PUBLICACIÓN

Design and Experimental Evaluation of a Peptide Antagonist against Amyloid β(1–42) Interactions with Calmodulin and Calbindin-D28k

Logo Open Access

ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Salazar J.; Poejo J.; Mata A.M.; Samhan-Arias A.K.; Gutierrez-Merino C.

2022 INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES

Catalysis (Q2), Computer Science Applications (Q1), Inorganic Chemistry (Q1), Medicine (miscellaneous) (Q1), Molecular Biology (Q2), Organic Chemistry (Q1), Physical and Theoretical Chemistry (Q1), Spectroscopy (Q1)

JCR: 5.6

SJR: 1.154

CITAS

10

DOI

10.3390/ijms23042289

EID

2-s2.0-85124894100

ISSN

1661-6596

EISSN

1422-0067

BIBTEX

@article{Mata2022,title = {Design and Experimental Evaluation of a Peptide Antagonist against Amyloid ?(1?42) Interactions with Calmodulin and Calbindin-D28k},journal = {International Journal of Molecular Sciences},year = {2022},volume = {23},number = {4},author = {Salazar, J. and Poejo, J. and Mata, A.M. and Samhan-Arias, A.K. and Gutierrez-Merino, C.}}

AUTORES DE LA UEX

Ana María Mata Durán

OTRAS PUBLICACIONES DE INTERÉS

Regulation Of Phosphorylase B By Amp: The Effects Of Enzyme Concentration
Kaempferol Protects Against Amyloid Β Overproduction And The Rise Of Phospho-Tau 217 And Phospho-Tau 181 In The Rat Cerebellum Induced By Acute 3-Nitropropionic Acid Administration
Neurotoxic Amyloid Β‐peptide And Tau Produce Cytokine‐like Effects On Pmca In Glioblastoma Cell Lines, Enhancing Its Activity And Isoforms Expression
Kaempferol Protects Against Amyloid Β Overproduction And The Rise Of Phospho-Tau 217 And Phospho-Tau 181 In The Rat Cerebellum Induced By Acute 3-Nitropropionic Acid Administration
Neurotoxic Amyloid Β‐peptide And Tau Produce Cytokine‐like Effects On Pmca In Glioblastoma Cell Lines, Enhancing Its Activity And Isoforms Expression
Unveiling The Agonistic Properties Of Preyssler-Type Polyoxotungstates On Purinergic P2 Receptors
Neurotoxic Amyloid Β‐peptide And Tau Produce Cytokine‐like Effects On Pmca In Glioblastoma Cell Lines, Enhancing Its Activity And Isoforms Expression
Unveiling The Agonistic Properties Of Preyssler-Type Polyoxotungstates On Purinergic P2 Receptors
An Update Of Kaempferol Protection Against Brain Damage Induced By Ischemia-Reperfusion And By 3-Nitropropionic Acid
Unveiling The Agonistic Properties Of Preyssler-Type Polyoxotungstates On Purinergic P2 Receptors
An Update Of Kaempferol Protection Against Brain Damage Induced By Ischemia-Reperfusion And By 3-Nitropropionic Acid
The Plasma Membrane Ca2+-Atpase, A Molecular Target For Tau-Induced Cytosolic Calcium Dysregulation
An Update Of Kaempferol Protection Against Brain Damage Induced By Ischemia-Reperfusion And By 3-Nitropropionic Acid
The Plasma Membrane Ca2+-Atpase, A Molecular Target For Tau-Induced Cytosolic Calcium Dysregulation
Store-Operated Calcium Entry Inhibition And Plasma Membrane Calcium Pump Upregulation Contribute To The Maintenance Of Resting Cytosolic Calcium Concentration In A1-Like Astrocytes
The Plasma Membrane Ca2+-Atpase, A Molecular Target For Tau-Induced Cytosolic Calcium Dysregulation
Store-Operated Calcium Entry Inhibition And Plasma Membrane Calcium Pump Upregulation Contribute To The Maintenance Of Resting Cytosolic Calcium Concentration In A1-Like Astrocytes
Brain Hydrophobic Peptides Antagonists Of Neurotoxic Amyloid Β Peptide Monomers/oligomers–protein Interactions
Store-Operated Calcium Entry Inhibition And Plasma Membrane Calcium Pump Upregulation Contribute To The Maintenance Of Resting Cytosolic Calcium Concentration In A1-Like Astrocytes
Brain Hydrophobic Peptides Antagonists Of Neurotoxic Amyloid Β Peptide Monomers/oligomers–protein Interactions
Hexa-Histidine, A Peptide With Versatile Applications In The Study Of Amyloid-Β(1–42) Molecular Mechanisms Of Action
Brain Hydrophobic Peptides Antagonists Of Neurotoxic Amyloid Β Peptide Monomers/oligomers–protein Interactions
Hexa-Histidine, A Peptide With Versatile Applications In The Study Of Amyloid-Β(1–42) Molecular Mechanisms Of Action
Are There Lipid Membrane-Domain Subtypes In Neurons With Different Roles In Calcium Signaling?
Hexa-Histidine, A Peptide With Versatile Applications In The Study Of Amyloid-Β(1–42) Molecular Mechanisms Of Action
Are There Lipid Membrane-Domain Subtypes In Neurons With Different Roles In Calcium Signaling?
Biochemical And Biophysical Characterization Of The Caveolin-2 Interaction With Membranes And Analysis Of The Protein Structural Alteration By The Presence Of Cholesterol
Are There Lipid Membrane-Domain Subtypes In Neurons With Different Roles In Calcium Signaling?
Biochemical And Biophysical Characterization Of The Caveolin-2 Interaction With Membranes And Analysis Of The Protein Structural Alteration By The Presence Of Cholesterol
Structural Features Of Cytochrome B5 –cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Biochemical And Biophysical Characterization Of The Caveolin-2 Interaction With Membranes And Analysis Of The Protein Structural Alteration By The Presence Of Cholesterol
Structural Features Of Cytochrome B5 –cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Inhibition Of Serca And Pmca Ca2+-Atpase Activities By Polyoxotungstates
Structural Features Of Cytochrome B5 –cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Inhibition Of Serca And Pmca Ca2+-Atpase Activities By Polyoxotungstates
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Inhibition Of Serca And Pmca Ca2+-Atpase Activities By Polyoxotungstates
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Special Issue “molecular And Cellular Mechanisms Of Action Of Markers Of Tissue Degeneration”
Internalized Amyloid-Β (1-42) Peptide Inhibits The Store-Operated Calcium Entry In Ht-22 Cells
Special Issue “molecular And Cellular Mechanisms Of Action Of Markers Of Tissue Degeneration”
Design And Experimental Evaluation Of A Peptide Antagonist Against Amyloid Β(1–42) Interactions With Calmodulin And Calbindin-D28k
Special Issue “molecular And Cellular Mechanisms Of Action Of Markers Of Tissue Degeneration”
Design And Experimental Evaluation Of A Peptide Antagonist Against Amyloid Β(1–42) Interactions With Calmodulin And Calbindin-D28k
Kaempferol Prevents The Activation Of Complement C3 Protein And The Generation Of Reactive A1 Astrocytes That Mediate Rat Brain Degeneration Induced By 3-Nitropropionic Acid
Design And Experimental Evaluation Of A Peptide Antagonist Against Amyloid Β(1–42) Interactions With Calmodulin And Calbindin-D28k
Kaempferol Prevents The Activation Of Complement C3 Protein And The Generation Of Reactive A1 Astrocytes That Mediate Rat Brain Degeneration Induced By 3-Nitropropionic Acid
Gold Compounds Inhibit The Ca2+-Atpase Activity Of Brain Pmca And Human Neuroblastoma Sh-Sy5y Cells And Decrease Cell Viability
Kaempferol Prevents The Activation Of Complement C3 Protein And The Generation Of Reactive A1 Astrocytes That Mediate Rat Brain Degeneration Induced By 3-Nitropropionic Acid
Gold Compounds Inhibit The Ca2+-Atpase Activity Of Brain Pmca And Human Neuroblastoma Sh-Sy5y Cells And Decrease Cell Viability
Sorcin Activates The Brain Pmca And Blocks The Inhibitory Effects Of Molecular Markers Of Alzheimer’s Disease On The Pump Activity
Gold Compounds Inhibit The Ca2+-Atpase Activity Of Brain Pmca And Human Neuroblastoma Sh-Sy5y Cells And Decrease Cell Viability
Sorcin Activates The Brain Pmca And Blocks The Inhibitory Effects Of Molecular Markers Of Alzheimer’s Disease On The Pump Activity
The Relevance Of Amyloid Β-Calmodulin Complexation In Neurons And Brain Degeneration In Alzheimer’s Disease
Sorcin Activates The Brain Pmca And Blocks The Inhibitory Effects Of Molecular Markers Of Alzheimer’s Disease On The Pump Activity
The Relevance Of Amyloid Β-Calmodulin Complexation In Neurons And Brain Degeneration In Alzheimer’s Disease
The Endoplasmic Reticulum Ca2+-Atpase Serca2b Is Upregulated In Activated Microglia And Its Inhibition Causes Opposite Effects On Migration And Phagocytosis
