PUBLICACIÓN

MPP+: Mechanism for its toxicity in cerebellar granule cells

ACCEDER A LA PUBLICACIÓN: Scopus Orcid

González-Polo, R.A., Soler, G., Fuentes, J.M.

2004 Molecular Neurobiology

CITAS

29

DOI

10.1385/mn:30:3:253

EID

2-s2.0-15244340545

ISSN

0893-7648

BIBTEX

@article{gonzalez-polo_mpp_2004, title= {{MPP}+: {Mechanism} for its toxicity in cerebellar granule cells}, volume= {30}, url= {https://www.scopus.com/inward/record.uri?eid=2-s2.0-15244340545&doi=10.1385%2fMN%3a30%3a3%3a253&partnerID=40&md5=67e285882b9a95ef6e770a191d04c1d9}, doi= {10.1385/MN:30:3:253}, number= {3}, journal= {Molecular Neurobiology}, author= {González-Polo, R.A. and Soler, G. and Fuentes, J.M.}, year= {2004}, pages= {253--264}}

AUTORES DE LA UEX

María Luisa Campo Guinea
Rosa Ana González Polo
José Manuel Fuentes Rodríguez

OTRAS PUBLICACIONES DE INTERÉS

The Influence Of Fasting And Cortisol Upon Arginosuccinate Synthetase, Arginosuccinase And Arginase Levels In Guinea Pig Liver
Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice
Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques
Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice
Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques
The Role Of Cardiolipin In Mitochondrial Function And Neurodegenerative Diseases
Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques
The Role Of Cardiolipin In Mitochondrial Function And Neurodegenerative Diseases
Immunological Aspects Of Central Neurodegeneration
The Role Of Cardiolipin In Mitochondrial Function And Neurodegenerative Diseases
Immunological Aspects Of Central Neurodegeneration
Role Of Tfeb In Huntington’s Disease
Immunological Aspects Of Central Neurodegeneration
Role Of Tfeb In Huntington’s Disease
Analysis Of Mitochondrial Translocases Tom And Tim By The Patch-Clamping Technique
Role Of Tfeb In Huntington’s Disease
Analysis Of Mitochondrial Translocases Tom And Tim By The Patch-Clamping Technique
Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects
Analysis Of Mitochondrial Translocases Tom And Tim By The Patch-Clamping Technique
Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects
Lysosomal Dysfunction: Connecting The Dots In The Landscape Of Human Diseases
Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects
Lysosomal Dysfunction: Connecting The Dots In The Landscape Of Human Diseases
Corrigendum: Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells (Journal Of Cellular Physiology, 10.1002/jcp.26855)
Lysosomal Dysfunction: Connecting The Dots In The Landscape Of Human Diseases
Corrigendum: Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells (Journal Of Cellular Physiology, 10.1002/jcp.26855)
Vitamin D Receptor Polymorphisms In A Spanish Cohort Of Parkinson’s Disease Patients
Corrigendum: Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells (Journal Of Cellular Physiology, 10.1002/jcp.26855)
Vitamin D Receptor Polymorphisms In A Spanish Cohort Of Parkinson’s Disease Patients
Links Between Paraquat And Parkinson’s Disease
Vitamin D Receptor Polymorphisms In A Spanish Cohort Of Parkinson’s Disease Patients
Links Between Paraquat And Parkinson’s Disease
Mitochondrial Dysfunction In Repeat Expansion Diseases
Links Between Paraquat And Parkinson’s Disease
Mitochondrial Dysfunction In Repeat Expansion Diseases
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction
Mitochondrial Dysfunction In Repeat Expansion Diseases
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction
Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction
Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease
Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease
Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease
Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress
Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress
Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress
Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles
In Vitro And In Vivo Models To Study The Biological And Pharmacological Properties Of Queen Bee Acid (Qba, 10-Hydroxy-2-Decenoic Acid): A Systematic Review
Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles
In Vitro And In Vivo Models To Study The Biological And Pharmacological Properties Of Queen Bee Acid (Qba, 10-Hydroxy-2-Decenoic Acid): A Systematic Review
Role Of Inflammatory Cytokines In The Conversion Of Mild Cognitive Impairment To Dementia: A Prospective Study.
In Vitro And In Vivo Models To Study The Biological And Pharmacological Properties Of Queen Bee Acid (Qba, 10-Hydroxy-2-Decenoic Acid): A Systematic Review
