Portal Investigación UEx

Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)

Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice

Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques

Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice

Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques

The Role Of Cardiolipin In Mitochondrial Function And Neurodegenerative Diseases

Heat Shock And Thermotolerance In Caenorhabditis Elegans: An Overview Of Laboratory Techniques

The Role Of Cardiolipin In Mitochondrial Function And Neurodegenerative Diseases

Immunological Aspects Of Central Neurodegeneration

The Role Of Cardiolipin In Mitochondrial Function And Neurodegenerative Diseases

Immunological Aspects Of Central Neurodegeneration

Role Of Tfeb In Huntington’s Disease

Immunological Aspects Of Central Neurodegeneration

Role Of Tfeb In Huntington’s Disease

Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects

Role Of Tfeb In Huntington’s Disease

Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects

Lysosomal Dysfunction: Connecting The Dots In The Landscape Of Human Diseases

Convergence Of Neuroinflammation, Microbiota, And Parkinson’s Disease: Therapeutic Insights And Prospects

Lysosomal Dysfunction: Connecting The Dots In The Landscape Of Human Diseases

Corrigendum: Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells (Journal Of Cellular Physiology, 10.1002/jcp.26855)

Lysosomal Dysfunction: Connecting The Dots In The Landscape Of Human Diseases

Corrigendum: Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells (Journal Of Cellular Physiology, 10.1002/jcp.26855)

Vitamin D Receptor Polymorphisms In A Spanish Cohort Of Parkinson’s Disease Patients

Corrigendum: Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells (Journal Of Cellular Physiology, 10.1002/jcp.26855)

Vitamin D Receptor Polymorphisms In A Spanish Cohort Of Parkinson’s Disease Patients

Mitochondrial Dysfunction In Repeat Expansion Diseases

Vitamin D Receptor Polymorphisms In A Spanish Cohort Of Parkinson’s Disease Patients

Mitochondrial Dysfunction In Repeat Expansion Diseases

Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction

Mitochondrial Dysfunction In Repeat Expansion Diseases

Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction

Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease

Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction

Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease

Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease

Comparative Genetic Analysis Of The Promoters Of The Atg16l1 And Atg5 Genes Associated With Sporadic Parkinson’s Disease

Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease

The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress

Changes In Liver Lipidomic Profile In G2019s-Lrrk2 Mouse Model Of Parkinson’s Disease

The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress

Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles

The Parkinsonian Lrrk2 R1441g Mutation Shows Macroautophagy-Mitophagy Dysregulation Concomitant With Endoplasmic Reticulum Stress

Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles

In Vitro And In Vivo Models To Study The Biological And Pharmacological Properties Of Queen Bee Acid (Qba, 10-Hydroxy-2-Decenoic Acid): A Systematic Review

Loss Of Keap1 Causes An Accumulation Of Nondegradative Organelles

In Vitro And In Vivo Models To Study The Biological And Pharmacological Properties Of Queen Bee Acid (Qba, 10-Hydroxy-2-Decenoic Acid): A Systematic Review

Role Of Inflammatory Cytokines In The Conversion Of Mild Cognitive Impairment To Dementia: A Prospective Study.

In Vitro And In Vivo Models To Study The Biological And Pharmacological Properties Of Queen Bee Acid (Qba, 10-Hydroxy-2-Decenoic Acid): A Systematic Review

Role Of Inflammatory Cytokines In The Conversion Of Mild Cognitive Impairment To Dementia: A Prospective Study.

Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria

Role Of Inflammatory Cytokines In The Conversion Of Mild Cognitive Impairment To Dementia: A Prospective Study.

Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria

Neuroprotective And Anti-Inflammatory Effects Of Linoleic Acid In Models Of Parkinson’s Disease: The Implication Of Lipid Droplets And Lipophagy

Biological Effects Of Olive Oil Phenolic Compounds On Mitochondria

Neuroprotective And Anti-Inflammatory Effects Of Linoleic Acid In Models Of Parkinson’s Disease: The Implication Of Lipid Droplets And Lipophagy

Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)

Neuroprotective And Anti-Inflammatory Effects Of Linoleic Acid In Models Of Parkinson’s Disease: The Implication Of Lipid Droplets And Lipophagy

Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)

Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.

Delay Of Egf-Stimulated Egfr Degradation In Myotonic Dystrophy Type 1 (Dm1)

Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1

Ambra1 Haploinsufficiency In Cd1 Mice Results In Metabolic Alterations And Exacerbates Age-Associated Retinal Degeneration.

