PUBLICACIÓN

Vitamin E blocks early events induced by 1-methyl-4-phenylpyridinium (MPP+) in cerebellar granule cells

ACCEDER A LA PUBLICACIÓN: Scopus Orcid

González-Polo RA, Soler G, Alvarez A, Fabregat I, Fuentes JM

2003 Journal of Neurochemistry

CITAS

44

DOI

10.1046/j.1471-4159.2003.01520.x

EID

2-s2.0-0037265851

ISSN

0022-3042

BIBTEX

@article{PMID:12558993, title = {Vitamin E blocks early events induced by 1-methyl-4-phenylpyridinium (MPP+) in cerebellar granule cells.}, author = {González-Polo RA and Soler G and Alvarez A and Fabregat I and Fuentes JM}, doi = {10.1046/j.1471-4159.2003.01520.x}, journal = {Journal of neurochemistry}, month = {01}, year = {2003} }

AUTORES DE LA UEX

Alberto Manuel Álvarez Barrientos
María Luisa Campo Guinea
Rosa Ana González Polo
José Manuel Fuentes Rodríguez

OTRAS PUBLICACIONES DE INTERÉS

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Er-Mitochondria Signaling In Parkinson's Disease Review-Article
[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
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[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
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[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
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Expression Of Concern: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways. J. Cell Sci. Doi: 10.1242/jcs.02878
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Expression Of Concern: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways. J. Cell Sci. Doi: 10.1242/jcs.02878
Expression Of Concern: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains. J. Cell Sci. (2002). 115, 3119-3130.
Expression Of Concern: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways. J. Cell Sci. Doi: 10.1242/jcs.02878
Expression Of Concern: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains. J. Cell Sci. (2002). 115, 3119-3130.
N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
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N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
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Trend Of Lipid Profile In General Population From Caceres Health Area
Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Trend Of Lipid Profile In General Population From Caceres Health Area
A Disulfide Bond In The Tim23 Complex Is Crucial For Voltage Gating And Mitochondrial Protein Import
Trend Of Lipid Profile In General Population From Caceres Health Area
A Disulfide Bond In The Tim23 Complex Is Crucial For Voltage Gating And Mitochondrial Protein Import
The Basics Of Autophagy
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Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice
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Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
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Autophagy-Related Proteins Are Functionally Active In Human Spermatozoa And May Be Involved In The Regulation Of Cell Survival And Motility
Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor
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Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
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Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
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Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
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Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
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Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
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Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
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Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells
Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells
Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg
Erratum To Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans [Fungal Genet. Biol. 74 (2015) 10-20] Doi: 10.1016/j.fgb.2014.10.016
Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg
Erratum To Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans [Fungal Genet. Biol. 74 (2015) 10-20] Doi: 10.1016/j.fgb.2014.10.016
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
Erratum To Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans [Fungal Genet. Biol. 74 (2015) 10-20] Doi: 10.1016/j.fgb.2014.10.016
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Control Of Autophagy In Parkinson’s Disease
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Control Of Autophagy In Parkinson’s Disease
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Control Of Autophagy In Parkinson’s Disease
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans
Toxicity And Autophagy In Neurodegenerative Disorders
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans
Toxicity And Autophagy In Neurodegenerative Disorders
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells.
Toxicity And Autophagy In Neurodegenerative Disorders
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells.
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans.
A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells.
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans.
Origen Molecular De La Compleja Enfermedad De Parkinson
Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans.
Origen Molecular De La Compleja Enfermedad De Parkinson
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol
Origen Molecular De La Compleja Enfermedad De Parkinson
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Links Between Paraquat And Parkinson's Disease
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Links Between Paraquat And Parkinson's Disease
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Links Between Paraquat And Parkinson's Disease
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol.
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol.
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol.
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis
Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1
The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis
The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway
The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis
The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway
Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition
The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway
Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition
Effect Of High Pressure Thermal Processing On Some Essential Nutrients And Immunological Components Present In Breast Milk
Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition
Effect Of High Pressure Thermal Processing On Some Essential Nutrients And Immunological Components Present In Breast Milk
Implication Of Autophagy In Parkinson's Disease
Effect Of High Pressure Thermal Processing On Some Essential Nutrients And Immunological Components Present In Breast Milk
Implication Of Autophagy In Parkinson's Disease
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
Implication Of Autophagy In Parkinson's Disease
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Paraquat, Between Apoptosis And Autophagy
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Paraquat, Between Apoptosis And Autophagy
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Paraquat, Between Apoptosis And Autophagy
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Cell Fusion Reprogramming Leads To A Specific Hepatic Expression Pattern During Mouse Bone Marrow Derived Hepatocyte Formation In Vivo