Role Of Inflammatory Cytokines In The Conversion Of Mild Cognitive Impairment To Dementia: A Prospective Study.
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
Role Of Inflammatory Cytokines In The Conversion Of Mild Cognitive Impairment To Dementia: A Prospective Study.
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
Neuroprotective And Anti-Inflammatory Effects Of Linoleic Acid In Models Of Parkinson’s Disease: The Implication Of Lipid Droplets And Lipophagy
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
Neuroprotective And Anti-Inflammatory Effects Of Linoleic Acid In Models Of Parkinson’s Disease: The Implication Of Lipid Droplets And Lipophagy
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Neuroprotective And Anti-Inflammatory Effects Of Linoleic Acid In Models Of Parkinson’s Disease: The Implication Of Lipid Droplets And Lipophagy
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1
Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1
Links Between Paraquat And Parkinson’s Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1
Links Between Paraquat And Parkinson’s Disease
Procedimientos, Experimentación E Investigación En Enfermedades Neurodegenerativas: Protocolos Para Un Laboratorio De Neurociencias
Links Between Paraquat And Parkinson’s Disease
Procedimientos, Experimentación E Investigación En Enfermedades Neurodegenerativas: Protocolos Para Un Laboratorio De Neurociencias
Cultivos Celulares
Procedimientos, Experimentación E Investigación En Enfermedades Neurodegenerativas: Protocolos Para Un Laboratorio De Neurociencias
Cultivos Celulares
Microscopía Electrónica
Cultivos Celulares
Microscopía Electrónica
Análisis De Extracto Proteico Por Western Blotting
Microscopía Electrónica
Análisis De Extracto Proteico Por Western Blotting
Extracción De Proteínas
Análisis De Extracto Proteico Por Western Blotting
Extracción De Proteínas
Técnica De Sobreexpresión Génica
Extracción De Proteínas
Técnica De Sobreexpresión Génica
Técnica De Silenciamiento Génico
Técnica De Sobreexpresión Génica
Técnica De Silenciamiento Génico
The Dual Role Of Necrostatin-1 In Parkinson's Disease Models
Técnica De Silenciamiento Génico
The Dual Role Of Necrostatin-1 In Parkinson's Disease Models
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction.
The Dual Role Of Necrostatin-1 In Parkinson's Disease Models
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction.
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress.
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction.
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress.
Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress.
Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease
Biología: Tenemos Una Buena Representación En La Base, Pero Si Asciendes En La Cúspide Va Habiendo Menos Visibilidad
Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease
Biología: Tenemos Una Buena Representación En La Base, Pero Si Asciendes En La Cúspide Va Habiendo Menos Visibilidad
Toxicity Of Necrostatin-1 In Parkinson’s Disease Models
Biología: Tenemos Una Buena Representación En La Base, Pero Si Asciendes En La Cúspide Va Habiendo Menos Visibilidad
Toxicity Of Necrostatin-1 In Parkinson’s Disease Models
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells
Toxicity Of Necrostatin-1 In Parkinson’s Disease Models
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells
Natural Products In The Promotion Of Healthspan And Longevity.
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells
Natural Products In The Promotion Of Healthspan And Longevity.
Aprendizaje Basado En Juegos De Rol: Una Aplicación A La Economía Del Cambio Climático
Natural Products In The Promotion Of Healthspan And Longevity.
Aprendizaje Basado En Juegos De Rol: Una Aplicación A La Economía Del Cambio Climático
Dual Role Of Mitochondrial Porin In Metabolite Transport Across The Outer Membrane And Protein Transfer To The Inner Membrane
Aprendizaje Basado En Juegos De Rol: Una Aplicación A La Economía Del Cambio Climático
Dual Role Of Mitochondrial Porin In Metabolite Transport Across The Outer Membrane And Protein Transfer To The Inner Membrane
Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack
Dual Role Of Mitochondrial Porin In Metabolite Transport Across The Outer Membrane And Protein Transfer To The Inner Membrane
Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack
The Paradigm Of Protein Acetylation In Parkinson's Disease
Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack
The Paradigm Of Protein Acetylation In Parkinson's Disease
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients
The Paradigm Of Protein Acetylation In Parkinson's Disease
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson's Disease Patients.
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson's Disease Patients.
Cholesterol And Multilamellar Bodies: Lysosomal Dysfunction In Gba-Parkinson Disease
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson's Disease Patients.
Cholesterol And Multilamellar Bodies: Lysosomal Dysfunction In Gba-Parkinson Disease
Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria
Cholesterol And Multilamellar Bodies: Lysosomal Dysfunction In Gba-Parkinson Disease
Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria
Vascular Risk Factors And Lesions Of Vascular Nature In Magnetic Resonance As Predictors Of Progression To Dementia In Patients With Mild Cognitive Impairment
Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria
Vascular Risk Factors And Lesions Of Vascular Nature In Magnetic Resonance As Predictors Of Progression To Dementia In Patients With Mild Cognitive Impairment
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells: In Vitro And In Vivo Studies
Vascular Risk Factors And Lesions Of Vascular Nature In Magnetic Resonance As Predictors Of Progression To Dementia In Patients With Mild Cognitive Impairment
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells: In Vitro And In Vivo Studies
Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells: In Vitro And In Vivo Studies
Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
Revisiting Trends On Mitochondrial Mega-Channels For The Import Of Proteins And Nucleic Acids
[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
Revisiting Trends On Mitochondrial Mega-Channels For The Import Of Proteins And Nucleic Acids
N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
Revisiting Trends On Mitochondrial Mega-Channels For The Import Of Proteins And Nucleic Acids
N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells
Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells
Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Trend Of Lipid Profile In General Population From Caceres Health Area
Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Trend Of Lipid Profile In General Population From Caceres Health Area
A Disulfide Bond In The Tim23 Complex Is Crucial For Voltage Gating And Mitochondrial Protein Import
Trend Of Lipid Profile In General Population From Caceres Health Area
A Disulfide Bond In The Tim23 Complex Is Crucial For Voltage Gating And Mitochondrial Protein Import
The Basics Of Autophagy
A Disulfide Bond In The Tim23 Complex Is Crucial For Voltage Gating And Mitochondrial Protein Import
The Basics Of Autophagy
Autophagy-Related Proteins Are Functionally Active In Human Spermatozoa And May Be Involved In The Regulation Of Cell Survival And Motility
The Basics Of Autophagy
Autophagy-Related Proteins Are Functionally Active In Human Spermatozoa And May Be Involved In The Regulation Of Cell Survival And Motility
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Autophagy-Related Proteins Are Functionally Active In Human Spermatozoa And May Be Involved In The Regulation Of Cell Survival And Motility
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis
Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis
Mitochondria: Key Organelle In Parkinson's Disease
Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis
Mitochondria: Key Organelle In Parkinson's Disease
G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway
Mitochondria: Key Organelle In Parkinson's Disease
G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway
Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment
G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway
Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Control Of Autophagy In Parkinson’s Disease
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Control Of Autophagy In Parkinson’s Disease
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Control Of Autophagy In Parkinson’s Disease
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Toxicity And Autophagy In Neurodegenerative Disorders
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Toxicity And Autophagy In Neurodegenerative Disorders
Origen Molecular De La Compleja Enfermedad De Parkinson
Toxicity And Autophagy In Neurodegenerative Disorders
Origen Molecular De La Compleja Enfermedad De Parkinson
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
