression Of Arginase Isoforms In Murine Macrophages And Dendritic Cells
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Th1/th2-Regulated Expression Of Arginase Isoforms In Murine Macrophages And Dendritic Cells
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Analysis Of Secreted Flavonoids Of Cistus Ladanifer L. By High- Performance Liquid Chromatography-Particle Beam Mass Spectrometry
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Analysis Of Secreted Flavonoids Of Cistus Ladanifer L. By High- Performance Liquid Chromatography-Particle Beam Mass Spectrometry
Quantitative Variation Of Flavonoids Among Individuals Of A Cistus Ladanifer Population
Analysis Of Secreted Flavonoids Of Cistus Ladanifer L. By High- Performance Liquid Chromatography-Particle Beam Mass Spectrometry
Quantitative Variation Of Flavonoids Among Individuals Of A Cistus Ladanifer Population
Role Of Ecological Variables In The Seasonal Variation Of Flavonoid Content Of Cistus Ladanifer Exudate
Quantitative Variation Of Flavonoids Among Individuals Of A Cistus Ladanifer Population
Role Of Ecological Variables In The Seasonal Variation Of Flavonoid Content Of Cistus Ladanifer Exudate
The Cytoplasmic Loop Between Putative Transmembrane Segments 6 And 7 In Sarcoplasmic Reticulum Ca2+-Atpase Binds Ca2+ And Is Functionally Important
Role Of Ecological Variables In The Seasonal Variation Of Flavonoid Content Of Cistus Ladanifer Exudate
The Cytoplasmic Loop Between Putative Transmembrane Segments 6 And 7 In Sarcoplasmic Reticulum Ca2+-Atpase Binds Ca2+ And Is Functionally Important
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
The Cytoplasmic Loop Between Putative Transmembrane Segments 6 And 7 In Sarcoplasmic Reticulum Ca2+-Atpase Binds Ca2+ And Is Functionally Important
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Hypothalamic Hypophyseal Inhibitory Factor (Hhif) Increases Intrasynaptosomal Free Calcium Concentration
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Hypothalamic Hypophyseal Inhibitory Factor (Hhif) Increases Intrasynaptosomal Free Calcium Concentration
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
Hypothalamic Hypophyseal Inhibitory Factor (Hhif) Increases Intrasynaptosomal Free Calcium Concentration
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
A Comparative Kinetic Analysis Of The Flavin-Photosensitized Oxidation And Reduction Of Plastocyanin And Cytochrome C6from Different Organisms
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
A Comparative Kinetic Analysis Of The Flavin-Photosensitized Oxidation And Reduction Of Plastocyanin And Cytochrome C6from Different Organisms
Physical State Of Bulk And Protein-Associated Lipid In Nicotinic Acetylcholine Receptor-Rich Membrane Studied By Laurdan Generalized Polarization And Fluorescence Energy Transfer
A Comparative Kinetic Analysis Of The Flavin-Photosensitized Oxidation And Reduction Of Plastocyanin And Cytochrome C6from Different Organisms
Physical State Of Bulk And Protein-Associated Lipid In Nicotinic Acetylcholine Receptor-Rich Membrane Studied By Laurdan Generalized Polarization And Fluorescence Energy Transfer
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
Physical State Of Bulk And Protein-Associated Lipid In Nicotinic Acetylcholine Receptor-Rich Membrane Studied By Laurdan Generalized Polarization And Fluorescence Energy Transfer
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Rate Of Na+/ca2+ Exchange Across The Plasma Membrane Of Synaptosomes Measured Using The Fluorescence Of Chlorotetracycline. Implications To Calcium Homeostasis In Synaptic Terminals
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Rate Of Na+/ca2+ Exchange Across The Plasma Membrane Of Synaptosomes Measured Using The Fluorescence Of Chlorotetracycline. Implications To Calcium Homeostasis In Synaptic Terminals
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Rate Of Na+/ca2+ Exchange Across The Plasma Membrane Of Synaptosomes Measured Using The Fluorescence Of Chlorotetracycline. Implications To Calcium Homeostasis In Synaptic Terminals
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Preferential Distribution Of The Fluorescent Phospholipid Probes Nbd-Phosphatidylcholine And Rhodamine-Phosphatidylethanolamine In The Exofacial Leaflet Of Acetylcholine Receptor-Rich Membranes From Torpedo Marmorata
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Preferential Distribution Of The Fluorescent Phospholipid Probes Nbd-Phosphatidylcholine And Rhodamine-Phosphatidylethanolamine In The Exofacial Leaflet Of Acetylcholine Receptor-Rich Membranes From Torpedo Marmorata
Expression Of Sarcoplasmic Reticulum Ca2+-Atpase In Yeast (Pii:0014-5793(94)01104-4)