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Cell Fusion Reprogramming Leads To A Specific Hepatic Expression Pattern During Mouse Bone Marrow Derived Hepatocyte Formation In Vivo
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Cell Fusion Reprogramming Leads To A Specific Hepatic Expression Pattern During Mouse Bone Marrow Derived Hepatocyte Formation In Vivo
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
270 Cd44+/epcam− Phenotype In Hepatocellular Carcinoma (Hcc) Cells Correlates With Increased Autocrine Tgf-Beta And Mesenchymal Traits
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
270 Cd44+/epcam− Phenotype In Hepatocellular Carcinoma (Hcc) Cells Correlates With Increased Autocrine Tgf-Beta And Mesenchymal Traits
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
270 Cd44+/epcam− Phenotype In Hepatocellular Carcinoma (Hcc) Cells Correlates With Increased Autocrine Tgf-Beta And Mesenchymal Traits
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Inhibition Of Autophagy By Tab2 And Tab3
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Inhibition Of Autophagy By Tab2 And Tab3
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Inhibition Of Autophagy By Tab2 And Tab3
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Active Commuting To School And Cognitive Performance In Adolescents: The Avena Study.
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Active Commuting To School And Cognitive Performance In Adolescents: The Avena Study.
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
Active Commuting To School And Cognitive Performance In Adolescents: The Avena Study.
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Comparative Analysis Of Carmellose 0.5% Versus Hyaluronate 0.15% In Dry Eye: A Flow Cytometric Study
Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry
Comparative Analysis Of Carmellose 0.5% Versus Hyaluronate 0.15% In Dry Eye: A Flow Cytometric Study
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Comparative Analysis Of Carmellose 0.5% Versus Hyaluronate 0.15% In Dry Eye: A Flow Cytometric Study
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
The Effect Of Preservative-Free Hp-Guar On Dry Eye After Phacoemulsification: A Flow Cytometric Study.
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
The Effect Of Preservative-Free Hp-Guar On Dry Eye After Phacoemulsification: A Flow Cytometric Study.
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
The Effect Of Preservative-Free Hp-Guar On Dry Eye After Phacoemulsification: A Flow Cytometric Study.
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Haematological Reference Values In Spanish Adolescents: The Avena Study.
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Haematological Reference Values In Spanish Adolescents: The Avena Study.
Isolation And Characterization Of A Putative Liver Progenitor Population After Treatment Of Fetal Rat Hepatocytes With Tgf-Beta
Haematological Reference Values In Spanish Adolescents: The Avena Study.
Isolation And Characterization Of A Putative Liver Progenitor Population After Treatment Of Fetal Rat Hepatocytes With Tgf-Beta
Ccaat/enhancer Binding Protein Β Deficiency Provides Cerebral Protection Following Excitotoxic Injury
Isolation And Characterization Of A Putative Liver Progenitor Population After Treatment Of Fetal Rat Hepatocytes With Tgf-Beta
Ccaat/enhancer Binding Protein Β Deficiency Provides Cerebral Protection Following Excitotoxic Injury
Deletion Of The Met Tyrosine Kinase In Liver Progenitor Oval Cells Increases Sensitivity To Apoptosis In Vitro
Ccaat/enhancer Binding Protein Β Deficiency Provides Cerebral Protection Following Excitotoxic Injury
Deletion Of The Met Tyrosine Kinase In Liver Progenitor Oval Cells Increases Sensitivity To Apoptosis In Vitro
Bcl6 Represses Nfκb Activity In Diffuse Large B-Cell Lymphomas
Deletion Of The Met Tyrosine Kinase In Liver Progenitor Oval Cells Increases Sensitivity To Apoptosis In Vitro
Bcl6 Represses Nfκb Activity In Diffuse Large B-Cell Lymphomas
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Bcl6 Represses Nfκb Activity In Diffuse Large B-Cell Lymphomas
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Arginase I Induction By Modified Lipoproteins In Macrophages: A Peroxisome Proliferator-Activated Receptor-Γ/δ-Mediated Effect That Links Lipid Metabolism And Immunity
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Arginase I Induction By Modified Lipoproteins In Macrophages: A Peroxisome Proliferator-Activated Receptor-Γ/δ-Mediated Effect That Links Lipid Metabolism And Immunity
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Arginase I Induction By Modified Lipoproteins In Macrophages: A Peroxisome Proliferator-Activated Receptor-Γ/δ-Mediated Effect That Links Lipid Metabolism And Immunity
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
Methylmalonic Acidaemia Leads To Increased Production Of Reactive Oxygen Species And Induction Of Apoptosis Through The Mitochondrial/caspase Pathway
Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines
Methylmalonic Acidaemia Leads To Increased Production Of Reactive Oxygen Species And Induction Of Apoptosis Through The Mitochondrial/caspase Pathway
Nitric Oxide Down-Regulates Caveolin-3 Levels Through The Interaction With Myogenin, Its Transcription Factor
Methylmalonic Acidaemia Leads To Increased Production Of Reactive Oxygen Species And Induction Of Apoptosis Through The Mitochondrial/caspase Pathway
Nitric Oxide Down-Regulates Caveolin-3 Levels Through The Interaction With Myogenin, Its Transcription Factor
P38α Mapk Can Positively Or Negatively Regulate Rac-1 Activity Depending On The Presence Of Serum
Nitric Oxide Down-Regulates Caveolin-3 Levels Through The Interaction With Myogenin, Its Transcription Factor
P38α Mapk Can Positively Or Negatively Regulate Rac-1 Activity Depending On The Presence Of Serum
Binding Of Cap70 To Inducible Nitric Oxide Synthase And Implications For The Vectorial Release Of Nitric Oxide In Polarized Cells
P38α Mapk Can Positively Or Negatively Regulate Rac-1 Activity Depending On The Presence Of Serum
Binding Of Cap70 To Inducible Nitric Oxide Synthase And Implications For The Vectorial Release Of Nitric Oxide In Polarized Cells
Mitochondrial Superoxide Mediates Heat-Induced Apoptotic-Like Death In Leishmania Infantum
Binding Of Cap70 To Inducible Nitric Oxide Synthase And Implications For The Vectorial Release Of Nitric Oxide In Polarized Cells
Mitochondrial Superoxide Mediates Heat-Induced Apoptotic-Like Death In Leishmania Infantum
Tlr4-Mediated Survival Of Macrophages Is Myd88 Dependent And Requires Tnf-Α Autocrine Signalling
Mitochondrial Superoxide Mediates Heat-Induced Apoptotic-Like Death In Leishmania Infantum
Tlr4-Mediated Survival Of Macrophages Is Myd88 Dependent And Requires Tnf-Α Autocrine Signalling
Role Of Peroxynitrite In Endothelial Damage Mediated By Cyclosporine A
Tlr4-Mediated Survival Of Macrophages Is Myd88 Dependent And Requires Tnf-Α Autocrine Signalling
Role Of Peroxynitrite In Endothelial Damage Mediated By Cyclosporine A
C-Jun N-Terminal Protein Kinase Signalling Pathway Mediates Lovastatin-Induced Rat Brain Neuroblast Apoptosis
Role Of Peroxynitrite In Endothelial Damage Mediated By Cyclosporine A
C-Jun N-Terminal Protein Kinase Signalling Pathway Mediates Lovastatin-Induced Rat Brain Neuroblast Apoptosis
Lovastatin Inhibits The Extracellular-Signal-Regulated Kinase Pathway In Immortalized Rat Brain Neuroblasts
C-Jun N-Terminal Protein Kinase Signalling Pathway Mediates Lovastatin-Induced Rat Brain Neuroblast Apoptosis
Lovastatin Inhibits The Extracellular-Signal-Regulated Kinase Pathway In Immortalized Rat Brain Neuroblasts
Negative Regulation Of Akt Activity By P38α Map Kinase In Cardiomyocytes Involves Membrane Localization Of Pp2a Through Interaction With Caveolin-1
