PUBLICACIÓN

Mitochondrial membrane permeabilization is a critical step of lysosome-initiated apoptosis induced by hydroxychloroquine

ACCEDER A LA PUBLICACIÓN: Scopus Orcid

2003 Oncogene

CITAS

361

DOI

10.1038/sj.onc.1206622

EID

2-s2.0-0038677510

ISSN

0950-9232

BIBTEX

@article{boya_mitochondrial_2003, title= {Mitochondrial membrane permeabilization is a critical step of lysosome-initiated apoptosis induced by hydroxychloroquine}, volume= {22}, url= {https://www.scopus.com/inward/record.uri?eid=2-s2.0-0038677510&doi=10.1038%2fsj.onc.1206622&partnerID=40&md5=356138bf34123d13d22bb321eef81355}, doi= {10.1038/sj.onc.1206622}, number= {25}, journal= {Oncogene}, author= {Boya, P. and Gonzalez-Polo, R.-A. and Poncet, D. and Andreau, K. and Vieira, H.L.A. and Roumier, T. and Perfettini, J.-L. and Kroemer, G.}, year= {2003}, pages= {3927--3936}}

AUTORES DE LA UEX

Rosa Ana González Polo

OTRAS PUBLICACIONES DE INTERÉS

Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects
Links Between Paraquat And Parkinson’s Disease
Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects
Links Between Paraquat And Parkinson’s Disease
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction
Links Between Paraquat And Parkinson’s Disease
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction
Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction
Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress
Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress
Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress
Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1
Cultivos Celulares
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1
Cultivos Celulares
Microscopía Electrónica
Cultivos Celulares
Microscopía Electrónica
Análisis De Extracto Proteico Por Western Blotting
Microscopía Electrónica
Análisis De Extracto Proteico Por Western Blotting
Extracción De Proteínas
Análisis De Extracto Proteico Por Western Blotting
Extracción De Proteínas
Técnica De Sobreexpresión Génica
Extracción De Proteínas
Técnica De Sobreexpresión Génica
Técnica De Silenciamiento Génico
Técnica De Sobreexpresión Génica
Técnica De Silenciamiento Génico
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress.
Técnica De Silenciamiento Génico
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress.
Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress.
Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease
Biología: Tenemos Una Buena Representación En La Base, Pero Si Asciendes En La Cúspide Va Habiendo Menos Visibilidad
Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease
Biología: Tenemos Una Buena Representación En La Base, Pero Si Asciendes En La Cúspide Va Habiendo Menos Visibilidad
Toxicity Of Necrostatin-1 In Parkinson’s Disease Models
Biología: Tenemos Una Buena Representación En La Base, Pero Si Asciendes En La Cúspide Va Habiendo Menos Visibilidad
Toxicity Of Necrostatin-1 In Parkinson’s Disease Models
Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack
Toxicity Of Necrostatin-1 In Parkinson’s Disease Models
Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack
The Paradigm Of Protein Acetylation In Parkinson's Disease
Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack
The Paradigm Of Protein Acetylation In Parkinson's Disease
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients
The Paradigm Of Protein Acetylation In Parkinson's Disease
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients
Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients
Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells
Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells
Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis
Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis
Mitochondria: Key Organelle In Parkinson's Disease
Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis
Mitochondria: Key Organelle In Parkinson's Disease
G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway
Mitochondria: Key Organelle In Parkinson's Disease
G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Control Of Autophagy In Parkinson’s Disease
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Control Of Autophagy In Parkinson’s Disease
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
Control Of Autophagy In Parkinson’s Disease
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Links Between Paraquat And Parkinson's Disease
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Links Between Paraquat And Parkinson's Disease
Implication Of Autophagy In Parkinson's Disease
Links Between Paraquat And Parkinson's Disease
Implication Of Autophagy In Parkinson's Disease
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
Implication Of Autophagy In Parkinson's Disease
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Autophagy And Parkinson's Disease: A Love-Hate Relationship
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor
The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death
Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor
The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death
Inhibition Of Macroautophagy Triggers Apoptosis
The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death
Inhibition Of Macroautophagy Triggers Apoptosis
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Inhibition Of Macroautophagy Triggers Apoptosis
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Viral Proteins Targeting Mitochondria: Controlling Cell Death
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Viral Proteins Targeting Mitochondria: Controlling Cell Death
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Viral Proteins Targeting Mitochondria: Controlling Cell Death
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family
Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine
The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family
Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine
Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin
Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine
Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects
WEB ACCESIBLE