Origen Molecular De La Compleja Enfermedad De Parkinson
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Links Between Paraquat And Parkinson's Disease
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Links Between Paraquat And Parkinson's Disease
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Links Between Paraquat And Parkinson's Disease
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Implication Of Autophagy In Parkinson's Disease
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Implication Of Autophagy In Parkinson's Disease
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
Implication Of Autophagy In Parkinson's Disease
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Paraquat, Between Apoptosis And Autophagy
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Paraquat, Between Apoptosis And Autophagy
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Paraquat, Between Apoptosis And Autophagy
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Inhibition Of Autophagy By Tab2 And Tab3
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Inhibition Of Autophagy By Tab2 And Tab3
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Inhibition Of Autophagy By Tab2 And Tab3
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Arginase I Induction By Modified Lipoproteins In Macrophages: A Peroxisome Proliferator-Activated Receptor-Γ/δ-Mediated Effect That Links Lipid Metabolism And Immunity
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Arginase I Induction By Modified Lipoproteins In Macrophages: A Peroxisome Proliferator-Activated Receptor-Γ/δ-Mediated Effect That Links Lipid Metabolism And Immunity
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Arginase I Induction By Modified Lipoproteins In Macrophages: A Peroxisome Proliferator-Activated Receptor-Γ/δ-Mediated Effect That Links Lipid Metabolism And Immunity
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Apoptosis
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Apoptosis
Awaking Tim22, A Dynamic Ligand-Gated Channel For Protein Insertion In The Mitochondrial Inner Membrane
Apoptosis
Awaking Tim22, A Dynamic Ligand-Gated Channel For Protein Insertion In The Mitochondrial Inner Membrane
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Awaking Tim22, A Dynamic Ligand-Gated Channel For Protein Insertion In The Mitochondrial Inner Membrane
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
A Fluorescence Assay For Peptide Translocation Into Mitochondria
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
A Fluorescence Assay For Peptide Translocation Into Mitochondria
Tim17p Regulates The Twin Pore Structure And Voltage Gating Of The Mitochondrial Protein Import Complex Tim23
A Fluorescence Assay For Peptide Translocation Into Mitochondria
Tim17p Regulates The Twin Pore Structure And Voltage Gating Of The Mitochondrial Protein Import Complex Tim23
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Tim17p Regulates The Twin Pore Structure And Voltage Gating Of The Mitochondrial Protein Import Complex Tim23
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Arginase I Induction During Leishmania Major Infection Mediates The Development Of Disease
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Arginase I Induction During Leishmania Major Infection Mediates The Development Of Disease
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase I Induction During Leishmania Major Infection Mediates The Development Of Disease
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor
The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death
Pk11195 Potently Sensitizes To Apoptosis Induction Independently From The Peripheral Benzodiazepin Receptor
The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death
Inhibition Of Macroautophagy Triggers Apoptosis
The Apoptosis/autophagy Paradox: Autophagic Vacuolization Before Apoptotic Death
Inhibition Of Macroautophagy Triggers Apoptosis
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Inhibition Of Macroautophagy Triggers Apoptosis
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Viral Proteins Targeting Mitochondria: Controlling Cell Death
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Viral Proteins Targeting Mitochondria: Controlling Cell Death
Electrophysiological Approaches To The Study Of Protein Translocation In Mitochondria
Viral Proteins Targeting Mitochondria: Controlling Cell Death
Electrophysiological Approaches To The Study Of Protein Translocation In Mitochondria
