Portal Investigación UEx

Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)

Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice

Embryo-Induced Changes In The Protein Profile Of Bovine Oviductal Extracellular Vesicles

Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice

Embryo-Induced Changes In The Protein Profile Of Bovine Oviductal Extracellular Vesicles

Neurotoxic Amyloid Β‐peptide And Tau Produce Cytokine‐like Effects On Pmca In Glioblastoma Cell Lines, Enhancing Its Activity And Isoforms Expression

Embryo-Induced Changes In The Protein Profile Of Bovine Oviductal Extracellular Vesicles

Neurotoxic Amyloid Β‐peptide And Tau Produce Cytokine‐like Effects On Pmca In Glioblastoma Cell Lines, Enhancing Its Activity And Isoforms Expression

Editorial: Go With The Vet-Flow! The Current Uses And New Frontiers Of Flow Cytometry In Veterinary Sciences

Neurotoxic Amyloid Β‐peptide And Tau Produce Cytokine‐like Effects On Pmca In Glioblastoma Cell Lines, Enhancing Its Activity And Isoforms Expression

Editorial: Go With The Vet-Flow! The Current Uses And New Frontiers Of Flow Cytometry In Veterinary Sciences

Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques

Editorial: Go With The Vet-Flow! The Current Uses And New Frontiers Of Flow Cytometry In Veterinary Sciences

Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques

The Role Of Cardiolipin In Mitochondrial Function And Neurodegenerative Diseases

Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques

The Role Of Cardiolipin In Mitochondrial Function And Neurodegenerative Diseases

Immunological Aspects Of Central Neurodegeneration

The Role Of Cardiolipin In Mitochondrial Function And Neurodegenerative Diseases

Immunological Aspects Of Central Neurodegeneration

Role Of Tfeb In Huntington’s Disease

Immunological Aspects Of Central Neurodegeneration

Role Of Tfeb In Huntington’s Disease

Immunophenotype Profile By Flow Cytometry Reveals Different Subtypes Of Extracellular Vesicles In Porcine Seminal Plasma

Role Of Tfeb In Huntington’s Disease

Immunophenotype Profile By Flow Cytometry Reveals Different Subtypes Of Extracellular Vesicles In Porcine Seminal Plasma

Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects

Immunophenotype Profile By Flow Cytometry Reveals Different Subtypes Of Extracellular Vesicles In Porcine Seminal Plasma

Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects

Stim1 Translocation To The Nucleus Protects Cells From Dna Damage

Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects

Stim1 Translocation To The Nucleus Protects Cells From Dna Damage

Lysosomal Dysfunction: Connecting The Dots In The Landscape Of Human Diseases

Stim1 Translocation To The Nucleus Protects Cells From Dna Damage

Lysosomal Dysfunction: Connecting The Dots In The Landscape Of Human Diseases

Corrigendum: Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells (Journal Of Cellular Physiology, 10.1002/jcp.26855)

Lysosomal Dysfunction: Connecting The Dots In The Landscape Of Human Diseases

Corrigendum: Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells (Journal Of Cellular Physiology, 10.1002/jcp.26855)

Vitamin D Receptor Polymorphisms In A Spanish Cohort Of Parkinson’s Disease Patients

Corrigendum: Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells (Journal Of Cellular Physiology, 10.1002/jcp.26855)

Vitamin D Receptor Polymorphisms In A Spanish Cohort Of Parkinson’s Disease Patients

Links Between Paraquat And Parkinson’s Disease

Vitamin D Receptor Polymorphisms In A Spanish Cohort Of Parkinson’s Disease Patients

Links Between Paraquat And Parkinson’s Disease

Mitochondrial Dysfunction In Repeat Expansion Diseases

Links Between Paraquat And Parkinson’s Disease

Mitochondrial Dysfunction In Repeat Expansion Diseases

Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction

Mitochondrial Dysfunction In Repeat Expansion Diseases

Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction

Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease

Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction

Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease

Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease

Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease

Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease

The Proteome Of Large Or Small Extracellular Vesicles In Pig Seminal Plasma Differs, Defining Sources And Biological Functions

Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease

The Proteome Of Large Or Small Extracellular Vesicles In Pig Seminal Plasma Differs, Defining Sources And Biological Functions

The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress

The Proteome Of Large Or Small Extracellular Vesicles In Pig Seminal Plasma Differs, Defining Sources And Biological Functions

The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress

Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles

The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress

Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles

In Vitro And In Vivo Models To Study The Biological And Pharmacological Properties Of Queen Bee Acid (Qba, 10-Hydroxy-2-Decenoic Acid): A Systematic Review

Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles

In Vitro And In Vivo Models To Study The Biological And Pharmacological Properties Of Queen Bee Acid (Qba, 10-Hydroxy-2-Decenoic Acid): A Systematic Review

Role Of Inflammatory Cytokines In The Conversion Of Mild Cognitive Impairment To Dementia: A Prospective Study.

In Vitro And In Vivo Models To Study The Biological And Pharmacological Properties Of Queen Bee Acid (Qba, 10-Hydroxy-2-Decenoic Acid): A Systematic Review

Role Of Inflammatory Cytokines In The Conversion Of Mild Cognitive Impairment To Dementia: A Prospective Study.

Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria

Role Of Inflammatory Cytokines In The Conversion Of Mild Cognitive Impairment To Dementia: A Prospective Study.

Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria

Neuroprotective And Anti-Inflammatory Effects Of Linoleic Acid In Models Of Parkinson’s Disease: The Implication Of Lipid Droplets And Lipophagy

Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria

Neuroprotective And Anti-Inflammatory Effects Of Linoleic Acid In Models Of Parkinson’s Disease: The Implication Of Lipid Droplets And Lipophagy

Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)

Neuroprotective And Anti-Inflammatory Effects Of Linoleic Acid In Models Of Parkinson’s Disease: The Implication Of Lipid Droplets And Lipophagy

Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)

Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.

Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)

Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1

Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1

Links Between Paraquat And Parkinson’s Disease

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1

Links Between Paraquat And Parkinson’s Disease

Procedimientos, Experimentación E Investigación En Enfermedades Neurodegenerativas: Protocolos Para Un Laboratorio De Neurociencias

Links Between Paraquat And Parkinson’s Disease

Procedimientos, Experimentación E Investigación En Enfermedades Neurodegenerativas: Protocolos Para Un Laboratorio De Neurociencias

Cultivos Celulares

Procedimientos, Experimentación E Investigación En Enfermedades Neurodegenerativas: Protocolos Para Un Laboratorio De Neurociencias

Cultivos Celulares

Microscopía Electrónica

Cultivos Celulares

Microscopía Electrónica

Análisis De Extracto Proteico Por Western Blotting

Microscopía Electrónica

Análisis De Extracto Proteico Por Western Blotting

Extracción De Proteínas

Análisis De Extracto Proteico Por Western Blotting

Extracción De Proteínas

Técnica De Sobreexpresión Génica

Extracción De Proteínas

Técnica De Sobreexpresión Génica

Técnica De Silenciamiento Génico

Técnica De Sobreexpresión Génica

Técnica De Silenciamiento Génico

The Dual Role Of Necrostatin-1 In Parkinson's Disease Models

Técnica De Silenciamiento Génico

The Dual Role Of Necrostatin-1 In Parkinson's Disease Models

Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction.

The Dual Role Of Necrostatin-1 In Parkinson's Disease Models

Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction.

The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress.

Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction.

The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress.

Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease

The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress.

Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease

Biología: Tenemos Una Buena Representación En La Base, Pero Si Asciendes En La Cúspide Va Habiendo Menos Visibilidad

Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease

Biología: Tenemos Una Buena Representación En La Base, Pero Si Asciendes En La Cúspide Va Habiendo Menos Visibilidad

Toxicity Of Necrostatin-1 In Parkinson’s Disease Models

Biología: Tenemos Una Buena Representación En La Base, Pero Si Asciendes En La Cúspide Va Habiendo Menos Visibilidad

Toxicity Of Necrostatin-1 In Parkinson’s Disease Models

Transcriptome Analysis Reveals That Fertilization With Cryopreserved Sperm Downregulates Genes Relevant For Early Embryo Development In The Horse

Toxicity Of Necrostatin-1 In Parkinson’s Disease Models

Transcriptome Analysis Reveals That Fertilization With Cryopreserved Sperm Downregulates Genes Relevant For Early Embryo Development In The Horse

Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells

Transcriptome Analysis Reveals That Fertilization With Cryopreserved Sperm Downregulates Genes Relevant For Early Embryo Development In The Horse

Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells

Natural Products In The Promotion Of Healthspan And Longevity.

Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells

Natural Products In The Promotion Of Healthspan And Longevity.