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1

Links Between Paraquat And Parkinson’s Disease

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (4th Edition)1

Links Between Paraquat And Parkinson’s Disease

Procedimientos, Experimentación E Investigación En Enfermedades Neurodegenerativas: Protocolos Para Un Laboratorio De Neurociencias

Links Between Paraquat And Parkinson’s Disease

Procedimientos, Experimentación E Investigación En Enfermedades Neurodegenerativas: Protocolos Para Un Laboratorio De Neurociencias

Cultivos Celulares

Procedimientos, Experimentación E Investigación En Enfermedades Neurodegenerativas: Protocolos Para Un Laboratorio De Neurociencias

Cultivos Celulares

Análisis De Extracto Proteico Por Western Blotting

Cultivos Celulares

Análisis De Extracto Proteico Por Western Blotting

Extracción De Proteínas

Análisis De Extracto Proteico Por Western Blotting

Extracción De Proteínas

Técnica De Sobreexpresión Génica

Extracción De Proteínas

Técnica De Sobreexpresión Génica

Técnica De Silenciamiento Génico

Técnica De Sobreexpresión Génica

Técnica De Silenciamiento Génico

The Dual Role Of Necrostatin-1 In Parkinson's Disease Models

Técnica De Silenciamiento Génico

The Dual Role Of Necrostatin-1 In Parkinson's Disease Models

Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction.

The Dual Role Of Necrostatin-1 In Parkinson's Disease Models

Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction.

Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease

Neuroprotective Properties Of Queen Bee Acid By Autophagy Induction.

Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease

Toxicity Of Necrostatin-1 In Parkinson’s Disease Models

Metabolic Alterations In Plasma From Patients With Familial And Idiopathic Parkinson's Disease

Toxicity Of Necrostatin-1 In Parkinson’s Disease Models

Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells

Toxicity Of Necrostatin-1 In Parkinson’s Disease Models

Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells

Natural Products In The Promotion Of Healthspan And Longevity.

Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells

Natural Products In The Promotion Of Healthspan And Longevity.

Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack

Natural Products In The Promotion Of Healthspan And Longevity.

Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack

The Paradigm Of Protein Acetylation In Parkinson's Disease

Mitophagy In Human Astrocytes Treated With The Antiretroviral Drug Efavirenz: Lack Of Evidence Or Evidence Of The Lack

The Paradigm Of Protein Acetylation In Parkinson's Disease

Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients

The Paradigm Of Protein Acetylation In Parkinson's Disease

Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients

Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson's Disease Patients.

Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson’s Disease Patients

Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson's Disease Patients.

Cholesterol And Multilamellar Bodies: Lysosomal Dysfunction In Gba-Parkinson Disease

Impaired Mitophagy And Protein Acetylation Levels In Fibroblasts From Parkinson's Disease Patients.

Cholesterol And Multilamellar Bodies: Lysosomal Dysfunction In Gba-Parkinson Disease

Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria

Cholesterol And Multilamellar Bodies: Lysosomal Dysfunction In Gba-Parkinson Disease

Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria

Vascular Risk Factors And Lesions Of Vascular Nature In Magnetic Resonance As Predictors Of Progression To Dementia In Patients With Mild Cognitive Impairment

Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria

Vascular Risk Factors And Lesions Of Vascular Nature In Magnetic Resonance As Predictors Of Progression To Dementia In Patients With Mild Cognitive Impairment

Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells: In Vitro And In Vivo Studies

Vascular Risk Factors And Lesions Of Vascular Nature In Magnetic Resonance As Predictors Of Progression To Dementia In Patients With Mild Cognitive Impairment

Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells: In Vitro And In Vivo Studies

Acetylome In Human Fibroblasts From Parkinson’s Disease Patients

Molecular Characterization Of Autophagic And Apoptotic Signaling Induced By Sorafenib In Liver Cancer Cells: In Vitro And In Vivo Studies

Acetylome In Human Fibroblasts From Parkinson’s Disease Patients

Er-Mitochondria Signaling In Parkinson's Disease Review-Article

Acetylome In Human Fibroblasts From Parkinson’s Disease Patients

Er-Mitochondria Signaling In Parkinson's Disease Review-Article

[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].

Er-Mitochondria Signaling In Parkinson's Disease Review-Article

[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].

