PUBLICACIÓN

Biological effects of olive oil phenolic compounds on mitochondria

ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Blanco-Benitez M. et al.

2022 Molecular and Cellular Oncology

Cancer Research (Q4), Molecular Medicine (Q3)

JCR: 2.1

SJR: 0.436

CITAS

18

DOI

10.1080/23723556.2022.2044263

EID

2-s2.0-85126761843

EISSN

2372-3556

BIBTEX

@article{Blanco_Ben_tez_2022,doi = {10.1080/23723556.2022.2044263},url = {https://doi.org/10.1080%2F23723556.2022.2044263},year = 2022,month = {mar},publisher = {Informa {UK} Limited},volume = {9},number = {1},author = {Mercedes Blanco-Ben{\'{\i}}tez and Ana Calder{\'{o}}n-Fern{\'{a}}ndez and Saray Canales-Cort{\'{e}}s and Eva Alegre-Cort{\'{e}}s and Elisabet Uribe-Carretero and Marta Paredes-Barquero and Alberto Gimenez-Bejarano and Gema Duque Gonz{\'{a}}lez and Patricia G{\'{o}}mez-Suaga and Juan Ortega-Vidal and Sof{\'{\i}}a Salido and Joaqu{\'{\i}}n Altarejos and Guadalupe Mart{\'{\i}}nez-Chac{\'{o}}n and Mireia Niso-Santano and Jos{\'{e}} M. Fuentes and Rosa A. Gonz{\'{a}}lez-Polo and Sokhna M. S. Yakhine-Diop},title = {Biological effects of olive oil phenolic compounds on mitochondria},journal = {Molecular {\&}amp$\mathsemicolon$ Cellular Oncology}}

AUTORES DE LA UEX

Patricia María Gómez Suaga
Eva Alegre Cortés
Rosa Ana González Polo
Mireia Niso Santano
Marta Paredes Barquero
José Manuel Fuentes Rodríguez

