PUBLICACIÓN

Cytoplasmic STAT3 Represses Autophagy by Inhibiting PKR Activity

Logo Open Access

ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Shen S. et al.

2012 MOLECULAR CELL

Cell Biology (Q1), Molecular Biology (Q1)

JCR: 15.28

SJR: 13.492

CITAS

240

DOI

10.1016/j.molcel.2012.09.013

EID

2-s2.0-84870901484

ISSN

1097-2765

EISSN

1097-4164

BIBTEX

@article{Niso-Santano2012,title = {Cytoplasmic STAT3 Represses Autophagy by Inhibiting PKR Activity},journal = {Molecular Cell},year = {2012},volume = {48},number = {5},pages = {667-680},author = {Shen, S. and Niso-Santano, M. and Adjemian, S. and Takehara, T. and Malik, S.A. and Minoux, H. and Souquere, S. and Mari{\~n}o, G. and Lachkar, S. and Senovilla, L. and Galluzzi, L. and Kepp, O. and Pierron, G. and Maiuri, M.C. and Hikita, H. and Kroemer, R. and Kroemer, G.}}

AUTORES DE LA UEX

Mireia Niso Santano

OTRAS PUBLICACIONES DE INTERÉS

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Links Between Paraquat And Parkinson’s Disease
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Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease
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The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress
Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress
Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles
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Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles
In Vitro And In Vivo Models To Study The Biological And Pharmacological Properties Of Queen Bee Acid (Qba, 10-Hydroxy-2-Decenoic Acid): A Systematic Review
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Neuroprotective And Anti-Inflammatory Effects Of Linoleic Acid In Models Of Parkinson’s Disease: The Implication Of Lipid Droplets And Lipophagy
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Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1
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Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity
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Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
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Er-Mitochondria Signaling In Parkinson's Disease Review-Article
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Er-Mitochondria Signaling In Parkinson's Disease Review-Article
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Er-Mitochondria Signaling In Parkinson's Disease Review-Article
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Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells
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Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
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Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy
Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
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Metabolomic Analyses Reveal That Anti-Aging Metabolites Are Depleted By Palmitate But Increased By Oleate In Vivo
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Spermidine Induces Autophagy By Inhibiting The Acetyltransferase Ep300
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Links Between Paraquat And Parkinson's Disease
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Links Between Paraquat And Parkinson's Disease
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Self-Consumption: The Interplay Of Autophagy And Apoptosis
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Regulation Of Autophagy By Stress-Responsive Transcription Factors
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Regulation Of Autophagy By Stress-Responsive Transcription Factors
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Regulation Of Autophagy By Stress-Responsive Transcription Factors
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Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Direct Interaction Between Stat3 And Eif2ak2 Controls Fatty Acid-Induced Autophagy
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Autophagy And Parkinson's Disease: A Love-Hate Relationship
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Autophagy And Parkinson's Disease: A Love-Hate Relationship
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.
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Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.
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Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
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The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway
Autophagic Removal Of Micronuclei
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway
Autophagic Removal Of Micronuclei
Autophagy Is Required For The Activation Of Nfκb
Autophagic Removal Of Micronuclei
Autophagy Is Required For The Activation Of Nfκb
Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))
Autophagy Is Required For The Activation Of Nfκb
Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))
Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity
Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))
Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins
An Immunosurveillance Mechanism Controls Cancer Cell Ploidy
Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins
An Immunosurveillance Mechanism Controls Cancer Cell Ploidy
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
An Immunosurveillance Mechanism Controls Cancer Cell Ploidy
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Inhibition Of Autophagy By Tab2 And Tab3
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Inhibition Of Autophagy By Tab2 And Tab3
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Inhibition Of Autophagy By Tab2 And Tab3
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Immunological Aspects Of Central Neurodegeneration
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