Preferential Distribution Of The Fluorescent Phospholipid Probes Nbd-Phosphatidylcholine And Rhodamine-Phosphatidylethanolamine In The Exofacial Leaflet Of Acetylcholine Receptor-Rich Membranes From Torpedo Marmorata
Expression Of Sarcoplasmic Reticulum Ca2+-Atpase In Yeast (Pii:0014-5793(94)01104-4)
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Expression Of Sarcoplasmic Reticulum Ca2+-Atpase In Yeast (Pii:0014-5793(94)01104-4)
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Modulation Of The Ca2+ Pump By The Hypothalamic-Hypophysary Inhibitory Factor
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Modulation Of The Ca2+ Pump By The Hypothalamic-Hypophysary Inhibitory Factor
Modulation Of The Ca 2+,mg2+-Atpase Of Sarcoplasmic Reticulum By The Hypothalamic Hypophyseal Inhibitory Factor
Modulation Of The Ca2+ Pump By The Hypothalamic-Hypophysary Inhibitory Factor
Modulation Of The Ca 2+,mg2+-Atpase Of Sarcoplasmic Reticulum By The Hypothalamic Hypophyseal Inhibitory Factor
Expression Of The Sarcoplasmic Reticulum Ca2+-Atpase In Yeast
Modulation Of The Ca 2+,mg2+-Atpase Of Sarcoplasmic Reticulum By The Hypothalamic Hypophyseal Inhibitory Factor
Expression Of The Sarcoplasmic Reticulum Ca2+-Atpase In Yeast
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Expression Of The Sarcoplasmic Reticulum Ca2+-Atpase In Yeast
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Seasonal Variation Of Exudate Of Cistus Ladanifer
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Seasonal Variation Of Exudate Of Cistus Ladanifer
Glycogen Phosphorolysis Can Form A Metabolic Shuttle To Support Ca2+ Uptake By Sarcoplasmic Reticulum Membranes In Skeletal Muscle
Seasonal Variation Of Exudate Of Cistus Ladanifer
Glycogen Phosphorolysis Can Form A Metabolic Shuttle To Support Ca2+ Uptake By Sarcoplasmic Reticulum Membranes In Skeletal Muscle
Advanced Course On Structure-Function Relationships Of Membrane Proteins: An Assessment Of Current Knowledge
Glycogen Phosphorolysis Can Form A Metabolic Shuttle To Support Ca2+ Uptake By Sarcoplasmic Reticulum Membranes In Skeletal Muscle
Advanced Course On Structure-Function Relationships Of Membrane Proteins: An Assessment Of Current Knowledge
Sedimentation Equilibrium Of Detergent-Solubilized Membrane Proteins In The Preparative Ultracentrifuge
Advanced Course On Structure-Function Relationships Of Membrane Proteins: An Assessment Of Current Knowledge
Sedimentation Equilibrium Of Detergent-Solubilized Membrane Proteins In The Preparative Ultracentrifuge
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Sedimentation Equilibrium Of Detergent-Solubilized Membrane Proteins In The Preparative Ultracentrifuge
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Properties Of The Purified Hypothalamic Pituitary Na/k-Atpase Inhibitor
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Properties Of The Purified Hypothalamic Pituitary Na/k-Atpase Inhibitor
Location Of Functional Centers In The Microsomal Cytochrome P450 System
Properties Of The Purified Hypothalamic Pituitary Na/k-Atpase Inhibitor
Location Of Functional Centers In The Microsomal Cytochrome P450 System
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Location Of Functional Centers In The Microsomal Cytochrome P450 System
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Modulation By Phosphorylation Of Glycogen Phosphorylase-Sarcoplasmic Reticulum Interaction
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Modulation By Phosphorylation Of Glycogen Phosphorylase-Sarcoplasmic Reticulum Interaction
Caracterización Por Técnicas Espectrofotométricas Del Ultravioleta Y Visible Edel Exudado De Cistus Ladanifer L. Como Respuesta Al Estrés Estival
Modulation By Phosphorylation Of Glycogen Phosphorylase-Sarcoplasmic Reticulum Interaction
Caracterización Por Técnicas Espectrofotométricas Del Ultravioleta Y Visible Edel Exudado De Cistus Ladanifer L. Como Respuesta Al Estrés Estival
Hemin And Hemeprotein Bleaching During Linoleic Acid Oxidation By Lipoxygenases
Caracterización Por Técnicas Espectrofotométricas Del Ultravioleta Y Visible Edel Exudado De Cistus Ladanifer L. Como Respuesta Al Estrés Estival
Hemin And Hemeprotein Bleaching During Linoleic Acid Oxidation By Lipoxygenases
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Hemin And Hemeprotein Bleaching During Linoleic Acid Oxidation By Lipoxygenases
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Modulation Of Calcium Fluxes Across Synaptosomal Plasma Membrane By Local Anesthetics
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Modulation Of Calcium Fluxes Across Synaptosomal Plasma Membrane By Local Anesthetics