Lovastatin Inhibits The Extracellular-Signal-Regulated Kinase Pathway In Immortalized Rat Brain Neuroblasts
Negative Regulation Of Akt Activity By P38α Map Kinase In Cardiomyocytes Involves Membrane Localization Of Pp2a Through Interaction With Caveolin-1
Apoptosis
Negative Regulation Of Akt Activity By P38α Map Kinase In Cardiomyocytes Involves Membrane Localization Of Pp2a Through Interaction With Caveolin-1
Apoptosis
Awaking Tim22, A Dynamic Ligand-Gated Channel For Protein Insertion In The Mitochondrial Inner Membrane
Apoptosis
Awaking Tim22, A Dynamic Ligand-Gated Channel For Protein Insertion In The Mitochondrial Inner Membrane
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Awaking Tim22, A Dynamic Ligand-Gated Channel For Protein Insertion In The Mitochondrial Inner Membrane
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
A Fluorescence Assay For Peptide Translocation Into Mitochondria
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
A Fluorescence Assay For Peptide Translocation Into Mitochondria
Tim17p Regulates The Twin Pore Structure And Voltage Gating Of The Mitochondrial Protein Import Complex Tim23
A Fluorescence Assay For Peptide Translocation Into Mitochondria
Tim17p Regulates The Twin Pore Structure And Voltage Gating Of The Mitochondrial Protein Import Complex Tim23
Heat-Induced Programmed Cell Death In Leishmania Infantum Is Reverted By Bcl-Xl Expression
Tim17p Regulates The Twin Pore Structure And Voltage Gating Of The Mitochondrial Protein Import Complex Tim23
Heat-Induced Programmed Cell Death In Leishmania Infantum Is Reverted By Bcl-Xl Expression
A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c
Heat-Induced Programmed Cell Death In Leishmania Infantum Is Reverted By Bcl-Xl Expression
A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c
Loss Of Apoptosis-Inducing Factor Leads To An Increase In Reactive Oxygen Species, And An Impairment Of Respiration That Can Be Reversed By Antioxidants [4]
A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c
Loss Of Apoptosis-Inducing Factor Leads To An Increase In Reactive Oxygen Species, And An Impairment Of Respiration That Can Be Reversed By Antioxidants [4]
Infiltration Of Inflammatory Cells Plays An Important Role In Matrix Metalloproteinase Expression And Activation In The Heart During Sepsis
Loss Of Apoptosis-Inducing Factor Leads To An Increase In Reactive Oxygen Species, And An Impairment Of Respiration That Can Be Reversed By Antioxidants [4]
Infiltration Of Inflammatory Cells Plays An Important Role In Matrix Metalloproteinase Expression And Activation In The Heart During Sepsis
Hematopoietic Mobilization In Mice Increases The Presence Of Bone Marrow-Derived Hepatocytes Via In Vivo Cell Fusion
Infiltration Of Inflammatory Cells Plays An Important Role In Matrix Metalloproteinase Expression And Activation In The Heart During Sepsis
Hematopoietic Mobilization In Mice Increases The Presence Of Bone Marrow-Derived Hepatocytes Via In Vivo Cell Fusion
Gastrin Induces Leukocyte-Endothelial Cell Interactions In Vivo And Contributes To The Inflammation Caused By Helicobacter Pylori
Hematopoietic Mobilization In Mice Increases The Presence Of Bone Marrow-Derived Hepatocytes Via In Vivo Cell Fusion
Gastrin Induces Leukocyte-Endothelial Cell Interactions In Vivo And Contributes To The Inflammation Caused By Helicobacter Pylori
Autocrine Production Of Tgf-Β Confers Resistance To Apoptosis After An Epithelial-Mesenchymal Transition Process In Hepatocytes: Role Of Egf Receptor Ligands
Gastrin Induces Leukocyte-Endothelial Cell Interactions In Vivo And Contributes To The Inflammation Caused By Helicobacter Pylori
Autocrine Production Of Tgf-Β Confers Resistance To Apoptosis After An Epithelial-Mesenchymal Transition Process In Hepatocytes: Role Of Egf Receptor Ligands
N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways
Autocrine Production Of Tgf-Β Confers Resistance To Apoptosis After An Epithelial-Mesenchymal Transition Process In Hepatocytes: Role Of Egf Receptor Ligands
N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science (2006) Vol. 119 (1558-1596))
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science (2006) Vol. 119 (1558-1596))
Erratum: A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c (Genes And Development (2006) 20 (307-320))
Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science (2006) Vol. 119 (1558-1596))
Erratum: A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c (Genes And Development (2006) 20 (307-320))
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Erratum: A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c (Genes And Development (2006) 20 (307-320))
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Identification Of A Nuclear Export Signal In The Kshv Latent Protein Lana2 Mediating Its Export From The Nucleus
Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb
Identification Of A Nuclear Export Signal In The Kshv Latent Protein Lana2 Mediating Its Export From The Nucleus
Discrepancies Between Nitroglycerin And No-Releasing Drugs On Mitochondrial Oxygen Consumption, Vasoactivity, And The Release Of No
Identification Of A Nuclear Export Signal In The Kshv Latent Protein Lana2 Mediating Its Export From The Nucleus
Discrepancies Between Nitroglycerin And No-Releasing Drugs On Mitochondrial Oxygen Consumption, Vasoactivity, And The Release Of No
Direct Interaction Between The Reductase Domain Of Endothelial Nitric Oxide Synthase And The Ryanodine Receptor
Discrepancies Between Nitroglycerin And No-Releasing Drugs On Mitochondrial Oxygen Consumption, Vasoactivity, And The Release Of No
Direct Interaction Between The Reductase Domain Of Endothelial Nitric Oxide Synthase And The Ryanodine Receptor
Pgc-1α Regulates The Mitochondrial Antioxidant Defense System In Vascular Endothelial Cells
Direct Interaction Between The Reductase Domain Of Endothelial Nitric Oxide Synthase And The Ryanodine Receptor
Pgc-1α Regulates The Mitochondrial Antioxidant Defense System In Vascular Endothelial Cells
Matrix Metalloproteinase 13 Mediates Nitric Oxide Activation Of Endothelial Cell Migration
Pgc-1α Regulates The Mitochondrial Antioxidant Defense System In Vascular Endothelial Cells
Matrix Metalloproteinase 13 Mediates Nitric Oxide Activation Of Endothelial Cell Migration
Arginase I Induction During Leishmania Major Infection Mediates The Development Of Disease
Matrix Metalloproteinase 13 Mediates Nitric Oxide Activation Of Endothelial Cell Migration
Arginase I Induction During Leishmania Major Infection Mediates The Development Of Disease
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase I Induction During Leishmania Major Infection Mediates The Development Of Disease
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor
The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death
Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor
The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death
Inhibition Of Macroautophagy Triggers Apoptosis
The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death
Inhibition Of Macroautophagy Triggers Apoptosis
Synthesis Of Nitric Oxide In Postganglionic Myenteric Neurons During Endotoxemia: Implications For Gastric Motor Function In Rats.
Inhibition Of Macroautophagy Triggers Apoptosis
Synthesis Of Nitric Oxide In Postganglionic Myenteric Neurons During Endotoxemia: Implications For Gastric Motor Function In Rats.
Source Of Early Reactive Oxygen Species In The Apoptosis Induced By Transforming Growth Factor-Β In Fetal Rat Hepatocytes
Synthesis Of Nitric Oxide In Postganglionic Myenteric Neurons During Endotoxemia: Implications For Gastric Motor Function In Rats.