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Electrophysiological Approaches To The Study Of Protein Translocation In Mitochondria
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Comparison Of The Tim And Tom Channel Activities Of The Mitochondrial Protein Import Complexes
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Comparison Of The Tim And Tom Channel Activities Of The Mitochondrial Protein Import Complexes
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Comparison Of The Tim And Tom Channel Activities Of The Mitochondrial Protein Import Complexes
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family
Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine
The Chemopreventive Agent N-(4-Hydroxyphenyl)retinamide Induces Apoptosis Through A Mitochondrial Pathway Regulated By Proteins From The Bcl-2 Family
Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine
Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin
Mitochondrial Membrane Permeabilization Is A Critical Step Of Lysosome-Initiated Apoptosis Induced By Hydroxychloroquine
Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Mitochondrion-Targeted Apoptosis Regulators Of Viral Origin
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Lithium Blocks The Pkb And Gsk3 Dephosphorylation Induced By Ceramide Through Protein Phosphatase-2a
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Lithium Blocks The Pkb And Gsk3 Dephosphorylation Induced By Ceramide Through Protein Phosphatase-2a
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Lithium Blocks The Pkb And Gsk3 Dephosphorylation Induced By Ceramide Through Protein Phosphatase-2a
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Different Dependence Of Lithium And Valproate On P13k/pkb Pathway
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Different Dependence Of Lithium And Valproate On P13k/pkb Pathway
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Different Dependence Of Lithium And Valproate On P13k/pkb Pathway
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Glu-256 Is A Main Structural Determinant For Oligomerisation Of Human Arginase I
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Glu-256 Is A Main Structural Determinant For Oligomerisation Of Human Arginase I
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Glu-256 Is A Main Structural Determinant For Oligomerisation Of Human Arginase I
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Inhibition Of Phosphatidylcholine Synthesis Precedes Apoptosis Induced By C2-Ceramide: Protection By Exogenous Phosphatidylcholine
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Inhibition Of Phosphatidylcholine Synthesis Precedes Apoptosis Induced By C2-Ceramide: Protection By Exogenous Phosphatidylcholine
Mcc And Psc, The Putative Protein Import Channels Of Mitochondria
Inhibition Of Phosphatidylcholine Synthesis Precedes Apoptosis Induced By C2-Ceramide: Protection By Exogenous Phosphatidylcholine
Mcc And Psc, The Putative Protein Import Channels Of Mitochondria
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Mcc And Psc, The Putative Protein Import Channels Of Mitochondria
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Signal Presequences Increase Mitochondrial Permeability And Open The Multiple Conductance Channel
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Signal Presequences Increase Mitochondrial Permeability And Open The Multiple Conductance Channel
Th1/th2-Regulated Expression Of Arginase Isoforms In Murine Macrophages And Dendritic Cells
Signal Presequences Increase Mitochondrial Permeability And Open The Multiple Conductance Channel
Th1/th2-Regulated Expression Of Arginase Isoforms In Murine Macrophages And Dendritic Cells
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Th1/th2-Regulated Expression Of Arginase Isoforms In Murine Macrophages And Dendritic Cells
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Anti-Mhv3 State Induced By Ifn Gamma In Macrophages Is Not Related To Arginine Metabolism
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Anti-Mhv3 State Induced By Ifn Gamma In Macrophages Is Not Related To Arginine Metabolism
Effects Of Carbonyl Cyanide Phenylhydrazones On Two Mitochondrial Ion Channel Activities
Anti-Mhv3 State Induced By Ifn Gamma In Macrophages Is Not Related To Arginine Metabolism
Effects Of Carbonyl Cyanide Phenylhydrazones On Two Mitochondrial Ion Channel Activities
Involvement Of Protein Kinase A In The Induction Of Arginase In Murine Bone Marrow-Derived Macrophages