Obesity Affects Β2 Adrenergic Regulation Of The Inflammatory Profile And Phenotype Of Circulating Monocytes From Exercised Animals

Natural Products In The Promotion Of Healthspan And Longevity.

Obesity Affects Β2 Adrenergic Regulation Of The Inflammatory Profile And Phenotype Of Circulating Monocytes From Exercised Animals

Anti-Inflammatory Effect Of Β2 Adrenergic Stimulation On Circulating Monocytes With A Pro-Inflammatory State In High-Fat Diet-Induced Obesity

Obesity Affects Β2 Adrenergic Regulation Of The Inflammatory Profile And Phenotype Of Circulating Monocytes From Exercised Animals

Anti-Inflammatory Effect Of Β2 Adrenergic Stimulation On Circulating Monocytes With A Pro-Inflammatory State In High-Fat Diet-Induced Obesity

Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)

Anti-Inflammatory Effect Of Β2 Adrenergic Stimulation On Circulating Monocytes With A Pro-Inflammatory State In High-Fat Diet-Induced Obesity

Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)

Extracellular Vesicles Isolated From Porcine Seminal Plasma Exhibit Different Tetraspanin Expression Profiles

Correction To: Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry (Journal Of Molecular Medicine, (2018), 96, 10, (1061-1079), 10.1007/s00109-018-1677-Y)

Extracellular Vesicles Isolated From Porcine Seminal Plasma Exhibit Different Tetraspanin Expression Profiles

Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack

Extracellular Vesicles Isolated From Porcine Seminal Plasma Exhibit Different Tetraspanin Expression Profiles

Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack

The Paradigm Of Protein Acetylation In Parkinson's Disease

Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack

The Paradigm Of Protein Acetylation In Parkinson's Disease

Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients

The Paradigm Of Protein Acetylation In Parkinson's Disease

Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients

Erratum: Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity (Cell Metabolism (2019) 30(4) (754–767.e9), (S1550413119303833), (10.1016/j.cmet.2019.07.010))

Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients

Erratum: Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity (Cell Metabolism (2019) 30(4) (754–767.e9), (S1550413119303833), (10.1016/j.cmet.2019.07.010))

Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity

Erratum: Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity (Cell Metabolism (2019) 30(4) (754–767.e9), (S1550413119303833), (10.1016/j.cmet.2019.07.010))

Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity

Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson's Disease Patients.

Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity

Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson's Disease Patients.

Cholesterol And Multilamellar Bodies: Lysosomal Dysfunction In Gba-Parkinson Disease

Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson's Disease Patients.

Cholesterol And Multilamellar Bodies: Lysosomal Dysfunction In Gba-Parkinson Disease

Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria

Cholesterol And Multilamellar Bodies: Lysosomal Dysfunction In Gba-Parkinson Disease

Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria

Vascular Risk Factors And Lesions Of Vascular Nature In Magnetic Resonance As Predictors Of Progression To Dementia In Patients With Mild Cognitive Impairment

Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria

Vascular Risk Factors And Lesions Of Vascular Nature In Magnetic Resonance As Predictors Of Progression To Dementia In Patients With Mild Cognitive Impairment

Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science Doi: 10.1242/jcs.02878)

Vascular Risk Factors And Lesions Of Vascular Nature In Magnetic Resonance As Predictors Of Progression To Dementia In Patients With Mild Cognitive Impairment

Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science Doi: 10.1242/jcs.02878)

Erratum: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains (Journal Of Cell Science (2002) 115 (3119-3130) Doi: 10.1242/jcs.209973)

Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science Doi: 10.1242/jcs.02878)

Erratum: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains (Journal Of Cell Science (2002) 115 (3119-3130) Doi: 10.1242/jcs.209973)

Correction: Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels (Doi:10.1242/jcs.01002).

Erratum: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains (Journal Of Cell Science (2002) 115 (3119-3130) Doi: 10.1242/jcs.209973)

Correction: Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels (Doi:10.1242/jcs.01002).

Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells: In Vitro And In Vivo Studies

Correction: Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels (Doi:10.1242/jcs.01002).

Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells: In Vitro And In Vivo Studies

Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry

Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells: In Vitro And In Vivo Studies

Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry

Variation In Malaria Infection And Immune Defence In Invasive And Endemic House Sparrows

Stim1 Deficiency Is Linked To Alzheimer’s Disease And Triggers Cell Death In Sh-Sy5y Cells By Upregulation Of L-Type Voltage-Operated Ca2+ Entry

Variation In Malaria Infection And Immune Defence In Invasive And Endemic House Sparrows

Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling

Variation In Malaria Infection And Immune Defence In Invasive And Endemic House Sparrows

Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling

Centriole Planar Polarity Assessment In Drosophila Wings

Aryl Hydrocarbon Receptor Promotes Liver Polyploidization And Inhibits Pi3k, Erk, And Wnt/β-Catenin Signaling

Centriole Planar Polarity Assessment In Drosophila Wings

Acetylome In Human Fibroblasts From Parkinson’s Disease Patients

Centriole Planar Polarity Assessment In Drosophila Wings

Acetylome In Human Fibroblasts From Parkinson’s Disease Patients

Er-Mitochondria Signaling In Parkinson's Disease Review-Article

Acetylome In Human Fibroblasts From Parkinson’s Disease Patients

Er-Mitochondria Signaling In Parkinson's Disease Review-Article

[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].

Er-Mitochondria Signaling In Parkinson's Disease Review-Article

[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].

Immune-Neuroendocrine Dysregulation In Patients With Osteoarthritis: A Revision And A Pilot Study

[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].

Immune-Neuroendocrine Dysregulation In Patients With Osteoarthritis: A Revision And A Pilot Study

Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor

Immune-Neuroendocrine Dysregulation In Patients With Osteoarthritis: A Revision And A Pilot Study

Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor

Expression Of Concern: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways. J. Cell Sci. Doi: 10.1242/jcs.02878

Lung Regeneration After Toxic Injury Is Improved In Absence Of Dioxin Receptor

Expression Of Concern: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways. J. Cell Sci. Doi: 10.1242/jcs.02878

Expression Of Concern: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains. J. Cell Sci. (2002). 115, 3119-3130.

Expression Of Concern: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways. J. Cell Sci. Doi: 10.1242/jcs.02878

Expression Of Concern: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains. J. Cell Sci. (2002). 115, 3119-3130.

N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease

Expression Of Concern: Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains. J. Cell Sci. (2002). 115, 3119-3130.

N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease

Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease

N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease

Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease

Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells

Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease

Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells

Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy

Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells

Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy

Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy

Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy

Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy

Trend Of Lipid Profile In General Population From Caceres Health Area

Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy

Trend Of Lipid Profile In General Population From Caceres Health Area

The Basics Of Autophagy

Trend Of Lipid Profile In General Population From Caceres Health Area

The Basics Of Autophagy

Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice

The Basics Of Autophagy

Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice

Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor

Pirna-Associated Proteins And Retrotransposons Are Differentially Expressed In Murine Testis And Ovary Of Aryl Hydrocarbon Receptor Deficient Mice

Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor

Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?

Alu Retrotransposons Promote Differentiation Of Human Carcinoma Cells Through The Aryl Hydrocarbon Receptor

Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?

Pink1 Deficiency Enhances Autophagy And Mitophagy Induction

Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?

Pink1 Deficiency Enhances Autophagy And Mitophagy Induction

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)

Pink1 Deficiency Enhances Autophagy And Mitophagy Induction

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)

Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)

Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?

Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?

Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines

Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?

Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines

Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis

Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines

Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis

Mitochondria: Key Organelle In Parkinson's Disease

Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis

Mitochondria: Key Organelle In Parkinson's Disease

G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway

Mitochondria: Key Organelle In Parkinson's Disease

G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway

Pharmacological Inhibition Of Fatty-Acid Oxidation Synergistically Enhances The Effect Of L-Asparaginase In Childhood All Cells

G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway

Pharmacological Inhibition Of Fatty-Acid Oxidation Synergistically Enhances The Effect Of L-Asparaginase In Childhood All Cells

Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment

Pharmacological Inhibition Of Fatty-Acid Oxidation Synergistically Enhances The Effect Of L-Asparaginase In Childhood All Cells

Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment

Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis

Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment

Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis

A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells

Dioxin Receptor Regulates Aldehyde Dehydrogenase To Block Melanoma Tumorigenesis And Metastasis

A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells

Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg

A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells

Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg

Erratum To Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans [Fungal Genet. Biol. 74 (2015) 10-20] Doi: 10.1016/j.fgb.2014.10.016

Implication Of Akt, Erk1/2 And Alternative P38mapk Signalling Pathways In Human Colon Cancer Cell Apoptosis Induced By Green Tea Egcg

Erratum To Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans [Fungal Genet. Biol. 74 (2015) 10-20] Doi: 10.1016/j.fgb.2014.10.016

Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?