Source Of Early Reactive Oxygen Species In The Apoptosis Induced By Transforming Growth Factor-Β In Fetal Rat Hepatocytes
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Source Of Early Reactive Oxygen Species In The Apoptosis Induced By Transforming Growth Factor-Β In Fetal Rat Hepatocytes
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Viral Proteins Targeting Mitochondria: Controlling Cell Death
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Viral Proteins Targeting Mitochondria: Controlling Cell Death
Resistance To Tgf-Beta-Induced Apoptosis In Regenerating Hepatocytes.
Viral Proteins Targeting Mitochondria: Controlling Cell Death
Resistance To Tgf-Beta-Induced Apoptosis In Regenerating Hepatocytes.
Palmitoylation Of Inducible Nitric-Oxide Synthase At Cys-3 Is Required For Proper Intracellular Traffic And Nitric Oxide Synthesis.
Resistance To Tgf-Beta-Induced Apoptosis In Regenerating Hepatocytes.
Palmitoylation Of Inducible Nitric-Oxide Synthase At Cys-3 Is Required For Proper Intracellular Traffic And Nitric Oxide Synthesis.
The Effect Of Milk Fermented By Yogurt Cultures Plus Lactobacillus Casei Dn-114001 On The Immune Response Of Subjects Under Academic Examination Stress.
Palmitoylation Of Inducible Nitric-Oxide Synthase At Cys-3 Is Required For Proper Intracellular Traffic And Nitric Oxide Synthesis.
The Effect Of Milk Fermented By Yogurt Cultures Plus Lactobacillus Casei Dn-114001 On The Immune Response Of Subjects Under Academic Examination Stress.
Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels.
The Effect Of Milk Fermented By Yogurt Cultures Plus Lactobacillus Casei Dn-114001 On The Immune Response Of Subjects Under Academic Examination Stress.
Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels.
P38 Alpha Mitogen-Activated Protein Kinase Sensitizes Cells To Apoptosis Induced By Different Stimuli.
Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels.
P38 Alpha Mitogen-Activated Protein Kinase Sensitizes Cells To Apoptosis Induced By Different Stimuli.
Electrophysiological Approaches To The Study Of Protein Translocation In Mitochondria
P38 Alpha Mitogen-Activated Protein Kinase Sensitizes Cells To Apoptosis Induced By Different Stimuli.
Electrophysiological Approaches To The Study Of Protein Translocation In Mitochondria
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Electrophysiological Approaches To The Study Of Protein Translocation In Mitochondria
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Comparison Of The Tim And Tom Channel Activities Of The Mitochondrial Protein Import Complexes
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Comparison Of The Tim And Tom Channel Activities Of The Mitochondrial Protein Import Complexes
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Comparison Of The Tim And Tom Channel Activities Of The Mitochondrial Protein Import Complexes
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family
Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine
The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family
Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine
Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin
Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine
Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Confocal Visualization Of Nitric Oxide In The Myenteric Plexus Of The Gastric Fundus Obtained From Endotoxin-Treated Rats
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Confocal Visualization Of Nitric Oxide In The Myenteric Plexus Of The Gastric Fundus Obtained From Endotoxin-Treated Rats
Long-Term Treatment With Insulin Induces Apoptosis In Brown Adipocytes: Role Of Oxidative Stress.
Confocal Visualization Of Nitric Oxide In The Myenteric Plexus Of The Gastric Fundus Obtained From Endotoxin-Treated Rats
Long-Term Treatment With Insulin Induces Apoptosis In Brown Adipocytes: Role Of Oxidative Stress.
Sustained Nitric Oxide Delivery Delays Nitric Oxide-Dependent Apoptosis In Macrophages: Contribution To The Physiological Function Of Activated Macrophages.
Long-Term Treatment With Insulin Induces Apoptosis In Brown Adipocytes: Role Of Oxidative Stress.
Sustained Nitric Oxide Delivery Delays Nitric Oxide-Dependent Apoptosis In Macrophages: Contribution To The Physiological Function Of Activated Macrophages.
Lithium Blocks The Pkb And Gsk3 Dephosphorylation Induced By Ceramide Through Protein Phosphatase-2a
Sustained Nitric Oxide Delivery Delays Nitric Oxide-Dependent Apoptosis In Macrophages: Contribution To The Physiological Function Of Activated Macrophages.
Lithium Blocks The Pkb And Gsk3 Dephosphorylation Induced By Ceramide Through Protein Phosphatase-2a
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Lithium Blocks The Pkb And Gsk3 Dephosphorylation Induced By Ceramide Through Protein Phosphatase-2a
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Evidence For Discoordinate Regulation Of The Hla-Dpb1 Gene
The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle
Evidence For Discoordinate Regulation Of The Hla-Dpb1 Gene
Tace/adam17-Tnf-Α Pathway In Rat Cortical Cultures After Exposure To Oxygen-Glucose Deprivation Or Glutamate
Evidence For Discoordinate Regulation Of The Hla-Dpb1 Gene
Tace/adam17-Tnf-Α Pathway In Rat Cortical Cultures After Exposure To Oxygen-Glucose Deprivation Or Glutamate
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Tace/adam17-Tnf-Α Pathway In Rat Cortical Cultures After Exposure To Oxygen-Glucose Deprivation Or Glutamate
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Aplidin Induces The Mitochondrial Apoptotic Pathway Via Oxidative Stress-Mediated Jnk And P38 Activation And Protein Kinase C Delta.
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Aplidin Induces The Mitochondrial Apoptotic Pathway Via Oxidative Stress-Mediated Jnk And P38 Activation And Protein Kinase C Delta.
Differential Role Of Ppar Gamma In The Regulation Of Ucp-1 And Adipogenesis By Tnf-Alpha In Brown Adipocytes.
Aplidin Induces The Mitochondrial Apoptotic Pathway Via Oxidative Stress-Mediated Jnk And P38 Activation And Protein Kinase C Delta.
Differential Role Of Ppar Gamma In The Regulation Of Ucp-1 And Adipogenesis By Tnf-Alpha In Brown Adipocytes.
Induction Of Apoptosis By Nitric Oxide In Macrophages Is Independent Of Apoptotic Volume Decrease.
Differential Role Of Ppar Gamma In The Regulation Of Ucp-1 And Adipogenesis By Tnf-Alpha In Brown Adipocytes.
Induction Of Apoptosis By Nitric Oxide In Macrophages Is Independent Of Apoptotic Volume Decrease.
In Vitro Fertilizing Capacity And Chromatin Condensation Of Deep Frozen Boar Semen Packaged In 0.5 And 5 Ml Straws.
Induction Of Apoptosis By Nitric Oxide In Macrophages Is Independent Of Apoptotic Volume Decrease.
In Vitro Fertilizing Capacity And Chromatin Condensation Of Deep Frozen Boar Semen Packaged In 0.5 And 5 Ml Straws.
Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains
In Vitro Fertilizing Capacity And Chromatin Condensation Of Deep Frozen Boar Semen Packaged In 0.5 And 5 Ml Straws.
Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains
Different Dependence Of Lithium And Valproate On P13k/pkb Pathway
Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains
Different Dependence Of Lithium And Valproate On P13k/pkb Pathway
Akt1/pkbalpha Protects Pc12 Cells Against The Parkinsonism-Inducing Neurotoxin 1-Methyl-4-Phenylpyridinium And Reduces The Levels Of Oxygen-Free Radicals.
Different Dependence Of Lithium And Valproate On P13k/pkb Pathway
Akt1/pkbalpha Protects Pc12 Cells Against The Parkinsonism-Inducing Neurotoxin 1-Methyl-4-Phenylpyridinium And Reduces The Levels Of Oxygen-Free Radicals.
Use Of Flow Cytometry And Confocal Microscopy Techniques To Investigate Early Cdcl2-Induced Nephrotoxicity In Vitro
