PUBLICACIÓN

In vitro and in vivo models to study the biological and pharmacological properties of queen bee acid (QBA, 10-hydroxy-2-decenoic acid): A systematic review

Logo Open Access

ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Paredes-Barquero M., Niso-Santano M., Fuentes J.M., Martinez-Chacon G.

2022 Journal of Functional Foods

Food Science (Q1), Medicine (miscellaneous) (Q1), Nutrition and Dietetics (Q2)

JCR: 5.6

SJR: 0.894

CITAS

9

DOI

10.1016/j.jff.2022.105143

EID

2-s2.0-85131923599

ISSN

1756-4646

BIBTEX

@article{Paredes_Barquero_2022,doi = {10.1016/j.jff.2022.105143},url = {https://doi.org/10.1016%2Fj.jff.2022.105143},year = 2022,month = {jul},publisher = {Elsevier {BV}},volume = {94},pages = {105143},author = {Marta Paredes-Barquero and Mireia Niso-Santano and Jos{\'{e}} M. Fuentes and Guadalupe Mart{\'{\i}}nez-Chac{\'{o}}n},title = {In vitro and in vivo models to study the biological and pharmacological properties of queen bee acid ({QBA}, 10-hydroxy-2-decenoic acid): A systematic review},journal = {Journal of Functional Foods}}