Erratum To Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans [Fungal Genet. Biol. 74 (2015) 10-20] Doi: 10.1016/j.fgb.2014.10.016

Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?

Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations

Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?

Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations

Control Of Autophagy In Parkinson’s Disease

Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations

Control Of Autophagy In Parkinson’s Disease

Unsaturated Fatty Acids Induce Non-Canonical Autophagy

Control Of Autophagy In Parkinson’s Disease

Unsaturated Fatty Acids Induce Non-Canonical Autophagy

Metabolomic Analyses Reveal That Anti-Aging Metabolites Are Depleted By Palmitate But Increased By Oleate In Vivo

Unsaturated Fatty Acids Induce Non-Canonical Autophagy

Metabolomic Analyses Reveal That Anti-Aging Metabolites Are Depleted By Palmitate But Increased By Oleate In Vivo

Novel Inducers Of Becn1-Independent Autophagy: Cis-Unsaturated Fatty Acids

Metabolomic Analyses Reveal That Anti-Aging Metabolites Are Depleted By Palmitate But Increased By Oleate In Vivo

Novel Inducers Of Becn1-Independent Autophagy: Cis-Unsaturated Fatty Acids

Spermidine Induces Autophagy By Inhibiting The Acetyltransferase Ep300

Novel Inducers Of Becn1-Independent Autophagy: Cis-Unsaturated Fatty Acids

Spermidine Induces Autophagy By Inhibiting The Acetyltransferase Ep300

Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach

Spermidine Induces Autophagy By Inhibiting The Acetyltransferase Ep300

Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach

Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans

Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach

Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans

Toxicity And Autophagy In Neurodegenerative Disorders

Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans

Toxicity And Autophagy In Neurodegenerative Disorders

A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells.

Toxicity And Autophagy In Neurodegenerative Disorders

A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells.

Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans.

A Mesenchymal-Like Phenotype And Expression Of Cd44 Predict Lack Of Apoptotic Response To Sorafenib In Liver Tumor Cells.

Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans.

Origen Molecular De La Compleja Enfermedad De Parkinson

Genetic Interactions Among Homologous Recombination Mutants In Candida Albicans.

Origen Molecular De La Compleja Enfermedad De Parkinson

The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol

Origen Molecular De La Compleja Enfermedad De Parkinson

The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol

Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression

The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol

Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression

G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy

Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression

G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy

Links Between Paraquat And Parkinson's Disease

G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy

Links Between Paraquat And Parkinson's Disease

Cancer Cell–autonomous Contribution Of Type I Interferon Signaling To The Efficacy Of Chemotherapy

Links Between Paraquat And Parkinson's Disease

Cancer Cell–autonomous Contribution Of Type I Interferon Signaling To The Efficacy Of Chemotherapy

Coffee Induces Autophagy In Vivo

Cancer Cell–autonomous Contribution Of Type I Interferon Signaling To The Efficacy Of Chemotherapy

Coffee Induces Autophagy In Vivo

Regulation Of Autophagy By Cytosolic Acetyl-Coenzyme A

Coffee Induces Autophagy In Vivo

Regulation Of Autophagy By Cytosolic Acetyl-Coenzyme A

Self-Consumption: The Interplay Of Autophagy And Apoptosis

Regulation Of Autophagy By Cytosolic Acetyl-Coenzyme A

Self-Consumption: The Interplay Of Autophagy And Apoptosis

Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease

Self-Consumption: The Interplay Of Autophagy And Apoptosis

Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease

The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol.

Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease

The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol.

Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1

The Dioxin Receptor Modulates Caveolin-1 Mobilization During Directional Migration: Role Of Cholesterol.

Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1

The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis

Phosphorylation Of Stim1 At Erk1/2 Target Sites Regulates Interaction With The Microtubule Plus-End Binding Protein Eb1

The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis

The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway

The Dioxin Receptor Has Tumor Suppressor Activity In Melanoma Growth And Metastasis

The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway

Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition

The Dioxin Receptor Controls Β1 Integrin Activation In Fibroblasts Through A Cbp-Csk-Src Pathway

Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition

Effect Of High Pressure Thermal Processing On Some Essential Nutrients And Immunological Components Present In Breast Milk

Dioxin Receptor Expression Inhibits Basal And Transforming Growth Factor Β-Induced Epithelial-To-Mesenchymal Transition

Effect Of High Pressure Thermal Processing On Some Essential Nutrients And Immunological Components Present In Breast Milk

Implication Of Autophagy In Parkinson's Disease

Effect Of High Pressure Thermal Processing On Some Essential Nutrients And Immunological Components Present In Breast Milk

Implication Of Autophagy In Parkinson's Disease

Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model

Implication Of Autophagy In Parkinson's Disease

Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model

The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway

Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model

The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway

Regulation Of Autophagy By Stress-Responsive Transcription Factors

The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway

Regulation Of Autophagy By Stress-Responsive Transcription Factors

Direct Interaction Between Stat3 And Eif2ak2 Controls Fatty Acid-Induced Autophagy

Regulation Of Autophagy By Stress-Responsive Transcription Factors

Direct Interaction Between Stat3 And Eif2ak2 Controls Fatty Acid-Induced Autophagy

Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer

Direct Interaction Between Stat3 And Eif2ak2 Controls Fatty Acid-Induced Autophagy

Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer

Paraquat, Between Apoptosis And Autophagy

Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer

Paraquat, Between Apoptosis And Autophagy

Autophagy And Parkinson's Disease: A Love-Hate Relationship

Paraquat, Between Apoptosis And Autophagy

Autophagy And Parkinson's Disease: A Love-Hate Relationship

Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.

Autophagy And Parkinson's Disease: A Love-Hate Relationship

Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy

Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy

Cell Fusion Reprogramming Leads To A Specific Hepatic Expression Pattern During Mouse Bone Marrow Derived Hepatocyte Formation In Vivo

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy

Cell Fusion Reprogramming Leads To A Specific Hepatic Expression Pattern During Mouse Bone Marrow Derived Hepatocyte Formation In Vivo

Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies

Cell Fusion Reprogramming Leads To A Specific Hepatic Expression Pattern During Mouse Bone Marrow Derived Hepatocyte Formation In Vivo

Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies

Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease

Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies

Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy

Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy

The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy

The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts

Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway

The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts

Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway

Autophagic Removal Of Micronuclei

Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway

Autophagic Removal Of Micronuclei

Autophagy Is Required For The Activation Of Nfκb

Autophagic Removal Of Micronuclei

Autophagy Is Required For The Activation Of Nfκb

Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))

Autophagy Is Required For The Activation Of Nfκb

Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))

Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity

Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))

Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity

Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer

Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity

Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer

Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins

Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer

Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins

An Immunosurveillance Mechanism Controls Cancer Cell Ploidy

Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins

An Immunosurveillance Mechanism Controls Cancer Cell Ploidy

270 Cd44+/epcam− Phenotype In Hepatocellular Carcinoma (Hcc) Cells Correlates With Increased Autocrine Tgf-Beta And Mesenchymal Traits

An Immunosurveillance Mechanism Controls Cancer Cell Ploidy

270 Cd44+/epcam− Phenotype In Hepatocellular Carcinoma (Hcc) Cells Correlates With Increased Autocrine Tgf-Beta And Mesenchymal Traits

Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress

270 Cd44+/epcam− Phenotype In Hepatocellular Carcinoma (Hcc) Cells Correlates With Increased Autocrine Tgf-Beta And Mesenchymal Traits

Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress

Inhibition Of Autophagy By Tab2 And Tab3

Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress

Inhibition Of Autophagy By Tab2 And Tab3

Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y

Inhibition Of Autophagy By Tab2 And Tab3

Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y

Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat

Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y

Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat

Active Commuting To School And Cognitive Performance In Adolescents: The Avena Study.

Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat

Active Commuting To School And Cognitive Performance In Adolescents: The Avena Study.

Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain

Active Commuting To School And Cognitive Performance In Adolescents: The Avena Study.

Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain

Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry

Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain

Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry

Comparative Analysis Of Carmellose 0.5% Versus Hyaluronate 0.15% In Dry Eye: A Flow Cytometric Study

Phosphorylation Of Stim1 At Erk1/2 Target Sites Modulates Store-Operated Calcium Entry

Comparative Analysis Of Carmellose 0.5% Versus Hyaluronate 0.15% In Dry Eye: A Flow Cytometric Study

The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells

Comparative Analysis Of Carmellose 0.5% Versus Hyaluronate 0.15% In Dry Eye: A Flow Cytometric Study

The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells

Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis

The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells

Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis

Dj-1 As A Modulator Of Autophagy: An Hypothesis

Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis

Dj-1 As A Modulator Of Autophagy: An Hypothesis

Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells

Dj-1 As A Modulator Of Autophagy: An Hypothesis

Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells

Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells

Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells

Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells

Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade

Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells

Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade

Nitric Oxide In Paraquat-Mediated Toxicity: A Review

Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade

Nitric Oxide In Paraquat-Mediated Toxicity: A Review

The Effect Of Preservative-Free Hp-Guar On Dry Eye After Phacoemulsification: A Flow Cytometric Study.