Effects Of Carbonyl Cyanide Phenylhydrazones On Two Mitochondrial Ion Channel Activities
Involvement Of Protein Kinase A In The Induction Of Arginase In Murine Bone Marrow-Derived Macrophages
Oxidation Of N(G)-Hydroxy-L-Arginine To Nitric Oxide Mediated By Respiratory Burst: An Alternative Pathway To No Synthesis
Involvement Of Protein Kinase A In The Induction Of Arginase In Murine Bone Marrow-Derived Macrophages
Oxidation Of N(G)-Hydroxy-L-Arginine To Nitric Oxide Mediated By Respiratory Burst: An Alternative Pathway To No Synthesis
Perspectives On The Mitochondrial Multiple Conductance Channel
Oxidation Of N(G)-Hydroxy-L-Arginine To Nitric Oxide Mediated By Respiratory Burst: An Alternative Pathway To No Synthesis
Perspectives On The Mitochondrial Multiple Conductance Channel
Arginase Induction By Suppressors Of Nitric Oxide Synthesis (Il-4, Il-10 And Pge2) In Murine Bone-Marrow-Derived Macrophages
Perspectives On The Mitochondrial Multiple Conductance Channel
Arginase Induction By Suppressors Of Nitric Oxide Synthesis (Il-4, Il-10 And Pge2) In Murine Bone-Marrow-Derived Macrophages
Reciprocal Regulation Of The Nitric Oxide Synthase/arginase Balance In Mouse Bone Marrow‐derived Macrophages By Th 1 And Th 2 Cytokines
Arginase Induction By Suppressors Of Nitric Oxide Synthesis (Il-4, Il-10 And Pge2) In Murine Bone-Marrow-Derived Macrophages
Reciprocal Regulation Of The Nitric Oxide Synthase/arginase Balance In Mouse Bone Marrow‐derived Macrophages By Th 1 And Th 2 Cytokines
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
Reciprocal Regulation Of The Nitric Oxide Synthase/arginase Balance In Mouse Bone Marrow‐derived Macrophages By Th 1 And Th 2 Cytokines
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
The Immune System And Autoaggression
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
The Immune System And Autoaggression
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
The Immune System And Autoaggression
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
Determination Of Arginase Activity In Macrophages: A Micromethod
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
Determination Of Arginase Activity In Macrophages: A Micromethod
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Determination Of Arginase Activity In Macrophages: A Micromethod
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
The Effect Of Antimycin A On Mouse Liver Inner Mitochondrial Membrane Channel Activity
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
The Effect Of Antimycin A On Mouse Liver Inner Mitochondrial Membrane Channel Activity
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
The Effect Of Antimycin A On Mouse Liver Inner Mitochondrial Membrane Channel Activity
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Arginine Catabolism In The Phototrophic Bacterium Rhodobacter Capsulatus E1f1: Purification And Properties Of Arginase
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Arginine Catabolism In The Phototrophic Bacterium Rhodobacter Capsulatus E1f1: Purification And Properties Of Arginase
Trypsin Digestion Of Arginase: Evidence For A Stable Conformation Manganese Directed
Arginine Catabolism In The Phototrophic Bacterium Rhodobacter Capsulatus E1f1: Purification And Properties Of Arginase
Trypsin Digestion Of Arginase: Evidence For A Stable Conformation Manganese Directed
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Trypsin Digestion Of Arginase: Evidence For A Stable Conformation Manganese Directed
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
Properties Of Arginase Immobilized In A Fibrin Clot
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
Properties Of Arginase Immobilized In A Fibrin Clot
The Regulation Of Glucose 6-Phosphate Dehydrogenase From Dicentrarchus Labrax (Bass) Liver
Properties Of Arginase Immobilized In A Fibrin Clot
The Regulation Of Glucose 6-Phosphate Dehydrogenase From Dicentrarchus Labrax (Bass) Liver
Mitochondrial Channel Activity Studied By Patch-Clamping Mitoplasts
The Regulation Of Glucose 6-Phosphate Dehydrogenase From Dicentrarchus Labrax (Bass) Liver
Mitochondrial Channel Activity Studied By Patch-Clamping Mitoplasts
Glucose-6-Phosphate Dehydrogenase From Dicentrarchus Labrax Liver: Kinetic Mechanism And Kinetics Of Nadph Inhibition
Mitochondrial Channel Activity Studied By Patch-Clamping Mitoplasts
Glucose-6-Phosphate Dehydrogenase From Dicentrarchus Labrax Liver: Kinetic Mechanism And Kinetics Of Nadph Inhibition
Circadian Rhythms In Enzymatic Activity Of Rat Liver Arginase And Glucose 6-Phosphate Dehydrogenase.
