PUBLICACIÓN

Delay of EGF-Stimulated EGFR Degradation in Myotonic Dystrophy Type 1 (DM1)

ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Alegre-Cortes E. et al.

2022 Cells

Biochemistry, Genetics and Molecular Biology (miscellaneous) (Q1)

JCR: 6

SJR: 1.537

CITAS

2

DOI

10.3390/cells11193018

EID

2-s2.0-85139760705

EISSN

2073-4409

AUTORES DE LA UEX

Patricia María Gómez Suaga
Pedro Javier Camello Almaraz
Eva Alegre Cortés
Rosa Ana González Polo
Mireia Niso Santano
Marta Paredes Barquero
José Manuel Fuentes Rodríguez

OTRAS PUBLICACIONES DE INTERÉS

Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)
Filamin A C-Terminal Fragment Modulates Orai1 Expression By Inhibition Of Protein Degradation
Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice
Filamin A C-Terminal Fragment Modulates Orai1 Expression By Inhibition Of Protein Degradation
Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice
Stimulating Vapb-Ptpip51 Er-Mitochondria Tethering Corrects Ftd/als Mutant Tdp43 Linked Ca2+ And Synaptic Defects
Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice
Stimulating Vapb-Ptpip51 Er-Mitochondria Tethering Corrects Ftd/als Mutant Tdp43 Linked Ca2+ And Synaptic Defects
Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques
Stimulating Vapb-Ptpip51 Er-Mitochondria Tethering Corrects Ftd/als Mutant Tdp43 Linked Ca2+ And Synaptic Defects
Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques
The Role Of Cardiolipin In Mitochondrial Function And Neurodegenerative Diseases
Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques
The Role Of Cardiolipin In Mitochondrial Function And Neurodegenerative Diseases
Immunological Aspects Of Central Neurodegeneration
The Role Of Cardiolipin In Mitochondrial Function And Neurodegenerative Diseases
Immunological Aspects Of Central Neurodegeneration
Role Of Tfeb In Huntington’s Disease
Immunological Aspects Of Central Neurodegeneration
Role Of Tfeb In Huntington’s Disease
Truncated Tau Interferes With The Autophagy And Endolysosomal Pathway And Results In Lipid Accumulation
Role Of Tfeb In Huntington’s Disease
Truncated Tau Interferes With The Autophagy And Endolysosomal Pathway And Results In Lipid Accumulation
Pharmacological Inhibition Reveals Participation Of The Endocytic Compartment In Positive Feedback Il-6 Secretion In Human Skeletal Myotubes
Truncated Tau Interferes With The Autophagy And Endolysosomal Pathway And Results In Lipid Accumulation
Pharmacological Inhibition Reveals Participation Of The Endocytic Compartment In Positive Feedback Il-6 Secretion In Human Skeletal Myotubes
Insight Into Endoplasmic Reticulum-Mitochondria Contacts In Human Amyotrophic Lateral Sclerosis
Pharmacological Inhibition Reveals Participation Of The Endocytic Compartment In Positive Feedback Il-6 Secretion In Human Skeletal Myotubes
Insight Into Endoplasmic Reticulum-Mitochondria Contacts In Human Amyotrophic Lateral Sclerosis
Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects
Insight Into Endoplasmic Reticulum-Mitochondria Contacts In Human Amyotrophic Lateral Sclerosis
Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects
Lysosomal Dysfunction: Connecting The Dots In The Landscape Of Human Diseases
Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects
Lysosomal Dysfunction: Connecting The Dots In The Landscape Of Human Diseases
Corrigendum: Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells (Journal Of Cellular Physiology, 10.1002/jcp.26855)
Lysosomal Dysfunction: Connecting The Dots In The Landscape Of Human Diseases
Corrigendum: Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells (Journal Of Cellular Physiology, 10.1002/jcp.26855)
Vitamin D Receptor Polymorphisms In A Spanish Cohort Of Parkinson’s Disease Patients
Corrigendum: Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells (Journal Of Cellular Physiology, 10.1002/jcp.26855)
Vitamin D Receptor Polymorphisms In A Spanish Cohort Of Parkinson’s Disease Patients
Links Between Paraquat And Parkinson’s Disease
Vitamin D Receptor Polymorphisms In A Spanish Cohort Of Parkinson’s Disease Patients
Links Between Paraquat And Parkinson’s Disease
Secretion Of Interleukin 6 In Human Skeletal Muscle Cultures Depends On Ca2+ Signalling
Links Between Paraquat And Parkinson’s Disease
Secretion Of Interleukin 6 In Human Skeletal Muscle Cultures Depends On Ca2+ Signalling
Mitochondrial Dysfunction In Repeat Expansion Diseases
Secretion Of Interleukin 6 In Human Skeletal Muscle Cultures Depends On Ca2+ Signalling
Mitochondrial Dysfunction In Repeat Expansion Diseases
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction
Mitochondrial Dysfunction In Repeat Expansion Diseases
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction
Editorial
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction
Editorial
Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease
Editorial
Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease
Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease
Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease
Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress
Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress
Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress
Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles
In Vitro And In Vivo Models To Study The Biological And Pharmacological Properties Of Queen Bee Acid (Qba, 10-Hydroxy-2-Decenoic Acid): A Systematic Review
Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles
In Vitro And In Vivo Models To Study The Biological And Pharmacological Properties Of Queen Bee Acid (Qba, 10-Hydroxy-2-Decenoic Acid): A Systematic Review
Role Of Inflammatory Cytokines In The Conversion Of Mild Cognitive Impairment To Dementia: A Prospective Study.
In Vitro And In Vivo Models To Study The Biological And Pharmacological Properties Of Queen Bee Acid (Qba, 10-Hydroxy-2-Decenoic Acid): A Systematic Review
Role Of Inflammatory Cytokines In The Conversion Of Mild Cognitive Impairment To Dementia: A Prospective Study.
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
Role Of Inflammatory Cytokines In The Conversion Of Mild Cognitive Impairment To Dementia: A Prospective Study.
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
The Presence Of Disease-Associated Tau Leads To Adverse Effects On The Autophagy And Endolysosomal Pathway In A Cell Model Of Tauopathy
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
The Presence Of Disease-Associated Tau Leads To Adverse Effects On The Autophagy And Endolysosomal Pathway In A Cell Model Of Tauopathy
Disruption Of The Vapb-Ptpip51 Er-Mitochondria Tethering Proteins In Post-Mortem Human Amyotrophic Lateral Sclerosis
The Presence Of Disease-Associated Tau Leads To Adverse Effects On The Autophagy And Endolysosomal Pathway In A Cell Model Of Tauopathy
Disruption Of The Vapb-Ptpip51 Er-Mitochondria Tethering Proteins In Post-Mortem Human Amyotrophic Lateral Sclerosis
The Interplay Between Autophagy And Rna Homeostasis: Implications For Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
Disruption Of The Vapb-Ptpip51 Er-Mitochondria Tethering Proteins In Post-Mortem Human Amyotrophic Lateral Sclerosis
The Interplay Between Autophagy And Rna Homeostasis: Implications For Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
Disruption Of Er-Mitochondria Tethering And Signalling In C9orf72-Associated Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