AUTORES DE LA UEX

Mireia Niso Santano
Marta Paredes Barquero
José Manuel Fuentes Rodríguez

OTRAS PUBLICACIONES DE INTERÉS

Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)
Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice
Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques
Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice
Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques
The Role Of Cardiolipin In Mitochondrial Function And Neurodegenerative Diseases
Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques
The Role Of Cardiolipin In Mitochondrial Function And Neurodegenerative Diseases
Immunological Aspects Of Central Neurodegeneration
The Role Of Cardiolipin In Mitochondrial Function And Neurodegenerative Diseases
Immunological Aspects Of Central Neurodegeneration
Role Of Tfeb In Huntington’s Disease
Immunological Aspects Of Central Neurodegeneration
Role Of Tfeb In Huntington’s Disease
Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects
Role Of Tfeb In Huntington’s Disease
Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects
Lysosomal Dysfunction: Connecting The Dots In The Landscape Of Human Diseases
Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects
Lysosomal Dysfunction: Connecting The Dots In The Landscape Of Human Diseases
Corrigendum: Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells (Journal Of Cellular Physiology, 10.1002/jcp.26855)
Lysosomal Dysfunction: Connecting The Dots In The Landscape Of Human Diseases
Corrigendum: Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells (Journal Of Cellular Physiology, 10.1002/jcp.26855)
Vitamin D Receptor Polymorphisms In A Spanish Cohort Of Parkinson’s Disease Patients
Corrigendum: Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells (Journal Of Cellular Physiology, 10.1002/jcp.26855)
Vitamin D Receptor Polymorphisms In A Spanish Cohort Of Parkinson’s Disease Patients
Links Between Paraquat And Parkinson’s Disease
Vitamin D Receptor Polymorphisms In A Spanish Cohort Of Parkinson’s Disease Patients
Links Between Paraquat And Parkinson’s Disease
Mitochondrial Dysfunction In Repeat Expansion Diseases
Links Between Paraquat And Parkinson’s Disease
Mitochondrial Dysfunction In Repeat Expansion Diseases
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction
Mitochondrial Dysfunction In Repeat Expansion Diseases
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction
Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction
Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease
Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease
Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease
Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress
Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress
Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress
Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles
In Vitro And In Vivo Models To Study The Biological And Pharmacological Properties Of Queen Bee Acid (Qba, 10-Hydroxy-2-Decenoic Acid): A Systematic Review
Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles
In Vitro And In Vivo Models To Study The Biological And Pharmacological Properties Of Queen Bee Acid (Qba, 10-Hydroxy-2-Decenoic Acid): A Systematic Review
Role Of Inflammatory Cytokines In The Conversion Of Mild Cognitive Impairment To Dementia: A Prospective Study.
In Vitro And In Vivo Models To Study The Biological And Pharmacological Properties Of Queen Bee Acid (Qba, 10-Hydroxy-2-Decenoic Acid): A Systematic Review
Role Of Inflammatory Cytokines In The Conversion Of Mild Cognitive Impairment To Dementia: A Prospective Study.
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
Role Of Inflammatory Cytokines In The Conversion Of Mild Cognitive Impairment To Dementia: A Prospective Study.
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
Neuroprotective And Anti-Inflammatory Effects Of Linoleic Acid In Models Of Parkinson’s Disease: The Implication Of Lipid Droplets And Lipophagy
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
Neuroprotective And Anti-Inflammatory Effects Of Linoleic Acid In Models Of Parkinson’s Disease: The Implication Of Lipid Droplets And Lipophagy
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Neuroprotective And Anti-Inflammatory Effects Of Linoleic Acid In Models Of Parkinson’s Disease: The Implication Of Lipid Droplets And Lipophagy
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1
Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1
Links Between Paraquat And Parkinson’s Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1
Links Between Paraquat And Parkinson’s Disease
Procedimientos, Experimentación E Investigación En Enfermedades Neurodegenerativas: Protocolos Para Un Laboratorio De Neurociencias
Links Between Paraquat And Parkinson’s Disease
Procedimientos, Experimentación E Investigación En Enfermedades Neurodegenerativas: Protocolos Para Un Laboratorio De Neurociencias
Cultivos Celulares
Procedimientos, Experimentación E Investigación En Enfermedades Neurodegenerativas: Protocolos Para Un Laboratorio De Neurociencias
Cultivos Celulares
Análisis De Extracto Proteico Por Western Blotting
Cultivos Celulares
Análisis De Extracto Proteico Por Western Blotting
Extracción De Proteínas
Análisis De Extracto Proteico Por Western Blotting
Extracción De Proteínas
Técnica De Sobreexpresión Génica
Extracción De Proteínas
Técnica De Sobreexpresión Génica
Técnica De Silenciamiento Génico
Técnica De Sobreexpresión Génica
Técnica De Silenciamiento Génico
The Dual Role Of Necrostatin-1 In Parkinson's Disease Models
Técnica De Silenciamiento Génico
The Dual Role Of Necrostatin-1 In Parkinson's Disease Models
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction.
The Dual Role Of Necrostatin-1 In Parkinson's Disease Models
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction.
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress.