N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease

[Detection And Classification Of Chronic Kidney Disease In Primary Care And Importance Of Albuminuria].

N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease

Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease

N370s-Gba1 Mutation Causes Lysosomal Cholesterol Accumulation In Parkinson's Disease

Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease

Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells

Mitochondria-Associated Membranes (Mams): Overview And Its Role In Parkinson’s Disease

Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells

Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy

Turnover Of Lipidated Lc3 And Autophagic Cargoes In Mammalian Cells

Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy

Trend Of Lipid Profile In General Population From Caceres Health Area

Fluorescent Fyve Chimeras To Quantify Ptdins3p Synthesis During Autophagy

Trend Of Lipid Profile In General Population From Caceres Health Area

The Basics Of Autophagy

Trend Of Lipid Profile In General Population From Caceres Health Area

The Basics Of Autophagy

Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?

The Basics Of Autophagy

Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?

Pink1 Deficiency Enhances Autophagy And Mitophagy Induction

Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?

Pink1 Deficiency Enhances Autophagy And Mitophagy Induction

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)

Pink1 Deficiency Enhances Autophagy And Mitophagy Induction

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)

Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition)

Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?

Erratum To: Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy (3rd Edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?

Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines

Pompe Disease And Autophagy: Partners In Crime, Or Cause And Consequence?

Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines

Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis

Mrna And Protein Dataset Of Autophagy Markers (Lc3 And P62) In Several Cell Lines

Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis

Mitochondria: Key Organelle In Parkinson's Disease

Ifdotmeter: A New Software Application For Automated Immunofluorescence Analysis

Mitochondria: Key Organelle In Parkinson's Disease

G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway

Mitochondria: Key Organelle In Parkinson's Disease

G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway

Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment

G2019s Mutation Of Lrrk2 Increases Autophagy Via Mek/erk Pathway

Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment

Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?

Association Between Subclinical Carotid Atherosclerosis, Hyperhomocysteinaemia And Mild Cognitive Impairment

Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?

Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations

Is The Modulation Of Autophagy The Future In The Treatment Of Neurodegenerative Diseases?

Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations

Control Of Autophagy In Parkinson’s Disease

Routine Western Blot To Check Autophagic Flux: Cautions And Recommendations

Control Of Autophagy In Parkinson’s Disease

Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach

Control Of Autophagy In Parkinson’s Disease

Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach

Toxicity And Autophagy In Neurodegenerative Disorders

Association Of Vascular Factors And Amnestic Mild Cognitive Impairment: A Comprehensive Approach

Toxicity And Autophagy In Neurodegenerative Disorders

Origen Molecular De La Compleja Enfermedad De Parkinson

Toxicity And Autophagy In Neurodegenerative Disorders

Origen Molecular De La Compleja Enfermedad De Parkinson

Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression

Origen Molecular De La Compleja Enfermedad De Parkinson

Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression

G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy

Mitochondrial Impairment Increases Fl-Pink1 Levels By Calcium-Dependent Gene Expression

G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy

Links Between Paraquat And Parkinson's Disease

G2019s Lrrk2 Mutant Fibroblasts From Parkinson's Disease Patients Show Increased Sensitivity To Neurotoxin 1-Methyl-4-Phenylpyridinium Dependent Of Autophagy

Links Between Paraquat And Parkinson's Disease

Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease

Links Between Paraquat And Parkinson's Disease

Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease

Implication Of Autophagy In Parkinson's Disease

Novel Insights Into The Neurobiology Underlying Lrrk2-Linked Parkinson's Disease

Implication Of Autophagy In Parkinson's Disease

Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model

Implication Of Autophagy In Parkinson's Disease

Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model

The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway

Autophagy, Mitochondria And 3-Nitropropionic Acid Joined In The Same Model

The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway

Paraquat, Between Apoptosis And Autophagy

The Lrrk2 G2019s Mutant Exacerbates Basal Autophagy Through Activation Of The Mek/erk Pathway

Paraquat, Between Apoptosis And Autophagy

Autophagy And Parkinson's Disease: A Love-Hate Relationship

Paraquat, Between Apoptosis And Autophagy

Autophagy And Parkinson's Disease: A Love-Hate Relationship

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy

Autophagy And Parkinson's Disease: A Love-Hate Relationship

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy

Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy

Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies

Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease

Parkinsons Disease: Leucine-Rich Repeat Kinase 2 And Autophagy, Intimate Enemies

Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy

Possible Involvement Of The Relationship Of Lrrk2 And Autophagy In Parkinson's Disease