Glucose-6-Phosphate Dehydrogenase From Dicentrarchus Labrax Liver: Kinetic Mechanism And Kinetics Of Nadph Inhibition
Circadian Rhythms In Enzymatic Activity Of Rat Liver Arginase And Glucose 6-Phosphate Dehydrogenase.
Nucleotide Effects On Kinetic Properties Of Mitochondril Atpase
Circadian Rhythms In Enzymatic Activity Of Rat Liver Arginase And Glucose 6-Phosphate Dehydrogenase.
Nucleotide Effects On Kinetic Properties Of Mitochondril Atpase
Effect Of Nucleotides And Inhibitory Anions On Mitochondrial Atpase. Its Ph Dependence.
Nucleotide Effects On Kinetic Properties Of Mitochondril Atpase
Effect Of Nucleotides And Inhibitory Anions On Mitochondrial Atpase. Its Ph Dependence.
Nucleotide Effects On Kinetic Properties Of Mitochondrial Atpase.
Effect Of Nucleotides And Inhibitory Anions On Mitochondrial Atpase. Its Ph Dependence.
Nucleotide Effects On Kinetic Properties Of Mitochondrial Atpase.
Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)
Nucleotide Effects On Kinetic Properties Of Mitochondrial Atpase.
Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)
Protonmotive Force And Photophosphorylation In Single Swollen Thylakoid Vesicles
Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)
Protonmotive Force And Photophosphorylation In Single Swollen Thylakoid Vesicles
Assay Of Atp Synthesis Using Single Giant Mitochondria
Protonmotive Force And Photophosphorylation In Single Swollen Thylakoid Vesicles
Assay Of Atp Synthesis Using Single Giant Mitochondria
Protonmotive Force In Giant Mitochondria
Assay Of Atp Synthesis Using Single Giant Mitochondria
Protonmotive Force In Giant Mitochondria
Energy Transduction, Ion Transport And Protonmotive Force: Are They Linked?
Protonmotive Force In Giant Mitochondria
Energy Transduction, Ion Transport And Protonmotive Force: Are They Linked?
Effects Of Heating On The Ion-Gating Function And Structural Domains Of The Acetylcholine Receptor
Energy Transduction, Ion Transport And Protonmotive Force: Are They Linked?
Effects Of Heating On The Ion-Gating Function And Structural Domains Of The Acetylcholine Receptor
Controlled Thermal Disruption Of The Ion Gating Function And The Structure Of The Acetylcholine Receptor
Effects Of Heating On The Ion-Gating Function And Structural Domains Of The Acetylcholine Receptor
Controlled Thermal Disruption Of The Ion Gating Function And The Structure Of The Acetylcholine Receptor
Three Catalytic Sites In Mitochondrial Atpase
Controlled Thermal Disruption Of The Ion Gating Function And The Structure Of The Acetylcholine Receptor
Three Catalytic Sites In Mitochondrial Atpase
Reductive Methylation As A Probe Of The Heat-Labile Α-Bungarotoxin-Acetylcholine Receptor Membrane Complex: Evidence For Surface Interactions
Three Catalytic Sites In Mitochondrial Atpase
Reductive Methylation As A Probe Of The Heat-Labile Α-Bungarotoxin-Acetylcholine Receptor Membrane Complex: Evidence For Surface Interactions
Nutritional Regulation Of Glycolysis In Rainbow Trout (Salmo Gairdneri R.)
Reductive Methylation As A Probe Of The Heat-Labile Α-Bungarotoxin-Acetylcholine Receptor Membrane Complex: Evidence For Surface Interactions
Nutritional Regulation Of Glycolysis In Rainbow Trout (Salmo Gairdneri R.)
Physico-Chemical Properties Of Guinea Pig Liver Arginosuccinate Synthetase
Nutritional Regulation Of Glycolysis In Rainbow Trout (Salmo Gairdneri R.)
Physico-Chemical Properties Of Guinea Pig Liver Arginosuccinate Synthetase
Arginine As Competitive Inhibitor Of Arginosuccinate Synthetase From Guinea Pig Liver
Physico-Chemical Properties Of Guinea Pig Liver Arginosuccinate Synthetase
Arginine As Competitive Inhibitor Of Arginosuccinate Synthetase From Guinea Pig Liver
Physico-Chemical Properties Of Guinea Pig Liver Arginase
Arginine As Competitive Inhibitor Of Arginosuccinate Synthetase From Guinea Pig Liver
Physico-Chemical Properties Of Guinea Pig Liver Arginase
Effect Of Ph On The Sensitivity Of Mitochondrial Atpase To Free Atp, Adp And Anions.
Physico-Chemical Properties Of Guinea Pig Liver Arginase
Effect Of Ph On The Sensitivity Of Mitochondrial Atpase To Free Atp, Adp And Anions.
The Influence Of Fasting And Cortisol Upon Arginosuccinate Synthetase, Arginosuccinase And Arginase Levels In Guinea Pig Liver
Effect Of Ph On The Sensitivity Of Mitochondrial Atpase To Free Atp, Adp And Anions.
The Influence Of Fasting And Cortisol Upon Arginosuccinate Synthetase, Arginosuccinase And Arginase Levels In Guinea Pig Liver
Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice
WEB ACCESIBLE