Akt1/pkbalpha Protects Pc12 Cells Against The Parkinsonism-Inducing Neurotoxin 1-Methyl-4-Phenylpyridinium And Reduces The Levels Of Oxygen-Free Radicals.
Use Of Flow Cytometry And Confocal Microscopy Techniques To Investigate Early Cdcl2-Induced Nephrotoxicity In Vitro
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Use Of Flow Cytometry And Confocal Microscopy Techniques To Investigate Early Cdcl2-Induced Nephrotoxicity In Vitro
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Lovastatin Induces Apoptosis Of Spontaneously Immortalized Rat Brain Neuroblasts: Involvement Of Nonsterol Isoprenoid Biosynthesis Inhibition
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Lovastatin Induces Apoptosis Of Spontaneously Immortalized Rat Brain Neuroblasts: Involvement Of Nonsterol Isoprenoid Biosynthesis Inhibition
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Lovastatin Induces Apoptosis Of Spontaneously Immortalized Rat Brain Neuroblasts: Involvement Of Nonsterol Isoprenoid Biosynthesis Inhibition
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Intracellular Water Motion Decreases In Apoptotic Macrophages After Caspase Activation.
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Intracellular Water Motion Decreases In Apoptotic Macrophages After Caspase Activation.
Activation Of Caspases Occurs Downstream From Radical Oxygen Species Production, Bcl-Xl Down-Regulation, And Early Cytochrome C Release In Apoptosis Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes.
Intracellular Water Motion Decreases In Apoptotic Macrophages After Caspase Activation.
Activation Of Caspases Occurs Downstream From Radical Oxygen Species Production, Bcl-Xl Down-Regulation, And Early Cytochrome C Release In Apoptosis Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes.
Reactive Oxygen Species (Ros) Mediates The Mitochondrial-Dependent Apoptosis Induced By Transforming Growth Factor (Beta) In Fetal Hepatocytes.
Activation Of Caspases Occurs Downstream From Radical Oxygen Species Production, Bcl-Xl Down-Regulation, And Early Cytochrome C Release In Apoptosis Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes.
Reactive Oxygen Species (Ros) Mediates The Mitochondrial-Dependent Apoptosis Induced By Transforming Growth Factor (Beta) In Fetal Hepatocytes.
Mitochondrial Involvement In Cocaine-Treated Rat Hepatocytes: Effect Of N-Acetylcysteine And Deferoxamine.
Reactive Oxygen Species (Ros) Mediates The Mitochondrial-Dependent Apoptosis Induced By Transforming Growth Factor (Beta) In Fetal Hepatocytes.
Mitochondrial Involvement In Cocaine-Treated Rat Hepatocytes: Effect Of N-Acetylcysteine And Deferoxamine.
Glu-256 Is A Main Structural Determinant For Oligomerisation Of Human Arginase I
Mitochondrial Involvement In Cocaine-Treated Rat Hepatocytes: Effect Of N-Acetylcysteine And Deferoxamine.
Glu-256 Is A Main Structural Determinant For Oligomerisation Of Human Arginase I
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Glu-256 Is A Main Structural Determinant For Oligomerisation Of Human Arginase I
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Effect Of N-Acetylcysteine And Deferoxamine On Endogenous Antioxidant Defense System Gene Expression In A Rat Hepatocyte Model Of Cocaine Cytotoxicity.
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Effect Of N-Acetylcysteine And Deferoxamine On Endogenous Antioxidant Defense System Gene Expression In A Rat Hepatocyte Model Of Cocaine Cytotoxicity.
Inhibition Of Phosphatidylcholine Synthesis Precedes Apoptosis Induced By C2-Ceramide: Protection By Exogenous Phosphatidylcholine
Effect Of N-Acetylcysteine And Deferoxamine On Endogenous Antioxidant Defense System Gene Expression In A Rat Hepatocyte Model Of Cocaine Cytotoxicity.
Inhibition Of Phosphatidylcholine Synthesis Precedes Apoptosis Induced By C2-Ceramide: Protection By Exogenous Phosphatidylcholine
Mcc And Psc, The Putative Protein Import Channels Of Mitochondria
Inhibition Of Phosphatidylcholine Synthesis Precedes Apoptosis Induced By C2-Ceramide: Protection By Exogenous Phosphatidylcholine
Mcc And Psc, The Putative Protein Import Channels Of Mitochondria
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Mcc And Psc, The Putative Protein Import Channels Of Mitochondria
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Fibronectin Regulates Morphology, Cell Organization And Gene Expression Of Rat Fetal Hepatocytes In Primary Culture.
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Fibronectin Regulates Morphology, Cell Organization And Gene Expression Of Rat Fetal Hepatocytes In Primary Culture.
Inhibition Of Pkb/akt1 By C2-Ceramide Involves Activation Of Ceramide-Activated Protein Phosphatase In Pc12 Cells.
Fibronectin Regulates Morphology, Cell Organization And Gene Expression Of Rat Fetal Hepatocytes In Primary Culture.
Inhibition Of Pkb/akt1 By C2-Ceramide Involves Activation Of Ceramide-Activated Protein Phosphatase In Pc12 Cells.
P38 Mitogen-Activated Protein Kinase Mediates Tumor Necrosis Factor-Alpha-Induced Apoptosis In Rat Fetal Brown Adipocytes.
Inhibition Of Pkb/akt1 By C2-Ceramide Involves Activation Of Ceramide-Activated Protein Phosphatase In Pc12 Cells.
P38 Mitogen-Activated Protein Kinase Mediates Tumor Necrosis Factor-Alpha-Induced Apoptosis In Rat Fetal Brown Adipocytes.
Noradrenaline Induces Brown Adipocytes Cell Growth Via Beta-Receptors By A Mechanism Dependent On Erks But Independent Of Camp And Pka.
P38 Mitogen-Activated Protein Kinase Mediates Tumor Necrosis Factor-Alpha-Induced Apoptosis In Rat Fetal Brown Adipocytes.
Noradrenaline Induces Brown Adipocytes Cell Growth Via Beta-Receptors By A Mechanism Dependent On Erks But Independent Of Camp And Pka.
Epidermal Growth Factor Impairs The Cytochrome C/caspase-3 Apoptotic Pathway Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes Via A Phosphoinositide 3-Kinase-Dependent Pathway.
Noradrenaline Induces Brown Adipocytes Cell Growth Via Beta-Receptors By A Mechanism Dependent On Erks But Independent Of Camp And Pka.
Epidermal Growth Factor Impairs The Cytochrome C/caspase-3 Apoptotic Pathway Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes Via A Phosphoinositide 3-Kinase-Dependent Pathway.
Changes In Antioxidant Defence Systems Induced By Cyclosporine A In Cultures Of Hepatocytes From 2- And 12-Month-Old Rats.
Epidermal Growth Factor Impairs The Cytochrome C/caspase-3 Apoptotic Pathway Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes Via A Phosphoinositide 3-Kinase-Dependent Pathway.
Changes In Antioxidant Defence Systems Induced By Cyclosporine A In Cultures Of Hepatocytes From 2- And 12-Month-Old Rats.
Contribution Of Cyclopentenone Prostaglandins To The Resolution Of Inflammation Through The Potentiation Of Apoptosis In Activated Macrophages.
Changes In Antioxidant Defence Systems Induced By Cyclosporine A In Cultures Of Hepatocytes From 2- And 12-Month-Old Rats.
Contribution Of Cyclopentenone Prostaglandins To The Resolution Of Inflammation Through The Potentiation Of Apoptosis In Activated Macrophages.
Hsp70 Induction By Cyclosporine A In Cultured Rat Hepatocytes: Effect Of Vitamin E Succinate.
Contribution Of Cyclopentenone Prostaglandins To The Resolution Of Inflammation Through The Potentiation Of Apoptosis In Activated Macrophages.
Hsp70 Induction By Cyclosporine A In Cultured Rat Hepatocytes: Effect Of Vitamin E Succinate.
Applications Of Confocal Mycroscopy To The Paper Industry: Slime And Deposit Examples
Hsp70 Induction By Cyclosporine A In Cultured Rat Hepatocytes: Effect Of Vitamin E Succinate.
Applications Of Confocal Mycroscopy To The Paper Industry: Slime And Deposit Examples
Applications Of Flow Cytometry To Clinical Microbiology
Applications Of Confocal Mycroscopy To The Paper Industry: Slime And Deposit Examples