The Interplay Between Autophagy And Rna Homeostasis: Implications For Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
Disruption Of Er-Mitochondria Tethering And Signalling In C9orf72-Associated Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
Orai1α, But Not Orai1β, Co-Localizes With Trpc1 And Is Required For Its Plasma Membrane Location And Activation In Hela Cells
Disruption Of Er-Mitochondria Tethering And Signalling In C9orf72-Associated Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
Orai1α, But Not Orai1β, Co-Localizes With Trpc1 And Is Required For Its Plasma Membrane Location And Activation In Hela Cells
Neuroprotective And Anti-Inflammatory Effects Of Linoleic Acid In Models Of Parkinson’s Disease: The Implication Of Lipid Droplets And Lipophagy
Orai1α, But Not Orai1β, Co-Localizes With Trpc1 And Is Required For Its Plasma Membrane Location And Activation In Hela Cells
Neuroprotective And Anti-Inflammatory Effects Of Linoleic Acid In Models Of Parkinson’s Disease: The Implication Of Lipid Droplets And Lipophagy
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Neuroprotective And Anti-Inflammatory Effects Of Linoleic Acid In Models Of Parkinson’s Disease: The Implication Of Lipid Droplets And Lipophagy
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1
Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1
Links Between Paraquat And Parkinson’s Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1
Links Between Paraquat And Parkinson’s Disease
Tmem97 Facilitates The Activation Of Soce By Downregulating The Association Of Cholesterol To Orai1 In Mda-Mb-231 Cells
Links Between Paraquat And Parkinson’s Disease
Tmem97 Facilitates The Activation Of Soce By Downregulating The Association Of Cholesterol To Orai1 In Mda-Mb-231 Cells
Monochloramine Effects On Gallbladder Contractility
Tmem97 Facilitates The Activation Of Soce By Downregulating The Association Of Cholesterol To Orai1 In Mda-Mb-231 Cells
Monochloramine Effects On Gallbladder Contractility
Procedimientos, Experimentación E Investigación En Enfermedades Neurodegenerativas: Protocolos Para Un Laboratorio De Neurociencias
Monochloramine Effects On Gallbladder Contractility
Procedimientos, Experimentación E Investigación En Enfermedades Neurodegenerativas: Protocolos Para Un Laboratorio De Neurociencias
Autophagy And Lysosomal Defects In Cells Expressing Disease-Associated Tau
Procedimientos, Experimentación E Investigación En Enfermedades Neurodegenerativas: Protocolos Para Un Laboratorio De Neurociencias
Autophagy And Lysosomal Defects In Cells Expressing Disease-Associated Tau
Cultivos Celulares
Autophagy And Lysosomal Defects In Cells Expressing Disease-Associated Tau
Cultivos Celulares
Extracción De Rna
Cultivos Celulares
Extracción De Rna
Técnica De Inmunoprecipitación
Extracción De Rna
Técnica De Inmunoprecipitación
Microscopía Electrónica
Técnica De Inmunoprecipitación
Microscopía Electrónica
Análisis De Extracto Proteico Por Western Blotting
Microscopía Electrónica
Análisis De Extracto Proteico Por Western Blotting
Extracción De Proteínas
Análisis De Extracto Proteico Por Western Blotting
Extracción De Proteínas
Técnica De Sobreexpresión Génica
Extracción De Proteínas
Técnica De Sobreexpresión Génica
Técnica De Silenciamiento Génico
Técnica De Sobreexpresión Génica
Técnica De Silenciamiento Génico
The Dual Role Of Necrostatin-1 In Parkinson's Disease Models
Técnica De Silenciamiento Génico
The Dual Role Of Necrostatin-1 In Parkinson's Disease Models
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction.
The Dual Role Of Necrostatin-1 In Parkinson's Disease Models
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction.
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress.
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction.
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress.
Fisiología Hepática
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress.
Fisiología Hepática
Bases Neurológicas De La Motilidad
Fisiología Hepática
Bases Neurológicas De La Motilidad
Autophagy Modulation In Animal Models Of Corneal Diseases: A Systematic Review
Bases Neurológicas De La Motilidad
Autophagy Modulation In Animal Models Of Corneal Diseases: A Systematic Review
Animal Models Used To Study Direct Peripheral Nerve Repair: A Systematic Review
Autophagy Modulation In Animal Models Of Corneal Diseases: A Systematic Review
Animal Models Used To Study Direct Peripheral Nerve Repair: A Systematic Review
Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease
Animal Models Used To Study Direct Peripheral Nerve Repair: A Systematic Review
Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease
No1, A New Sigma 2 Receptor/tmem97 Fluorescent Ligand, Downregulates Soce And Promotes Apoptosis In The Triple Negative Breast Cancer Cell Lines
Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease
No1, A New Sigma 2 Receptor/tmem97 Fluorescent Ligand, Downregulates Soce And Promotes Apoptosis In The Triple Negative Breast Cancer Cell Lines
Age-Induced Differential Changes In The Central And Colonic Human Circadian Oscillators
No1, A New Sigma 2 Receptor/tmem97 Fluorescent Ligand, Downregulates Soce And Promotes Apoptosis In The Triple Negative Breast Cancer Cell Lines
Age-Induced Differential Changes In The Central And Colonic Human Circadian Oscillators
Disruption Of Endoplasmic Reticulum-Mitochondria Tethering Proteins In Post-Mortem Alzheimer's Disease Brain
Age-Induced Differential Changes In The Central And Colonic Human Circadian Oscillators
Disruption Of Endoplasmic Reticulum-Mitochondria Tethering Proteins In Post-Mortem Alzheimer's Disease Brain
Biología: Tenemos Una Buena Representación En La Base, Pero Si Asciendes En La Cúspide Va Habiendo Menos Visibilidad
Disruption Of Endoplasmic Reticulum-Mitochondria Tethering Proteins In Post-Mortem Alzheimer's Disease Brain
Biología: Tenemos Una Buena Representación En La Base, Pero Si Asciendes En La Cúspide Va Habiendo Menos Visibilidad
Toxicity Of Necrostatin-1 In Parkinson’s Disease Models
Biología: Tenemos Una Buena Representación En La Base, Pero Si Asciendes En La Cúspide Va Habiendo Menos Visibilidad
Toxicity Of Necrostatin-1 In Parkinson’s Disease Models
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells
Toxicity Of Necrostatin-1 In Parkinson’s Disease Models
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells
Natural Products In The Promotion Of Healthspan And Longevity.
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells
Natural Products In The Promotion Of Healthspan And Longevity.
Lmtk2 Binds To Kinesin Light Chains To Mediate Anterograde Axonal Transport Of Cdk5/p35 And Lmtk2 Levels Are Reduced In Alzheimer's Disease Brains
Natural Products In The Promotion Of Healthspan And Longevity.
Lmtk2 Binds To Kinesin Light Chains To Mediate Anterograde Axonal Transport Of Cdk5/p35 And Lmtk2 Levels Are Reduced In Alzheimer's Disease Brains
The Vapb-Ptpip51 Endoplasmic Reticulum-Mitochondria Tethering Proteins Are Present In Neuronal Synapses And Regulate Synaptic Activity
Lmtk2 Binds To Kinesin Light Chains To Mediate Anterograde Axonal Transport Of Cdk5/p35 And Lmtk2 Levels Are Reduced In Alzheimer's Disease Brains
The Vapb-Ptpip51 Endoplasmic Reticulum-Mitochondria Tethering Proteins Are Present In Neuronal Synapses And Regulate Synaptic Activity
