PUBLICACIÓN

Resistance to TGF-beta-induced apoptosis in regenerating hepatocytes.

ACCEDER A LA PUBLICACIÓN: Orcid

Herrera B, Alvarez AM, Beltrán J, Valdés F, Fabregat I, Fernández M

2004

DOI

10.1002/jcp.20078

BIBTEX

@article{PMID:15389556, title = {Resistance to TGF-beta-induced apoptosis in regenerating hepatocytes.}, author = {Herrera B and Alvarez AM and Beltrán J and Valdés F and Fabregat I and Fernández M}, doi = {10.1002/jcp.20078}, journal = {Journal of cellular physiology}, month = {12}, year = {2004} }

AUTORES DE LA UEX

Alberto Manuel Álvarez Barrientos

OTRAS PUBLICACIONES DE INTERÉS

Not Available
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Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
Immunophenotype Profile By Flow Cytometry Reveals Different Subtypes Of Extracellular Vesicles In Porcine Seminal Plasma
Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
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Stim1 Translocation To The Nucleus Protects Cells From Dna Damage
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The Proteome Of Large Or Small Extracellular Vesicles In Pig Seminal Plasma Differs, Defining Sources And Biological Functions
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Transcriptome Analysis Reveals That Fertilization With Cryopreserved Sperm Downregulates Genes Relevant For Early Embryo Development In The Horse
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Extracellular Vesicles Isolated From Porcine Seminal Plasma Exhibit Different Tetraspanin Expression Profiles
Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science Doi: 10.1242/jcs.02878)
Erratum: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains (Journal Of Cell Science (2002) 115 (3119-3130) Doi: 10.1242/jcs.209973)
Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science Doi: 10.1242/jcs.02878)
Erratum: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains (Journal Of Cell Science (2002) 115 (3119-3130) Doi: 10.1242/jcs.209973)
Correction: Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels (Doi:10.1242/jcs.01002).
Erratum: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains (Journal Of Cell Science (2002) 115 (3119-3130) Doi: 10.1242/jcs.209973)
Correction: Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels (Doi:10.1242/jcs.01002).
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Correction: Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels (Doi:10.1242/jcs.01002).
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Variation In Malaria Infection And Immune Defence In Invasive And Endemic House Sparrows
Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry
Variation In Malaria Infection And Immune Defence In Invasive And Endemic House Sparrows
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
Variation In Malaria Infection And Immune Defence In Invasive And Endemic House Sparrows
Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling
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Immune-Neuroendocrine Dysregulation In Patients With Osteoarthritis: A Revision And A Pilot Study
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Immune-Neuroendocrine Dysregulation In Patients With Osteoarthritis: A Revision And A Pilot Study
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Immune-Neuroendocrine Dysregulation In Patients With Osteoarthritis: A Revision And A Pilot Study
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Expression Of Concern: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways. J. Cell Sci. Doi: 10.1242/jcs.02878
Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor
Expression Of Concern: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways. J. Cell Sci. Doi: 10.1242/jcs.02878
Expression Of Concern: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains. J. Cell Sci. (2002). 115, 3119-3130.
Expression Of Concern: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways. J. Cell Sci. Doi: 10.1242/jcs.02878
Expression Of Concern: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains. J. Cell Sci. (2002). 115, 3119-3130.
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
Expression Of Concern: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains. J. Cell Sci. (2002). 115, 3119-3130.
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells
Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells
Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells
Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg
Erratum To Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans [Fungal Genet. Biol. 74 (2015) 10-20] Doi: 10.1016/j.fgb.2014.10.016
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Erratum To Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans [Fungal Genet. Biol. 74 (2015) 10-20] Doi: 10.1016/j.fgb.2014.10.016
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Erratum To Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans [Fungal Genet. Biol. 74 (2015) 10-20] Doi: 10.1016/j.fgb.2014.10.016
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells.
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells.
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans.
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells.
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans.
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans.
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol.
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol.
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol.
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis
The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway
The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis
The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway
Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition
The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway
Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition
Effect Of High Pressure Thermal Processing On Some Essential Nutrients And Immunological Components Present In Breast Milk
Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition
Effect Of High Pressure Thermal Processing On Some Essential Nutrients And Immunological Components Present In Breast Milk
Cell Fusion Reprogramming Leads To A Specific Hepatic Expression Pattern During Mouse Bone Marrow Derived Hepatocyte Formation In Vivo
Effect Of High Pressure Thermal Processing On Some Essential Nutrients And Immunological Components Present In Breast Milk
Cell Fusion Reprogramming Leads To A Specific Hepatic Expression Pattern During Mouse Bone Marrow Derived Hepatocyte Formation In Vivo
270 Cd44+/epcam− Phenotype In Hepatocellular Carcinoma (Hcc) Cells Correlates With Increased Autocrine Tgf-Beta And Mesenchymal Traits
Cell Fusion Reprogramming Leads To A Specific Hepatic Expression Pattern During Mouse Bone Marrow Derived Hepatocyte Formation In Vivo
270 Cd44+/epcam− Phenotype In Hepatocellular Carcinoma (Hcc) Cells Correlates With Increased Autocrine Tgf-Beta And Mesenchymal Traits
Active Commuting To School And Cognitive Performance In Adolescents: The Avena Study.
270 Cd44+/epcam− Phenotype In Hepatocellular Carcinoma (Hcc) Cells Correlates With Increased Autocrine Tgf-Beta And Mesenchymal Traits
Active Commuting To School And Cognitive Performance In Adolescents: The Avena Study.
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Active Commuting To School And Cognitive Performance In Adolescents: The Avena Study.