Applications Of Flow Cytometry To Clinical Microbiology
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Applications Of Flow Cytometry To Clinical Microbiology
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Signal Presequences Increase Mitochondrial Permeability And Open The Multiple Conductance Channel
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Signal Presequences Increase Mitochondrial Permeability And Open The Multiple Conductance Channel
Age-Related Changes On Parameters Of Experimentally-Induced Liver Injury And Regeneration.
Signal Presequences Increase Mitochondrial Permeability And Open The Multiple Conductance Channel
Age-Related Changes On Parameters Of Experimentally-Induced Liver Injury And Regeneration.
Apoptotic Response To Tgf-Beta In Fetal Hepatocytes Depends Upon Their State Of Differentiation.
Age-Related Changes On Parameters Of Experimentally-Induced Liver Injury And Regeneration.
Apoptotic Response To Tgf-Beta In Fetal Hepatocytes Depends Upon Their State Of Differentiation.
Nonviral Transfer Of Genes To Pig Primary Keratinocytes. Induction Of Angiogenesis By Composite Grafts Of Modified Keratinocytes Overexpressing Vegf Driven By A Keratin Promoter.
Apoptotic Response To Tgf-Beta In Fetal Hepatocytes Depends Upon Their State Of Differentiation.
Nonviral Transfer Of Genes To Pig Primary Keratinocytes. Induction Of Angiogenesis By Composite Grafts Of Modified Keratinocytes Overexpressing Vegf Driven By A Keratin Promoter.
Cocaine Cytotoxicity In Hepatocyte Cultures From Phenobarbital-Induced Rats: Involvement Of Reactive Oxygen Species And Expression Of Antioxidant Defense Systems.
Nonviral Transfer Of Genes To Pig Primary Keratinocytes. Induction Of Angiogenesis By Composite Grafts Of Modified Keratinocytes Overexpressing Vegf Driven By A Keratin Promoter.
Cocaine Cytotoxicity In Hepatocyte Cultures From Phenobarbital-Induced Rats: Involvement Of Reactive Oxygen Species And Expression Of Antioxidant Defense Systems.
Nitric Oxide Induces Tyrosine Nitration And Release Of Cytochrome C Preceding An Increase Of Mitochondrial Transmembrane Potential In Macrophages.
Cocaine Cytotoxicity In Hepatocyte Cultures From Phenobarbital-Induced Rats: Involvement Of Reactive Oxygen Species And Expression Of Antioxidant Defense Systems.
Nitric Oxide Induces Tyrosine Nitration And Release Of Cytochrome C Preceding An Increase Of Mitochondrial Transmembrane Potential In Macrophages.
Th1/th2-Regulated Expression Of Arginase Isoforms In Murine Macrophages And Dendritic Cells
Nitric Oxide Induces Tyrosine Nitration And Release Of Cytochrome C Preceding An Increase Of Mitochondrial Transmembrane Potential In Macrophages.
Th1/th2-Regulated Expression Of Arginase Isoforms In Murine Macrophages And Dendritic Cells
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Th1/th2-Regulated Expression Of Arginase Isoforms In Murine Macrophages And Dendritic Cells
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
The Effect Of Non-Genotoxic Carcinogens, Phenobarbital And Clofibrate, On The Relationship Between Reactive Oxygen Species, Antioxidant Enzyme Expression And Apoptosis.
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
The Effect Of Non-Genotoxic Carcinogens, Phenobarbital And Clofibrate, On The Relationship Between Reactive Oxygen Species, Antioxidant Enzyme Expression And Apoptosis.
Influence Of Aminoguanidine On Parameters Of Liver Injury And Regeneration Induced In Rats By A Necrogenic Dose Of Thioacetamide.
The Effect Of Non-Genotoxic Carcinogens, Phenobarbital And Clofibrate, On The Relationship Between Reactive Oxygen Species, Antioxidant Enzyme Expression And Apoptosis.
Influence Of Aminoguanidine On Parameters Of Liver Injury And Regeneration Induced In Rats By A Necrogenic Dose Of Thioacetamide.
Hla-Dpbeta Residue 69 Plays A Crucial Role In Allorecognition.
Influence Of Aminoguanidine On Parameters Of Liver Injury And Regeneration Induced In Rats By A Necrogenic Dose Of Thioacetamide.
Hla-Dpbeta Residue 69 Plays A Crucial Role In Allorecognition.
Transforming Growth Factor-Beta (Tgf-Beta) And Egf Promote Cord-Like Structures That Indicate Terminal Differentiation Of Fetal Hepatocytes In Primary Culture.
Hla-Dpbeta Residue 69 Plays A Crucial Role In Allorecognition.
Transforming Growth Factor-Beta (Tgf-Beta) And Egf Promote Cord-Like Structures That Indicate Terminal Differentiation Of Fetal Hepatocytes In Primary Culture.
P42/p44 Mitogen-Activated Protein Kinases Activation Is Required For The Insulin-Like Growth Factor-I/insulin Induced Proliferation, But Inhibits Differentiation, In Rat Fetal Brown Adipocytes.
Transforming Growth Factor-Beta (Tgf-Beta) And Egf Promote Cord-Like Structures That Indicate Terminal Differentiation Of Fetal Hepatocytes In Primary Culture.
P42/p44 Mitogen-Activated Protein Kinases Activation Is Required For The Insulin-Like Growth Factor-I/insulin Induced Proliferation, But Inhibits Differentiation, In Rat Fetal Brown Adipocytes.
Flow Cytometric Studies Of Nucleoside Transport Regulation In Single Chromaffin Cells.
P42/p44 Mitogen-Activated Protein Kinases Activation Is Required For The Insulin-Like Growth Factor-I/insulin Induced Proliferation, But Inhibits Differentiation, In Rat Fetal Brown Adipocytes.
Flow Cytometric Studies Of Nucleoside Transport Regulation In Single Chromaffin Cells.
Necrogenic And Regenerative Responses Of Liver Of Newly Weaned Rats Against A Sublethal Dose Of Thioacetamide.
Flow Cytometric Studies Of Nucleoside Transport Regulation In Single Chromaffin Cells.
Necrogenic And Regenerative Responses Of Liver Of Newly Weaned Rats Against A Sublethal Dose Of Thioacetamide.
Microbiological Induced Corrosion In The Paper Industry
Necrogenic And Regenerative Responses Of Liver Of Newly Weaned Rats Against A Sublethal Dose Of Thioacetamide.
Microbiological Induced Corrosion In The Paper Industry
Anti-Mhv3 State Induced By Ifn Gamma In Macrophages Is Not Related To Arginine Metabolism
Microbiological Induced Corrosion In The Paper Industry
Anti-Mhv3 State Induced By Ifn Gamma In Macrophages Is Not Related To Arginine Metabolism
Effects Of Carbonyl Cyanide Phenylhydrazones On Two Mitochondrial Ion Channel Activities
Anti-Mhv3 State Induced By Ifn Gamma In Macrophages Is Not Related To Arginine Metabolism
Effects Of Carbonyl Cyanide Phenylhydrazones On Two Mitochondrial Ion Channel Activities
Involvement Of Protein Kinase A In The Induction Of Arginase In Murine Bone Marrow-Derived Macrophages
Effects Of Carbonyl Cyanide Phenylhydrazones On Two Mitochondrial Ion Channel Activities
Involvement Of Protein Kinase A In The Induction Of Arginase In Murine Bone Marrow-Derived Macrophages
Oxidation Of N(G)-Hydroxy-L-Arginine To Nitric Oxide Mediated By Respiratory Burst: An Alternative Pathway To No Synthesis
Involvement Of Protein Kinase A In The Induction Of Arginase In Murine Bone Marrow-Derived Macrophages
Oxidation Of N(G)-Hydroxy-L-Arginine To Nitric Oxide Mediated By Respiratory Burst: An Alternative Pathway To No Synthesis
Tnf-Alpha Induces Apoptosis In Rat Fetal Brown Adipocytes In Primary Culture.
Oxidation Of N(G)-Hydroxy-L-Arginine To Nitric Oxide Mediated By Respiratory Burst: An Alternative Pathway To No Synthesis
Tnf-Alpha Induces Apoptosis In Rat Fetal Brown Adipocytes In Primary Culture.
Cycloheximide Prevents Apoptosis, Reactive Oxygen Species Production, And Glutathione Depletion Induced By Transforming Growth Factor Beta In Fetal Rat Hepatocytes In Primary Culture.
Tnf-Alpha Induces Apoptosis In Rat Fetal Brown Adipocytes In Primary Culture.
Cycloheximide Prevents Apoptosis, Reactive Oxygen Species Production, And Glutathione Depletion Induced By Transforming Growth Factor Beta In Fetal Rat Hepatocytes In Primary Culture.