Nitric Oxide In Paraquat-Mediated Toxicity: A Review

The Effect Of Preservative-Free Hp-Guar On Dry Eye After Phacoemulsification: A Flow Cytometric Study.

Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species

The Effect Of Preservative-Free Hp-Guar On Dry Eye After Phacoemulsification: A Flow Cytometric Study.

Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species

Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole

Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species

Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole

Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy

Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole

Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy

Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity

Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy

Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity

Haematological Reference Values In Spanish Adolescents: The Avena Study.

Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity

Haematological Reference Values In Spanish Adolescents: The Avena Study.

Isolation And Characterization Of A Putative Liver Progenitor Population After Treatment Of Fetal Rat Hepatocytes With Tgf-Beta

Haematological Reference Values In Spanish Adolescents: The Avena Study.

Isolation And Characterization Of A Putative Liver Progenitor Population After Treatment Of Fetal Rat Hepatocytes With Tgf-Beta

Ccaat/enhancer Binding Protein Β Deficiency Provides Cerebral Protection Following Excitotoxic Injury

Isolation And Characterization Of A Putative Liver Progenitor Population After Treatment Of Fetal Rat Hepatocytes With Tgf-Beta

Ccaat/enhancer Binding Protein Β Deficiency Provides Cerebral Protection Following Excitotoxic Injury

Deletion Of The Met Tyrosine Kinase In Liver Progenitor Oval Cells Increases Sensitivity To Apoptosis In Vitro

Ccaat/enhancer Binding Protein Β Deficiency Provides Cerebral Protection Following Excitotoxic Injury

Deletion Of The Met Tyrosine Kinase In Liver Progenitor Oval Cells Increases Sensitivity To Apoptosis In Vitro

Bcl6 Represses Nfκb Activity In Diffuse Large B-Cell Lymphomas

Deletion Of The Met Tyrosine Kinase In Liver Progenitor Oval Cells Increases Sensitivity To Apoptosis In Vitro

Bcl6 Represses Nfκb Activity In Diffuse Large B-Cell Lymphomas

Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways

Bcl6 Represses Nfκb Activity In Diffuse Large B-Cell Lymphomas

Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways

Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis

Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways

Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes

Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes

Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes

Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines

Methylmalonic Acidaemia Leads To Increased Production Of Reactive Oxygen Species And Induction Of Apoptosis Through The Mitochondrial/caspase Pathway

Mechanisms Involved In Resveratrol-Induced Apoptosis And Cell Cycle Arrest In Prostate Cancer-Derived Cell Lines

Methylmalonic Acidaemia Leads To Increased Production Of Reactive Oxygen Species And Induction Of Apoptosis Through The Mitochondrial/caspase Pathway

Nitric Oxide Down-Regulates Caveolin-3 Levels Through The Interaction With Myogenin, Its Transcription Factor

Methylmalonic Acidaemia Leads To Increased Production Of Reactive Oxygen Species And Induction Of Apoptosis Through The Mitochondrial/caspase Pathway

Nitric Oxide Down-Regulates Caveolin-3 Levels Through The Interaction With Myogenin, Its Transcription Factor

P38α Mapk Can Positively Or Negatively Regulate Rac-1 Activity Depending On The Presence Of Serum

Nitric Oxide Down-Regulates Caveolin-3 Levels Through The Interaction With Myogenin, Its Transcription Factor

P38α Mapk Can Positively Or Negatively Regulate Rac-1 Activity Depending On The Presence Of Serum

Binding Of Cap70 To Inducible Nitric Oxide Synthase And Implications For The Vectorial Release Of Nitric Oxide In Polarized Cells

P38α Mapk Can Positively Or Negatively Regulate Rac-1 Activity Depending On The Presence Of Serum

Binding Of Cap70 To Inducible Nitric Oxide Synthase And Implications For The Vectorial Release Of Nitric Oxide In Polarized Cells

Mitochondrial Superoxide Mediates Heat-Induced Apoptotic-Like Death In Leishmania Infantum

Binding Of Cap70 To Inducible Nitric Oxide Synthase And Implications For The Vectorial Release Of Nitric Oxide In Polarized Cells

Mitochondrial Superoxide Mediates Heat-Induced Apoptotic-Like Death In Leishmania Infantum

Tlr4-Mediated Survival Of Macrophages Is Myd88 Dependent And Requires Tnf-Α Autocrine Signalling

Mitochondrial Superoxide Mediates Heat-Induced Apoptotic-Like Death In Leishmania Infantum

Tlr4-Mediated Survival Of Macrophages Is Myd88 Dependent And Requires Tnf-Α Autocrine Signalling

Role Of Peroxynitrite In Endothelial Damage Mediated By Cyclosporine A

Tlr4-Mediated Survival Of Macrophages Is Myd88 Dependent And Requires Tnf-Α Autocrine Signalling

Role Of Peroxynitrite In Endothelial Damage Mediated By Cyclosporine A

C-Jun N-Terminal Protein Kinase Signalling Pathway Mediates Lovastatin-Induced Rat Brain Neuroblast Apoptosis

Role Of Peroxynitrite In Endothelial Damage Mediated By Cyclosporine A

C-Jun N-Terminal Protein Kinase Signalling Pathway Mediates Lovastatin-Induced Rat Brain Neuroblast Apoptosis

Lovastatin Inhibits The Extracellular-Signal-Regulated Kinase Pathway In Immortalized Rat Brain Neuroblasts

C-Jun N-Terminal Protein Kinase Signalling Pathway Mediates Lovastatin-Induced Rat Brain Neuroblast Apoptosis

Lovastatin Inhibits The Extracellular-Signal-Regulated Kinase Pathway In Immortalized Rat Brain Neuroblasts

Negative Regulation Of Akt Activity By P38α Map Kinase In Cardiomyocytes Involves Membrane Localization Of Pp2a Through Interaction With Caveolin-1

Lovastatin Inhibits The Extracellular-Signal-Regulated Kinase Pathway In Immortalized Rat Brain Neuroblasts

Negative Regulation Of Akt Activity By P38α Map Kinase In Cardiomyocytes Involves Membrane Localization Of Pp2a Through Interaction With Caveolin-1

Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?

Negative Regulation Of Akt Activity By P38α Map Kinase In Cardiomyocytes Involves Membrane Localization Of Pp2a Through Interaction With Caveolin-1

Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?

Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells

Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?

Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells

Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy

Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells

Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy

Heat-Induced Programmed Cell Death In Leishmania Infantum Is Reverted By Bcl-Xl Expression

Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy

Heat-Induced Programmed Cell Death In Leishmania Infantum Is Reverted By Bcl-Xl Expression

A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c

Heat-Induced Programmed Cell Death In Leishmania Infantum Is Reverted By Bcl-Xl Expression

A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c

Loss Of Apoptosis-Inducing Factor Leads To An Increase In Reactive Oxygen Species, And An Impairment Of Respiration That Can Be Reversed By Antioxidants [4]

A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c

Loss Of Apoptosis-Inducing Factor Leads To An Increase In Reactive Oxygen Species, And An Impairment Of Respiration That Can Be Reversed By Antioxidants [4]

Infiltration Of Inflammatory Cells Plays An Important Role In Matrix Metalloproteinase Expression And Activation In The Heart During Sepsis

Loss Of Apoptosis-Inducing Factor Leads To An Increase In Reactive Oxygen Species, And An Impairment Of Respiration That Can Be Reversed By Antioxidants [4]

Infiltration Of Inflammatory Cells Plays An Important Role In Matrix Metalloproteinase Expression And Activation In The Heart During Sepsis

Hematopoietic Mobilization In Mice Increases The Presence Of Bone Marrow-Derived Hepatocytes Via In Vivo Cell Fusion

Infiltration Of Inflammatory Cells Plays An Important Role In Matrix Metalloproteinase Expression And Activation In The Heart During Sepsis

Hematopoietic Mobilization In Mice Increases The Presence Of Bone Marrow-Derived Hepatocytes Via In Vivo Cell Fusion

Gastrin Induces Leukocyte-Endothelial Cell Interactions In Vivo And Contributes To The Inflammation Caused By Helicobacter Pylori

Hematopoietic Mobilization In Mice Increases The Presence Of Bone Marrow-Derived Hepatocytes Via In Vivo Cell Fusion