The Relevance Of Amyloid Β-Calmodulin Complexation In Neurons And Brain Degeneration In Alzheimer’s Disease
The Endoplasmic Reticulum Ca2+-Atpase Serca2b Is Upregulated In Activated Microglia And Its Inhibition Causes Opposite Effects On Migration And Phagocytosis
Binding Of Amyloid Β(1–42)‐calmodulin Complexes To Plasma Membrane Lipid Rafts In Cerebellar Granule Neurons Alters Resting Cytosolic Calcium Homeostasis
The Endoplasmic Reticulum Ca2+-Atpase Serca2b Is Upregulated In Activated Microglia And Its Inhibition Causes Opposite Effects On Migration And Phagocytosis
Binding Of Amyloid Β(1–42)‐calmodulin Complexes To Plasma Membrane Lipid Rafts In Cerebellar Granule Neurons Alters Resting Cytosolic Calcium Homeostasis
Structural Features Of Cytochrome B5–cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Binding Of Amyloid Β(1–42)‐calmodulin Complexes To Plasma Membrane Lipid Rafts In Cerebellar Granule Neurons Alters Resting Cytosolic Calcium Homeostasis
Structural Features Of Cytochrome B5–cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Human Erythrocytes Exposure To Juglone Leads To An Increase Of Superoxide Anion Production Associated With Cytochrome B5 Reductase Uncoupling
Structural Features Of Cytochrome B5–cytochrome B5 Reductase Complex Formation And Implications For The Intramolecular Dynamics Of Cytochrome B5 Reductase
Human Erythrocytes Exposure To Juglone Leads To An Increase Of Superoxide Anion Production Associated With Cytochrome B5 Reductase Uncoupling
Early Reactive A1 Astrocytes Induction By The Neurotoxin 3-Nitropropionic Acid In Rat Brain
Human Erythrocytes Exposure To Juglone Leads To An Increase Of Superoxide Anion Production Associated With Cytochrome B5 Reductase Uncoupling
Early Reactive A1 Astrocytes Induction By The Neurotoxin 3-Nitropropionic Acid In Rat Brain
Modulation Of Cyp2c9 Activity And Hydrogen Peroxide Production By Cytochrome B 5
Early Reactive A1 Astrocytes Induction By The Neurotoxin 3-Nitropropionic Acid In Rat Brain
Modulation Of Cyp2c9 Activity And Hydrogen Peroxide Production By Cytochrome B 5
Ligand Accessibility To Heme Cytochrome B 5 Coordinating Sphere And Enzymatic Activity Enhancement Upon Tyrosine Ionization
Modulation Of Cyp2c9 Activity And Hydrogen Peroxide Production By Cytochrome B 5
Ligand Accessibility To Heme Cytochrome B 5 Coordinating Sphere And Enzymatic Activity Enhancement Upon Tyrosine Ionization
Methylene Blue Blocks And Reverses The Inhibitory Effect Of Tau On Pmca Function
Ligand Accessibility To Heme Cytochrome B 5 Coordinating Sphere And Enzymatic Activity Enhancement Upon Tyrosine Ionization
Methylene Blue Blocks And Reverses The Inhibitory Effect Of Tau On Pmca Function
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Methylene Blue Blocks And Reverses The Inhibitory Effect Of Tau On Pmca Function
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Methyl-Β-Cyclodextrin Impairs The Phosphorylation Of The Β2 Subunit Of L-Type Calcium Channels And Cytosolic Calcium Homeostasis In Mature Cerebellar Granule Neurons
Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)
Methyl-Β-Cyclodextrin Impairs The Phosphorylation Of The Β2 Subunit Of L-Type Calcium Channels And Cytosolic Calcium Homeostasis In Mature Cerebellar Granule Neurons
Creatine Protects Against Cytosolic Calcium Dysregulation, Mitochondrial Depolarization And Increase Of Reactive Oxygen Species Production In Rotenone-Induced Cell Death Of Cerebellar Granule Neurons
Methyl-Β-Cyclodextrin Impairs The Phosphorylation Of The Β2 Subunit Of L-Type Calcium Channels And Cytosolic Calcium Homeostasis In Mature Cerebellar Granule Neurons
Creatine Protects Against Cytosolic Calcium Dysregulation, Mitochondrial Depolarization And Increase Of Reactive Oxygen Species Production In Rotenone-Induced Cell Death Of Cerebellar Granule Neurons
Cytochrome B5 Reductase Is The Component From Neuronal Synaptic Plasma Membrane Vesicles That Generates Superoxide Anion Upon Stimulation By Cytochrome C
Creatine Protects Against Cytosolic Calcium Dysregulation, Mitochondrial Depolarization And Increase Of Reactive Oxygen Species Production In Rotenone-Induced Cell Death Of Cerebellar Granule Neurons
Cytochrome B5 Reductase Is The Component From Neuronal Synaptic Plasma Membrane Vesicles That Generates Superoxide Anion Upon Stimulation By Cytochrome C
Topography Of Human Cytochrome B5/cytochrome B5 Reductase Interacting Domain And Redox Alterations Upon Complex Formation
Cytochrome B5 Reductase Is The Component From Neuronal Synaptic Plasma Membrane Vesicles That Generates Superoxide Anion Upon Stimulation By Cytochrome C
Topography Of Human Cytochrome B5/cytochrome B5 Reductase Interacting Domain And Redox Alterations Upon Complex Formation
Peroxidase-Like Activity Of Cytochrome B5 Is Triggered Upon Hemichrome Formation In Alkaline Ph
Topography Of Human Cytochrome B5/cytochrome B5 Reductase Interacting Domain And Redox Alterations Upon Complex Formation
Peroxidase-Like Activity Of Cytochrome B5 Is Triggered Upon Hemichrome Formation In Alkaline Ph
Methylene Blue Activates The Pmca Activity And Cross-Interacts With Amyloid Β-Peptide, Blocking Aβ-Mediated Pmca Inhibition
Peroxidase-Like Activity Of Cytochrome B5 Is Triggered Upon Hemichrome Formation In Alkaline Ph
Methylene Blue Activates The Pmca Activity And Cross-Interacts With Amyloid Β-Peptide, Blocking Aβ-Mediated Pmca Inhibition
Functional Interplay Between Plasma Membrane Ca2+-Atpase, Amyloid Β-Peptide And Tau
Methylene Blue Activates The Pmca Activity And Cross-Interacts With Amyloid Β-Peptide, Blocking Aβ-Mediated Pmca Inhibition
Functional Interplay Between Plasma Membrane Ca2+-Atpase, Amyloid Β-Peptide And Tau
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Functional Interplay Between Plasma Membrane Ca2+-Atpase, Amyloid Β-Peptide And Tau
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Phospholipids And Calmodulin Modulate The Inhibition Of Pmca Activity By Tau
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Phospholipids And Calmodulin Modulate The Inhibition Of Pmca Activity By Tau
High Affinity Binding Of Amyloid Β-Peptide To Calmodulin: Structural And Functional Implications
Phospholipids And Calmodulin Modulate The Inhibition Of Pmca Activity By Tau
High Affinity Binding Of Amyloid Β-Peptide To Calmodulin: Structural And Functional Implications
Correlation Between The Potency Of Flavonoids For Cytochrome C Reduction And Inhibition Of Cardiolipin-Induced Peroxidase Activity
High Affinity Binding Of Amyloid Β-Peptide To Calmodulin: Structural And Functional Implications
Correlation Between The Potency Of Flavonoids For Cytochrome C Reduction And Inhibition Of Cardiolipin-Induced Peroxidase Activity
An Improved Method For Expression And Purification Of Functional Human Ca2+ Transporter Pmca4b In Saccharomyces Cerevisiae
Correlation Between The Potency Of Flavonoids For Cytochrome C Reduction And Inhibition Of Cardiolipin-Induced Peroxidase Activity
An Improved Method For Expression And Purification Of Functional Human Ca2+ Transporter Pmca4b In Saccharomyces Cerevisiae
High Expression Of Cytochrome B 5 Reductase Isoform 3/cytochrome B 5 System In The Cerebellum And Pyramidal Neurons Of Adult Rat Brain
An Improved Method For Expression And Purification Of Functional Human Ca2+ Transporter Pmca4b In Saccharomyces Cerevisiae
High Expression Of Cytochrome B 5 Reductase Isoform 3/cytochrome B 5 System In The Cerebellum And Pyramidal Neurons Of Adult Rat Brain
Inhibition Of Pmca Activity By Tau As A Function Of Aging And Alzheimer's Neuropathology
High Expression Of Cytochrome B 5 Reductase Isoform 3/cytochrome B 5 System In The Cerebellum And Pyramidal Neurons Of Adult Rat Brain
Inhibition Of Pmca Activity By Tau As A Function Of Aging And Alzheimer's Neuropathology
Cytochrome B5 As A Pleiotropic Metabolic Modulator In Mammalian Cells