Neuronal Mitochondrial Morphology And Transmembrane Potential Are Severely Altered By Hypothyroidism During Rat Brain Development.
Cycloheximide Prevents Apoptosis, Reactive Oxygen Species Production, And Glutathione Depletion Induced By Transforming Growth Factor Beta In Fetal Rat Hepatocytes In Primary Culture.
Neuronal Mitochondrial Morphology And Transmembrane Potential Are Severely Altered By Hypothyroidism During Rat Brain Development.
Age-Dependent Modifications In Rat Hepatocyte Antioxidant Defense Systems.
Neuronal Mitochondrial Morphology And Transmembrane Potential Are Severely Altered By Hypothyroidism During Rat Brain Development.
Age-Dependent Modifications In Rat Hepatocyte Antioxidant Defense Systems.
Rapid Up-Regulation Of Ikappabbeta And Abrogation Of Nf-Kappab Activity In Peritoneal Macrophages Stimulated With Lipopolysaccharide.
Age-Dependent Modifications In Rat Hepatocyte Antioxidant Defense Systems.
Rapid Up-Regulation Of Ikappabbeta And Abrogation Of Nf-Kappab Activity In Peritoneal Macrophages Stimulated With Lipopolysaccharide.
Phenotypic Characterization Of A Candida Albicans Strain Deficient In Its Major Exoglucanase.
Rapid Up-Regulation Of Ikappabbeta And Abrogation Of Nf-Kappab Activity In Peritoneal Macrophages Stimulated With Lipopolysaccharide.
Phenotypic Characterization Of A Candida Albicans Strain Deficient In Its Major Exoglucanase.
Perspectives On The Mitochondrial Multiple Conductance Channel
Phenotypic Characterization Of A Candida Albicans Strain Deficient In Its Major Exoglucanase.
Perspectives On The Mitochondrial Multiple Conductance Channel
Epidermal Growth Factor, But Not Hepatocyte Growth Factor, Suppresses The Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocytes In Primary Culture.
Perspectives On The Mitochondrial Multiple Conductance Channel
Epidermal Growth Factor, But Not Hepatocyte Growth Factor, Suppresses The Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocytes In Primary Culture.
Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocyte Primary Cultures: Involvement Of Reactive Oxygen Intermediates.
Epidermal Growth Factor, But Not Hepatocyte Growth Factor, Suppresses The Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocytes In Primary Culture.
Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocyte Primary Cultures: Involvement Of Reactive Oxygen Intermediates.
Arginase Induction By Suppressors Of Nitric Oxide Synthesis (Il-4, Il-10 And Pge2) In Murine Bone-Marrow-Derived Macrophages
Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocyte Primary Cultures: Involvement Of Reactive Oxygen Intermediates.
Arginase Induction By Suppressors Of Nitric Oxide Synthesis (Il-4, Il-10 And Pge2) In Murine Bone-Marrow-Derived Macrophages
Reciprocal Regulation Of The Nitric Oxide Synthase/arginase Balance In Mouse Bone Marrow‐derived Macrophages By Th 1 And Th 2 Cytokines
Arginase Induction By Suppressors Of Nitric Oxide Synthesis (Il-4, Il-10 And Pge2) In Murine Bone-Marrow-Derived Macrophages
Reciprocal Regulation Of The Nitric Oxide Synthase/arginase Balance In Mouse Bone Marrow‐derived Macrophages By Th 1 And Th 2 Cytokines
Transforming Growth Factor Beta Modulates Growth And Differentiation Of Fetal Hepatocytes In Primary Culture.
Reciprocal Regulation Of The Nitric Oxide Synthase/arginase Balance In Mouse Bone Marrow‐derived Macrophages By Th 1 And Th 2 Cytokines
Transforming Growth Factor Beta Modulates Growth And Differentiation Of Fetal Hepatocytes In Primary Culture.
Differentiation Of Rat Brown Adipocytes During Late Foetal Development: Role Of Insulin-Like Growth Factor I.
Transforming Growth Factor Beta Modulates Growth And Differentiation Of Fetal Hepatocytes In Primary Culture.
Differentiation Of Rat Brown Adipocytes During Late Foetal Development: Role Of Insulin-Like Growth Factor I.
The Role Of Hla-Dp Beta Residue 69 In The Definition Of Antibody-Binding Epitopes.
Differentiation Of Rat Brown Adipocytes During Late Foetal Development: Role Of Insulin-Like Growth Factor I.
The Role Of Hla-Dp Beta Residue 69 In The Definition Of Antibody-Binding Epitopes.
Relationship Between Antioxidant Systems, Intracellular Thiols And Dna Ploidy In Liver Of Rats During Experimental Cirrhogenesis.
The Role Of Hla-Dp Beta Residue 69 In The Definition Of Antibody-Binding Epitopes.
Relationship Between Antioxidant Systems, Intracellular Thiols And Dna Ploidy In Liver Of Rats During Experimental Cirrhogenesis.
Splenic B Lymphocyte Programmed Cell Death Is Prevented By Nitric Oxide Release Through Mechanisms Involving Sustained Bcl-2 Levels.
Relationship Between Antioxidant Systems, Intracellular Thiols And Dna Ploidy In Liver Of Rats During Experimental Cirrhogenesis.
Splenic B Lymphocyte Programmed Cell Death Is Prevented By Nitric Oxide Release Through Mechanisms Involving Sustained Bcl-2 Levels.
Two Different Subpopulations Of Ig-Bearing Cells In Lymphoid Organs Of Rainbow Trout.
Splenic B Lymphocyte Programmed Cell Death Is Prevented By Nitric Oxide Release Through Mechanisms Involving Sustained Bcl-2 Levels.
Two Different Subpopulations Of Ig-Bearing Cells In Lymphoid Organs Of Rainbow Trout.
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
Two Different Subpopulations Of Ig-Bearing Cells In Lymphoid Organs Of Rainbow Trout.
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
Cocaine-Induced Liver Injury In Mice Elicits Specific Changes In Dna Ploidy And Induces Programmed Death Of Hepatocytes
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
Cocaine-Induced Liver Injury In Mice Elicits Specific Changes In Dna Ploidy And Induces Programmed Death Of Hepatocytes
Yeast Exo-Beta-Glucanases Can Be Used As Efficient And Readily Detectable Reporter Genes In Saccharomyces Cerevisiae.
Cocaine-Induced Liver Injury In Mice Elicits Specific Changes In Dna Ploidy And Induces Programmed Death Of Hepatocytes
Yeast Exo-Beta-Glucanases Can Be Used As Efficient And Readily Detectable Reporter Genes In Saccharomyces Cerevisiae.
Molecular Analysis Of An Hla-Dp Mutant Cell Line Selected For Its Resistance To Killing By Hla-Dpw2-Specific T-Cell Clones.
Yeast Exo-Beta-Glucanases Can Be Used As Efficient And Readily Detectable Reporter Genes In Saccharomyces Cerevisiae.
Molecular Analysis Of An Hla-Dp Mutant Cell Line Selected For Its Resistance To Killing By Hla-Dpw2-Specific T-Cell Clones.
The Immune System And Autoaggression
Molecular Analysis Of An Hla-Dp Mutant Cell Line Selected For Its Resistance To Killing By Hla-Dpw2-Specific T-Cell Clones.
The Immune System And Autoaggression
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
The Immune System And Autoaggression
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
Determination Of Arginase Activity In Macrophages: A Micromethod
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
Determination Of Arginase Activity In Macrophages: A Micromethod
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Determination Of Arginase Activity In Macrophages: A Micromethod
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Relationship Between Genomic Dna Ploidy And Parameters Of Liver Damage During Necrosis And Regeneration Induced By Thioacetamide
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Relationship Between Genomic Dna Ploidy And Parameters Of Liver Damage During Necrosis And Regeneration Induced By Thioacetamide
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
Relationship Between Genomic Dna Ploidy And Parameters Of Liver Damage During Necrosis And Regeneration Induced By Thioacetamide
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
Differential Proliferative Response Of Cultured Fetal And Regenerating Hepatocytes To Growth Factors And Hormones.
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