Adenylyl Cyclase Type 8 Overexpression Impairs Phosphorylation-Dependent Orai1 Inactivation And Promotes Migration In Mda-Mb-231 Breast Cancer Cells
The Vapb-Ptpip51 Endoplasmic Reticulum-Mitochondria Tethering Proteins Are Present In Neuronal Synapses And Regulate Synaptic Activity
Adenylyl Cyclase Type 8 Overexpression Impairs Phosphorylation-Dependent Orai1 Inactivation And Promotes Migration In Mda-Mb-231 Breast Cancer Cells
Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack
Adenylyl Cyclase Type 8 Overexpression Impairs Phosphorylation-Dependent Orai1 Inactivation And Promotes Migration In Mda-Mb-231 Breast Cancer Cells
Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack
The Paradigm Of Protein Acetylation In Parkinson's Disease
Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack
The Paradigm Of Protein Acetylation In Parkinson's Disease
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients
The Paradigm Of Protein Acetylation In Parkinson's Disease
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients
A Pre-Clinical Rat Model For The Study Of Ischemia-Reperfusion Injury In Reconstructive Microsurgery
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients
A Pre-Clinical Rat Model For The Study Of Ischemia-Reperfusion Injury In Reconstructive Microsurgery
Erratum: Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity (Cell Metabolism (2019) 30(4) (754–767.e9), (S1550413119303833), (10.1016/j.cmet.2019.07.010))
A Pre-Clinical Rat Model For The Study Of Ischemia-Reperfusion Injury In Reconstructive Microsurgery
Erratum: Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity (Cell Metabolism (2019) 30(4) (754–767.e9), (S1550413119303833), (10.1016/j.cmet.2019.07.010))
Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity
Erratum: Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity (Cell Metabolism (2019) 30(4) (754–767.e9), (S1550413119303833), (10.1016/j.cmet.2019.07.010))
Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson's Disease Patients.
Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson's Disease Patients.
Cholesterol And Multilamellar Bodies: Lysosomal Dysfunction In Gba-Parkinson Disease
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson's Disease Patients.
Cholesterol And Multilamellar Bodies: Lysosomal Dysfunction In Gba-Parkinson Disease
Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria
Cholesterol And Multilamellar Bodies: Lysosomal Dysfunction In Gba-Parkinson Disease
Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria
Vascular Risk Factors And Lesions Of Vascular Nature In Magnetic Resonance As Predictors Of Progression To Dementia In Patients With Mild Cognitive Impairment
Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria
Vascular Risk Factors And Lesions Of Vascular Nature In Magnetic Resonance As Predictors Of Progression To Dementia In Patients With Mild Cognitive Impairment
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells: In Vitro And In Vivo Studies
Vascular Risk Factors And Lesions Of Vascular Nature In Magnetic Resonance As Predictors Of Progression To Dementia In Patients With Mild Cognitive Impairment
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells: In Vitro And In Vivo Studies
Efhb Is A Novel Cytosolic Ca 2+ Sensor That Modulates Stim1-Saraf Interaction
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells: In Vitro And In Vivo Studies
Efhb Is A Novel Cytosolic Ca 2+ Sensor That Modulates Stim1-Saraf Interaction
Secreción Pancreática Exocrina
Efhb Is A Novel Cytosolic Ca 2+ Sensor That Modulates Stim1-Saraf Interaction
Secreción Pancreática Exocrina
Disruption Of Er-Mitochondria Signalling In Fronto-Temporal Dementia And Related Amyotrophic Lateral Sclerosis
Secreción Pancreática Exocrina
Disruption Of Er-Mitochondria Signalling In Fronto-Temporal Dementia And Related Amyotrophic Lateral Sclerosis
Parkinson Disease-Associated Mutations In Lrrk2 Cause Centrosomal Defects Via Rab8a Phosphorylation
Disruption Of Er-Mitochondria Signalling In Fronto-Temporal Dementia And Related Amyotrophic Lateral Sclerosis
Parkinson Disease-Associated Mutations In Lrrk2 Cause Centrosomal Defects Via Rab8a Phosphorylation
Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Parkinson Disease-Associated Mutations In Lrrk2 Cause Centrosomal Defects Via Rab8a Phosphorylation
Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
Octodon Degus, A New Model To Study The Agonist And Plexus-Induced Response In The Urinary Bladder
[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
Octodon Degus, A New Model To Study The Agonist And Plexus-Induced Response In The Urinary Bladder
Er-Mitochondria Signaling Regulates Autophagy
Octodon Degus, A New Model To Study The Agonist And Plexus-Induced Response In The Urinary Bladder
Er-Mitochondria Signaling Regulates Autophagy
Α-Synuclein Binds To The Er–mitochondria Tethering Protein Vapb To Disrupt Ca2+ Homeostasis And Mitochondrial Atp Production
Er-Mitochondria Signaling Regulates Autophagy
Α-Synuclein Binds To The Er–mitochondria Tethering Protein Vapb To Disrupt Ca2+ Homeostasis And Mitochondrial Atp Production
The Er-Mitochondria Tethering Complex Vapb-Ptpip51 Regulates Autophagy
Α-Synuclein Binds To The Er–mitochondria Tethering Protein Vapb To Disrupt Ca2+ Homeostasis And Mitochondrial Atp Production
The Er-Mitochondria Tethering Complex Vapb-Ptpip51 Regulates Autophagy
N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
The Er-Mitochondria Tethering Complex Vapb-Ptpip51 Regulates Autophagy
N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells
Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells
Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy
Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy
Trend Of Lipid Profile In General Population From Caceres Health Area
Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy
Trend Of Lipid Profile In General Population From Caceres Health Area
Mitochondria-Associated Membranes (Mams)
Trend Of Lipid Profile In General Population From Caceres Health Area
Mitochondria-Associated Membranes (Mams)
Propagation Of Intracellular Ca2+ Signals In Aged Exocrine Cells
Mitochondria-Associated Membranes (Mams)
Propagation Of Intracellular Ca2+ Signals In Aged Exocrine Cells
Als/ftd-Associated Fus Activates Gsk-3β To Disrupt The Vapb–ptpip51 Interaction And Er–mitochondria Associations
Propagation Of Intracellular Ca2+ Signals In Aged Exocrine Cells
Als/ftd-Associated Fus Activates Gsk-3β To Disrupt The Vapb–ptpip51 Interaction And Er–mitochondria Associations
Iron Overload Causes Endolysosomal Deficits Modulated By Naadp-Regulated 2-Pore Channels And Rab7a
Als/ftd-Associated Fus Activates Gsk-3β To Disrupt The Vapb–ptpip51 Interaction And Er–mitochondria Associations
Iron Overload Causes Endolysosomal Deficits Modulated By Naadp-Regulated 2-Pore Channels And Rab7a
There's Something Wrong With My Mam; The Er-Mitochondria Axis And Neurodegenerative Diseases
Iron Overload Causes Endolysosomal Deficits Modulated By Naadp-Regulated 2-Pore Channels And Rab7a
There's Something Wrong With My Mam; The Er-Mitochondria Axis And Neurodegenerative Diseases
The Basics Of Autophagy
There's Something Wrong With My Mam; The Er-Mitochondria Axis And Neurodegenerative Diseases
The Basics Of Autophagy
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