Inhibition Of Pmca Activity By Tau As A Function Of Aging And Alzheimer's Neuropathology
Cytochrome B5 As A Pleiotropic Metabolic Modulator In Mammalian Cells
Presynaptic Control Of Glycine Transporter 2 (Glyt2) By Physical And Functional Association With Plasma Membrane Ca2+-Atpase (Pmca) And Na+-Ca2+ Exchanger (Ncx)
Cytochrome B5 As A Pleiotropic Metabolic Modulator In Mammalian Cells
Presynaptic Control Of Glycine Transporter 2 (Glyt2) By Physical And Functional Association With Plasma Membrane Ca2+-Atpase (Pmca) And Na+-Ca2+ Exchanger (Ncx)
Heterologous Microarray Analysis Of Transcriptome Alterations In Mus Spretus Mice Living In An Industrial Settlement
Presynaptic Control Of Glycine Transporter 2 (Glyt2) By Physical And Functional Association With Plasma Membrane Ca2+-Atpase (Pmca) And Na+-Ca2+ Exchanger (Ncx)
Heterologous Microarray Analysis Of Transcriptome Alterations In Mus Spretus Mice Living In An Industrial Settlement
The Decrease Of Nad(P)h:quinone Oxidoreductase 1 Activity And Increase Of Ros Production By Nadph Oxidases Are Early Biomarkers In Doxorubicin Cardiotoxicity
Heterologous Microarray Analysis Of Transcriptome Alterations In Mus Spretus Mice Living In An Industrial Settlement
The Decrease Of Nad(P)h:quinone Oxidoreductase 1 Activity And Increase Of Ros Production By Nadph Oxidases Are Early Biomarkers In Doxorubicin Cardiotoxicity
Caveolin-Rich Lipid Rafts Of The Plasma Membrane Of Mature Cerebellar Granule Neurons Are Microcompartments For Calcium/reactive Oxygen And Nitrogen Species Cross-Talk Signaling
The Decrease Of Nad(P)h:quinone Oxidoreductase 1 Activity And Increase Of Ros Production By Nadph Oxidases Are Early Biomarkers In Doxorubicin Cardiotoxicity
Caveolin-Rich Lipid Rafts Of The Plasma Membrane Of Mature Cerebellar Granule Neurons Are Microcompartments For Calcium/reactive Oxygen And Nitrogen Species Cross-Talk Signaling
Purified Nadh-Cytochrome B5 Reductase Is A Novel Superoxide Anion Source Inhibited By Apocynin: Sensitivity To Nitric Oxide And Peroxynitrite
Caveolin-Rich Lipid Rafts Of The Plasma Membrane Of Mature Cerebellar Granule Neurons Are Microcompartments For Calcium/reactive Oxygen And Nitrogen Species Cross-Talk Signaling
Purified Nadh-Cytochrome B5 Reductase Is A Novel Superoxide Anion Source Inhibited By Apocynin: Sensitivity To Nitric Oxide And Peroxynitrite
High Levels Of Mn2+ Inhibit Secretory Pathway Ca 2+/mn2+-Atpase (Spca) Activity And Cause Golgi Fragmentation In Neurons And Glia
Purified Nadh-Cytochrome B5 Reductase Is A Novel Superoxide Anion Source Inhibited By Apocynin: Sensitivity To Nitric Oxide And Peroxynitrite
High Levels Of Mn2+ Inhibit Secretory Pathway Ca 2+/mn2+-Atpase (Spca) Activity And Cause Golgi Fragmentation In Neurons And Glia
Evaluation Of Manganese Uptake And Toxicity In Mouse Brain During Continuous Mncl2 Administration Using Osmotic Pumps
High Levels Of Mn2+ Inhibit Secretory Pathway Ca 2+/mn2+-Atpase (Spca) Activity And Cause Golgi Fragmentation In Neurons And Glia
Evaluation Of Manganese Uptake And Toxicity In Mouse Brain During Continuous Mncl2 Administration Using Osmotic Pumps
Calmodulin Antagonizes Amyloid-Β Peptides-Mediated Inhibition Of Brain Plasma Membrane Ca 2+-Atpase
Evaluation Of Manganese Uptake And Toxicity In Mouse Brain During Continuous Mncl2 Administration Using Osmotic Pumps
Calmodulin Antagonizes Amyloid-Β Peptides-Mediated Inhibition Of Brain Plasma Membrane Ca 2+-Atpase
Stimulation And Clustering Of Cytochrome B 5 Reductase In Caveolin-Rich Lipid Microdomains Is An Early Event In Oxidative Stress-Mediated Apoptosis Of Cerebellar Granule Neurons
Calmodulin Antagonizes Amyloid-Β Peptides-Mediated Inhibition Of Brain Plasma Membrane Ca 2+-Atpase
Stimulation And Clustering Of Cytochrome B 5 Reductase In Caveolin-Rich Lipid Microdomains Is An Early Event In Oxidative Stress-Mediated Apoptosis Of Cerebellar Granule Neurons
L-Type Voltage-Operated Calcium Channels, N-Methyl-D-Aspartate Receptors And Neuronal Nitric-Oxide Synthase Form A Calcium/redox Nano-Transducer Within Lipid Rafts
Stimulation And Clustering Of Cytochrome B 5 Reductase In Caveolin-Rich Lipid Microdomains Is An Early Event In Oxidative Stress-Mediated Apoptosis Of Cerebellar Granule Neurons
L-Type Voltage-Operated Calcium Channels, N-Methyl-D-Aspartate Receptors And Neuronal Nitric-Oxide Synthase Form A Calcium/redox Nano-Transducer Within Lipid Rafts
Impairment Of The Activity Of The Plasma Membrane Ca2+-Atpase In Alzheimer's Disease
L-Type Voltage-Operated Calcium Channels, N-Methyl-D-Aspartate Receptors And Neuronal Nitric-Oxide Synthase Form A Calcium/redox Nano-Transducer Within Lipid Rafts
Impairment Of The Activity Of The Plasma Membrane Ca2+-Atpase In Alzheimer's Disease
Complex I And Cytochrome C Are Molecular Targets Of Flavonoids That Inhibit Hydrogen Peroxide Production By Mitochondria
Impairment Of The Activity Of The Plasma Membrane Ca2+-Atpase In Alzheimer's Disease
Complex I And Cytochrome C Are Molecular Targets Of Flavonoids That Inhibit Hydrogen Peroxide Production By Mitochondria
Neuroprotective Actions Of Flavonoids
Complex I And Cytochrome C Are Molecular Targets Of Flavonoids That Inhibit Hydrogen Peroxide Production By Mitochondria
Neuroprotective Actions Of Flavonoids
Early Disruption Of The Actin Cytoskeleton In Cultured Cerebellar Granule Neurons Exposed To 3-Morpholinosydnonimine-Oxidative Stress Is Linked To Alterations Of The Cytosolic Calcium Concentration
Neuroprotective Actions Of Flavonoids
Early Disruption Of The Actin Cytoskeleton In Cultured Cerebellar Granule Neurons Exposed To 3-Morpholinosydnonimine-Oxidative Stress Is Linked To Alterations Of The Cytosolic Calcium Concentration
Reactivity Of Hydrogen Sulfide With Peroxynitrite And Other Oxidants Of Biological Interest
Early Disruption Of The Actin Cytoskeleton In Cultured Cerebellar Granule Neurons Exposed To 3-Morpholinosydnonimine-Oxidative Stress Is Linked To Alterations Of The Cytosolic Calcium Concentration
Reactivity Of Hydrogen Sulfide With Peroxynitrite And Other Oxidants Of Biological Interest
Peroxynitrite-Mediated Oxidative Modifications Of Myosin And Implications On Structure And Function
Reactivity Of Hydrogen Sulfide With Peroxynitrite And Other Oxidants Of Biological Interest
Peroxynitrite-Mediated Oxidative Modifications Of Myosin And Implications On Structure And Function
L-Type Calcium Channels And Cytochrome B5 Reductase Are Components Of Protein Complexes Tightly Associated With Lipid Rafts Microdomains Of The Neuronal Plasma Membrane
Peroxynitrite-Mediated Oxidative Modifications Of Myosin And Implications On Structure And Function
L-Type Calcium Channels And Cytochrome B5 Reductase Are Components Of Protein Complexes Tightly Associated With Lipid Rafts Microdomains Of The Neuronal Plasma Membrane
Silencing The Spca1 (Secretory Pathway Ca2+-Atpase Isoform 1) Impairs Ca2+ Homeostasis In The Golgi And Disturbs Neural Polarity
L-Type Calcium Channels And Cytochrome B5 Reductase Are Components Of Protein Complexes Tightly Associated With Lipid Rafts Microdomains Of The Neuronal Plasma Membrane
Silencing The Spca1 (Secretory Pathway Ca2+-Atpase Isoform 1) Impairs Ca2+ Homeostasis In The Golgi And Disturbs Neural Polarity
Ontogeny Of Atp Hydrolysis And Isoform Expression Of The Plasma Membrane Ca2+-Atpase In Mouse Brain
Silencing The Spca1 (Secretory Pathway Ca2+-Atpase Isoform 1) Impairs Ca2+ Homeostasis In The Golgi And Disturbs Neural Polarity
Ontogeny Of Atp Hydrolysis And Isoform Expression Of The Plasma Membrane Ca2+-Atpase In Mouse Brain
Altered Ca2+ Dependence Of Synaptosomal Plasma Membrane Ca 2+-Atpase In Human Brain Affected By Alzheimer's Disease
Ontogeny Of Atp Hydrolysis And Isoform Expression Of The Plasma Membrane Ca2+-Atpase In Mouse Brain
Altered Ca2+ Dependence Of Synaptosomal Plasma Membrane Ca 2+-Atpase In Human Brain Affected By Alzheimer's Disease
Kaempferol Protects Against Rat Striatal Degeneration Induced By 3-Nitropropionic Acid