Modulation Of The Ca2+,mg2+‐atpase Activity Of Synaptosomal Plasma Membrane By The Local Anesthetics Dibucaine And Lidocaine
Modulation Of Calcium Fluxes Across Synaptosomal Plasma Membrane By Local Anesthetics
Modulation Of The Ca2+,mg2+‐atpase Activity Of Synaptosomal Plasma Membrane By The Local Anesthetics Dibucaine And Lidocaine
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Modulation Of The Ca2+,mg2+‐atpase Activity Of Synaptosomal Plasma Membrane By The Local Anesthetics Dibucaine And Lidocaine
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Interaction Of The Local Anesthetics Dibucaine And Tetracaine With Sarcoplasmic Reticulum Membranes. Differential Scanning Calorimetry And Fluorescence Studies
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Interaction Of The Local Anesthetics Dibucaine And Tetracaine With Sarcoplasmic Reticulum Membranes. Differential Scanning Calorimetry And Fluorescence Studies
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Interaction Of The Local Anesthetics Dibucaine And Tetracaine With Sarcoplasmic Reticulum Membranes. Differential Scanning Calorimetry And Fluorescence Studies
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Local Anesthetic-Divalent Cation Binding Center Interaction
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Local Anesthetic-Divalent Cation Binding Center Interaction
Comparative Fluorescence Properties Of Lipoxygenases
Local Anesthetic-Divalent Cation Binding Center Interaction
Comparative Fluorescence Properties Of Lipoxygenases
Effects Of Local Anesthetics On The Passive Permeability Of Sarcoplasmic Reticulum Vesicles To Ca2+ And Mg2+
Comparative Fluorescence Properties Of Lipoxygenases
Effects Of Local Anesthetics On The Passive Permeability Of Sarcoplasmic Reticulum Vesicles To Ca2+ And Mg2+
The Position Of The Atp Binding Site On The (Ca2+ + Mg2+)-Atpase
Effects Of Local Anesthetics On The Passive Permeability Of Sarcoplasmic Reticulum Vesicles To Ca2+ And Mg2+
The Position Of The Atp Binding Site On The (Ca2+ + Mg2+)-Atpase
Long-Wavelength Fluorescence Of Tyrosine And Tryptophan Solutions.
The Position Of The Atp Binding Site On The (Ca2+ + Mg2+)-Atpase
Long-Wavelength Fluorescence Of Tyrosine And Tryptophan Solutions.
Local Anesthetics Inhibit The Ca2+,mg2+‐atpase Activity Of Rat Brain Synaptosomes
Long-Wavelength Fluorescence Of Tyrosine And Tryptophan Solutions.
Local Anesthetics Inhibit The Ca2+,mg2+‐atpase Activity Of Rat Brain Synaptosomes
Dependence Of The Fluorescence Of Fluorescein Labelled (Ca2+, Mg2+)-Atpase Upon The Lipid To Protein Ratio In Sarcoplasmic Reticulum Reconstituted Systems
Local Anesthetics Inhibit The Ca2+,mg2+‐atpase Activity Of Rat Brain Synaptosomes
Dependence Of The Fluorescence Of Fluorescein Labelled (Ca2+, Mg2+)-Atpase Upon The Lipid To Protein Ratio In Sarcoplasmic Reticulum Reconstituted Systems
On The Rate Of Association And Dissociation Processes Of Intrinsic Membrane Proteins
Dependence Of The Fluorescence Of Fluorescein Labelled (Ca2+, Mg2+)-Atpase Upon The Lipid To Protein Ratio In Sarcoplasmic Reticulum Reconstituted Systems
On The Rate Of Association And Dissociation Processes Of Intrinsic Membrane Proteins
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. Ii. Protein Distribution And Aggregation State In Biological Membranes
On The Rate Of Association And Dissociation Processes Of Intrinsic Membrane Proteins
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. Ii. Protein Distribution And Aggregation State In Biological Membranes
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. I. Lateral Phase Separation In Unilamellar Vesicles Formed By Binary Phospholipid Mixtures
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. Ii. Protein Distribution And Aggregation State In Biological Membranes
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. I. Lateral Phase Separation In Unilamellar Vesicles Formed By Binary Phospholipid Mixtures
Relationship Between The Activity Of Pancreatic Phospholipase A2 And The Physical State Of The Phospholipid Substrate.
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. I. Lateral Phase Separation In Unilamellar Vesicles Formed By Binary Phospholipid Mixtures
Relationship Between The Activity Of Pancreatic Phospholipase A2 And The Physical State Of The Phospholipid Substrate.
Regulation Of Phosphorylase B By Amp: The Effects Of Enzyme Concentration
Relationship Between The Activity Of Pancreatic Phospholipase A2 And The Physical State Of The Phospholipid Substrate.
Regulation Of Phosphorylase B By Amp: The Effects Of Enzyme Concentration
Editorial For The Special Issue “mapk In Health And Disease”
WEB ACCESIBLE