The Basics Of Autophagy
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis
Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis
Mitochondria: Key Organelle In Parkinson's Disease
Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis
Mitochondria: Key Organelle In Parkinson's Disease
G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway
Mitochondria: Key Organelle In Parkinson's Disease
G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway
Pharmacological Inhibition Of Fatty-Acid Oxidation Synergistically Enhances The Effect Of L-Asparaginase In Childhood All Cells
G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway
Pharmacological Inhibition Of Fatty-Acid Oxidation Synergistically Enhances The Effect Of L-Asparaginase In Childhood All Cells
Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment
Pharmacological Inhibition Of Fatty-Acid Oxidation Synergistically Enhances The Effect Of L-Asparaginase In Childhood All Cells
Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment
Alterations In Late Endocytic Trafficking Related To The Pathobiology Of Lrrk2-Linked Parkinson's Disease
Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment
Alterations In Late Endocytic Trafficking Related To The Pathobiology Of Lrrk2-Linked Parkinson's Disease
Autophagic Pathology And Calcium Deregulation In Neurodegeneration
Alterations In Late Endocytic Trafficking Related To The Pathobiology Of Lrrk2-Linked Parkinson's Disease
Autophagic Pathology And Calcium Deregulation In Neurodegeneration
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
Autophagic Pathology And Calcium Deregulation In Neurodegeneration
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Control Of Autophagy In Parkinson’s Disease
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Control Of Autophagy In Parkinson’s Disease
Unsaturated Fatty Acids Induce Non-Canonical Autophagy
Control Of Autophagy In Parkinson’s Disease
Unsaturated Fatty Acids Induce Non-Canonical Autophagy
Metabolomic Analyses Reveal That Anti-Aging Metabolites Are Depleted By Palmitate But Increased By Oleate In Vivo
Unsaturated Fatty Acids Induce Non-Canonical Autophagy
Metabolomic Analyses Reveal That Anti-Aging Metabolites Are Depleted By Palmitate But Increased By Oleate In Vivo
Novel Inducers Of Becn1-Independent Autophagy: Cis-Unsaturated Fatty Acids
Metabolomic Analyses Reveal That Anti-Aging Metabolites Are Depleted By Palmitate But Increased By Oleate In Vivo
Novel Inducers Of Becn1-Independent Autophagy: Cis-Unsaturated Fatty Acids
Spermidine Induces Autophagy By Inhibiting The Acetyltransferase Ep300
Novel Inducers Of Becn1-Independent Autophagy: Cis-Unsaturated Fatty Acids
Spermidine Induces Autophagy By Inhibiting The Acetyltransferase Ep300
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Spermidine Induces Autophagy By Inhibiting The Acetyltransferase Ep300
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Toxicity And Autophagy In Neurodegenerative Disorders
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Toxicity And Autophagy In Neurodegenerative Disorders
Origen Molecular De La Compleja Enfermedad De Parkinson
Toxicity And Autophagy In Neurodegenerative Disorders
Origen Molecular De La Compleja Enfermedad De Parkinson
Characterization Of The Motor Inhibitory Role Of Colonic Mucosa Under Chemical Stimulation In Mice
Origen Molecular De La Compleja Enfermedad De Parkinson
Characterization Of The Motor Inhibitory Role Of Colonic Mucosa Under Chemical Stimulation In Mice
Age-Related Changes In Mitochondrial Function Of Mouse Colonic Smooth Muscle: Beneficial Effects Of Melatonin
Characterization Of The Motor Inhibitory Role Of Colonic Mucosa Under Chemical Stimulation In Mice
Age-Related Changes In Mitochondrial Function Of Mouse Colonic Smooth Muscle: Beneficial Effects Of Melatonin
Uso Del Campus Virtual Para La Evaluación Continuada En El Aula En Grupos Grandes
Age-Related Changes In Mitochondrial Function Of Mouse Colonic Smooth Muscle: Beneficial Effects Of Melatonin
Uso Del Campus Virtual Para La Evaluación Continuada En El Aula En Grupos Grandes
Lrrk2 Delays Degradative Receptor Trafficking By Impeding Late Endosomal Budding Through Decreasing Rab7 Activity
Uso Del Campus Virtual Para La Evaluación Continuada En El Aula En Grupos Grandes
Lrrk2 Delays Degradative Receptor Trafficking By Impeding Late Endosomal Budding Through Decreasing Rab7 Activity
Upstream Deregulation Of Calcium Signaling In Parkinson's Disease
Lrrk2 Delays Degradative Receptor Trafficking By Impeding Late Endosomal Budding Through Decreasing Rab7 Activity
Upstream Deregulation Of Calcium Signaling In Parkinson's Disease
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
Upstream Deregulation Of Calcium Signaling In Parkinson's Disease
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Links Between Paraquat And Parkinson's Disease
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Links Between Paraquat And Parkinson's Disease
Cancer Cell–autonomous Contribution Of Type I Interferon Signaling To The Efficacy Of Chemotherapy
Links Between Paraquat And Parkinson's Disease
Cancer Cell–autonomous Contribution Of Type I Interferon Signaling To The Efficacy Of Chemotherapy
Coffee Induces Autophagy In Vivo
Cancer Cell–autonomous Contribution Of Type I Interferon Signaling To The Efficacy Of Chemotherapy
Coffee Induces Autophagy In Vivo
Regulation Of Autophagy By Cytosolic Acetyl-Coenzyme A
Coffee Induces Autophagy In Vivo
Regulation Of Autophagy By Cytosolic Acetyl-Coenzyme A
Self-Consumption: The Interplay Of Autophagy And Apoptosis
Regulation Of Autophagy By Cytosolic Acetyl-Coenzyme A
Self-Consumption: The Interplay Of Autophagy And Apoptosis
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Self-Consumption: The Interplay Of Autophagy And Apoptosis
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Mtor Pathway And Ca2+ Stores Mobilization In Aged Smooth Muscle Cells
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Mtor Pathway And Ca2+ Stores Mobilization In Aged Smooth Muscle Cells
Implication Of Autophagy In Parkinson's Disease
Mtor Pathway And Ca2+ Stores Mobilization In Aged Smooth Muscle Cells
Implication Of Autophagy In Parkinson's Disease
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
Implication Of Autophagy In Parkinson's Disease
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Regulation Of Autophagy By Stress-Responsive Transcription Factors
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Regulation Of Autophagy By Stress-Responsive Transcription Factors
Direct Interaction Between Stat3 And Eif2ak2 Controls Fatty Acid-Induced Autophagy
Regulation Of Autophagy By Stress-Responsive Transcription Factors
Direct Interaction Between Stat3 And Eif2ak2 Controls Fatty Acid-Induced Autophagy
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Direct Interaction Between Stat3 And Eif2ak2 Controls Fatty Acid-Induced Autophagy
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Paraquat, Between Apoptosis And Autophagy
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Paraquat, Between Apoptosis And Autophagy
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Paraquat, Between Apoptosis And Autophagy
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Aging Impairs Ca2+ Sensitization Pathways In Gallbladder Smooth Muscle