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Comparative Analysis Of Carmellose 0.5% Versus Hyaluronate 0.15% In Dry Eye: A Flow Cytometric Study
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Comparative Analysis Of Carmellose 0.5% Versus Hyaluronate 0.15% In Dry Eye: A Flow Cytometric Study
The Effect Of Preservative-Free Hp-Guar On Dry Eye After Phacoemulsification: A Flow Cytometric Study.
Comparative Analysis Of Carmellose 0.5% Versus Hyaluronate 0.15% In Dry Eye: A Flow Cytometric Study
The Effect Of Preservative-Free Hp-Guar On Dry Eye After Phacoemulsification: A Flow Cytometric Study.
Haematological Reference Values In Spanish Adolescents: The Avena Study.
The Effect Of Preservative-Free Hp-Guar On Dry Eye After Phacoemulsification: A Flow Cytometric Study.
Haematological Reference Values In Spanish Adolescents: The Avena Study.
Isolation And Characterization Of A Putative Liver Progenitor Population After Treatment Of Fetal Rat Hepatocytes With Tgf-Beta
Haematological Reference Values In Spanish Adolescents: The Avena Study.
Isolation And Characterization Of A Putative Liver Progenitor Population After Treatment Of Fetal Rat Hepatocytes With Tgf-Beta
Ccaat/enhancer Binding Protein Β Deficiency Provides Cerebral Protection Following Excitotoxic Injury
Isolation And Characterization Of A Putative Liver Progenitor Population After Treatment Of Fetal Rat Hepatocytes With Tgf-Beta
Ccaat/enhancer Binding Protein Β Deficiency Provides Cerebral Protection Following Excitotoxic Injury
Deletion Of The Met Tyrosine Kinase In Liver Progenitor Oval Cells Increases Sensitivity To Apoptosis In Vitro
Ccaat/enhancer Binding Protein Β Deficiency Provides Cerebral Protection Following Excitotoxic Injury
Deletion Of The Met Tyrosine Kinase In Liver Progenitor Oval Cells Increases Sensitivity To Apoptosis In Vitro
Bcl6 Represses Nfκb Activity In Diffuse Large B-Cell Lymphomas
Deletion Of The Met Tyrosine Kinase In Liver Progenitor Oval Cells Increases Sensitivity To Apoptosis In Vitro
Bcl6 Represses Nfκb Activity In Diffuse Large B-Cell Lymphomas
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
Bcl6 Represses Nfκb Activity In Diffuse Large B-Cell Lymphomas
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
Methylmalonic Acidaemia Leads To Increased Production Of Reactive Oxygen Species And Induction Of Apoptosis Through The Mitochondrial/caspase Pathway
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
Methylmalonic Acidaemia Leads To Increased Production Of Reactive Oxygen Species And Induction Of Apoptosis Through The Mitochondrial/caspase Pathway
Nitric Oxide Down-Regulates Caveolin-3 Levels Through The Interaction With Myogenin, Its Transcription Factor
Methylmalonic Acidaemia Leads To Increased Production Of Reactive Oxygen Species And Induction Of Apoptosis Through The Mitochondrial/caspase Pathway
Nitric Oxide Down-Regulates Caveolin-3 Levels Through The Interaction With Myogenin, Its Transcription Factor
P38α Mapk Can Positively Or Negatively Regulate Rac-1 Activity Depending On The Presence Of Serum
Nitric Oxide Down-Regulates Caveolin-3 Levels Through The Interaction With Myogenin, Its Transcription Factor
P38α Mapk Can Positively Or Negatively Regulate Rac-1 Activity Depending On The Presence Of Serum
Binding Of Cap70 To Inducible Nitric Oxide Synthase And Implications For The Vectorial Release Of Nitric Oxide In Polarized Cells
P38α Mapk Can Positively Or Negatively Regulate Rac-1 Activity Depending On The Presence Of Serum
Binding Of Cap70 To Inducible Nitric Oxide Synthase And Implications For The Vectorial Release Of Nitric Oxide In Polarized Cells
Mitochondrial Superoxide Mediates Heat-Induced Apoptotic-Like Death In Leishmania Infantum
Binding Of Cap70 To Inducible Nitric Oxide Synthase And Implications For The Vectorial Release Of Nitric Oxide In Polarized Cells
Mitochondrial Superoxide Mediates Heat-Induced Apoptotic-Like Death In Leishmania Infantum
Tlr4-Mediated Survival Of Macrophages Is Myd88 Dependent And Requires Tnf-Α Autocrine Signalling
Mitochondrial Superoxide Mediates Heat-Induced Apoptotic-Like Death In Leishmania Infantum
Tlr4-Mediated Survival Of Macrophages Is Myd88 Dependent And Requires Tnf-Α Autocrine Signalling
Role Of Peroxynitrite In Endothelial Damage Mediated By Cyclosporine A
Tlr4-Mediated Survival Of Macrophages Is Myd88 Dependent And Requires Tnf-Α Autocrine Signalling
Role Of Peroxynitrite In Endothelial Damage Mediated By Cyclosporine A
C-Jun N-Terminal Protein Kinase Signalling Pathway Mediates Lovastatin-Induced Rat Brain Neuroblast Apoptosis
Role Of Peroxynitrite In Endothelial Damage Mediated By Cyclosporine A
C-Jun N-Terminal Protein Kinase Signalling Pathway Mediates Lovastatin-Induced Rat Brain Neuroblast Apoptosis
Lovastatin Inhibits The Extracellular-Signal-Regulated Kinase Pathway In Immortalized Rat Brain Neuroblasts
C-Jun N-Terminal Protein Kinase Signalling Pathway Mediates Lovastatin-Induced Rat Brain Neuroblast Apoptosis
Lovastatin Inhibits The Extracellular-Signal-Regulated Kinase Pathway In Immortalized Rat Brain Neuroblasts
Negative Regulation Of Akt Activity By P38α Map Kinase In Cardiomyocytes Involves Membrane Localization Of Pp2a Through Interaction With Caveolin-1
Lovastatin Inhibits The Extracellular-Signal-Regulated Kinase Pathway In Immortalized Rat Brain Neuroblasts