OTRAS PUBLICACIONES DE INTERÉS

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The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress.
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Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells
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Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
Er-Mitochondria Signaling Regulates Autophagy
[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
Er-Mitochondria Signaling Regulates Autophagy
Α-Synuclein Binds To The Er–mitochondria Tethering Protein Vapb To Disrupt Ca2+ Homeostasis And Mitochondrial Atp Production
Er-Mitochondria Signaling Regulates Autophagy
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Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
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Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy
Trend Of Lipid Profile In General Population From Caceres Health Area
Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy
Trend Of Lipid Profile In General Population From Caceres Health Area
Als/ftd-Associated Fus Activates Gsk-3β To Disrupt The Vapb–ptpip51 Interaction And Er–mitochondria Associations
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There's Something Wrong With My Mam; The Er-Mitochondria Axis And Neurodegenerative Diseases
Iron Overload Causes Endolysosomal Deficits Modulated By Naadp-Regulated 2-Pore Channels And Rab7a
There's Something Wrong With My Mam; The Er-Mitochondria Axis And Neurodegenerative Diseases
The Basics Of Autophagy
There's Something Wrong With My Mam; The Er-Mitochondria Axis And Neurodegenerative Diseases
The Basics Of Autophagy
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
The Basics Of Autophagy
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
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Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis
Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis
Mitochondria: Key Organelle In Parkinson's Disease
Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis
Mitochondria: Key Organelle In Parkinson's Disease
G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway
Mitochondria: Key Organelle In Parkinson's Disease
G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway
Pharmacological Inhibition Of Fatty-Acid Oxidation Synergistically Enhances The Effect Of L-Asparaginase In Childhood All Cells
G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway
Pharmacological Inhibition Of Fatty-Acid Oxidation Synergistically Enhances The Effect Of L-Asparaginase In Childhood All Cells
Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment
Pharmacological Inhibition Of Fatty-Acid Oxidation Synergistically Enhances The Effect Of L-Asparaginase In Childhood All Cells
Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment
Alterations In Late Endocytic Trafficking Related To The Pathobiology Of Lrrk2-Linked Parkinson's Disease
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Autophagic Pathology And Calcium Deregulation In Neurodegeneration
Alterations In Late Endocytic Trafficking Related To The Pathobiology Of Lrrk2-Linked Parkinson's Disease
Autophagic Pathology And Calcium Deregulation In Neurodegeneration
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Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
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Metabolomic Analyses Reveal That Anti-Aging Metabolites Are Depleted By Palmitate But Increased By Oleate In Vivo
Unsaturated Fatty Acids Induce Non-Canonical Autophagy
Metabolomic Analyses Reveal That Anti-Aging Metabolites Are Depleted By Palmitate But Increased By Oleate In Vivo
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Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Spermidine Induces Autophagy By Inhibiting The Acetyltransferase Ep300
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Toxicity And Autophagy In Neurodegenerative Disorders
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Toxicity And Autophagy In Neurodegenerative Disorders
Origen Molecular De La Compleja Enfermedad De Parkinson
Toxicity And Autophagy In Neurodegenerative Disorders
Origen Molecular De La Compleja Enfermedad De Parkinson
Lrrk2 Delays Degradative Receptor Trafficking By Impeding Late Endosomal Budding Through Decreasing Rab7 Activity
Origen Molecular De La Compleja Enfermedad De Parkinson
Lrrk2 Delays Degradative Receptor Trafficking By Impeding Late Endosomal Budding Through Decreasing Rab7 Activity
Upstream Deregulation Of Calcium Signaling In Parkinson's Disease
Lrrk2 Delays Degradative Receptor Trafficking By Impeding Late Endosomal Budding Through Decreasing Rab7 Activity
Upstream Deregulation Of Calcium Signaling In Parkinson's Disease
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
Upstream Deregulation Of Calcium Signaling In Parkinson's Disease
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Links Between Paraquat And Parkinson's Disease
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Links Between Paraquat And Parkinson's Disease
Cancer Cell–autonomous Contribution Of Type I Interferon Signaling To The Efficacy Of Chemotherapy
Links Between Paraquat And Parkinson's Disease
Cancer Cell–autonomous Contribution Of Type I Interferon Signaling To The Efficacy Of Chemotherapy
Coffee Induces Autophagy In Vivo
Cancer Cell–autonomous Contribution Of Type I Interferon Signaling To The Efficacy Of Chemotherapy
Coffee Induces Autophagy In Vivo
Regulation Of Autophagy By Cytosolic Acetyl-Coenzyme A
Coffee Induces Autophagy In Vivo
Regulation Of Autophagy By Cytosolic Acetyl-Coenzyme A
Self-Consumption: The Interplay Of Autophagy And Apoptosis
Regulation Of Autophagy By Cytosolic Acetyl-Coenzyme A
Self-Consumption: The Interplay Of Autophagy And Apoptosis
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Self-Consumption: The Interplay Of Autophagy And Apoptosis
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Implication Of Autophagy In Parkinson's Disease
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Implication Of Autophagy In Parkinson's Disease
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
Implication Of Autophagy In Parkinson's Disease
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Regulation Of Autophagy By Stress-Responsive Transcription Factors
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Regulation Of Autophagy By Stress-Responsive Transcription Factors
Direct Interaction Between Stat3 And Eif2ak2 Controls Fatty Acid-Induced Autophagy
Regulation Of Autophagy By Stress-Responsive Transcription Factors
Direct Interaction Between Stat3 And Eif2ak2 Controls Fatty Acid-Induced Autophagy
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Direct Interaction Between Stat3 And Eif2ak2 Controls Fatty Acid-Induced Autophagy
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Paraquat, Between Apoptosis And Autophagy
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Paraquat, Between Apoptosis And Autophagy
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Paraquat, Between Apoptosis And Autophagy
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.
A Link Between Autophagy And The Pathophysiology Of Lrrk2 In Parkinson's Disease
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.
A Link Between Autophagy And The Pathophysiology Of Lrrk2 In Parkinson's Disease
A Link Between Lrrk2, Autophagy And Naadp-Mediated Endolysosomal Calcium Signalling
A Link Between Autophagy And The Pathophysiology Of Lrrk2 In Parkinson's Disease
A Link Between Lrrk2, Autophagy And Naadp-Mediated Endolysosomal Calcium Signalling
Stone-Isolated Carbonatogenic Bacteria As Inoculants In Bioconsolidation Treatments For Historical Limestone
A Link Between Lrrk2, Autophagy And Naadp-Mediated Endolysosomal Calcium Signalling
Stone-Isolated Carbonatogenic Bacteria As Inoculants In Bioconsolidation Treatments For Historical Limestone
Leucine-Rich Repeat Kinase 2 Regulates Autophagy Through A Calcium-Dependent Pathway Involving Naadp
Stone-Isolated Carbonatogenic Bacteria As Inoculants In Bioconsolidation Treatments For Historical Limestone
Leucine-Rich Repeat Kinase 2 Regulates Autophagy Through A Calcium-Dependent Pathway Involving Naadp
Lrrk2 As A Modulator Of Lysosomal Calcium Homeostasis With Downstream Effects On Autophagy
Leucine-Rich Repeat Kinase 2 Regulates Autophagy Through A Calcium-Dependent Pathway Involving Naadp
Lrrk2 As A Modulator Of Lysosomal Calcium Homeostasis With Downstream Effects On Autophagy
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Lrrk2 As A Modulator Of Lysosomal Calcium Homeostasis With Downstream Effects On Autophagy
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway
Autophagic Removal Of Micronuclei
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway
Autophagic Removal Of Micronuclei
Autophagy Is Required For The Activation Of Nfκb
Autophagic Removal Of Micronuclei
Autophagy Is Required For The Activation Of Nfκb
Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))
Autophagy Is Required For The Activation Of Nfκb
Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))
Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity
Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))
Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins
An Immunosurveillance Mechanism Controls Cancer Cell Ploidy
Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins
An Immunosurveillance Mechanism Controls Cancer Cell Ploidy
Posttranslational Modifications As Versatile Regulators Of Parkin Function
An Immunosurveillance Mechanism Controls Cancer Cell Ploidy
Posttranslational Modifications As Versatile Regulators Of Parkin Function
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Posttranslational Modifications As Versatile Regulators Of Parkin Function
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Inhibition Of Autophagy By Tab2 And Tab3
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Inhibition Of Autophagy By Tab2 And Tab3
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Inhibition Of Autophagy By Tab2 And Tab3
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor
The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death
Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor
The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death
Inhibition Of Macroautophagy Triggers Apoptosis
The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death
Inhibition Of Macroautophagy Triggers Apoptosis
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Inhibition Of Macroautophagy Triggers Apoptosis
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Viral Proteins Targeting Mitochondria: Controlling Cell Death
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Viral Proteins Targeting Mitochondria: Controlling Cell Death
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Viral Proteins Targeting Mitochondria: Controlling Cell Death
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family
Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine
The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family
Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine
Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin
Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine
Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
The Immune System And Autoaggression
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
The Immune System And Autoaggression
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
The Immune System And Autoaggression
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)
Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice
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