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy

The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy

The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts

Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress

The Mapk1/3 Pathway Is Essential For The Deregulation Of Autophagy Observed In G2019s Lrrk2 Mutant Fibroblasts

Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress

Inhibition Of Autophagy By Tab2 And Tab3

Ask1 Overexpression Accelerates Paraquat-Induced Autophagy Via Endoplasmic Reticulum Stress

Inhibition Of Autophagy By Tab2 And Tab3

Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y

Inhibition Of Autophagy By Tab2 And Tab3

Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y

Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat

Fipronil Is A Powerful Uncoupler Of Oxidative Phosphorylation That Triggers Apoptosis In Human Neuronal Cell Line Shsy5y

Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat

Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain

Protective Effect Of The Glial Cell Line-Derived Neurotrophic Factor (Gdnf) On Human Mesencephalic Neuron-Derived Cells Against Neurotoxicity Induced By Paraquat

Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain

The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells

Genetic Screening For The Lrrk2, G2019s And R1441 Codon Mutations In Parkinson's Disease Patients From Extremadura, Spain

The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells

Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis

The Neuroprotective Effect Of Talipexole From Paraquat-Induced Cell Death In Dopaminergic Neuronal Cells

Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis

Dj-1 As A Modulator Of Autophagy: An Hypothesis

Activation Of Apoptosis Signal-Regulating Kinase 1 Is A Key Factor In Paraquat-Induced Cell Death: Modulation By The Nrf2/trx Axis

Dj-1 As A Modulator Of Autophagy: An Hypothesis

Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells

Dj-1 As A Modulator Of Autophagy: An Hypothesis

Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells

Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells

Effect Of Paraquat Exposure On Nitric Oxide-Responsive Genes In Rat Mesencephalic Cells

Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells

Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade

Curcumin Exposure Induces Expression Of The Parkinson's Disease-Associated Leucine-Rich Repeat Kinase 2 (Lrrk2) In Rat Mesencephalic Cells

Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade

Nitric Oxide In Paraquat-Mediated Toxicity: A Review

Paraquat Exposure Induces Nuclear Translocation Of Glyceraldehyde-3-Phosphate Dehydrogenase (Gapdh) And The Activation Of The Nitric Oxide-Gapdh-Siah Cell Death Cascade

Nitric Oxide In Paraquat-Mediated Toxicity: A Review

Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species

Nitric Oxide In Paraquat-Mediated Toxicity: A Review

Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species

Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole

Curcumin Enhances Paraquat-Induced Apoptosis Of N27 Mesencephalic Cells Via The Generation Of Reactive Oxygen Species

Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole

Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy

Nitric Oxide-Mediated Toxicity In Paraquat-Exposed Sh-Sy5y Cells: A Protective Role Of 7-Nitroindazole

Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy

Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity

Silencing Dj-1 Reveals Its Contribution In Paraquat-Induced Autophagy

Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity

Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways

Schistosoma Mansoni Arginase Shares Functional Similarities With Human Orthologs But Depends Upon Disulphide Bridges For Enzymatic Activity

Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways

Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis

Identification Of Genes Associated With Paraquat-Induced Toxicity In Sh-Sy5y Cells By Pcr Array Focused On Apoptotic Pathways

Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes

Age-Related Alteration Of Arginase Activity Impacts On Severity Of Leishmaniasis

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes

Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?

Guidelines For The Use And Interpretation Of Assays For Monitoring Autophagy In Higher Eukaryotes

Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?

Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells

Relationship Between Autophagy And Apoptotic Cell Death In Human Neuroblastoma Cells Treated With Paraquat: Could Autophagy Be A Brake In Paraquat-Induced Apoptotic Death?

Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells

Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy

Inhibition Of Paraquat-Induced Autophagy Accelerates The Apoptotic Cell Death In Neuroblastoma Sh-Sy5y Cells

Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy

Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells

Arginase Activity Mediates Reversible T Cell Hyporesponsiveness In Human Pregnancy

Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells

Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death

Th1/th2 Cytokines: An Easy Model To Study Gene Expression In Immune Cells

Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death

Suppression Of T-Cell Functions By Human Granulocyte Arginase

Low Concentrations Of Paraquat Induces Early Activation Of Extracellular Signal-Regulated Kinase 1/2, Protein Kinase B, And C-Jun N-Terminal Kinase 1/2 Pathways: Role Of C-Jun N-Terminal Kinase In Paraquat-Induced Cell Death