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Aging Impairs Ca2+ Sensitization Pathways In Gallbladder Smooth Muscle
Circadian System Functionality, Hippocampal Oxidative Stress, And Spatial Memory In The Appsweps1de9 Transgenic Model Of Alzheimer Disease: Effects Of Melatonin Or Ramelteon
Aging Impairs Ca2+ Sensitization Pathways In Gallbladder Smooth Muscle
Circadian System Functionality, Hippocampal Oxidative Stress, And Spatial Memory In The Appsweps1de9 Transgenic Model Of Alzheimer Disease: Effects Of Melatonin Or Ramelteon
Aging-Induced Alterations In Female Rat Colon Smooth Muscle: The Protective Effects Of Hormonal Therapy
Circadian System Functionality, Hippocampal Oxidative Stress, And Spatial Memory In The Appsweps1de9 Transgenic Model Of Alzheimer Disease: Effects Of Melatonin Or Ramelteon
Aging-Induced Alterations In Female Rat Colon Smooth Muscle: The Protective Effects Of Hormonal Therapy
Nfatc3 Regulates Trypsinogen Activation, Neutrophil Recruitment, And Tissue Damage In Acute Pancreatitis In Mice
Aging-Induced Alterations In Female Rat Colon Smooth Muscle: The Protective Effects Of Hormonal Therapy
Nfatc3 Regulates Trypsinogen Activation, Neutrophil Recruitment, And Tissue Damage In Acute Pancreatitis In Mice
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.
Nfatc3 Regulates Trypsinogen Activation, Neutrophil Recruitment, And Tissue Damage In Acute Pancreatitis In Mice
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.
A Link Between Autophagy And The Pathophysiology Of Lrrk2 In Parkinson's Disease
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.
A Link Between Autophagy And The Pathophysiology Of Lrrk2 In Parkinson's Disease
A Link Between Lrrk2, Autophagy And Naadp-Mediated Endolysosomal Calcium Signalling
A Link Between Autophagy And The Pathophysiology Of Lrrk2 In Parkinson's Disease
A Link Between Lrrk2, Autophagy And Naadp-Mediated Endolysosomal Calcium Signalling
Stone-Isolated Carbonatogenic Bacteria As Inoculants In Bioconsolidation Treatments For Historical Limestone
A Link Between Lrrk2, Autophagy And Naadp-Mediated Endolysosomal Calcium Signalling
Stone-Isolated Carbonatogenic Bacteria As Inoculants In Bioconsolidation Treatments For Historical Limestone
Leucine-Rich Repeat Kinase 2 Regulates Autophagy Through A Calcium-Dependent Pathway Involving Naadp
Stone-Isolated Carbonatogenic Bacteria As Inoculants In Bioconsolidation Treatments For Historical Limestone
Leucine-Rich Repeat Kinase 2 Regulates Autophagy Through A Calcium-Dependent Pathway Involving Naadp
Lrrk2 As A Modulator Of Lysosomal Calcium Homeostasis With Downstream Effects On Autophagy
Leucine-Rich Repeat Kinase 2 Regulates Autophagy Through A Calcium-Dependent Pathway Involving Naadp
Lrrk2 As A Modulator Of Lysosomal Calcium Homeostasis With Downstream Effects On Autophagy
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Lrrk2 As A Modulator Of Lysosomal Calcium Homeostasis With Downstream Effects On Autophagy
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway
Autophagic Removal Of Micronuclei
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway
Autophagic Removal Of Micronuclei
Autophagy Is Required For The Activation Of Nfκb
Autophagic Removal Of Micronuclei
Autophagy Is Required For The Activation Of Nfκb
Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))
Autophagy Is Required For The Activation Of Nfκb
Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))
Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity
Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))
Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins
An Immunosurveillance Mechanism Controls Cancer Cell Ploidy
Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins
An Immunosurveillance Mechanism Controls Cancer Cell Ploidy
Melatonin Induces Neural Sod2 Expression Independent Of The Nf-Kappab Pathway And Improves The Mitochondrial Population And Function In Old Mice
An Immunosurveillance Mechanism Controls Cancer Cell Ploidy
Melatonin Induces Neural Sod2 Expression Independent Of The Nf-Kappab Pathway And Improves The Mitochondrial Population And Function In Old Mice
Melatonin, And To A Lesser Extent Growth Hormone, Restores Colonic Smooth Muscle Physiology In Old Rats
Melatonin Induces Neural Sod2 Expression Independent Of The Nf-Kappab Pathway And Improves The Mitochondrial Population And Function In Old Mice
Melatonin, And To A Lesser Extent Growth Hormone, Restores Colonic Smooth Muscle Physiology In Old Rats
Aging Differentially Modifies Agonist-Evoked Mouse Detrusor Contraction And Calcium Signals
Melatonin, And To A Lesser Extent Growth Hormone, Restores Colonic Smooth Muscle Physiology In Old Rats
Aging Differentially Modifies Agonist-Evoked Mouse Detrusor Contraction And Calcium Signals
Posttranslational Modifications As Versatile Regulators Of Parkin Function
Aging Differentially Modifies Agonist-Evoked Mouse Detrusor Contraction And Calcium Signals
Posttranslational Modifications As Versatile Regulators Of Parkin Function
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Posttranslational Modifications As Versatile Regulators Of Parkin Function
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Inhibition Of Autophagy By Tab2 And Tab3
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Inhibition Of Autophagy By Tab2 And Tab3
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Inhibition Of Autophagy By Tab2 And Tab3
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Capítulo 62. Bases Neurológicas De La Motilidad
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Capítulo 62. Bases Neurológicas De La Motilidad
Fisiología Hepática
Capítulo 62. Bases Neurológicas De La Motilidad
Fisiología Hepática
Melatonin, A Potential Therapeutic Agent For Smooth Muscle-Related Pathological Conditions And Aging
Fisiología Hepática
Melatonin, A Potential Therapeutic Agent For Smooth Muscle-Related Pathological Conditions And Aging
Tempol Protects The Gallbladder Against Ischemia/reperfusion
Melatonin, A Potential Therapeutic Agent For Smooth Muscle-Related Pathological Conditions And Aging
Tempol Protects The Gallbladder Against Ischemia/reperfusion
Simultaneous Measurement Of Serotonin And Melatonin From The Intestine Of Old Mice: The Effects Of Daily Melatonin Supplementation
Tempol Protects The Gallbladder Against Ischemia/reperfusion
Simultaneous Measurement Of Serotonin And Melatonin From The Intestine Of Old Mice: The Effects Of Daily Melatonin Supplementation
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
Simultaneous Measurement Of Serotonin And Melatonin From The Intestine Of Old Mice: The Effects Of Daily Melatonin Supplementation
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Spontaneous Calcium Oscillations In Urinary Bladder Smooth Muscle Cells
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Spontaneous Calcium Oscillations In Urinary Bladder Smooth Muscle Cells
Developmental Changes In Ca2+ Homeostasis And Contractility In Gallbladder Smooth Muscle
Spontaneous Calcium Oscillations In Urinary Bladder Smooth Muscle Cells
Developmental Changes In Ca2+ Homeostasis And Contractility In Gallbladder Smooth Muscle
Pancreatic Calcium Signaling: Role In Health And Disease
Developmental Changes In Ca2+ Homeostasis And Contractility In Gallbladder Smooth Muscle
Pancreatic Calcium Signaling: Role In Health And Disease