Gastrin Induces Leukocyte-Endothelial Cell Interactions In Vivo And Contributes To The Inflammation Caused By Helicobacter Pylori

Autocrine Production Of Tgf-Β Confers Resistance To Apoptosis After An Epithelial-Mesenchymal Transition Process In Hepatocytes: Role Of Egf Receptor Ligands

Gastrin Induces Leukocyte-Endothelial Cell Interactions In Vivo And Contributes To The Inflammation Caused By Helicobacter Pylori

Autocrine Production Of Tgf-Β Confers Resistance To Apoptosis After An Epithelial-Mesenchymal Transition Process In Hepatocytes: Role Of Egf Receptor Ligands

N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways

Autocrine Production Of Tgf-Β Confers Resistance To Apoptosis After An Epithelial-Mesenchymal Transition Process In Hepatocytes: Role Of Egf Receptor Ligands

N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways

Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells

N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways

Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells

Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death

Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells

Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death

Suppression Of T-Cell Functions By Human Granulocyte Arginase

Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death

Suppression Of T-Cell Functions By Human Granulocyte Arginase

Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain

Suppression Of T-Cell Functions By Human Granulocyte Arginase

Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain

Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science (2006) Vol. 119 (1558-1596))

Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain

Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science (2006) Vol. 119 (1558-1596))

Erratum: A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c (Genes And Development (2006) 20 (307-320))

Erratum: N-Terminal Palmitoylation Within The Appropriate Amino Acid Environment Conveys On Nos2 The Ability To Progress Along The Intracellular Sorting Pathways (Journal Of Cell Science (2006) Vol. 119 (1558-1596))

Erratum: A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c (Genes And Development (2006) 20 (307-320))

Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))

Erratum: A Mechanism Of Ap-1 Suppression Through Interaction Of C-Fos With Lamin A/c (Genes And Development (2006) 20 (307-320))

Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))

Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model

Erratum: Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb (International Journal Of Cancer (2005) 115 (74-84))

Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model

Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb

Immortalized Mouse Mammary Fibroblasts Lacking Dioxin Receptor Have Impaired Tumorigenicity In A Subcutaneous Mouse Xenograft Model

Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb

Identification Of A Nuclear Export Signal In The Kshv Latent Protein Lana2 Mediating Its Export From The Nucleus

Resveratrol-Induced Apoptosis In Mcf-7 Human Breast Cancer Cells Involves A Caspase-Independent Mechanism With Downregulation Of Bcl-2 And Nf-Κb

Identification Of A Nuclear Export Signal In The Kshv Latent Protein Lana2 Mediating Its Export From The Nucleus

Discrepancies Between Nitroglycerin And No-Releasing Drugs On Mitochondrial Oxygen Consumption, Vasoactivity, And The Release Of No

Identification Of A Nuclear Export Signal In The Kshv Latent Protein Lana2 Mediating Its Export From The Nucleus

Discrepancies Between Nitroglycerin And No-Releasing Drugs On Mitochondrial Oxygen Consumption, Vasoactivity, And The Release Of No

Direct Interaction Between The Reductase Domain Of Endothelial Nitric Oxide Synthase And The Ryanodine Receptor

Discrepancies Between Nitroglycerin And No-Releasing Drugs On Mitochondrial Oxygen Consumption, Vasoactivity, And The Release Of No

Direct Interaction Between The Reductase Domain Of Endothelial Nitric Oxide Synthase And The Ryanodine Receptor

Pgc-1α Regulates The Mitochondrial Antioxidant Defense System In Vascular Endothelial Cells

Direct Interaction Between The Reductase Domain Of Endothelial Nitric Oxide Synthase And The Ryanodine Receptor

Pgc-1α Regulates The Mitochondrial Antioxidant Defense System In Vascular Endothelial Cells

Matrix Metalloproteinase 13 Mediates Nitric Oxide Activation Of Endothelial Cell Migration

Pgc-1α Regulates The Mitochondrial Antioxidant Defense System In Vascular Endothelial Cells

Matrix Metalloproteinase 13 Mediates Nitric Oxide Activation Of Endothelial Cell Migration

Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity

Matrix Metalloproteinase 13 Mediates Nitric Oxide Activation Of Endothelial Cell Migration

Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity

Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo

Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity

Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo

Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity

Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo

Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity

Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor

Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity

Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor

The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death

Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor

The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death

Inhibition Of Macroautophagy Triggers Apoptosis

The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death

Inhibition Of Macroautophagy Triggers Apoptosis

Synthesis Of Nitric Oxide In Postganglionic Myenteric Neurons During Endotoxemia: Implications For Gastric Motor Function In Rats.

Inhibition Of Macroautophagy Triggers Apoptosis

Synthesis Of Nitric Oxide In Postganglionic Myenteric Neurons During Endotoxemia: Implications For Gastric Motor Function In Rats.

Source Of Early Reactive Oxygen Species In The Apoptosis Induced By Transforming Growth Factor-Β In Fetal Rat Hepatocytes

Synthesis Of Nitric Oxide In Postganglionic Myenteric Neurons During Endotoxemia: Implications For Gastric Motor Function In Rats.

Source Of Early Reactive Oxygen Species In The Apoptosis Induced By Transforming Growth Factor-Β In Fetal Rat Hepatocytes

Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells

Source Of Early Reactive Oxygen Species In The Apoptosis Induced By Transforming Growth Factor-Β In Fetal Rat Hepatocytes

Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells

Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants

Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells

Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants

Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells

Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants

Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells

Viral Proteins Targeting Mitochondria: Controlling Cell Death

Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells

Viral Proteins Targeting Mitochondria: Controlling Cell Death

Resistance To Tgf-Beta-Induced Apoptosis In Regenerating Hepatocytes.

Viral Proteins Targeting Mitochondria: Controlling Cell Death

Resistance To Tgf-Beta-Induced Apoptosis In Regenerating Hepatocytes.

Palmitoylation Of Inducible Nitric-Oxide Synthase At Cys-3 Is Required For Proper Intracellular Traffic And Nitric Oxide Synthesis.

Resistance To Tgf-Beta-Induced Apoptosis In Regenerating Hepatocytes.

Palmitoylation Of Inducible Nitric-Oxide Synthase At Cys-3 Is Required For Proper Intracellular Traffic And Nitric Oxide Synthesis.

The Effect Of Milk Fermented By Yogurt Cultures Plus Lactobacillus Casei Dn-114001 On The Immune Response Of Subjects Under Academic Examination Stress.

Palmitoylation Of Inducible Nitric-Oxide Synthase At Cys-3 Is Required For Proper Intracellular Traffic And Nitric Oxide Synthesis.

The Effect Of Milk Fermented By Yogurt Cultures Plus Lactobacillus Casei Dn-114001 On The Immune Response Of Subjects Under Academic Examination Stress.

Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels.

The Effect Of Milk Fermented By Yogurt Cultures Plus Lactobacillus Casei Dn-114001 On The Immune Response Of Subjects Under Academic Examination Stress.

Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels.

P38 Alpha Mitogen-Activated Protein Kinase Sensitizes Cells To Apoptosis Induced By Different Stimuli.

Induction Of Nitric Oxide Synthase-2 Proceeds With The Concomitant Downregulation Of The Endogenous Caveolin Levels.

P38 Alpha Mitogen-Activated Protein Kinase Sensitizes Cells To Apoptosis Induced By Different Stimuli.

Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells

P38 Alpha Mitogen-Activated Protein Kinase Sensitizes Cells To Apoptosis Induced By Different Stimuli.

Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells

Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells

Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells

Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells

The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family

Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells

The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family

Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine

The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family

Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine

Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin

Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine

Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin

Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase

Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin

Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase

Confocal Visualization Of Nitric Oxide In The Myenteric Plexus Of The Gastric Fundus Obtained From Endotoxin-Treated Rats

Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase

Confocal Visualization Of Nitric Oxide In The Myenteric Plexus Of The Gastric Fundus Obtained From Endotoxin-Treated Rats

Long-Term Treatment With Insulin Induces Apoptosis In Brown Adipocytes: Role Of Oxidative Stress.

Confocal Visualization Of Nitric Oxide In The Myenteric Plexus Of The Gastric Fundus Obtained From Endotoxin-Treated Rats

Long-Term Treatment With Insulin Induces Apoptosis In Brown Adipocytes: Role Of Oxidative Stress.

Sustained Nitric Oxide Delivery Delays Nitric Oxide-Dependent Apoptosis In Macrophages: Contribution To The Physiological Function Of Activated Macrophages.

Long-Term Treatment With Insulin Induces Apoptosis In Brown Adipocytes: Role Of Oxidative Stress.

Sustained Nitric Oxide Delivery Delays Nitric Oxide-Dependent Apoptosis In Macrophages: Contribution To The Physiological Function Of Activated Macrophages.