Altered Ca2+ Dependence Of Synaptosomal Plasma Membrane Ca 2+-Atpase In Human Brain Affected By Alzheimer's Disease
Kaempferol Protects Against Rat Striatal Degeneration Induced By 3-Nitropropionic Acid
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Kaempferol Protects Against Rat Striatal Degeneration Induced By 3-Nitropropionic Acid
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Hydrogen Sulfide Is A Reversible Inhibitor Of The Nadh Oxidase Activity Of Synaptic Plasma Membranes
Clustering Of Plasma Membrane-Bound Cytochrome B5 Reductase Within 'lipid Raft' Microdomains Of The Neuronal Plasma Membrane
Hydrogen Sulfide Is A Reversible Inhibitor Of The Nadh Oxidase Activity Of Synaptic Plasma Membranes
Activity And Localization Of The Secretory Pathway Ca2+-Atpase Isoform 1 (Spca1) In Different Areas Of The Mouse Brain During Postnatal Development
Hydrogen Sulfide Is A Reversible Inhibitor Of The Nadh Oxidase Activity Of Synaptic Plasma Membranes
Activity And Localization Of The Secretory Pathway Ca2+-Atpase Isoform 1 (Spca1) In Different Areas Of The Mouse Brain During Postnatal Development
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Activity And Localization Of The Secretory Pathway Ca2+-Atpase Isoform 1 (Spca1) In Different Areas Of The Mouse Brain During Postnatal Development
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Reduction Of Ascorbate Free Radical By The Plasma Membrane Of Synaptic Terminals From Rat Brain
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Hydrogen Sulfide Raises Cytosolic Calcium In Neurons Through Activation Of L-Type Ca2+ Channels
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Vanadium And Cadmium In Vivo Effects In Teleost Cardiac Muscle: Metal Accumulation And Oxidative Stress Markers
Vanadate Induces Necrotic Death In Neonatal Rat Cardiomyocytes Through Mitochondrial Membrane Depolarization
Vanadium And Cadmium In Vivo Effects In Teleost Cardiac Muscle: Metal Accumulation And Oxidative Stress Markers
Functional And Immunocytochemical Evidence For The Expression And Localization Of The Secretory Pathway Ca2+-Atpase Isoform 1 (Spca1) In Cerebellum Relative To Other Ca2+ Pumps
Vanadium And Cadmium In Vivo Effects In Teleost Cardiac Muscle: Metal Accumulation And Oxidative Stress Markers
Functional And Immunocytochemical Evidence For The Expression And Localization Of The Secretory Pathway Ca2+-Atpase Isoform 1 (Spca1) In Cerebellum Relative To Other Ca2+ Pumps
Developmental Distribution Of Plasma Membrane Ca2+-Atpase Isoforms In Chick Cerebellum
Functional And Immunocytochemical Evidence For The Expression And Localization Of The Secretory Pathway Ca2+-Atpase Isoform 1 (Spca1) In Cerebellum Relative To Other Ca2+ Pumps
Developmental Distribution Of Plasma Membrane Ca2+-Atpase Isoforms In Chick Cerebellum
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Developmental Distribution Of Plasma Membrane Ca2+-Atpase Isoforms In Chick Cerebellum
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Biological Effects Of Decavanadate: Muscle Contraction, In Vivo Oxidative Stress, And Mitochondrial Toxicity
Blood Micromolar Concentrations Of Kaempferol Afford Protection Against Ischemia/reperfusion-Induced Damage In Rat Brain
Biological Effects Of Decavanadate: Muscle Contraction, In Vivo Oxidative Stress, And Mitochondrial Toxicity
Vanadium Distribution, Lipid Peroxidation And Oxidative Stress Markers Upon Decavanadate In Vivo Administration
Biological Effects Of Decavanadate: Muscle Contraction, In Vivo Oxidative Stress, And Mitochondrial Toxicity
Vanadium Distribution, Lipid Peroxidation And Oxidative Stress Markers Upon Decavanadate In Vivo Administration
Mitochondria As A Target For Decavanadate Toxicity In Sparus Aurata Heart
Vanadium Distribution, Lipid Peroxidation And Oxidative Stress Markers Upon Decavanadate In Vivo Administration
Mitochondria As A Target For Decavanadate Toxicity In Sparus Aurata Heart
Decavanadate Induces Mitochondrial Membrane Depolarization And Inhibits Oxygen Consumption
Mitochondria As A Target For Decavanadate Toxicity In Sparus Aurata Heart
Decavanadate Induces Mitochondrial Membrane Depolarization And Inhibits Oxygen Consumption
Binding Modes Of Decavanadate To Myosin And Inhibition Of The Actomyosin Atpase Activity
Decavanadate Induces Mitochondrial Membrane Depolarization And Inhibits Oxygen Consumption
Binding Modes Of Decavanadate To Myosin And Inhibition Of The Actomyosin Atpase Activity
The Plasma Membrane Ca2+-Atpase Isoform 4 Is Localized In Lipid Rafts Of Cerebellum Synaptic Plasma Membranes
Binding Modes Of Decavanadate To Myosin And Inhibition Of The Actomyosin Atpase Activity
The Plasma Membrane Ca2+-Atpase Isoform 4 Is Localized In Lipid Rafts Of Cerebellum Synaptic Plasma Membranes
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
The Plasma Membrane Ca2+-Atpase Isoform 4 Is Localized In Lipid Rafts Of Cerebellum Synaptic Plasma Membranes
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Regionalization Of Plasma Membrane-Bound Flavoproteins Of Cerebellar Granule Neurons In Culture By Fluorescence Energy Transfer Imaging
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Decavanadate Interactions With Actin: Inhibition Of G-Actin Polymerization And Stabilization Of Decameric Vanadate
Inhibition Of Skeletal Muscle S1-Myosin Atpase By Peroxynitrite
Decavanadate Interactions With Actin: Inhibition Of G-Actin Polymerization And Stabilization Of Decameric Vanadate
Peroxynitrite Induces F-Actin Depolymerization And Blockade Of Myosin Atpase Stimulation
Decavanadate Interactions With Actin: Inhibition Of G-Actin Polymerization And Stabilization Of Decameric Vanadate
Peroxynitrite Induces F-Actin Depolymerization And Blockade Of Myosin Atpase Stimulation
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Peroxynitrite Induces F-Actin Depolymerization And Blockade Of Myosin Atpase Stimulation
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Transfemoral Selective Intraluminal Wiring Technique For Transient Middle Cerebral Artery Occlusion In Rats
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Vanadate Oligomers: In Vivo Effects In Hepatic Vanadium Accumulation And Stress Markers
Alteration Of Cytosolic Free Calcium Homeostasis By Sin-1: High Sensitivity Of L-Type Ca2+ Channels To Extracellular Oxidative/nitrosative Stress In Cerebellar Granule Cells
Vanadate Oligomers: In Vivo Effects In Hepatic Vanadium Accumulation And Stress Markers
A Developmental Profile Of The Levels Of Calcium Pumps In Chick Cerebellum
Vanadate Oligomers: In Vivo Effects In Hepatic Vanadium Accumulation And Stress Markers
A Developmental Profile Of The Levels Of Calcium Pumps In Chick Cerebellum
Calcium Pumps In The Central Nervous System
A Developmental Profile Of The Levels Of Calcium Pumps In Chick Cerebellum
Calcium Pumps In The Central Nervous System
Localization Of Intracellular And Plasma Membrane Ca2+-Atpases In The Cerebellum
Calcium Pumps In The Central Nervous System
Localization Of Intracellular And Plasma Membrane Ca2+-Atpases In The Cerebellum
Glycogen Synthase Kinase-3 Plays A Crucial Role In Tau Exon 10 Splicing And Intranuclear Distribution Of Sc35: Implications For Alzheimer's Disease
Localization Of Intracellular And Plasma Membrane Ca2+-Atpases In The Cerebellum
Glycogen Synthase Kinase-3 Plays A Crucial Role In Tau Exon 10 Splicing And Intranuclear Distribution Of Sc35: Implications For Alzheimer's Disease
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Glycogen Synthase Kinase-3 Plays A Crucial Role In Tau Exon 10 Splicing And Intranuclear Distribution Of Sc35: Implications For Alzheimer's Disease
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Modulation Of Sarcoplasmic Reticulum Ca2+-Atpase By Chronic And Acute Exposure To Peroxynitrite
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Kaempferol Blocks Oxidative Stress In Cerebellar Granule Cells And Reveals A Key Role For Reactive Oxygen Species Production At The Plasma Membrane In The Commitment To Apoptosis
