PUBLICACIÓN

Suppression of T-cell functions by human granulocyte arginase

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ACCEDER A LA PUBLICACIÓN: Scopus Orcid

Munder M. et al.

2006 Blood

CITAS

336

DOI

10.1182/blood-2006-11-010389

EID

2-s2.0-33748127113

ISSN

0006-4971

EISSN

0006-4971

BIBTEX

@article { fuentes2006,title = {Suppression of T-cell functions by human granulocyte arginase},journal = {Blood},year = {2006},volume = {108},number = {5},pages = {1627-1634},author = {Munder, M. and Schneider, H. and Luckner, C. and Giese, T. and Langhans, C.-D. and Fuentes, J.M. and Kropf, P. and Mueller, I. and Kolb, A. and Modolell, M. and Ho, A.D.}}

AUTORES DE LA UEX

José Manuel Fuentes Rodríguez

OTRAS PUBLICACIONES DE INTERÉS

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Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques
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Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques
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Vitamin D Receptor Polymorphisms In A Spanish Cohort Of Parkinson’s Disease Patients
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Mitochondrial Dysfunction In Repeat Expansion Diseases
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Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease
Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction
Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease
Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease
Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease
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Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles
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Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria
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Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.
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Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1
Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1
Links Between Paraquat And Parkinson’s Disease
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Links Between Paraquat And Parkinson’s Disease
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Cultivos Celulares
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Extracción De Proteínas
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Extracción De Proteínas
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Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
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Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
Acetylome In Human Fibroblasts From Parkinson’s Disease Patients
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
Er-Mitochondria Signaling In Parkinson's Disease Review-Article
[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].
N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease
N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease
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Trend Of Lipid Profile In General Population From Caceres Health Area
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Trend Of Lipid Profile In General Population From Caceres Health Area
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Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?
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Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)
Pink1 Deficiency Enhances Autophagy And Mitophagy Induction
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Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
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Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
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Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment
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Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment
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Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Control Of Autophagy In Parkinson’s Disease
Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations
Control Of Autophagy In Parkinson’s Disease
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Control Of Autophagy In Parkinson’s Disease
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Toxicity And Autophagy In Neurodegenerative Disorders
Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach
Toxicity And Autophagy In Neurodegenerative Disorders
Origen Molecular De La Compleja Enfermedad De Parkinson
Toxicity And Autophagy In Neurodegenerative Disorders
Origen Molecular De La Compleja Enfermedad De Parkinson
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
Origen Molecular De La Compleja Enfermedad De Parkinson
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Links Between Paraquat And Parkinson's Disease
G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy
Links Between Paraquat And Parkinson's Disease
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Links Between Paraquat And Parkinson's Disease
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Implication Of Autophagy In Parkinson's Disease
Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease
Implication Of Autophagy In Parkinson's Disease
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
Implication Of Autophagy In Parkinson's Disease
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Paraquat, Between Apoptosis And Autophagy
The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway
Paraquat, Between Apoptosis And Autophagy
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Paraquat, Between Apoptosis And Autophagy
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Autophagy And Parkinson's Disease: A Love-Hate Relationship
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Inhibition Of Autophagy By Tab2 And Tab3
Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress
Inhibition Of Autophagy By Tab2 And Tab3
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Inhibition Of Autophagy By Tab2 And Tab3
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Dj-1 As A Modulator Of Autophagy: An Hypothesis
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide In Paraquat-Mediated Toxicity: A Review
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Suppression Of T-Cell Functions By Human Granulocyte Arginase
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Diagnostic Performance Of Arginase Activity In Colorectal Cancer
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
The Immune System And Autoaggression
Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase
The Immune System And Autoaggression
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
The Immune System And Autoaggression
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)
Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.
Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)
Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice
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