Suppression Of T-Cell Functions By Human Granulocyte Arginase

Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain

Suppression Of T-Cell Functions By Human Granulocyte Arginase

Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain

Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity

Expression In Yeast And Purification Of A Membrane Protein, Serca1a, Using A Biotinylated Acceptor Domain

Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity

Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo

Heat Shock Proteins Protect Both Mpp+ And Paraquat Neurotoxicity

Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo

Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity

Arginase And Polyamine Synthesis Are Key Factors In The Regulation Of Experimental Leishmaniasis In Vivo

Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity

Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells

Arginase I Is Constitutively Expressed In Human Granulocytes And Participates In Fungicidal Activity

Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells

Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants

Paraquat-Induced Apoptotic Cell Death In Cerebellar Granule Cells

Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants

Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells

Protection Against Mpp+ Neurotoxicity In Cerebellar Granule Cells By Antioxidants

Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells

Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells

Mpp+: Mechanism For Its Toxicity In Cerebellar Granule Cells

Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells

Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells

Mpp+ Causes Inhibition Of Cellular Energy Supply In Cerebellar Granule Cells

Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells

Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase

Vitamin E Blocks Early Events Induced By 1-Methyl-4-Phenylpyridinium (Mpp+) In Cerebellar Granule Cells

Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase

Diagnostic Performance Of Arginase Activity In Colorectal Cancer

Involvement Of The Cytoplasmic Loop L6-7 In The Entry Mechanism For Transport Of Ca2+ Through The Sarcoplasmic Reticulum Ca2+-Atpase

Diagnostic Performance Of Arginase Activity In Colorectal Cancer

Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells

Diagnostic Performance Of Arginase Activity In Colorectal Cancer

Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells

Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism

Mechanisms Of Mpp+ Incorporation Into Cerebellar Granule Cells

Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism

Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells

Differential Regulation Of Nitric Oxide Synthase-2 And Arginase-1 By Type 1/type 2 Cytokines In Vivo: Granulomatous Pathology Is Shaped By The Pattern Of L-Arginine Metabolism

Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells

Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase

Lithium Inhibits Caspase 3 Activation And Dephosphorylation Of Pkb And Gsk3 Induced By K+ Deprivation In Cerebellar Granule Cells

Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase

Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.

Implications Of The S-Shaped Domain In The Quaternary Structure Of Human Arginase

Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.

Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+

Clean Western Blots Of Membrane Proteins After Yeast Heterologous Expression Following A Shortened Version Of The Method Of Perini Et Al.

Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+

Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons

Different Mechanisms Of Protection Against Apoptosis By Valproate And Li+

Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons

Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase

Partial Lithium-Associated Protection Against Apoptosis Induced By C2- Ceramide In Cerebellar Granule Neurons

Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase

The Immune System And Autoaggression

Immunological Identity Of The Two Different Molecular Mass Constitutive Subunits Of Liver Arginase

The Immune System And Autoaggression

Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle

The Immune System And Autoaggression

Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle

Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase

Oscillations In Rat Liver Cytosolic Enzyme Activities Of The Urea Cycle

Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase

Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase

Kinetics And Inhibition By Some Aminoacids Of Lactating Rat Mammary Gland Arginase

Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase

Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación

Kinetics Of Manganese Reconstitution And Thiol Group Exposition In Dialyzed Rat Mammary Gland Arginase

Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación

Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages

Importancia Fisiológica De La Arginasa De Glándula Mamaria En Lactación

Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages

Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp

Parallel Induction Of Nitric Oxide And Glucose-6-Phosphate Dehydrogenase In Activated Bone Marrow Derived Macrophages

Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp

An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)

Unfolding And Trypsin Inactivation Studies Reveal A Conformation Drift Of Glucose-6-Phosphate Dehydrogenase Upon Binding Of Nadp

An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)

Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.

An Arginine Regulated Γguanidobutyrate Ureahydrolase From Tench Liver (Tinca Tinca L.)

Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.

Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)

Physico-Chemical Properties Of Hepatocyte Plasma-Membrane-Bound Arginase.

Glucosa Y Glucosa 6-Fosfato Deshidrogenasas En Hígado De Tenca (Tinca Tinca)

Novel Fkbp Prolyl Isomerase 1a (Fkbp12) Ligand Promotes Functional Improvement In Sod1g93a Amyotrophic Lateral Sclerosis (Als) Mice