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Fluorescence Measurements Of Steady State Peroxynitrite Production Upon Sin-1 Decomposition: Nadh Versus Dihydrodichlorofluorescein And Dihydrorhodamine 123
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Decavanadate Binding To A High Affinity Site Near The Myosin Catalytic Centre Inhibits F-Actin-Stimulated Myosin Atpase Activity
Inhibition Of Mouth Skeletal Muscle Relaxation By Flavonoids Of Cistus Ladanifer L.: A Plant Defense Mechanism Against Herbivores
Decavanadate Binding To A High Affinity Site Near The Myosin Catalytic Centre Inhibits F-Actin-Stimulated Myosin Atpase Activity
The Interaction Of Ethanol With Reconstituted Synaptosomal Plasma Membrane Ca 2+-Atpase
Decavanadate Binding To A High Affinity Site Near The Myosin Catalytic Centre Inhibits F-Actin-Stimulated Myosin Atpase Activity
The Interaction Of Ethanol With Reconstituted Synaptosomal Plasma Membrane Ca 2+-Atpase
Ca2+ Transport By The Synaptosomal Plasma Membrane Ca 2+-Atpase And The Effect Of Thioridazinet
The Interaction Of Ethanol With Reconstituted Synaptosomal Plasma Membrane Ca 2+-Atpase
Ca2+ Transport By The Synaptosomal Plasma Membrane Ca 2+-Atpase And The Effect Of Thioridazinet
Localization Of Endoplasmic Reticulum And Plasma Membrane Ca 2+-Atpases In Subcellular Fractions And Sections Of Pig Cerebellum
Ca2+ Transport By The Synaptosomal Plasma Membrane Ca 2+-Atpase And The Effect Of Thioridazinet
Localization Of Endoplasmic Reticulum And Plasma Membrane Ca 2+-Atpases In Subcellular Fractions And Sections Of Pig Cerebellum
The Trapping Of The Oh Radical By Coenzyme Q. A Theoretical And Experimental Study
Localization Of Endoplasmic Reticulum And Plasma Membrane Ca 2+-Atpases In Subcellular Fractions And Sections Of Pig Cerebellum
The Trapping Of The Oh Radical By Coenzyme Q. A Theoretical And Experimental Study
Erratum: Effect Of Spermine On The Activity Of Synaptosomal Plasma Membrane Ca2+-Atpase Reconstituted In Neutral Or Acidic Phospholipids (Biochimica Et Biophysica Acta (2003) 1611 (197-203))
The Trapping Of The Oh Radical By Coenzyme Q. A Theoretical And Experimental Study
Erratum: Effect Of Spermine On The Activity Of Synaptosomal Plasma Membrane Ca2+-Atpase Reconstituted In Neutral Or Acidic Phospholipids (Biochimica Et Biophysica Acta (2003) 1611 (197-203))
Effect Of Spermine On The Activity Of Synaptosomal Plasma Membrane Ca2+-Atpase Reconstituted In Neutral Or Acidic Phospholipids
Erratum: Effect Of Spermine On The Activity Of Synaptosomal Plasma Membrane Ca2+-Atpase Reconstituted In Neutral Or Acidic Phospholipids (Biochimica Et Biophysica Acta (2003) 1611 (197-203))
Effect Of Spermine On The Activity Of Synaptosomal Plasma Membrane Ca2+-Atpase Reconstituted In Neutral Or Acidic Phospholipids
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
Effect Of Spermine On The Activity Of Synaptosomal Plasma Membrane Ca2+-Atpase Reconstituted In Neutral Or Acidic Phospholipids
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Inhibition Of Oxidative Stress Produced By Plasma Membrane Nadh Oxidase Delays Low-Potassium-Induced Apoptosis Of Cerebellar Granule Cells
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
The Nadh Oxidase Activity Of The Plasma Membrane Of Synaptosomes Is A Major Source Of Superoxide Anion And Is Inhibited By Peroxynitrite
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
Quenching Of Myosin Intrinsic Fluorescence Unravels The Existence Of A High Affinity Binding Site For Decavanadate
Synaptosomal Plasma Membrane Ca2+ Pump Activity Inhibition By Repetitive Micromolar Onoo- Pulses
Quenching Of Myosin Intrinsic Fluorescence Unravels The Existence Of A High Affinity Binding Site For Decavanadate
Sphingomyelin Composition And Physical Asymmetries In Native Acetylcholine Receptor-Rich Membranes
Quenching Of Myosin Intrinsic Fluorescence Unravels The Existence Of A High Affinity Binding Site For Decavanadate
Sphingomyelin Composition And Physical Asymmetries In Native Acetylcholine Receptor-Rich Membranes
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Sphingomyelin Composition And Physical Asymmetries In Native Acetylcholine Receptor-Rich Membranes
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Distribution Of The Intracellular Ca2+-Atpase Isoform 2b In Pig Brain Subcellular Fractions And Cross-Reaction With A Monoclonal Antibody Raised Against The Enzyme Isoform 1
The Content Of Glycogen Phosphorylase And Glycogen In Preparations Of Sarcoplasmic Reticulum - Glycogenolytic Complex Is Enhanced In Diabetic Rat Skeletal Muscle
Distribution Of The Intracellular Ca2+-Atpase Isoform 2b In Pig Brain Subcellular Fractions And Cross-Reaction With A Monoclonal Antibody Raised Against The Enzyme Isoform 1
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
Distribution Of The Intracellular Ca2+-Atpase Isoform 2b In Pig Brain Subcellular Fractions And Cross-Reaction With A Monoclonal Antibody Raised Against The Enzyme Isoform 1
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
The Expression Of Plasma Membrane Ca2+ Pump Isoforms In Cerebellar Granule Neurons Is Modulated By Ca2+
Potassium-Induced Apoptosis In Rat Cerebellar Granule Cells Involves Cell-Cycle Blockade At The G1/s Transition
The Expression Of Plasma Membrane Ca2+ Pump Isoforms In Cerebellar Granule Neurons Is Modulated By Ca2+
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
The Expression Of Plasma Membrane Ca2+ Pump Isoforms In Cerebellar Granule Neurons Is Modulated By Ca2+
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Ph And Ligand Binding Modulate The Strength Of Protein-Protein Interactions In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Analysis Of Secreted Flavonoids Of Cistus Ladanifer L. By High- Performance Liquid Chromatography-Particle Beam Mass Spectrometry
Plausible Stoichiometry Of The Interacting Nucleotide-Binding Sites In The Ca2+-Atpase From Sarcoplasmic Reticulum Membranes
Analysis Of Secreted Flavonoids Of Cistus Ladanifer L. By High- Performance Liquid Chromatography-Particle Beam Mass Spectrometry
Ca2+ Transport By Reconstituted Synaptosomal Atpase Is Associated With H+ Countertransport And Net Charge Displacement
Analysis Of Secreted Flavonoids Of Cistus Ladanifer L. By High- Performance Liquid Chromatography-Particle Beam Mass Spectrometry
Ca2+ Transport By Reconstituted Synaptosomal Atpase Is Associated With H+ Countertransport And Net Charge Displacement
Characterization Of The Intracellular And The Plasma Membrane Ca2+- Atpases In Fractionated Pig Brain Membranes Using Calcium Pump Inhibitors
Ca2+ Transport By Reconstituted Synaptosomal Atpase Is Associated With H+ Countertransport And Net Charge Displacement
Characterization Of The Intracellular And The Plasma Membrane Ca2+- Atpases In Fractionated Pig Brain Membranes Using Calcium Pump Inhibitors
Quantitative Variation Of Flavonoids Among Individuals Of A Cistus Ladanifer Population
Characterization Of The Intracellular And The Plasma Membrane Ca2+- Atpases In Fractionated Pig Brain Membranes Using Calcium Pump Inhibitors
Quantitative Variation Of Flavonoids Among Individuals Of A Cistus Ladanifer Population
Role Of Ecological Variables In The Seasonal Variation Of Flavonoid Content Of Cistus Ladanifer Exudate
Quantitative Variation Of Flavonoids Among Individuals Of A Cistus Ladanifer Population
Role Of Ecological Variables In The Seasonal Variation Of Flavonoid Content Of Cistus Ladanifer Exudate
Evidence That An Ant1-Serca1 Monoclonal Antibody Recognizes The Serca2b Calcium Pump In Pig Brain
Role Of Ecological Variables In The Seasonal Variation Of Flavonoid Content Of Cistus Ladanifer Exudate
Evidence That An Ant1-Serca1 Monoclonal Antibody Recognizes The Serca2b Calcium Pump In Pig Brain
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Evidence That An Ant1-Serca1 Monoclonal Antibody Recognizes The Serca2b Calcium Pump In Pig Brain