Negative Regulation Of Akt Activity By P38α Map Kinase In Cardiomyocytes Involves Membrane Localization Of Pp2a Through Interaction With Caveolin-1
Heat-Induced Programmed Cell Death In Leishmania Infantum Is Reverted By Bcl-Xl Expression
Negative Regulation Of Akt Activity By P38α Map Kinase In Cardiomyocytes Involves Membrane Localization Of Pp2a Through Interaction With Caveolin-1
Heat-Induced Programmed Cell Death In Leishmania Infantum Is Reverted By Bcl-Xl Expression
A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c
Heat-Induced Programmed Cell Death In Leishmania Infantum Is Reverted By Bcl-Xl Expression
A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c
Loss Of Apoptosis-Inducing Factor Leads To An Increase In Reactive Oxygen Species, And An Impairment Of Respiration That Can Be Reversed By Antioxidants [4]
A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c
Loss Of Apoptosis-Inducing Factor Leads To An Increase In Reactive Oxygen Species, And An Impairment Of Respiration That Can Be Reversed By Antioxidants [4]
Infiltration Of Inflammatory Cells Plays An Important Role In Matrix Metalloproteinase Expression And Activation In The Heart During Sepsis
Loss Of Apoptosis-Inducing Factor Leads To An Increase In Reactive Oxygen Species, And An Impairment Of Respiration That Can Be Reversed By Antioxidants [4]
Infiltration Of Inflammatory Cells Plays An Important Role In Matrix Metalloproteinase Expression And Activation In The Heart During Sepsis
Hematopoietic Mobilization In Mice Increases The Presence Of Bone Marrow-Derived Hepatocytes Via In Vivo Cell Fusion
Infiltration Of Inflammatory Cells Plays An Important Role In Matrix Metalloproteinase Expression And Activation In The Heart During Sepsis
Hematopoietic Mobilization In Mice Increases The Presence Of Bone Marrow-Derived Hepatocytes Via In Vivo Cell Fusion
Gastrin Induces Leukocyte-Endothelial Cell Interactions In Vivo And Contributes To The Inflammation Caused By Helicobacter Pylori
Hematopoietic Mobilization In Mice Increases The Presence Of Bone Marrow-Derived Hepatocytes Via In Vivo Cell Fusion
Gastrin Induces Leukocyte-Endothelial Cell Interactions In Vivo And Contributes To The Inflammation Caused By Helicobacter Pylori
Autocrine Production Of Tgf-Β Confers Resistance To Apoptosis After An Epithelial-Mesenchymal Transition Process In Hepatocytes: Role Of Egf Receptor Ligands
Gastrin Induces Leukocyte-Endothelial Cell Interactions In Vivo And Contributes To The Inflammation Caused By Helicobacter Pylori
Autocrine Production Of Tgf-Β Confers Resistance To Apoptosis After An Epithelial-Mesenchymal Transition Process In Hepatocytes: Role Of Egf Receptor Ligands
N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways
Autocrine Production Of Tgf-Β Confers Resistance To Apoptosis After An Epithelial-Mesenchymal Transition Process In Hepatocytes: Role Of Egf Receptor Ligands
N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways
Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science (2006) Vol. 119 (1558-1596))
N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways
Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science (2006) Vol. 119 (1558-1596))
Erratum: A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c (Genes And Development (2006) 20 (307-320))
Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science (2006) Vol. 119 (1558-1596))
Erratum: A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c (Genes And Development (2006) 20 (307-320))
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Erratum: A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c (Genes And Development (2006) 20 (307-320))
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Identification Of A Nuclear Export Signal In The Kshv Latent Protein Lana2 Mediating Its Export From The Nucleus
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Identification Of A Nuclear Export Signal In The Kshv Latent Protein Lana2 Mediating Its Export From The Nucleus
Discrepancies Between Nitroglycerin And No-Releasing Drugs On Mitochondrial Oxygen Consumption, Vasoactivity, And The Release Of No
Identification Of A Nuclear Export Signal In The Kshv Latent Protein Lana2 Mediating Its Export From The Nucleus
Discrepancies Between Nitroglycerin And No-Releasing Drugs On Mitochondrial Oxygen Consumption, Vasoactivity, And The Release Of No
Direct Interaction Between The Reductase Domain Of Endothelial Nitric Oxide Synthase And The Ryanodine Receptor
Discrepancies Between Nitroglycerin And No-Releasing Drugs On Mitochondrial Oxygen Consumption, Vasoactivity, And The Release Of No
Direct Interaction Between The Reductase Domain Of Endothelial Nitric Oxide Synthase And The Ryanodine Receptor
Pgc-1α Regulates The Mitochondrial Antioxidant Defense System In Vascular Endothelial Cells
Direct Interaction Between The Reductase Domain Of Endothelial Nitric Oxide Synthase And The Ryanodine Receptor
Pgc-1α Regulates The Mitochondrial Antioxidant Defense System In Vascular Endothelial Cells
Matrix Metalloproteinase 13 Mediates Nitric Oxide Activation Of Endothelial Cell Migration
Pgc-1α Regulates The Mitochondrial Antioxidant Defense System In Vascular Endothelial Cells
Matrix Metalloproteinase 13 Mediates Nitric Oxide Activation Of Endothelial Cell Migration
Synthesis Of Nitric Oxide In Postganglionic Myenteric Neurons During Endotoxemia: Implications For Gastric Motor Function In Rats.