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Pancreatic Calcium Signaling: Role In Health And Disease
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Age-Related Alterations In Ca2+ Signals And Mitochondrial Membrane Potential In Exocrine Cells Are Prevented By Melatonin
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Age-Related Alterations In Ca2+ Signals And Mitochondrial Membrane Potential In Exocrine Cells Are Prevented By Melatonin
Melatonin Restores Impaired Contractility In Aged Guinea Pig Urinary Bladder
Age-Related Alterations In Ca2+ Signals And Mitochondrial Membrane Potential In Exocrine Cells Are Prevented By Melatonin
Melatonin Restores Impaired Contractility In Aged Guinea Pig Urinary Bladder
Protective Effect Of Melatonin On Ca2+ Homeostasis And Contractility In Acute Cholecystitis
Melatonin Restores Impaired Contractility In Aged Guinea Pig Urinary Bladder
Protective Effect Of Melatonin On Ca2+ Homeostasis And Contractility In Acute Cholecystitis
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Protective Effect Of Melatonin On Ca2+ Homeostasis And Contractility In Acute Cholecystitis
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Effects Of Melatonin On Gallbladder Neuromuscular Function In Acute Cholecystitis
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Effects Of Melatonin On Gallbladder Neuromuscular Function In Acute Cholecystitis
Ca2+ Extrusion In Aged Smooth Muscle Cells
Effects Of Melatonin On Gallbladder Neuromuscular Function In Acute Cholecystitis
Ca2+ Extrusion In Aged Smooth Muscle Cells
Aging Impairs Neurogenic Contraction In Guinea Pig Urinary Bladder: Role Of Oxidative Stress And Melatonin
Ca2+ Extrusion In Aged Smooth Muscle Cells
Aging Impairs Neurogenic Contraction In Guinea Pig Urinary Bladder: Role Of Oxidative Stress And Melatonin
Effect Of Melatonin On Age Associated Changes In Guinea Pig Bladder Function
Aging Impairs Neurogenic Contraction In Guinea Pig Urinary Bladder: Role Of Oxidative Stress And Melatonin
Effect Of Melatonin On Age Associated Changes In Guinea Pig Bladder Function
Melatonin And Digestive Motility
Effect Of Melatonin On Age Associated Changes In Guinea Pig Bladder Function
Melatonin And Digestive Motility
Calcium Controls Smooth Muscle Trpc Gene Transcription Via The Camk/calcineurin-Dependent Pathways
Melatonin And Digestive Motility
Calcium Controls Smooth Muscle Trpc Gene Transcription Via The Camk/calcineurin-Dependent Pathways
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Calcium Controls Smooth Muscle Trpc Gene Transcription Via The Camk/calcineurin-Dependent Pathways
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Melatonin Treatment Reverts Age-Related Changes In Guinea Pig Gallbladder Neuromuscular Transmission And Contractility
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Melatonin Treatment Reverts Age-Related Changes In Guinea Pig Gallbladder Neuromuscular Transmission And Contractility
Mitochondrial Reactive Oxygen Species And Ca2+ Signaling
Melatonin Treatment Reverts Age-Related Changes In Guinea Pig Gallbladder Neuromuscular Transmission And Contractility
Mitochondrial Reactive Oxygen Species And Ca2+ Signaling
Mitochondrial Production Of Oxidants Is Necessary For Physiological Calcium Oscillations
Mitochondrial Reactive Oxygen Species And Ca2+ Signaling
Mitochondrial Production Of Oxidants Is Necessary For Physiological Calcium Oscillations
Changes In Guinea Pig Gallbladder Smooth Muscle Ca2+ Homeostasis By Acute Acalculous Cholecystitis
Mitochondrial Production Of Oxidants Is Necessary For Physiological Calcium Oscillations
Changes In Guinea Pig Gallbladder Smooth Muscle Ca2+ Homeostasis By Acute Acalculous Cholecystitis
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Changes In Guinea Pig Gallbladder Smooth Muscle Ca2+ Homeostasis By Acute Acalculous Cholecystitis
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor
The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death
Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor
The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death
Inhibition Of Macroautophagy Triggers Apoptosis
The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death
Inhibition Of Macroautophagy Triggers Apoptosis
Non-Stimulated Ca2+ Leak Pathway In Cerebellar Granule Neurones
Inhibition Of Macroautophagy Triggers Apoptosis
Non-Stimulated Ca2+ Leak Pathway In Cerebellar Granule Neurones
Characterization Of Intracellular Ca2+ Stores In Gallbladder Smooth Muscle
Non-Stimulated Ca2+ Leak Pathway In Cerebellar Granule Neurones
Characterization Of Intracellular Ca2+ Stores In Gallbladder Smooth Muscle
Disruption Of The Filamentous Actin Cytoskeleton Is Necessary For The Activation Of Capacitative Calcium Entry In Naive Smooth Muscle Cells
Characterization Of Intracellular Ca2+ Stores In Gallbladder Smooth Muscle
Disruption Of The Filamentous Actin Cytoskeleton Is Necessary For The Activation Of Capacitative Calcium Entry In Naive Smooth Muscle Cells
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Disruption Of The Filamentous Actin Cytoskeleton Is Necessary For The Activation Of Capacitative Calcium Entry In Naive Smooth Muscle Cells
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Viral Proteins Targeting Mitochondria: Controlling Cell Death
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Viral Proteins Targeting Mitochondria: Controlling Cell Death
Cyclic Amp-Mediated Inhibition Of Gallbladder Contractility: Role Of K + Channel Activation And Ca 2+ Signaling
Viral Proteins Targeting Mitochondria: Controlling Cell Death
Cyclic Amp-Mediated Inhibition Of Gallbladder Contractility: Role Of K + Channel Activation And Ca 2+ Signaling
Coactivation Of Capacitative Calcium Entry And L-Type Calcium Channels In Guinea Pig Gallbladder
Cyclic Amp-Mediated Inhibition Of Gallbladder Contractility: Role Of K + Channel Activation And Ca 2+ Signaling
Coactivation Of Capacitative Calcium Entry And L-Type Calcium Channels In Guinea Pig Gallbladder
Chemical Mediators Of Gallbladder Dysmotility
Coactivation Of Capacitative Calcium Entry And L-Type Calcium Channels In Guinea Pig Gallbladder
Chemical Mediators Of Gallbladder Dysmotility
Mitochondrial Polarisation Status And [Ca2+]i Signalling In Rat Cerebellar Granule Neurones Aged In Vitro
Chemical Mediators Of Gallbladder Dysmotility
Mitochondrial Polarisation Status And [Ca2+]i Signalling In Rat Cerebellar Granule Neurones Aged In Vitro
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Mitochondrial Polarisation Status And [Ca2+]i Signalling In Rat Cerebellar Granule Neurones Aged In Vitro
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family
Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine
The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family
Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine
Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin
Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine
Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Contribution Of Different Phospholipases And Arachidonic Acid Metabolites In The Response Of Gallbladder Smooth Muscle To Cholecystokinin
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Contribution Of Different Phospholipases And Arachidonic Acid Metabolites In The Response Of Gallbladder Smooth Muscle To Cholecystokinin
