PUBLICACIÓN

Pharmacological inhibition of fatty-acid oxidation synergistically enhances the effect of l-asparaginase in childhood ALL cells

ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Hermanova I. et al.

2016 LEUKEMIA

Anesthesiology and Pain Medicine (Q1), Cancer Research (Q1), Hematology (Q1), Oncology (Q1)

JCR: 11.702

SJR: 5.041

CITAS

37

DOI

10.1038/leu.2015.213

EID

2-s2.0-84953368007

ISSN

0887-6924

EISSN

1476-5551

BIBTEX

@article{Niso-Santano2016,title = {Pharmacological inhibition of fatty-acid oxidation synergistically enhances the effect of l-asparaginase in childhood ALL cells},journal = {Leukemia},year = {2016},volume = {30},number = {1},pages = {209-218},author = {Hermanova, I. and Arruabarrena-Aristorena, A. and Valis, K. and Nuskova, H. and Alberich-Jorda, M. and Fiser, K. and Fernandez-Ruiz, S. and Kavan, D. and Pecinova, A. and Niso-Santano, M. and Zaliova, M. and Novak, P. and Houstek, J. and Mracek, T. and Kroemer, G. and Carracedo, A. and Trka, J. and Starkova, J.}}

AUTORES DE LA UEX

Mireia Niso Santano

OTRAS PUBLICACIONES DE INTERÉS

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Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles
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Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1
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Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity
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Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity
Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
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Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
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Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells
Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
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Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy
Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
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Pharmacological Inhibition Of Fatty-Acid Oxidation Synergistically Enhances The Effect Of L-Asparaginase In Childhood All Cells
G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway
Pharmacological Inhibition Of Fatty-Acid Oxidation Synergistically Enhances The Effect Of L-Asparaginase In Childhood All Cells
Unsaturated Fatty Acids Induce Non-Canonical Autophagy
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Unsaturated Fatty Acids Induce Non-Canonical Autophagy
Metabolomic Analyses Reveal That Anti-Aging Metabolites Are Depleted By Palmitate But Increased By Oleate In Vivo
Unsaturated Fatty Acids Induce Non-Canonical Autophagy
Metabolomic Analyses Reveal That Anti-Aging Metabolites Are Depleted By Palmitate But Increased By Oleate In Vivo
Novel Inducers Of Becn1-Independent Autophagy: Cis-Unsaturated Fatty Acids
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Novel Inducers Of Becn1-Independent Autophagy: Cis-Unsaturated Fatty Acids
Spermidine Induces Autophagy By Inhibiting The Acetyltransferase Ep300
Novel Inducers Of Becn1-Independent Autophagy: Cis-Unsaturated Fatty Acids
Spermidine Induces Autophagy By Inhibiting The Acetyltransferase Ep300
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
Spermidine Induces Autophagy By Inhibiting The Acetyltransferase Ep300
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
Links Between Paraquat And Parkinson's Disease
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
Links Between Paraquat And Parkinson's Disease
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Links Between Paraquat And Parkinson's Disease
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Regulation Of Autophagy By Cytosolic Acetyl-Coenzyme A
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Regulation Of Autophagy By Cytosolic Acetyl-Coenzyme A
Self-Consumption: The Interplay Of Autophagy And Apoptosis
Regulation Of Autophagy By Cytosolic Acetyl-Coenzyme A
Self-Consumption: The Interplay Of Autophagy And Apoptosis
Implication Of Autophagy In Parkinson's Disease
Self-Consumption: The Interplay Of Autophagy And Apoptosis
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Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Regulation Of Autophagy By Stress-Responsive Transcription Factors
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Regulation Of Autophagy By Stress-Responsive Transcription Factors
Direct Interaction Between Stat3 And Eif2ak2 Controls Fatty Acid-Induced Autophagy
Regulation Of Autophagy By Stress-Responsive Transcription Factors
Direct Interaction Between Stat3 And Eif2ak2 Controls Fatty Acid-Induced Autophagy
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Direct Interaction Between Stat3 And Eif2ak2 Controls Fatty Acid-Induced Autophagy
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Autophagy And Parkinson's Disease: A Love-Hate Relationship
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Autophagy And Parkinson's Disease: A Love-Hate Relationship
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway
Autophagic Removal Of Micronuclei
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway
Autophagic Removal Of Micronuclei
Autophagy Is Required For The Activation Of Nfκb
Autophagic Removal Of Micronuclei
Autophagy Is Required For The Activation Of Nfκb
Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))
Autophagy Is Required For The Activation Of Nfκb
Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))
Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity
Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))
Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins
An Immunosurveillance Mechanism Controls Cancer Cell Ploidy
Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins
An Immunosurveillance Mechanism Controls Cancer Cell Ploidy
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
An Immunosurveillance Mechanism Controls Cancer Cell Ploidy
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Inhibition Of Autophagy By Tab2 And Tab3
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Inhibition Of Autophagy By Tab2 And Tab3
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Inhibition Of Autophagy By Tab2 And Tab3
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
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Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
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Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
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Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
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Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
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Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
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Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
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Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
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