Differential Proliferative Response Of Cultured Fetal And Regenerating Hepatocytes To Growth Factors And Hormones.
Flow Cytometric Analysis Of African Swine Fever Virus-Induced Plasma Membrane Proteins And Their Humoral Immune Response In Infected Pigs.
Differential Proliferative Response Of Cultured Fetal And Regenerating Hepatocytes To Growth Factors And Hormones.
Flow Cytometric Analysis Of African Swine Fever Virus-Induced Plasma Membrane Proteins And Their Humoral Immune Response In Infected Pigs.
Flow Cytometric Analysis Of Saccharomyces Cerevisiae Autolytic Mutants And Protoplasts.
Flow Cytometric Analysis Of African Swine Fever Virus-Induced Plasma Membrane Proteins And Their Humoral Immune Response In Infected Pigs.
Flow Cytometric Analysis Of Saccharomyces Cerevisiae Autolytic Mutants And Protoplasts.
The Effect Of Antimycin A On Mouse Liver Inner Mitochondrial Membrane Channel Activity
Flow Cytometric Analysis Of Saccharomyces Cerevisiae Autolytic Mutants And Protoplasts.
The Effect Of Antimycin A On Mouse Liver Inner Mitochondrial Membrane Channel Activity
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
The Effect Of Antimycin A On Mouse Liver Inner Mitochondrial Membrane Channel Activity
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Arginine Catabolism In The Phototrophic Bacterium Rhodobacter Capsulatus E1f1: Purification And Properties Of Arginase
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Arginine Catabolism In The Phototrophic Bacterium Rhodobacter Capsulatus E1f1: Purification And Properties Of Arginase
Trypsin Digestion Of Arginase: Evidence For A Stable Conformation Manganese Directed
Arginine Catabolism In The Phototrophic Bacterium Rhodobacter Capsulatus E1f1: Purification And Properties Of Arginase
Trypsin Digestion Of Arginase: Evidence For A Stable Conformation Manganese Directed
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Trypsin Digestion Of Arginase: Evidence For A Stable Conformation Manganese Directed
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
Properties Of Arginase Immobilized In A Fibrin Clot
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
Properties Of Arginase Immobilized In A Fibrin Clot
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Properties Of Arginase Immobilized In A Fibrin Clot
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The Regulation Of Glucose 6-Phosphate Dehydrogenase From Dicentrarchus Labrax (Bass) Liver
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The Regulation Of Glucose 6-Phosphate Dehydrogenase From Dicentrarchus Labrax (Bass) Liver
Mitochondrial Channel Activity Studied By Patch-Clamping Mitoplasts
The Regulation Of Glucose 6-Phosphate Dehydrogenase From Dicentrarchus Labrax (Bass) Liver
Mitochondrial Channel Activity Studied By Patch-Clamping Mitoplasts
Glucose-6-Phosphate Dehydrogenase From Dicentrarchus Labrax Liver: Kinetic Mechanism And Kinetics Of Nadph Inhibition
Mitochondrial Channel Activity Studied By Patch-Clamping Mitoplasts
Glucose-6-Phosphate Dehydrogenase From Dicentrarchus Labrax Liver: Kinetic Mechanism And Kinetics Of Nadph Inhibition
Circadian Rhythms In Enzymatic Activity Of Rat Liver Arginase And Glucose 6-Phosphate Dehydrogenase.
Glucose-6-Phosphate Dehydrogenase From Dicentrarchus Labrax Liver: Kinetic Mechanism And Kinetics Of Nadph Inhibition
Circadian Rhythms In Enzymatic Activity Of Rat Liver Arginase And Glucose 6-Phosphate Dehydrogenase.
Nucleotide Effects On Kinetic Properties Of Mitochondril Atpase
Circadian Rhythms In Enzymatic Activity Of Rat Liver Arginase And Glucose 6-Phosphate Dehydrogenase.
Nucleotide Effects On Kinetic Properties Of Mitochondril Atpase
Effect Of Nucleotides And Inhibitory Anions On Mitochondrial Atpase. Its Ph Dependence.
Nucleotide Effects On Kinetic Properties Of Mitochondril Atpase
Effect Of Nucleotides And Inhibitory Anions On Mitochondrial Atpase. Its Ph Dependence.
Nucleotide Effects On Kinetic Properties Of Mitochondrial Atpase.
Effect Of Nucleotides And Inhibitory Anions On Mitochondrial Atpase. Its Ph Dependence.
Nucleotide Effects On Kinetic Properties Of Mitochondrial Atpase.
Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)
Nucleotide Effects On Kinetic Properties Of Mitochondrial Atpase.
Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)
Protonmotive Force And Photophosphorylation In Single Swollen Thylakoid Vesicles
Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)
Protonmotive Force And Photophosphorylation In Single Swollen Thylakoid Vesicles
Assay Of Atp Synthesis Using Single Giant Mitochondria
Protonmotive Force And Photophosphorylation In Single Swollen Thylakoid Vesicles
Assay Of Atp Synthesis Using Single Giant Mitochondria
Protonmotive Force In Giant Mitochondria
Assay Of Atp Synthesis Using Single Giant Mitochondria
Protonmotive Force In Giant Mitochondria
Energy Transduction, Ion Transport And Protonmotive Force: Are They Linked?
Protonmotive Force In Giant Mitochondria
Energy Transduction, Ion Transport And Protonmotive Force: Are They Linked?
Effects Of Heating On The Ion-Gating Function And Structural Domains Of The Acetylcholine Receptor
Energy Transduction, Ion Transport And Protonmotive Force: Are They Linked?
Effects Of Heating On The Ion-Gating Function And Structural Domains Of The Acetylcholine Receptor
Controlled Thermal Disruption Of The Ion Gating Function And The Structure Of The Acetylcholine Receptor
Effects Of Heating On The Ion-Gating Function And Structural Domains Of The Acetylcholine Receptor
Controlled Thermal Disruption Of The Ion Gating Function And The Structure Of The Acetylcholine Receptor
Three Catalytic Sites In Mitochondrial Atpase
Controlled Thermal Disruption Of The Ion Gating Function And The Structure Of The Acetylcholine Receptor
Three Catalytic Sites In Mitochondrial Atpase
Reductive Methylation As A Probe Of The Heat-Labile Α-Bungarotoxin-Acetylcholine Receptor Membrane Complex: Evidence For Surface Interactions
Three Catalytic Sites In Mitochondrial Atpase
Reductive Methylation As A Probe Of The Heat-Labile Α-Bungarotoxin-Acetylcholine Receptor Membrane Complex: Evidence For Surface Interactions
Nutritional Regulation Of Glycolysis In Rainbow Trout (Salmo Gairdneri R.)
Reductive Methylation As A Probe Of The Heat-Labile Α-Bungarotoxin-Acetylcholine Receptor Membrane Complex: Evidence For Surface Interactions
Nutritional Regulation Of Glycolysis In Rainbow Trout (Salmo Gairdneri R.)
Physico-Chemical Properties Of Guinea Pig Liver Arginosuccinate Synthetase
Nutritional Regulation Of Glycolysis In Rainbow Trout (Salmo Gairdneri R.)
Physico-Chemical Properties Of Guinea Pig Liver Arginosuccinate Synthetase
Arginine As Competitive Inhibitor Of Arginosuccinate Synthetase From Guinea Pig Liver
Physico-Chemical Properties Of Guinea Pig Liver Arginosuccinate Synthetase
Arginine As Competitive Inhibitor Of Arginosuccinate Synthetase From Guinea Pig Liver
Physico-Chemical Properties Of Guinea Pig Liver Arginase
Arginine As Competitive Inhibitor Of Arginosuccinate Synthetase From Guinea Pig Liver
Physico-Chemical Properties Of Guinea Pig Liver Arginase
Effect Of Ph On The Sensitivity Of Mitochondrial Atpase To Free Atp, Adp And Anions.
Physico-Chemical Properties Of Guinea Pig Liver Arginase
Effect Of Ph On The Sensitivity Of Mitochondrial Atpase To Free Atp, Adp And Anions.
The Influence Of Fasting And Cortisol Upon Arginosuccinate Synthetase, Arginosuccinase And Arginase Levels In Guinea Pig Liver
Effect Of Ph On The Sensitivity Of Mitochondrial Atpase To Free Atp, Adp And Anions.
The Influence Of Fasting And Cortisol Upon Arginosuccinate Synthetase, Arginosuccinase And Arginase Levels In Guinea Pig Liver
Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice
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