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction.
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress.
Autophagy Modulation In Animal Models Of Corneal Diseases: A Systematic Review
The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress.
Autophagy Modulation In Animal Models Of Corneal Diseases: A Systematic Review
Animal Models Used To Study Direct Peripheral Nerve Repair: A Systematic Review
Autophagy Modulation In Animal Models Of Corneal Diseases: A Systematic Review
Animal Models Used To Study Direct Peripheral Nerve Repair: A Systematic Review
Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease
Animal Models Used To Study Direct Peripheral Nerve Repair: A Systematic Review
Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease
Toxicity Of Necrostatin-1 In Parkinson’s Disease Models
Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease
Toxicity Of Necrostatin-1 In Parkinson’s Disease Models
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells
Toxicity Of Necrostatin-1 In Parkinson’s Disease Models
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells
Natural Products In The Promotion Of Healthspan And Longevity.
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells
Natural Products In The Promotion Of Healthspan And Longevity.
Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack
Natural Products In The Promotion Of Healthspan And Longevity.
Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack
The Paradigm Of Protein Acetylation In Parkinson's Disease
Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack
The Paradigm Of Protein Acetylation In Parkinson's Disease
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients
The Paradigm Of Protein Acetylation In Parkinson's Disease
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients
A Pre-Clinical Rat Model For The Study Of Ischemia-Reperfusion Injury In Reconstructive Microsurgery
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients
A Pre-Clinical Rat Model For The Study Of Ischemia-Reperfusion Injury In Reconstructive Microsurgery
Erratum: Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity (Cell Metabolism (2019) 30(4) (754–767.e9), (S1550413119303833), (10.1016/j.cmet.2019.07.010))
A Pre-Clinical Rat Model For The Study Of Ischemia-Reperfusion Injury In Reconstructive Microsurgery
Erratum: Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity (Cell Metabolism (2019) 30(4) (754–767.e9), (S1550413119303833), (10.1016/j.cmet.2019.07.010))
Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity
Erratum: Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity (Cell Metabolism (2019) 30(4) (754–767.e9), (S1550413119303833), (10.1016/j.cmet.2019.07.010))
Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson's Disease Patients.
Acyl-Coa-Binding Protein Is A Lipogenic Factor That Triggers Food Intake And Obesity
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson's Disease Patients.
Cholesterol And Multilamellar Bodies: Lysosomal Dysfunction In Gba-Parkinson Disease
Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson's Disease Patients.
Cholesterol And Multilamellar Bodies: Lysosomal Dysfunction In Gba-Parkinson Disease
Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria
Cholesterol And Multilamellar Bodies: Lysosomal Dysfunction In Gba-Parkinson Disease
Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria
Vascular Risk Factors And Lesions Of Vascular Nature In Magnetic Resonance As Predictors Of Progression To Dementia In Patients With Mild Cognitive Impairment
Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria
Vascular Risk Factors And Lesions Of Vascular Nature In Magnetic Resonance As Predictors Of Progression To Dementia In Patients With Mild Cognitive Impairment
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells: In Vitro And In Vivo Studies
Vascular Risk Factors And Lesions Of Vascular Nature In Magnetic Resonance As Predictors Of Progression To Dementia In Patients With Mild Cognitive Impairment
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells: In Vitro And In Vivo Studies
Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells: In Vitro And In Vivo Studies
Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells
Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells
Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy
Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy
Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy
Trend Of Lipid Profile In General Population From Caceres Health Area
Inhibitor Of Growth Protein 4 Interacts With Beclin 1 And Represses Autophagy
Trend Of Lipid Profile In General Population From Caceres Health Area
The Basics Of Autophagy
Trend Of Lipid Profile In General Population From Caceres Health Area
The Basics Of Autophagy
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
The Basics Of Autophagy
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis
Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines
Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis
Mitochondria: Key Organelle In Parkinson's Disease
Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis
Mitochondria: Key Organelle In Parkinson's Disease
G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway
Mitochondria: Key Organelle In Parkinson's Disease
G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway
Pharmacological Inhibition Of Fatty-Acid Oxidation Synergistically Enhances The Effect Of L-Asparaginase In Childhood All Cells
G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway
Pharmacological Inhibition Of Fatty-Acid Oxidation Synergistically Enhances The Effect Of L-Asparaginase In Childhood All Cells
Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment
Pharmacological Inhibition Of Fatty-Acid Oxidation Synergistically Enhances The Effect Of L-Asparaginase In Childhood All Cells
Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Control Of Autophagy In Parkinson’s Disease
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Control Of Autophagy In Parkinson’s Disease
Unsaturated Fatty Acids Induce Non-Canonical Autophagy
Control Of Autophagy In Parkinson’s Disease
Unsaturated Fatty Acids Induce Non-Canonical Autophagy
Metabolomic Analyses Reveal That Anti-Aging Metabolites Are Depleted By Palmitate But Increased By Oleate In Vivo
Unsaturated Fatty Acids Induce Non-Canonical Autophagy
Metabolomic Analyses Reveal That Anti-Aging Metabolites Are Depleted By Palmitate But Increased By Oleate In Vivo
Novel Inducers Of Becn1-Independent Autophagy: Cis-Unsaturated Fatty Acids
Metabolomic Analyses Reveal That Anti-Aging Metabolites Are Depleted By Palmitate But Increased By Oleate In Vivo
Novel Inducers Of Becn1-Independent Autophagy: Cis-Unsaturated Fatty Acids
Spermidine Induces Autophagy By Inhibiting The Acetyltransferase Ep300
Novel Inducers Of Becn1-Independent Autophagy: Cis-Unsaturated Fatty Acids
Spermidine Induces Autophagy By Inhibiting The Acetyltransferase Ep300
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Spermidine Induces Autophagy By Inhibiting The Acetyltransferase Ep300
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Toxicity And Autophagy In Neurodegenerative Disorders
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Toxicity And Autophagy In Neurodegenerative Disorders
Origen Molecular De La Compleja Enfermedad De Parkinson
Toxicity And Autophagy In Neurodegenerative Disorders
Origen Molecular De La Compleja Enfermedad De Parkinson
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
Origen Molecular De La Compleja Enfermedad De Parkinson
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Links Between Paraquat And Parkinson's Disease
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Links Between Paraquat And Parkinson's Disease
Cancer Cell–autonomous Contribution Of Type I Interferon Signaling To The Efficacy Of Chemotherapy
Links Between Paraquat And Parkinson's Disease
Cancer Cell–autonomous Contribution Of Type I Interferon Signaling To The Efficacy Of Chemotherapy
Coffee Induces Autophagy In Vivo
Cancer Cell–autonomous Contribution Of Type I Interferon Signaling To The Efficacy Of Chemotherapy
Coffee Induces Autophagy In Vivo
Regulation Of Autophagy By Cytosolic Acetyl-Coenzyme A
Coffee Induces Autophagy In Vivo
Regulation Of Autophagy By Cytosolic Acetyl-Coenzyme A
Self-Consumption: The Interplay Of Autophagy And Apoptosis
Regulation Of Autophagy By Cytosolic Acetyl-Coenzyme A
Self-Consumption: The Interplay Of Autophagy And Apoptosis
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Self-Consumption: The Interplay Of Autophagy And Apoptosis
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Implication Of Autophagy In Parkinson's Disease
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Implication Of Autophagy In Parkinson's Disease
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
Implication Of Autophagy In Parkinson's Disease
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Regulation Of Autophagy By Stress-Responsive Transcription Factors
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Regulation Of Autophagy By Stress-Responsive Transcription Factors
Direct Interaction Between Stat3 And Eif2ak2 Controls Fatty Acid-Induced Autophagy
Regulation Of Autophagy By Stress-Responsive Transcription Factors
Direct Interaction Between Stat3 And Eif2ak2 Controls Fatty Acid-Induced Autophagy
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Direct Interaction Between Stat3 And Eif2ak2 Controls Fatty Acid-Induced Autophagy
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Paraquat, Between Apoptosis And Autophagy
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Paraquat, Between Apoptosis And Autophagy
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Paraquat, Between Apoptosis And Autophagy
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway.
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway
Autophagic Removal Of Micronuclei
Direct Molecular Interactions Between Beclin 1 And The Canonical Nfκb Activation Pathway
Autophagic Removal Of Micronuclei
Autophagy Is Required For The Activation Of Nfκb
Autophagic Removal Of Micronuclei
Autophagy Is Required For The Activation Of Nfκb
Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))
Autophagy Is Required For The Activation Of Nfκb
Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))
Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity
Autophagy Is Required For The Activation Of Nfkb (Cell Cycle 11, 1 (194-199))
Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Cytoplasmic Stat3 Represses Autophagy By Inhibiting Pkr Activity
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins
Prognostic Impact Of Vitamin B6 Metabolism In Lung Cancer
Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins
An Immunosurveillance Mechanism Controls Cancer Cell Ploidy
Pro-Autophagic Polyphenols Reduce The Acetylation Of Cytoplasmic Proteins
An Immunosurveillance Mechanism Controls Cancer Cell Ploidy
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
An Immunosurveillance Mechanism Controls Cancer Cell Ploidy
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Inhibition Of Autophagy By Tab2 And Tab3
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Inhibition Of Autophagy By Tab2 And Tab3
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Inhibition Of Autophagy By Tab2 And Tab3
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
The Immune System And Autoaggression
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
The Immune System And Autoaggression
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
The Immune System And Autoaggression
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)
Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice
WEB ACCESIBLE