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Hypothalamic Hypophyseal Inhibitory Factor (Hhif) Increases Intrasynaptosomal Free Calcium Concentration
Structural Changes Of The Sarcoplasmic Reticulum Ca(Ii)-Atpase Nucleotide Binding Domain By Ph And La(Iii)
Hypothalamic Hypophyseal Inhibitory Factor (Hhif) Increases Intrasynaptosomal Free Calcium Concentration
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
Hypothalamic Hypophyseal Inhibitory Factor (Hhif) Increases Intrasynaptosomal Free Calcium Concentration
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
A Comparative Kinetic Analysis Of The Flavin-Photosensitized Oxidation And Reduction Of Plastocyanin And Cytochrome C6from Different Organisms
Ca2+ Uptake Coupled To Glycogen Phosphorolysis In The Glycogenolytic-Sarcoplasmic Reticulum Complex From Rat Skeletal Muscle
A Comparative Kinetic Analysis Of The Flavin-Photosensitized Oxidation And Reduction Of Plastocyanin And Cytochrome C6from Different Organisms
Physical State Of Bulk And Protein-Associated Lipid In Nicotinic Acetylcholine Receptor-Rich Membrane Studied By Laurdan Generalized Polarization And Fluorescence Energy Transfer
A Comparative Kinetic Analysis Of The Flavin-Photosensitized Oxidation And Reduction Of Plastocyanin And Cytochrome C6from Different Organisms
Physical State Of Bulk And Protein-Associated Lipid In Nicotinic Acetylcholine Receptor-Rich Membrane Studied By Laurdan Generalized Polarization And Fluorescence Energy Transfer
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
Physical State Of Bulk And Protein-Associated Lipid In Nicotinic Acetylcholine Receptor-Rich Membrane Studied By Laurdan Generalized Polarization And Fluorescence Energy Transfer
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
Purification Of The Synaptosomal Plasma Membrane (Ca2+ + Mg2+)-Atpase From Pig Brain
Involvement Of Free Radicals In Signalling Of Low-Potassium Induced Apoptosis In Cultured Cerebellar Granule Cells.
Purification Of The Synaptosomal Plasma Membrane (Ca2+ + Mg2+)-Atpase From Pig Brain
Correction: Purification Of The Synaptosomal Plasma Membrane (Ca2+ + Mg2+)-Atpase From Pig Brain (Biochemical Journal (1996) 315 (183-187))
Purification Of The Synaptosomal Plasma Membrane (Ca2+ + Mg2+)-Atpase From Pig Brain
Correction: Purification Of The Synaptosomal Plasma Membrane (Ca2+ + Mg2+)-Atpase From Pig Brain (Biochemical Journal (1996) 315 (183-187))
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Correction: Purification Of The Synaptosomal Plasma Membrane (Ca2+ + Mg2+)-Atpase From Pig Brain (Biochemical Journal (1996) 315 (183-187))
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Rate Of Na+/ca2+ Exchange Across The Plasma Membrane Of Synaptosomes Measured Using The Fluorescence Of Chlorotetracycline. Implications To Calcium Homeostasis In Synaptic Terminals
Inactivation Of Ecto-Atpase Activity Of Rat Brain Synaptosomes
Rate Of Na+/ca2+ Exchange Across The Plasma Membrane Of Synaptosomes Measured Using The Fluorescence Of Chlorotetracycline. Implications To Calcium Homeostasis In Synaptic Terminals
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Rate Of Na+/ca2+ Exchange Across The Plasma Membrane Of Synaptosomes Measured Using The Fluorescence Of Chlorotetracycline. Implications To Calcium Homeostasis In Synaptic Terminals
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Fluorescnece Anisotropy Of Fluorescein Phosphatidylethanolthiocarbamide In Lipid Bilayers And In Ca2+-Atpase/lipid Reconstituted Systems
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Preferential Distribution Of The Fluorescent Phospholipid Probes Nbd-Phosphatidylcholine And Rhodamine-Phosphatidylethanolamine In The Exofacial Leaflet Of Acetylcholine Receptor-Rich Membranes From Torpedo Marmorata
Interaction Between Glycogen Phosphorylase And Sarcoplasmic Reticulum Membranes And Its Functional Implications
Preferential Distribution Of The Fluorescent Phospholipid Probes Nbd-Phosphatidylcholine And Rhodamine-Phosphatidylethanolamine In The Exofacial Leaflet Of Acetylcholine Receptor-Rich Membranes From Torpedo Marmorata
The Modulation Of Ca2+ Binding To Sarcoplasmic Reticulum Atpase By Atp Analogues Is Ph-Dependent
Preferential Distribution Of The Fluorescent Phospholipid Probes Nbd-Phosphatidylcholine And Rhodamine-Phosphatidylethanolamine In The Exofacial Leaflet Of Acetylcholine Receptor-Rich Membranes From Torpedo Marmorata
The Modulation Of Ca2+ Binding To Sarcoplasmic Reticulum Atpase By Atp Analogues Is Ph-Dependent
Identification Of Two Types Of Ca2+ Transport Atpases In Pig Brain By Specific Antibodies
The Modulation Of Ca2+ Binding To Sarcoplasmic Reticulum Atpase By Atp Analogues Is Ph-Dependent
Identification Of Two Types Of Ca2+ Transport Atpases In Pig Brain By Specific Antibodies
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Identification Of Two Types Of Ca2+ Transport Atpases In Pig Brain By Specific Antibodies
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Intrasynaptosomal Free Mg2+ Concentration Measured With The Fluorescent Indicator Mag-Fura-2: Modulation By Na+ Gradient And By Extrasynaptosomal Atp
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Modulation Of The Ca2+ Pump By The Hypothalamic-Hypophysary Inhibitory Factor
Mg2+-Adp Protects Against Inactivation Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase By N-Cyclohexyl-N′-(4-Dimethylamino-Α-Naphthyl) Carbodiimide
Modulation Of The Ca2+ Pump By The Hypothalamic-Hypophysary Inhibitory Factor
Modulation Of The Ca 2+,mg2+-Atpase Of Sarcoplasmic Reticulum By The Hypothalamic Hypophyseal Inhibitory Factor
Modulation Of The Ca2+ Pump By The Hypothalamic-Hypophysary Inhibitory Factor
Modulation Of The Ca 2+,mg2+-Atpase Of Sarcoplasmic Reticulum By The Hypothalamic Hypophyseal Inhibitory Factor
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Modulation Of The Ca 2+,mg2+-Atpase Of Sarcoplasmic Reticulum By The Hypothalamic Hypophyseal Inhibitory Factor
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Thermal Unfolding Of Monomeric Ca(Ii),mg(Ii)-Atpase From Sarcoplasmic Reticulum Of Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Fluorescence Energy Transfer As A Tool To Locate Functional Sites In Membrane Proteins
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Differential Scanning Calorimetry Study Of The Thermal Unfolding Of Sarcoplasmic Reticulum Ca2+, Mg2+-Atpase From Rabbit Skeletal Muscle
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Seasonal Variation Of Exudate Of Cistus Ladanifer
Quantification And Removal Of Glycogen Phosphorylase And Other Enzymes Associated With Sarcoplasmic Reticulum Membrane Preparations
Seasonal Variation Of Exudate Of Cistus Ladanifer
Glycogen Phosphorolysis Can Form A Metabolic Shuttle To Support Ca2+ Uptake By Sarcoplasmic Reticulum Membranes In Skeletal Muscle
Seasonal Variation Of Exudate Of Cistus Ladanifer
Glycogen Phosphorolysis Can Form A Metabolic Shuttle To Support Ca2+ Uptake By Sarcoplasmic Reticulum Membranes In Skeletal Muscle
Advanced Course On Structure-Function Relationships Of Membrane Proteins: An Assessment Of Current Knowledge
Glycogen Phosphorolysis Can Form A Metabolic Shuttle To Support Ca2+ Uptake By Sarcoplasmic Reticulum Membranes In Skeletal Muscle
Advanced Course On Structure-Function Relationships Of Membrane Proteins: An Assessment Of Current Knowledge
Evidence For The Lumenal Location Of The 53 Kda Glycoprotein Of Sarcoplasmic Reticulum
Advanced Course On Structure-Function Relationships Of Membrane Proteins: An Assessment Of Current Knowledge
Evidence For The Lumenal Location Of The 53 Kda Glycoprotein Of Sarcoplasmic Reticulum
Reactivity Of Lysyl Residues On The (Ca2+-Mg2+)-Atpase To 7-Amino-4-Methylcoumarin-3-Acetic Acid Succinimidyl Ester
Evidence For The Lumenal Location Of The 53 Kda Glycoprotein Of Sarcoplasmic Reticulum
Reactivity Of Lysyl Residues On The (Ca2+-Mg2+)-Atpase To 7-Amino-4-Methylcoumarin-3-Acetic Acid Succinimidyl Ester