Matrix Metalloproteinase 13 Mediates Nitric Oxide Activation Of Endothelial Cell Migration
Synthesis Of Nitric Oxide In Postganglionic Myenteric Neurons During Endotoxemia: Implications For Gastric Motor Function In Rats.
Source Of Early Reactive Oxygen Species In The Apoptosis Induced By Transforming Growth Factor-Β In Fetal Rat Hepatocytes
Synthesis Of Nitric Oxide In Postganglionic Myenteric Neurons During Endotoxemia: Implications For Gastric Motor Function In Rats.
Source Of Early Reactive Oxygen Species In The Apoptosis Induced By Transforming Growth Factor-Β In Fetal Rat Hepatocytes
Resistance To Tgf-Beta-Induced Apoptosis In Regenerating Hepatocytes.
Source Of Early Reactive Oxygen Species In The Apoptosis Induced By Transforming Growth Factor-Β In Fetal Rat Hepatocytes
Resistance To Tgf-Beta-Induced Apoptosis In Regenerating Hepatocytes.
Palmitoylation Of Inducible Nitric-Oxide Synthase At Cys-3 Is Required For Proper Intracellular Traffic And Nitric Oxide Synthesis.
Resistance To Tgf-Beta-Induced Apoptosis In Regenerating Hepatocytes.
Palmitoylation Of Inducible Nitric-Oxide Synthase At Cys-3 Is Required For Proper Intracellular Traffic And Nitric Oxide Synthesis.
The Effect Of Milk Fermented By Yogurt Cultures Plus Lactobacillus Casei Dn-114001 On The Immune Response Of Subjects Under Academic Examination Stress.
Palmitoylation Of Inducible Nitric-Oxide Synthase At Cys-3 Is Required For Proper Intracellular Traffic And Nitric Oxide Synthesis.
The Effect Of Milk Fermented By Yogurt Cultures Plus Lactobacillus Casei Dn-114001 On The Immune Response Of Subjects Under Academic Examination Stress.
Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels.
The Effect Of Milk Fermented By Yogurt Cultures Plus Lactobacillus Casei Dn-114001 On The Immune Response Of Subjects Under Academic Examination Stress.
Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels.
P38 Alpha Mitogen-Activated Protein Kinase Sensitizes Cells To Apoptosis Induced By Different Stimuli.
Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels.
P38 Alpha Mitogen-Activated Protein Kinase Sensitizes Cells To Apoptosis Induced By Different Stimuli.
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
P38 Alpha Mitogen-Activated Protein Kinase Sensitizes Cells To Apoptosis Induced By Different Stimuli.
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Confocal Visualization Of Nitric Oxide In The Myenteric Plexus Of The Gastric Fundus Obtained From Endotoxin-Treated Rats
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Confocal Visualization Of Nitric Oxide In The Myenteric Plexus Of The Gastric Fundus Obtained From Endotoxin-Treated Rats
Long-Term Treatment With Insulin Induces Apoptosis In Brown Adipocytes: Role Of Oxidative Stress.
Confocal Visualization Of Nitric Oxide In The Myenteric Plexus Of The Gastric Fundus Obtained From Endotoxin-Treated Rats
Long-Term Treatment With Insulin Induces Apoptosis In Brown Adipocytes: Role Of Oxidative Stress.
Sustained Nitric Oxide Delivery Delays Nitric Oxide-Dependent Apoptosis In Macrophages: Contribution To The Physiological Function Of Activated Macrophages.
Long-Term Treatment With Insulin Induces Apoptosis In Brown Adipocytes: Role Of Oxidative Stress.
Sustained Nitric Oxide Delivery Delays Nitric Oxide-Dependent Apoptosis In Macrophages: Contribution To The Physiological Function Of Activated Macrophages.
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Sustained Nitric Oxide Delivery Delays Nitric Oxide-Dependent Apoptosis In Macrophages: Contribution To The Physiological Function Of Activated Macrophages.
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Evidence For Discoordinate Regulation Of The Hla-Dpb1 Gene
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Evidence For Discoordinate Regulation Of The Hla-Dpb1 Gene
Tace/adam17-Tnf-Α Pathway In Rat Cortical Cultures After Exposure To Oxygen-Glucose Deprivation Or Glutamate
Evidence For Discoordinate Regulation Of The Hla-Dpb1 Gene
Tace/adam17-Tnf-Α Pathway In Rat Cortical Cultures After Exposure To Oxygen-Glucose Deprivation Or Glutamate
Aplidin Induces The Mitochondrial Apoptotic Pathway Via Oxidative Stress-Mediated Jnk And P38 Activation And Protein Kinase C Delta.
Tace/adam17-Tnf-Α Pathway In Rat Cortical Cultures After Exposure To Oxygen-Glucose Deprivation Or Glutamate
Aplidin Induces The Mitochondrial Apoptotic Pathway Via Oxidative Stress-Mediated Jnk And P38 Activation And Protein Kinase C Delta.
Differential Role Of Ppar Gamma In The Regulation Of Ucp-1 And Adipogenesis By Tnf-Alpha In Brown Adipocytes.
Aplidin Induces The Mitochondrial Apoptotic Pathway Via Oxidative Stress-Mediated Jnk And P38 Activation And Protein Kinase C Delta.
Differential Role Of Ppar Gamma In The Regulation Of Ucp-1 And Adipogenesis By Tnf-Alpha In Brown Adipocytes.
Induction Of Apoptosis By Nitric Oxide In Macrophages Is Independent Of Apoptotic Volume Decrease.
Differential Role Of Ppar Gamma In The Regulation Of Ucp-1 And Adipogenesis By Tnf-Alpha In Brown Adipocytes.
Induction Of Apoptosis By Nitric Oxide In Macrophages Is Independent Of Apoptotic Volume Decrease.