The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle

Sustained Nitric Oxide Delivery Delays Nitric Oxide-Dependent Apoptosis In Macrophages: Contribution To The Physiological Function Of Activated Macrophages.

The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle

Evidence For Discoordinate Regulation Of The Hla-Dpb1 Gene

The Antiproliferative Activity Of Resveratrol Results In Apoptosis In Mcf-7 But Not In Mda-Mb-231 Human Breast Cancer Cells: Cell-Specific Alteration Of The Cell Cycle

Evidence For Discoordinate Regulation Of The Hla-Dpb1 Gene

Tace/adam17-Tnf-Α Pathway In Rat Cortical Cultures After Exposure To Oxygen-Glucose Deprivation Or Glutamate

Evidence For Discoordinate Regulation Of The Hla-Dpb1 Gene

Tace/adam17-Tnf-Α Pathway In Rat Cortical Cultures After Exposure To Oxygen-Glucose Deprivation Or Glutamate

Diagnostic Performance Of Arginase Activity In Colorectal Cancer

Tace/adam17-Tnf-Α Pathway In Rat Cortical Cultures After Exposure To Oxygen-Glucose Deprivation Or Glutamate

Diagnostic Performance Of Arginase Activity In Colorectal Cancer

Aplidin Induces The Mitochondrial Apoptotic Pathway Via Oxidative Stress-Mediated Jnk And P38 Activation And Protein Kinase C Delta.

Diagnostic Performance Of Arginase Activity In Colorectal Cancer

Aplidin Induces The Mitochondrial Apoptotic Pathway Via Oxidative Stress-Mediated Jnk And P38 Activation And Protein Kinase C Delta.

Differential Role Of Ppar Gamma In The Regulation Of Ucp-1 And Adipogenesis By Tnf-Alpha In Brown Adipocytes.

Aplidin Induces The Mitochondrial Apoptotic Pathway Via Oxidative Stress-Mediated Jnk And P38 Activation And Protein Kinase C Delta.

Differential Role Of Ppar Gamma In The Regulation Of Ucp-1 And Adipogenesis By Tnf-Alpha In Brown Adipocytes.

Induction Of Apoptosis By Nitric Oxide In Macrophages Is Independent Of Apoptotic Volume Decrease.

Differential Role Of Ppar Gamma In The Regulation Of Ucp-1 And Adipogenesis By Tnf-Alpha In Brown Adipocytes.

Induction Of Apoptosis By Nitric Oxide In Macrophages Is Independent Of Apoptotic Volume Decrease.

In Vitro Fertilizing Capacity And Chromatin Condensation Of Deep Frozen Boar Semen Packaged In 0.5 And 5 Ml Straws.

Induction Of Apoptosis By Nitric Oxide In Macrophages Is Independent Of Apoptotic Volume Decrease.

In Vitro Fertilizing Capacity And Chromatin Condensation Of Deep Frozen Boar Semen Packaged In 0.5 And 5 Ml Straws.

Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains

In Vitro Fertilizing Capacity And Chromatin Condensation Of Deep Frozen Boar Semen Packaged In 0.5 And 5 Ml Straws.

Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains

Akt1/pkbalpha Protects Pc12 Cells Against The Parkinsonism-Inducing Neurotoxin 1-Methyl-4-Phenylpyridinium And Reduces The Levels Of Oxygen-Free Radicals.

Distance-Dependent Cellular Palmitoylation Of De-Novo-Designed Sequences And Their Translocation To Plasma Membrane Subdomains

Akt1/pkbalpha Protects Pc12 Cells Against The Parkinsonism-Inducing Neurotoxin 1-Methyl-4-Phenylpyridinium And Reduces The Levels Of Oxygen-Free Radicals.

Use Of Flow Cytometry And Confocal Microscopy Techniques To Investigate Early Cdcl2-Induced Nephrotoxicity In Vitro

Akt1/pkbalpha Protects Pc12 Cells Against The Parkinsonism-Inducing Neurotoxin 1-Methyl-4-Phenylpyridinium And Reduces The Levels Of Oxygen-Free Radicals.

Use Of Flow Cytometry And Confocal Microscopy Techniques To Investigate Early Cdcl2-Induced Nephrotoxicity In Vitro

Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells

Use Of Flow Cytometry And Confocal Microscopy Techniques To Investigate Early Cdcl2-Induced Nephrotoxicity In Vitro

Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells

Lovastatin Induces Apoptosis Of Spontaneously Immortalized Rat Brain Neuroblasts: Involvement Of Nonsterol Isoprenoid Biosynthesis Inhibition

Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells

Lovastatin Induces Apoptosis Of Spontaneously Immortalized Rat Brain Neuroblasts: Involvement Of Nonsterol Isoprenoid Biosynthesis Inhibition

Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism

Lovastatin Induces Apoptosis Of Spontaneously Immortalized Rat Brain Neuroblasts: Involvement Of Nonsterol Isoprenoid Biosynthesis Inhibition

Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism

Intracellular Water Motion Decreases In Apoptotic Macrophages After Caspase Activation.

Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism

Intracellular Water Motion Decreases In Apoptotic Macrophages After Caspase Activation.

Activation Of Caspases Occurs Downstream From Radical Oxygen Species Production, Bcl-Xl Down-Regulation, And Early Cytochrome C Release In Apoptosis Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes.

Intracellular Water Motion Decreases In Apoptotic Macrophages After Caspase Activation.

Activation Of Caspases Occurs Downstream From Radical Oxygen Species Production, Bcl-Xl Down-Regulation, And Early Cytochrome C Release In Apoptosis Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes.

Reactive Oxygen Species (Ros) Mediates The Mitochondrial-Dependent Apoptosis Induced By Transforming Growth Factor (Beta) In Fetal Hepatocytes.

Activation Of Caspases Occurs Downstream From Radical Oxygen Species Production, Bcl-Xl Down-Regulation, And Early Cytochrome C Release In Apoptosis Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes.

Reactive Oxygen Species (Ros) Mediates The Mitochondrial-Dependent Apoptosis Induced By Transforming Growth Factor (Beta) In Fetal Hepatocytes.

Mitochondrial Involvement In Cocaine-Treated Rat Hepatocytes: Effect Of N-Acetylcysteine And Deferoxamine.

Reactive Oxygen Species (Ros) Mediates The Mitochondrial-Dependent Apoptosis Induced By Transforming Growth Factor (Beta) In Fetal Hepatocytes.

Mitochondrial Involvement In Cocaine-Treated Rat Hepatocytes: Effect Of N-Acetylcysteine And Deferoxamine.

Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells

Mitochondrial Involvement In Cocaine-Treated Rat Hepatocytes: Effect Of N-Acetylcysteine And Deferoxamine.

Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells

Effect Of N-Acetylcysteine And Deferoxamine On Endogenous Antioxidant Defense System Gene Expression In A Rat Hepatocyte Model Of Cocaine Cytotoxicity.

Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells

Effect Of N-Acetylcysteine And Deferoxamine On Endogenous Antioxidant Defense System Gene Expression In A Rat Hepatocyte Model Of Cocaine Cytotoxicity.

Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase

Effect Of N-Acetylcysteine And Deferoxamine On Endogenous Antioxidant Defense System Gene Expression In A Rat Hepatocyte Model Of Cocaine Cytotoxicity.

Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase

Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.

Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase

Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.

Fibronectin Regulates Morphology, Cell Organization And Gene Expression Of Rat Fetal Hepatocytes In Primary Culture.

Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.

Fibronectin Regulates Morphology, Cell Organization And Gene Expression Of Rat Fetal Hepatocytes In Primary Culture.

Inhibition Of Pkb/akt1 By C2-Ceramide Involves Activation Of Ceramide-Activated Protein Phosphatase In Pc12 Cells.

Fibronectin Regulates Morphology, Cell Organization And Gene Expression Of Rat Fetal Hepatocytes In Primary Culture.

Inhibition Of Pkb/akt1 By C2-Ceramide Involves Activation Of Ceramide-Activated Protein Phosphatase In Pc12 Cells.

P38 Mitogen-Activated Protein Kinase Mediates Tumor Necrosis Factor-Alpha-Induced Apoptosis In Rat Fetal Brown Adipocytes.

Inhibition Of Pkb/akt1 By C2-Ceramide Involves Activation Of Ceramide-Activated Protein Phosphatase In Pc12 Cells.

P38 Mitogen-Activated Protein Kinase Mediates Tumor Necrosis Factor-Alpha-Induced Apoptosis In Rat Fetal Brown Adipocytes.

Noradrenaline Induces Brown Adipocytes Cell Growth Via Beta-Receptors By A Mechanism Dependent On Erks But Independent Of Camp And Pka.

