PUBLICACIÓN

The presence of disease-associated tau leads to adverse effects on the autophagy and endolysosomal pathway in a cell model of tauopathy

ACCEDER A LA PUBLICACIÓN: Scopus

Pollack S.J., Dakkak D., Guo T., Gomez-Suaga P., Noble W., Hanger D.P.

2022 Alzheimer's and Dementia

Cellular and Molecular Neuroscience (Q1), Developmental Neuroscience (Q1), Epidemiology (Q1), Geriatrics and Gerontology (Q1), Health Policy (Q1), Neurology (clinical) (Q1), Psychiatry and Mental Health (Q1)

JCR: 14

SJR: 3.288

CITAS

0

DOI

10.1002/alz.063630

EID

2-s2.0-85144383545

ISSN

1552-5260

EISSN

1552-5279

AUTORES DE LA UEX

Patricia María Gómez Suaga

OTRAS PUBLICACIONES DE INTERÉS

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Stimulating Vapb-Ptpip51 Er-Mitochondria Tethering Corrects Ftd/als Mutant Tdp43 Linked Ca2+ And Synaptic Defects
Truncated Tau Interferes With The Autophagy And Endolysosomal Pathway And Results In Lipid Accumulation
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Truncated Tau Interferes With The Autophagy And Endolysosomal Pathway And Results In Lipid Accumulation
Insight Into Endoplasmic Reticulum-Mitochondria Contacts In Human Amyotrophic Lateral Sclerosis
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The Presence Of Disease-Associated Tau Leads To Adverse Effects On The Autophagy And Endolysosomal Pathway In A Cell Model Of Tauopathy
Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
The Presence Of Disease-Associated Tau Leads To Adverse Effects On The Autophagy And Endolysosomal Pathway In A Cell Model Of Tauopathy
Disruption Of The Vapb-Ptpip51 Er-Mitochondria Tethering Proteins In Post-Mortem Human Amyotrophic Lateral Sclerosis
The Presence Of Disease-Associated Tau Leads To Adverse Effects On The Autophagy And Endolysosomal Pathway In A Cell Model Of Tauopathy
Disruption Of The Vapb-Ptpip51 Er-Mitochondria Tethering Proteins In Post-Mortem Human Amyotrophic Lateral Sclerosis
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Disruption Of The Vapb-Ptpip51 Er-Mitochondria Tethering Proteins In Post-Mortem Human Amyotrophic Lateral Sclerosis
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Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
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Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)
Autophagy And Lysosomal Defects In Cells Expressing Disease-Associated Tau
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The Vapb-Ptpip51 Endoplasmic Reticulum-Mitochondria Tethering Proteins Are Present In Neuronal Synapses And Regulate Synaptic Activity
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The Vapb-Ptpip51 Endoplasmic Reticulum-Mitochondria Tethering Proteins Are Present In Neuronal Synapses And Regulate Synaptic Activity
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Parkinson Disease-Associated Mutations In Lrrk2 Cause Centrosomal Defects Via Rab8a Phosphorylation
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