In Vitro Fertilizing Capacity And Chromatin Condensation Of Deep Frozen Boar Semen Packaged In 0.5 And 5 Ml Straws.
Induction Of Apoptosis By Nitric Oxide In Macrophages Is Independent Of Apoptotic Volume Decrease.
In Vitro Fertilizing Capacity And Chromatin Condensation Of Deep Frozen Boar Semen Packaged In 0.5 And 5 Ml Straws.
Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains
In Vitro Fertilizing Capacity And Chromatin Condensation Of Deep Frozen Boar Semen Packaged In 0.5 And 5 Ml Straws.
Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains
Akt1/pkbalpha Protects Pc12 Cells Against The Parkinsonism-Inducing Neurotoxin 1-Methyl-4-Phenylpyridinium And Reduces The Levels Of Oxygen-Free Radicals.
Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains
Akt1/pkbalpha Protects Pc12 Cells Against The Parkinsonism-Inducing Neurotoxin 1-Methyl-4-Phenylpyridinium And Reduces The Levels Of Oxygen-Free Radicals.
Use Of Flow Cytometry And Confocal Microscopy Techniques To Investigate Early Cdcl2-Induced Nephrotoxicity In Vitro
Akt1/pkbalpha Protects Pc12 Cells Against The Parkinsonism-Inducing Neurotoxin 1-Methyl-4-Phenylpyridinium And Reduces The Levels Of Oxygen-Free Radicals.
Use Of Flow Cytometry And Confocal Microscopy Techniques To Investigate Early Cdcl2-Induced Nephrotoxicity In Vitro
Lovastatin Induces Apoptosis Of Spontaneously Immortalized Rat Brain Neuroblasts: Involvement Of Nonsterol Isoprenoid Biosynthesis Inhibition
Use Of Flow Cytometry And Confocal Microscopy Techniques To Investigate Early Cdcl2-Induced Nephrotoxicity In Vitro
Lovastatin Induces Apoptosis Of Spontaneously Immortalized Rat Brain Neuroblasts: Involvement Of Nonsterol Isoprenoid Biosynthesis Inhibition
Intracellular Water Motion Decreases In Apoptotic Macrophages After Caspase Activation.
Lovastatin Induces Apoptosis Of Spontaneously Immortalized Rat Brain Neuroblasts: Involvement Of Nonsterol Isoprenoid Biosynthesis Inhibition
Intracellular Water Motion Decreases In Apoptotic Macrophages After Caspase Activation.
Activation Of Caspases Occurs Downstream From Radical Oxygen Species Production, Bcl-Xl Down-Regulation, And Early Cytochrome C Release In Apoptosis Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes.
Intracellular Water Motion Decreases In Apoptotic Macrophages After Caspase Activation.
Activation Of Caspases Occurs Downstream From Radical Oxygen Species Production, Bcl-Xl Down-Regulation, And Early Cytochrome C Release In Apoptosis Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes.
Reactive Oxygen Species (Ros) Mediates The Mitochondrial-Dependent Apoptosis Induced By Transforming Growth Factor (Beta) In Fetal Hepatocytes.
Activation Of Caspases Occurs Downstream From Radical Oxygen Species Production, Bcl-Xl Down-Regulation, And Early Cytochrome C Release In Apoptosis Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes.
Reactive Oxygen Species (Ros) Mediates The Mitochondrial-Dependent Apoptosis Induced By Transforming Growth Factor (Beta) In Fetal Hepatocytes.
Mitochondrial Involvement In Cocaine-Treated Rat Hepatocytes: Effect Of N-Acetylcysteine And Deferoxamine.
Reactive Oxygen Species (Ros) Mediates The Mitochondrial-Dependent Apoptosis Induced By Transforming Growth Factor (Beta) In Fetal Hepatocytes.
Mitochondrial Involvement In Cocaine-Treated Rat Hepatocytes: Effect Of N-Acetylcysteine And Deferoxamine.
Effect Of N-Acetylcysteine And Deferoxamine On Endogenous Antioxidant Defense System Gene Expression In A Rat Hepatocyte Model Of Cocaine Cytotoxicity.
Mitochondrial Involvement In Cocaine-Treated Rat Hepatocytes: Effect Of N-Acetylcysteine And Deferoxamine.
Effect Of N-Acetylcysteine And Deferoxamine On Endogenous Antioxidant Defense System Gene Expression In A Rat Hepatocyte Model Of Cocaine Cytotoxicity.
Fibronectin Regulates Morphology, Cell Organization And Gene Expression Of Rat Fetal Hepatocytes In Primary Culture.
Effect Of N-Acetylcysteine And Deferoxamine On Endogenous Antioxidant Defense System Gene Expression In A Rat Hepatocyte Model Of Cocaine Cytotoxicity.
Fibronectin Regulates Morphology, Cell Organization And Gene Expression Of Rat Fetal Hepatocytes In Primary Culture.
Inhibition Of Pkb/akt1 By C2-Ceramide Involves Activation Of Ceramide-Activated Protein Phosphatase In Pc12 Cells.
Fibronectin Regulates Morphology, Cell Organization And Gene Expression Of Rat Fetal Hepatocytes In Primary Culture.
Inhibition Of Pkb/akt1 By C2-Ceramide Involves Activation Of Ceramide-Activated Protein Phosphatase In Pc12 Cells.
P38 Mitogen-Activated Protein Kinase Mediates Tumor Necrosis Factor-Alpha-Induced Apoptosis In Rat Fetal Brown Adipocytes.
Inhibition Of Pkb/akt1 By C2-Ceramide Involves Activation Of Ceramide-Activated Protein Phosphatase In Pc12 Cells.
P38 Mitogen-Activated Protein Kinase Mediates Tumor Necrosis Factor-Alpha-Induced Apoptosis In Rat Fetal Brown Adipocytes.