P38 Mitogen-Activated Protein Kinase Mediates Tumor Necrosis Factor-Alpha-Induced Apoptosis In Rat Fetal Brown Adipocytes.

Noradrenaline Induces Brown Adipocytes Cell Growth Via Beta-Receptors By A Mechanism Dependent On Erks But Independent Of Camp And Pka.

Epidermal Growth Factor Impairs The Cytochrome C/caspase-3 Apoptotic Pathway Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes Via A Phosphoinositide 3-Kinase-Dependent Pathway.

Noradrenaline Induces Brown Adipocytes Cell Growth Via Beta-Receptors By A Mechanism Dependent On Erks But Independent Of Camp And Pka.

Epidermal Growth Factor Impairs The Cytochrome C/caspase-3 Apoptotic Pathway Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes Via A Phosphoinositide 3-Kinase-Dependent Pathway.

Changes In Antioxidant Defence Systems Induced By Cyclosporine A In Cultures Of Hepatocytes From 2- And 12-Month-Old Rats.

Epidermal Growth Factor Impairs The Cytochrome C/caspase-3 Apoptotic Pathway Induced By Transforming Growth Factor Beta In Rat Fetal Hepatocytes Via A Phosphoinositide 3-Kinase-Dependent Pathway.

Changes In Antioxidant Defence Systems Induced By Cyclosporine A In Cultures Of Hepatocytes From 2- And 12-Month-Old Rats.

Contribution Of Cyclopentenone Prostaglandins To The Resolution Of Inflammation Through The Potentiation Of Apoptosis In Activated Macrophages.

Changes In Antioxidant Defence Systems Induced By Cyclosporine A In Cultures Of Hepatocytes From 2- And 12-Month-Old Rats.

Contribution Of Cyclopentenone Prostaglandins To The Resolution Of Inflammation Through The Potentiation Of Apoptosis In Activated Macrophages.

Hsp70 Induction By Cyclosporine A In Cultured Rat Hepatocytes: Effect Of Vitamin E Succinate.

Contribution Of Cyclopentenone Prostaglandins To The Resolution Of Inflammation Through The Potentiation Of Apoptosis In Activated Macrophages.

Hsp70 Induction By Cyclosporine A In Cultured Rat Hepatocytes: Effect Of Vitamin E Succinate.

Applications Of Confocal Mycroscopy To The Paper Industry: Slime And Deposit Examples

Hsp70 Induction By Cyclosporine A In Cultured Rat Hepatocytes: Effect Of Vitamin E Succinate.

Applications Of Confocal Mycroscopy To The Paper Industry: Slime And Deposit Examples

Applications Of Flow Cytometry To Clinical Microbiology

Applications Of Confocal Mycroscopy To The Paper Industry: Slime And Deposit Examples

Applications Of Flow Cytometry To Clinical Microbiology

Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+

Applications Of Flow Cytometry To Clinical Microbiology

Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+

Age-Related Changes On Parameters Of Experimentally-Induced Liver Injury And Regeneration.

Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+

Age-Related Changes On Parameters Of Experimentally-Induced Liver Injury And Regeneration.

Apoptotic Response To Tgf-Beta In Fetal Hepatocytes Depends Upon Their State Of Differentiation.

Age-Related Changes On Parameters Of Experimentally-Induced Liver Injury And Regeneration.

Apoptotic Response To Tgf-Beta In Fetal Hepatocytes Depends Upon Their State Of Differentiation.

Nonviral Transfer Of Genes To Pig Primary Keratinocytes. Induction Of Angiogenesis By Composite Grafts Of Modified Keratinocytes Overexpressing Vegf Driven By A Keratin Promoter.

Apoptotic Response To Tgf-Beta In Fetal Hepatocytes Depends Upon Their State Of Differentiation.

Nonviral Transfer Of Genes To Pig Primary Keratinocytes. Induction Of Angiogenesis By Composite Grafts Of Modified Keratinocytes Overexpressing Vegf Driven By A Keratin Promoter.

Cocaine Cytotoxicity In Hepatocyte Cultures From Phenobarbital-Induced Rats: Involvement Of Reactive Oxygen Species And Expression Of Antioxidant Defense Systems.

Nonviral Transfer Of Genes To Pig Primary Keratinocytes. Induction Of Angiogenesis By Composite Grafts Of Modified Keratinocytes Overexpressing Vegf Driven By A Keratin Promoter.

Cocaine Cytotoxicity In Hepatocyte Cultures From Phenobarbital-Induced Rats: Involvement Of Reactive Oxygen Species And Expression Of Antioxidant Defense Systems.

Nitric Oxide Induces Tyrosine Nitration And Release Of Cytochrome C Preceding An Increase Of Mitochondrial Transmembrane Potential In Macrophages.

Cocaine Cytotoxicity In Hepatocyte Cultures From Phenobarbital-Induced Rats: Involvement Of Reactive Oxygen Species And Expression Of Antioxidant Defense Systems.

Nitric Oxide Induces Tyrosine Nitration And Release Of Cytochrome C Preceding An Increase Of Mitochondrial Transmembrane Potential In Macrophages.

Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons

Nitric Oxide Induces Tyrosine Nitration And Release Of Cytochrome C Preceding An Increase Of Mitochondrial Transmembrane Potential In Macrophages.

Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons

The Effect Of Non-Genotoxic Carcinogens, Phenobarbital And Clofibrate, On The Relationship Between Reactive Oxygen Species, Antioxidant Enzyme Expression And Apoptosis.

Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons

The Effect Of Non-Genotoxic Carcinogens, Phenobarbital And Clofibrate, On The Relationship Between Reactive Oxygen Species, Antioxidant Enzyme Expression And Apoptosis.

Influence Of Aminoguanidine On Parameters Of Liver Injury And Regeneration Induced In Rats By A Necrogenic Dose Of Thioacetamide.

The Effect Of Non-Genotoxic Carcinogens, Phenobarbital And Clofibrate, On The Relationship Between Reactive Oxygen Species, Antioxidant Enzyme Expression And Apoptosis.

Influence Of Aminoguanidine On Parameters Of Liver Injury And Regeneration Induced In Rats By A Necrogenic Dose Of Thioacetamide.

Hla-Dpbeta Residue 69 Plays A Crucial Role In Allorecognition.

Influence Of Aminoguanidine On Parameters Of Liver Injury And Regeneration Induced In Rats By A Necrogenic Dose Of Thioacetamide.

Hla-Dpbeta Residue 69 Plays A Crucial Role In Allorecognition.

Transforming Growth Factor-Beta (Tgf-Beta) And Egf Promote Cord-Like Structures That Indicate Terminal Differentiation Of Fetal Hepatocytes In Primary Culture.

Hla-Dpbeta Residue 69 Plays A Crucial Role In Allorecognition.

Transforming Growth Factor-Beta (Tgf-Beta) And Egf Promote Cord-Like Structures That Indicate Terminal Differentiation Of Fetal Hepatocytes In Primary Culture.

P42/p44 Mitogen-Activated Protein Kinases Activation Is Required For The Insulin-Like Growth Factor-I/insulin Induced Proliferation, But Inhibits Differentiation, In Rat Fetal Brown Adipocytes.

Transforming Growth Factor-Beta (Tgf-Beta) And Egf Promote Cord-Like Structures That Indicate Terminal Differentiation Of Fetal Hepatocytes In Primary Culture.

P42/p44 Mitogen-Activated Protein Kinases Activation Is Required For The Insulin-Like Growth Factor-I/insulin Induced Proliferation, But Inhibits Differentiation, In Rat Fetal Brown Adipocytes.

Flow Cytometric Studies Of Nucleoside Transport Regulation In Single Chromaffin Cells.

P42/p44 Mitogen-Activated Protein Kinases Activation Is Required For The Insulin-Like Growth Factor-I/insulin Induced Proliferation, But Inhibits Differentiation, In Rat Fetal Brown Adipocytes.

Flow Cytometric Studies Of Nucleoside Transport Regulation In Single Chromaffin Cells.

Necrogenic And Regenerative Responses Of Liver Of Newly Weaned Rats Against A Sublethal Dose Of Thioacetamide.

Flow Cytometric Studies Of Nucleoside Transport Regulation In Single Chromaffin Cells.

Necrogenic And Regenerative Responses Of Liver Of Newly Weaned Rats Against A Sublethal Dose Of Thioacetamide.

Microbiological Induced Corrosion In The Paper Industry

Necrogenic And Regenerative Responses Of Liver Of Newly Weaned Rats Against A Sublethal Dose Of Thioacetamide.

Microbiological Induced Corrosion In The Paper Industry

Tnf-Alpha Induces Apoptosis In Rat Fetal Brown Adipocytes In Primary Culture.