Labeling The (Ca2+-Mg2+)-Atpase Of Sarcoplasmic Reticulum At Glu-439 With 5-(Bromomethyl)fluorescein
Reactivity Of Lysyl Residues On The (Ca2+-Mg2+)-Atpase To 7-Amino-4-Methylcoumarin-3-Acetic Acid Succinimidyl Ester
Labeling The (Ca2+-Mg2+)-Atpase Of Sarcoplasmic Reticulum At Glu-439 With 5-(Bromomethyl)fluorescein
Localization Of Cys-344 On The (Ca2+ -Mg2+)-Atpase Of Sarcoplasmic Reticulum Using Resonance Energy Transfer
Labeling The (Ca2+-Mg2+)-Atpase Of Sarcoplasmic Reticulum At Glu-439 With 5-(Bromomethyl)fluorescein
Localization Of Cys-344 On The (Ca2+ -Mg2+)-Atpase Of Sarcoplasmic Reticulum Using Resonance Energy Transfer
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Localization Of Cys-344 On The (Ca2+ -Mg2+)-Atpase Of Sarcoplasmic Reticulum Using Resonance Energy Transfer
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Properties Of The Purified Hypothalamic Pituitary Na/k-Atpase Inhibitor
Effect Of Immobilization On The Activity Of Rat Hepatic Microsomal Cytochrome P450 Enzymes
Properties Of The Purified Hypothalamic Pituitary Na/k-Atpase Inhibitor
Location Of Functional Centers In The Microsomal Cytochrome P450 System
Properties Of The Purified Hypothalamic Pituitary Na/k-Atpase Inhibitor
Location Of Functional Centers In The Microsomal Cytochrome P450 System
Definition Of Surface-Exposed And Trans-Membranous Regions Of The (Ca2+-Mg2+)-Atpase Of Sarcoplasmic Reticulum Using Anti-Peptide Antibodies
Location Of Functional Centers In The Microsomal Cytochrome P450 System
Definition Of Surface-Exposed And Trans-Membranous Regions Of The (Ca2+-Mg2+)-Atpase Of Sarcoplasmic Reticulum Using Anti-Peptide Antibodies
Studies Of The Structure And Function Of Sarcoplasmic Reticulum (Ca2+-Mg2+)-Atpase Using Immunological Approaches
Definition Of Surface-Exposed And Trans-Membranous Regions Of The (Ca2+-Mg2+)-Atpase Of Sarcoplasmic Reticulum Using Anti-Peptide Antibodies
Studies Of The Structure And Function Of Sarcoplasmic Reticulum (Ca2+-Mg2+)-Atpase Using Immunological Approaches
An Examination Of The Transmembranous Organisation Of The Sarcoplasmic Reticulum (Ca2+-Mg2+)-Atpase Using Antipeptide Antibodies
Studies Of The Structure And Function Of Sarcoplasmic Reticulum (Ca2+-Mg2+)-Atpase Using Immunological Approaches
An Examination Of The Transmembranous Organisation Of The Sarcoplasmic Reticulum (Ca2+-Mg2+)-Atpase Using Antipeptide Antibodies
Definition Of An Epitope At The Nucleotide Binding Site Of The (Ca2+-Mg2+)-Atpase Of Sarcoplasmic Reticulum By Fluorescein Labelling
An Examination Of The Transmembranous Organisation Of The Sarcoplasmic Reticulum (Ca2+-Mg2+)-Atpase Using Antipeptide Antibodies
Definition Of An Epitope At The Nucleotide Binding Site Of The (Ca2+-Mg2+)-Atpase Of Sarcoplasmic Reticulum By Fluorescein Labelling
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Definition Of An Epitope At The Nucleotide Binding Site Of The (Ca2+-Mg2+)-Atpase Of Sarcoplasmic Reticulum By Fluorescein Labelling
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Differential Scanning Calorimetry Study Of Glycogen Phosphorylase B-Detergent Interactions
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Modulation By Phosphorylation Of Glycogen Phosphorylase-Sarcoplasmic Reticulum Interaction
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Modulation By Phosphorylation Of Glycogen Phosphorylase-Sarcoplasmic Reticulum Interaction
Caracterización Por Técnicas Espectrofotométricas Del Ultravioleta Y Visible Edel Exudado De "cistus Ladanifer L." Como Respuesta Al Estrés Estival
Modulation By Phosphorylation Of Glycogen Phosphorylase-Sarcoplasmic Reticulum Interaction
Caracterización Por Técnicas Espectrofotométricas Del Ultravioleta Y Visible Edel Exudado De "cistus Ladanifer L." Como Respuesta Al Estrés Estival
Chemical Crosslinking And Enzyme Kinetics Provide No Evidence For A Regulatory Role For The 53 Kda Glycoprotein Of Sarcoplasmic Reticulum In Calcium Transport
Caracterización Por Técnicas Espectrofotométricas Del Ultravioleta Y Visible Edel Exudado De "cistus Ladanifer L." Como Respuesta Al Estrés Estival
Chemical Crosslinking And Enzyme Kinetics Provide No Evidence For A Regulatory Role For The 53 Kda Glycoprotein Of Sarcoplasmic Reticulum In Calcium Transport
Mapping Epitopes On The (Ca2+mg2+)-Atpase Of Sarcoplasmic Reticulum Using Fusion Proteins
Chemical Crosslinking And Enzyme Kinetics Provide No Evidence For A Regulatory Role For The 53 Kda Glycoprotein Of Sarcoplasmic Reticulum In Calcium Transport
Mapping Epitopes On The (Ca2+mg2+)-Atpase Of Sarcoplasmic Reticulum Using Fusion Proteins
The Effect Of A Monoclonal Antibody On Specific Steps Of The Reaction Sequence Of The (Ca2+-Mg2+)-Atpase From Sarcoplasmic Reticulum
Mapping Epitopes On The (Ca2+mg2+)-Atpase Of Sarcoplasmic Reticulum Using Fusion Proteins
The Effect Of A Monoclonal Antibody On Specific Steps Of The Reaction Sequence Of The (Ca2+-Mg2+)-Atpase From Sarcoplasmic Reticulum
Hemin And Hemeprotein Bleaching During Linoleic Acid Oxidation By Lipoxygenases
The Effect Of A Monoclonal Antibody On Specific Steps Of The Reaction Sequence Of The (Ca2+-Mg2+)-Atpase From Sarcoplasmic Reticulum
Hemin And Hemeprotein Bleaching During Linoleic Acid Oxidation By Lipoxygenases
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Hemin And Hemeprotein Bleaching During Linoleic Acid Oxidation By Lipoxygenases
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Effect Of The Detergent C12e8 On The Binding Of Monoclonal Antibodies To The (Ca2+-Mg2+)-Atpase Of Rabbit Skeletal Sarcoplasmic Reticulum
Kinetic Characterization Of The Normal And Procaine‐perturbed Reaction Cycles Of The Sarcoplasmic Reticulum Calcium Pump
Effect Of The Detergent C12e8 On The Binding Of Monoclonal Antibodies To The (Ca2+-Mg2+)-Atpase Of Rabbit Skeletal Sarcoplasmic Reticulum
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Effect Of The Detergent C12e8 On The Binding Of Monoclonal Antibodies To The (Ca2+-Mg2+)-Atpase Of Rabbit Skeletal Sarcoplasmic Reticulum
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Modulation Of Calcium Fluxes Across Synaptosomal Plasma Membrane By Local Anesthetics
Modulation Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+)-Atpase By Pentobarbital
Modulation Of Calcium Fluxes Across Synaptosomal Plasma Membrane By Local Anesthetics
Modulation Of The Ca2+,mg2+‐atpase Activity Of Synaptosomal Plasma Membrane By The Local Anesthetics Dibucaine And Lidocaine
Modulation Of Calcium Fluxes Across Synaptosomal Plasma Membrane By Local Anesthetics
Modulation Of The Ca2+,mg2+‐atpase Activity Of Synaptosomal Plasma Membrane By The Local Anesthetics Dibucaine And Lidocaine
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Modulation Of The Ca2+,mg2+‐atpase Activity Of Synaptosomal Plasma Membrane By The Local Anesthetics Dibucaine And Lidocaine
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Distribution Of Two Distinct Ca2+ -Atpase-Like Proteins And Their Relationships To The Agonist-Sensitive Calcium Store In Adrenal Chromaffin Cells
Distances Between Functional Sites Of The Ca2++ Mg2+‐atpase From Sarcoplasmic Reticulum Using Co2+ As A Spectroscopic Ruler
Distribution Of Two Distinct Ca2+ -Atpase-Like Proteins And Their Relationships To The Agonist-Sensitive Calcium Store In Adrenal Chromaffin Cells
Interaction Of The Local Anesthetics Dibucaine And Tetracaine With Sarcoplasmic Reticulum Membranes. Differential Scanning Calorimetry And Fluorescence Studies
Distribution Of Two Distinct Ca2+ -Atpase-Like Proteins And Their Relationships To The Agonist-Sensitive Calcium Store In Adrenal Chromaffin Cells
Interaction Of The Local Anesthetics Dibucaine And Tetracaine With Sarcoplasmic Reticulum Membranes. Differential Scanning Calorimetry And Fluorescence Studies
Effects Of Atpase Activity Of Monoclonal Antibodies Raised Against (Ca2+ + Mg2+)-Atpase From Rabbit Skeletal Muscle Sarcoplasmic Reticulum And Their Correlation With Epitope Location
Interaction Of The Local Anesthetics Dibucaine And Tetracaine With Sarcoplasmic Reticulum Membranes. Differential Scanning Calorimetry And Fluorescence Studies
Effects Of Atpase Activity Of Monoclonal Antibodies Raised Against (Ca2+ + Mg2+)-Atpase From Rabbit Skeletal Muscle Sarcoplasmic Reticulum And Their Correlation With Epitope Location
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Effects Of Atpase Activity Of Monoclonal Antibodies Raised Against (Ca2+ + Mg2+)-Atpase From Rabbit Skeletal Muscle Sarcoplasmic Reticulum And Their Correlation With Epitope Location
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Modulation Of (Ca2+ + Mg2+)-Atpases And Ca2+ Fluxes Through The Plasma Membrane Of Synaptosomes And Sarcoplasmic Reticulum By Local Anaesthetics
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Probing The Nucleotide-Binding Site Of Sarcoplasmic Reticulum (Ca2+-Mg2+)-Atpase With Anti-Fluorescein Antibodies
Inhibition Of The Sarcoplasmic Reticulum (Ca2+ + Mg2+-Atpase By Zn(Ii)
Probing The Nucleotide-Binding Site Of Sarcoplasmic Reticulum (Ca2+-Mg2+)-Atpase With Anti-Fluorescein Antibodies
Evidence For The Cytoplasmic Location Of The N- And C-Terminal Segments Of Sarcoplasmic Reticulum (Ca2+mg2+)-Atpase
Probing The Nucleotide-Binding Site Of Sarcoplasmic Reticulum (Ca2+-Mg2+)-Atpase With Anti-Fluorescein Antibodies
Evidence For The Cytoplasmic Location Of The N- And C-Terminal Segments Of Sarcoplasmic Reticulum (Ca2+mg2+)-Atpase
Local Anesthetic-Divalent Cation Binding Center Interaction
Evidence For The Cytoplasmic Location Of The N- And C-Terminal Segments Of Sarcoplasmic Reticulum (Ca2+mg2+)-Atpase
Local Anesthetic-Divalent Cation Binding Center Interaction
Probing The Nucleotide Binding Site Of Sarcoplasmic Reticulum (Ca2+-Mg2)-Atpase With Anti-Fluorescein Antibodies
Local Anesthetic-Divalent Cation Binding Center Interaction
Probing The Nucleotide Binding Site Of Sarcoplasmic Reticulum (Ca2+-Mg2)-Atpase With Anti-Fluorescein Antibodies
Use Of Antibodies To Detect Chemically Cross-Linked Products From Sarcoplasmic Reticulum
Probing The Nucleotide Binding Site Of Sarcoplasmic Reticulum (Ca2+-Mg2)-Atpase With Anti-Fluorescein Antibodies
Use Of Antibodies To Detect Chemically Cross-Linked Products From Sarcoplasmic Reticulum
Comparative Fluorescence Properties Of Lipoxygenases
Use Of Antibodies To Detect Chemically Cross-Linked Products From Sarcoplasmic Reticulum
Comparative Fluorescence Properties Of Lipoxygenases
Effect Of Monoclonal Antibodies Raised Against Ca2+,mg2+-Atpase From Rabbit Skeletal Muscle Sarcoplasmic Reticulum On Atpase Activity And Its Correlation With Epitope Location
Comparative Fluorescence Properties Of Lipoxygenases
Effect Of Monoclonal Antibodies Raised Against Ca2+,mg2+-Atpase From Rabbit Skeletal Muscle Sarcoplasmic Reticulum On Atpase Activity And Its Correlation With Epitope Location
Effects Of Local Anesthetics On The Passive Permeability Of Sarcoplasmic Reticulum Vesicles To Ca2+ And Mg2+
Effect Of Monoclonal Antibodies Raised Against Ca2+,mg2+-Atpase From Rabbit Skeletal Muscle Sarcoplasmic Reticulum On Atpase Activity And Its Correlation With Epitope Location
Effects Of Local Anesthetics On The Passive Permeability Of Sarcoplasmic Reticulum Vesicles To Ca2+ And Mg2+
The Position Of The Atp Binding Site On The (Ca2+ + Mg2+)-Atpase
Effects Of Local Anesthetics On The Passive Permeability Of Sarcoplasmic Reticulum Vesicles To Ca2+ And Mg2+
The Position Of The Atp Binding Site On The (Ca2+ + Mg2+)-Atpase
Long-Wavelength Fluorescence Of Tyrosine And Tryptophan Solutions.
The Position Of The Atp Binding Site On The (Ca2+ + Mg2+)-Atpase
Long-Wavelength Fluorescence Of Tyrosine And Tryptophan Solutions.
Local Anesthetics Inhibit The Ca2+,mg2+‐atpase Activity Of Rat Brain Synaptosomes
Long-Wavelength Fluorescence Of Tyrosine And Tryptophan Solutions.
Local Anesthetics Inhibit The Ca2+,mg2+‐atpase Activity Of Rat Brain Synaptosomes
Dependence Of The Fluorescence Of Fluorescein Labelled (Ca2+, Mg2+)-Atpase Upon The Lipid To Protein Ratio In Sarcoplasmic Reticulum Reconstituted Systems
Local Anesthetics Inhibit The Ca2+,mg2+‐atpase Activity Of Rat Brain Synaptosomes
Dependence Of The Fluorescence Of Fluorescein Labelled (Ca2+, Mg2+)-Atpase Upon The Lipid To Protein Ratio In Sarcoplasmic Reticulum Reconstituted Systems
Redox Interconversion Of Escherichia Coli Glutathione Reductase. A Study With Permeabilized And Intact Cells
Dependence Of The Fluorescence Of Fluorescein Labelled (Ca2+, Mg2+)-Atpase Upon The Lipid To Protein Ratio In Sarcoplasmic Reticulum Reconstituted Systems
Redox Interconversion Of Escherichia Coli Glutathione Reductase. A Study With Permeabilized And Intact Cells
Redox Interconversion Of Glutathione Reductase From Escherichia Coli. A Study With Pure Enzyme And Cell-Free Extracts
Redox Interconversion Of Escherichia Coli Glutathione Reductase. A Study With Permeabilized And Intact Cells
Redox Interconversion Of Glutathione Reductase From Escherichia Coli. A Study With Pure Enzyme And Cell-Free Extracts
The Redox Interconversion Mechanism Of Saccahromyces Cerevisiae Glutathione Reductase
Redox Interconversion Of Glutathione Reductase From Escherichia Coli. A Study With Pure Enzyme And Cell-Free Extracts
The Redox Interconversion Mechanism Of Saccahromyces Cerevisiae Glutathione Reductase
Purification By Affinity Chromatography Of Glutathione Reductase (Ec 1.6.4.2) From Escherichia Coli And Characterization Of Such Enzyme
The Redox Interconversion Mechanism Of Saccahromyces Cerevisiae Glutathione Reductase
Purification By Affinity Chromatography Of Glutathione Reductase (Ec 1.6.4.2) From Escherichia Coli And Characterization Of Such Enzyme
Reversible Inactivation Of Saccharomyces Cerevisiae Glutathione Reductase Under Reducing Conditions
Purification By Affinity Chromatography Of Glutathione Reductase (Ec 1.6.4.2) From Escherichia Coli And Characterization Of Such Enzyme
Reversible Inactivation Of Saccharomyces Cerevisiae Glutathione Reductase Under Reducing Conditions
On The Rate Of Association And Dissociation Processes Of Intrinsic Membrane Proteins
Reversible Inactivation Of Saccharomyces Cerevisiae Glutathione Reductase Under Reducing Conditions
On The Rate Of Association And Dissociation Processes Of Intrinsic Membrane Proteins
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. Ii. Protein Distribution And Aggregation State In Biological Membranes
On The Rate Of Association And Dissociation Processes Of Intrinsic Membrane Proteins
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. Ii. Protein Distribution And Aggregation State In Biological Membranes
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. I. Lateral Phase Separation In Unilamellar Vesicles Formed By Binary Phospholipid Mixtures
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. Ii. Protein Distribution And Aggregation State In Biological Membranes
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. I. Lateral Phase Separation In Unilamellar Vesicles Formed By Binary Phospholipid Mixtures
Relationship Between The Activity Of Pancreatic Phospholipase A2 And The Physical State Of The Phospholipid Substrate.
Quantitation Of The Förster Energy Transfer For Two-Dimensional Systems. I. Lateral Phase Separation In Unilamellar Vesicles Formed By Binary Phospholipid Mixtures
Relationship Between The Activity Of Pancreatic Phospholipase A2 And The Physical State Of The Phospholipid Substrate.
Regulation Of Phosphorylase B By Amp: The Effects Of Enzyme Concentration
Relationship Between The Activity Of Pancreatic Phospholipase A2 And The Physical State Of The Phospholipid Substrate.
Regulation Of Phosphorylase B By Amp: The Effects Of Enzyme Concentration
Kaempferol Protects Against Amyloid Β Overproduction And The Rise Of Phospho-Tau 217 And Phospho-Tau 181 In The Rat Cerebellum Induced By Acute 3-Nitropropionic Acid Administration
WEB ACCESIBLE