Noradrenaline Induces Brown Adipocytes Cell Growth Via Beta-Receptors By A Mechanism Dependent On Erks But Independent Of Camp And Pka.
P38 Mitogen-Activated Protein Kinase Mediates Tumor Necrosis Factor-Alpha-Induced Apoptosis In Rat Fetal Brown Adipocytes.
Noradrenaline Induces Brown Adipocytes Cell Growth Via Beta-Receptors By A Mechanism Dependent On Erks But Independent Of Camp And Pka.
Epidermal Growth Factor Impairs The Cytochrome C/caspase-3 Apoptotic Pathway Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes Via A Phosphoinositide 3-Kinase-Dependent Pathway.
Noradrenaline Induces Brown Adipocytes Cell Growth Via Beta-Receptors By A Mechanism Dependent On Erks But Independent Of Camp And Pka.
Epidermal Growth Factor Impairs The Cytochrome C/caspase-3 Apoptotic Pathway Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes Via A Phosphoinositide 3-Kinase-Dependent Pathway.
Changes In Antioxidant Defence Systems Induced By Cyclosporine A In Cultures Of Hepatocytes From 2- And 12-Month-Old Rats.
Epidermal Growth Factor Impairs The Cytochrome C/caspase-3 Apoptotic Pathway Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes Via A Phosphoinositide 3-Kinase-Dependent Pathway.
Changes In Antioxidant Defence Systems Induced By Cyclosporine A In Cultures Of Hepatocytes From 2- And 12-Month-Old Rats.
Contribution Of Cyclopentenone Prostaglandins To The Resolution Of Inflammation Through The Potentiation Of Apoptosis In Activated Macrophages.
Changes In Antioxidant Defence Systems Induced By Cyclosporine A In Cultures Of Hepatocytes From 2- And 12-Month-Old Rats.
Contribution Of Cyclopentenone Prostaglandins To The Resolution Of Inflammation Through The Potentiation Of Apoptosis In Activated Macrophages.
Hsp70 Induction By Cyclosporine A In Cultured Rat Hepatocytes: Effect Of Vitamin E Succinate.
Contribution Of Cyclopentenone Prostaglandins To The Resolution Of Inflammation Through The Potentiation Of Apoptosis In Activated Macrophages.
Hsp70 Induction By Cyclosporine A In Cultured Rat Hepatocytes: Effect Of Vitamin E Succinate.
Applications Of Confocal Mycroscopy To The Paper Industry: Slime And Deposit Examples
Hsp70 Induction By Cyclosporine A In Cultured Rat Hepatocytes: Effect Of Vitamin E Succinate.
Applications Of Confocal Mycroscopy To The Paper Industry: Slime And Deposit Examples
Applications Of Flow Cytometry To Clinical Microbiology
Applications Of Confocal Mycroscopy To The Paper Industry: Slime And Deposit Examples
Applications Of Flow Cytometry To Clinical Microbiology
Age-Related Changes On Parameters Of Experimentally-Induced Liver Injury And Regeneration.
Applications Of Flow Cytometry To Clinical Microbiology
Age-Related Changes On Parameters Of Experimentally-Induced Liver Injury And Regeneration.
Apoptotic Response To Tgf-Beta In Fetal Hepatocytes Depends Upon Their State Of Differentiation.
Age-Related Changes On Parameters Of Experimentally-Induced Liver Injury And Regeneration.
Apoptotic Response To Tgf-Beta In Fetal Hepatocytes Depends Upon Their State Of Differentiation.
Nonviral Transfer Of Genes To Pig Primary Keratinocytes. Induction Of Angiogenesis By Composite Grafts Of Modified Keratinocytes Overexpressing Vegf Driven By A Keratin Promoter.
Apoptotic Response To Tgf-Beta In Fetal Hepatocytes Depends Upon Their State Of Differentiation.
Nonviral Transfer Of Genes To Pig Primary Keratinocytes. Induction Of Angiogenesis By Composite Grafts Of Modified Keratinocytes Overexpressing Vegf Driven By A Keratin Promoter.
Cocaine Cytotoxicity In Hepatocyte Cultures From Phenobarbital-Induced Rats: Involvement Of Reactive Oxygen Species And Expression Of Antioxidant Defense Systems.
Nonviral Transfer Of Genes To Pig Primary Keratinocytes. Induction Of Angiogenesis By Composite Grafts Of Modified Keratinocytes Overexpressing Vegf Driven By A Keratin Promoter.
Cocaine Cytotoxicity In Hepatocyte Cultures From Phenobarbital-Induced Rats: Involvement Of Reactive Oxygen Species And Expression Of Antioxidant Defense Systems.
Nitric Oxide Induces Tyrosine Nitration And Release Of Cytochrome C Preceding An Increase Of Mitochondrial Transmembrane Potential In Macrophages.
Cocaine Cytotoxicity In Hepatocyte Cultures From Phenobarbital-Induced Rats: Involvement Of Reactive Oxygen Species And Expression Of Antioxidant Defense Systems.
Nitric Oxide Induces Tyrosine Nitration And Release Of Cytochrome C Preceding An Increase Of Mitochondrial Transmembrane Potential In Macrophages.
The Effect Of Non-Genotoxic Carcinogens, Phenobarbital And Clofibrate, On The Relationship Between Reactive Oxygen Species, Antioxidant Enzyme Expression And Apoptosis.
Nitric Oxide Induces Tyrosine Nitration And Release Of Cytochrome C Preceding An Increase Of Mitochondrial Transmembrane Potential In Macrophages.
The Effect Of Non-Genotoxic Carcinogens, Phenobarbital And Clofibrate, On The Relationship Between Reactive Oxygen Species, Antioxidant Enzyme Expression And Apoptosis.
Influence Of Aminoguanidine On Parameters Of Liver Injury And Regeneration Induced In Rats By A Necrogenic Dose Of Thioacetamide.
The Effect Of Non-Genotoxic Carcinogens, Phenobarbital And Clofibrate, On The Relationship Between Reactive Oxygen Species, Antioxidant Enzyme Expression And Apoptosis.
Influence Of Aminoguanidine On Parameters Of Liver Injury And Regeneration Induced In Rats By A Necrogenic Dose Of Thioacetamide.
Hla-Dpbeta Residue 69 Plays A Crucial Role In Allorecognition.
Influence Of Aminoguanidine On Parameters Of Liver Injury And Regeneration Induced In Rats By A Necrogenic Dose Of Thioacetamide.
Hla-Dpbeta Residue 69 Plays A Crucial Role In Allorecognition.
Transforming Growth Factor-Beta (Tgf-Beta) And Egf Promote Cord-Like Structures That Indicate Terminal Differentiation Of Fetal Hepatocytes In Primary Culture.
Hla-Dpbeta Residue 69 Plays A Crucial Role In Allorecognition.
Transforming Growth Factor-Beta (Tgf-Beta) And Egf Promote Cord-Like Structures That Indicate Terminal Differentiation Of Fetal Hepatocytes In Primary Culture.
P42/p44 Mitogen-Activated Protein Kinases Activation Is Required For The Insulin-Like Growth Factor-I/insulin Induced Proliferation, But Inhibits Differentiation, In Rat Fetal Brown Adipocytes.
Transforming Growth Factor-Beta (Tgf-Beta) And Egf Promote Cord-Like Structures That Indicate Terminal Differentiation Of Fetal Hepatocytes In Primary Culture.
P42/p44 Mitogen-Activated Protein Kinases Activation Is Required For The Insulin-Like Growth Factor-I/insulin Induced Proliferation, But Inhibits Differentiation, In Rat Fetal Brown Adipocytes.
Flow Cytometric Studies Of Nucleoside Transport Regulation In Single Chromaffin Cells.
P42/p44 Mitogen-Activated Protein Kinases Activation Is Required For The Insulin-Like Growth Factor-I/insulin Induced Proliferation, But Inhibits Differentiation, In Rat Fetal Brown Adipocytes.
Flow Cytometric Studies Of Nucleoside Transport Regulation In Single Chromaffin Cells.
Necrogenic And Regenerative Responses Of Liver Of Newly Weaned Rats Against A Sublethal Dose Of Thioacetamide.
Flow Cytometric Studies Of Nucleoside Transport Regulation In Single Chromaffin Cells.
Necrogenic And Regenerative Responses Of Liver Of Newly Weaned Rats Against A Sublethal Dose Of Thioacetamide.
Microbiological Induced Corrosion In The Paper Industry
Necrogenic And Regenerative Responses Of Liver Of Newly Weaned Rats Against A Sublethal Dose Of Thioacetamide.
Microbiological Induced Corrosion In The Paper Industry
Tnf-Alpha Induces Apoptosis In Rat Fetal Brown Adipocytes In Primary Culture.
Microbiological Induced Corrosion In The Paper Industry
Tnf-Alpha Induces Apoptosis In Rat Fetal Brown Adipocytes In Primary Culture.
Cycloheximide Prevents Apoptosis, Reactive Oxygen Species Production, And Glutathione Depletion Induced By Transforming Growth Factor Beta In Fetal Rat Hepatocytes In Primary Culture.
Tnf-Alpha Induces Apoptosis In Rat Fetal Brown Adipocytes In Primary Culture.
Cycloheximide Prevents Apoptosis, Reactive Oxygen Species Production, And Glutathione Depletion Induced By Transforming Growth Factor Beta In Fetal Rat Hepatocytes In Primary Culture.
Neuronal Mitochondrial Morphology And Transmembrane Potential Are Severely Altered By Hypothyroidism During Rat Brain Development.
Cycloheximide Prevents Apoptosis, Reactive Oxygen Species Production, And Glutathione Depletion Induced By Transforming Growth Factor Beta In Fetal Rat Hepatocytes In Primary Culture.
Neuronal Mitochondrial Morphology And Transmembrane Potential Are Severely Altered By Hypothyroidism During Rat Brain Development.
Age-Dependent Modifications In Rat Hepatocyte Antioxidant Defense Systems.
Neuronal Mitochondrial Morphology And Transmembrane Potential Are Severely Altered By Hypothyroidism During Rat Brain Development.
Age-Dependent Modifications In Rat Hepatocyte Antioxidant Defense Systems.
Rapid Up-Regulation Of Ikappabbeta And Abrogation Of Nf-Kappab Activity In Peritoneal Macrophages Stimulated With Lipopolysaccharide.
Age-Dependent Modifications In Rat Hepatocyte Antioxidant Defense Systems.
Rapid Up-Regulation Of Ikappabbeta And Abrogation Of Nf-Kappab Activity In Peritoneal Macrophages Stimulated With Lipopolysaccharide.
Phenotypic Characterization Of A Candida Albicans Strain Deficient In Its Major Exoglucanase.
Rapid Up-Regulation Of Ikappabbeta And Abrogation Of Nf-Kappab Activity In Peritoneal Macrophages Stimulated With Lipopolysaccharide.
Phenotypic Characterization Of A Candida Albicans Strain Deficient In Its Major Exoglucanase.
Epidermal Growth Factor, But Not Hepatocyte Growth Factor, Suppresses The Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocytes In Primary Culture.
Phenotypic Characterization Of A Candida Albicans Strain Deficient In Its Major Exoglucanase.
Epidermal Growth Factor, But Not Hepatocyte Growth Factor, Suppresses The Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocytes In Primary Culture.
Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocyte Primary Cultures: Involvement Of Reactive Oxygen Intermediates.
Epidermal Growth Factor, But Not Hepatocyte Growth Factor, Suppresses The Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocytes In Primary Culture.
Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocyte Primary Cultures: Involvement Of Reactive Oxygen Intermediates.
Transforming Growth Factor Beta Modulates Growth And Differentiation Of Fetal Hepatocytes In Primary Culture.
Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocyte Primary Cultures: Involvement Of Reactive Oxygen Intermediates.
Transforming Growth Factor Beta Modulates Growth And Differentiation Of Fetal Hepatocytes In Primary Culture.
Differentiation Of Rat Brown Adipocytes During Late Foetal Development: Role Of Insulin-Like Growth Factor I.
Transforming Growth Factor Beta Modulates Growth And Differentiation Of Fetal Hepatocytes In Primary Culture.
Differentiation Of Rat Brown Adipocytes During Late Foetal Development: Role Of Insulin-Like Growth Factor I.
The Role Of Hla-Dp Beta Residue 69 In The Definition Of Antibody-Binding Epitopes.
Differentiation Of Rat Brown Adipocytes During Late Foetal Development: Role Of Insulin-Like Growth Factor I.
The Role Of Hla-Dp Beta Residue 69 In The Definition Of Antibody-Binding Epitopes.
Relationship Between Antioxidant Systems, Intracellular Thiols And Dna Ploidy In Liver Of Rats During Experimental Cirrhogenesis.
The Role Of Hla-Dp Beta Residue 69 In The Definition Of Antibody-Binding Epitopes.
Relationship Between Antioxidant Systems, Intracellular Thiols And Dna Ploidy In Liver Of Rats During Experimental Cirrhogenesis.
Splenic B Lymphocyte Programmed Cell Death Is Prevented By Nitric Oxide Release Through Mechanisms Involving Sustained Bcl-2 Levels.
Relationship Between Antioxidant Systems, Intracellular Thiols And Dna Ploidy In Liver Of Rats During Experimental Cirrhogenesis.
Splenic B Lymphocyte Programmed Cell Death Is Prevented By Nitric Oxide Release Through Mechanisms Involving Sustained Bcl-2 Levels.
Two Different Subpopulations Of Ig-Bearing Cells In Lymphoid Organs Of Rainbow Trout.
Splenic B Lymphocyte Programmed Cell Death Is Prevented By Nitric Oxide Release Through Mechanisms Involving Sustained Bcl-2 Levels.
Two Different Subpopulations Of Ig-Bearing Cells In Lymphoid Organs Of Rainbow Trout.
Cocaine-Induced Liver Injury In Mice Elicits Specific Changes In Dna Ploidy And Induces Programmed Death Of Hepatocytes
Two Different Subpopulations Of Ig-Bearing Cells In Lymphoid Organs Of Rainbow Trout.
Cocaine-Induced Liver Injury In Mice Elicits Specific Changes In Dna Ploidy And Induces Programmed Death Of Hepatocytes
Yeast Exo-Beta-Glucanases Can Be Used As Efficient And Readily Detectable Reporter Genes In Saccharomyces Cerevisiae.
Cocaine-Induced Liver Injury In Mice Elicits Specific Changes In Dna Ploidy And Induces Programmed Death Of Hepatocytes
Yeast Exo-Beta-Glucanases Can Be Used As Efficient And Readily Detectable Reporter Genes In Saccharomyces Cerevisiae.
Molecular Analysis Of An Hla-Dp Mutant Cell Line Selected For Its Resistance To Killing By Hla-Dpw2-Specific T-Cell Clones.
Yeast Exo-Beta-Glucanases Can Be Used As Efficient And Readily Detectable Reporter Genes In Saccharomyces Cerevisiae.
Molecular Analysis Of An Hla-Dp Mutant Cell Line Selected For Its Resistance To Killing By Hla-Dpw2-Specific T-Cell Clones.
Relationship Between Genomic Dna Ploidy And Parameters Of Liver Damage During Necrosis And Regeneration Induced By Thioacetamide
Molecular Analysis Of An Hla-Dp Mutant Cell Line Selected For Its Resistance To Killing By Hla-Dpw2-Specific T-Cell Clones.
Relationship Between Genomic Dna Ploidy And Parameters Of Liver Damage During Necrosis And Regeneration Induced By Thioacetamide
Differential Proliferative Response Of Cultured Fetal And Regenerating Hepatocytes To Growth Factors And Hormones.
Relationship Between Genomic Dna Ploidy And Parameters Of Liver Damage During Necrosis And Regeneration Induced By Thioacetamide
Differential Proliferative Response Of Cultured Fetal And Regenerating Hepatocytes To Growth Factors And Hormones.
Flow Cytometric Analysis Of African Swine Fever Virus-Induced Plasma Membrane Proteins And Their Humoral Immune Response In Infected Pigs.
Differential Proliferative Response Of Cultured Fetal And Regenerating Hepatocytes To Growth Factors And Hormones.
Flow Cytometric Analysis Of African Swine Fever Virus-Induced Plasma Membrane Proteins And Their Humoral Immune Response In Infected Pigs.
Flow Cytometric Analysis Of Saccharomyces Cerevisiae Autolytic Mutants And Protoplasts.
Flow Cytometric Analysis Of African Swine Fever Virus-Induced Plasma Membrane Proteins And Their Humoral Immune Response In Infected Pigs.
Flow Cytometric Analysis Of Saccharomyces Cerevisiae Autolytic Mutants And Protoplasts.
Not Available
Flow Cytometric Analysis Of Saccharomyces Cerevisiae Autolytic Mutants And Protoplasts.
Not Available
Embryo-Induced Changes In The Protein Profile Of Bovine Oviductal Extracellular Vesicles
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