Microbiological Induced Corrosion In The Paper Industry

Tnf-Alpha Induces Apoptosis In Rat Fetal Brown Adipocytes In Primary Culture.

Cycloheximide Prevents Apoptosis, Reactive Oxygen Species Production, And Glutathione Depletion Induced By Transforming Growth Factor Beta In Fetal Rat Hepatocytes In Primary Culture.

Tnf-Alpha Induces Apoptosis In Rat Fetal Brown Adipocytes In Primary Culture.

Cycloheximide Prevents Apoptosis, Reactive Oxygen Species Production, And Glutathione Depletion Induced By Transforming Growth Factor Beta In Fetal Rat Hepatocytes In Primary Culture.

Neuronal Mitochondrial Morphology And Transmembrane Potential Are Severely Altered By Hypothyroidism During Rat Brain Development.

Cycloheximide Prevents Apoptosis, Reactive Oxygen Species Production, And Glutathione Depletion Induced By Transforming Growth Factor Beta In Fetal Rat Hepatocytes In Primary Culture.

Neuronal Mitochondrial Morphology And Transmembrane Potential Are Severely Altered By Hypothyroidism During Rat Brain Development.

Age-Dependent Modifications In Rat Hepatocyte Antioxidant Defense Systems.

Neuronal Mitochondrial Morphology And Transmembrane Potential Are Severely Altered By Hypothyroidism During Rat Brain Development.

Age-Dependent Modifications In Rat Hepatocyte Antioxidant Defense Systems.

Rapid Up-Regulation Of Ikappabbeta And Abrogation Of Nf-Kappab Activity In Peritoneal Macrophages Stimulated With Lipopolysaccharide.

Age-Dependent Modifications In Rat Hepatocyte Antioxidant Defense Systems.

Rapid Up-Regulation Of Ikappabbeta And Abrogation Of Nf-Kappab Activity In Peritoneal Macrophages Stimulated With Lipopolysaccharide.

Phenotypic Characterization Of A Candida Albicans Strain Deficient In Its Major Exoglucanase.

Rapid Up-Regulation Of Ikappabbeta And Abrogation Of Nf-Kappab Activity In Peritoneal Macrophages Stimulated With Lipopolysaccharide.

Phenotypic Characterization Of A Candida Albicans Strain Deficient In Its Major Exoglucanase.

Epidermal Growth Factor, But Not Hepatocyte Growth Factor, Suppresses The Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocytes In Primary Culture.

Phenotypic Characterization Of A Candida Albicans Strain Deficient In Its Major Exoglucanase.

Epidermal Growth Factor, But Not Hepatocyte Growth Factor, Suppresses The Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocytes In Primary Culture.

Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocyte Primary Cultures: Involvement Of Reactive Oxygen Intermediates.

Epidermal Growth Factor, But Not Hepatocyte Growth Factor, Suppresses The Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocytes In Primary Culture.

Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocyte Primary Cultures: Involvement Of Reactive Oxygen Intermediates.

Transforming Growth Factor Beta Modulates Growth And Differentiation Of Fetal Hepatocytes In Primary Culture.

Apoptosis Induced By Transforming Growth Factor-Beta In Fetal Hepatocyte Primary Cultures: Involvement Of Reactive Oxygen Intermediates.

Transforming Growth Factor Beta Modulates Growth And Differentiation Of Fetal Hepatocytes In Primary Culture.

Differentiation Of Rat Brown Adipocytes During Late Foetal Development: Role Of Insulin-Like Growth Factor I.

Transforming Growth Factor Beta Modulates Growth And Differentiation Of Fetal Hepatocytes In Primary Culture.

Differentiation Of Rat Brown Adipocytes During Late Foetal Development: Role Of Insulin-Like Growth Factor I.

The Role Of Hla-Dp Beta Residue 69 In The Definition Of Antibody-Binding Epitopes.

Differentiation Of Rat Brown Adipocytes During Late Foetal Development: Role Of Insulin-Like Growth Factor I.

The Role Of Hla-Dp Beta Residue 69 In The Definition Of Antibody-Binding Epitopes.

Relationship Between Antioxidant Systems, Intracellular Thiols And Dna Ploidy In Liver Of Rats During Experimental Cirrhogenesis.

The Role Of Hla-Dp Beta Residue 69 In The Definition Of Antibody-Binding Epitopes.

Relationship Between Antioxidant Systems, Intracellular Thiols And Dna Ploidy In Liver Of Rats During Experimental Cirrhogenesis.

Splenic B Lymphocyte Programmed Cell Death Is Prevented By Nitric Oxide Release Through Mechanisms Involving Sustained Bcl-2 Levels.

Relationship Between Antioxidant Systems, Intracellular Thiols And Dna Ploidy In Liver Of Rats During Experimental Cirrhogenesis.

Splenic B Lymphocyte Programmed Cell Death Is Prevented By Nitric Oxide Release Through Mechanisms Involving Sustained Bcl-2 Levels.

Two Different Subpopulations Of Ig-Bearing Cells In Lymphoid Organs Of Rainbow Trout.

Splenic B Lymphocyte Programmed Cell Death Is Prevented By Nitric Oxide Release Through Mechanisms Involving Sustained Bcl-2 Levels.

Two Different Subpopulations Of Ig-Bearing Cells In Lymphoid Organs Of Rainbow Trout.

Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase

Two Different Subpopulations Of Ig-Bearing Cells In Lymphoid Organs Of Rainbow Trout.

Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase

Cocaine-Induced Liver Injury In Mice Elicits Specific Changes In Dna Ploidy And Induces Programmed Death Of Hepatocytes

Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase

Cocaine-Induced Liver Injury In Mice Elicits Specific Changes In Dna Ploidy And Induces Programmed Death Of Hepatocytes

Yeast Exo-Beta-Glucanases Can Be Used As Efficient And Readily Detectable Reporter Genes In Saccharomyces Cerevisiae.

Cocaine-Induced Liver Injury In Mice Elicits Specific Changes In Dna Ploidy And Induces Programmed Death Of Hepatocytes

Yeast Exo-Beta-Glucanases Can Be Used As Efficient And Readily Detectable Reporter Genes In Saccharomyces Cerevisiae.

Molecular Analysis Of An Hla-Dp Mutant Cell Line Selected For Its Resistance To Killing By Hla-Dpw2-Specific T-Cell Clones.

Yeast Exo-Beta-Glucanases Can Be Used As Efficient And Readily Detectable Reporter Genes In Saccharomyces Cerevisiae.

Molecular Analysis Of An Hla-Dp Mutant Cell Line Selected For Its Resistance To Killing By Hla-Dpw2-Specific T-Cell Clones.

The Immune System And Autoaggression

Molecular Analysis Of An Hla-Dp Mutant Cell Line Selected For Its Resistance To Killing By Hla-Dpw2-Specific T-Cell Clones.

The Immune System And Autoaggression

Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle

The Immune System And Autoaggression

Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle

Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase

Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle

Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase

Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase

Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase

Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase

Relationship Between Genomic Dna Ploidy And Parameters Of Liver Damage During Necrosis And Regeneration Induced By Thioacetamide

Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase

Relationship Between Genomic Dna Ploidy And Parameters Of Liver Damage During Necrosis And Regeneration Induced By Thioacetamide

Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación

Relationship Between Genomic Dna Ploidy And Parameters Of Liver Damage During Necrosis And Regeneration Induced By Thioacetamide

Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación

Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages

Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación

Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages

Differential Proliferative Response Of Cultured Fetal And Regenerating Hepatocytes To Growth Factors And Hormones.

Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages

Differential Proliferative Response Of Cultured Fetal And Regenerating Hepatocytes To Growth Factors And Hormones.

Flow Cytometric Analysis Of African Swine Fever Virus-Induced Plasma Membrane Proteins And Their Humoral Immune Response In Infected Pigs.

Differential Proliferative Response Of Cultured Fetal And Regenerating Hepatocytes To Growth Factors And Hormones.

Flow Cytometric Analysis Of African Swine Fever Virus-Induced Plasma Membrane Proteins And Their Humoral Immune Response In Infected Pigs.

Flow Cytometric Analysis Of Saccharomyces Cerevisiae Autolytic Mutants And Protoplasts.

Flow Cytometric Analysis Of African Swine Fever Virus-Induced Plasma Membrane Proteins And Their Humoral Immune Response In Infected Pigs.

Flow Cytometric Analysis Of Saccharomyces Cerevisiae Autolytic Mutants And Protoplasts.

Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp

Flow Cytometric Analysis Of Saccharomyces Cerevisiae Autolytic Mutants And Protoplasts.

Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp

An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)

Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp

An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)

Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.

An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)

Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.

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Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.

Not Available

Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)

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Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)

Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice