PUBLICACIÓN

ACCEDER A LA PUBLICACIÓN: Scopus Orcid Dialnet Zenodo OpenAlex Dehesa Repositorio Institucional Catalogo Bibliotecas UEx

Cellular and Molecular Neuroscience (Q1), Developmental Neuroscience (Q1), Epidemiology (Q1), Geriatrics and Gerontology (Q1), Health Policy (Q1), Neurology (clinical) (Q1), Psychiatry and Mental Health (Q1)

JCR: 16.655

SJR: 5.059

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AUTORES DE LA UEX

OTRAS PUBLICACIONES DE INTERÉS

Posttranslational Modifications As Versatile Regulators Of Parkin Function
Stim1-Containing Contact Sites Promote Direct Calcium Flux From The Endoplasmic Reticulum To Mitochondria
Stimulating Vapb-Ptpip51 Er-Mitochondria Tethering Corrects Ftd/als Mutant Tdp43 Linked Ca2+ And Synaptic Defects
Stim1-Containing Contact Sites Promote Direct Calcium Flux From The Endoplasmic Reticulum To Mitochondria
Stimulating Vapb-Ptpip51 Er-Mitochondria Tethering Corrects Ftd/als Mutant Tdp43 Linked Ca2+ And Synaptic Defects
Truncated Tau Interferes With The Autophagy And Endolysosomal Pathway And Results In Lipid Accumulation
Stimulating Vapb-Ptpip51 Er-Mitochondria Tethering Corrects Ftd/als Mutant Tdp43 Linked Ca2+ And Synaptic Defects
Truncated Tau Interferes With The Autophagy And Endolysosomal Pathway And Results In Lipid Accumulation
Insight Into Endoplasmic Reticulum-Mitochondria Contacts In Human Amyotrophic Lateral Sclerosis
Truncated Tau Interferes With The Autophagy And Endolysosomal Pathway And Results In Lipid Accumulation
Insight Into Endoplasmic Reticulum-Mitochondria Contacts In Human Amyotrophic Lateral Sclerosis
Mitochondrial Dysfunction In Repeat Expansion Diseases
Insight Into Endoplasmic Reticulum-Mitochondria Contacts In Human Amyotrophic Lateral Sclerosis
Mitochondrial Dysfunction In Repeat Expansion Diseases
Editorial
Mitochondrial Dysfunction In Repeat Expansion Diseases
Editorial
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
Editorial
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
The Presence Of Disease-Associated Tau Leads To Adverse Effects On The Autophagy And Endolysosomal Pathway In A Cell Model Of Tauopathy
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
The Presence Of Disease-Associated Tau Leads To Adverse Effects On The Autophagy And Endolysosomal Pathway In A Cell Model Of Tauopathy
Disruption Of The Vapb-Ptpip51 Er-Mitochondria Tethering Proteins In Post-Mortem Human Amyotrophic Lateral Sclerosis
The Presence Of Disease-Associated Tau Leads To Adverse Effects On The Autophagy And Endolysosomal Pathway In A Cell Model Of Tauopathy
Disruption Of The Vapb-Ptpip51 Er-Mitochondria Tethering Proteins In Post-Mortem Human Amyotrophic Lateral Sclerosis
The Interplay Between Autophagy And Rna Homeostasis: Implications For Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
Disruption Of The Vapb-Ptpip51 Er-Mitochondria Tethering Proteins In Post-Mortem Human Amyotrophic Lateral Sclerosis
The Interplay Between Autophagy And Rna Homeostasis: Implications For Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
Disruption Of Er-Mitochondria Tethering And Signalling In C9orf72-Associated Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
The Interplay Between Autophagy And Rna Homeostasis: Implications For Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
Disruption Of Er-Mitochondria Tethering And Signalling In C9orf72-Associated Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Disruption Of Er-Mitochondria Tethering And Signalling In C9orf72-Associated Amyotrophic Lateral Sclerosis And Frontotemporal Dementia
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Autophagy And Lysosomal Defects In Cells Expressing Disease-Associated Tau
Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Autophagy And Lysosomal Defects In Cells Expressing Disease-Associated Tau
Disruption Of Endoplasmic Reticulum-Mitochondria Tethering Proteins In Post-Mortem Alzheimer's Disease Brain
Autophagy And Lysosomal Defects In Cells Expressing Disease-Associated Tau
Disruption Of Endoplasmic Reticulum-Mitochondria Tethering Proteins In Post-Mortem Alzheimer's Disease Brain
Lmtk2 Binds To Kinesin Light Chains To Mediate Anterograde Axonal Transport Of Cdk5/p35 And Lmtk2 Levels Are Reduced In Alzheimer's Disease Brains
Disruption Of Endoplasmic Reticulum-Mitochondria Tethering Proteins In Post-Mortem Alzheimer's Disease Brain
Lmtk2 Binds To Kinesin Light Chains To Mediate Anterograde Axonal Transport Of Cdk5/p35 And Lmtk2 Levels Are Reduced In Alzheimer's Disease Brains
The Vapb-Ptpip51 Endoplasmic Reticulum-Mitochondria Tethering Proteins Are Present In Neuronal Synapses And Regulate Synaptic Activity
Lmtk2 Binds To Kinesin Light Chains To Mediate Anterograde Axonal Transport Of Cdk5/p35 And Lmtk2 Levels Are Reduced In Alzheimer's Disease Brains
The Vapb-Ptpip51 Endoplasmic Reticulum-Mitochondria Tethering Proteins Are Present In Neuronal Synapses And Regulate Synaptic Activity
Disruption Of Er-Mitochondria Signalling In Fronto-Temporal Dementia And Related Amyotrophic Lateral Sclerosis
The Vapb-Ptpip51 Endoplasmic Reticulum-Mitochondria Tethering Proteins Are Present In Neuronal Synapses And Regulate Synaptic Activity
Disruption Of Er-Mitochondria Signalling In Fronto-Temporal Dementia And Related Amyotrophic Lateral Sclerosis
Parkinson Disease-Associated Mutations In Lrrk2 Cause Centrosomal Defects Via Rab8a Phosphorylation
Disruption Of Er-Mitochondria Signalling In Fronto-Temporal Dementia And Related Amyotrophic Lateral Sclerosis
Parkinson Disease-Associated Mutations In Lrrk2 Cause Centrosomal Defects Via Rab8a Phosphorylation
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
Parkinson Disease-Associated Mutations In Lrrk2 Cause Centrosomal Defects Via Rab8a Phosphorylation
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
Er-Mitochondria Signaling Regulates Autophagy
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
Er-Mitochondria Signaling Regulates Autophagy
Α-Synuclein Binds To The Er–mitochondria Tethering Protein Vapb To Disrupt Ca2+ Homeostasis And Mitochondrial Atp Production
Er-Mitochondria Signaling Regulates Autophagy
Α-Synuclein Binds To The Er–mitochondria Tethering Protein Vapb To Disrupt Ca2+ Homeostasis And Mitochondrial Atp Production
The Er-Mitochondria Tethering Complex Vapb-Ptpip51 Regulates Autophagy
Α-Synuclein Binds To The Er–mitochondria Tethering Protein Vapb To Disrupt Ca2+ Homeostasis And Mitochondrial Atp Production
The Er-Mitochondria Tethering Complex Vapb-Ptpip51 Regulates Autophagy
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
The Er-Mitochondria Tethering Complex Vapb-Ptpip51 Regulates Autophagy
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
Als/ftd-Associated Fus Activates Gsk-3β To Disrupt The Vapb–ptpip51 Interaction And Er–mitochondria Associations
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
Als/ftd-Associated Fus Activates Gsk-3β To Disrupt The Vapb–ptpip51 Interaction And Er–mitochondria Associations
Iron Overload Causes Endolysosomal Deficits Modulated By Naadp-Regulated 2-Pore Channels And Rab7a
Als/ftd-Associated Fus Activates Gsk-3β To Disrupt The Vapb–ptpip51 Interaction And Er–mitochondria Associations
Iron Overload Causes Endolysosomal Deficits Modulated By Naadp-Regulated 2-Pore Channels And Rab7a
There's Something Wrong With My Mam; The Er-Mitochondria Axis And Neurodegenerative Diseases
Iron Overload Causes Endolysosomal Deficits Modulated By Naadp-Regulated 2-Pore Channels And Rab7a
There's Something Wrong With My Mam; The Er-Mitochondria Axis And Neurodegenerative Diseases
Alterations In Late Endocytic Trafficking Related To The Pathobiology Of Lrrk2-Linked Parkinson's Disease
There's Something Wrong With My Mam; The Er-Mitochondria Axis And Neurodegenerative Diseases
Alterations In Late Endocytic Trafficking Related To The Pathobiology Of Lrrk2-Linked Parkinson's Disease
Autophagic Pathology And Calcium Deregulation In Neurodegeneration
Alterations In Late Endocytic Trafficking Related To The Pathobiology Of Lrrk2-Linked Parkinson's Disease
Autophagic Pathology And Calcium Deregulation In Neurodegeneration
Lrrk2 Delays Degradative Receptor Trafficking By Impeding Late Endosomal Budding Through Decreasing Rab7 Activity
Autophagic Pathology And Calcium Deregulation In Neurodegeneration
Lrrk2 Delays Degradative Receptor Trafficking By Impeding Late Endosomal Budding Through Decreasing Rab7 Activity
Upstream Deregulation Of Calcium Signaling In Parkinson's Disease
Lrrk2 Delays Degradative Receptor Trafficking By Impeding Late Endosomal Budding Through Decreasing Rab7 Activity
Upstream Deregulation Of Calcium Signaling In Parkinson's Disease
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Upstream Deregulation Of Calcium Signaling In Parkinson's Disease
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
A Link Between Autophagy And The Pathophysiology Of Lrrk2 In Parkinson's Disease
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
A Link Between Autophagy And The Pathophysiology Of Lrrk2 In Parkinson's Disease
A Link Between Lrrk2, Autophagy And Naadp-Mediated Endolysosomal Calcium Signalling
A Link Between Autophagy And The Pathophysiology Of Lrrk2 In Parkinson's Disease
A Link Between Lrrk2, Autophagy And Naadp-Mediated Endolysosomal Calcium Signalling
Stone-Isolated Carbonatogenic Bacteria As Inoculants In Bioconsolidation Treatments For Historical Limestone
A Link Between Lrrk2, Autophagy And Naadp-Mediated Endolysosomal Calcium Signalling
Stone-Isolated Carbonatogenic Bacteria As Inoculants In Bioconsolidation Treatments For Historical Limestone
Leucine-Rich Repeat Kinase 2 Regulates Autophagy Through A Calcium-Dependent Pathway Involving Naadp
Stone-Isolated Carbonatogenic Bacteria As Inoculants In Bioconsolidation Treatments For Historical Limestone
Leucine-Rich Repeat Kinase 2 Regulates Autophagy Through A Calcium-Dependent Pathway Involving Naadp
Lrrk2 As A Modulator Of Lysosomal Calcium Homeostasis With Downstream Effects On Autophagy
Leucine-Rich Repeat Kinase 2 Regulates Autophagy Through A Calcium-Dependent Pathway Involving Naadp
Lrrk2 As A Modulator Of Lysosomal Calcium Homeostasis With Downstream Effects On Autophagy
Posttranslational Modifications As Versatile Regulators Of Parkin Function
Lrrk2 As A Modulator Of Lysosomal Calcium Homeostasis With Downstream Effects On Autophagy
Posttranslational Modifications As Versatile Regulators Of Parkin Function
Stim1-Containing Contact Sites Promote Direct Calcium Flux From The Endoplasmic Reticulum To Mitochondria
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