Role Of Mitochondria In Ca2+ Oscillations And Shape Of Ca2+ Signals In Pancreatic Acinar Cells
Contribution Of Different Phospholipases And Arachidonic Acid Metabolites In The Response Of Gallbladder Smooth Muscle To Cholecystokinin
Role Of Mitochondria In Ca2+ Oscillations And Shape Of Ca2+ Signals In Pancreatic Acinar Cells
Xod-Catalyzed Ros Generation Mobilizes Calcium From Intracellular Stores In Mouse Pancreatic Acinar Cells
Role Of Mitochondria In Ca2+ Oscillations And Shape Of Ca2+ Signals In Pancreatic Acinar Cells
Xod-Catalyzed Ros Generation Mobilizes Calcium From Intracellular Stores In Mouse Pancreatic Acinar Cells
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Xod-Catalyzed Ros Generation Mobilizes Calcium From Intracellular Stores In Mouse Pancreatic Acinar Cells
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Effect Of Xanthine Oxidase-Catalyzed Reactive Oxygen Species Generation On Secretagogue-Evoked Calcium Mobilization In Mouse Pancreatic Acinar Cells
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Effect Of Xanthine Oxidase-Catalyzed Reactive Oxygen Species Generation On Secretagogue-Evoked Calcium Mobilization In Mouse Pancreatic Acinar Cells
Relaxation Of Canine Gallbladder To Nerve Stimulation Involves Adrenergic And Non-Adrenergic Non-Cholinergic Mechanisms
Effect Of Xanthine Oxidase-Catalyzed Reactive Oxygen Species Generation On Secretagogue-Evoked Calcium Mobilization In Mouse Pancreatic Acinar Cells
Relaxation Of Canine Gallbladder To Nerve Stimulation Involves Adrenergic And Non-Adrenergic Non-Cholinergic Mechanisms
Vanadate Inhibits The Calcium Extrusion In Rat Pancreatic Acinar Cells
Relaxation Of Canine Gallbladder To Nerve Stimulation Involves Adrenergic And Non-Adrenergic Non-Cholinergic Mechanisms
Vanadate Inhibits The Calcium Extrusion In Rat Pancreatic Acinar Cells
A Redox-Based Mechanism For The Contractile And Relaxing Effects Of No In The Guinea-Pig Gall Bladder
Vanadate Inhibits The Calcium Extrusion In Rat Pancreatic Acinar Cells
A Redox-Based Mechanism For The Contractile And Relaxing Effects Of No In The Guinea-Pig Gall Bladder
Release Of Calcium From Mitochondrial And Nonmitochondrial Intracellular Stores In Mouse Pancreatic Acinar Cells By Hydrogen Peroxide
A Redox-Based Mechanism For The Contractile And Relaxing Effects Of No In The Guinea-Pig Gall Bladder
Release Of Calcium From Mitochondrial And Nonmitochondrial Intracellular Stores In Mouse Pancreatic Acinar Cells By Hydrogen Peroxide
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Release Of Calcium From Mitochondrial And Nonmitochondrial Intracellular Stores In Mouse Pancreatic Acinar Cells By Hydrogen Peroxide
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Differential Involvement Of Vacuolar H+-Atpase In The Refilling Of Thapsigargin- And Agonist-Mobilized Ca2+ Stores
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Differential Involvement Of Vacuolar H+-Atpase In The Refilling Of Thapsigargin- And Agonist-Mobilized Ca2+ Stores
Activation Of Tyrosine Kinase Pathway By Vanadate In Gallbladder Smooth Muscle
Differential Involvement Of Vacuolar H+-Atpase In The Refilling Of Thapsigargin- And Agonist-Mobilized Ca2+ Stores
Activation Of Tyrosine Kinase Pathway By Vanadate In Gallbladder Smooth Muscle
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Activation Of Tyrosine Kinase Pathway By Vanadate In Gallbladder Smooth Muscle
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Role Of Proton Gradients And Vacuolar H+-Atpases In The Refilling Of Intracellular Calcium Stores In Exocrine Cells
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Role Of Proton Gradients And Vacuolar H+-Atpases In The Refilling Of Intracellular Calcium Stores In Exocrine Cells
Effects Of Antioxidants On Calcium Signal Induced By Cholecystokinin In Mouse Pancreatic Acinar Cells
Role Of Proton Gradients And Vacuolar H+-Atpases In The Refilling Of Intracellular Calcium Stores In Exocrine Cells
Effects Of Antioxidants On Calcium Signal Induced By Cholecystokinin In Mouse Pancreatic Acinar Cells
Effect Of Dephostatin On Intracellular Free Calcium Concentration And Amylase Secretion In Isolated Rat Pancreatic Acinar Cells
Effects Of Antioxidants On Calcium Signal Induced By Cholecystokinin In Mouse Pancreatic Acinar Cells
Effect Of Dephostatin On Intracellular Free Calcium Concentration And Amylase Secretion In Isolated Rat Pancreatic Acinar Cells
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Effect Of Dephostatin On Intracellular Free Calcium Concentration And Amylase Secretion In Isolated Rat Pancreatic Acinar Cells
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Growth Factors And The Exocrine Pancreas
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Growth Factors And The Exocrine Pancreas
Phenylarsine Oxide Evokes Intracellular Calcium Increases And Amylase Secretion In Isolated Rat Pancreatic Acinar Cells
Growth Factors And The Exocrine Pancreas
Phenylarsine Oxide Evokes Intracellular Calcium Increases And Amylase Secretion In Isolated Rat Pancreatic Acinar Cells
Calcium Binding Capacity Of The Cytosol And Endoplasmic Reticulum Of Mouse Pancreatic Acinar Cells
Phenylarsine Oxide Evokes Intracellular Calcium Increases And Amylase Secretion In Isolated Rat Pancreatic Acinar Cells
Calcium Binding Capacity Of The Cytosol And Endoplasmic Reticulum Of Mouse Pancreatic Acinar Cells
Oxidizing Effects Of Vanadate On Calcium Mobilization And Amylase Release In Rat Pancreatic Acinar Cells
Calcium Binding Capacity Of The Cytosol And Endoplasmic Reticulum Of Mouse Pancreatic Acinar Cells
Oxidizing Effects Of Vanadate On Calcium Mobilization And Amylase Release In Rat Pancreatic Acinar Cells
Sequential Activation Of Different Ca2+ Entry Pathways Upon Cholinergic Stimulation In Mouse Pancreatic Acinar Cells
Oxidizing Effects Of Vanadate On Calcium Mobilization And Amylase Release In Rat Pancreatic Acinar Cells
Sequential Activation Of Different Ca2+ Entry Pathways Upon Cholinergic Stimulation In Mouse Pancreatic Acinar Cells
Factores De Crecimiento Y Páncreas Exocrino
Sequential Activation Of Different Ca2+ Entry Pathways Upon Cholinergic Stimulation In Mouse Pancreatic Acinar Cells
Factores De Crecimiento Y Páncreas Exocrino
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Factores De Crecimiento Y Páncreas Exocrino
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Sex-Related Difference In Histaminergic Control Of Exocrine Pancreatic Secretion In The Guinea-Pig
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Sex-Related Difference In Histaminergic Control Of Exocrine Pancreatic Secretion In The Guinea-Pig
Neurotransmitters Involved In The Mechanical Response Of Guinea Pig Gallbladder To Electrical Field Stimulation
Sex-Related Difference In Histaminergic Control Of Exocrine Pancreatic Secretion In The Guinea-Pig
Neurotransmitters Involved In The Mechanical Response Of Guinea Pig Gallbladder To Electrical Field Stimulation
Histamine Does Not Potentiate Cyclic Amp-Mediated Amylase Secretion In The Guinea-Pig Pancreatic Acinar Cells
Neurotransmitters Involved In The Mechanical Response Of Guinea Pig Gallbladder To Electrical Field Stimulation
Histamine Does Not Potentiate Cyclic Amp-Mediated Amylase Secretion In The Guinea-Pig Pancreatic Acinar Cells
Free Cytosolic Calcium Levels Modify Intracellular Ph In Rat Pancreatic Acini
Histamine Does Not Potentiate Cyclic Amp-Mediated Amylase Secretion In The Guinea-Pig Pancreatic Acinar Cells
Free Cytosolic Calcium Levels Modify Intracellular Ph In Rat Pancreatic Acini
Intracellular Ph And Calcium Signalling In Rat Pancreatic Acinar Cells
Free Cytosolic Calcium Levels Modify Intracellular Ph In Rat Pancreatic Acini
Intracellular Ph And Calcium Signalling In Rat Pancreatic Acinar Cells
Stimulus-Secretion Pathway For Histamine In The Exocrine Pancreas
Intracellular Ph And Calcium Signalling In Rat Pancreatic Acinar Cells
Stimulus-Secretion Pathway For Histamine In The Exocrine Pancreas
Simultaneous Presence Of Camp And Cgmp Exert A Co-Ordinated Inhibitory Effect On The Agonist Evoked Ca2+ Signal In Pancreatic Acinar Cells
Stimulus-Secretion Pathway For Histamine In The Exocrine Pancreas
Simultaneous Presence Of Camp And Cgmp Exert A Co-Ordinated Inhibitory Effect On The Agonist Evoked Ca2+ Signal In Pancreatic Acinar Cells
Calcium Dependence Of Calcium Extrusion And Calcium Uptake In Mouse Pancreatic Acinar Cells
Simultaneous Presence Of Camp And Cgmp Exert A Co-Ordinated Inhibitory Effect On The Agonist Evoked Ca2+ Signal In Pancreatic Acinar Cells
Calcium Dependence Of Calcium Extrusion And Calcium Uptake In Mouse Pancreatic Acinar Cells
Cholinergic Dependence Of Pancreatic Response To Cholecystokinin In Rats And Guinea Pigs
Calcium Dependence Of Calcium Extrusion And Calcium Uptake In Mouse Pancreatic Acinar Cells
Cholinergic Dependence Of Pancreatic Response To Cholecystokinin In Rats And Guinea Pigs
Hormonas Gastrointestinales
Cholinergic Dependence Of Pancreatic Response To Cholecystokinin In Rats And Guinea Pigs
Hormonas Gastrointestinales
Secreción Pancreática Exocrina
Hormonas Gastrointestinales
Secreción Pancreática Exocrina
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
Secreción Pancreática Exocrina
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
Hormonal Control Of Exocrine Pancreatic Secretion In The Isolated Intact Rat Pancreas
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
Hormonal Control Of Exocrine Pancreatic Secretion In The Isolated Intact Rat Pancreas
The Immune System And Autoaggression
Hormonal Control Of Exocrine Pancreatic Secretion In The Isolated Intact Rat Pancreas
The Immune System And Autoaggression
Interaction Between Secretin And Nerve‐mediated Amylase Secretion In The Isolated Exocrine Rat Pancreas
The Immune System And Autoaggression
Interaction Between Secretin And Nerve‐mediated Amylase Secretion In The Isolated Exocrine Rat Pancreas
Receptores Sensoriales
Interaction Between Secretin And Nerve‐mediated Amylase Secretion In The Isolated Exocrine Rat Pancreas
Receptores Sensoriales
Transmisión Sináptica
Receptores Sensoriales
Transmisión Sináptica
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
Transmisión Sináptica
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Effect Of Secretin On Vagal Stimulation-Evoked Exocrine Pancreatic Secretion In The Rat
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Effect Of Secretin On Vagal Stimulation-Evoked Exocrine Pancreatic Secretion In The Rat
Effects Of Phorbol Esters And Secretin On Acetylcholine-Evoked Exocrine Pancreatic Secretion In The Anaesthetized Rat
Effect Of Secretin On Vagal Stimulation-Evoked Exocrine Pancreatic Secretion In The Rat
Effects Of Phorbol Esters And Secretin On Acetylcholine-Evoked Exocrine Pancreatic Secretion In The Anaesthetized Rat
Mechanism Of Action Of Magnesium On Cholecystokinin-Octapeptide-Evoked Secretory Responses In The Isolated Exocrine Rat Pancreas
Effects Of Phorbol Esters And Secretin On Acetylcholine-Evoked Exocrine Pancreatic Secretion In The Anaesthetized Rat
Mechanism Of Action Of Magnesium On Cholecystokinin-Octapeptide-Evoked Secretory Responses In The Isolated Exocrine Rat Pancreas
Inhibitory Interactions Between Stimulus-Secretion Pathways In The Exocrine Rat Pancreas
Mechanism Of Action Of Magnesium On Cholecystokinin-Octapeptide-Evoked Secretory Responses In The Isolated Exocrine Rat Pancreas
Inhibitory Interactions Between Stimulus-Secretion Pathways In The Exocrine Rat Pancreas
Effect Of Phorbol Ester On Vagal Stimulation And Acetylcholine-Evoked Exocrine Pancreatic Secretion And Cytosolic Free Calcium In The Rat
Inhibitory Interactions Between Stimulus-Secretion Pathways In The Exocrine Rat Pancreas
Effect Of Phorbol Ester On Vagal Stimulation And Acetylcholine-Evoked Exocrine Pancreatic Secretion And Cytosolic Free Calcium In The Rat
Effects Of Secretin On Vagal Stimulation-Evoked Exocrime Pancreatic Secretion In The Rat
Effect Of Phorbol Ester On Vagal Stimulation And Acetylcholine-Evoked Exocrine Pancreatic Secretion And Cytosolic Free Calcium In The Rat
Effects Of Secretin On Vagal Stimulation-Evoked Exocrime Pancreatic Secretion In The Rat
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
Effects Of Secretin On Vagal Stimulation-Evoked Exocrime Pancreatic Secretion In The Rat
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
El Páncreas Perfundido Y Aislado Como Modelo De Estudio De La Secreción
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
El Páncreas Perfundido Y Aislado Como Modelo De Estudio De La Secreción
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
El Páncreas Perfundido Y Aislado Como Modelo De Estudio De La Secreción
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
Secretagogue-Evoked Time-Course Changes On Pancreatic Juice Secretion In The Anaesthetized Rat
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
Secretagogue-Evoked Time-Course Changes On Pancreatic Juice Secretion In The Anaesthetized Rat
Interaction Between Secretin And Cholecystokinin‐octapeptide In The Exocrine Rat Pancreas In Vivo And In Vitro
Secretagogue-Evoked Time-Course Changes On Pancreatic Juice Secretion In The Anaesthetized Rat
Interaction Between Secretin And Cholecystokinin‐octapeptide In The Exocrine Rat Pancreas In Vivo And In Vitro
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Interaction Between Secretin And Cholecystokinin‐octapeptide In The Exocrine Rat Pancreas In Vivo And In Vitro
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
Effects Of Phorbol Ester On Cholecystokinin Octapeptide‐evoked Exocrine Pancreatic Secretion In The Rat.
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
Effects Of Phorbol Ester On Cholecystokinin Octapeptide‐evoked Exocrine Pancreatic Secretion In The Rat.
Effects Of Phorbol Esters And Secretin On Pancreatic Juice Secretion In The Anaesthetized Rat
Effects Of Phorbol Ester On Cholecystokinin Octapeptide‐evoked Exocrine Pancreatic Secretion In The Rat.
Effects Of Phorbol Esters And Secretin On Pancreatic Juice Secretion In The Anaesthetized Rat
Interactions Between Cholecystokinin And Phorbol Ester In The Pancreas Of The Anaesthetized Rat And Permeabilized Acini
Effects Of Phorbol Esters And Secretin On Pancreatic Juice Secretion In The Anaesthetized Rat
Interactions Between Cholecystokinin And Phorbol Ester In The Pancreas Of The Anaesthetized Rat And Permeabilized Acini
Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)
Interactions Between Cholecystokinin And Phorbol Ester In The Pancreas Of The Anaesthetized Rat And Permeabilized Acini
Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)
Filamin A C-Terminal Fragment Modulates Orai1 Expression By Inhibition Of Protein Degradation
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